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Journal articles on the topic 'Dysprosody'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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1

Harris, Robert, Klaus L. Leenders, and Bauke M. de Jong. "Speech dysprosody but no music ‘dysprosody’ in Parkinson’s disease." Brain and Language 163 (December 2016): 1–9. http://dx.doi.org/10.1016/j.bandl.2016.08.008.

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2

Sidtis, John J., and Diana Van Lancker Sidtis. "A Neurobehavioral Approach to Dysprosody." Seminars in Speech and Language 24, no. 2 (2003): 093–106. http://dx.doi.org/10.1055/s-2003-38901.

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3

Hird, K., and K. Kirsner. "Dysprosody Following Acquired Neurogenic Impairment." Brain and Language 45, no. 1 (July 1993): 46–60. http://dx.doi.org/10.1006/brln.1993.1032.

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4

Lebrun, Yvan, Anne Lessinnes, Luc De Vresse, and Chantal Leleux. "Dysprosody and the non-dominant hemisphere." Language Sciences 7, no. 1 (April 1985): 41–52. http://dx.doi.org/10.1016/s0388-0001(85)80011-5.

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5

Haley, Katarina L. "Dysprosody and the Foreign Accent Syndrome." Perspectives on Neurophysiology and Neurogenic Speech and Language Disorders 19, no. 3 (October 2009): 90–96. http://dx.doi.org/10.1044/nnsld19.3.90.

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Abstract Purpose: The sudden emergence of a foreign accent in an individual's native language has been described in the literature for over 60 years. In one of the most famous cases, the terms prosody and dysprosody first were introduced to the literature. The purpose of this paper is to summarize the prosodic changes seen in the foreign accent syndrome (FAS) and to review its etiology and clinical course. Method: Case studies were reviewed, with an emphasis on information about clinical presentation and course and on speech changes affecting stress, rate, duration, and intonation. Results and Conclusions: In the majority of published cases with FAS, there has been documented focal brain injury in the left cerebral hemisphere, and the foreign accent has emerged after a period of recovery from muteness, nonfluent aphasia, and/or motor speech disorder. In other cases, a psychogenic etiology has been established or suggested. Stress, rate, and duration changes are similar to those seen in nonfluent aphasia and apraxia of speech, whereas intonation changes are more specific to the foreign accent presentation. Information about recovery and psychosocial consequences of the accented speech is sparse and these areas are in need of further study. In particular, there is a need for detailed and clinically oriented case studies with longitudinal follow-up.

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6

Graff-Radford, J., D. A. Drubach, E. A. Strand, and K. A. Josephs. "Fluorodeoxyglucose F18 PET in progressive emotional dysprosody." Neurology 79, no. 5 (July 18, 2012): 480–81. http://dx.doi.org/10.1212/wnl.0b013e31826170e0.

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7

Pinto, José Antonio, Renato José Corso, Ana Cláudia Rocha Guilherme, Sı́lvia Rebelo Pinho, and Monica de Oliveira Nóbrega. "Dysprosody nonassociated with neurological diseases—a case report." Journal of Voice 18, no. 1 (March 2004): 90–96. http://dx.doi.org/10.1016/j.jvoice.2003.07.005.

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8

FEINSTEIN, ANTHONY, and ANDREW HATTERSLEY. "SINGLE CASE STUDY Ganser Symptoms, Dissociation, and Dysprosody." Journal of Nervous and Mental Disease 176, no. 11 (November 1988): 692–93. http://dx.doi.org/10.1097/00005053-198811000-00009.

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9

Bandini, A., F. Giovannelli, S. Orlandi, S. D. Barbagallo, M. Cincotta, P. Vanni, R. Chiaramonti, A. Borgheresi, G. Zaccara, and C. Manfredi. "Automatic identification of dysprosody in idiopathic Parkinson's disease." Biomedical Signal Processing and Control 17 (March 2015): 47–54. http://dx.doi.org/10.1016/j.bspc.2014.07.006.

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10

GANDOUR, J. "Dysprosody in Broca's aphasia: A case study*1." Brain and Language 37, no. 2 (August 1989): 232–57. http://dx.doi.org/10.1016/0093-934x(89)90017-5.

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11

Prins, Yopie. "Historical Poetics, Dysprosody, and The Science of English Verse." PMLA/Publications of the Modern Language Association of America 123, no. 1 (January 2008): 229–34. http://dx.doi.org/10.1632/pmla.2008.123.1.229.

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“The sound of poetry, the poetry of sound” resonated as Marjorie Perloff's theme for the 2006 MLA Convention, where one could hear about this topic at panels, poetry readings, and the Presidential Forum. Addressing a large audience at the forum, Charles Bernstein tapped the microphone and loudly intoned, “Is this working? Can you hear me?” The moment was a self-conscious performance, perhaps parody, of lyric utterance addressed to “you” from “me”: even before beginning his speech, Bernstein called our attention to the amplification of voice. Instead of addressing any particular you, singular or plural, he seemed to address the microphone, a mediating apparatus that makes possible but also interrupts the intimacy of address that lyric poetry (after John Stuart Mill) invites us to overhear. Through the microphone, Bernstein gave new overtones to Mill's definition of poetry as “overheard,” which could also mean hearing it too much, making it too loud, overworking the metaphor of the voice that we think is speaking directly to us. Although we tend to think of sound as immediate (is it?), the sound of poetry is never heard without mediation, and we should attend to the medium.

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12

Samuel, C., A. Louis-Dreyfus, J. Couillet, B. Roubeau, S. Bakchine, B. Bussel, and P. Azouvi. "Dysprosody after severe closed head injury: an acoustic analysis." Journal of Neurology, Neurosurgery & Psychiatry 64, no. 4 (April 1, 1998): 482–85. http://dx.doi.org/10.1136/jnnp.64.4.482.

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13

Penner, Nicholas Miller, Ingo Hertr, Heike. "Dysprosody in Parkinson's disease: an investigation of intonation patterns." Clinical Linguistics & Phonetics 15, no. 7 (January 2001): 551–66. http://dx.doi.org/10.1080/02699200110078140.

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14

Caekebeke, J. F., A. Jennekens-Schinkel, M. E. van der Linden, O. J. Buruma, and R. A. Roos. "The interpretation of dysprosody in patients with Parkinson's disease." Journal of Neurology, Neurosurgery & Psychiatry 54, no. 2 (February 1, 1991): 145–48. http://dx.doi.org/10.1136/jnnp.54.2.145.

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15

Ballard, Kirrie J., Donald A. Robin, Patricia McCabe, and Jeannie McDonald. "A Treatment for Dysprosody in Childhood Apraxia of Speech." Journal of Speech, Language, and Hearing Research 53, no. 5 (October 2010): 1227–45. http://dx.doi.org/10.1044/1092-4388(2010/09-0130).

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16

Peters, A. S., J. Remi, C. Vollmar, J. A. Gonzalez-Victores, J. P. S. Cunha, and S. Noachtar. "Dysprosody during epileptic seizures lateralizes to the nondominant hemisphere." Neurology 77, no. 15 (September 28, 2011): 1482–86. http://dx.doi.org/10.1212/wnl.0b013e318232abae.

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17

Ma, Joan K. Y., Tara Whitehill, and Katherine S. K. Cheung. "Dysprosody and stimulus effects in Cantonese speakers with Parkinson's disease." International Journal of Language & Communication Disorders 45, no. 6 (November 2010): 645–55. http://dx.doi.org/10.3109/13682820903434813.

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18

Skodda, Sabine, Heiko Rinsche, and Uwe Schlegel. "Progression of dysprosody in Parkinson's disease over time-A longitudinal study." Movement Disorders 24, no. 5 (April 15, 2009): 716–22. http://dx.doi.org/10.1002/mds.22430.

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19

Ballard, Kirrie J., Rosemary Varley, and Diane Kendall. "Promising Approaches to Treatment of Apraxia of Speech: Preliminary Evidence and Directions for the Future." Perspectives on Neurophysiology and Neurogenic Speech and Language Disorders 20, no. 3 (October 2010): 87–93. http://dx.doi.org/10.1044/nnsld20.3.87.

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This paper provides a brief review of three interventions for apraxia of speech (AOS) that are in preliminary stages of development with evidence at the level of Phase I intervention trials. The first, Rapid Syllable Transition Treatment (ReST), combines intervention for dysprosody and segmental accuracy in apraxia of speech (AOS). The second is a word-level neurobiological approach that incorporates a substantial sensory-perceptual stimulation component and encourages error-free productions during practice. The third, phono-motor rehabilitation of AOS, provides intensive multi-sensory therapy for production of single sounds and words. These early studies have provided motivation for refining and exploring these interventions further.

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20

Bandini, A., F. Giovannelli, M. Cincotta, P. Vanni, R. Chiaramonti, A. Borgheresi, G. Zaccara, S. Orlandi, and C. Manfredi. "75. Automatic detection of dysprosody patterns in patients with idiopathic Parkinson’s disease." Clinical Neurophysiology 126, no. 1 (January 2015): e17-e18. http://dx.doi.org/10.1016/j.clinph.2014.10.094.

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21

Zainaee, Shahryar, Ramin Mahdipour, Masoud Mahdavi Rashed, and Davood Sobhani-Rad. "Dysgraphia and dysprosody in a patient with arteriovenous malformation: a case report." Neurocase 27, no. 3 (May 4, 2021): 259–65. http://dx.doi.org/10.1080/13554794.2021.1929332.

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22

Michel, A., A. Perillat-Mercerot, L. Damamme, F. Bizouard, and A. Yelnik. "Dysphonia and dysprosody after a stroke: A psychomotor and speech-language co-therapy." Annals of Physical and Rehabilitation Medicine 56 (October 2013): e261-e262. http://dx.doi.org/10.1016/j.rehab.2013.07.682.

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23

Neumann, Katrin, Harald A. Euler, Malte Kob, Alexander Wolff von Gudenberg, Anne-Lise Giraud, Tobias Weissgerber, and Christian A. Kell. "Assisted and unassisted recession of functional anomalies associated with dysprosody in adults who stutter." Journal of Fluency Disorders 55 (March 2018): 120–34. http://dx.doi.org/10.1016/j.jfludis.2017.09.003.

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24

Shoaib, Maria, and Muhammad Taimoor Khan. "Therapeutic deep brain stimulation worsening dysprosody in Parkinson’s disease – an unexplored entity." Neuropsychiatric Disease and Treatment Volume 14 (March 2018): 767–68. http://dx.doi.org/10.2147/ndt.s160803.

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25

McCabe, Patricia, Anita G. Macdonald-D’Silva, Lauren J. van Rees, Kirrie J. Ballard, and Joanne Arciuli. "Orthographically sensitive treatment for dysprosody in children with Childhood Apraxia of Speech using ReST intervention." Developmental Neurorehabilitation 17, no. 2 (April 2014): 137–45. http://dx.doi.org/10.3109/17518423.2014.906002.

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26

Skodda, Sabine, Wenke Visser, and Uwe Schlegel. "Gender-Related Patterns of Dysprosody in Parkinson Disease and Correlation Between Speech Variables and Motor Symptoms." Journal of Voice 25, no. 1 (January 2011): 76–82. http://dx.doi.org/10.1016/j.jvoice.2009.07.005.

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27

Uehara, Rieko, Sachiyo Muranishi, and Mitsuru Kawamura. "Dysprosody Due to Lesion in the Inferior Portion of the Left Central Gyrus. A Case Report." Japan Journal of Logopedics and Phoniatrics 39, no. 3 (1998): 286–90. http://dx.doi.org/10.5112/jjlp.39.286.

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28

Fukunaga, Shinya, Haruki Tokida, Masashi Shiomi, Masahiro Ikeno, Shinsuke Nagami, and Ken Nakatani. "Dysprosody Remaining in a Non-Right-Handed Patient with Apraxia of Speech due to Right Hemisphere Lesion." Japan Journal of Logopedics and Phoniatrics 61, no. 1 (2020): 67–75. http://dx.doi.org/10.5112/jjlp.61.67.

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29

Mashima, Kyoko, Mika Konishi, Toshiki Tezuka, Daisuke Ito, and Masaru Mimura. "A case of tauopathy with auditory agnosia and dysprosody diagnosed by [18F]PM-PBB3 tau PET scan." Neurological Sciences 42, no. 8 (May 4, 2021): 3471–74. http://dx.doi.org/10.1007/s10072-021-05287-y.

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30

Noorsham, Abdul Haleem, Mohamad Muhaimin Abdullah, Sanihah Abdul Halim, Abdul Rahman Izaini Ghani, Zamzuri Idris, and Jafri Malin Abdullah. "Neurological Examination Techniques of Speech in Bahasa Malaysia for Adults: Simple Approach Practiced in Hospital Universiti Sains Malaysia." Malaysian Journal of Medical Sciences 27, no. 6 (December 29, 2020): 148–82. http://dx.doi.org/10.21315/mjms2020.27.6.14.

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There are four classification levels for speech disorders namely dysphonia, dysarthria, dysprosody and dysphasia. In general, speech examination mainly focuses on three main components that are spontaneous speech, auditory comprehension, and oral motor examination. Quick bedside assessment on speech in Bahasa Malaysia is essential to assist the speech language therapist (SLT) and other physicians to determine the disorders. Speech therapy is also essential in monitoring and continuous assessment for patients with speech and language disorders such as dysphasia and dysarthria. Speech clinicians in Hospital Universiti Sains Malaysia (HUSM) have been adapting two most widely used batteries of speech assessment tools namely Western aphasia battery-revised (WAB-R) by Andrew Kertesz and Boston diagnostic aphasia examination (BDAE). These tools have been modified into simple and validated speech assessments in Bahasa Malaysia. This video manuscript will demonstrate the use of both tools in performing bedside speech assessment for patients with speech disorders. The Bahasa Malaysia speech examination should not be difficult when WAB-R and BDAE speech assessment tools are applied. The aim of this simple approach using the adapted version of BDAE and WAB-R is to assist the clinician to achieve quick and accurate diagnosis with a validated scoring system.

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31

Ponsot, Emmanuel, Juan José Burred, Pascal Belin, and Jean-Julien Aucouturier. "Cracking the social code of speech prosody using reverse correlation." Proceedings of the National Academy of Sciences 115, no. 15 (March 26, 2018): 3972–77. http://dx.doi.org/10.1073/pnas.1716090115.

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Human listeners excel at forming high-level social representations about each other, even from the briefest of utterances. In particular, pitch is widely recognized as the auditory dimension that conveys most of the information about a speaker’s traits, emotional states, and attitudes. While past research has primarily looked at the influence of mean pitch, almost nothing is known about how intonation patterns, i.e., finely tuned pitch trajectories around the mean, may determine social judgments in speech. Here, we introduce an experimental paradigm that combines state-of-the-art voice transformation algorithms with psychophysical reverse correlation and show that two of the most important dimensions of social judgments, a speaker’s perceived dominance and trustworthiness, are driven by robust and distinguishing pitch trajectories in short utterances like the word “Hello,” which remained remarkably stable whether male or female listeners judged male or female speakers. These findings reveal a unique communicative adaptation that enables listeners to infer social traits regardless of speakers’ physical characteristics, such as sex and mean pitch. By characterizing how any given individual’s mental representations may differ from this generic code, the method introduced here opens avenues to explore dysprosody and social-cognitive deficits in disorders like autism spectrum and schizophrenia. In addition, once derived experimentally, these prototypes can be applied to novel utterances, thus providing a principled way to modulate personality impressions in arbitrary speech signals.

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32

Hartzheim, Daphne, Yunjung Kim, and Ariel Johnson. "Dysprosodie in preschool children with autism spectrum disorders." Journal of the Acoustical Society of America 142, no. 4 (October 2017): 2641. http://dx.doi.org/10.1121/1.5014682.

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33

Heidler, Maria-Dorothea. "Thalamische Aphasien." Zeitschrift für Neuropsychologie 20, no. 2 (January 2009): 127–39. http://dx.doi.org/10.1024/1016-264x.20.2.127.

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Primär nach linksseitigen thalamischen Läsionen manifestieren sich zwei sprachpathologische Syndrome, die charakterisiert sind durch eine entweder verminderte oder aber flüssige Spontansprachenproduktion, Wortabrufstörungen infolge mangelhafter semantischer Selektion, Perseverationen, Paraphasien, relativ intakte schriftsprachliche, syntaktische und repetitive Fähigkeiten, moderate auditive und visuelle Sprachverständnisstörungen sowie fakultative sprechmotorische Beeinträchtigungen wie Hypophonie und/oder Dysprosodie. Diese als «thalamische Aphasien» bezeichneten Syndrome zeigen einen im Vergleich zu kortikalen Aphasien distinkten Pathomechanismus, da vor allem kognitive Basisprozesse (wie Aufmerksamkeit, Gedächtnis, Exekutivfunktionen, Antrieb etc.) beeinträchtigt sind, die sekundär zu ineffizienten Sprachverarbeitungsprozessen führen. Zudem führen thalamische Läsionen oft zu Amnesien, Orientierungsstörungen und Anosognosie, die auf sprachlicher Ebene u. a. thematische Instabilität, Fragmentation und Konfabulationen zur Folge haben.

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34

Michel, A., A. Perillat-Mercerot, L. Damamme, F. Bizouard, and A. Yelnik. "Dysphonie et dysprosodie après AVC : rééducation en cothérapie psychomotricité et orthophonie." Annals of Physical and Rehabilitation Medicine 56 (October 2013): e260-e261. http://dx.doi.org/10.1016/j.rehab.2013.07.679.

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35

Paolini, Susy, Lucia Paciaroni, Antonio Manca, Roberto Rossi, Daniela Fornarelli, Stefano F. Cappa, Angela M. Abbatecola, and Osvaldo Scarpino. "Change of Accent as an Atypical Onset of non Fluent Primary Progressive Aphasia." Behavioural Neurology 27, no. 2 (2013): 221–27. http://dx.doi.org/10.1155/2013/278976.

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Language disorders can be the first symptom of many neurodegenerative diseases, including Alzheimer's disease (AD) and primary progressive aphasia (PPA). The main variants of PPA are: the non-fluent/agrammatic variant, the semantic variant and the logopenic variant.Several additional variants of PPA, however, have been described and are considered as atypical presentations.We describe the case of a woman presenting a progressive isolated language disturbance, characterized by an early dysprosodia, phonological and semantic paraphasias, agrammatism, impairment in repetition, writing of non-words and sentence comprehension. This clinical picture pointed to an atypical presentation of the non-fluent variety. The frequent symptom overlap between the different variants of PPA, most likely reflecting differences in the topography of the pathological changes, needs to be considered in the definition of diagnostic criteria.

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36

Cercy, Steven P., and Joshua E. Kuluva. "Gelastic epilepsy and dysprosodia in a case of late-onset right frontal seizures." Epilepsy & Behavior 16, no. 2 (October 2009): 360–65. http://dx.doi.org/10.1016/j.yebeh.2009.08.007.

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37

Scarpa, Ester Mirian. "Aquisição, afasia e a hierarquia prosódica." Cadernos de Estudos Lingüísticos 40 (August 10, 2011): 61–76. http://dx.doi.org/10.20396/cel.v40i0.8637120.

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Studies on language acquisition have shown that the child exhibits a top-down trajectory in the acquisition of the prosodic hierarchy, starting with the organisation of the upper (intonational) prosodic levels. Rhythmic readjustments and postlexical secondary stress are later acquisitions. Prosodic disturbances of aphasia and dysarthria have been connected to the question of brain-damage lateralisation and linguistic processing. Subjects damaged in their right hemisphere are said to be dysprosodic; they produce few Fo variations, Fo flattenning, slow tempo. Prosody is said to be reasonably preserved in Broca´s subjects and well preserved in fluent (Wernicke) aphasia subjects. A comparative study was carried out with two subjects, one aphasic and one dysarthric. Some prosodic difficulties were observed in the speech of fluent aphasic subjects, related to the prosodic hierarchy, to the metrical grid and to syllable structure, respectively. On the other hand, the correct placement of pauses in the frontiers of upper domains of the dysarthric subject shows preservation of the prosodic hierarchy. Pitch-direction is also preserved, with short pitch-range. In both cases, the upper domains of the prosodic hierarchy are preserved.

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38

Frota, Sónia, Marisa Cruz, Rita Cardoso, Isabel Guimarães, Joaquim Ferreira, Serge Pinto, and Marina Vigário. "(Dys)Prosody in Parkinson’s Disease: Effects of Medication and Disease Duration on Intonation and Prosodic Phrasing." Brain Sciences 11, no. 8 (August 20, 2021): 1100. http://dx.doi.org/10.3390/brainsci11081100.

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The phonology of prosody has received little attention in studies of motor speech disorders. The present study investigates the phonology of intonation (nuclear contours) and speech chunking (prosodic phrasing) in Parkinson’s disease (PD) as a function of medication intake and duration of the disease. Following methods of the prosodic and intonational phonology frameworks, we examined the ability of 30 PD patients to use intonation categories and prosodic phrasing structures in ways similar to 20 healthy controls to convey similar meanings. Speech data from PD patients were collected before and after a dopaminomimetic drug intake and were phonologically analyzed in relation to nuclear contours and intonational phrasing. Besides medication, disease duration and the presence of motor fluctuations were also factors included in the analyses. Overall, PD patients showed a decreased ability to use nuclear contours and prosodic phrasing. Medication improved intonation regardless of disease duration but did not help with dysprosodic phrasing. In turn, disease duration and motor fluctuations affected phrasing patterns but had no impact on intonation. Our study demonstrated that the phonology of prosody is impaired in PD, and prosodic categories and structures may be differently affected, with implications for the understanding of PD neurophysiology and therapy.

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39

Pinto, S., A. Ghio, B. Teston, and F. Viallet. "La dysarthrie au cours de la maladie de Parkinson. Histoire naturelle de ses composantes : dysphonie, dysprosodie et dysarthrie." Revue Neurologique 166, no. 10 (October 2010): 800–810. http://dx.doi.org/10.1016/j.neurol.2010.07.005.

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40

VANLANCKERSIDTIS, D., N. PACHANA, J. CUMMINGS, and J. SIDTIS. "Dysprosodic speech following basal ganglia insult: Toward a conceptual framework for the study of the cerebral representation of prosody." Brain and Language 97, no. 2 (May 2006): 135–53. http://dx.doi.org/10.1016/j.bandl.2005.09.001.

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41

Van Lancker Sidtis, Diana, Yoonji Kim, Ji Sook Ahn, and John Sidtis. "Do singing and talking arise from the same or different neurological systems? Dissociations of pitch, timing, and rhythm in two dysprosodic singers." Psychomusicology: Music, Mind, and Brain 31, no. 1 (March 2021): 18–34. http://dx.doi.org/10.1037/pmu0000270.

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42

"Expressive dysprosody in schizophrenia — a right frontal deficit?" Schizophrenia Research 1, no. 2-3 (March 1988): 189–90. http://dx.doi.org/10.1016/0920-9964(88)90136-3.

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43

Visser, W., U. Schlegel, and S. Skodda. "Dysprosodie bei Patienten mit idiopathischem Parkinson-Syndrom." Aktuelle Neurologie 35, S 01 (September 2008). http://dx.doi.org/10.1055/s-0028-1086809.

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44

Gürtler, S., K. Franke, and A. Ebner. "Ictal Dysprosodia: Documentation by Fourier Spectral Analysis." Klinische Neurophysiologie 35, no. 03 (August 25, 2004). http://dx.doi.org/10.1055/s-2004-831997.

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45

Rinsche, H., W. Visser, U. Schlegel, and S. Skodda. "Zeitlicher Verlauf der Dysprosodie bei Patienten mit idiopathischem Parkinson-Syndrom." Aktuelle Neurologie 35, S 01 (September 2008). http://dx.doi.org/10.1055/s-0028-1087009.

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46

Eckert, O., T. Schmitz-Hübsch, T. Klockgether, U. Schlegel, and S. Skodda. "Dysprosodie bei Patienten mit spinozerebellärer Ataxie (SCA) und idiopathischem Parkinson-Syndrom (IPS)." Aktuelle Neurologie 35, S 01 (September 2008). http://dx.doi.org/10.1055/s-0028-1086890.

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47

Visser, W., A. Flaßkamp, U. Schlegel, and S. Skodda. "Einfluss von L-Dopa auf die Dysprosodie bei Patienten mit idiopathischem Parkinson-Syndrom." Aktuelle Neurologie 34, S 2 (2007). http://dx.doi.org/10.1055/s-2007-987893.

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