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Relevant bibliographies by topics / Emotion Upregulation

Academic literature on the topic 'Emotion Upregulation'

Author: Grafiati

Published: 10 December 2022

Last updated: 29 January 2023

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Journal articles on the topic "Emotion Upregulation"

1

Salimzadeh, Raheleh, Nathan C. Hall, and Alenoush Saroyan. "Stress, Emotion Regulation, and Well-Being among Canadian Faculty Members in Research-Intensive Universities." Social Sciences 9, no. 12 (December 10, 2020): 227. http://dx.doi.org/10.3390/socsci9120227.

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Existing research reveals the academic profession to be stressful and emotion-laden. Recent evidence further shows job-related stress and emotion regulation to impact faculty well-being and productivity. The present study recruited 414 Canadian faculty members from 13 English-speaking research-intensive universities. We examined the associations between perceived stressors, emotion regulation strategies, including reappraisal, suppression, adaptive upregulation of positive emotions, maladaptive downregulation of positive emotions, as well as adaptive and maladaptive downregulation of negative emotions, and well-being outcomes (emotional exhaustion, job satisfaction, quitting intentions, psychological maladjustment, and illness symptoms). Additionally, the study explored the moderating role of stress, gender, and years of experience in the link between emotion regulation and well-being as well as the interactions between adaptive and maladaptive emotion regulation strategies in predicting well-being. The results revealed that cognitive reappraisal was a health-beneficial strategy, whereas suppression and maladaptive strategies for downregulating positive and negative emotions were detrimental. Strategies previously defined as adaptive for downregulating negative emotions and upregulating positive emotions did not significantly predict well-being. In contrast, strategies for downregulating negative emotions previously defined as dysfunctional showed the strongest maladaptive associations with ill health. Practical implications and directions for future research are also discussed.

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2

Schelhorn, Iris, Swantje Schlüter, Kerstin Paintner, Youssef Shiban, Ricardo Lugo, Marie Meyer, and Stefan Sütterlin. "Emotions and emotion up-regulation during the COVID-19 pandemic in Germany." PLOS ONE 17, no. 1 (January 7, 2022): e0262283. http://dx.doi.org/10.1371/journal.pone.0262283.

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In stressful situations such as the COVID-19-pandemic, unpleasant emotions are expected to increase while pleasant emotions will likely decrease. Little is known about the role cognitive appraisals, information management, and upregulating pleasant emotions can play to support emotion regulation in a pandemic. In an online survey (N = 1682), we investigated predictors of changes in pleasant and unpleasant emotions in a German sample (aged 18–88 years) shortly after the first restrictions were imposed. Crisis self-efficacy and felt restriction were predictors of changes in unpleasant emotions and joy alike. The application of emotion up-regulation strategies was weakly associated with changes in joy. Among the different upregulation strategies, only “savouring the moment” predicted changes in joy. Our study informs future research perspectives assessing the role of upregulating pleasant emotions under challenging circumstances.

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3

Min, Jungwon, Kaoru Nashiro, Hyun Joo Yoo, Christine Cho, Padideh Nasseri, Shelby L. Bachman, Shai Porat, et al. "Emotion Downregulation Targets Interoceptive Brain Regions While Emotion Upregulation Targets Other Affective Brain Regions." Journal of Neuroscience 42, no. 14 (February 22, 2022): 2973–85. http://dx.doi.org/10.1523/jneurosci.1865-21.2022.

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4

Brom, Mirte, Ellen Laan, Walter Everaerd, Philip Spinhoven, Baptist Trimbos, and Stephanie Both. "The Influence of Emotion Upregulation on the Expectation of Sexual Reward." Journal of Sexual Medicine 13, no. 1 (January 2016): 105–19. http://dx.doi.org/10.1016/j.jsxm.2015.11.003.

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5

Pavlov, Sergei V., Natalia V. Reva, Konstantin V. Loktev, Alexei V. Tumyalis, Vladimir V. Korenyok, and Lyubomir I. Aftanas. "The temporal dynamics of cognitive reappraisal: Cardiovascular consequences of downregulation of negative emotion and upregulation of positive emotion." Psychophysiology 51, no. 2 (October 17, 2013): 178–86. http://dx.doi.org/10.1111/psyp.12159.

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6

Johnston, Stephen, D. E. J. Linden, D. Healy, R. Goebel, I. Habes, and S. G. Boehm. "Upregulation of emotion areas through neurofeedback with a focus on positive mood." Cognitive, Affective, & Behavioral Neuroscience 11, no. 1 (November 25, 2010): 44–51. http://dx.doi.org/10.3758/s13415-010-0010-1.

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7

Walsh, Erin C., Tory A. Eisenlohr-Moul, Jared Minkel, Joshua Bizzell, Chris Petty, Andrew Crowther, Hannah Carl, Moria J. Smoski, and Gabriel S. Dichter. "Pretreatment brain connectivity during positive emotion upregulation predicts decreased anhedonia following behavioral activation therapy for depression." Journal of Affective Disorders 243 (January 2019): 188–92. http://dx.doi.org/10.1016/j.jad.2018.09.065.

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8

Scheggi, Simona, Graziano Pinna, Giulia Braccagni, Maria De Montis, and Carla Gambarana. "PPARα Signaling: A Candidate Target in Psychiatric Disorder Management." Biomolecules 12, no. 5 (May 20, 2022): 723. http://dx.doi.org/10.3390/biom12050723.

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Peroxisome proliferator-activator receptors (PPARs) regulate lipid and glucose metabolism, control inflammatory processes, and modulate several brain functions. Three PPAR isoforms have been identified, PPARα, PPARb/d, and PPARg, which are expressed in different tissues and cell types. Hereinafter, we focus on PPARα involvement in the pathophysiology of neuropsychiatric and neurodegenerative disorders, which is underscored by PPARα localization in neuronal circuits involved in emotion modulation and stress response, and its role in neurodevelopment and neuroinflammation. A multiplicity of downstream pathways modulated by PPARα activation, including glutamatergic neurotransmission, upregulation of brain-derived neurotrophic factor, and neurosteroidogenic effects, encompass mechanisms underlying behavioral regulation. Modulation of dopamine neuronal firing in the ventral tegmental area likely contributes to PPAR effects in depression, anhedonia, and autism spectrum disorder (ASD). Based on robust preclinical evidence and the initial results of clinical studies, future clinical trials should assess the efficacy of PPARα agonists in the treatment of mood and neurodevelopmental disorders, such as depression, schizophrenia, and ASD.

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9

Xu, Zitong, JunFan Fang, Xuaner Xiang, HaiJu Sun, SiSi Wang, Jianqiao Fang, and Junying Du. "Electroacupuncture Alleviates Pain-Related Emotion by Upregulating the Expression of NPS and Its Receptor NPSR in the Anterior Cingulate Cortex and Hypothalamus." Evidence-Based Complementary and Alternative Medicine 2020 (February 10, 2020): 1–16. http://dx.doi.org/10.1155/2020/8630368.

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Objective. Electroacupuncture (EA) is reported effective in alleviating pain-related emotion; however, the underlying mechanism of its effects still needs to be elucidated. The NPS-NPSR system has been validated for the involvement in the modulation of analgesia and emotional behavior. Here, we aimed to investigate the role of the NPS-NPSR system in the anterior cingulate cortex (ACC), hypothalamus, and central amygdala (CeA) in the use of EA to relieve affective pain modeled by complete Freund’s adjuvant- (CFA-) evoked conditioned place aversion (C-CPA). Materials and Methods. CFA injection combined with a CPA paradigm was introduced to establish the C-CPA model, and the elevated O-maze (EOM) was used to test the behavioral changes after model establishment. We further explored the expression of NPS and NPSR at the protein and gene levels in the brain regions of interest by immunofluorescence staining and quantitative real-time PCR. Results. We observed that EA stimulation delivered to the bilateral Zusanli (ST36) and Kunlun (BL60) acupoints remarkably inhibited sensory pain, pain-evoked place aversion, and anxiety-like behavior. The current study showed that EA significantly enhanced the protein expression of this peptide system in the ACC and hypothalamus, while the elevated expression of NPSR protein alone was just confined to the affected side in the CeA. Moreover, EA remarkably upregulated the mRNA expression of NPS in CeA, ACC, and hypothalamus and NPSR mRNA in the hypothalamus and CeA. Conclusions. These data suggest the effectiveness of EA in alleviating affective pain, and these benefits may at least partially be attributable to the upregulation of the NPS-NPSR system in the ACC and hypothalamus.

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10

Linden, D., and T. Lancaster. "Functional Magnetic Resonance Imaging (FMRI)-based neurofeedback as a new treatment tool for depression." European Psychiatry 26, S2 (March 2011): 937. http://dx.doi.org/10.1016/s0924-9338(11)72642-6.

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We investigated whether depressed patients who received fMRI-based neurofeedback are able to upregulate the activity in brain areas devoted to positive emotion processing and thereby establish improvements in mood state. Eight medicated patients with major depression participated in four separate fMRI sessions, each of which consisted of an emotion localiser and three neurofeedback runs. Target areas were selected individually with a functional localiser that identified the region most responsive to positive affective images. The target areas were in uni- or bilateral prefrontal cortex, insula or amygdala. During neurofeedback runs, patients received real-time feedback about activation levels in the target area. Each patient learnt to increase target area activity over successive sessions. Depression scores on the 17-item Hamilton Depression Rating Scale improved significantly. No such improvement was seen in a non-neurofeedback control group (N = 8) that was matched for symptom severity, demographics and medication and used the same cognitive/affective strategies that were employed successfully by the neurofeedback group, but outside the scanner. This group difference in treatment effects was supported by a significant interaction between the factors time (pre/post-intervention) and group (neurofeedback/controls) on the repeated measures ANOVA (F(1,14) = 10.15, p = .007). The neurofeedback group showed increasing activity in the ventral striatum and regions involved in cognitive control as training progressed. Upregulation of brain areas responsive to positive affective cues through fMRI-neurofeedback is thus a promising tool in the treatment of depression. The novelty of the present approach consists in the combination of biological and cognitive factors in the same intervention.

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