Relevant bibliographies by topics / Emotion processing deficits

Academic literature on the topic 'Emotion processing deficits'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Emotion processing deficits"

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Chaudhary, Shefali, Simon Zhornitsky, Herta H. Chao, Christopher H. van Dyck, and Chiang-Shan R. Li. "Emotion Processing Dysfunction in Alzheimer’s Disease: An Overview of Behavioral Findings, Systems Neural Correlates, and Underlying Neural Biology." American Journal of Alzheimer's Disease & Other Dementias® 37 (January 2022): 153331752210828. http://dx.doi.org/10.1177/15333175221082834.

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We described behavioral studies to highlight emotional processing deficits in Alzheimer’s disease (AD). The findings suggest prominent deficit in recognizing negative emotions, pronounced effect of positive emotion on enhancing memory, and a critical role of cognitive deficits in manifesting emotional processing dysfunction in AD. We reviewed imaging studies to highlight morphometric and functional markers of hippocampal circuit dysfunction in emotional processing deficits. Despite amygdala reactivity to emotional stimuli, hippocampal dysfunction conduces to deficits in emotional memory. Finally, the reviewed studies implicating major neurotransmitter systems in anxiety and depression in AD supported altered cholinergic and noradrenergic signaling in AD emotional disorders. Overall, the studies showed altered emotions early in the course of illness and suggest the need of multimodal imaging for further investigations. Particularly, longitudinal studies with multiple behavioral paradigms translatable between preclinical and clinical models would provide data to elucidate the time course and underlying neurobiology of emotion processing dysfunction in AD.
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Schönenberg, Michael, Alexander Schneidt, Eva Wiedemann, and Aiste Jusyte. "Processing of Dynamic Affective Information in Adults With ADHD." Journal of Attention Disorders 23, no. 1 (March 30, 2015): 32–39. http://dx.doi.org/10.1177/1087054715577992.

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Objective: ADHD has been repeatedly linked to problems in social functioning. Although some theories assume that the emotion recognition deficits are explained by general attentional deficits, mounting evidence suggests that they may actually constitute a distinct impairment. However, it remains unclear whether the deficient processing affects specific emotional categories or may generalize to all basic emotions. The present study aims to investigate these questions by assessing the sensitivity to all six basic emotions in adults with ADHD. Method: The participants judged the emotion onset in animated morph clips displaying facial expressions that slowly changed from neutral to emotional. Results: ADHD participants exhibited an impaired recognition of sad and fearful facial expressions. Conclusion: The present findings indicate that ADHD is possibly associated with a specific deficit in the recognition of facial emotions signaling negative social feedback.
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CADIEUX, NICOLE L., and KEVIN W. GREVE. "Emotion processing in Alzheimer's disease." Journal of the International Neuropsychological Society 3, no. 5 (September 1997): 411–19. http://dx.doi.org/10.1017/s1355617797004116.

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Emotion processing deficits may have an important effect on the quality of life of Alzheimer's disease (AD) patients and their families, yet there are few studies in this area and little is known about the cause of such deficits in AD. This study sought to determine if some AD patients have a disruption in a specific right hemisphere emotion processing system, and to determine if the processing of emotional facial expression is more vulnerable to the pathology of AD than is the perception of emotional prosody. It was specifically hypothesized that patients with greater right hemisphere dysfunction (low spatial AD patients) would be impaired on emotion processing tasks relative to those with predominantly left hemisphere dysfunction (low verbal AD patients). Both groups showed impairment on emotion processing tasks but for different reasons. The low verbal patients performed poorly on the affect processing measures because they had difficulty comprehending and/or remembering the task instructions. In contrast, low spatial AD patients have emotion processing deficits that are independent of language and/or memory and may be due to a more general visuoperceptual deficit that affects the perception of static but not dynamic affective stimuli. (JINS, 1997, 3, 411–419.)
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Philip, R. C. M., H. C. Whalley, A. C. Stanfield, R. Sprengelmeyer, I. M. Santos, A. W. Young, A. P. Atkinson, et al. "Deficits in facial, body movement and vocal emotional processing in autism spectrum disorders." Psychological Medicine 40, no. 11 (January 27, 2010): 1919–29. http://dx.doi.org/10.1017/s0033291709992364.

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BackgroundPrevious behavioural and neuroimaging studies of emotion processing in autistic spectrum disorder (ASD) have focused on the use of facial stimuli. To date, however, no studies have examined emotion processing in autism across a broad range of social signals.MethodThis study addressed this issue by investigating emotion processing in a group of 23 adults with ASD and 23 age- and gender-matched controls. Recognition of basic emotions (‘happiness’, ‘sadness’, ‘anger’, disgust' and ‘fear’) was assessed from facial, body movement and vocal stimuli. The ability to make social judgements (such as approachability) from facial stimuli was also investigated.ResultsSignificant deficits in emotion recognition were found in the ASD group relative to the control group across all stimulus domains (faces, body movements and voices). These deficits were seen across a range of emotions. The ASD group were also impaired in making social judgements compared to the control group and this correlated with impairments in basic emotion recognition.ConclusionsThis study demonstrates that there are significant and broad-ranging deficits in emotion processing in ASD present across a range of stimulus domains and in the auditory and visual modality; they cannot therefore be accounted for simply in terms of impairments in face processing or in the visual modality alone. These results identify a core deficit affecting the processing of a wide range of emotional information in ASD, which contributes to the impairments in social function seen in people with this condition.
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Ye, Wei. "The Atypical Emotional Processing in Bipolar Disorder and Relevant Interventions." Journal of Education, Humanities and Social Sciences 8 (February 7, 2023): 169–74. http://dx.doi.org/10.54097/ehss.v8i.4244.

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Individuals with bipolar disorder (BD) demonstrate impaired functioning including cognitive deficits, neurological damage, and psychosocial dysfunction. Atypical emotional processing, the lack of normal functioning in identifying human emotions, is one of the most significant impairments observed in individuals with BD. Such abnormal emotional processing has different causes. Deficits in emotion perception and recognition can contribute to the impaired social functioning in separation or in combination, which can further impede social functioning. Both facial emotion recognition deficits (FERD) and alexithymia are proven to be powerful predictors of BD development. Relevant interventions including Mindfulness-Based Cognitive Therapy for Children (MBCT-C), Dialectical Behavior Therapy (DBT), and Positive emotion regulation (PRE) intervention are verified to be effective in treatment for BD patients with deficits in emotional processing. By examining recent literature, this review encapsulates the relationships between atypical emotion recognition and perception and impaired social functioning, the relationship between deficits in emotional processing and BD symptoms, and efficacy of the interventions focusing on mindfulness, presenting a more comprehensive understanding in this area. It can provide some guidance to the prevention and intervention studies and practices for at-risk adolescents.
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Castro, L. C., and M. Martins. "Emotional Processing in Eating Disorders." European Psychiatry 24, S1 (January 2009): 1. http://dx.doi.org/10.1016/s0924-9338(09)70974-5.

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Background:Patients with eating disorders are reported to show emotional processing deficits compared to healthy controls.Aim:To study and discuss the role of emotional processing in eating disorders.Methods:Review of the literature.Results:Several studies found a marked impairment in emotional processing in eating disorder patients. These emotion-processing deficits seem to be independent of affective symptoms. Different studies address different concepts as emotional awareness, alexithymia and facial or voice emotional recognition. The emotional processing and functioning of eating disorder patients highly impact their relational styles and behaviour.Discussion:The literature suggests global emotion-processing deficits in eating disorders. The emotion-processing deficits in eating disorder patients may contribute to the poor interpersonal communication, lack of social cognition and lack of empathy frequently seen in these patients. It is extremely important a better understanding of these complex relationships as they can act as maintenance factors of the eating disorder, contributing to social isolation and therapeutic failure. The prognostic implications of these findings, as well as potential therapeutical interventions are topics valuable for future research in this area.
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Grace, Sally A., Wei Lin Toh, Ben Buchanan, David J. Castle, and Susan L. Rossell. "Impaired Recognition of Negative Facial Emotions in Body Dysmorphic Disorder." Journal of the International Neuropsychological Society 25, no. 08 (May 17, 2019): 884–89. http://dx.doi.org/10.1017/s1355617719000419.

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Abstract Objectives: Patients with body dysmorphic disorder (BDD) have difficulty in recognising facial emotions, and there is evidence to suggest that there is a specific deficit in identifying negative facial emotions, such as sadness and anger. Methods: This study investigated facial emotion recognition in 19 individuals with BDD compared with 21 healthy control participants who completed a facial emotion recognition task, in which they were asked to identify emotional expressions portrayed in neutral, happy, sad, fearful, or angry faces. Results: Compared to the healthy control participants, the BDD patients were generally less accurate in identifying all facial emotions but showed specific deficits for negative emotions. The BDD group made significantly more errors when identifying neutral, angry, and sad faces than healthy controls; and were significantly slower at identifying neutral, angry, and happy faces. Conclusions: These findings add to previous face-processing literature in BDD, suggesting deficits in identifying negative facial emotions. There are treatment implications as future interventions would do well to target such deficits.
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McDonald, Skye. "Are You Crying or Laughing? Emotion Recognition Deficits After Severe Traumatic Brain Injury." Brain Impairment 6, no. 1 (May 1, 2005): 56–67. http://dx.doi.org/10.1375/brim.6.1.56.65481.

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AbstractTo date there has been little research concerning the neuropsychological mechanisms of emotion perception deficits following traumatic brain injury (TBI), although such deficits are well documented. This paper considers two major issues. First, are emotion-processing deficits found regardless of the media of presentation? In a recent study examining this issue, adults with severe TBI were found to have particular problems identifying emotions from conversational tone, as well as difficulties when presented with still photographs and audiovisual dynamic displays (videoed vignettes). They were relatively normal when asked to classify emotions on the basis of moving visual displays without sound. This may reflect the fact that the parietal cortices, important for processing movement, are relatively unscathed in TBI. The second issue concerns whether emotion recognition is facilitated by empathic emotional responses and whether these are diminished in people with TBI. Evidence is presented for a relation between subjective reports of diminished emotional experience and emotion recognition accuracy. Finally, preliminary data suggests that people with TBI may fail to have empathic reactions when asked to passively view emotional expressions.
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Mier, D., C. Sauer, S. Lis, C. Esslinger, J. Wilhelm, B. Gallhofer, and P. Kirsch. "Neuronal correlates of affective theory of mind in schizophrenia out-patients: evidence for a baseline deficit." Psychological Medicine 40, no. 10 (January 8, 2010): 1607–17. http://dx.doi.org/10.1017/s0033291709992133.

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BackgroundSchizophrenia out-patients have deficits in affective theory of mind (ToM) but also on more basal levels of social cognition, such as the processing of neutral and emotional expressions. These deficits are associated with changes in brain activation in the amygdala and the superior temporal sulcus (STS). However, until now there have been no studies that examined these different levels of social cognition and their neurobiological underpinnings in patients within one design.MethodSixteen medicated schizophrenia out-patients and 16 matched healthy controls were studied with functional magnetic resonance imaging (fMRI) during a social cognition task that allows the investigation of affective ToM (aToM), emotion recognition and the processing of neutral facial expressions.ResultsPatients showed a deficit in emotion recognition and a more prominent deficit in aToM. The performance in aToM and in emotion recognition was correlated in the control group but not in the schizophrenia group. Region-of-interest analysis of functional brain imaging data revealed no difference between groups during aToM, but a hyperactivation in the schizophrenia group in the left amygdala and right STS during emotion recognition and the processing of neutral facial expressions.ConclusionsThe results indicate that schizophrenia out-patients have deficits at several levels of social cognition and provide the first evidence that deficits on higher-order social cognitive processes in schizophrenia may be traced back to an aberrant processing of faces per se.
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Keutmann, M. K., R. E. Gur, and R. C. Gur. "Understanding emotion processing in schizophrenia." Die Psychiatrie 07, no. 04 (October 2010): 217–26. http://dx.doi.org/10.1055/s-0038-1669583.

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SummaryImpaired emotional functioning is a prominent feature of schizophrenia. Although positive symptoms have traditionally attracted more attention and targeted treatment, negative symptoms, including flat affect, are increasingly recognized as the more debilitating and resistant to intervention. We describe studies examining affect processing in schizophrenia, focusing on facial affect with initial findings in vocal affect, or prosody. Deficits in schizophrenia are pronounced, and studies with functional neuroimaging indicate that the neural substrates for these deficits center on the amygdala and its projections. The abnormalities are highly correlated with symptom severity and functional outcome. While there is quite extensive work on affect recognition abnormalities, deficits have also been documented in the ability to express affect on the face and in voice, and perhaps to a lesser extent in the experience of emotion. These abnormalities can be better studied when methods for quantitative analysis of emotional expression are available. Recognizing the existence of such deficits and their neural substrates will lead to improved approaches to pharmacological and behavioral treatment.
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Dissertations / Theses on the topic "Emotion processing deficits"

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Schafer, Molly Clark. "Emotional Processing Deficits in Parkinson's Disease." Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/3687.

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Introduction: Parkinson’s disease (PD) is known to cause detrimental effects to motor function and cognition. The motor effects of the disease in turn impact emotion expression in patients with PD. There is conflicting evidence in research, however, as to whether PD also affects emotion comprehension, and if so, what emotions in particular are affected and across what modalities. This study aimed to investigate the effects of PD on a broad range of skills involved in basic and complex emotion comprehension. Whether these effects extend into other areas associated with emotion processing, such as social cognition and autobiographical memory, was also explored. Methods: Sixteen patients with PD participated in the study along with sixteen control subjects who were matched for age, gender, education level and estimated premorbid intelligence. The PD participants, on average, were in the moderate phase of the disease and taking PD medication, including dopamine. Participants were tested on a range of recognition measures including prototypical and morphed facial expressions with reduced intensity (40 and 80%), emotion prosody, written emotion vignettes, emotional imagery, pictures of emotion, social cognition, and a cued autobiographical memory task. A mood inventory was given, and disease severity and duration were noted. Results: The PD group did not show pervasive deficits in emotion recognition overall. Deficits were demonstrated in prosody recognition, specifically with fearful tones, and in an incongruent prosody task, specifically with angry and neutral tones. The PD group was not able to recognise facial expressions of disgust (mixed intensities) as well as controls, with the result showing a trend toward significance. PD participants were also significantly worse in Theory of Mind (TOM) reasoning but not at another social cognition measure involving recognising social emotions through expressions from the eyes only. There were no differences between the groups across all other tests. Discussion: PD is thought to cause subtle deficits in emotion comprehension which are only elucidated through complex tasks. The effects of PD on complex processing also impact TOM performance, which relies on skills involved in complex emotion recognition. Effects of mood and disease factors on performance were circumscribed. Evidence suggested that the basal ganglia and fronto-striatal connections play a role in emotion comprehension.
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Schafer, Molly Clark. "Emotional Processing Deficits in Parkinson's Disease." University of Sydney, 2008. http://hdl.handle.net/2123/3687.

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Doctor of Clinical Neuropsychology/Master of Science
Introduction: Parkinson’s disease (PD) is known to cause detrimental effects to motor function and cognition. The motor effects of the disease in turn impact emotion expression in patients with PD. There is conflicting evidence in research, however, as to whether PD also affects emotion comprehension, and if so, what emotions in particular are affected and across what modalities. This study aimed to investigate the effects of PD on a broad range of skills involved in basic and complex emotion comprehension. Whether these effects extend into other areas associated with emotion processing, such as social cognition and autobiographical memory, was also explored. Methods: Sixteen patients with PD participated in the study along with sixteen control subjects who were matched for age, gender, education level and estimated premorbid intelligence. The PD participants, on average, were in the moderate phase of the disease and taking PD medication, including dopamine. Participants were tested on a range of recognition measures including prototypical and morphed facial expressions with reduced intensity (40 and 80%), emotion prosody, written emotion vignettes, emotional imagery, pictures of emotion, social cognition, and a cued autobiographical memory task. A mood inventory was given, and disease severity and duration were noted. Results: The PD group did not show pervasive deficits in emotion recognition overall. Deficits were demonstrated in prosody recognition, specifically with fearful tones, and in an incongruent prosody task, specifically with angry and neutral tones. The PD group was not able to recognise facial expressions of disgust (mixed intensities) as well as controls, with the result showing a trend toward significance. PD participants were also significantly worse in Theory of Mind (TOM) reasoning but not at another social cognition measure involving recognising social emotions through expressions from the eyes only. There were no differences between the groups across all other tests. Discussion: PD is thought to cause subtle deficits in emotion comprehension which are only elucidated through complex tasks. The effects of PD on complex processing also impact TOM performance, which relies on skills involved in complex emotion recognition. Effects of mood and disease factors on performance were circumscribed. Evidence suggested that the basal ganglia and fronto-striatal connections play a role in emotion comprehension.
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Hubble, Kelly. "Antisocial behaviour in adolescents : exploring and improving emotion processing deficits." Thesis, Cardiff University, 2015. http://orca.cf.ac.uk/89388/.

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Antisocial behaviour in childhood and adolescence is associated with a range of negative outcomes in later life, which are costly to both society and to the antisocial individual themselves. Because the effectiveness of current interventions appears to be limited, it has been argued that treatment efforts should focus more on designing interventions that target neuropsychological correlates of antisocial behaviour. Two important correlates are impaired facial emotion recognition and empathy; these deficits have been proposed to cause antisocial behaviour because they involve an inability to understand and appropriately respond to the distress of others.
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Johnston, Patrick James. "Facial emotion processing deficits in schizophrenia : an integrative cognitive neurosciences approach." Thesis, Northumbria University, 2004. http://nrl.northumbria.ac.uk/7327/.

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Schizophrenia is a debilitating disease associated with deficits in a wide variety of cognitive, social and emotional domains. This includes deficits in facial affect recognition, which have been suggested to be linked to problems in judging social intentions and as leading to social withdrawal. A number of behavioural studies report a greater deficit in the recognition of negative emotions (particularly fear). a parallel strand of research has examined the neural substrates of facial emotion recognition using neuroimaging techniques, and has reported reductions in levels of amygdala activation to fearful faces in schizophrenia patients. These finding, have, in combination, been interpreted as indicating a negative emotion specific deficit in schizophrenia underpinned by aberrant functioning of limbic structures including the amygdala. The current thesis attempts to deconstruct this formulation from two perspectives, a psychometric or task conceptualisation perspective, and a neuroimaging perspective.
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Buxton, Sharon L. "Shadows of emotion: emotional processing deficits in Parkinson's disease and their impact on social relationships." Thesis, University of Auckland, 2011. http://hdl.handle.net/2292/6582.

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Evidence suggests individuals with Parkinson's disease (PD) may be impaired at production and recognition of affective prosody and facial expressions of emotion. When united with social psychology's assertion that nonverbal cues, including facial expressions and voice tone, facilitate effective communication within social relationships, it suggests that these impairments impact negatively on quality of life for individuals with PD. This thesis aimed to investigate emotional processing deficits in PD, systematically, and their possible affect on social relationships. Thirty PD and 30 closely-matched control participants participated in three studies. Study 1 found that raters were less able to recognise emotional expressions from facial expressions and affective prosody in the PD group than the Control group. Study 2, using the Aprosodia Battery (Ross et al., 1997), found PD participants were impaired at recognition of affective prosody. A novel task was developed to assess recognition of facial expressions, comprising prototypical facial expressions and more subtle facial expressions. The PD group was impaired at recognition of subtle facial expressions but not prototypical facial expressions. Positive associations were found between performance on some recognition and production tasks in the PD group, providing support for an overall impairment in emotional processing in PD. When the PD group was subdivided based on side of symptom-onset, the group who developed right-sided symptoms first were more impaired compared to the Control group, than those who had left-sided onset of symptoms, suggesting a possible left-hemispheric lateralisation of emotional processing. Study 3, using a semi-structured interview, found that individuals with PD were less satisfied with their relationships with close others and acquaintances than the Control group, and that this was associated with how well the PD participants felt that others understood their facial expressions and affective prosody. Findings support the contribution of the basal ganglia to emotional processing, possibly as part of the thalamo-cortical loops, specifically, the skeleto-motor circuit, the lateral orbitofrontal circuit and the anterior cingulate circuit.
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Kopf, Juliane [Verfasser], Andreas [Gutachter] Reif, and Paul [Gutachter] Pauli. "Emotion processing and working memory deficits in Bipolar Disorder: interactions and changes from acute to remitted state / Juliane Kopf ; Gutachter: Andreas Reif, Paul Pauli." Würzburg : Universität Würzburg, 2018. http://d-nb.info/1160187835/34.

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Lothian, Sharon. "Emotional processing deficits in colorectal cancer : a theoretical overview and empirical investigation." Thesis, University of Southampton, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268646.

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Baur, Ramona [Verfasser], and Andreas [Gutachter] Mühlberger. "Adult Attention-Deficit/Hyperactivity Disorder (ADHD), Emotion Processing, and Emotion Regulation in Virtual Reality / Ramona Baur ; Gutachter: Andreas Mühlberger." Würzburg : Universität Würzburg, 2016. http://d-nb.info/1124396047/34.

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Bui, Kim-Kim. "Face Processing in Schizophrenia : Deficit in Face Perception or in Recognition of Facial Emotions?" Thesis, University of Skövde, School of Humanities and Informatics, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-3349.

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Schizophrenia is a psychiatric disorder characterized by social dysfunction. People with schizophrenia misinterpret social information and it is suggested that this difficulty may result from visual processing deficits. As faces are one of the most important sources of social information it is hypothesized that people suffering from the disorder have impairments in the visual face processing system. It is unclear which mechanism of the face processing system is impaired but two types of deficits are most often proposed: a deficit in face perception in general (i.e., processing of facial features as such) and a deficit in facial emotion processing (i.e., recognition of emotional facial expressions). Due to the contradictory evidence from behavioural, electrophysiological as well as neuroimaging studies offering support for the involvement of one or the other deficit in schizophrenia it is early to make any conclusive statements as to the nature and level of impairment. Further studies are needed for a better understanding of the key mechanism and abnormalities underlying social dysfunction in schizophrenia.

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Serrano, Verenea J. "Exploring Social Information Processing of Emotion Content and its Relationship with Social Outcomes in Children at-risk for Attention-Deficit/Hyperactivity Disorder." Ohio University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1501163848926904.

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Books on the topic "Emotion processing deficits"

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Timoney, Linden R., and Mark D. Holder. Emotional Processing Deficits and Happiness. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2.

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Carrión, Victor G., John A. Turner, and Carl F. Weems. Emotion Processing. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190201968.003.0003.

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Prolonged difficulty identifying and regulating emotions is another essential symptom of PTSD, and has been associated with hormonal dysregulation, social and academic difficulties, and structural and functional brain deficits in youth and adults. Individual subject variance in personality, disposition, sex, and genotype has been shown to uniquely modulate the prefrontal and limbic brain regions associated with emotion processing. The current chapter examines how the component processes of emotion regulation, such as fear conditioning, can be dysregulated by the experience of traumatic stress, by which the brain centers that manage reactions to emotionally charged stimuli are over- or underactivated. The preclinical literature that serves as the basis for our understanding of these systems is reviewed, as well as studies of adults and children who have experienced trauma. Future directions, such as clinical care based on neuroendocrine research, are also discussed.
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Hamilton, Nancy A., Ruth Ann Atchley, Lauren Boddy, Erik Benau, and Ronald Freche. Emotion Regulation and Cognitive Control in Pain Processing. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190627898.003.0003.

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Chronic pain is a multidimensional phenomenon characterized by deficits at the behavioral, social, and affective levels of functioning. Depression and anxiety disorders are overrepresented among pain patients, suggesting that pain affects processes of emotion regulation. Conceptualizing the experience of chronic pain within a motivational organizing perspective offers a useful framework for understanding the emotional experiences of individuals living with chronic pain and how they balance harm-avoidant goals with generative approach oriented goals. To that end this chapter also integrates theories of emotion regulation (ER) and cognitive control to shed additional light on the problem of living with chronic pain, and it introduces a theory, consistent with findings from affective neuroscience, suggesting that painful flare-ups may be driven by anticipatory pain reactions in addition to somatic signals.
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Buhlmann, Ulrike, and Andrea S. Hartmann. Cognitive and Emotional Processing in Body Dysmorphic Disorder. Edited by Katharine A. Phillips. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190254131.003.0022.

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According to current cognitive-behavioral models, body dysmorphic disorder (BDD) is characterized by a vicious cycle between maladaptive appearance-related thoughts and information-processing biases, as well as maladaptive behaviors and negative emotions such as feelings of shame, disgust, anxiety, and depression. This chapter provides an overview of findings on cognitive characteristics such as dysfunctional beliefs, information-processing biases for threat (e.g., selective attention, interpretation), and implicit associations (e.g., low self-esteem, strong physical attractiveness stereotype, and high importance of attractiveness). The chapter also reviews face recognition abnormalities and emotion recognition deficits and biases (e.g., misinterpreting neutral faces as angry) as well as facial discrimination ability. These studies suggest that BDD is associated with dysfunctional beliefs about one’s own appearance, information-processing biases, emotion recognition deficits and biases, and selective processing of appearance-related information. Future steps to stimulate more research and clinical implications are discussed.
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Quadt, Lisa, Hugo D. Critchley, and Sarah N. Garfinkel. Interoception and emotion: Shared mechanisms and clinical implications. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0007.

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Internal states of bodily arousal contribute to emotional feeling states and behaviors. This chapter details the influence of interoceptive processing on emotion and describes how deficits in interoceptive ability may underpin aberrant emotional processes characteristic of clinical conditions. The representation and control of bodily physiology (e.g. heart rate and blood pressure) and the encoding of emotional experience and behavior share neural substrates within forebrain regions coupled to ascending neuromodulatory systems. This functional architecture provides a basis for dynamic embodiment of emotion. This chapter will approach the relationship between interoception and emotion within the interoceptive predictive processing framework and describe how emotional states could be the product of interoceptive prediction error minimization.
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May, Joshua. The Limits of Emotion. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811572.003.0002.

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Empirical research apparently suggests that emotions play an integral role in moral judgment. The evidence for sentimentalism is diverse, but it is rather weak and has generally been overblown. There is no evidence that our moral concepts themselves are partly composed of or necessarily dependent on emotions. While the moral/conventional distinction may partly characterize the essence of moral judgment, moral norms needn’t be backed by affect in order to transcend convention. Priming people with incidental emotions like disgust doesn’t make them moralize actions. Finally, moral judgment can only be somewhat impaired by damage to areas of the brain that are generally associated with emotional processing (as in acquired sociopathy and frontotemporal dementia). While psychopaths exhibit both emotional and rational deficits, the latter alone can explain any minor defects in moral cognition.
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Newman, Jennifer, and Charles R. Marmar. Executive Function in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0015.

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This chapter discusses the role of executive function in post-traumatic stress disorder (PTSD), which is far from fully understood. Deficits are subtle and findings are often inconsistent. Impairments have been related to worsening of psychological symptoms, functioning, and quality of life. They can also negatively impact treatment. Functional imaging shows that neurocognitive deficits in PTSD may be related to an imbalance in brain connectivity, where emotion processing is enhanced and control is reduced. Structural findings show abnormalities in brain regions involved in higher-level functions. However, findings are often discrepant. Factors related to these inconclusive results are considered, including developmental course, premorbid functioning, and comorbidities such as traumatic brain injury, depression, substance use, attention deficit hyperactivity disorder, health behaviors, and medical concerns. Treatment implications, limitations of this work, and future directions are presented. The aim of future research is to advance scientific understanding of PTSD, neurocognitive impairments, and related conditions, with the goal of improving outcomes for those who encounter trauma.
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Goodkind, Madeleine S., and Amit Etkin. Functional Neurocircuitry and Neuroimaging Studies of Anxiety Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0034.

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Anxiety and fear serve adaptive functions and include wide-ranging subjective, physiological, behavioral, and cognitive responses. When these reactions are present chronically, and to a heightened degree that generalizes to signals beyond those that are objectively dangerous, one sees emergence of clinical anxiety disorders. Historically, anxiety disorders have been conceptualized as disruptions in fear processing, though more recent accounts also highlight changes in emotional reactivity beyond fear and deficits in emotion regulation. In this chapter, we review the neural circuitry relevant for fear processing and for emotional reactivity and regulation more broadly. We then review neuroimaging studies of social anxiety disorder, specific phobia, generalized anxiety disorder, panic disorder, and posttraumatic stress disorder. We highlight areas of overlap between disorders as well as disorder-specific perturbations.
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Soloff, Paul, and Christian Schmahl. Suicide and Nonsuicidal Self-Injury. Edited by Christian Schmahl, K. Luan Phan, Robert O. Friedel, and Larry J. Siever. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199362318.003.0011.

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This chapter reviews current data on the prevalence of suicidal behavior and non-suicidal self-injury (NSSI) in patients with PDs; the characteristics of attempters versus completers; and the epidemiology of NSSI in borderline personality disorder (BPD). In addition, it presents explanatory models for suicide and NSSI. Also, there are comprehensive discussions of the neurobiological mechanisms involved in both suicidality and NSSI focusing on the structural and functional neuroimaging of emotion dysregulation, impulsivity, executive cognitive deficits, affective interference and cognitive function, and the Endogenous Opioid System. The chapter concludes with a detailed description of pain processing as it interacts with NSSI.
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Maren, Stephen. Neural Circuits for Context Processing in Aversive Learning and Memory. Edited by Israel Liberzon and Kerry J. Ressler. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190215422.003.0005.

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The nature and properties of emotional expression depend importantly on not only the stimuli that elicit emotional responses, but also the context in which those stimuli are experienced. Deficits in context processing have been associated with a variety of cognitive-emotional disorders, including post-traumatic stress disorder (PTSD). These deficits can be localized to specific neural circuits underlying context processing in the mammalian brain. In particular, the hippocampus has been implicated through numerous animal and human studies to be involved both in normal contextual memory formation, but also in discrimination of trauma-related cues. Decreased hippocampal functioning, as is observed in PTSD, is associated with increased generalization of fear and threat responses as well as deficits in extinction of fear. Understanding context processing offers the opportunity to further understand the biology of PTSD and to target new approaches to therapeutics.
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Book chapters on the topic "Emotion processing deficits"

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Timoney, Linden R., and Mark D. Holder. "Definition of Alexithymia." In Emotional Processing Deficits and Happiness, 1–6. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_1.

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Timoney, Linden R., and Mark D. Holder. "The History of the Construct and Etiology of Alexithymia." In Emotional Processing Deficits and Happiness, 7–11. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_2.

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Timoney, Linden R., and Mark D. Holder. "The Importance of Research on Alexithymia." In Emotional Processing Deficits and Happiness, 13–15. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_3.

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Timoney, Linden R., and Mark D. Holder. "Measurement of Alexithymia." In Emotional Processing Deficits and Happiness, 17–33. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_4.

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Timoney, Linden R., and Mark D. Holder. "Recommendations for Measurement." In Emotional Processing Deficits and Happiness, 35–39. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_5.

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Timoney, Linden R., and Mark D. Holder. "Correlates of Alexithymia." In Emotional Processing Deficits and Happiness, 41–60. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_6.

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Timoney, Linden R., and Mark D. Holder. "Alexithymia and Personality." In Emotional Processing Deficits and Happiness, 61–68. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_7.

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Timoney, Linden R., and Mark D. Holder. "Alexithymia and Subjective Well-Being." In Emotional Processing Deficits and Happiness, 69–82. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_8.

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Timoney, Linden R., and Mark D. Holder. "Summary and Recommendations for Future Research." In Emotional Processing Deficits and Happiness, 83–88. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7177-2_9.

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Rice, Frances, Shiri Davidovich, and Sandra Dunsmuir. "Emotion Regulation and Depression." In Emotion Regulation and Psychopathology in Children and Adolescents, edited by Cecilia A. Essau, Sara Leblanc, and Thomas H. Ollendick, 171–95. Oxford University Press, 2017. http://dx.doi.org/10.1093/med:psych/9780198765844.003.0009.

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Chapter 9 delineates the symptomology, epidemiology and risk factors associated with depression in children and adolescents. Youth depression involves an assemblage of co-occurring symptoms causing impairment and dysfunction across school, interpersonal relationships, and extracurricular activities. Core diagnostic features of depressive disorders include: depressed mood, loss of interest or pleasure in activities and decreased energy. Depressed youth demonstrate deficits in processing emotional material, which deleteriously impacts regulatory efforts. These impairments include low levels of bias for positive information. Until recently CBT was considered the gold-standard treatment for depression; however, research demonstrates there is limited quality evidence to suggest one treatment modality offers a preferred approach. Thus, interventions should consider the child’s age, cognitive competency, and educational experience when determining treatment options. A novel intervention with auspicious, preliminary findings is Thinking about Reward in Young People (TRY), which incorporates CBT and behavioral activation, with an emphasis on increasing reward-processing.
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Conference papers on the topic "Emotion processing deficits"

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Silva, Rogeria Cristina, Raquel Luíza Carvalho, and Marcia Cristina Dourado. "THE IMPACT OF ALZHEIMER’S DISEASE ON EMOTIONAL PROCESSING." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda053.

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Background: Emotional processing involves the ability of the individual to infer emotional information. There is no consensus about how Alzheimer’s disease (AD) affects emotional processing. Objective: We aim to systematically review the impact of AD on emotion processing Method: We conducted a search based on the Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA). The literature search was performed using the electronic databases MEDLINE (Pubmed) and Science Citation Index (ISI). The following descriptors were used in the review process: emotion or emotional processing, cognition or cognitive functions and Alzheimer disease or Alzheimer’s disease. This systematic review was recorded in the International Prospective Register of Systematic Reviews (PROSPERO) under the number CRD42018115891. Results: We identified 425 articles, 19 of which met our criteria. Visual emotional stimuli were the most used among studies. Most studies used tasks of emotional naming, discrimination, identification and correspondence. The results were contradictory. Many studies reported that individuals with AD were impaired on emotional perception tasks, while others results reported preserved skills. The relationship between emotional processing and cognition is also unclear. Some studies suggested that general cognitive performance affects performance in emotional perception tasks among people with AD, but other studies have shown deficits in recognizing emotion, regardless cognitive performance. Conclusions: Studies are scarce, present contradictory results, and report impairment in emotional processing in relation to cognition. Moreover, analyzes of correlation between emotion processing and cognitive functioning failed to reveal clear relationships.
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Pereira, Nayara Karina Ferreira, Fábio Ricardo de Oliveira Galvão, Vaneza Mirele Gomes dos Santos, Enio Walker Azevedo Cacho, Marcos Hortes Nisihara Chagas, and Roberta de Oliveira Cacho. "Evaluation of social cognition in post stroke patients: a hemispheric analysis." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.247.

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Introduction: Social cognition is an increasingly explored field of research in the study of post-acquired brain injury psychiatric disorders, encompasses a number of domains including theory of mind (ToM). The ToM refers to the ability to understand, infer emotions, and predict the behavior of others. ToM deficits can impair information processing, and the laterality of the brain injury can influence the level of impairment. Objectives: To investigate whether laterality of the post-stroke hemispheric lesion influences performance on ToM tasks. Methods: Cross-sectional observational study, with chronic post-stroke subjects, aged ≥18 years, without cognitive deficits suggestive by Mini Mental State Examination (MMSE). The volunteers were submitted to a single evaluation using the Theory of Mind Task Battery (ToM TB) in its version translated and adapted to Portuguese (Brazil). The participants were allocated into two groups: group with right hemisphere injury (RHI) and group with left hemisphere injury (LHI). Comparative statistics were performed based on the ToM TB scores between the two groups using Student’s t-test. Results: Thirty-eight participants (RHI=22 and LHI=16) were included. There was no difference between the RHI and LHI group regarding social cognition assessed by ToM TB (t=1.63; p=0.11). Conclusion: The laterality of brain injury appears to not influence the level of performance on ToM tasks in stroke survivors.
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Marcu, Gabriela Mariana. "Particularities of Electrophysiological and Psychoneurological Assessment of Mild Traumatic Brain Injury." In World Lumen Congress 2021, May 26-30, 2021, Iasi, Romania. LUMEN Publishing House, 2022. http://dx.doi.org/10.18662/wlc2021/38.

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Neuropsychological functioning after mTBI is individualized and dynamic, with no currently known predictors and usually having a trajectory of gradual improvement. It is still a challenge to identify specific cognitive profiles associated with mTBI. One of the causes is the transient character of TBI symptoms as they are not appearing immediately after the injury. Another explanation resides in the individual and group variability of cognitive impairements following mTBI, which also affects the standardisation of the neuropsychological tests to use in mTBI assessment batteries (Iverson et al., 2013; Prince & Bruhns, 2017; Tulsky et al., 2017). Presently concussion has no accepted definition or diagnostic criteria. Also, there is no standard (or gold standard) for screening or properly identifying and diagnosing all population with concussion. (Borg et al., 2004). Patients with mTBI could evolve in a bunch of physical, cognitive, and emotional symptoms (Permenter et al., 2021) that are usually known as post-concussion syndrome (PCS). In terms of symptoms, we target neuropsychological evaluation of four key domains (“higher-order attention”, “executive function”, “episodic memory”, and “speed of information processing”) implicated in chronic impairment after mTBI. Alternatively, studies on the EEG frequency domain shed new light on the possibility to have a diagnostic marker based on QEEG patterns identified in the mTBI population and some prognostic factors for the PCS syndrome.(Rapp et al., 2015; Thornton & Carmody, 2009). Given the particularities of neuropsychological functioning after mTBI we emphasize the need of a mixed methodology, using both electrophysiological and psychoneurological tools, to provide the best sensitivity and specificity in assessing cognitive and functional deficits and in predicting further PCS.
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