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Published: 12 May 2023

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1

Carlos, Kaski Juan, and Holt David W, eds. Myocardial damage: Early detection by novel biochemical markers. Dordrecht: Kluwer Academic, 1998.

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2

Policy, Toronto Working Group on Cholesterol. Detection and management of asymptomatic hypercholesterolemia: A policy document. [Toronto?: s.n., 1989.

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3

Shen, Ze-ping. An application of neural networks for the detection of coronary heart disease. Uxbridge: Brunel University, 1994.

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4

McCarthy, Joseph C. Early hip disorders: Advances in detection and minimally invasive treatment. New York: Springer, 2011.

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5

editor, Mordini E. (Emilio), and Green Manfred editor, eds. Internet-based intelligence in public health emergencies: Early detection and response in disease outbreak crises. Amsterdam, Netherlands: IOS Press, 2011.

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6

name, No. Early hip disorders: Advances in detection and minimally invasive treatment. New York, NY: Springer, 2003.

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7

Report of the Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults. Bethesda, Md.?]: U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, National Cholesterol Education Program, 1989.

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8

Rosen, Shara. Trends in the early diagnosis of cardiovascular disease: Worldwide market opportunities. New York: Kalorama Information, 2001.

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9

Fitzgerald, Rebecca C. Pre-invasive disease: Pathogenesis and clinical management. New York: Springer, 2011.

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10

Patlak, Margie. Mammography and beyond: Developing technologies for the early detection of breast cancer : a non-technical summary. Edited by National Cancer Policy Board (U.S.). Committee on the Early Detection of Breast Cancer and National Research Council (U.S.). Commission on Life Sciences. Washington, D.C: National Academy Press, 2001.

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11

Wells, F. O. Preventive medicine: Working for patients : a review of the therapeutic areas where screening programmes and the early detection and treatment of disease will benefit patients and the NHS. London: Association of the British Pharmaceutical Industry, 1989.

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12

S, Luxenberg Jay, and Better Health Foundation, eds. You can't live forever, you can live 10 years longer with better health: A practical guide to reducing your risk of dying early from heart disease, cancer, stroke, osteoporosis & accidents. San Francisco, Calif: Better Health Foundation & UCSF/Mount Zion, 1993.

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13

Myocardial Damage: Early Detection by Novel Biochemical Markers. Springer Netherlands, 2010.

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14

(Editor), J. C. Kaski, and David W. Holt (Editor), eds. Myocardial Damage: Early Detection by Novel Biochemical Markers. Springer, 1998.

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15

Holt, David W., and Juan Carlos Kaski. Myocardial Damage: Early Detection by Novel Biochemical Markers. Springer London, Limited, 2013.

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16

Tribouilloy, Christophe, Patrizio Lancellotti, Ferande Peters, José Juan Gómez de Diego, and Luc A. Pierard. Heart valve disease (aortic valve disease): aortic regurgitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0033.

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Echocardiography is the cornerstone examination for the assessment of aortic regurgitation (AR): it provides reliable evaluation of the aortic valve and allows diagnosis and identification of the mechanism of regurgitation. The specific aetiology of the disease can be identified in the majority of cases. A combination of quantitative and quantitative Doppler and two-dimensional (2D) echocardiographic parameters allows the evaluation of the severity of AR and determination of the haemodynamic and left ventricular function repercussions. Echocardiography allows the detection of associated lesions of the aortic root or other valves. In symptomatic patients, echocardiography is essential to confirm the severity of AR. In asymptomatic patients with moderate or severe AR, echocardiography is essential for regular follow-up, by providing precise and reproducible measurements of LV dimensions and function, and for identifying patients who should be considered for elective surgical intervention. In most cases, transthoracic echocardiography (TTE) provides all of the necessary information and transoesophageal echocardiography in usually not required. Real-time three-dimensional (3D) TTE can be complementary to 2D echocardiography for the assessment of the mechanism and quantification of AR by increasing the level of confidence, especially when 2D echocardiographic data are inconclusive or discordant with clinical findings. Tissue Doppler imaging and especially the speckle tracking method are promising approaches to detect early LV dysfunction in patients with asymptomatic severe AR. Echocardiography is therefore the key examination for the assessment of AR and at the centre of the strategic discussion concerning the indications and timing of surgery.
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17

Morrison, Karen. Prevention of cerebrovascular disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0348.

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Stroke is the main cause of neurological morbidity in adults and the third most common cause of death worldwide after ischaemic heart disease and cancer (all forms combined). It is more common in older people, with three-quarters of strokes occurring in people over 65 years of age, and estimates are that overall stroke morbidity will double by the early 2020s. The worldwide figure of increasing incidence of stroke detection masks the fact that mortality from stroke has actually been falling in developed countries since the latter half of the twentieth century while the mortality has continued to rise in China, Asia, and eastern Europe. This chapter discusses prevention of cerebrovascular disease, and includes strategies to reduce the risk of thromboembolic stroke and cerebral haemorrhage.
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18

Hoyle, Nicholas R., Thompson Michael, Richard Morgan, Subrayal M. Reddy, and Damien Arrigan. Early Detection of Biomarkers for Disease. Royal Society of Chemistry, The, 2017.

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19

Seipel, L., and Franz Loogen. Detection of Ischemic Myocardium with Exercise. Springer London, Limited, 2012.

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20

Seipel, L., and Franz Loogen. Detection of Ischemic Myocardium with Exercise. Springer London, Limited, 2011.

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21

Hagendorff, Andreas. Cardiac involvement in systemic diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0020.

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Systemic diseases are generally an interdisciplinary challenge in clinical practice. Systemic diseases are able to induce tissue damage in different organs with ongoing duration of the illness. The heart and the circulation are important targets in systemic diseases. The cardiac involvement in systemic diseases normally introduces a chronic process of alterations in cardiac tissue, which causes cardiac failure in the end stage of the diseases or causes dangerous and life-threatening problems by induced acute cardiac events, such as myocardial infarction due to coronary thrombosis. Thus, diagnostic methods—especially imaging techniques—are required, which can be used for screening as well as for the detection of early stages of the diseases. Two-dimensional echocardiography is the predominant diagnostic technique in cardiology for the detection of injuries in cardiac tissue—e.g. the myocardium, endocardium, and the pericardium—due to the overall availability of the non-invasive procedure.The quality of the echocardiography and the success rate of detecting cardiac pathologies in patients with primary non-cardiac problems depend on the competence and expertise of the investigator. Especially in this scenario clinical knowledge about the influence of the systemic disease on cardiac anatomy and physiology is essential for central diagnostic problem. Therefore the primary echocardiography in these patients should be performed by an experienced clinician or investigator. It is possible to detect changes of cardiac morphology and function at different stages of systemic diseases as well as complications of the systemic diseases by echocardiography.The different parts of this chapter will show proposals for qualified transthoracic echocardiography focusing on cardiac structures which are mainly involved in different systemic diseases.
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22

Early Detection of Alzheimer's Disease: A Neuropsychological Approach. Academic & Scientific Publishers, 2008.

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23

Gao, Youhe. Urine: Promising Biomarker Source for Early Disease Detection. Springer, 2019.

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24

Albert, Marilyn S., and Guy M. Mckhann. Neuroethical Issues In Early Detection Of Alzheimer’S Disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780199570706.013.0135.

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25

Gao, Youhe. Urine: Promising Biomarker Source for Early Disease Detection. Springer Singapore Pte. Limited, 2020.

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26

Chan, Dennis. Early Detection in Alzheimer's Disease: Biological and Technological Advances. Academic Press, 2021.

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27

Chan, Dennis. Early Detection in Alzheimer's Disease: Biological and Technological Advances. Elsevier Science & Technology Books, 2021.

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28

Tragante, Vinicius, Anna Pilbrow, and Katrina Poppe, eds. Improving Early Detection and Risk Prediction in Heart Failure. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-322-1.

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29

C, Morris John, and Jeffrey M. Burns. Mild Cognitive Impairment and Early Alzheimer's Disease: Detection and Diagnosis. Wiley & Sons, Incorporated, John, 2008.

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30

Edoh, Thierry. Pre-Screening Systems for Early Disease Prediction, Detection, and Prevention. IGI Global, 2018.

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31

Peacock, Linzi, and Rachel Hignett. Acquired heart disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0041.

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Heart disease in pregnancy is a leading cause of maternal death worldwide. In the United Kingdom and United States, heart disease in pregnancy is the commonest cause of maternal death. In Europe, over 1% of maternal deaths are attributable to structural heart disease. In addition, heart disease in pregnancy is a significant cause of severe maternal and fetal morbidity. Whilst the vast majority of women with heart disease in pregnancy have underlying congenital heart disease, most maternal deaths are due to acquired heart disease (AHD). As the risk factors for AHD become ever more prevalent, the expectation is that disease burden from AHD in pregnancy will also increase. Women with AHD benefit from preconception or early assessment in pregnancy by a multidisciplinary team including obstetricians, cardiologists, and obstetric anaesthetists. Risk assessment using the modified World Health Organization classification of cardiac disease in pregnancy will inform frequency of review in pregnancy. A detailed plan for delivery should be agreed in the third trimester. Where possible, a vaginal delivery is advised: caesarean delivery is reserved for women with obstetric indications or with specific severe underlying cardiac conditions. Slow incremental epidural analgesia is usually recommended to reduce the cardiorespiratory work of labour and an assisted second-stage delivery will limit exertion due to pushing. Neuraxial anaesthesia for operative delivery is becoming a more familiar approach and techniques such as low-dose spinal component combined spinal–epidural or slow incremental epidural top-up maximize haemodynamic stability. Invasive monitoring is often beneficial. Post-delivery care is safely delivered in a high dependency or intensive therapy setting. This chapter looks at the general principles of management of women with AHD, and then examines in detail ischaemic heart disease, arrhythmias, cardiac transplantation, aortic pathology and aortic dissection, cardiomyopathy, valvular heart disease, and infective endocarditis.
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32

Bass, Cristina, Barbara Bauce, and Gaetano Thiene. Arrhythmogenic right ventricular cardiomyopathy: diagnosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0360.

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Arrhythmogenic cardiomyopathy is a heart muscle disease clinically characterized by life-threatening ventricular arrhythmias and pathologically by an acquired and progressive dystrophy of the ventricular myocardium with fibrofatty replacement. The clinical manifestations of arrhythmogenic cardiomyopathy vary according to the ‘phenotypic’ stage of the underlying disease process. Since there is no ‘gold standard’ to reach the diagnosis of arrhythmogenic cardiomyopathy, multiple categories of diagnostic information have been combined. Different diagnostic categories include right ventricular morphofunctional abnormalities (by echocardiography and/or angiography and/or cardiovascular magnetic resonance imaging), histopathological features on endomyocardial biopsy, electrocardiogram, arrhythmias, and family history, including genetics. The diagnostic criteria were revised in 2010 to improve diagnostic sensitivity, but with the important prerequisite of maintaining diagnostic specificity. Quantitative parameters have been put forward and abnormalities are defined based on the comparison with normal subject data. A definite diagnosis of arrhythmogenic cardiomyopathy is achieved when two major, or one major and two minor, or four minor criteria from different categories are met. The main differential diagnoses are idiopathic right ventricular outflow tract tachycardia, myocarditis, sarcoidosis, dilated cardiomyopathy, right ventricular infarction, congenital heart diseases with right ventricular overload, and athlete’s heart. Among diagnostic tools, contrast-enhanced cardiovascular magnetic resonance is playing a major role in detecting subepicardial-midmural left ventricular free wall involvement, even preceding morphofunctional abnormalities. Moreover, electroanatomical mapping is an invasive tool able to detect early right ventricular free wall involvement in terms of low-voltage areas. Both techniques are increasingly used in the diagnostic work-up although are not yet part of diagnostic criteria.
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33

Lemiere, Jurgen. Huntington's Disease: Early Detection & Progression of Cognitive Changes in Patients & Asymptomatic Mutation Carriers (Acta Biomedica Lovaniensia). Leuven Univ Pr, 2004.

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34

Rose, Kelly. Heart Illness Explained: Heart or Cardiovascular Disease, Its Symptoms and Early Warning Signals, and How to Avoid Them. Independently Published, 2022.

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35

Dob, Daryl P., Elspeth E. Pickering, and Michael A. Gatzoulis. Moderate to complex congenital heart disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0040.

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Children born with congenital heart disease no longer face the prospect of early death and a poor quality of life. In fact, most neonates with moderate to complex congenital heart disease have a survival rate to adulthood of over 80%. The ratio of adults to children with congenital heart disease is increasing, due to better surgical repairs, and longer survival with a better quality of life. In the Western world, there are more adults than children alive with congenital heart disease. This remarkable medical effort has allowed young women with congenital heart disease to mature to an age where they wish to have babies of their own. Early generations of women, palliated with Mustard or Senning repairs, have shown it is possible to face the cardiovascular challenges of pregnancy and survive. As the number of women with congenital heart disease is predicted to grow by 25% in the next decade and more women with congenital heart disease become pregnant; a better understanding of moderate to complex heart disease, different surgical repair procedures, and residual anomalies is paramount. This chapter examines the management of parturients with transposition complexes (both classical and congenitally corrected), tetralogy of Fallot, the Fontan circulation, Eisenmenger’s syndrome, and congenital aortic stenosis, taking into consideration the effect of pregnancy, labour, delivery, and anaesthesia on each circulation.
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36

Fye, W. Bruce. President Roosevelt’s Secret Hypertensive Heart Disease. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199982356.003.0007.

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Franklin D. Roosevelt’s health was a factor in the 1944 election. Presidential press secretary Stephen Early and White House physician Ross McIntire worried that Thomas Dewey might win if voters learned that Roosevelt had severe hypertension and had an episode of congestive heart failure. Three weeks before the election, Mayo cardiologist Arlie Barnes visited the Bethesda Naval Medical Center where he learned that some physicians suspected that Roosevelt had a “serious heart ailment.” When Barnes returned to Rochester he mentioned this to a few friends. Word of this conversation reached Early, and FBI agents interrogated Barnes and other Mayo physicians two weeks before the election. During the campaign’s closing days, White House insiders orchestrated events designed to showcase a healthy commander in chief. Stifling rumors about Roosevelt’s health was part of their strategy to defeat Dewey. Less than three months into his fourth term, Roosevelt had a stroke and died.
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37

Karatasakis, G., and G. D. Athanassopoulos. Cardiomyopathies. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0019.

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Echocardiography is a key diagnostic method in the management of patients with cardiomyopathies.The main echocardiographic findings of hypertrophic cardiomyopathy are asymmetric hypertrophy of the septum, increased echogenicity of the myocardium, systolic anterior motion, turbulent left ventricular (LV) outflow tract blood flow, intracavitary gradient of dynamic nature, mid-systolic closure of the aortic valve and mitral regurgitation. The degree of hypertrophy and the magnitude of the obstruction have prognostic meaning. Echocardiography plays a fundamental role not only in diagnostic process, but also in management of patients, prognostic stratification, and evaluation of therapeutic intervention effects.In idiopathic dilated cardiomyopathy, echocardiography reveals dilation and impaired contraction of the LV or both ventricles. The biplane Simpson’s method incorporates much of the shape of the LV in calculation of volume; currently, three-dimensional echocardiography accurately evaluates LV volumes. Deformation parameters might be used for detection of early ventricular involvement. Stress echocardiography using dobutamine or dipyridamole may contribute to risk stratification, evaluating contractile reserve and left anterior descending flow reserve. LV dyssynchrony assessment is challenging and in patients with biventricular pacing already applied, optimization of atrio-interventricular delays should be done. Specific characteristics of right ventricular dysplasia and isolated LV non-compaction can be recognized, resulting in an increasing frequency of their prevalence. Rare forms of cardiomyopathy related with neuromuscular disorders can be studied at an earlier stage of ventricular involvement.Restrictive and infiltrative cardiomyopathies are characterized by an increase in ventricular stiffness with ensuing diastolic dysfunction and heart failure. A variety of entities may produce this pathological disturbance with amyloidosis being the most prevalent. Storage diseases (Fabry, Gaucher, Hurler) are currently treatable and early detection of ventricular involvement is of paramount importance for successful treatment. Traditional differentiation between constrictive pericarditis (surgically manageable) and the rare cases of restrictive cardiomyopathy should be properly performed.
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38

Fitzgerald, Rebecca C. Pre-Invasive Disease: Pathogenesis and Clinical Management. Springer, 2014.

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39

Martinez, Jaun Jair. An international telemedicine model design: Using e-health to improve chronic disease early detection and initial management in rural areas. 2005.

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40

Pasquet, Agnes, Marcia Barbosa, and Jo-Nan Liao. Systemic disease and other conditions: the heart during pregnancy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0056.

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Normal pregnancy represents a haemodynamic challenge for the heart. The main changes are increases in blood (plasma) volume and cardiac output, and a decrease in systemic vascular resistance. These change start early during pregnancy and will be maximal around the 24th week of gestation. This translates into echocardiographic changes such as increase in ventricular volume, stroke volume, and changes in geometry. Peripartum cardiomyopathy is a left ventricular dysfunction without any underlying cause, arising near the end of the gestation or in the early postpartum period. Echocardiography is the key to confirm the diagnosis and also to exclude other causes of cardiomyopathy or to explain clinical signs.
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41

Kelly, Laura, and William P. Stanford. Implementation of Personalized Precision Medicine: Expanding the Clinical Vision Towards Prevention, Early Detection and Precision Treatment of Disease to Drive Extended Healthspan. Elsevier Science & Technology, 2023.

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42

Kelly, Laura, and William P. Stanford. Implementation of Personalized Precision Medicine: Expanding the Clinical Vision Towards Prevention, Early Detection and Precision Treatment of Disease to Drive Extended Healthspan. Elsevier Science & Technology Books, 2023.

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43

Wise, Matt, and Paul Frost. Critical illness. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0147.

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Critical illness can be considered to be any disease process which causes physiological instability that leads to disability or death within minutes or hours. Fortunately, physiological instability associated with critical illness is easily detected by perturbations of simple clinical observations such as blood pressure, heart rate, respiratory rate, oxygen saturations, level of consciousness, and urine output. Individual abnormalities in these observations are sensitive for the presence of critical illness but non-specific. Specificity for critical illness improves as the number of abnormal clinical observations increases. Over recent years, a greater appreciation of the importance of deviations in simple clinical observations as a method of detecting critical illness has led to the development of a number of ‘early warning’ or ‘track and trigger’ systems. These systems attribute a score according to the magnitude and number of abnormal observations that are present, and a high score prompts immediate medical review. Although intuitively sensible, the evidence that these systems are effective in ameliorating or preventing critical illness is currently lacking. This chapter looks at the approach to diagnosis of critical illness, including the pitfalls in diagnosis.
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44

Options, Easy Health. Nature's Simple Solutions for Avoiding Heart Disease - and How NOT to Become a Statistic: Your Step-by-tep Guide to Prevention, Detection, and Natural Treatment Strategies. Easy Health Options, 2008.

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45

Chneiweiss, Hervé. Anticipating a therapeutically elusive neurodegenerative condition: Ethical considerations for the preclinical detection of Alzheimer’s disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198786832.003.0016.

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Among neurodegenerative disorders, Alzheimer’s disease has held a special position during the last 40 years. It represents a huge burden of disease with more than 40 million people affected worldwide. The economic effect it has on society is enormous, and the specific challenges of dementia are tremendous. Now that science has demonstrated that the disease starts two or three decades before any symptoms occur, possibilities exist for diagnosis or testing increasingly early through the capabilities of predictive medicine. The related ethical debate is on the multiple meanings and the impact of preclinical diagnosis of Alzheimer’s disease before the onset of symptoms. To guide this discussion, this chapter draws upon lessons from other fields of medicine and the identification of high-risk individuals bearing pathogenic genetic mutations that predispose them to the disease. It concludes with thoughts on value and choice in the complex, fine balance between anticipating, knowing, and doing.
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46

Keshav, Satish, and Alexandra Kent. Screening for gastrointestinal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0354.

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This chapter discusses screening for gastrointestinal disease, including Barrett’s oesophagus (BO), colorectal cancer, and hepatocellular cancer (HCC). In patients with BO, approximately 5% will develop dysplasia, and 10%–50% of the low-grade dysplasias will progress to high-grade dysplasia or adenocarcinoma within 2–5 years. Thus, screening for BO has been developed to reduce the development of adenocarcinoma via the early detection of high-grade dysplasia or cancer in situ. The main aim of colorectal cancer screening is the early detection of polyps and cancers, at a time when treatment is likely to be more effective. Similarly, early detection of HCC is advantageous, as the prognosis in advanced disease is very poor. This chapter describes the current processes of screening for these diseases, and the impact of this screening, as well as screening for gastrointestinal cancer in specific groups.
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47

Role of emotional expression, selected personality factors, and early childhood experience in the development of cancer, rheumatoid arthritis, coronary heart disease, and health. 1988.

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48

Grassberger, Martin, Ronald A. Sherman, and Olga S. Gileva. Biotherapy - History, Principles and Practice: A Practical Guide to the Diagnosis and Treatment of Disease using Living Organisms. Springer, 2013.

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49

Kim, Christopher, Martin Grassberger, Ronald A. Sherman, Olga S. Gileva, and Kosta Mumcuoglu. Biotherapy - History, Principles and Practice: A Practical Guide to the Diagnosis and Treatment of Disease Using Living Organisms. Springer London, Limited, 2013.

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50

Kim, Christopher, Martin Grassberger, Ronald A. Sherman, Olga S. Gileva, and Kosta Mumcuoglu. Biotherapy - History, Principles and Practice: A Practical Guide to the Diagnosis and Treatment of Disease using Living Organisms. Springer, 2013.

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