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Books on the topic 'Emotion regulation mechanisms'
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1
Krivoyekov, Syergyey, and Roman Ayzman. Psychophysiology. ru: INFRA-M Academic Publishing LLC., 2015. http://dx.doi.org/10.12737/10884.
Full textAbstract:
Psychophysiology — the science studying interrelation of mentality of the person and physiological processes. Fundamental knowledge of work of a brain, first of all, of nervous regulation of functions of an organism, the general and specific features of the highest falls within the scope of its interests
nervous activity, the defining character and behavior of the person,
psychophysiological mechanisms of regulation of functional states.
In the book neurophysiological bases of coding and information processing in nervous system, neural mechanisms of feelings, perceptions, memories, training, motivations and emotions, thinking and the speech, attention, consciousness, behavior, mental activity are stated. Separate
the section is devoted to physiological bases of mental changes at various functional, extreme and pathophysiological states (a stress, post-stressful frustration, addiktivny states, depressions, etc.) and to ways of their correction. Authors tried to pay special attention to disclosure of specifics of psychophysiology of the person,
to difference of physiological mechanisms of regulation of mental functions of the person in comparison with model researches on animals.
For simplification of work on discipline and the best digestion of material
the textbook is supplied with the glossary.
For students, undergraduates, graduate students and teachers of psychological and medical faculties of higher education institutions.
2
Todd, Rebecca Mary Ruth. Cortical mechanisms of emotion regulation in young children responding to angry, neutral, and happy faces. 2005.
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3
Todd, Rebecca Mary Ruth. Cortical mechanisms of emotion regulation in young children responding to angry, neutral, and happy faces. 2005.
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4
Todd, Rebecca Mary Ruth. Cortical mechanisms of emotion regulation in young children responding to angry, neutral, and happy faces. 2005.
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5
Beauchaine, Theodore P., Aimee R. Zisner, and Elizabeth P. Hayden. Neurobiological Mechanisms of Psychopathology and Treatment Action. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.54.
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In recent years, it has become increasingly clear that common forms of psychopathology derive from complex interactions among neurobiological vulnerabilities and environmental adversities. These interactions can alter neurobehavioral development to yield progressively intractable forms of psychopathology across childhood and adolescence. This chapter focuses on neurobiological mechanisms of trait impulsivity, trait anxiety, stress reactivity, and emotion regulation/executive function. How these traits confer vulnerability to externalizing disorders, internalizing disorders, heterotypic comorbidity, and heterotypic continuity is described. Next, neurobiological mechanisms of treatment response are considered. Trait impulsivity and trait anxiety are highly heritable and derive initially from subcortical structures that mature early in life. In contrast, emotion regulation and executive function, which modulate trait impulsivity and trait anxiety, are more sensitive to environmental influence and derive from cortical structures that mature into young adulthood. Neurobiological mechanisms of psychosocial treatment response are represented largely in the cortex and its neuromodulatory connections with the subcortex.
6
Snyder, James. Coercive Family Processes and the Development of Child Social Behavior and Self-Regulation. Edited by Thomas J. Dishion and James Snyder. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199324552.013.10.
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This chapter (1) examines the multiple ways in which coercive processes may be manifested during family interaction in addition to their more blatant, aversive forms, including emotion dismissing, invalidating, intrusive/controlling social actions; (2) assesses the role of higher cognitive processing and control in coercive social interaction in the context of previous assumptions that coercive processes are primarily overlearned and automatic; (3) examines the utility of extensions of environmental main effects models of coercive processes by explicitly focusing on synergistic models that involve child temperamental self-regulatory capacities (reflecting underlying molecular genetic and neurobiological mechanisms); and (4) assesses the role of coercive family processes in relation to borderline features and trauma/PTSD.
7
Miu, Andrei C., Judith R. Homberg, and Klaus-Peter Lesch, eds. Genes, brain, and emotions. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198793014.001.0001.
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With the advent of methods from behavioral genetics, molecular biology, and cognitive neuroscience, affective science has recently started to approach genetic influences on emotion, and the underlying intermediate neural mechanisms through which genes and experience shape emotion. The aim of this volume is to offer a comprehensive account of current research in the genetics of emotion, written by leading researchers, with extensive sections focused on methods, intermediate phenotypes, and clinical and translational work. Major methodological approaches are reviewed in the first section, including the two traditional “workhorses” in the field, twin studies and gene–environment interaction studies, and the more recently developed epigenetic modification assays, genome-wide association studies, and optogenetic methods. Parts 2 and 3 focus on a variety of psychological (e.g. fear conditioning, emotional action control, emotion regulation, emotional memory, decision-making) and biological (e.g. neural activity assessed using functional neuroimaging, electroencephalography, and psychophysiological methods; telomere length) mechanisms, respectively, that may be viewed as intermediate phenotypes in the pathways between genes and emotional experience. Part 4 concentrates on the genetics of emotional dysregulation in neuropsychiatric disorders (e.g. post-traumatic stress disorder, eating disorders, obsessive–compulsive disorder, Tourette’s syndrome), including factors contributing to the risk and persistence of these disorders (e.g. child maltreatment, personality, emotional resilience, impulsivity). In addition, two chapters in Part 4 review genetic influences on the response to psychotherapy (i.e. therapygenetics) and pharmacological interventions (i.e. pharmacogenetics) in anxiety and affective disorders.
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Dishion, Thomas J., and James Snyder. Coercion Dynamics. Edited by Thomas J. Dishion and James Snyder. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199324552.013.29.
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This chapter summarizes the history of research focused on coercive relationship dynamics among family members and peers. It is plausible that evolutionary mechanisms are at play that account for the cross-generational repetition of conflict and coercion and the alarming transformations in human behavior that lead to more serious forms of violence. Considerable progress has been made in understanding the genetic and neurobiological underpinnings of emotion-regulation patterns that define vulnerability to coercive relationships. Coercive relationship dynamics can be subtle and laden with many emotions, but ultimately, the core dynamic is that conflict is solved by emotional manipulation rather than by negotiation. More nuanced forms of coercion are also implicated in some patterns of depression, anxiety, and suicide ideation and attempts. There are several evidence-based prevention and treatment strategies for youth problem behavior and marital relationships. Successful prevention and intervention must skillfully motivate and manage changes in these overlearned patterns of behavior.
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Carrión, Victor G., John A. Turner, and Carl F. Weems. Comorbidity in Pediatric PTSD. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190201968.003.0007.
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The current chapter focuses on the idea that the constructs reviewed in the previous chapters such as executive function, emotion regulation, and memory processing abnormalities are not unique to PTSD. Many of the neurobiological correlates of PTSD are also found in depression, anxiety, and substance use disorders. The research reviewed in the current chapter suggests that the constructs of executive function, emotion regulation, and memory processing abnormalities may underlie these conditions and define core mechanisms across mental health disorders. Future directions, such as a shift in the field to incorporate neuroscience into personalized treatment for PTSD, as well as current imperatives, such as the need to assess for trauma histories in differential diagnosis, are discussed.
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Vögele, Claus, Annika P. C. Lutz, and E. Leigh Gibson. Mood, Emotions, and Eating Disorders. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.8.
Full textAbstract:
Mood and emotions are intrinsically involved with eating. This chapter discusses basic mechanisms, findings, and models that help our understanding of the interactions between eating and emotions, in both clinical and nonclinical populations. The finding that negative affect predicts EDs transdiagnostically, and that comorbidity with depressive disorders and anxiety disorders is the norm among patients with EDs suggests that EDs may not necessarily be restricted to domains of eating behavior and body image but may also be associated with significant difficulties in affective functioning. This chapter reviews the evidence relating to the notion that EDs are disturbances of mood regulation, in which regulatory strategies specifically related to eating and the body are used to diminish negative affect associated with food, body image, or stress.
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Magcamit, Michael. Ethnoreligious Otherings and Passionate Conflicts. Oxford University PressOxford, 2022. http://dx.doi.org/10.1093/oso/9780192847751.001.0001.
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Abstract Departing from the mainstream practice and conventional wisdom of materialist and rationalist accounts of internal and intrastate conflicts, the book demonstrates how and why emotions, symbolic predispositions, and perceptions are just as powerful and useful in understanding and explaining these phenomena. By uncovering the invisible albeit concrete emotive, symbolic, and perceptual causal mechanisms underpinning ethnoreligious otherings and the resulting violent protracted conflicts, the book aims to help address the incongruence between how the actual actors operating within these contexts think and act and the existing theories and models of how they are expected to behave. Accordingly, the book has three main goals. First, to highlight the centrality of emotions, symbolic predispositions, and perceptions in providing a more holistic and realistic understanding of otherings and conflicts. Second, to illustrate how the ethnoreligious othering framework developed and applied in the study bolsters and advances process tracing explanations by systematically incorporating context-specific intersubjective meanings into causal accounts of the events under investigation. And third, to emphasize the importance of recognizing religion and nationalism as legitimate constituents and instruments of contemporary realpolitik by underlining their enduring security utility and essence at individual, group, and state levels. As argued and established throughout the book, because the causal mechanisms driving ethnoreligious otherings and passionate conflicts are simultaneously emitting and are propelled by deeply entrenched emotions, symbolic predispositions, and perceptions, achieving durable peace settlement requires reconciliation initiatives and regulation strategies that directly and unapologetically incorporate and address these neglected “immaterial” and “irrational” forces.
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Buckholtz, Joshua W., and Andreas Meyer-Lindenberg. Genetic Perspectives on the Neurochemistry of Human Aggression and Violence. Edited by Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.009.
Full textAbstract:
Violence is a devastating social phenomenon that is costly both to affected individuals and to society at large. Pathological aggression, especially reactive/impulsive aggression, is a cardinal symptom common to several psychiatric disorders—including antisocial personality disorder, borderline personality disorder, and psychopathy—that are associated with risk for violence. Thus, understanding the factors that predispose people to impulsive violence represents a crucial goal for psychology, neuroscience, and psychiatry. Although we are far from a full understanding of the etiopathophysiology of violence, impulsive aggression is heritable, suggesting that genetic mechanisms may be important for determining individual variation in susceptibility. This chapter synthesizes available preclinical and human data to propose a compelling neurogenetic mechanism for violence, specifically arguing that a genetically determined excess in serotonin signaling during a critical developmental period leads to dysregulation within a key corticolimbic circuit for emotional arousal and regulation, inhibitory control, and social cognition.
13
Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.
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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only to clinical neurologists but also to psychiatrists and physical therapists. The scope includes the mechanisms and abnormalities of DNA/RNA metabolism, proteostasis, vesicular biogenesis, and axonal transport and mechanisms of neurodegeneration; the role of the mitochondria in cell function and death mechanisms; ion channels, neurotransmission and mechanisms of channelopathies and synaptopathies; the functions of astrocytes, oligodendrocytes, and microglia and their involvement in disease; the local circuits and synaptic interactions at the level of the cerebral cortex, thalamus, basal ganglia, cerebellum, brainstem, and spinal cord transmission regulating sensory processing, behavioral state, and motor functions; the peripheral and central mechanisms of pain and homeostasis; and networks involved in emotion, memory, language, and executive function.
14
Gorman, Jack M. Life Events Shape Us. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190850128.003.0003.
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Psychiatry downplayed the importance of life events in causing mental illness from the 1960s on, favoring a view that all disorders except one are the result of abnormal genes affecting chemical processes in the brain. Studying the exception, posttraumatic stress disorder (PTSD), when it was defined in 1980 helped lead to renewed recognition that early life adversity is central to all psychiatric conditions. At the same time, neuroscientists showed that early life experiences are capable of changing life-long behavior and brain function in laboratory animals. One mechanism by which this occurs is through the epigenetic regulation of gene expression. Epigenetics is the way that the expression levels of genes are controlled without changing the underlying genetic code. Epigenetics is an attractive way of understanding how individual life experiences are translated in the brain into each person’s unique set of emotions, behaviors, abilities, and risks for psychiatric abnormalities.
15
Littlefield, Andrew K., and Kenneth J. Sher. Personality and Substance Use Disorders. Edited by Kenneth J. Sher. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199381678.013.006.
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Individual differences in personality have long been linked to the use and misuse of alcohol, tobacco, cannabis, and other drugs. Broadly, personality characteristics of high neuroticism and behavioral undercontrol/impulsivity appear to robustly relate to several substance use disorders (SUDs), although other traits have also been linked to SUDs. Much of the genetic basis of SUDs appears to be mediated by personality traits, which may relate to SUDs through a variety of non-mutually exclusive mechanisms that may work additively and synergistically, are indexed by various motivations associated with reward seeking and regulating negative emotion, and also relate to self-control and environment selection. Considerable change occurs in personality over the life course, and recent data show that the course of substance use and SUDs is associated with personality change. Although much progress has been made, several lines of future research could be pursued to further our understanding of the personality–SUD relation.
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Gibson, Matthew. Pride and Shame in Child and Family Social Work. Policy Press, 2019. http://dx.doi.org/10.1332/policypress/9781447344797.001.0001.
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This book reports on the first study into the role of pride and shame in social work practice. The concepts of pride, shame, guilt, humiliation, and embarrassment are outlined and analysed, providing a new framework for understanding and researching these emotions in professional practice. It is argued that these emotions are inherently part of practice, influencing what social workers do and how they do it. Such emotions are considered in the context of wider institutional processes that construct ideal forms of practice, which are then used to evaluate social workers’ actions and praise, shame, or humiliate them accordingly. The threat of shame, and promise of praise, influence most social workers to enact or conform to the standard, thereby regulating their practice. These emotions can, therefore, be considered to be strategically used as a mechanism of control by constructing contextually specific boundaries for shameful and praiseworthy behaviour that are policed within the organisation. While some social workers feel proud to act in such a manner in some contexts, often resulting in a difficult experience for the parents, many social workers feel constrained, believing they are no longer doing social work. Indeed, some social workers feel ashamed or guilty of what they are doing and seek to resist these attempts at control through acts of compromising, concealing, and influencing. This book provides a new theory for pride and shame in organisations and specifically outlines a theory for the role pride and shame play in leadership, management, and individual social work practice.