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Books on the topic 'Dyscrasite'

Author: Grafiati

Published: 6 September 2023

Last updated: 7 September 2023

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1

Roccaro, Aldo M., and Irene M. Ghobrial, eds. Plasma Cell Dyscrasias. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5.

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2

Kelly, John J., Robert A. Kyle, and Norman Latov. Polyneuropathies Associated with Plasma Cell Dyscrasias. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2065-4.

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3

Minetti, Luigi, Giuseppe D’Amico, and Claudio Ponticelli, eds. The Kidney in Plasma Cell Dyscrasias. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1315-8.

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4

1928-, Kyle Robert A., and Latov Norman, eds. Polyneuropathies associated with plasma cell dyscrasias. Boston: Nijhoff, 1987.

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5

L, Minetti, D'Amico G. 1929-, and Ponticelli C, eds. The Kidney in plasma cell dyscrasias. Dordrecht: Kluwer Academic Publishers, 1988.

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6

Zimmerman, Todd M., and Shaji K. Kumar, eds. Biology and Management of Unusual Plasma Cell Dyscrasias. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4419-6848-7.

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7

W, Pruzanski, and Keystone E. C, eds. Paraproteins in disease: Investigation of plasma cell dyscrasia. Edinburgh: Churchill Livingstone, 1985.

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8

W, Pruzanski, and Keystone E. C, eds. Paraproteins in desease: Investigation of plasma cell dyscrasia. Edinburgh: Churchill Livingstone, 1985.

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9

Ghobrial, Irene M., and Aldo M. Roccaro. Plasma Cell Dyscrasias. Springer, 2018.

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10

Plasma Cell Dyscrasias. Springer, 2016.

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11

Ghobrial, Irene M., and Aldo M. Roccaro. Plasma Cell Dyscrasias. Springer London, Limited, 2016.

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12

D'Amico, G., Luigi Minetti, and G. Ponticelli. Kidney in Plasma Cell Dyscrasias. Springer London, Limited, 2012.

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13

Herrera, G. A., ed. The Kidney in Plasma Cell Dyscrasias. S. Karger AG, 2006. http://dx.doi.org/10.1159/isbn.978-3-318-01397-9.

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14

Ronco, Pierre M. Kidney involvement in plasma cell dyscrasias. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0150.

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Monoclonal proliferations of the B-cell lineage are characterized by abnormal and uncontrolled expansion of a single clone of B cells at different maturation stages, with a variable degree of differentiation to immunoglobulin-secreting plasma cells. Therefore, they are usually associated with the production and secretion in blood of a monoclonal immunoglobulin and/or a fragment thereof which may become deposited in tissues. These deposits can take the form of casts (in myeloma cast nephropathy), crystals (in myeloma-associated Fanconi syndrome), fibrils (in light-chain and exceptional heavy-chain amyloidosis), or granular precipitates (in monoclonal immunoglobulin deposition disease). They may disrupt organ structure and function, inducing life-threatening complications. All of the pathologic entities related to immunoglobulin deposition principally involve the kidney, which is not only explained by the high levels of renal plasma flow and glomerular filtration rate, but also by the sieving properties of the glomerular capillary wall and by the prominent role of the renal tubule in LC handling and catabolism.The different renal (and other) manifestations are related to the unique physicochemical characteristics of each paraprotein or immunoglobulin fragment, and the rate of their production.

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15

Herrera, G. A. The Kidney in Plasma Cell Dyscrasias. Not Avail, 2006.

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16

The kidney in plasma cell dyscrasias. Basel: Karger, 2007.

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17

The kidney in plasma cell dyscrasias. Springer, 2011.

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18

J, Kelly John, Norman Latov, and Robert A. Kyle. Polyneuropathies Associated with Plasma Cell Dyscrasias. Springer, 2011.

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19

Polyneuropathies Associated with Plasma Cell Dyscrasias. Springer, 2011.

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20

J, Kelly John, Norman Latov, and Robert A. Kyle. Polyneuropathies Associated with Plasma Cell Dyscrasias. Springer London, Limited, 2012.

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21

D'Amico, G., Luigi Minetti, and G. Ponticelli. The kidney in plasma cell dyscrasias. Springer, 2011.

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22

Zimmerman, Todd M., and Shaji K. Kumar. Biology and Management of Unusual Plasma Cell Dyscrasias. Springer, 2016.

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23

Zimmerman, Todd M., and Shaji K. Kumar. Biology and Management of Unusual Plasma Cell Dyscrasias. Springer London, Limited, 2016.

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24

Biology and Management of Unusual Plasma Cell Dyscrasias. Springer, 2018.

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25

Biology and Management of Unusual Plasma Cell Dyscrasias. Springer, 2016.

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26

Skarin, Arthur T. Slide Atlas of Diagnostic Oncology Unit 13: Slide Atlas of Multiple Myeloma and Plasma Cell Dyscrasias. Mosby, Incorporated, 1991.

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27

Elham, Bayat. Neurologic Manifestations of Hematological Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0193.

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A wide sprectum of hematologic disorders affect the central and peripheral nervous system. These disorders include porphyria, thrombotic thrombocytopenic purpura-hemolytic uremic syndromes, sickle cell disease, plasma cell dyscrasias, monoclonal gammopathy, primary systemic amyloidosis, primary systemic amyloidosis, Waldonstrom’s macroglobulinemia, myeloproliferative syndromes, cryoglobulinemia, and polycythemia vera. Some, like porphyria, cause both central and peripheral nervous system manifestations including sensory/motor peripheral neuropathy, dysautonomia, pain, seizures, and abdominal pain. Others such as sickle cell disease primarily affect the brain and cause both clinically apparent strokes associated with a vasculopathy of large intracranial blood vessels, as well as less obvious microstrokes that cause progressive cognitive decline if not treated.

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28

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff. Bone and soft tissue malignancies. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199689842.003.0025.

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Haematological malignancies examines the epidemiology, genetics, clinical presentation and classification of these diseases, and presents current treatment approaches for each. First are the acute leukaemias, and the management of acute lymphoblastic leukaemia (ALL) and acute myeloid leukaemia (AML). Chronic myeloid leukaemia, its genetics and sensitivity to tyrosine kinase inhibitors, is described. Myelodysplastic syndromes and their management, are followed by chronic lymphoid leukaemias, a large heterogeneous group of diseases, and their treatment. Hodgkin lymphoma, its pathology and presentation, staging and role of PET scanning, is described along with current treatment with chemotherapy and limited radiotherapy. Non-Hodgkin lymphoma is another heterogeneous group of diseases, divided into low-grade and high-grade pathology, and varying in their genetics, presentation, and management. Rituximab is a key component of chemotherapy regimens against B-cell lymphoma. Myeloma and other plasma cell dyscrasias are described, and treatment options reviewed. Myeloma remains incurable, but with appropriate management consistent with prolonged good quality life. Treatment includes chemotherapy, bisphosphonate therapy, analgesics and radiotherapy, Throughout this chapter is emphasised the importance of clinical trials in driving the rapid improvements in treatment of these diseases.

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