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Books on the topic 'Encephalopaty'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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1

Food and Agriculture Organization of the United Nations. Regional Office for Latin America and the Caribbean. and Asociación Panamericana de Ciencias Veterinarias., eds. Novedades sobre la encefalopatia espongiforme de los bovinos =: News on the bovine spongiform encephalopaty. Santiago, Chile: Oficina Regional de la FAO para América Latina y el Caribe, 1996.

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2

Mullen, Kevin D., and Ravi K. Prakash, eds. Hepatic Encephalopathy. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-836-8.

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3

McCandless, David W., ed. Metabolic Encephalopathy. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-79112-8.

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4

Butterworth, Roger F., and Gilles Pomier Layrargues, eds. Hepatic Encephalopathy. Totowa, NJ: Humana Press, 1989. http://dx.doi.org/10.1007/978-1-4612-4506-3.

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5

1941-, McCandless David W., ed. Metabolic encephalopathy. New York: Springer, 2009.

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6

Hepatic encephalopathy. Boston: Butterworth-Heinemann, 1992.

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7

Gibbs, Clarence J., ed. Bovine Spongiform Encephalopathy. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-2406-8.

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8

Swanson, Janice C. Bovine spongiform encephalopathy. Beltsville, Md: National Agricultural Library, 1990.

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9

Pratt, Kendra. Bovine spongiform encephalopathy. Riverdale, MD: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, Veterinary Services, 1995.

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10

United States. Animal and Plant Health Inspection Service. Veterinary Services. Bovine spongiform encephalopathy. Hyattsville, Md.?]: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, Veterinary Services, 1994.

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11

Golden, Charles J., and Matthew R. Zusman. Chronic Traumatic Encephalopathy (CTE). Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23288-7.

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12

Scientific, Enfer. Bovine spongiform encephalopathy test. Cashel, Co. Tipperary: Enfer Scientific, 1997.

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13

F, Butterworth Roger, and Pomier Layrargues Gilles, eds. Hepatic encephalopathy: Pathophysiology and treatment. Clifton, N.J: Humana Press, 1989.

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14

Grisolía, Santiago, and Vicente Felipo, eds. Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2484-7.

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15

Häussinger, D., G. Kircheis, and F. Schliess, eds. Hepatic Encephalopathy and Nitrogen Metabolism. Dordrecht: Springer Netherlands, 2006. http://dx.doi.org/10.1007/1-4020-4456-9.

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16

1923-, Grisolía Santiago, and Felipo Vicente, eds. Cirrhosis, hyperammonemia, and hepatic encephalopathy. New York: Plenum Press, 1993.

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17

United States. Animal and Plant Health Inspection Service, ed. Bovine spongiform encephalopathy: An overview. 2nd ed. [Washington, D.C.]: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, 2006.

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18

1951-, Häussinger D., Kircheis G, and Schliess F, eds. Hepatic encephalopathy and nitrogen metabolism. Berlin: Springer, 2006.

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19

Becker, Geoffrey S. Mad cow disease (Bovine spongiform encephalopathy). Edited by Copeland Curtis W and Lister Sarah A. New York: Nova Science Publishers, 2008.

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20

Nieuwenhuizen, Onno van. Cerebral visual disturbance in infantile encephalopathy. Dordrecht: Nijhoff for Bartiméus Foundation, Doorn, Netherlands, 1987.

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21

Felipo, Vicente, and Santiago Grisolia, eds. Hepatic Encephalopathy, Hyperammonemia, and Ammonia Toxicity. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-1989-8.

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22

Bajaj, Jasmohan S., ed. Diagnosis and Management of Hepatic Encephalopathy. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-76798-7.

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23

van Nieuwenhuizen, Onno. Cerebral Visual Disturbance in Infantile Encephalopathy. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3305-7.

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24

McCandless, David W., ed. Cerebral Energy Metabolism and Metabolic Encephalopathy. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1209-3.

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25

Grisolía, Santiago, Vicente Felipo, and María-Dolores Miñana, eds. Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5826-8.

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26

1941-, McCandless David W., ed. Cerebral energy metabolism and metabolic encephalopathy. New York: Plenum Press, 1985.

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27

Vicente, Felipo, and Grisolía Santiago 1923-, eds. Hepatic encephalopathy, hyperammonemia, and ammonia toxicity. New York: Plenum Press, 1994.

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28

Ridgway, Tom. Mad cow disease: Bovine spongiform encephalopathy. New York, NY: Rosen Pub. Group, 2002.

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29

1924-, Gibbs Clarence J., ed. Bovine spongiform encephalopathy: The BSE dilemma. New York: Springer, 1996.

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30

United States. Animal and Plant Health Inspection Service. Emergency programs alert: Bovine spongiform encephalopathy. Washington, D.C.]: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, 1992.

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31

International Symposium on Cirrhosis, Hepatic Encephalopathy, and Ammonium Toxicity, Molecular Basis and Clinical Aspects (1989 Valencia, Spain). Cirrhosis, hepatic encephalopathy, and ammonium toxicity. New York: Plenum Press, 1990.

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32

Jones, E. Anthony, Alfred J. Meijer, and Robert A. F. M. Chamuleau, eds. Encephalopathy and Nitrogen Metabolism in Liver Failure. Dordrecht: Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-010-0159-5.

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33

Felipo, Vicente, and Santiago Grisolía, eds. Advances in Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5945-0.

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34

United States. Food Safety and Inspection Service. Bovine spongiform encephalopathy (BSE) response plan summary. Washington, D.C.?]: U.S. Dept. of Agriculture, Animal and Plant Health Inspection Service, Food Safety and Inspection Service, 1998.

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35

Great Britain. Department of Health. Chronic fatigue syndrome/myalgic encephalopathy (CFS/ME). London: Department of Health, 2004.

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36

Felipo, Vicente. Advances in Cirrhosis, Hyperammonemia, and Hepatic Encephalopathy. Boston, MA: Springer US, 1997.

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37

Novedades sobre la encefalopatia espongiforme de los bovinos y otras temas de interés =: News on the bovine spongiform encephalopaty and other interesting issues. Santiago, Chile: Oficina Regional de la FAO para América Latina y el Caribe, 1996.

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38

Hepatic Encephalopathy. Humana Press, 2012.

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39

Alvis, Bret, and Amy Robertson. Hepatic Encephalopathy. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0075.

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Hepatic encephalopathy is a complication of both acute and chronic liver failure. The disease can range from mild cognitive deficits to deep coma. Ammonia accumulation and inflammation are the two most accepted causes of hepatic encephalopathy. It is important to confirm an elevated serum ammonia level and rule out alternative causes of neurological derangements. Nonabsorbable disaccharides are the mainstay of treatment; however, the only definitive treatment is liver transplantation. Pertinent anesthetic considerations include avoiding benzodiazepines, understanding the implications of diminished hepatic function, and recognizing factors that may contribute to increased intracranial cerebral pressure. Proper assessment and management of the patient presenting with hepatic encephalopathy will be discussed.
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40

Miller, Aaron E., and Teresa M. DeAngelis. Hashimoto’s Encephalopathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0018.

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Hashimoto’s encephalopathy (HE) is a rare but potentially corticosteroid responsive disorder, which should be considered in cases of encephalopathy of unclear etiology. Hashimoto’s thyroiditis usually does not accompany HE and thyroid function is generally clinically normal. In this chapter, we review the differential diagnostic suspicions for a case of encephalopathy of unclear etiology and respective aspects that raise concern for HE. In addition, we discuss the utility of diagnostic testing and options for therapeutic management.
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41

Mullen, Kevin D., and Ravi K. Prakash. Hepatic Encephalopathy. Humana Press, 2012.

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42

Ridola, Lorenzo, and Oliviero Riggio, eds. Hepatic Encephalopathy. MDPI, 2023. http://dx.doi.org/10.3390/books978-3-0365-6339-8.

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43

McCandless, David W. Metabolic Encephalopathy. Springer, 2011.

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44

Spongiform Encephalopathy. Stationery Office Books, 1992.

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45

Mullen, Kevin D., and Ravi K. Prakash. Hepatic Encephalopathy. Humana, 2014.

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46

Sawant, Prabha. Hepatic Encephalopathy. Elsevier - Health Sciences Division, 2009.

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47

Mullen, Kevin D., and Ravi K. Prakash. Hepatic Encephalopathy. Humana Press, 2012.

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48

Misulis, Karl E., and E. Lee Murray. Hypoxic-Ischemic Encephalopathy. Edited by Karl E. Misulis and E. Lee Murray. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190259419.003.0014.

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Hypoxic-ischemic encephalopathy (HIE) is one of the most common reasons for hospital neurology consultation. The reasons for neurologic consultations are usually medical management, evaluation and management of myoclonus and seizure activity, and prognosis.
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49

Magalhaes, Eric, Angelo Polito, Andréa Polito, and Tarek Sharshar. Sepsis-Associated Encephalopathy. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0032.

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Brain dysfunction is a major complication of sepsis and is characterized by alteration of consciousness, ranging from delirium to coma and marked electroencephalographic changes. It reflects a constellation of dynamic biological mechanisms, including neurotransmitter imbalance, macro- and microcirculatory dysfunction resulting in ischaemia, endothelial activation, alteration of the blood-brain barrier impairment with passage of neurotoxic mediators, activation of microglial cells within the central nervous system, cumulatively resulting in a neuroinflammatory state. Sepsis-associated brain dysfunction is associated with increased mortality and long-term cognitive decline, whose mechanisms might include microglial activation, axonopathy, or cerebral microinfarction. There is no specific treatment, other than the management of the underlying septic source, correction of physiological and metabolic abnormalities, and limiting the use of medications with neurotoxic effects.
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50

Lister, Sarah A. Bovine Spongiform Encephalopathy. Nova Science Publishers, Incorporated, 2009.

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