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Journal articles on the topic 'Effects of sleep'

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Published: 10 December 2022
Last updated: 29 January 2023

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1

Sprott, Richard L. "Sleep effects." Experimental Gerontology 26, no. 2-3 (January 1991): 215. http://dx.doi.org/10.1016/0531-5565(91)90013-c.

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2

Bonnet, Michael H. "Differentiating sleep continuity effects from sleep stage effects." Journal of Sleep Research 9, no. 4 (December 18, 2000): 403–4. http://dx.doi.org/10.1046/j.1365-2869.2000.0215a.x.

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3

Sundelin, Tina, Mats Lekander, Kimmo Sorjonen, and John Axelsson. "Negative effects of restricted sleep on facial appearance and social appeal." Royal Society Open Science 4, no. 5 (May 2017): 160918. http://dx.doi.org/10.1098/rsos.160918.

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The importance of assessing evolutionarily relevant social cues suggests that humans should be sensitive to others' sleep history, as this may indicate something about their health as well as their capacity for social interaction. Recent findings show that acute sleep deprivation and looking tired are related to decreased attractiveness and health, as perceived by others. This suggests that one might also avoid contact with sleep-deprived, or sleepy-looking, individuals, as a strategy to reduce health risk and poor interactions. In this study, 25 participants (14 females, age range 18–47 years) were photographed after 2 days of sleep restriction and after normal sleep, in a balanced design. The photographs were rated by 122 raters (65 females, age range 18–65 years) on how much they would like to socialize with the participants. They also rated participants' attractiveness, health, sleepiness and trustworthiness. The results show that raters were less inclined to socialize with individuals who had gotten insufficient sleep. Furthermore, when sleep-restricted, participants were perceived as less attractive, less healthy and more sleepy. There was no difference in perceived trustworthiness. These findings suggest that naturalistic sleep loss can be detected in a face and that people are less inclined to interact with a sleep-deprived individual.
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4

Bonnet, M. H. "Cognitive Effects of Sleep and Sleep Fragmentation." Sleep 16, suppl_8 (December 1993): S65—S67. http://dx.doi.org/10.1093/sleep/16.suppl_8.s65.

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5

Cavaglia, F., D. Pires-Barreira, L. Paula, A. Matos-Pires, and F. Arriaga. "Sleep deprivation effects on subjective sleep clomplaints." European Psychiatry 17 (May 2002): 209. http://dx.doi.org/10.1016/s0924-9338(02)80892-6.

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6

Ebrahim, Irshaad O., Colin M. Shapiro, Adrian J. Williams, and Peter B. Fenwick. "Alcohol and Sleep I: Effects on Normal Sleep." Alcoholism: Clinical and Experimental Research 37, no. 4 (January 24, 2013): 539`—549. http://dx.doi.org/10.1111/acer.12006.

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7

Holsboer-Trachsler, E., U. Hemmeter, and E. Seifritz. "Sleep-EEG and neuroendocrine effects of sleep deprivation." Biological Psychiatry 42, no. 1 (July 1997): 8S—9S. http://dx.doi.org/10.1016/s0006-3223(97)86895-3.

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8

Bonnet, Michael H., and Donna L. Arand. "Clinical effects of sleep fragmentation versus sleep deprivation." Sleep Medicine Reviews 7, no. 4 (August 2003): 297–310. http://dx.doi.org/10.1053/smrv.2001.0245.

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9

Rimmele, Ulrike, and Arielle Tambini. "Sleep, Sleep Alterations, Stress—Combined Effects on Memory?" Sleep 38, no. 12 (December 2015): 1835–36. http://dx.doi.org/10.5665/sleep.5214.

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10

Pagel, J. F. "MEDICATION EFFECTS ON SLEEP." Dental Clinics of North America 45, no. 4 (October 2001): 855–65. http://dx.doi.org/10.1016/s0011-8532(22)00496-7.

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11

Lindseth, Glenda, Paul Lindseth, and Mark Thompson. "Nutritional Effects on Sleep." Western Journal of Nursing Research 35, no. 4 (August 4, 2011): 497–513. http://dx.doi.org/10.1177/0193945911416379.

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12

Roux, Francoise J., and Meir H. Kryger. "Medication Effects on Sleep." Clinics in Chest Medicine 31, no. 2 (June 2010): 397–405. http://dx.doi.org/10.1016/j.ccm.2010.02.008.

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13

Panagiotou, Irene, and Kyriaki Mystakidou. "Non-Analgesic Effects of Opioids: Opioids’ Effects on Sleep (Including Sleep Apnea)." Current Pharmaceutical Design 18, no. 37 (October 18, 2012): 6025–33. http://dx.doi.org/10.2174/138161212803582450.

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14

Gupta, PD. "Menstrual cycle effects on sleep." Clinical Journal of Obstetrics and Gynecology 5, no. 2 (April 5, 2022): 042–43. http://dx.doi.org/10.29328/journal.cjog.1001105.

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Sleep and menstrual cycle both are normal physiological processes in women’s life but they are regulated by different centers. Sleep is a daily rhythm whereas the menstrual cycle lasts for 28 days. During this period the estrogen peaks twice. We have shown earlier that there is an inverse relationship between estrogen and the hormone melatonin which aids sleep. Because of this menstruating women will have sleep disorders.
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15

Endo, Takuro, Corinne Roth, Hans-Peter Landolt, Esther Werth, Daniel Aeschbach, Peter Achermann, and Alexander A. Borbély. "Selective REM sleep deprivation in humans: effects on sleep and sleep EEG." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 4 (April 1, 1998): R1186—R1194. http://dx.doi.org/10.1152/ajpregu.1998.274.4.r1186.

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To investigate rapid eye movement (REM) sleep regulation, eight healthy young men were deprived of REM sleep for three consecutive nights. In a three-night control sleep deprivation (CD) session 2 wk later, the subjects were repeatedly awakened from non-REM sleep in an attempt to match the awakenings during the REM sleep deprivation (RD) nights. During the RD nights the number of sleep interruptions required to prevent REM sleep increased within and across consecutive nights. REM sleep was reduced to 9.2% of baseline (CD nights: 80.7%) and rose to 140.1% in the first recovery night. RD gave rise to changes in the EEG power spectra of REM sleep. Power in the 8.25- to 11-Hz range was reduced in the first recovery night, an effect that gradually subsided but was still present in the third recovery night. The rising REM sleep propensity, as reflected by the increase of interventions within and across RD nights, and the moderate REM sleep rebound during recovery can be accounted for by a compensatory response that serves REM sleep homeostasis. The changes in the electroencephalogram power spectra, which were observed during enhanced REM sleep propensity, may be a sign of an altered quality of REM sleep.
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16

Frank, Marcos G., Roger Morrissette, and H. Craig Heller. "Effects of sleep deprivation in neonatal rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 275, no. 1 (July 1, 1998): R148—R157. http://dx.doi.org/10.1152/ajpregu.1998.275.1.r148.

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This investigation represents the first systematic study of sleep homeostasis in developing mammals that spans the preweaning and postweaning periods. Neonatal rats from 12 to 24 days of postnatal life ( P12– P24) were anesthetized with Metofane (methoxyflurane) and implanted with miniaturized electroencephalographic (EEG) and electromyographic electrodes. After 48 h of recovery, neonatal rats were sleep deprived for 3 h by either gentle handling or forced locomotion. We find that 3-h sleep deprivation produces dramatically different compensatory responses at different stages of postnatal development. In striking contrast to adult rats, sleep deprivation does not increase slow-wave sleep EEG delta (0.5–4.0 Hz) activity in rats younger than P24. However, P12– P20rats do show evidence of sleep regulation because they show compensatory increases in sleep time and sleep continuity during recovery. In P12 rats, ∼90% of total slow wave sleep time lost during the sleep-deprivation period was recovered during subsequent sleep. A similar recovery of active sleep time was observed in P20– P24rats. These findings suggest not only that sleep is regulated in neonatal rats but that the accumulation and/or discharge of sleep need changes dramatically between the third and fourth postnatal weeks.
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17

Rainville, G., and Cheryl Lampkin. "ELECTRONIC USE AND SLEEP, SURPRISING BEDFELLOWS: RESTFULNESS EFFECTS OF ELECTRONICS USE PRIOR TO BEDTIME FOR THE 40-PLUS." Innovation in Aging 3, Supplement_1 (November 2019): S526. http://dx.doi.org/10.1093/geroni/igz038.1936.

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Abstract Getting restful sleep is essential to well-being but stress and poor sleep habits may make sleeping through the night challenging. This research explored life event stressors and pre-sleep activities among 2,464 randomly selected Americans age 40 and older (using Ipsos’ KnowledgeNetwork panel) to determine their joint effects on mental well-being. Respondents reported how often they engaged in twelve individual behaviors within an hour of going to sleep. These behaviors (found to be inter-correlated) were combined using EFA into four factors representing levels of engagement in each of four classes of pre-sleep activities: pre-sleep electronics use (e.g. texting/e-mail before bed), deep relaxation activities, reliance on sleep-aids, and “nightowl” behaviors (i.e., snacking). Counter to expectations, only electronics use had significant conditional effects on the path between a life events stressor index (a count of current, potentially stressful life events) and scores on the positively-framed Warwick Edinburgh well-being scale (WEMWBS). How often one sleeps through the night also had unexpected effects in a conditional path analysis. A somewhat-involved relationship emerges between each of the theoretically-relevant measures. First, the negative impact of stress is moderated by sleeping through the night. Sleeping through the night is, counter to previous studies on electronics use and sleep, mediated by the use of electronics prior to sleep. We propose that mechanisms (such as the nature of backlighting used in electronics) that hamper restfulness may be offset by relaxation effects or by setting one’s ducks in a row by texting/emailing before going to sleep.
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18

DeMartinis, Nicholas, and Andrew Winokur. "Effects of Psychiatric Medications on Sleep and Sleep Disorders." CNS & Neurological Disorders - Drug Targets 6, no. 1 (February 1, 2007): 17–29. http://dx.doi.org/10.2174/187152707779940835.

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19

Mulrooney, Thomas F. "Sleep Deprivation: Clinical Issues, Pharmacology, and Sleep Loss Effects." Annals of Internal Medicine 142, no. 6 (March 15, 2005): 480. http://dx.doi.org/10.7326/0003-4819-142-6-200503150-00031.

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20

Kelly, T. L., M. M. Mitler, and M. H. Bonnet. "Sleep latency measures of caffeine effects during sleep deprivation." Electroencephalography and Clinical Neurophysiology 102, no. 5 (May 1997): 397–400. http://dx.doi.org/10.1016/s0921-884x(97)96135-x.

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21

Thomas, Karen A., and Shuyuann Wang Foreman. "Infant Sleep and Feeding Pattern: Effects on Maternal Sleep." Journal of Midwifery & Women's Health 50, no. 5 (September 10, 2005): 399–404. http://dx.doi.org/10.1016/j.jmwh.2005.04.010.

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22

Hayashi, Mitsuo, and Hiroshi Ogino. "The effects of sleep effort on sleep onset process." Proceedings of the Annual Convention of the Japanese Psychological Association 83 (September 11, 2019): 3D—023–3D—023. http://dx.doi.org/10.4992/pacjpa.83.0_3d-023.

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23

Lee, Kathryn A. "Sleep Deprivation: Clinical Issues, Pharmacology, and Sleep Loss Effects." Sleep 28, no. 6 (June 2005): 769. http://dx.doi.org/10.1093/sleep/28.6.769.

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24

Zolfaghari, Sheida, Chun Yao, Cynthia Thompson, Nadia Gosselin, Alex Desautels, Thien Thanh Dang-Vu, Ronald B. Postuma, and Julie Carrier. "Effects of menopause on sleep quality and sleep disorders." Menopause 27, no. 3 (March 2020): 295–304. http://dx.doi.org/10.1097/gme.0000000000001462.

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25

Penetar, D. M., H. C. Sing, D. R. Thorne, M. L. Thomas, J. B. Fertig, A. S. D. Schelling, J. C. Sealock, P. A. Newhouse, and G. Belenky. "Amphetamine effects on recovery sleep following total sleep deprivation." Human Psychopharmacology: Clinical and Experimental 6, no. 4 (December 1991): 319–23. http://dx.doi.org/10.1002/hup.470060409.

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26

Åkerstedt, Torbjörn, Ken Hume, David Minors, and Jim Waterhouse. "Experimental separation of time of day and homeostatic influences on sleep." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 4 (April 1, 1998): R1162—R1168. http://dx.doi.org/10.1152/ajpregu.1998.274.4.r1162.

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The purpose of the present study was to evaluate the simultaneous effects on sleep of prior time awake (PRW) and time of day (TOD). Eight male subjects spent 13 days in an isolated sleep lab and had three 8-h baseline sleeps and then 18 4-h sleeps, distributed to provide three sleeps starting at 2400, 0400, 0800, 1200, 1600, and 2000. The three sleeps were preceded by 4, 8, and 12 h of PRW, respectively. ANOVA showed that TST and subjective sleepiness increased with PRW and with closeness to the trough of the circadian rhythm of rectal temperature, whereas sleep latency showed the opposite pattern, and rapid eye movement sleep (REM) latency strongly decreased with PRW and with closeness to the trough. Slow-wave sleep (SWS) increased with PRW, whereas SWS latency and final time awake decreased. REM sleep increased with closeness to the circadian trough, and time awake decreased. Multiple-regression analysis showed that REM latency was closely related to increased SWS in the first sleep cycle, reduced SWS latency, and increased PRW [a short PRW before sleep at noon yielded an extremely short (14 min) REM latency]. Sleep latency and final time awake showed almost exactly the same relationship to TOD and PRW. It is concluded that both homeostatic and circadian influences simultaneously affect sleep, that REM latency is very sensitive to the need for SWS, and that the circadian acrophase strongly interferes with sleep. It should be emphasized that the conclusions should not be extrapolated to longer (>12 h) wake spans.
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27

Block, Sandra. "The Sleepy Student: The Effects of Sleep Disorders in Students." NASNewsletter 21, no. 6 (November 2006): 13–14. http://dx.doi.org/10.1177/104747570602100605.

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28

Kundermann, Bernd, Jürgen-Christian Krieg, Wolfgang Schreiber, and Stefan Lautenbacher. "The Effects of Sleep Deprivation on Pain." Pain Research and Management 9, no. 1 (2004): 25–32. http://dx.doi.org/10.1155/2004/949187.

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Chronic pain syndromes are associated with alterations in sleep continuity and sleep architecture. One perspective of this relationship, which has not received much attention to date, is that disturbances of sleep affect pain. To fathom this direction of cause, experimental human and animal studies on the effects of sleep deprivation on pain processing were reviewed. According to the majority of the studies, sleep deprivation produces hyperalgesic changes. Furthermore, sleep deprivation can counteract analgesic effects of pharmacological treatments involving opioidergic and serotoninergic mechanisms of action. The heterogeneity of the human data and the exclusive interest in rapid eye movement sleep deprivation in animals so far do not allow us to draw firm conclusions as to whether the hyperalgesic effects are due to the deprivation of specific sleep stages or whether they result from a generalized disruption of sleep continuity. The significance of opioidergic and serotoninergic processes as mediating mechanisms of the hyperalgesic changes produced by sleep deprivation are discussed.
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29

Pal, Dinesh, William J. Lipinski, Amanda J. Walker, Ashley M. Turner, and George A. Mashour. "State-specific Effects of Sevoflurane Anesthesia on Sleep Homeostasis." Anesthesiology 114, no. 2 (February 1, 2011): 302–10. http://dx.doi.org/10.1097/aln.0b013e318204e064.

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Background Prolonged propofol administration does not result in signs of sleep deprivation, and propofol anesthesia appears to satisfy the homeostatic need for both rapid eye movement (REM) and non-REM (NREM) sleep. In the current study, the effects of sevoflurane on recovery from total sleep deprivation were investigated. Methods Ten male rats were instrumented for electrophysiologic recordings under three conditions: (1) 36-h ad libitum sleep; (2) 12-h sleep deprivation followed by 24-h ad libitum sleep; and (3) 12-h sleep deprivation, followed by 6-h sevoflurane exposure, followed by 18-h ad libitum sleep. The percentage of waking, NREM sleep, and REM sleep, as well as NREM sleep δ power, were calculated and compared for all three conditions. Results Total sleep deprivation resulted in significantly increased NREM and REM sleep for 12-h postdeprivation. Sevoflurane exposure after deprivation eliminated the homeostatic increase in NREM sleep and produced a significant decrease in the NREM sleep δ power during the postanesthetic period, indicating a complete recovery from the effects of deprivation. However, sevoflurane did not affect the time course of REM sleep recovery, which required 12 h after deprivation and anesthetic exposure. Conclusion Unlike propofol, sevoflurane anesthesia has differential effects on NREM and REM sleep homeostasis. These data confirm the previous hypothesis that inhalational agents do not satisfy the homeostatic need for REM sleep, and that the relationship between sleep and anesthesia is likely to be agent and state specific.
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30

Vyas, Umesh Kumar. "Effects of quetiapine on sleep." Indian Journal of Sleep Medicine 10, no. 2 (2015): 70–73. http://dx.doi.org/10.5958/0974-0155.2015.00010.8.

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31

Weiss, Margaret D. "73.3 Stimulant Effects on Sleep." Journal of the American Academy of Child & Adolescent Psychiatry 61, no. 10 (October 2022): S102—S103. http://dx.doi.org/10.1016/j.jaac.2022.07.419.

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32

Vyas, Umesh Kumar. "Effects of quetiapine on sleep." Indian Journal of Sleep Medicine 10, no. 2 (2015): 70–73. http://dx.doi.org/10.5005/ijsm-10-2-70.

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33

Steiger, A., I. A. Antonijevic, S. Bohlhalter, R. M. Frieboes, E. Friess, and H. Murck. "Effects of Hormones on Sleep." Hormone Research in Paediatrics 49, no. 3-4 (1998): 125–30. http://dx.doi.org/10.1159/000023158.

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34

Taskar, Varsha, and Max Hirshkowitz. "Health Effects of Sleep Deprivation." Clinical Pulmonary Medicine 10, no. 1 (January 2003): 47–52. http://dx.doi.org/10.1097/00045413-200301000-00008.

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35

Parish, James M., and John W. Shepard. "Cardiovascular Effects of Sleep Disorders." Chest 97, no. 5 (May 1990): 1220–26. http://dx.doi.org/10.1378/chest.97.5.1220.

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36

Obermeyer, William H., and Ruth M. Benca. "EFFECTS OF DRUGS ON SLEEP." Otolaryngologic Clinics of North America 32, no. 2 (September 1999): 289–302. http://dx.doi.org/10.1016/s0030-6665(05)70131-6.

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37

Obermeyer, William H., and Ruth M. Benca. "EFFECTS OF DRUGS ON SLEEP." Neurologic Clinics 14, no. 4 (November 1996): 827–40. http://dx.doi.org/10.1016/s0733-8619(05)70287-5.

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38

Moser, Neal J., Barbara A. Phillips, Gordon Guthrie, and Gary Barnett. "Effects of Dexamethasone on Sleep." Pharmacology & Toxicology 79, no. 2 (August 1996): 100–102. http://dx.doi.org/10.1111/j.1600-0773.1996.tb00249.x.

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39

Dodson, Katherine J. "Cardiovascular Effects of Sleep Apnea." Journal for Nurse Practitioners 4, no. 6 (June 2008): 439–44. http://dx.doi.org/10.1016/j.nurpra.2008.02.017.

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40

LEARD-HANSSON, JAN, and LAURENCE GUTTMACHER. "Neurocognitive Effects of Sleep Apnea." Clinical Psychiatry News 35, no. 6 (June 2007): 48. http://dx.doi.org/10.1016/s0270-6644(07)70387-9.

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41

Hellwig, Jennifer. "Health Effects of Sleep Routines." Nursing for Women's Health 20, no. 1 (February 2016): 15. http://dx.doi.org/10.1016/s1751-4851(16)00034-9.

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42

Youngstedt, Shawn D. "Effects of Exercise on Sleep." Clinics in Sports Medicine 24, no. 2 (April 2005): 355–65. http://dx.doi.org/10.1016/j.csm.2004.12.003.

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43

Baker, Fiona C., and Kathryn Aldrich Lee. "Menstrual Cycle Effects on Sleep." Sleep Medicine Clinics 13, no. 3 (September 2018): 283–94. http://dx.doi.org/10.1016/j.jsmc.2018.04.002.

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44

Trambowicz, K., P. Gorzelak-Pabiś, and M. Broncel. "Statins And Sleep – Clinical Effects." Atherosclerosis 287 (August 2019): e202. http://dx.doi.org/10.1016/j.atherosclerosis.2019.06.613.

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45

VAN DEN HOOFDAKKER, R. H., A. L. BOUHUYS, and D. GM BEERSMA. "ANTIDEPRESSANT EFFECTS OF SLEEP DEPRIVATION." Behavioural Pharmacology 3, Supplement (April 1992): 5. http://dx.doi.org/10.1097/00008877-199204001-00010.

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46

Ansfield, Matthew E., Daniel M. Wegner, and Robin Bowser. "Ironic effects of sleep urgency." Behaviour Research and Therapy 34, no. 7 (July 1996): 523–31. http://dx.doi.org/10.1016/0005-7967(96)00031-9.

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47

Ueno, Ryuji, Hirotaka Onoe, Osamu Hayaishi, Ichiro Fujita, Hitoo Nishino, and Yutaka Oomura. "Sleep inducing effects of PGD2." Neuroscience Research Supplements 5 (January 1987): S173. http://dx.doi.org/10.1016/0921-8696(87)90357-4.

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48

Zhao, Mingxia, Houzhen Tuo, Shuhui Wang, and Lin Zhao. "The Effects of Dietary Nutrition on Sleep and Sleep Disorders." Mediators of Inflammation 2020 (June 25, 2020): 1–7. http://dx.doi.org/10.1155/2020/3142874.

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Sleep disorder significantly affects the life quality of a large number of people but is still an underrecognized disease. Dietary nutrition is believed to play a significant impact on sleeping wellness. Many nutritional supplements have been used trying to benefit sleep wellness. However, the relationship between nutritional components and sleep is complicated. Nutritional factors vary dramatically with different diet patterns and depend significantly on the digestive and metabiotic functions of each individual. Moreover, nutrition can profoundly affect the hormones and inflammation status which directly or indirectly contribute to insomnia. In this review, we summarized the role of major nutritional factors, carbohydrates, lipids, amino acids, and vitamins on sleep and sleep disorders and discussed the potential mechanisms.
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49

Burchakov, Denis Igorevich. "Сircadian rhythm and metabolic effects of melatonin." Obesity and metabolism 12, no. 1 (February 13, 2015): 46–51. http://dx.doi.org/10.14341/omet2015146-51.

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Sleep is a highly important process, required for normal organ and system function. Researchers assume, that during sleep brain shifts to internal body signals. Therefore, any sleep disturbance will disrupt health. Industrial and post-industrial society links high stress level and sleep problems. Excess light stimulation in living space, including bedroom, disorganize circadian rhythm of melatonin. Besides regulation this hormone has antioxidant and adaptogen functions. From psychological standpoint the same high-stress social context depletes the adaptation resources. To normalize sleep function we can utilize both sleep hygiene measures and modern pharmacotherapy. There are melatonin-based drugs, which help to restore sleep-wake cycle, augment adaptive capability and in some cases empower the existing treatment for specific somatic maladies. From a clinical and chronobiological standpoint melatonin is useful in broad spectrum of disorders.
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50

McNamara, Frances, and Colin E. Sullivan. "Sleep-Disordered Breathing and Its Effects on Sleep in Infants." Sleep 19, no. 1 (January 1996): 4–12. http://dx.doi.org/10.1093/sleep/19.1.4.

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