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Books on the topic 'Dysfunctional eating'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Hedges, Peter. What's eating Gilbert Grape? New York: Poseidon Press, 1991.

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Hedges, Peter. What's eating Gilbert Grape. London: Mandarin, 1994.

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What's eating Gilbert Grape. London: Mandarin, 1993.

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4

Books, Pocket, and Copyright Paperback Collection (Library of Congress), eds. What's eating Gilbert Grape? New York: Pocket Books, 1994.

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5

S, Cromwell Florence, ed. Occupational therapy for people with eating dysfunctions. New York: Haworth Press, 1986.

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6

Cohealing: The shared quest for optimal well-being. [California?]: Millennia Books, 1997.

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7

She's come undone. New York: Washington Square Press, 1992.

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8

Meadow, Rosalyn M. Women's conflicts about eating and sexuality: The relationship between food and sex. New York: Harrington Park Press, 1992.

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9

Lillie, Weiss, ed. Women's conflicts about eating and sexuality: The relationship between food and sex. New York: Haworth Press, 1992.

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10

Excessive appetites: A psychological view of addictions. Chichester: Wiley, 1992.

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11

Excessive appetites: A psychological view of addictions. Chichester: Wiley, 1985.

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12

Herbert, Beate M., and Olga Pollatos. The relevance of interoception for eating behavior and eating disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0009.

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The importance of interoception for adaptive and maladaptive behavior, as well as for psychopathology, has gained growing interest, and dysfunctional interoception has been recognized as representing a core impairment across psychosomatic and psychiatric disorders. Eating is intrinsically guided by interoceptive signals and is directly associated with homeostatic psychophysiological needs, well-being, and survival. This chapter provides conceptually and empirically drawn conclusions focusing on the relevance of distinguishable dimensions of interoception for shaping eating behavior and body weight, and for eating disorders. Going beyond eating behavior per se, anorexia and bulimia nervosa are conceptualized as characterized by profound impairment of the self, with dysfunctional interoception at its core. Predictive coding models are addressed to integrate conclusions and empirical findings tentatively.

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13

What's Eating Gilbert Grape. Simon & Schuster, 1999.

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14

What's Eating Gilbert Grape. Tandem Library, 1999.

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15

Hedges, Peter. What's Eating Gilbert Grape. RosettaBooks, 2014.

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16

Prevalence of disordered eating and menstrual dysfunction in female intercollegiate athletes. 1995.

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17

Prevalence of disordered eating and menstrual dysfunction in female intercollegiate athletes. 1995.

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18

Prevalence of disordered eating and menstrual dysfunction in female intercollegiate athletes. 1995.

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19

Baskin, Janie. Paint Me a Monster. Enslow Publishing, LLC, 2014.

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20

Paint me a monster. 2014.

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21

Baskin, Janie. Paint Me a Monster. Enslow Publishing, LLC, 2014.

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22

Caga, Jashelle, and Matthew C. Kiernan. Bulbar dysfunction in ALS: Psychological implications. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757726.003.0015.

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Bulbar dysfunction typically manifests as speech and swallowing impairment in amyotrophic lateral sclerosis (ALS). Timely assessment of changes in speech and swallowing is imperative, given its negative prognostic implication and impact on psychological well-being. The progressive loss of the ability to speak and swallow can also result in threats to the self-concept, which may compound issues with social interaction. The use of communication devices to accommodate loss of speech appears to be beneficial in reducing patient distress and caregiver burden. Implementation of interventions to manage problems eating secondary to swallowing impairment can also result in marked improvements in patients’ and caregivers’ quality of life. However, the success of these interventions depends on intact cognitive and behavioural functioning, which may be compromised in patients with bulbar dysfunction. Assessment of bulbar dysfunction should therefore be considered in the context of cognitive and behavioural change, to maximize patient and caregiver psychological well-being.

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23

Abrahams, Sharon, and Christopher Crockford. Cognitive and behavioural dysfunction in ALS and its assessment. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757726.003.0008.

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Cognitive and behavioural dysfunction in amyotrophic lateral sclerosis (ALS) occurs in up to half of patients with a spectrum from ALS with no cognitive or behavioural impairment to ALS with frontotemporal dementia (FTD). ~ 15% have a full blown ALS-FTD syndrome, while ~ 35% show milder and specific deficits on verbal fluency, executive and language functions and social cognition. Patients may show a behavioural syndrome that ranges from mild specific difficulties to changes that fulfil diagnostic criteria for behavioural variant-FTD. Apathy is the most prevalent symptom, but disinhibition, perseveration, loss of sympathy/empathy, and change in eating behaviour are also described. The importance of assessment is increasingly recognized. A distinction is made between brief assessment tools useful within ALS clinics and more extensive neuropsychological assessment by a qualified clinical neuropsychologist. Newly developed assessments specifically designed for ALS are available and will make valuable contribution to clinical care.

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24

A, Linden R. W., ed. The scientific basis of eating: Taste and smell, salivation, mastication and swallowing, and their dysfunctions. Basel: Karger, 1998.

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25

Johnson, J. J. Believarexic. Peachtree Publishing Company, 2017.

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26

Believarexic. Peachtree Publishing Company, 2015.

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27

Bohon, Cara. Research Domain Criteria. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.2.

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A primary goal of the research domain criteria (RDoC) project from the National Institute of Mental Health in the United States is to better characterize and understand the pathology and etiology of mental illness by examining constructs with biological underpinnings and their effects on psychiatric symptoms. This endeavor shows promise in helping to better conceptualize dysfunction in the field of eating disorders, where there appears to be great heterogeneity within diagnostic groups. Treatments designed for a particular diagnosis may result in improved remission rates if they instead target underlying mechanisms of eating disorder symptoms. This system is not without challenge and limitations, however. This chapter includes a brief review of relevant literature on the proposed RDoC functional domains in eating disorders and discussion of the benefits and costs of this type of approach in improving patient care.

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28

Double Duty: Gay Lesbian. Mac Publishing, 1990.

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29

Double Duty: Sexual Abuse. Mac Publishing, 1990.

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30

Black, Claudia. Double Duty. Ballantine Books, 1991.

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31

Double Duty: Food Addiction. Mac Publishing, 1990.

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32

Cromwell, Florence S. Occupational Therapy for People With Eating Dysfunctions (Occupational Therapy in Health Care Ser. 2) (Occupational Therapy in Health Care Ser. 2). Haworth Press, 1986.

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33

Roger, W. A. Ph D. Linden. The Scientific Basis of Eating: Taste and Smell, Salivation, Mastication and Swallowing and Their Dysfunctions (Frontiers of Oral Biology, Vol. 9). S. Karger Publishers (USA), 1998.

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34

Dr. Sebi Cure for Erectile Dysfunction Made Simple: The Absolute Guide on How to Cure ED Through Dr. Sebi Alkaline Diet Eating Habits. Independently Published, 2020.

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35

Hawton, Keith, Paul M. Salkovskis, Joan Kirk, and David M. Clark, eds. Cognitive Behaviour Therapy for Psychiatric Problems. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780192615879.001.0001.

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This title provides a summary of the cognitive behavioural principles on which the therapy is based, followed by a detailed account of how to carry out a cognitive–behavioural assessment. Subsequent chapters provide self-contained descriptions of how to use the therapy to treat particular conditions: panic and generalized anxiety, phobic disorders, depression, obsessional disorders, somatic problems, eating disorders, chronic psychiatric handicaps, marital problems, and sexual dysfunctions. A final chapter provides a description of problem-solving training. Each chapter describes the condition, assessment procedures, factors likely to be important in formulating the problem, and then the treatment, step-by-step. Particular attention is paid to overcoming difficulties encountered during treatment, and extensive use is made of clinical material and case illustrations. This is an excellent guide to the practice of cognitive behaviour therapy for all those beginning to use the technique.

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36

Excessive Appetites: A Psychological View of Addictions, 2nd Edition. Wiley, 2001.

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37

Excessive Appetites: A Psychological View of Addictions. 2nd ed. Wiley, 2001.

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38

Orford, Jim. Excessive Appetites: A Psychological View of Addictions. John Wiley & Sons Ltd (Import), 1995.

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39

Orford, Jim. Excessive Appetites: A Psychological View of Addictions. Wiley & Sons, Incorporated, John, 2008.

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40

Richards, C. Steven, and Michael W. O'Hara, eds. The Oxford Handbook of Depression and Comorbidity. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.001.0001.

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Depression is frequently associated with other psychiatric disorders, chronic health problems, and distressed close relationships. This comorbidity between depression and other disorders and problems is important. Furthermore, there has been a large increase in research on depressive comorbidity. Therefore, a book of 37 state-of-the-art reviews by experts will be helpful to teachers, researchers, practitioners, developers of relevant policies, and students in these areas. The comorbidity of depression with other psychiatric disorders is addressed in chapters focusing on panic disorder, post-traumatic stress disorder, social anxiety disorder, generalized anxiety disorder, alcohol-use disorders, eating disorders, conduct disorder, personality disorders, sexual dysfunctions, schizophrenia, suicide, and bipolar disorder. The comorbidity of depression and chronic health problems is addressed in chapters focusing on cardiovascular disease, cancer, pain, obesity, sleep disorders, multiple sclerosis, acquired immune deficiency syndrome, kidney disease, dementia, and women's health. The comorbidity of depression and distressed close relationships is addressed in chapters on intimate relationships, family relationships, and perinatal depression. There are also chapters on diagnostic issues, theory and constructs, models of comorbidity between depression and anxiety, assessment strategies, multidisciplinary treatments, community interventions, treatment in ethnic minority groups, psychosocial interventions for depressed cancer patients, and cognitive therapy for comorbid depression. Finally, in an effort to integrate the material, there are introduction, big picture, and epilogue chapters. The 37 chapters in this book reflect a scholarly and evidence-based perspective on depressive comorbidity. Moreover, the chapters address a wide array of relevant issues, including etiology, assessment, diagnosis, course, theory, research, practice, treatment, and clinical guidelines. In summary, this edited book includes 37 chapters on depression and comorbidity, and thereby provides a comprehensive, scholarly, and empirically-based compendium of reviews on this topic.

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41

Quântica, Sabrina. Depressão, em busca da libertação - Um estudo sobre a cura sem medicamentos. Brazil Publishing, 2021. http://dx.doi.org/10.31012/978-65-5861-424-1.

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Is it possible to develop techniques and mechanisms so that women can get out of a depression without the help of medication, even though they are in a deep state of apathy? It is scientifically proven that moving, eating well, cultivating good relationships and even meditating are actions that help the human being to become fuller and happier. The recipe is easy. There is a step-by-step that, most of the time, provides positive results to those who follow it. But what about when the individual is listless? At that stage of unwillingness, lack of strength and courage to change the stage? He may be fully aware of what he should do, how he should act and how much it would be beneficial for his well-being, yet he still does not find the strength to act. It is a feeling of pain and anguish that does not pass and there is no desire to do anything to pass. Would it be possible to use or create a revolutionary technique or set of techniques, "magical" that help the start of psychological / emotional change, without the aid of drugs? There are numerous studies that demonstrate both the ineffectiveness and the various side effects of antidepressant and psychotropic drugs. To check if there are mechanisms that help individuals to restore mental and emotional health without the aid of allopathy is to find a way out so that less people, in addition to not getting rid of depression, become dependent on the medications they use. Nowadays depression is often diagnosed and treated incorrectly, especially among women – one in seven is medicated. If there are effective mechanisms to change a depressive stage in a natural way, there will be a reduction in the consumption of medications, thus avoiding serious side effects such as dependence, reactions and physical changes such as disorderly weight loss or gain, sexual dysfunction and incapacity to live in society.

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42

Reddy, Ugan, and Nicholas Hirsch. Diagnosis, assessment, and management of myasthenia gravis and paramyasthenic syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0244.

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Diseases that affect the neuromuscular junction (NMJ) interfere with normal nerve transmission and cause weakness of voluntary muscles. The two most commonly encountered are acquired myasthenia gravis (MG) and the Lambert–Eaton myasthenic syndrome (LEMS). Acquired MG is an autoimmune disease in which antibodies are directed towards receptors at the NMJ. In 85% of patients, IgG antibodies against the postsynaptic acetylcholine receptor (AChR) are found (seropositive MG). The thymus gland appears to be involved in the production of these which cause an increase rate of degradation of AChR resulting in a decreased receptor density resulting in a reduced postsynaptic end-plate potential following motor nerve stimulation and leading to muscle weakness. Although all voluntary muscles can be affected, ocular, bulbar, respiratory, and proximal limb weakness predominates. In the majority of seronegative patients, an antibody directed towards a NMJ protein called muscle specific tyrosine kinase (MUSK) is found. Anti-MUSK MG is characterized by severe bulbar and respiratory muscle weakness. Diagnosis of MG requires a high degree of clinical suspicion coupled with pharmacological and electrophysiological testing, and detection of the various causative antibodies. Treatment of MG involves enhancing neuromuscular transmission with long-acting anticholinesterase agents and immunosuppression. Acute exacerbations are treated with either plasma exchange or intravenous immunoglobulin. Myasthenic crisis is associated with severe muscle weakness that necessitates tracheal intubation and mechanical ventilation. LEMS is an autoimmune disease in which IgG antibodies are directed towards the pre-synaptic voltage-gated calcium channels at the NMJ. It is often associated with malignant disease (usually small cell carcinoma of the lung). Autonomic dysfunction is prominent and patients show abnormal responses to neuromuscular blocking drugs.

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