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Published: 10 December 2022
Last updated: 29 January 2023

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1

Perrins, E. J. Ischaemic heart disease. Guildford: Update - Siebert, 1987.

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2

Bray, Colin. Ischaemic heart disease. London: Update, 1986.

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3

Rankin, A. C. Ischaemic heart disease. Guildford: Update-Siebert, 1988.

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4

M, Fox Kim, ed. Ischaemic heart disease. Lancaster, England: MTP Press, 1987.

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5

Fox, Kim M., ed. Ischaemic Heart Disease. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3211-1.

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6

Meade, T. W. Haemostatic variables, thrombosis and ischaemic heart disease. Amsterdam: Excerpta Medica, 1995.

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7

Gunnell, David. The invasive management of ischaemic heart disease. Bristol: Health Care Evaluation Unit, University of Bristol, 1994.

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8

de Luna, A. Bays, and M. Fiol-Sala, eds. The Surface Electrocardiography in Ischaemic Heart Disease. Oxford, UK: Blackwell Publishing, 2007. http://dx.doi.org/10.1002/9780470696248.

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9

Edwards, Eric William. Population variation for risk variables in ischaemic heart disease. Oxford: OxfordPolytechnic, 1992.

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10

Porter, Marilyn. Human plasma glutathione peroxidase as a risk factor for ischaemic heart disease. Manchester: University of Manchester, 1996.

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11

Wright, L. The practice nurse and secondary prevention o Ischaemic heart disease for myocardial infarction patients. Oxford: Oxford Brookes University, 1996.

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12

Joensuu, Tapio. Sairaalahoitoon tai kuolemaan johtanut sepelvaltimotauti Suomessa vuosina 1972-1985 =: Ischaemic heart disease leading to hospitalization or death in Finland, 1972-1985. Helsinki: Valtion painatuskeskus, 1989.

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13

Rowe, Michael. Angioplasty and other percutaneous interventional techniques in the treatment of ischaemic heart disease: A literature review. [Canberra]: Australian Institute of Health, 1989.

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14

Morran, Nan. Health education: An examination of its effectiveness in changing risk behaviours implicated in ischaemic heart disease. London: PEL, 1992.

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15

Hamdan, Haytham Kamal. The importance of quantitative thallium-201 single photon emission computed tomography in patients with ischaemic heart disease. Birmingham: University of Birmingham, 1996.

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16

R, Lichtlen Paul, ed. New therapy of ischaemic heart disease and hypertension: Proceedings of the symposium held in Geneva, 18-20 April 1985. Amsterdam: Excerpta Medica, 1986.

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17

Tighe, Paula. The influence of folate and related B-vitamins on plasma homocysteine in ischaemic heart disease patients and healthy controls. [S.l: The Author), 2004.

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18

National Commission on Macroeconomics and Health (Sri Lanka) and Health Economics Study Programme, eds. The economic cost of five common diseases in Sri Lanka: Asthma, hypertension, ischaemic heart disease, diarrhoea, and viral fever. Colombo: National Commission on Macroeconomics and Health, Ministry of Healthcare and Nutrition, 2006.

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19

The effect of calcium antagonists on the normoxic and the ischaemic myocardium: Studies in rat and guinea-pig cardiac preparations. Amsterdam: [s.n.], 1989.

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20

Carlos, Kaski Juan, and Holt David W, eds. Myocardial damage: Early detection by novel biochemical markers. Dordrecht: Kluwer Academic, 1998.

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21

Vanderpump. The incidence of thyroid disorders and diabetes mellitus in the community and the relationship of thyroid failure with the development of ischaemic heart disease. Birmingham: University of Birmingham, 1995.

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22

Rosen, Shara. Trends in the early diagnosis of cardiovascular disease: Worldwide market opportunities. New York: Kalorama Information, 2001.

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23

S, Luxenberg Jay, and Better Health Foundation, eds. You can't live forever, you can live 10 years longer with better health: A practical guide to reducing your risk of dying early from heart disease, cancer, stroke, osteoporosis & accidents. San Francisco, Calif: Better Health Foundation & UCSF/Mount Zion, 1993.

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24

Peacock, Linzi, and Rachel Hignett. Acquired heart disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0041.

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Heart disease in pregnancy is a leading cause of maternal death worldwide. In the United Kingdom and United States, heart disease in pregnancy is the commonest cause of maternal death. In Europe, over 1% of maternal deaths are attributable to structural heart disease. In addition, heart disease in pregnancy is a significant cause of severe maternal and fetal morbidity. Whilst the vast majority of women with heart disease in pregnancy have underlying congenital heart disease, most maternal deaths are due to acquired heart disease (AHD). As the risk factors for AHD become ever more prevalent, the expectation is that disease burden from AHD in pregnancy will also increase. Women with AHD benefit from preconception or early assessment in pregnancy by a multidisciplinary team including obstetricians, cardiologists, and obstetric anaesthetists. Risk assessment using the modified World Health Organization classification of cardiac disease in pregnancy will inform frequency of review in pregnancy. A detailed plan for delivery should be agreed in the third trimester. Where possible, a vaginal delivery is advised: caesarean delivery is reserved for women with obstetric indications or with specific severe underlying cardiac conditions. Slow incremental epidural analgesia is usually recommended to reduce the cardiorespiratory work of labour and an assisted second-stage delivery will limit exertion due to pushing. Neuraxial anaesthesia for operative delivery is becoming a more familiar approach and techniques such as low-dose spinal component combined spinal–epidural or slow incremental epidural top-up maximize haemodynamic stability. Invasive monitoring is often beneficial. Post-delivery care is safely delivered in a high dependency or intensive therapy setting. This chapter looks at the general principles of management of women with AHD, and then examines in detail ischaemic heart disease, arrhythmias, cardiac transplantation, aortic pathology and aortic dissection, cardiomyopathy, valvular heart disease, and infective endocarditis.
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25

Morrison, Karen. Prevention of cerebrovascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0348.

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Stroke is the main cause of neurological morbidity in adults and the third most common cause of death worldwide after ischaemic heart disease and cancer (all forms combined). It is more common in older people, with three-quarters of strokes occurring in people over 65 years of age, and estimates are that overall stroke morbidity will double by the early 2020s. The worldwide figure of increasing incidence of stroke detection masks the fact that mortality from stroke has actually been falling in developed countries since the latter half of the twentieth century while the mortality has continued to rise in China, Asia, and eastern Europe. This chapter discusses prevention of cerebrovascular disease, and includes strategies to reduce the risk of thromboembolic stroke and cerebral haemorrhage.
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26

Ischaemic heart disease. Reed Healthcare Communications, 1990.

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27

Falk, Erling, Pim J. De Feyter, and P. K. Shah. Ischaemic Heart Disease. Blackwell Publishing Limited, 2007.

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28

Cowan, ed. Ischaemic heart disease. Sutton: Reed Healthcare Communications, 1994.

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29

G, Julian Desmond, ed. Ischaemic heart disease. Update-Siebert, 1987.

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30

Fox, K. Ischaemic Heart Disease. Springer, 2012.

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31

Nihoyannopoulos, Petros, and Fausto Pinto. Ischaemic heart disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0012.

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Echocardiography with its multiple modalities plays a central role in the evaluation of patients with known or suspected coronary artery disease, starting from the differential diagnosis of the patient presenting with acute chest pain. In the patient presenting with acute myocardial infarction (raised troponins) whether it is with ST-segment elevation or without, echocardiography is the first imaging modality used in order to ascertain the presence and extent of LV dysfunction and the presence of complications. In the absence of myocardial infarction (negative troponins), echocardiography will play an important diagnostic role in identifying the presence of reversible myocardial ischaemia. Stress echocardiography in many institutions is now the preferred stress modality associated with imaging as it is cost-effective and does not use ionizing radiation. Finally, echocardiography plays a pivotal role in the assessment of myocardial viability since the presence and extent of viable myocardium may guide therapeutic strategies. It has been stressed that laboratories and individuals need to have experience and be accredited by the authorities so that the results of echocardiographic investigations will be credible.
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32

Ischaemic Heart Disease. Oxford University Press, 2002.

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33

Hemker, H. C., J. H. de Haas, and H. A. Snellen. Ischaemic Heart Disease. Springer, 2012.

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34

Hemker, H. C., J. H. de Haas, and H. A. Snellen. Ischaemic Heart Disease. Springer Netherlands, 2011.

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35

Fox, K. M. Ischaemic Heart Disease. Springer, 2011.

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36

Purcell, Henry. Ischaemic Heart Disease Compendium. Current Medical Literature, 2003.

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37

Ravi, Pillai, and Wright John E. C, eds. Surgery for ischaemic heart disease. Oxford: Oxford University Press, 1999.

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38

Mimura, Goro. Lipids and Ischaemic Heart Disease. Elsevier, 1985.

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39

Atheroma: Atherosclerosis in Ischaemic Heart Disease. Science Press Ltd, 1990.

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40

Fox, Kate, and W. J. Remme. ACE Inhibition and Ischaemic Heart Disease. 2nd ed. Science Press, 2004.

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41

Hearse, David. Metabolic Approaches to Ischaemic Heart Disease. Science Press, 1998.

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42

A, Poole-Wilson P., and Sheridan D. J, eds. Atheroma: Atherosclerosis in ischaemic heart disease. London: Science Press, 1990.

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43

Alonso Salinas, Gonzalo Luis, Marina Pascual Izco, Covadonga Fernández-Golfín, Luigi P. Badano, and José Luis Zamorano. Ischaemic heart disease: acute coronary syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0029.

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Transthoracic echocardiography (TTE) is a non-invasive and accessible tool that should be widely used in the evaluation of patients with suspected or known acute coronary syndrome (ACS). Its role is crucial in the management of patients with suspected ACS without electrocardiographic changes or elevation of cardiac markers, allowing the formulation of differential diagnosis between cardiac and extracardiac aetiologies. If the ACS is confirmed, initial assessment of regional and global left and right ventricle contractile function is fundamental in establishing the management strategy and may help in the risk stratification of these patients. TTE can also characterize the ischaemic myocardium in the acute phase, exposing any myocardial regional wall motion abnormalities. Furthermore, TTE is an excellent tool for the initial assessment of the aetiology of cardiogenic shock. It provides additional information regarding the haemodynamic status of the patient, including filling pressures and stroke volume, and it may rule out other causes of shock; thus, immediate TTE, or transoesophageal echocardiography if necessary, should be performed when cardiogenic shock is suspected. In the chronic phase, TTE plays an important role in characterizing myocardial infarction scar and its extent. TTE can accurately differentiate viable myocardium from scar tissue, and may guide revascularization if needed, improving patient care.
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44

Picano, Eugenio, Fausto Pinto, and Blazej Michalski. Ischaemic heart disease: coronary artery anomalies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0030.

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Coronary anomalies occur in less than 1% of the general population and their clinical presentation can range anywhere from a benign incidental finding to the cause of sudden cardiac death. Since congenital coronary arteries anomalies are often considered as the first cause of cardiac death in young athletes in Europe, careful attention has to be paid in this specific subpopulation in case of suggestive symptoms. Although focused expert echocardiography is the first-line imaging tool, coronary computed tomography or radiation-free magnetic resonance imaging are recommended for more definitive definition of the coronary course in persons suspected of having coronary artery anomalies. Most coronary anomalies belong to the group of anomalous origin. Aneurysms are defined as dilations of a coronary vessel 1.5 times the normal adjacent coronary artery segment. Coronary artery fistulas are communications between one or more coronary arteries and a cardiac chamber (coronary-cameral), the pulmonary artery, or a venous structure (such as the sinus or superior vena cava).
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45

J, Davies M., and Woolf Neville, eds. Atheroma: Atherosclerosis in ischaemic heart disease. London: Science Press, 1990.

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46

Ruiz-Villalba, Adrián, Nikolaos Frangogiannis, and José Maria Pérez-Pomares. Origin and diversity of cardiac fibroblasts: developmental substrates of adult cardiac fibrosis. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0012.

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Cardiac connective tissues are primarily formed by cardiac fibroblasts (CF) of diverse embryonic origins. Whereas CF specific roles in cardiac morphogenesis remain under-researched, their involvement in adult cardiac fibrosis is clinically relevant. Cardiac fibrosis is a common element of several chronic cardiac conditions characterized by the loss of ventricular wall mechanical function, ultimately driving to heart failure. In the ischaemic heart early reparative fibrosis evidences the very restricted regenerative potential of the myocardium. In non-ischaemic diseases fibrosis is activated by unknown signals. We summarize current knowledge on the origin of CFs and their developmental roles, and discuss the differential disease-dependent response of different CF subpopulations to various pathological stimuli. We also describe the characteristic cell-cell and cell-matrix interactions that determine the fibrotic remodelling of the myocardium. We analyse experimental models for the study of cardiac fibrosis, and suggest future directions in the search for new markers and therapeutic targets.
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47

Delcourt, Candice, and Craig Anderson. Diagnosis and assessment of stroke. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0235.

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Approximately 20 million strokes occur in the world each year and over one-quarter of these are fatal. This makes stroke the second most common cause of death, after ischaemic heart disease, and strokes are responsible for 6 million deaths (almost 10% of all deaths) annually. Stroke has major consequences in terms of residual physical disability, depression, dementia, epilepsy, and carer burden. Moreover, around 20% of survivors experience a further stroke or serious vascular event within a few years of the index event. Ischaemic stroke contributes the greatest share of the impact of stroke, with a rate of approximately 1 in 1000 person-years and accounting for between 60% (in Asia) and 90% (in Western ‘white’ populations) of all strokes around the world. Diagnosis and assessment are essentially clinical and confirmed by CT or MRI scanning. Prognostication is difficult in the early phase of haemorrhagic stroke and in ischaemic stroke is affected by the availability and timely use of treatments to recanalize the occluded vessel.
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48

Stringfellow, Cynthia. Ischaemic Heart Disease, Poor Man's Disease in a Rich Man's Society. United Health-Grimsby & Scunthorpe Health Authority, 1993.

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49

Hearse. Metabolic Approaches to Ischaemic Heart Disease and Its Management. Science Press Inc., 1998.

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50

Samuel, Sclarovsky, ed. Electrocardiography of acute myocardial ischaemic syndromes. London: M. Dunitz, 1999.

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