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Academic literature on the topic 'Dyscrasite'

Author: Grafiati

Published: 6 September 2023

Last updated: 7 September 2023

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Journal articles on the topic "Dyscrasite"

1

Osadchii, E., D. Boström, S. Lunin, and E. Rosén. "Thermodynamic properties of dyscrasite (Ag 3 Sb) at isostatic pressures 4000-8000 bar." Physics and Chemistry of Minerals 25, no. 4 (March 19, 1998): 288–91. http://dx.doi.org/10.1007/s002690050116.

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Kalinin, Arkadii A., Yevgeny E. Savchenko, and Ekaterina A. Selivanova. "Mustard Gold in the Oleninskoe Gold Deposit, Kolmozero–Voronya Greenstone Belt, Kola Peninsula, Russia." Minerals 9, no. 12 (December 13, 2019): 786. http://dx.doi.org/10.3390/min9120786.

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The Oleninskoe intrusion-related gold–silver deposit is the first deposit in the Precambrian of the Fennoscandian Shield, where mustard gold has been identified. The mustard gold replaces küstelite with impurities of Sb and, probably, gold-bearing dyscrasite and aurostibite. The mosaic structure of the mustard gold grains is due to different orientations and sizes of pores in the matrix of noble metals. Zonation in the mustard gold grains is connected with mobilization and partial removal of silver from küstelite, corresponding enrichment of the residual matter in gold, and also with the change in the composition of the substance filling the pores. Micropores in the mustard gold are filled with iron, antimony or thallium oxides, silver chlorides, bromides, and sulfides. The formation of mustard gold with chlorides and bromides shows that halogens played an important role in the remobilization of noble metals at the stage of hypergene transformation of the Oleninskoe deposit.

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Zachariáš, Jiří, and Matěj Němec. "Gold to aurostibite transformation and formation of Au-Ag-Sb phases: the Krásná Hora deposit, Czech Republic." Mineralogical Magazine 81, no. 4 (August 2017): 987–99. http://dx.doi.org/10.1180/minmag.2016.080.145.

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AbstractRare phases of the Au–Ag–Sb system were recognized in the Krásná Hora Sb-Au deposit (Sb 1.5–3 wt.%; Au 3–5 ppm), Czech Republic which correspond to auriferous dyscrasite (up to 7 at.% Au), auriferous allargentum (up to 34 at.% Au), and an unnamed phase with composition similar to the eutectics (E1, E2) of the experimental Au–Ag–Sb system. The dominant ore mineral is stibnite with rare native antimony, native gold and a Ag-Au alloy. Textural relationships are well established: stibnite (early) →gold → aurostibite → native antimony (late). Gold is present in four generations: Au-1 (0–15 at.% Ag) is the most abundant type; Au-2 (20–70 at.% Ag) forms thin rims along intra-grain boundaries of Au-1; Au-3 and Au-4 are rare and almost pure (∼0 at.% Ag). The formationof most of the Au-2 and of Au-Ag-Sb phases is associated with Ag-mobilization coupled with the Au-1 to aurostibite transformation via dissolution-precipitation and solid-state diffusion processes at temperatures <200°C.

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Hartshorn, Edward A., Gary M. Levin, and C. Lindsay DeVane. "A Review of Cyclic Antidepressant-Induced Blood Dyscrasias." Annals of Pharmacotherapy 26, no. 3 (March 1992): 378–83. http://dx.doi.org/10.1177/106002809202600313.

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OBJECTIVE: To review the literature for cases of blood dyscrasias associated with cyclic antidepressants. Several types of blood dyscrasias are discussed. DATA SOURCES: All references were selected through the use of MEDLINE. Indexing terms were blood, abnormalities, dyscrasias, antidepressants, agranulocytosis, and eosinophilia. The only constraints were English language and human subjects. STUDY SELECTION: All cases were included except for letters to the editor of various journals when pertinent data such as doses and additional medications were omitted. DATA SYNTHESIS: The review provides a table listing the different blood dyscrasias and the drug the patient was receiving. The table also includes time of onset, time to recovery, and several symptoms for each patient. CONCLUSIONS: Common symptoms of various blood dyscrasias are discussed. The chemical structures of the antidepressants are related to phenothiazines, which are also implicated in causing blood dyscrasias. Recommendations for treatment of both the dyscrasia and depression are discussed.

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Latif, Zahid, Faraz Jabbar, and Brendan D. Kelly. "Clozapine and blood dyscrasia." Psychiatrist 35, no. 1 (January 2011): 27–29. http://dx.doi.org/10.1192/pb.bp.109.026054.

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SummaryClozapine is an effective antipsychotic medication but is associated with agranulocytosis, neutropenia and leucopenia. The reintroduction of clozapine improved management of treatment-resistant schizophrenia, yet resulted in a paradoxical situation whereby the risk of blood dyscrasias is rigorously managed but other adverse effects (e.g. seizures, cardiovascular complications) are less well monitored. Monitoring of weight, lipids, plasma glucose and other metabolic parameters is recommended. There is also a need to reconsider routine haematological monitoring with other medications associated with blood dyscrasia (e.g. phenothiazines, carbamazepine). In particular, individuals who develop clozapine-induced blood dyscrasia may require haematological monitoring during treatment with other antipsychotics.

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Sutherland, Rory A., and Richard I. Crawford. "Scalp biopsy identifies systemic amyloidosis presenting as isolated telogen effluvium: A case report." SAGE Open Medical Case Reports 7 (January 2019): 2050313X1984778. http://dx.doi.org/10.1177/2050313x19847783.

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AL amyloidosis is a complication of B-cell dyscrasias and multiple myeloma, manifest as deposition of antibody fragments in many different organs, including the skin. We describe a rare case of this systemic disease which presented with isolated scalp alopecia. Further investigation led to the diagnosis of an occult plasma-cell dyscrasia, showing the benefit of including systemic amyloidosis in the differential diagnosis of alopecia. The biopsy finding of cutaneous amyloidosis should prompt further workup to exclude an underlying pathology.

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Dunk, Louisa R., Linda J. Annan, and Christopher D. Andrews. "Rechallenge with clozapine following leucopenia or neutropenia during previous therapy." British Journal of Psychiatry 188, no. 3 (March 2006): 255–63. http://dx.doi.org/10.1192/bjp.188.3.255.

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BackgroundFurther treatment with clozapine is contraindicated in any patient who has previously experienced leucopenia or neutropenia during clozapine therapy.AimsTo investigate the results of such a rechallenge in 53 patients.MethodAn analysis was made of the demographic, haematological and outcome data of patients in the UK and Ireland who were rechallenged with clozapine following leucopenia or neutropenia during previous clozapine therapy.ResultsOf 53 patients who were rechallenged, 20 (38%) experienced a further blood dyscrasia. In 17 of these 20 patients (85%) the second blood dyscrasia was more severe(P<0.001), in 12(60%) it lasted longer (P=0.0368) and in 17 (85%) it occurred more quickly on rechallenge (P<0.001). Of the original 53 patients, 55% (29 patients) are still receiving clozapine.ConclusionsNo clear risk factor for repeat blood dyscrasias was identified. Despite this, after risks and benefits have been considered, rechallenge may well be justified in some patients.

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Harris, Donald C., Andrew C. Roberts, J. H. Gilles Laflamme, and Chris J. Stanley. "Criddleite, TlAg2Au3Sb10S10, a New Gold-Bearing Mineral from Hemlo, Ontario, Canada." Mineralogical Magazine 52, no. 368 (December 1988): 691–97. http://dx.doi.org/10.1180/minmag.1988.052.368.13.

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AbstractCriddleite, ideally TlAg2Au3Sb10S10, is a rare constituent within the Hemlo gold deposit, Hemlo, Ontario, Canada. The mineral occurs as 20 to 50 µm-sized lath-like, tabular or anhedral grains usually surrounding or penetrating aurostibite, or associated with native antimony, native gold and stibnite. Criddleite is opaque with a metallic lustre and a black streak. It has been synthesized by reacting TlSbS2 and high purity Ag, Au, Sb and S in an evacuated silica glass tube at 400 °C. The measured density of the synthetic material is 6.86; the calculated density is 6.57 g/cm3. The difference is due to minor admixed aurostibite, native antimony and a dyscrasite-like phase within the charge. VHN25 is 94–129. Mohs hardness (calc.) = 3–3 ½. In reflected plane-polarized light in air, natural criddleite is weakly bireflectant with a discernible reflectance pleochroism from grey-blue to slightly greenish grey-blue. The mineral has a distinct to moderate anisotropy with rotation tints in shades of buff to slate grey. Reflectance spectra and colour values for both natural and synthetic criddleite are given. X-ray study showed that synthetic criddleite is monoclinic (pseudotetragonal) with refined unit-cell parameters a = 20.015(2), b = 8.075(2), c = 7.831(2) Å, β = 92.01(2)°, V = 1264.9 ± 1.0 Å3 and a:b:c = 2.4786: 1:0.9698. The space group choices are A2/m(12), A2(5) or Am(8), diffraction aspect A*/*. The seven strongest lines in the X-ray powder diffraction pattern [d in Å (I) (hkl)] are: 5.63(90) (011), 3.91(50) (002), 3.456(50) (320), 2.860(70) (700), 2.813(100) (022), 2.018(60) (040) and 1.959(70) (004). Electron microprobe analyses are reported of natural criddleite in five polished sections of drill core from four holes. The averaged empirical formulae, based on 26 atoms, are Tl0.92Ag1.99Au2.93Sb9.87S10.28 (natural) and Tl0.94Ag2.03Au2.89Sb9.76S10.38 (synthetic).

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Munshi, Tariq, Farooq Naeem, Mohammed Ayub, Saeed Farooq, Davit Khachatryan, Selim Asmer, Peter Wang, et al. "The haematological side effects of clozapine: literature review and meta-analysis." BJPsych Open 7, S1 (June 2021): S274. http://dx.doi.org/10.1192/bjo.2021.729.

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AimsThis review and meta-analysis aim to estimate the cumulative incidence of clozapine induced agranulocytosis and leukopenia the impact of the associated factors such as dose of clozapine, duration of follow-up, gender and race on the cumulative incidence.BackgroundClozapine is the only medication licensed for treatment-resistant schizophrenia. There has been a renewed interest in the role of Clozapine in the treatment of Schizophrenia based on strong evidence that favours its efficacy and safety. Despite the evidence that Clozapine has superior efficacy and has been recommended for treatment-resistant cases by the national guidelines, the drug is underutilised.MethodWe included all studies in which clozapine was used for a psychotic illness. We included studies which provided data on two primary indices; Leucopenia or agranulocytosis and neutropenia; defined according to the cut off used by CPMS for total WBC and neutrophil count. Additionally we included studies reporting another blood dyscrasia or death due to agranulocytosis. Studies were identified by searching AMED, BIOSIS, CINAHL, EMBASE, MEDLINE, PsycINFO, PubMed, and registries of Clinical Trials and their monthly updates, hand searches, gray literature, and conference proceedings from the first available date until 2nd February, 2015. The search was updated on 15th March, 2017. The Protocol was initiated and then registered with PROSPERO International prospective register of systematic reviews University of York, Centre for Reviews and Dissemination.ResultThe cumulative incidence of the agranulocytosis in all studies was 00.32 % (CI 00.1-0.63). The cumulative incidence in all studies for different types of blood dyscrasia were following: leucopenia 00.96 % (CI 0.39-1.70), neutropenia 2.93 % (CI 1.49-4.72), other blood dyscrasias 4.64% (CI 2.34-7.52) and any blood dyscrasia was 2.23 (CI 1.46-3.12).ConclusionThe limitations of this review are mainly due to the nature of evidence from the included studies. We adopted a broad inclusion criteria to include all the available evidence. Number of patients started on Clozapine may be withdrawn from the Clozapine on the earliest signs of blood dyscrasias since the introduction of Clozapine monitoring services. This means that the true incidence of agranulocytosis and neutropenia may be higher and this may be a major bias in finding the true incidence of Clozapine induced neutropenia.

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Michaeli, J., R. Niesvizky, D. Siegel, M. Ladanyi, PH Lieberman, and DA Filippa. "Proteinaceous (angiocentric sclerosing) lymphadenopathy: a polyclonal systemic, nonamyloid deposition disorder." Blood 86, no. 3 (August 1, 1995): 1159–62. http://dx.doi.org/10.1182/blood.v86.3.1159.1159.

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Abstract Proteinaceous lymphadenopathy with hypergammaglobulinemia (PLWH) is an exceedingly rare disease of unknown etiology. Described primarily as a pathologic entity, relatively little is known about its clinical manifestations or its response to therapy. The disease is often referred to and treated as an unusual form of plasma cell dyscrasia or light chain deposition disease. We have recently encountered a young patient with PLWH who presented with generalized lymphadenopathy, marked liver function abnormalities, hypocomplementemia, cryoglobulinemia, decreased T4/T8 ratio, and ophthalmopathy. Contrary to the notion that PLWH is a clonal disorder, we found no evidence of clonality in this patient. The most characteristic finding in this and in another patient, previously seen at our institution, was marked angiocentric hyaline sclerosis of the small and mid-sized blood vessels of involved lymph nodes and organs. Based on these findings, we propose the term angiocentric sclerosing lymphadenopathy, which more accurately defines this clinicopathologic entity that appears to be distinct from light chain deposition disease and other plasma cell dyscrasias.

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Dissertations / Theses on the topic "Dyscrasite"

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Ali, H. S. "Drug induced blood dyscrasias : Aminoglutethimide and related compounds." Thesis, Cardiff University, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.377874.

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Limpas, Yvon. "Le POEMS syndrome : entité particulière au sein des dyscrasies plasmocytaires." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25325.

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Paletta, Dominique Raoux. "Syndrome de Crow-Fukase : à propos d'une observation et revue de la littérature." Montpellier 1, 1988. http://www.theses.fr/1988MON11235.

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Soubrier, Martin. "Le Syndrome POEMS." Clermont-Ferrand 1, 2003. http://www.theses.fr/2003CLF1MM11.

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Gutknecht-Dignat, Sandrine. "Le syndrome d'hyperperméabilité capillaire idiopathique : à propos d'un cas." Montpellier 1, 1998. http://www.theses.fr/1998MON11040.

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CARLISI, Melania. "EVALUATION AND POTENTIAL ROLE OF BONE MARROW TISSUE-RESIDENT MEMORY T-CELLS IN PATIENTS WITH PLASMA CELL DYSCRASIAS." Doctoral thesis, Università degli Studi di Palermo, 2020. http://hdl.handle.net/10447/400371.

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MILANI, PAOLO. "The use of mass spectrometry for the diagnosis of plasma cell dyscrasias: focus on light chain (AL) amyloidosis patterns." Doctoral thesis, Università degli studi di Pavia, 2019. http://hdl.handle.net/11571/1285988.

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Kiran, Aramanda Shanmukha. "Exotic eutectic microstructures." Thesis, 2021. https://etd.iisc.ac.in/handle/2005/5476.

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Solidification of eutectic systems delivers compelling examples of microstructure formation, which makes the phenomena intriguing to many engineers and scientists. Therefore, eutectic solidification is extensively studied experimentally, theoretically, and numerically. However, some exotic eutectic microstructures are still not understood, particularly the systems that exhibit anisotropy. The main objective of this thesis is to explore the microstructure formation in exotic systems during solidification. As directional solidification is a convenient way (imposes the temperature gradient, solidification velocity accurately and independently) to study the microstructure formation, the first part of the work was directed towards the development of directional solidification configuration. Subsequently, we investigated the microstructure formation in three different exotic eutectic systems. In the first system (Sn-Zn eutectic), we study the formation of two-phase microstructures with endeavors to bring new inferences, as the volume percentage of the (Zn)-phase in the eutectic is less than 10%, so one would expect it to form rods in the matrix of (Sn)-phase; instead, thin lamellae are observed. We claim that the rod-lamellar transition and well-defined lamellar orientation are due to the anisotropy of the free energy of the solid-solid interfaces. We deploy various methods/experiments for confirming the evidence of solid-solid interface anisotropy. We also provide the crystallographic orientation relationships between BCT-(Sn) and HCP-(Zn) in steady-state microstructures. In the second system (SnTe-Te eutectic), we investigate the evolution of complex patterns due to the addition of ternary impurities in the Sn-Te eutectic system that contains trigonal (Te) and an intermetallic SnTe phase with a cubic crystal structure. In this work, we examine the origin of such a microstructure that arises due to a two-phase growth instability induced by impurity addition. The binary eutectics (Sn-Te) and ternary eutectics (Sn-Te with an impurity addition) are directionally solidified at different interfacial velocities in order to study morphological evolution. The binary alloy exhibits a rod-like or an interconnected string of rods morphology, while the addition of a third component leads to a diffusive instability (similar to a Mullins-Sekerka instability) that results in the formation of two-phase colonies. The onset of instability depends on both the growth velocity and impurity concentration, while the growth direction of the cells is normal to the (0001) of (Te) and (111) of SnTe. Through the extensive use of multiple characterization techniques, we have explored the morphological characteristics and crystallography of these colonies. The colonies have a complex internal structure that bears a three-fold symmetry reminiscent of the trigonal symmetry of the (Te) crystal, arising possibly because of strong anisotropy in the solid-liquid interfacial energy or in the kinetics of growth. For the different impurity additions (Ag, Cu, Ge, In, Sb), the internal eutectic morphology of the colony due to the addition of Ag, Cu, Ge, In is different from that observed for the addition of Sb. The latter leads to the formation of lamellae, while a rod-like feature could be observed for impurities Ag, Cu, Ge, In. In the third system (Ag-Cu-Sb), we have investigated the formation of three-phase microstructures of the Ag-Cu-Sb eutectic system that contains two intermetallic compounds, i.e., silver antimonide-Ag3Sb, copper antimonide-Cu2Sb and an antimony rich solid-solution (Sb). A vast range of microstructures in this system arise due to the possibilities of both invariant reactions giving rise to three-phase eutectic growth as well univariant reactions that are amenable to diffusive instabilities giving rise to microstructures involving two-phase colonies along with three-phase eutectic morphologies. The different ternary compositions are morphologically and crystallographically investigated at various velocities. The invariant three-phase eutectic reactions give rise to exotic hollow, dog bone, and fibrous (Sb) crystals along with lamellar/rod type morphologies of the Cu2Sb phase, while the Ag3Sb has a continuous morphology. The different microstructures have an underlying crystallographic basis and distinct growth mechanisms that are influenced by the crystal orientation with the imposed temperature gradient. Among the compositions that give rise to two-phase colonies, an exciting structure emerges in the Ag3Sb-(Sb) two-phase colonies that exhibit a complex 3-fold fish skeleton structure reminiscent of the rhombohedral (Sb) crystals. Similarly, the Ag3Sb-Cu2Sb colonies exhibit a complex plate morphology influenced by the anisotropic nature of the Ag3Sb-Cu2Sb interface.

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Havyarimana, Sandra. "Evaluation of prognostic markers in plasma cell dyscrasias." Thesis, 2017. https://hdl.handle.net/10539/24917.

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A research report submitted to the Faculty of Health Sciences, University of the Witwatersrand, Johannesburg, in partial fulfilment of the requirements for the degree of Master of Medicine in the Branch of Pathology (Haematology). Johannesburg, 2017.
Introduction: Plasma cell dyscrasias (PCDs) are a heterogeneous group of diseases in which there is expansion of clonal plasma cells which may produce monoclonal immunoglobulins. CD200 is a membrane glycoprotein of the type I immunoglobulin superfamily transmembrane glycoprotein. It is postulated that CD200 is expressed by the plasma cells in a significant number of cases of multiple myeloma (MM), but normal plasma cells do not express it. It may thus assist in confirming clonality of plasma cells in PCDs. Aims: To evaluate CD200 expression as a potential diagnostic and prognostic marker in plasma cell dyscrasias and to compare the results with other known prognostic factors. Methods: CD200 expression was evaluated by immunophenotypic analysis on normal, reactive and clonal plasma cells and expressed as a mean fluorescent intensity (MFI) value. Cut-off for positive CD200 was established. A negative population was established using different methodologies to exclude reactive plasma cells. CD200 was then compared with diagnostic and prognostic markers currently in use in our laboratory. Results: An MFI of ≥ 30 was the cut-off for CD200 positivity. CD200 was expressed by 76.5% of PCDs cases. CD200 did not predict anaemia, hypercalcaemia or renal dysfuction which are biochemical criteria for the diagnosis of multiple myeloma. CD200 expression did not correlate with traditional prognostic markers such as beta 2 microglobulin (β2M), lactate dehydrogenase (LDH) or genetic abnormalities (by fluorescence in situ hybridization analysis). Cases with plasma cell CD200 expression had a higher monoclonal band level compared to those without CD200 expression. Conclusion: CD200 is a useful tool to evaluate plasma cell clonality; however in our settings where there is a high prevalence of infectious diseases, particularly HIV infection, CD200 might yield false positive results most likely because of small oligoclones of reactive plasma cells. CD200 as a prognostic tool in PCDs could not be confirmed in this small study.
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Phipps, Jonathan E. "Toward Personalized Medicine: The potential role of RNA interference in Plasma Cell Dyscrasia." 2011. http://trace.tennessee.edu/utk_graddiss/1217.

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A major contributor to mortality in patients with plasma cell dyscrasias (PCDs); i.e., multiple myeloma, light chain deposition disease and AL amyloidosis is the deposition as insoluble aggregates of monoclonal immunoglobulin light chain proteins (LC) in the kidneys and other organs. Currently anti-plasma cell chemotherapies are used to reduce LC synthesis, and slow deposition. While effective, these treatments are toxic, non-specific, expensive, and might not be appropriate in all cases, making the identification of an alternate means of reducing toxic LC species desirable. To this end, we have investigated whether RNA interference (RNAi) could achieve these goals. Human (RPMI 8226, Bur) and transfected mouse myeloma (SP2/O-lambda 6) cells which produce measureable quantities of human LC protein were used as model systems for testing the efficacy of both synthetic small interfering RNAs (siRNAs) and short hairpin RNA (shRNA) expression vectors in reducing LC synthesis. Sequencing of LC genes provided the basis for design of siRNA duplexes targeting either the variable (V) or joining (J) regions of individual LCs, or the constant (C) region of either kappa or lambda LC isotypes. Myeloma lines were transfected with siRNAs using lipid-based transfection media. Cells receiving non-silencing siRNAs served as controls. Exposure of myeloma lines to siRNAs was well tolerated and no cytotoxicity was observed. LC mRNA expression was shown to be reduced ≥40% in 8226 and SP2/O- lambda 6 cell lines receiving siRNA treatment as compared with untreated controls. Exposure to siRNAs was also effective in significantly reducing both intracellular and secreted LC protein levels in cell lines tested as evidenced by flow cytometry or enzyme-linked immunosorbent assays (ELISAs). Effective siRNA nucleotide sequences were used to generate shRNA cassettes which were ligated into lentiviral expression vectors under the control of the RNA polymerase III promoter, U6. These expression systems were used to generate replication incompetent lentiviral particles. Exposure of 8226 to lentiviral particles resulted in significant knockdown of LC mRNA and protein both in vitro and in xenograft tumor bearing immune compromised mice. These results provide positive evidence for the ability of RNAi based approaches to reduce LC secretion in models of PCD.

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Books on the topic "Dyscrasite"

1

Roccaro, Aldo M., and Irene M. Ghobrial, eds. Plasma Cell Dyscrasias. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5.

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Kelly, John J., Robert A. Kyle, and Norman Latov. Polyneuropathies Associated with Plasma Cell Dyscrasias. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2065-4.

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Minetti, Luigi, Giuseppe D’Amico, and Claudio Ponticelli, eds. The Kidney in Plasma Cell Dyscrasias. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1315-8.

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1928-, Kyle Robert A., and Latov Norman, eds. Polyneuropathies associated with plasma cell dyscrasias. Boston: Nijhoff, 1987.

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L, Minetti, D'Amico G. 1929-, and Ponticelli C, eds. The Kidney in plasma cell dyscrasias. Dordrecht: Kluwer Academic Publishers, 1988.

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Zimmerman, Todd M., and Shaji K. Kumar, eds. Biology and Management of Unusual Plasma Cell Dyscrasias. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4419-6848-7.

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W, Pruzanski, and Keystone E. C, eds. Paraproteins in disease: Investigation of plasma cell dyscrasia. Edinburgh: Churchill Livingstone, 1985.

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W, Pruzanski, and Keystone E. C, eds. Paraproteins in desease: Investigation of plasma cell dyscrasia. Edinburgh: Churchill Livingstone, 1985.

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Ghobrial, Irene M., and Aldo M. Roccaro. Plasma Cell Dyscrasias. Springer, 2018.

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Plasma Cell Dyscrasias. Springer, 2016.

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Book chapters on the topic "Dyscrasite"

1

Castroviejo, Ricardo. "Dyscrasite (dy, dyscrasite, stibiotriargentite)." In A Practical Guide to Ore Microscopy—Volume 1, 265–68. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-12654-3_44.

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Speer, Tod W., Christin A. Knowlton, Michelle Kolton Mackay, Charlie Ma, Lu Wang, Larry C. Daugherty, Brandon J. Fisher, et al. "Dyscrasias." In Encyclopedia of Radiation Oncology, 168. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-540-85516-3_596.

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Mateos, María-Victoria, and Ola Landgren. "MGUS and Smoldering Multiple Myeloma: Diagnosis and Epidemiology." In Plasma Cell Dyscrasias, 3–12. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_1.

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Neri, Paola, Nizar J. Bahlis, Claudia Paba-Prada, and Paul Richardson. "Treatment of Relapsed/Refractory Multiple Myeloma." In Plasma Cell Dyscrasias, 169–94. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_10.

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Lonial, Sagar. "Treatment of MM: Upcoming Novel Therapies." In Plasma Cell Dyscrasias, 195–205. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_11.

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Lee, Susan J., and Ivan Borrello. "Role of the Immune Response in Disease Progression and Therapy in Multiple Myeloma." In Plasma Cell Dyscrasias, 207–25. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_12.

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Jethava, Yogesh S., and Frits van Rhee. "Transplantation for Multiple Myeloma." In Plasma Cell Dyscrasias, 227–50. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_13.

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Eda, Homare, Loredana Santo, G. David Roodman, and Noopur Raje. "Bone Disease in Multiple Myeloma." In Plasma Cell Dyscrasias, 251–70. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_14.

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Dispenzieri, Angela, and Giampaolo Merlini. "Immunoglobulin Light Chain Systemic Amyloidosis." In Plasma Cell Dyscrasias, 273–318. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_15.

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Kapoor, Prashant, Stephen M. Ansell, and Esteban Braggio. "Waldenstrom Macroglobulinemia: Genomic Aberrations and Treatment." In Plasma Cell Dyscrasias, 321–61. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40320-5_16.

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