Relevant bibliographies by topics / Elephantiasis

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Academic literature on the topic 'Elephantiasis'

Author: Grafiati
Published: 4 June 2021
Last updated: 30 July 2024

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Journal articles on the topic "Elephantiasis"

1

Mulyaningsih, Budi, Sitti Rahmah Umniyati, Ernaningsih Ernaningsih, Tri Baskoro Tunggul Satoto, Tridjoko Hadianto, and Siti Isti’anah. "Deteksi filariasis dan vektornya di wilayah kerja Dinas Kesehatan Kabupaten Bantul, Daerah Istimewa Yogyakarta." Journal of Community Empowerment for Health 2, no. 1 (June 14, 2019): 53. http://dx.doi.org/10.22146/jcoemph.41524.

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In Bantul, Yogyakarta Special Region there have been reported 6 elephantiasis cases and ware suspected as filariasis. Elephantiasis is classified into two, namely filarial elephantiasis and nonfilarial elephantiasis. Filarial elephantiasis caused by lymphatic filarial worms infection and nonfilarial elephantiasis can be caused by podoconiosis, leprosy, tuberculosis, or chlamydia infection. The aim of the study was to ascertain whether elephantiasis cases are caused by filariasis. Activities carried out in the work area of the Bantul District Health Office in July 2016 i.e.: (1) patient location survey, (2) interviews with patients, their families, and surrounding communities, (3) examination of patients, and (4) observe the environment around the patient's residence to ensure existence of vector mosquito breeding places. This study found two people with elephantiasis, patients from Depok, Gilangharjo, Pandak, Bantul was suspected elephantiasis due to Brugia malayi infection. Patient from Cawan, Argodadi, Sedayu, Bantul was suspected elephantiasis due to podoconiosis. In Cawan found many breeding sites for the Anopheles vagus mosquito.
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Susilowati, Dyah. "UTILIZATION OF ROSMARIN LEAF OIL (Rosmarinus officinalis L) ON Culex Quinquefasciatus MOSQUITO LARVA AS A FILARIASIS VECTOR (ELEPHANT FOOT DISEASE)." Jurnal Farmasi (Journal of Pharmacy) 1, no. 1 (October 20, 2018): 27–33. http://dx.doi.org/10.37013/jf.v1i1.60.

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Elephantiasis (filariasis / elephantiasis) is still endemic in hundreds of districts in Indonesia and has become a health problem for the world community in accordance with the resolution of the World Health Assembly (WHA) in 1997. This is caused by Culex quinquefasciatus mosquitoes as one of many filariasis vectors found in urban areas (Gandahusada et al., 1998), while the number of filariasis chronic cases reported until 2009 was 11,914 cases. Filariasis / elephantiasisi is a chronic infectious disease caused by filarial worm infection transmitted by various types of mosquitoes in the lymph nodes, this disease is chronic (chronic) and if it does not get treatment can cause permanent disability in the form of enlargement of the legs, arms and genitals both women and men man. Rosmarin (Rosmarinus officinalis L.) is a multifunctional plant in Indonesia especially abroad, one of its benefits is as larvicides. Because rosmarin has a chemical compound, one of them is essential oil which has larvicidal effectiveness on Culex quinquefasciatus mosquito larvae as vector filariasis (elephantiasis) Essential oil was obtained by distillation of fresh rosmarin leaves, then each extract was made with 1000 ppm stock solution and then made 6 series of concentration of 12.5 ppm to 300 ppm then tested on 20 larvae of instar III Culex quinquefasciatus, Observations were made after 24 hours and calculated LC90 uses an analysis of the orbit. The results obtained were only essential oils of Rosmarin leaves (Rosmarinus officinalis L.) which had larvicidal activity against Culex quinquefasciatus mosquito larvae, with LC90 182,9756 ppm.
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Sethi, Surendra Kumar, Kavita Jain, Neena Jain, and Sohan Lal Yadav. "Anesthetic management for a cesarean section in a parturient with huge vulvar and lower limbs elephantiasis: a case report." Journal of Society of Anesthesiologists of Nepal 4, no. 2 (October 1, 2018): 91–93. http://dx.doi.org/10.3126/jsan.v4i2.21210.

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Elephantiasis is caused by accumulation of lymph in soft tissues due to long standing lymphatic obstruction. Elephantiasis of female external genitalia is very rare and its association with limb elephantiasis is also quite very rare. A 20 years old, primigravida patient with 38 weeks’ gestation presented with massive swelling of lower limbs along with vulvar edema posted for emergency cesarean section diagnosed as elephantiasis of lower limbs and vulva which is usually rare and has not been reported yet. Elephantiasis in pregnancy poses difficulty for an anesthesiologist while providing neuraxial anesthesia to such a patient. We report the anesthetic management of a primigravida patient with both vulvar and lower limbs elephantiasis posted for emergency cesarean section.Journal of Society of Anesthesiologists of NepalVol. 4, No. 2, 2017, Page: 91-93
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Gupta, Somesh, C. Ajith, Amrinder J. Kanwar, Virendra N. Sehgal, Bhushan Kumar, and Uttam Mete. "Genital elephantiasis and sexually transmitted infections – revisited." International Journal of STD & AIDS 17, no. 3 (March 1, 2006): 157–66. http://dx.doi.org/10.1258/095646206775809150.

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Genital elephantiasis is an important medical problem in the tropics. It usually affects young and productive age group, and is associated with physical disability and extreme mental anguish. The majority of cases are due to filariasis; however, a small but significant proportion of patients develop genital elephantiasis due to bacterial sexually transmitted infections (STIs), mainly lymphogranuloma venereum (LGV) and donovanosis. STI-related genital elephantiasis should be differentiated from elephantiasis due to other causes, including filariasis, tuberculosis, haematological malignancies, iatrogenic, or dermatological diseases. Laboratory investigations like microscopy of tissue smear and nucleic acid amplification test for donovanosis, and serology and polymerase chain reaction for LGV may help in the diagnosis, but in endemic areas, in the absence of laboratory facilities, diagnosis largely depends on clinical characteristics. The causative agent of LGV, Chlamydia trachomatis serovar L1–L3, is a lymphotropic organism which leads to the development of thrombolymphangitis and perilymphangitis, and lymphadenitis. Long-standing oedema, fibrosis and lymphogranulomatous infiltration result in the final picture of elephantiasis. Elephantiasis in donovanosis is mainly due to constriction of the lymphatics which are trapped in the chronic granulomatous inflammatory response generated by the causative agent, Calymmatobacterium (Klebsiella) granulomatis. The LGV-associated genital elephantiasis should be treated with a prolonged course of doxycycline given orally, while donovanosis should be treated with azithromycin or trimethoprim-sulphamethoxazole combination given for a minimum of three weeks. Genital elephantiasis is not completely reversible with medical therapy alone and often needs to be reduced surgically.
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Palanisamy, ArunPrasath, KamalakannanKutuwa Kanakaram, Sivasubramanian Vadivel, and Srivenkateswaran Kothandapany. "Vulval elephantiasis." Indian Dermatology Online Journal 6, no. 5 (2015): 371. http://dx.doi.org/10.4103/2229-5178.164470.

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Famularo, Giuseppe, Laura Conversano, and Liborio Manente . "Elephantiasis neuromatosa." Our Dermatology Online 11, no. 1 (January 2, 2020): 53–55. http://dx.doi.org/10.7241/ourd.20201.13.

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Singh, Nidhi, Rashmi Kumari, and DevinderMohan Thappa. "Elephantiasis tropica." International Journal of Advanced Medical and Health Research 1, no. 1 (2014): 26. http://dx.doi.org/10.4103/2349-4220.134447.

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Zugor, V., R. Horch, D. Engehausen, and G. Schott. "Penoskrotale Elephantiasis." Aktuelle Urologie 38, no. 3 (May 2007): 252–54. http://dx.doi.org/10.1055/s-2006-932156.

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Meng, Zhaowei, Mei Zhu, and Jian Tan. "Elephantiasis Legs." American Journal of the Medical Sciences 347, no. 3 (March 2014): 248. http://dx.doi.org/10.1097/maj.0b013e31826d626c.

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Chiu, Hsien-Yi, and Yi-Hua Liao. "Elephantiasis Neuromatosa." New England Journal of Medicine 368, no. 18 (May 2, 2013): e23. http://dx.doi.org/10.1056/nejmicm1206146.

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Dissertations / Theses on the topic "Elephantiasis"

1

Bhatnagar, Barkha. "An examination of the effects of ivermectin on Brugia malayi adult worms /." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=100768.

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Brugia malayi is one of the causative agents of the disabling and disfiguring disease known as Lymphatic Filariasis (LF). This infection is a well-established ailment in tropical and subtropical countries and recently the drug ivermectin has been introduced for the LF control programs. Ivermectin (IVM) is an excellent microfilaricide, but is not markedly macrofilaricidal. However, it causes a long-lasting reduction in the production of new larvae by female worms, suggesting that adult stages are affected. However, the mechanism by which IVM produces such effect in the adult worm is not well understood. One major reason is our incomplete understanding about the biological effect of IVM on adult stages. The present study was carried out to examine the in vitro effects of IVM on B. malayi adult worms using Brugia-gerbil animal model. And also to have some leads in understanding the drug-uptake and location of probable targets in the worm body by using fluorescent labeled IVM and confocal microscopy.
The antifilarial effects of IVM were examined using three parameters: mf release by female worms, and motility, and viability in both male and female worms. The results reported in this study demonstrate that although IVM did not kill the adult worm, but showed significant antifilarial effects on B. malayi adult stages when examined in an in vitro system. Confocal microscopy images of the worms incubated in bodipy FITC-IVM showed strong specific localization signal in the anterior cephalic region of both male and female worms. These observations suggest the early/initial interactions of the drug with its probable receptors that could be located specifically in the head region.
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Schwab, Anne Elisabeth. "The genetics of potential albendazole and ivermectin resistance in lymphatic filariae /." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103006.

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A current initiative to eliminate lymphatic filariasis (LF), headed by the World Health Organization, aims to interrupt transmission of the disease through yearly community-wide treatment with the broad spectrum anthelmintic albendazole (ABZ), in combination with ivermectin (IVM) or diethylcarbamazine (DEC). Over the years, the use of both ABZ and IVM in the treatment of veterinary parasites has led to widespread anthelmintic resistance against these drugs. In this study, we genotyped microfilaria of Wuchereria bancrofti, a causative agent of LF, in order to detect the presence of mutations which confer ABZ resistance in other parasites, and we identified such mutations in worms obtained from untreated patients in Ghana and Burkina Faso, West Africa. Microfilaria from patients who had been treated with ABZ + IVM, had a significantly higher frequency of the resistant genotype, and this frequency was even higher in worms from patients that had received two rounds of treatment. In addition, the untreated population of microfilaria had an excess of homozygotes in the population. This excess homozygosity was equivalent to a Wright's Inbreeding Statistic of FIT= 0.44, and we found that the population was significantly subdivided between patients. In order to better understand the mechanisms and factors involved in the potential spread of ABZ resistance, caused by such mutations, through a population of Culex-transmitted W. bancrofti, we developed a deterministic model that incorporates genotype structure into the epidemiological model EPIFIL. This model predicts that the combination of ABZ + DEC leads to stronger selection for the resistant genotype than ABZ + IVM, and that drug efficacy assumptions are an important factor affecting the spread of drug resistance. Treatment coverage, non-random mating, initial allele frequency and number of treatments also had substantial impact on the speed and magnitude of the spread of ABZ resistance. When we expanded this model to include potential IVM-resistance alleles we found that, under ABZ + IVM treatment, selection for resistance to either drug is enhanced by the presence of resistance against the second drug. Similarly, excess homozygosity caused by parasite non-random mating may increase selection for a dominant IVM resistance allele through enhancing the spread of a recessive ABZ resistance allele. Resistence developed more slowly when it was inherited as a polygenic trait. Results from this study suggest that resistance monitoring is crucial, as resistance may not be apparent until treatment is stopped, recrudescence occurs and treatment is reapplied.
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Bailey, Michelle. "Core Promoter Function in Brugia malayi." Scholar Commons, 2010. http://scholarcommons.usf.edu/etd/3492.

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Previous studies have indicated that the promoters of the human filarial parasite Brugia malayi are unusual in that they do not exhibit the CAAT or TATAA sequences usually found in the core domains of promoters of most eukaryotic organisms. Analysis of the promoters of the ribosomal proteins showed that the region flanking the splice leader (SL) addition site plays an important role in transcription and may function as the core promoter domain in B. malayi. To test the hypothesis that the SL addition domain is the most important essential region of the ribosomal protein promoters, the SL addition site of the BmRPL13 gene was replaced with the SL addition domains from other ribosomal protein genes from B. malayi. The promoter activity of the replacement constructs were tested using a transient transfection dual luciferase assay. Promoter activity with RPL13 replacement constructs was correlated with that seen in the wild type promoters, suggesting that roughly 80% of the variations seen in promoter activity among ribosomal protein promoters is due to variation in the SL core promoter domain.
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Risse, Marcus [Verfasser]. "Zur Pathogenese der akuten Lymphangitis und Elephantiasis beim Pferd : eine histologische, immunhistochemische und transmissionselektronenmikroskopische Studie / vorgelegt von Marcus Risse." 2004. http://d-nb.info/972258418/34.

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Books on the topic "Elephantiasis"

1

B, Nutman Thomas, ed. Lymphatic filariasis. London: Imperial College Press, 2000.

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WHO Expert Committee on Filariasis. Lymphatic filariasis: the disease and its control: Fifth report of the WHO Expert Commmittee on Filariasis. Geneva: World Health Organization, 1992.

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3

Podoconiosis: Non-filarial elephantiasis. Oxford: Oxford University Press, 1990.

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(Editor), James N. Parker, and Philip Parker (Editor), eds. Elephantiasis A Medical Dictionary. Icon Health Publications, 2004.

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Liveing, Robert. Elephantiasis Graecorum Or, True Leprosy. Palmer Press, 2010.

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Saengtharatip, Seeviga. Human lymphatic filariases in Thailand: Analysis of microfilaremia distribution in human populations. 1995.

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7

Keratosen. Ichthyosis. Morbus Darier. Atrophien. Sclerodermie. Elephantiasis. Springer, 2031.

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Price, E. W. Podoconiosis: Non-filarial Elephantiasis (Oxford medical publications). Oxford University Press, USA, 1991.

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9

Sonntag, Erich. Krampfadern : (Einschliesslich Ekzem, Beingeschwür, Venenentzündung und Elephantiasis). de Gruyter GmbH, Walter, 2019.

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Keratosen; Ichthyosis; Morbus Darier; Atrophien; Sclerodermie; Elephantiasis. Springer London, Limited, 2013.

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Book chapters on the topic "Elephantiasis"

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Dunford, James C., Louis A. Somma, David Serrano, C. Roxanne Rutledge, John L. Capinera, Guy Smagghe, Eli Shaaya, et al. "Elephantiasis." In Encyclopedia of Entomology, 1297. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6359-6_3528.

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Gooch, Jan W. "Elephantiasis." In Encyclopedic Dictionary of Polymers, 889. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13631.

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Mehlhorn, Heinz. "Tropical Elephantiasis." In Encyclopedia of Parasitology, 2886. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_3294.

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Mehlhorn, Heinz. "Elephantiasis Tropica." In Encyclopedia of Parasitology, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-642-27769-6_1026-2.

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Mehlhorn, Heinz. "Tropical Elephantiasis." In Encyclopedia of Parasitology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_3294-2.

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Mehlhorn, Heinz. "Elephantiasis Tropica." In Encyclopedia of Parasitology, 881–83. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_1026.

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Du Port, François. "Treatment of Elephantiasis." In The Decade of Medicine or The Physician of the Rich and the Poor, 210–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73715-2_257.

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Chastain, Stephen L., and Bruce H. Gray. "Elephantiasis Nostras Verrucosa." In Atlas of Clinical Vascular Medicine, 94–95. Oxford, UK: Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118618189.ch47.

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Sodahlon, Yao, Mwele Malecela, and John O. Gyapong. "Lymphatic Filariasis (Elephantiasis)." In Neglected Tropical Diseases - Sub-Saharan Africa, 159–86. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-25471-5_8.

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Goel, Trilok Chandra, and Apul Goel. "Chronic Lymphedema–Elephantiasis." In Lymphatic Filariasis, 165–68. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-2257-9_20.

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Conference papers on the topic "Elephantiasis"

1

Bertram, Christopher D., Charles Macaskill, and James E. Moore. "Towards a Realistic Model of a Lymphatic Network: Improved Methods of Solution of the Equations for Many Lymphangions in Series." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14434.

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Lymphedema of the arm is a common complication of surgery involving removal of armpit lymph nodes. It is currently incurable, although it can be palliated by massage. Equivalent situations occur following lymphatic interference elsewhere in the body. In the developing world, lymphedema is also caused by elephantiasis.
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Parsons, Kevin D., Timothy Kassis, and J. Brandon Dixon. "Design of an In Vitro Migration Chamber for Quantifying the Homing Patterns of Parasitic Worms." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80711.

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Lymphatic filariasis, a parasitic disease often resulting in severe lymphatic dysfunction and lymphedema, is perpetuated by an invasion of worms, delivered through mosquito bites, that reside, mature, and reproduce in the human lymphatic system. The disease cycle begins with stage 3 larvae (L3) leaving the mosquito and penetrating the dermal layer of the human while the mosquito is feeding where it eventually makes its way to a collecting lymphatic vessel where it resides for its adult life (up to 10 years) [1]. While many infected individuals will remain asymptomatic, a subset of patients will develop reconstruction of the tissue structure and the extreme swelling of the arms, legs, genitals and/or breasts. This elephantiasis occurs in over 10 million people worldwide and has a harsh negative effect on the infected individual’s ability to work and function in society.
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