Academic literature on the topic 'EDHF'

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Journal articles on the topic "EDHF"

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Hasunuma, K., T. Yamaguchi, D. M. Rodman, R. F. O'Brien, and I. F. McMurtry. "Effects of inhibitors of EDRF and EDHF on vasoreactivity of perfused rat lungs." American Journal of Physiology-Lung Cellular and Molecular Physiology 260, no. 2 (February 1, 1991): L97—L104. http://dx.doi.org/10.1152/ajplung.1991.260.2.l97.

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Recent studies indicate that the endothelium of isolated rat pulmonary arteries releases two different factors, endothelium-derived relaxing factor (EDRF) and hyperpolarizing factor (EDHF), which participate in histamine- and acetylcholine-induced relaxation. There is evidence for EDRF vasoreactivity in perfused lungs, but a role for EDHF, which hyperpolarizes vascular smooth muscle by activating membrane K+ channels, has not been reported. We used the inhibitors of EDRF, 20 microM hemoglobin, 200 microM NG-mono-methyl-L-arginine, and 2 mM L-canavanine, the nonselective blocker of K+ channels,
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Park, Yoonjung, Stefano Capobianco, Xue Gao, John R. Falck, Kevin C. Dellsperger, and Cuihua Zhang. "Role of EDHF in type 2 diabetes-induced endothelial dysfunction." American Journal of Physiology-Heart and Circulatory Physiology 295, no. 5 (November 2008): H1982—H1988. http://dx.doi.org/10.1152/ajpheart.01261.2007.

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Endothelium-derived hyperpolarizing factor (EDHF) plays a crucial role in modulating vasomotor tone, especially in microvessels when nitric oxide-dependent control is compromised such as in diabetes. Epoxyeicosatrienoic acids (EETs), potassium ions (K+), and hydrogen peroxide (H2O2) are proposed as EDHFs. However, the identity (or identities) of EDHF-dependent endothelial dilators has not been clearly elucidated in diabetes. We assessed the mechanisms of EDHF-induced vasodilation in wild-type (WT, normal), db/db (advanced type 2 diabetic) mice, and db/db mice null for TNF (dbTNF−/dbTNF−). In d
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Wang, Xuemei, Greg Trottier, and Rodger Loutzenhiser. "Determinants of renal afferent arteriolar actions of bradykinin: evidence that multiple pathways mediate responses attributed to EDHF." American Journal of Physiology-Renal Physiology 285, no. 3 (September 2003): F540—F549. http://dx.doi.org/10.1152/ajprenal.00127.2003.

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The determinants of bradykinin (BK)-induced afferent arteriolar vasodilation were investigated in the in vitro perfused hydronephrotic rat kidney. BK elicited a concentration-dependent vasodilation of afferent arterioles that had been preconstricted with ANG II (0.1 nmol/l), but this dilation was transient in character. Pretreatment with the nitric oxide synthase inhibitor Nω-nitro-l-arginine methyl ester (100 μmol/l) and the cyclooxygenase inhibitor ibuprofen (10 μmol/l) did not prevent this dilation when tone was established by ANG II but fully blocked the response when tone was established
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Luksha, Leanid, Henry Nisell, Natallia Luksha, Marius Kublickas, Kjell Hultenby, and Karolina Kublickiene. "Endothelium-derived hyperpolarizing factor in preeclampsia: heterogeneous contribution, mechanisms, and morphological prerequisites." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 294, no. 2 (February 2008): R510—R519. http://dx.doi.org/10.1152/ajpregu.00458.2007.

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We hypothesized that in preeclampsia (PE), contribution of endothelium-derived hyperpolarizing factor (EDHF) and the mechanism/s of its action differ from that in normal pregnancy (NP). We aimed to assess endothelial function and morphology in arteries from NP and PE with particular focus on EDHF. Arteries (≈200 μm) were dissected from subcutaneous fat biopsies obtained from women undergoing cesarean section. With the use of wire myography, responses to the endothelium-dependent agonist bradykinin (BK) were determined before and after inhibition of pathways relevant to EDHF activity. The overa
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Hayakawa, H., Y. Hirata, E. Suzuki, T. Sugimoto, H. Matsuoka, K. Kikuchi, T. Nagano, M. Hirobe, and T. Sugimoto. "Mechanisms for altered endothelium-dependent vasorelaxation in isolated kidneys from experimental hypertensive rats." American Journal of Physiology-Heart and Circulatory Physiology 264, no. 5 (May 1, 1993): H1535—H1541. http://dx.doi.org/10.1152/ajpheart.1993.264.5.h1535.

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To study mechanisms for attenuated endothelium-dependent vasorelaxation in hypertension, we examined the effects of acetylcholine (ACh) on renal vascular resistance (RVR) and release rates of endothelium-derived relaxing factor (EDRF) in kidneys isolated from spontaneously hypertensive rats (SHR), deoxycorticosterone acetate (DOCA) salt-hypertensive (DOCA salt) rats, and Dahl salt-sensitive (Dahl S) rats. Decreases in RVR by ACh were smaller in hypertensive rats than in their normotensive controls. The release rate of nitric oxide into the perfusate, which was estimated using nitrite-nitrate a
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You, Junping, Elke M. Golding, and Robert M. Bryan. "Arachidonic acid metabolites, hydrogen peroxide, and EDHF in cerebral arteries." American Journal of Physiology-Heart and Circulatory Physiology 289, no. 3 (September 2005): H1077—H1083. http://dx.doi.org/10.1152/ajpheart.01046.2004.

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We tested the hypotheses that EDHF in rat middle cerebral arteries (MCAs) involves 1) metabolism of arachidonic acid through the epoxygenase pathway, 2) metabolism of arachidonic acid through the lipoxygenase pathway, or 3) reactive oxygen species. EDHF-mediated dilations were elicited in isolated and pressurized rat MCAs by activation of endothelial P2Y2 receptors with either UTP or ATP. All studies were conducted after the inhibition of nitric oxide synthase and cyclooxygenase with Nω-nitro-l-arginine methyl ester (10 μM) and indomethacin (10 μM), respectively. The inhibition of epoxygenase
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You, Junping, Sean P. Marrelli, and Robert M. Bryan. "Role of Cytoplasmic Phospholipase A2 in Endothelium-Derived Hyperpolarizing Factor Dilations of Rat Middle Cerebral Arteries." Journal of Cerebral Blood Flow & Metabolism 22, no. 10 (October 2002): 1239–47. http://dx.doi.org/10.1097/01.wcb.0000037996.34930.2e.

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Very little is known regarding the mechanism of action for the endothelium-derived hyperpolarizing factor (EDHF) response in cerebral vessels. The authors tested two hypotheses: (1) activation of the cytoplasmic form of phospholipase A2 (cPLA2) is involved with EDHF-mediated dilations in rat middle cerebral arteries; and (2) activation of the cPLA2 involves an increase in endothelial Ca2+ through activation of phospholipase C. Middle cerebral arteries were isolated from the rat, pressurized to 85 mm Hg, and luminally perfused. The EDHF response was elicited by luminal application of uridine tr
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Chilian, William M., and Ryoji Koshida. "EDHF and NO." Circulation Research 89, no. 8 (October 12, 2001): 648–49. http://dx.doi.org/10.1161/res.89.8.648.

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Félétou, Michel, and Paul M. Vanhoutte. "EDHF: an update." Clinical Science 117, no. 4 (July 16, 2009): 139–55. http://dx.doi.org/10.1042/cs20090096.

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The endothelium controls vascular tone not only by releasing NO and prostacyclin, but also by other pathways causing hyperpolarization of the underlying smooth muscle cells. This characteristic was at the origin of the term ‘endothelium-derived hyperpolarizing factor’ (EDHF). However, this acronym includes different mechanisms. Arachidonic acid metabolites derived from the cyclo-oxygenases, lipoxygenases and cytochrome P450 pathways, H2O2, CO, H2S and various peptides can be released by endothelial cells. These factors activate different families of K+ channels and hyperpolarization of the vas
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Félétou, Michel, and Paul M. Vanhoutte. "The Alternative: EDHF." Journal of Molecular and Cellular Cardiology 31, no. 1 (January 1999): 15–22. http://dx.doi.org/10.1006/jmcc.1998.0840.

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Dissertations / Theses on the topic "EDHF"

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Höpfl, Bernd. "EDHF initiiert fortgeleitete Gefäßdilatationen in Arteriolen des Skelettmuskels." Diss., lmu, 2005. http://nbn-resolving.de/urn:nbn:de:bvb:19-31718.

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Schach, Christian Oliver. "Mechanismus der EDHF-vermittelten Gefässdilatation der Arteria Interlobaris Suis /." Frankfurt a.M, 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000254594.

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Burnette, Ethan Williams. "Endothelium-derived hyperpolarizing factor (EDHF) in rat mesenteric artery." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ66130.pdf.

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Charisis, Ioannis. "Role of calcium-dependent potassium channels and mitochondria in the EDHF phenomenon." Thesis, Cardiff University, 2008. http://orca.cf.ac.uk/55773/.

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Background Acetylcholine (ACh) and the calcium ionophore A23187 are both known to trigger EDHF-type responses in the rabbit iliac artery via endothelial cell hyperpolarization mediated by the opening of calcium-dependent potassium channels (Kca). In addition, ACh and A23187 also stimulate the release of hydrogen peroxide (H2O2) from the endothelium. Aims of study 1) To determine the relative contribution of different Kca channel subtypes to ACh- and A23187-evoked responses. 2) To determine whether there is a connection between the activation of endothelial Kca channels and the release of H2O2.
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Harris, David. "Pharmacological studies on EDHF and anandamide in the rat mesenteric arterial bed." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366599.

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Chauhan, Sharmila Deepa. "Mechanisms of endothelium-dependant dilation : a study of EDHF and endothelial dysfunction." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397184.

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Mather, Simon. "Endothelial cell modulation of vascular tone by EDHF and gap junctional communication." Thesis, University of Bath, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.418594.

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Young, Elisa, and elisayoung@iprimus com au. "Endothelial dysfunction in insulin resistance: The role of EDHF and gap junction communication." RMIT University. Medical Sciences, 2007. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080110.162249.

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Background: Endothelial dysfunction is a key factor in the development of vascular complications in insulin resistance and diabetes and recent studies have established that endothelium-derived hyperpolarising factor (EDHF) plays an important physiological role in endothelium-derived relaxation responses, especially in small arteries and arterioles. Objective: This project aimed to identify the role of, and characterise, EDHF in animal models of insulin resistance, including the obese Zucker rat (OZR) as well as the fructose-fed (FF) Sprague-Dawley rat. Methods: Vascular function was st
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Neininger, Michael. "Zelluläre Mechanismen der Dilatation isolierter Widerstandsarterien durch NO-Donatoren und den Endothelabhängigen Hyperpolarisierenden Faktor EDHF." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-27038.

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Riexinger, Tobias. "Ein Cytochrom P450 2C8/9-abhängig gebildeter EDHF hyperpolarisiert Thrombozyten und hemmt deren Adhäsion an Endothelzellen." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-89394.

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Books on the topic "EDHF"

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Feletou, Michel. EDHF: The complete story. Boca Raton, FL: CRC/Taylor & Francis, 2006.

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Aic, Aʻvān Ke. Edhi. 2nd ed. Karachi: Mehran Press International, 1987.

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Ymeri, Hysen. Stërvitëm edhe Adem Jasharin. Tiranë: Emal, 2009.

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Pëllumbi, Servet. Edhe politika do moral. Tiranë: Rinia & ISPS, 2005.

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Ndreko, Merita. "--edhe gjysma e Botës". Tiranë: Shtëpia Botuese "Dora d'Istria", 2005.

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Kraja, Mehmet. Edhe të çmendurit fluturojnë: Roman. Tiranë: Onufri, 2004.

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Kraja, Mehmet. Edhe të çmendurit fluturojnë: Roman. Tiranë: Onufri, 2012.

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Benzo, Raúl. El umbral de Edaf: Cuentos. Montevideo: Ediciones de la Banda Oriental, 1990.

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Dashi, Kujtim. Edhe një gotë Filipit: Tregime. Tiranë: Botimet Toena, 1998.

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Beqja, Hamit. Edhe engjëlli edhe djalli janë brenda te i gjalli: Rreth dyzimit të personalitetit njerëzor. Tiranë: Albinform, 1995.

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Book chapters on the topic "EDHF"

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"References." In EDHF, 177–277. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.bmatt.

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"The Blood Vessel Wall." In EDHF, 1–38. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch1.

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"Endothelium-Derived Mediators." In EDHF, 39–53. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch2.

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"Endothelium-Dependent Hyperpolarizations." In EDHF, 55–132. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch3.

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"EDHF and Endothelial Dysfunction." In EDHF, 145–60. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch5.

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"EDHF and Therapeutic Interventions." In EDHF, 161–72. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch6.

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"Conclusions." In EDHF, 173–75. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch7.

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"EDHF and the Physiological Control of Blood Flow." In EDHF, 133–44. CRC Press, 2005. http://dx.doi.org/10.1201/9781420023312.ch4.

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"Endothelium and smooth muscle pathways for conduction along resistance microvessels." In Edhf 2000, 50–59. CRC Press, 2003. http://dx.doi.org/10.1201/b12802-10.

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"Membrane potential and calcium responses evoked by acetylcholine in submucosal arterioles of the guinea-pig small intestine." In Edhf 2000, 60–68. CRC Press, 2003. http://dx.doi.org/10.1201/b12802-11.

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Conference papers on the topic "EDHF"

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Orie, Nelson, Aysha Bakhamis, Moataz Bashah, Mohammed Alsayrafi, and Vidya Mohamed Ali. "EDHF Contribution To Microvascular Dilatation Is Not Linked To Endothelial Dysfunction In Morbidly Obese Qataris." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2014. http://dx.doi.org/10.5339/qfarc.2014.hbpp0360.

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Lau, Rachel Y. W., and Hilary J. Kahn. "Information modelling of EDIF." In the 30th international. New York, New York, USA: ACM Press, 1993. http://dx.doi.org/10.1145/157485.164892.

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Griffith, T. M., D. H. Edwards, R. L. Davies, T. J. Harrison, and K. L. Evans. "EDRF AND RESISTANCE VESSELS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643721.

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The influence of endothelium on vasomotionin resistance vessels was studied in an isolated, buffer-perfused rabbit ear preparation using novel microangiographic techniques and haemoglobin as a specific inhibitor of EDRF. In constricted preparations acetylcholine and Substance P, whose action is EDRF-dependent in large vessels, induceddilatation in vessels down to 25um in diameter which was inhibited by haemoglobin. Log (IC50>q) values calculated from diameter changes were the same in the central artery, GO, and its first three generations of branch vessels Gl, G2 and G3 (ie down to 7Oum) be
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Patterson, Jesse, and Thidapat Chantem. "EDF-hv." In RTNS '16: 24th International Conference on Real-Time Networks and Systems. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2997465.2997491.

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Huang, Benxiong, Fan Zhang, Yan Wang, and Xiaoling Wang. "IEEE 802.11e EDCF performance evaluation." In Asia-Pacific Optical and Wireless Communications, edited by Chih-Lin I, Jiann-An Tsai, and Hequan Wu. SPIE, 2004. http://dx.doi.org/10.1117/12.520233.

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Bardou-Jacquet, Antoine. "EDF la vallée." In the 29th International Conference. New York, New York, USA: ACM Press, 2002. http://dx.doi.org/10.1145/2931127.2931211.

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Roberts, M. "CEDIF: a data driven EDIF reader." In the 1989 26th ACM/IEEE conference. New York, New York, USA: ACM Press, 1989. http://dx.doi.org/10.1145/74382.74538.

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Shahdad, M. "An interface between VHDL and EDIF." In 24th ACM/IEEE conference proceedings. New York, New York, USA: ACM Press, 1987. http://dx.doi.org/10.1145/37888.37958.

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Shahdad, M. "An interface between VHDL and EDIF." In COMPCON Spring 88. IEEE, 1988. http://dx.doi.org/10.1109/cmpcon.1988.4881.

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Eurich, J. P. "EDIF: test generation and fault simulation." In COMPCON Spring 88. IEEE, 1988. http://dx.doi.org/10.1109/cmpcon.1988.4887.

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Reports on the topic "EDHF"

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Bui, Dai. Revision of a Non-Preemptive EDF Packet Scheduling Algorithm. Fort Belvoir, VA: Defense Technical Information Center, April 2009. http://dx.doi.org/10.21236/ada538864.

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Teixeira, Joao. Unified Cloud and Mixing Parameterizations of the Marine Boundary Layer: EDMF and PDF-Based Cloud Approaches. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada598085.

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Teixeira, Joao. Unified Cloud and Mixing Parameterizations of the Marine Boundary Layer: EDMF and PDF-based Cloud Approaches. Fort Belvoir, VA: Defense Technical Information Center, September 2012. http://dx.doi.org/10.21236/ada574017.

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Teixeira, Joao. Collaborative Project: An Integrated Parameterization of Boundary Layer and Convective Mixing: The Eddy-Diffusivity/Mass-Flux (EDMF) Approach. Office of Scientific and Technical Information (OSTI), June 2019. http://dx.doi.org/10.2172/1529045.

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Burchell, Timothy D. Experimental Plan for EDF Energy Creep Rabbit Graphite Irradiations- Rev. 2 (replaces Rev. 0 ORNL/TM/2013/49). Office of Scientific and Technical Information (OSTI), July 2014. http://dx.doi.org/10.2172/1143548.

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Emmitt, George D., Ralph Foster, and Stephan de Wekker. Investigation of the Representation of OLEs and Terrain Effects Within the Costal Zone in the EDMF Parameterization Scheme: An Airborne Doppler Wind Lidar Perspective. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada612728.

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Emmitt, George D., Ralph Foster, Stephan De Wekker, and Steven Greco. Investigation of the Representation of OLEs and Terrain Effects Within the Coastal Zone in the EDMF Parameterization Scheme: An Airborne Doppler Wind Lidar Perspective. Fort Belvoir, VA: Defense Technical Information Center, July 2012. http://dx.doi.org/10.21236/ada564621.

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Emmitt, George D., Ralph Foster, and Stephen de Wekker. Investigation of the Representation of OLEs and Terrain Effects Within the Coastal Zone in the EDMF Parameterization Scheme: An Airborne Doppler Wind Lidar Perspective. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada590709.

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Emmitt, George D., Ralph Foster, and Stephan De Wekker. Investigation of the Representation of OLEs and Terrain Effects within the Coastal Zone in the EDMF Parameterization Scheme: An Airborne Doppler Wind Lidar Perspective. Fort Belvoir, VA: Defense Technical Information Center, October 2015. http://dx.doi.org/10.21236/ada627183.

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Comerford, Milo, and Simeon Dukic. Ekstremizmi Online: Sfidat dhe Mundësitë në Ballkanin Perëndimor. RESOLVE Network, February 2021. http://dx.doi.org/10.37805/pn2021.7.wb.

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Ballkani Perëndimor përballet me një sfidë të dyfishtë si pasojë e ekstremizmit online. Platformat online po lehtësojnë shënjestrimin specifik të rajonit nga narrativa të ndryshme ekstremiste ndërkombëtare. Ndërkohë, historia dhe gjeopolitika rajonale po përvetësohen për të justifikuar veprimet dhe narrativat ekstremiste në mbarë botën. Sidoqoftë, ky fenomen është pjesë e një tendence më të gjerë, e cila vë në dukje sfidën gjithnjë e më të madhe që paraqet përhapja e ideologjive ekstremiste transkombëtare në platformat online, si ato xhihadiste të dhun
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