Academic literature on the topic 'Dynamic hyperinflation'
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Journal articles on the topic "Dynamic hyperinflation"
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McCarren, Bredge. "Dynamic pulmonary hyperinflation." Australian Journal of Physiotherapy 38, no. 3 (1992): 175–79. http://dx.doi.org/10.1016/s0004-9514(14)60560-2.
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van der Meer, Akke-Nynke, Kim de Jong, Aranka Hoekstra-Kuik, Elisabeth H. Bel, and Anneke ten Brinke. "Dynamic hyperinflation impairs daily life activity in asthma." European Respiratory Journal 53, no. 4 (January 24, 2019): 1801500. http://dx.doi.org/10.1183/13993003.01500-2018.
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IntroductionDynamic hyperinflation has been documented in asthma, yet its impact on overall health and daily life activities is unclear. We assessed the prevalence of dynamic hyperinflation in moderate to severe asthma and its relationship with the scores of a set of specific and general respiratory health questionnaires.Methods77 nonsmoking asthma patients (Global Initiative for Asthma steps 4–5) were recruited consecutively and completed five questionnaires: Asthma Control Questionnaire, Clinical COPD (chronic obstructive pulmonary disease) Questionnaire, St George's Respiratory Questionnaire, London Chest Activity of Daily Living scale (LCADL) and Shortness of Breath with Daily Activities (SOBDA). Dynamic hyperinflation was defined as ≥10% reduction in inspiratory capacity induced by standardised metronome-paced tachypnoea. Associations between level of dynamic hyperinflation and questionnaire scores were assessed and adjusted for asthma severity.Results81% (95% CI 71.7–89.4%) of patients showed dynamic hyperinflation. Higher levels of dynamic hyperinflation were related to poorer scores on all questionnaires (r=0.228–0.385, p<0.05). After adjustment for asthma severity, dynamic hyperinflation remained associated with poorer scores on LCADL (p=0.027) and SOBDA (p=0.031).ConclusionDynamic hyperinflation is associated with poorer overall health and impaired daily life activities, independent of asthma severity. Because of its major impact on everyday life activities, dynamic hyperinflation is an important target for treatment in asthma.
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van Dijk, Marlies, Karin Klooster, Jorine E. Hartman, Nick H. T. ten Hacken, and Dirk-Jan Slebos. "Change in Dynamic Hyperinflation After Bronchoscopic Lung Volume Reduction in Patients with Emphysema." Lung 198, no. 5 (July 24, 2020): 795–801. http://dx.doi.org/10.1007/s00408-020-00382-x.
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Abstract Background and Purpose In patients with severe emphysema, dynamic hyperinflation is superimposed on top of already existing static hyperinflation. Static hyperinflation reduces significantly after bronchoscopic lung volume reduction (BLVR). In this study, we investigated the effect of BLVR compared to standard of care (SoC) on dynamic hyperinflation. Methods Dynamic hyperinflation was induced by a manually paced tachypnea test (MPT) and was defined by change in inspiratory capacity (IC) measured before and after MPT. Static and dynamic hyperinflation measurements were performed both at baseline and 6 months after BLVR with endobronchial valves or coils (treatment group) or SoC (control group). Results Eighteen patients underwent BLVR (78% female, 57 (43–67) years, FEV1 25(18–37) %predicted, residual volume 231 (182–376) %predicted). Thirteen patients received SoC (100% female, 59 (44–74) years, FEV1 25 (19–37) %predicted, residual volume 225 (152–279) %predicted. The 6 months median change in dynamic hyperinflation in the treatment group was: + 225 ml (range − 113 to + 803) (p < 0.01) vs 0 ml (− 1067 to + 500) in the control group (p = 0.422). An increase in dynamic hyperinflation was significantly associated with a decrease in residual volume (r = − 0.439, p < 0.01). Conclusion Bronchoscopic lung volume reduction increases the ability for dynamic hyperinflation in patients with severe emphysema. We propose this is a consequence of improved static hyperinflation.
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Sutherland, Tim J. T., Jan O. Cowan, and D. Robin Taylor. "Dynamic Hyperinflation with Bronchoconstriction." American Journal of Respiratory and Critical Care Medicine 177, no. 9 (May 2008): 970–75. http://dx.doi.org/10.1164/rccm.200711-1738oc.
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van der Meer, Akke-Nynke, Kim de Jong, Aranka Hoekstra-Kuik, Elisabeth H. Bel, and Anneke ten Brinke. "Targeting dynamic hyperinflation in moderate-to-severe asthma: a randomised controlled trial." ERJ Open Research 7, no. 3 (June 3, 2021): 00738–2020. http://dx.doi.org/10.1183/23120541.00738-2020.
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BackgroundDynamic hyperinflation is highly prevalent in moderate-to-severe asthma, which may significantly impede activities of daily life. We hypothesised that dynamic hyperinflation in asthma is due to inflammation of large and small airways and can be reduced by systemic anti-inflammatory treatment. Therefore, we investigated the effect of systemic glucocorticoids on dynamic hyperinflation in moderate-to-severe asthma patients and explored the relationships between inflammatory markers and changes in dynamic hyperinflation.MethodsIn this randomised placebo-controlled trial we included 32 asthma patients on inhaled glucocorticoid therapy showing dynamic hyperinflation, defined by a ≥10% reduction in inspiratory capacity measured by standardised metronome-paced tachypnea test. Patients received either triamcinolone (80 mg) or placebo intramuscularly. Before and 2 weeks after treatment, patients completed respiratory health questionnaires, had blood eosinophils and exhaled nitric oxide levels measured, and underwent lung function and dynamic hyperinflation testing.ResultsAfter adjustment for potential confounders, dynamic hyperinflation was significantly reduced by 28.1% in the triamcinolone group and increased by 9.4% in the placebo group (p=0.027). In the triamcinolone-treated patients, the reduction in dynamic hyperinflation was greater in patients with higher blood eosinophils at baseline (r=−0.592, p=0.020) and tended to be associated with a reduction in blood eosinophils (r=0.412, p=0.127) and exhaled nitric oxide (r=0.442, p=0.099).ConclusionsThis exploratory study suggests that dynamic hyperinflation in asthma can be reduced by systemic anti-inflammatory treatment, particularly in patients with elevated blood eosinophils. This supports the hypothesis that dynamic hyperinflation in asthma is due to airway inflammation and should be considered an important target for treatment.
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van Dijk, Marlies, Jorine E. Hartman, Sonja W. S. Augustijn, Nick H. T. ten Hacken, Karin Klooster, and Dirk-Jan Slebos. "Comparison of Multiple Diagnostic Tests to Measure Dynamic Hyperinflation in Patients with Severe Emphysema Treated with Endobronchial Coils." Lung 199, no. 2 (March 9, 2021): 195–98. http://dx.doi.org/10.1007/s00408-021-00430-0.
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Abstract Purpose For this study, we aimed to compare dynamic hyperinflation measured by cardiopulmonary exercise testing (CPET), a six-minute walking test (6-MWT), and a manually paced tachypnea test (MPT) in patients with severe emphysema who were treated with endobronchial coils. Additionally, we investigated whether dynamic hyperinflation changed after treatment with endobronchial coils. Methods Dynamic hyperinflation was measured with CPET, 6-MWT, and an MPT in 29 patients before and after coil treatment. Results There was no significant change in dynamic hyperinflation after treatment with coils. Comparison of CPET and MPT showed a strong association (rho 0.660, p < 0.001) and a moderate agreement (BA-plot, 202 ml difference in favor of MPT). There was only a moderate association of the 6-MWT with CPET (rho 0.361, p 0.024). Conclusion MPT can be a suitable alternative to CPET to measure dynamic hyperinflation in severe emphysema but may overestimate dynamic hyperinflation possibly due to a higher breathing frequency.
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Van Meerhaeghe, A. "Flow limitation and dynamic hyperinflation." European Respiratory Journal 25, no. 4 (April 1, 2005): 772. http://dx.doi.org/10.1183/09031936.05.00009905.
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Gelb, Arthur F., Carlos A. Gutierrez, Idelle M. Weisman, Randy Newsom, Colleen Flynn Taylor, and Noe Zamel. "Simplified Detection of Dynamic Hyperinflation." Chest 126, no. 6 (December 2004): 1855–60. http://dx.doi.org/10.1378/chest.126.6.1855.
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Hannink, Jorien D. C., Hanneke A. C. van Helvoort, P. N. Richard Dekhuijzen, and Yvonne F. Heijdra. "Dynamic Hyperinflation During Daily Activities." Chest 137, no. 5 (May 2010): 1116–21. http://dx.doi.org/10.1378/chest.09-1847.
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Maurer, J. R. "Simplified Detection of Dynamic Hyperinflation." Yearbook of Pulmonary Disease 2006 (January 2006): 76–77. http://dx.doi.org/10.1016/s8756-3452(08)70069-0.
Full textDissertations / Theses on the topic "Dynamic hyperinflation"
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Orszag, Peter Richard. "Dynamic analysis of regime shifts under uncertainty : applications to hyperinflation and privatization." Thesis, London School of Economics and Political Science (University of London), 1997. http://etheses.lse.ac.uk/2221/.
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Many policy decisions involve discontinuous and irreversible shifts, often in the face of substantial uncertainty. This dissertation studies two types of regime shifts under uncertainty: stabilization from hyperinflation, and privatization of a government corporation. In both cases, policy-makers face decisions that involve uncertainty and irreversibility. These topics are examined using a theoretical model (Chapter 1), econometric evidence (Chapter 2), and a case study (Chapter 3). The first chapter presents a real options model of stabilization given uncertainty over the behavior of inflation. The model is used to argue that despite the costs of hyperinflation, stabilization delays do not necessarily indicate either irrationality or the presence of political economy effects. The second chapter constructs a comprehensive data set of hyperinflations over the past 35 years. The data set is used to examine the empirical regularities of high inflation episodes, and to test the predictions of the model presented in Chapter 1. We conclude that (1) high inflation is rare; (2) most high inflation episodes are short; (3) most economies experience only one high inflation; (4) high inflation is noisy but not necessarily explosive; (5) many high inflations end without a large fall in inflation; and (6) there is weak evidence supporting the real options approach to stabilizations. The third chapter studies another type of regime shift, that of moving a corporation from public to private ownership. The proposed privatization of the U.S. Enrichment Corporation (USEC) illustrates the costs and benefits of privatization. While a private firm would be more efficient, privatization may endanger a crucial nuclear non-proliferation program of the U.S. government. The chapter examines the uranium enrichment market; analyzes the proposed privatization of USEC within a framework developed by Jones, Tandon and Vogelsang (1990); and extends the analysis to incorporate limited information and irreversibility.
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Laveneziana, Pierantonio. "Dynamic lung hyperinflation as the common pathway for exercise-induced dyspnoea in cardio-respiratory diseases." Phd thesis, Université Pierre et Marie Curie - Paris VI, 2012. http://tel.archives-ouvertes.fr/tel-00831616.
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Les patients atteints de BPCO de stade I, d'ICC et d'HTAP peuvent présenter une diminution des débits aériens à bas volume pulmonaire. Il s'agit d'un déterminant majeur de la distension thoracique dynamique, particulièrement délétère, et facteur important de la dyspnée d'exercice. Nos travaux montrent sans ambiguïté une forte association entre la distension thoracique dynamique (limitant l'augmentation du volume courant) et la dyspnée à l'effort chez ces patients. Le corollaire de ces constatations est que des interventions thérapeutiques qui réduisent la distension thoracique devraient diminuer la dyspnée d'effort et améliorer la tolérance à l'exercice, et ce y compris dans des situations cliniques où les anomalies de la mécanique respiratoire ne sont a priori pas le primum movens de la maladie. Et en effet, la réduction de la dyspnée d'effort est bien corrélée avec la réduction du volume pulmonaire induite directement par des interventions pharmacologiques ou indirectement par des interventions non-pharmacologiques. De plus, du point de vue thérapeutique, la mise en évidence dans la troisième étude d'une propension à la distension thoracique induite par l'exercice chez certains patients atteints d'HTAP qui présentent une nette diminution des débits aériens à bas volume pulmonaire peut elle fournir une base théorique à l'adjonction de bronchodilatateurs aux traitements à visée hémodynamique. En conclusion, cette thèse contribue à une meilleure connaissance de la physiopathologie de la dyspnée d'exercice dans le contexte de la BPCO à un stade précoce, de l'ICC et de l'HTAP, en mettant en évidence le rôle d'un mécanisme pathogénétique qui n'avait pas été décrit auparavant.
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Kavila, William. "A dynamic analysis of the influence of monetary policy on the general price level in Zimbabwe under periods of hyperinflation and dollarisation." Thesis, Nelson Mandela Metropolitan University, 2015. http://hdl.handle.net/10948/3889.
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This thesis analyses the influence of monetary policy on the general price level in Zimbabwe during periods of hyperinflation and dollarisation. The first part of the analysis covers the period January 2006 to July 2008 when the country experienced high inflation and ultimately hyperinflation. The second part covers the period 2009 to 2012, when the country adopted the multi-currency system and became fully dollarised. In terms of motivation, the study firstly sought to empirically examine the factors that led to hyperinflation in Zimbabwe, paying particular attention to the influence of monetary policy. Secondly, the thesis sought to determine the major factors that influenced price formation in a dollarised Zimbabwean economy; a completely new macro-economic environment. A significant development in this new macro-economic environment was the loss of monetary policy autonomy of the central bank, which also contributed to the relevance of the study. This thesis makes two contributions. The first contribution is the finding that hyperinflation in Zimbabwe was caused by expansionary monetary policy as a result of the activities of an unrestrained and unaccountable central bank. The second contribution was the empirical finding that in the fully dollarised economy inflation is largely determined by external factors. This implies that the domestic economy has no control over domestic inflation developments and as such, Zimbabwean authorities should formulate appropriate economic policies to respond to the impact of external shocks on domestic price formation when the need arises. The role of monetary policy in Zimbabwe’s hyperinflation episode is assessed using the Autoregressive Distributed Lag (ARDL) and the Error Correction Model (ECM) approaches with monthly data from January 2006 to July 2008. The impact of monetary policy on hyperinflation is captured by the coefficient of broad money supply and the interest rate. Results indicate that hyperinflation was caused by expansionary monetary policy, the exchange rate premium and inflation expectations for both the short and long term. Zimbabwe’s hyperinflation episode which peaked during the period 2007 to 2008 brings to the fore the importance of ensuring that the central bank is independent in executing its mandate of influencing the monetary policy process in a manner that ensures price stability. The ARDL and ECM approaches are also used to explore the dynamics of inflation in the dollarised Zimbabwean economy, with monthly data from January 2009 to December 2012. The main drivers of inflation under the multi-currency system were found to be the United States of America dollar/South African rand exchange rate, international oil prices, inflation expectations and the South African inflation rate. The findings contrast with the hyperinflationary era, where empirical studies have cited excessive money supply growth as the major driver of inflation dynamics in Zimbabwe. The results also suggest a higher exchange rate pass-through to domestic prices, consistent with empirical literature which postulates that inflation in dollarised economies is largely explained by movements in the exchange rate of major trading partners and international prices. The policy implication from the analysis is the need for policy makers to aggressively promote policies that ensure increased productivity of the economy. An improvement in productivity would influence the relative prices of tradable and non-tradable goods and ultimately the general price level in the economy. The study also quantified the independence of the Reserve Bank of Zimbabwe (RBZ) using the Mathew (2006), “new index for institutional quality” and the results showed that the RBZ is not an independent central bank. The central bank is found to have a low index of central bank independence (CBI), against a high level of inflation. While this relationship does not imply causality it can be inferred that the lack of independence of the RBZ could have influenced inflation dynamics in Zimbabwe. Only a subordinated central bank can be compelled to engage in inflationary deficit financing and also fund quasi-fiscal activities. The provisions of the RBZ Act [Chapter 22:15] in their current form make the central bank an appendage of the Ministry of Finance and Economic Development and this has, to a large extent, resulted in conflict between the political goals of government and the central bank’s primary objective of achieving price stability. In the event that Zimbabwe reintroduces its own currency in future, the achievement of the primary goal of price stability by the central bank will only be realised if the apex bank is given more autonomy.
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Gazzana, Marcelo Basso. "Investigação da hiperinsuflação pulmonar dinâmica durante o exercício e sua relação com a força dos músculos inspiratórios em pacientes com hipertensão arterial pulmonar." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2015. http://hdl.handle.net/10183/119416.
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Introdução: A redução da capacidade inspiratória (CI) induzida pelo exercício observada em alguns pacientes com hipertensão arterial pulmonar (HAP) poderia potencialmente ser influenciada por disfunção muscular respiratória. Objetivos: Investigar se há alguma relação entre CI e força muscular respiratória antes e após o exercício máximo e estudar o papel da pressão muscular respiratória e da CI na dispneia e na capacidade de exercício em pacientes com HAP. Métodos: 27 pacientes com HAP e 12 controles saudáveis pareados foram comparados. Todos os participantes foram submetidos a teste de exercício cardiopulmonar (TECP) com determinação seriada da CI. As pressões inspiratória e expiratória máximas (PImáx e PEmáx, respectivamente) foram medidas antes, no pico e após o exercício. Resultados: Os pacientes tiveram menor volume expiratório forçado no primeiro segundo (VEF1), capacidade vital forçada (CVF) (com relação VEF1/CVF semelhante) e capacidade aeróbia máxima e maior dispneia no exercício. A PImáx e a PEmáx foram significativamente menores nos pacientes com HAP que nos controles. Entretanto, a variação pós exercício em relação ao repouso não foi significativamente diferente nos dois grupos. Os pacientes apresentaram redução significativa da CI do repouso ao pico do exercício em comparação aos controles. 17/27 pacientes (63%) apresentaram redução da CI durante o exercício. Considerando-se apenas os pacientes, não houve associação entre CI e PImáx ou PEmáx (pré, pós exercício ou mudança do repouso). Comparando-se os pacientes com e sem redução da CI, não houve diferença na proporção de pacientes que apresentaram redução da PImáx (41 vs 44%) ou da PEmáx (76 vs 89%) após o exercício. Da mesma forma, nenhuma diferença na PImáx ou PEmáx foi observada no exercício comparando estes subgrupos. Conclusões: Em resumo, a força muscular respiratória foi significativamente menor em pacientes com HAP em comparação com controles e uma proporção significativa de pacientes com HAP apresentaram redução da CI durante o exercício. No entanto, não foram observadas associações entre CI e alterações de força muscular respiratória com o exercício, sugerindo que ocorra verdadeira hiperinsuflação dinâmica. Além disso, o único parâmetro relacionado com a dispneia induzida pelo exercício foi a CI no repouso e com capacidade aeróbia no pico foi a magnitude da redução da PEmáx após o exercício.Rationale: The exercise induced inspiratory capacity (IC) reduction observed in some patients with pulmonary arterial hypertension (PAH) could potentially be influenced by respiratory muscle dysfunction. Aims: To investigate if there is any relationship between IC and respiratory muscle strength before and after maximal exercise and to study the contribution of respiratory muscle pressure and IC in exercise dyspnea and capacity in PAH patients. Methods: 27 patients with PAH and 12 healthy matched controls were compared. All participants underwent cardiopulmonary exercise test (CPET) with serial IC measurements. Inspiratory and expiratory maximal mouth pressure (PImax and PEmax, respectively) were measured before and at peak/post exercise. Results: Patients had lower forced expiratory volume in 1 s (FEV1), forced vital capacity (FVC) (with similar FEV1/FVC ratio) and peak aerobic capacity and higher exercise dyspnea. PImax and PEmax were significantly lower in PAH patients compared to controls. However, post exercise variations from rest were not significant different in either group. Patients presented significant rest-to-peak reduction in IC compared to controls. 17/27 patients (63%) exhibited IC reduction during exercise. Considering only patients, there was no association between IC and PImax or PEmax (pre, post exercise or change from rest). Comparing patients with and without IC reduction, there was no difference in the proportion of patients presenting inspiratory (41 vs 44%) or expiratory (76 vs 89%) pressure reduction after exercise, respectively. In the same way, no difference in both inspiratory and expiratory respiratory pressure change with exercise was observed comparing these subgroups. Conclusions: In summary, respiratory muscle strength was significantly lower in PAH patients compared to controls and a significant proportion of PAH presented IC reduction during exercise. Nonetheless, no associations between IC and respiratory muscle strength changes with exercise were observed, suggesting a true dynamic lung hyperinflation. Additionally, the only parameter associated with exercise induced dyspnea was resting IC and with peak aerobic capacity was the magnitude of PEmax reduction after exercise.
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Silva, Aline Grandi da. "Efeitos da perda de peso na hiperinsuflação pulmonar dinâmica em asmáticos obesos." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-27092018-122111/.
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Introdução: Adultos obesos com asma apresentam maior ocorrencia e intensidade de hiperinsuflação dinâmica (HD) e limitação do fluxo expiratório (LFE) em comparação aos asmáticos não obesos e a perda de peso parece melhorar a mecânica respiratória durante o exercício. Contudo, desconhece-se, até o momento, estudos que tenham avaliado o efeito da perda de peso na HD em asmáticos obesos. Objetivo: Avaliar o efeito de um programa de perda de peso na hiperinsuflação pulmonar dinâmica em asmáticos obesos. Métodos: Trata-se de um estudo secundário a um ensaio clinico randomizado no qual 42 pacientes com asma moderada ou grave foram previamente submetidos a um programa de perda de peso (dieta e psicologia associados ou não ao treinamento físico, 2vezes/semana, 60 minutos/sessão durante 3 meses). Posteriormente, foram divididos em 2 grupos de acordo com a % da perda de peso: (grupo >= 5%, n=19) e (grupo < 5%, n=23). Antes e após as intervenções, foram avaliados a HD e a LFE (exercício com carga constante) assim como os fatores de saúde relacionados a qualidade de vida (FSRQV), o controle da asma, a força e endurance muscular de quadríceps, a composição corporal e a função pulmonar. A comparação entre os dados categóricos foi realizada pelo teste qui-quadrado e entre os dados numéricos pela ANOVA de dois fatores com medidas repetidas. A associação entre a perda de peso e a melhora da HD foi analisada pelo teste de Correlação de Pearson. O nível de significância estatística foi ajustado para 5% (p <= 0,05). Resultados: O grupo >= 5% apresentou redução clinicamente significante da HD em relação ao grupo < 5% pós intervenção (-9,1 ± 14,5% vs. -12,5 ± 13,5%, respectivamente), que foi acompanhado por um retardo significante no tempo de inicio da HD e LFE. Além disso, o grupo >= 5% obteve melhora clinicamente significativa nos FSRQV e no controle da asma. Também foi observado uma correlação entre a redução da circunferência da cintura e o aumento da CI (r = -0,45, p = 0,05) no grupo >= 5%. Não foi encontrada diferença nos volumes pulmonares avaliados. Conclusão: A perda de peso moderada ( >= 5% do peso corporal), principalmente na presença da diminuição da circunferência da cintura, melhora a HD em adultos obesos com asma. Além disso, o grupo que perdeu mais peso também retardou o tempo de início da HD e da LFE durante a progressão do exercício, apresentando melhora nos FSRQV e controle clínico da asmaRationale: Obese adults with asthma develop dynamic hyperinflation (DH) and expiratory flow limitation (EFL) more likely than no obese asthmatics and weight loss seems to improve the breathing mechanic during exercise. However, studies to evaluate the effect of weight loss on DH in obese asthmatics are unknown. Objective: To evaluate the effect of a weight loss program on dynamic pulmonary hyperinflation in obese asthmatics. Methods: This was a secondary study of a randomized clinical trial in which 42 subjects with moderate or severe asthma previously participated in a weight loss program (diet and psychology associated or not with physical training, 2x/ week, 60 min/ session for 3 months). Posteriorly, they were divided into 2 groups according to %weight loss: (group >= 5%, n = 19) and (group < 5%, n = 23). Before and after the intervention, DH and EFL (constant load exercise), health-related quality of life (HRQoL), asthma control, quadriceps muscle strength and endurance, body composition and lung function were assessed. The comparison between the categorical data was performed using the chi-square test and between the numerical data by two-way ANOVA with repeated measures. The association between weight loss and DH improvement was analyzed by the Pearson\'s correlation test. The level of statistical significance was adjusted to 5% (p <= 0.05). Results: Group >= 5% presented a clinically significant reduction of DH compared to group < 5% post intervention (-9.1±14.5% vs. - 12.5±13.5%, respectively), that was following by a significant delay at the onset time for both DH and EFL. Besides, group >= 5% obtained clinically significant improvement in the HRQoL and asthma control. Furthermore, was observed a correlation between reduction waist circumference and increased IC (r=-0.45, p=0.05) in the group >= 5%. No difference was found in the lung volumes evaluated. Conclusion: A moderate weight loss ( >= 5% body weight) mainly with the decrease in waist circumference can improved DH in obese adults with asthma. In addition the greater weight loss group also delayed the onset time of DH and EFL during the progression of the exercise and presented an improvement in the asthma clinical control and in the HRQoL
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Pinto, Jana?na Maria Dantas. "Efeitos da respira??o em freno-labial sobre os volumes pulmonares e o padr?o de hiperinsufla??o din?mica em pacientes com asma." Universidade Federal do Rio Grande do Norte, 2011. http://repositorio.ufrn.br:8080/jspui/handle/123456789/16720.
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Previous issue date: 2011-08-29Objectives: To evaluate how to develop dynamic hyperinflation (DH) during exercise, the influence of pursed-lip breathing in (PLB) on breathing pattern and operating volume in patients with asthma. Methods: We studied 12 asthmatic patients in three moments: (1) anthropometry and spirometry, (2) submaximal incremental cycle ergometer test in spontaneous breathing and (3), submaximal incremental test on a cycle ergometer with PLB using the Opto-electronic plethysmography. Results: Evaluating the end-expiratory lung volume (EEV) during submaximal incremental test in spontaneous breathing, patients were divided into euvolume and hyperinflated. The RFL has increased significantly, the variation of the EEV group euvolume (1.4L) and decreased in group hyperinflated (0.272L). In group volume observed a significant increase of 140% in Vt at baseline, before exercise, comparing the RFL and spontaneous breathing. Hyperinflated group was observed that the RFL induced significant increases of Vt at all times of the test incremental baseline, 50%, 100% load and 66% recovery, 250%, 61.5% and 66% respectively. Respiratory rate decreased significantly with PLB at all times of the submaximal incremental test in the group euvolume. The speed of shortening of inspiratory muscles (VtRcp/Ti) in the hyperinflated increased from 1.6 ? 0.8L/s vs. 2.55 ? 0.9L/s, whereas in the RFL euvolume group ranged from 0.72 ? 0.31L/s vs. 0.65 ? 0.2L/s. The velocity of shortening of the expiratory muscles (VtAb/Te) showed similarity in response to RFL. In group hyperinflated varied vs. 0.89 ? 0.47 vs. 0.80 ? 0.36 and ? 1.17 ? 1L vs. 0.78 ? 0.6 for group euvolume. Conclusion: Different behavior in relation to EEV in patients with moderate asthma were observed, the HD and decreased EEV in response to exercise. The breathing pattern was modulated by both RFL performance as at home, making it more efficient
Objetivos: Avaliar como se desenvolve a hiperinsufla??o din?mica (HD) e as poss?veis modifica??es dos volumes pulonares operacionais durante o exerc?cio em respira??o espont?nea e respira??o em freno-labial (RFL) em pacientes asm?ticos. M?todos: Foram avaliados 12 pacientes asm?ticos em 3 momentos: (1) antropometria e espirometria, (2) teste incremental subm?ximo no cicloerg?metro em respira??o espont?nea e (3), teste incremental subm?ximo no cicloerg?metro com RFL utilizando a Pletismografia Opto-eletr?nica. Resultados: Avaliando o volume pulmonar expirat?rio final (EEV) durante o teste incremental subm?ximo em respira??o espont?nea, os pacientes foram divididos em grupo euvolume e grupo hiperinsuflado. A RFL aumentou, significativamente, a varia??o do EEV no grupo euvolume (1.4L) e diminuiu (0.272L) no grupo hiperinsuflado. No grupo euvolume observou-se aumento significativo de 140% no Vt na situa??o basal, pr?-exerc?cio, comparando a RFL e a respira??o espont?nea. No grupo hiperinsuflado foi observado que a RFL induziu aumentos significativos do Vt em todos os momentos do teste incremental, basal, 50%, 100% da carga e recupera??o em 66%, 250%, 61.5% e 66% respectivamente. A frequ?ncia respirat?ria diminuiu significativamente com a RFL em todos os momentos do teste incremental subm?ximo no grupo euvolume. A velocidade encurtamento dos m?sculos inspirat?rios (VtRcp/Ti) no grupo hiperinsuflado aumentou de 1.6 ? 0.8L/s vs. 2.55 ? 0.9L/s, com a RFL enquanto que no grupo euvolume variou de 0.72 ? 0.31L/s vs. 0.65 ? 0.2 L/s. A velocidade de encurtamento dos m?sculos expirat?rios (VtAb/Te) demonstrou similaridade em resposta a RFL. No grupo hiperinsuflado variou 0.89 ? 0.47 vs. 0.80 ? 0.36 e no grupo euvolume em 1.17 ? 1L vs. 0.78 ? 0.6. Conclus?o: Diferentes comportamentos em rela??o ao EEV nos pacientes com asma moderada foram observados, a HD e diminui??o do EEV em resposta ao exerc?cio. O padr?o respirat?rio foi modulado pela RFL tanto em exerc?cio como no repouso, tornando-o mais eficiente
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Monteiro, Mariane Borba. "Efeitos da pressão expiratória positiva na hiperinsuflação dinâmica em pacientes portadores de doença pulmonar obstrutiva crônica submetidos ao exercício." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/17760.
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A hiperinsuflação dinâmica (HD) é considerada um importante contribuinte para a sensação de dispnéia e interrupção do esforço físico em pacientes portadores de doença pulmonar obstrutiva crônica (DPOC). Diversas estratégias são testadas para tentar amenizar a HD e, frequentemente, utiliza-se a capacidade inspiratória (CI) para avaliar esse efeito. Os objetivos deste estudo foram verificar a presença de HD através da pletismografia logo após a suspensão do exercício e avaliar os efeitos da pressão expiratória positiva em via aérea (EPAP) na HD em pacientes portadores de DPOC submetidos ao exercício. Foram incluídos portadores de DPOC moderada a muito grave, de ambos os sexos, considerados capazes de realizarem o teste de esforço. Todos os participantes submeteram-se à mensuração de fluxos expiratórios, volumes e capacidades pulmonares, além da análise de difusão dos gases através da pletismografia. Essas medidas foram feitas antes e após o uso do broncodilatador. A seguir utilizou-se um protocolo de exercício submáximo e nova prova de função pulmonar era realizada imediatamente após o esforço físico para avaliar a presença de hiperinsuflação, ainda sob efeito do broncodilatador. Os pacientes que apresentaram sinal de HD na pletismografia foram convidados a retornar após 48 horas para repetir o mesmo protocolo de estudo, porém com uso de máscara de EPAP durante o exercício. Os parâmetros de função pulmonar foram analisados e comparados nos diferentes momentos e entre os protocolos. A amostra foi composta inicialmente por 46 pacientes, com média de idade de 65±8,5 anos, sendo 32 (70%) do sexo masculino, 25 (54%) com doença em estágio IV. Do total, 17(37%) apresentaram HD na pletismografia realizada após o teste de exercício. Após o exercício, observou-se diferença significativa entre pacientes com e sem HD apenas nas variáveis: CI (p<0,0001), CI/CPT (p=0,001), CRF/CPT (p=0,002). O uso da EPAP durante o exercício aplicado em 17 pacientes com HD não alterou de maneira significativa a capacidade pulmonar total (CPT; p=0,64), a capacidade residual funcional (CRF; p=0,09) e o volume residual (VR; p=0,10) quando comparado aos valores obtidos após exercício sem EPAP. Entretanto na comparação da CI observou-se uma menor perda de CI (p=0,02) com o uso da máscara. Verificou-se diferença significativa na comparação da relação CI/CPT antes e após o exercício em cada protocolo, ambos apresentando uma queda do valor com o exercício. Na comparação entre protocolos observou-se diferença significativa (p=0,01), representado uma queda menor da relação CI/CPT no protocolo com EPAP. Também se observaram relações VR/CPT e CRF/CPT significativamente menores (p=0,03) após o exercício com EPAP em relação ao exercício isolado. Conclui-se que 37% dos 46 pacientes apresentaram HD, detectada através da redução da CI e da sua relação com a CPT, quando avaliados imediatamente após o teste de exercício através da pletismografia. O uso da EPAP através de máscara facial reduziu a HD em teste de exercício submáximo, observado através da redução significativa da queda da CI e da relação CI/CPT, e pela menor alteração das relações VR/CPT e CRF/CPT.Dynamic hyperinflation (DH) contributes substantially to the sensation of dyspnea and the interruption of physical exercise in patients with chronic obstructive pulmonary disease (COPD). Several strategies have been tested to mitigate DH, and inspiratory capacity (IC) is often used to measure it. The purpose of this study was investigate the presence of DH immediately after exercise interruption using plethysmography and to evaluate the effects of expiratory positive airway pressure (EPAP) on DH of patients with COPD that underwent a exercise test. The study enrolled men and women with moderate to very severe COPD who were able to perform a exercise test. All participants underwent measurement of expiratory flows, volumes and lung capacities, and gas diffusion using plethysmography before and after the use of bronchodilators. A submaximal exercise test and repeated pulmonary function tests were conducted immediately after physical exercise to evaluate hyperinflation, still under the effect of the bronchodilator. The patients with DH according to plethysmography were invited to return 48 hours later to repeat the same protocol using an EPAP mask during exercise test. Pulmonary function parameters were analyzed and compared at the different time points and between the two tests. The sample consisted of 46 patients whose mean age was 65±8.5 years; 32 (70%) were men, and 25 (54%) had stage IV disease. Plethysmography performed after the exercise test revealed DH in 17 (37%) participants. After exercise, there was a significant difference between patients with and without DH only in IC (p<0.0001), IC/TLC (p=0.001), and FRC/TLC (p=0.002). The use of EPAP during exercise in 17 patients with DH did not significantly change total lung capacity (TLC; p=0.64), functional residual capacity (FRC; p=0.09), or residual volume (RV; p=0.10) when compared with the values obtained after exercise without EPAP. However, there was a lower loss of IC (p=0.02) in the EPAP mask group. There was a significant difference in IC/TLC before and after the exercise in each test, and both groups had a decrease in this value after exercise. The comparison between groups revealed a significant difference (p=0.01) and a smaller decrease in the IC/TLC ratio in the EPAP group. Moreover, significantly lower RV/TLC and FRC/TLC (p=0.03) were found after exercise with EPAP than after exercise alone. Of the 46 study patients, 37% developed DH, detected by a reduction in IC and in IC/TLC when evaluated immediately after exercise test using plethysmography. The use of EPAP delivered by face mask reduced DH in submaximal exercise tests, indicated by a significant reduction in IC and IC/TLC decreases and smaller changes in RV/TLC and FRC/TLC.
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Sabapathy, Surendran, and n/a. "Acute and Chronic Adaptations To Intermittent and Continuous Exercise in Chronic Obstructive Pulmonary Disease Patients." Griffith University. School of Physiotherapy and Exercise Science, 2006. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20070115.170236.
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The primary aim of this thesis was to develop a better understanding of the physiology and perceptual responses associated with the performance of continuous (CE) and intermittent exercise (IE) in patients with moderate chronic obstructive pulmonary disease (COPD). A secondary aim was to examine factors that could potentially limit exercise tolerance in COPD patients, particularly in relation to the dynamics of the cardiovascular system and muscle metabolism. The results of the four studies conducted to achieve these aims are presented in this thesis. In Study 1, the physiological, metabolic and perceptual responses to an acute bout of IE and CE were examined in 10 individuals with moderate COPD. Each subject completed an incremental exercise test to exhaustion on a cycle ergometer. Subjects then performed IE (1 min exercise: 1 min rest ratio) and CE tests at 70% of peak power in random order on separate days. Gas exchange, heart rate, plasma lactate concentration, ratings of breathlessness, inspiratory capacity and the total amount of work completed were measured during each exercise test. Subjects were able to complete a significantly greater amount of work during IE (71 ± 32 kJ) compared with CE (31 ± 24 kJ). Intermittent exercise was associated with significantly lower values for oxygen uptake, expired ventilation and plasma lactate concentration when compared with CE. Subjects also reported a significantly lower rating of breathlessness during IE compared to CE. The degree of dynamic lung hyperinflation (change in end-expiratory lung volume) was lower during IE (0.23 ± 0.07 L) than during CE (0.52 ± 0.13 L). The results suggest that IE may be superior to CE as a mode of training for patients with COPD. The greater amount of total work performed and the lower measured physiological responses attained with intermittent exercise could potentially allow greater training adaptations to be achieved in individuals with more limited lung function. The purpose of Study 2 was to compare the adaptations to 8 wk of supervised intermittent and continuous cycle ergometry training, performed at the same relative intensity and matched for total work completed, in patients with COPD. Nineteen subjects with moderate COPD were stratified according to age, gender, and pulmonary function, and then randomly assigned to either an IE (1 min exercise: 1 min rest ratio) or CE training group. Subjects trained 3 d per week for 8 wk and completed 30 min of exercise. Initial training intensity, i.e., the power output applied during the CE bouts and during the exercise interval of the IE bouts, was determined as 50% of the peak power output achieved during incremental exercise and was increased by 5% each week after 2 wk of training. The total amount of work performed was not significantly different (P=0.74) between the CE (750 ± 90 kJ) and IE (707 ± 92 kJ) groups. The subjects who performed IE (N=9) experienced significantly lower levels of perceived breathlessness and lower limb fatigue during the exercise-training bouts than the group who performed CE (N=10). However, exercise capacity (peak oxygen uptake) and exercise tolerance (peak power output and 6-min walk distance) improved to a similar extent in both training groups. During submaximal constant-load exercise, the improved (faster) phase II oxygen uptake kinetic response with training was independent of exercise mode. Furthermore, training-induced reductions in submaximal exercise heart rate, carbon dioxide output, expired ventilation and blood lactate concentrations were not different between the two training modes. Exercise training also resulted in an equivalent reduction for both training modes in the degree of dynamic hyperinflation observed during incremental exercise. Thus, when total work performed and relative intensity were the same for both training modes, 8 wk of CE or IE training resulted in similar functional improvements and physiological adaptations in patients with moderate COPD. Study 3 examined the relationship between exercise capacity (peak oxygen uptake) and lower limb vasodilatory capacity in 9 patients with moderate COPD and 9 healthy age-matched control subjects. While peak oxygen uptake was significantly lower in the COPD patients (15.8 ± 3.5 mL·min-1·kg-1) compared to the control subjects (25.2 ± 3.5 mL·kg-1·min-1), there were no significant differences between groups in peak calf blood flow or peak calf conductance measured 7 s post-ischemia. Peak oxygen uptake was significantly correlated with peak calf blood flow and peak conductance in the control group, whereas there was no significant relationship found between these variables in the COPD group. However, the rate of decay in blood flow following ischemia was significantly slower (p less than 0.05) for the COPD group (-0.036 ± 0.005 mL·100 mL-1·min-1·s-1) when compared to the control group (-0.048 ± 0.015 mL·100 mL-1·min-1·s-1). The results of this study suggest that the lower peak exercise capacity in patients with moderate COPD is not related to a loss in leg vasodilatory capacity. Study 4 examined the dynamics of oxygen uptake kinetics during high-intensity constant-load cycling performed at 70% of the peak power attained during an incremental exercise test in 7 patients with moderate COPD and 7 healthy age-matched controls. The time constant of the primary component (phase II) of oxygen uptake was significantly slower in the COPD patients (82 ± 8 s) when compared to healthy control subjects (44 ± 4 s). Moreover, the oxygen cost per unit increment in power output for the primary component and the overall response were significantly higher in patients with COPD than in healthy control subjects. A slow component was observed in 5 of the 7 patients with COPD (49 ± 11 mL·min-1), whereas all of the control subjects demonstrated a slow component of oxygen uptake (213 ± 35 mL·min-1). The slow component comprised a significantly greater proportion of the total oxygen uptake response in the healthy control group (18 ± 2%) than in the COPD group (10 ± 2%). In the COPD patients, the slow component amplitude was significantly correlated with the decrease in inspiratory capacity (r = -0.88, P less than 0.05; N=5), indicating that the magnitude of the slow component was larger in individuals who experienced a greater degree of dynamic hyperinflation. This study demonstrated that most patients with moderate COPD are able to exercise at intensities high enough to elicit a slow component of oxygen uptake during constant-load exercise. The significant correlation observed between the slow component amplitude and the degree of dynamic hyperinflation suggests that the work of breathing may contribute to the slow component in patients with COPD.
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Christev, Atanas Christev. "On the dynamics of hyperinflation and learning /." Search for this dissertation online, 2003. http://wwwlib.umi.com/cr/ksu/main.
Full textBooks on the topic "Dynamic hyperinflation"
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Fernández, Roque B. Real interest rate and the dynamics of hiperinflation [i.e. hyperinflation]: The case of Argentina. Buenos Aires: C.E.M.A., 1990.
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Kreit, John W. Dynamic Hyperinflation and Intrinsic Positive End-Expiratory Pressure. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0010.
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Dynamic hyperinflation and intrinsic PEEP almost always occur in patients with severe obstructive lung disease, in whom slowing of expiratory flow prevents complete exhalation. Occasionally, patients without airflow obstruction develop dynamic hyperinflation when expiratory time, is excessively shortened by a rapid respiratory rate, a long set inspiratory time (TI), or both. Dynamic Hyperinflation and Intrinsic Positive End-Expiratory Pressure describes the causes of dynamic hyperinflation and the mechanisms of its adverse effects, including reduced cardiac output and blood pressure, pulmonary barotrauma, and ineffective ventilator triggering. The chapter also describes how to screen for and measure intrinsic PEEP, and how to reduce or eliminate its adverse effects.
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Tuxen, David V. Pathophysiology and causes of airflow limitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0110.
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Exacerbations of asthma or chronic obstructive pulmonary disease (COPD) can be life-threatening emergencies, and require careful management to minimize the risks of morbidity and mortality. Prompt, full bronchodilator therapy, careful observation and appropriate mechanical ventilation technique is required. Dynamic hyperinflation of the lungs occurs in all patients, and must be careful assessed and regulated. Excessive dynamic hyperinflation can result in respiratory tamponade, hypotension, circulatory failure, pneumothoraces and, in severe cases, cardiac arrest. Intravenous or continuous nebulized salbutamol commonly causes lactic acidosis that should be detected and managed. Prolonged paralysis during difficult mechanical ventilation can result in severe necrotizing myopathy. Pneumothoraces in ventilated patients with asthma are usually under tension, redistribute ventilation to the contralateral lung, and risk a second tension pneumothorax. Patients surviving mechanical ventilation for asthma and COPD have an increased risk of recurrence and death. All these problems require awareness, avoidance or detection and management
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Sampson, Brett G., and Andrew D. Bersten. Therapeutic approach to bronchospasm and asthma. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0111.
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The optimal management of bronchospasm and acute asthma is reliant upon confirmation of the diagnosis of asthma, detection of life-threatening complications, recognition of β2 agonist toxicity, and exclusion of important asthma mimics (such as vocal cord dysfunction and left ventricular failure). β2 agonists, anticholinergics, and corticosteroids are the mainstay of treatment. β2 agonists should be preferentially administered by metered dose inhaler via a spacer, and corticosteroids by the oral route, reserving nebulized (and intravenous) salbutamol, as well as intravenous hydrocortisone, for situations when these routes are not possible. A single intravenous dose of magnesium may be of benefit in severe asthma, but repeat dosing is likely to cause serious side effects. Parenteral administration of adrenaline may prevent the need for intubation in the patient in extremis. Aminophylline has an unfavourable side effect profile and has not been shown to offer additional benefit in adults. However, it does have a role in paediatric asthma. Unproven medical therapies with potential benefit include ketamine, heliox, inhalational anaesthetics, and leukotriene antagonists. The need for ventilatory support is usually preceded by worsening dynamic hyperinflation, exhaustion, hypoxia, reduced conscious state, or a combination of these. While non-invasive ventilation may have a temporizing role to allow time for response to medical therapy, there is insufficient evidence for its use, and should not delay invasive ventilation. If invasive ventilation is indicated, a strategy of hypoventilation and permissive hypercapnoea, minimizes barotrauma and dynamic hyperinflation. Extracorporeal support may have a role as a rescue therapy.
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Kreit, John W., and John A. Kellum. Mechanical Ventilation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.001.0001.
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Mechanical Ventilation—Physiology and Practice provides a comprehensive review of the physiological principles underlying mechanical ventilation, as well as practical approaches to the management of patients with respiratory failure. The book explains instrumentation and terminology, ventilator modes and breath types, ventilator alarms, how to write ventilator orders, and how to diagnose and correct patient–ventilator asynchrony. It also discusses the physiological assessment of the mechanically ventilated patient and the diagnosis and management of dynamic hyperinflation, and describes how to manage patients with the acute respiratory distress syndrome (ARDS), severe obstructive lung disease, and right ventricular failure; how to “wean” patients from the ventilator; and how and when to use noninvasive ventilation.
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Kreit, John W. Severe Obstructive Lung Disease. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0013.
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Although chronic obstructive lung disease, asthma, bronchiectasis, and bronchiolitis have very different causes, clinical features, and therapies, they share the same underlying pathophysiology. They are referred to as obstructive lung diseases because airway narrowing causes increased resistance and slowing of expiratory gas flow. Mechanical ventilation of patients with severe obstructive lung disease often produces two problems that must be recognized and effectively managed: over-ventilation and dynamic hyperinflation. Severe Obstructive Lung Disease reviews these two major adverse consequences of mechanical ventilation in patients with severe air flow obstruction. The chapter explains how to detect and correct both of these problems and provides guidelines for managing patients with respiratory failure caused by severe obstructive lung disease.
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Lucangelo, Umberto, and Massimo Ferluga. Pulmonary mechanical dysfunction in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0084.
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In intensive care units practitioners are confronted every day with mechanically-ventilated patients and should be able to sort out from all the data available from modern ventilators to tailored patient ventilatory strategy. Real-time visualization of pressure, flow and tidal volume provide valuable information on the respiratory system, to optimize ventilatory support and avoiding complications associated with mechanical ventilation. Early determination of patient–ventilator asynchrony, air-trapping, and variation in respiratory parameters is important during mechanical ventilation. A correct evaluation of data becomes mandatory to avoid a prolonged need for ventilatory support. During dynamic hyperinflation the lungs do not have time to reach the functional residual capacity at the end of expiration, increasing the work of breathing and promoting patient-ventilator asynchrony. Expiratory capnogram provides qualitative information on the waveform patterns associated with mechanical ventilation and quantitative estimation of expired CO2. The concept of dead space accounts for those lung areas that are ventilated but not perfused. Calculations derived from volumetric capnography are useful indicators of pulmonary embolism. Moreover, alveolar dead space is increased in acute lung injury and its value decreased in case of positive end-expiratory pressure (PEEP)-induced recruitment, whereas PEEP-induced overdistension tends to increment alveolar dead space.
Book chapters on the topic "Dynamic hyperinflation"
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Rossi, A., G. Polese, and G. Brandi. "Dynamic Hyperinflation." In Ventilatory Failure, 199–218. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84554-3_12.
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Laveneziana, Pierantonio, Katherine A. Webb, and Denis E. O’Donnell. "Static and Dynamic Hyperinflation in Chronic Obstructive Pulmonary Disease." In Mechanics of Breathing, 73–97. Milano: Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5647-3_7.
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Milic-Emili, J., S. B. Gottfried, and A. Rossi. "Dynamic Hyperinflation: Intrinsic Peep and its Ramifications in Patients with Respiratory Failure." In Anesthesia and the Lung, 261–68. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0899-4_28.
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Milic-Emili, J., S. B. Gottfried, and A. Rossi. "Dynamic Hyperinflation: Intrinsic PEEP and Its Ramifications in Patients with Respiratory Failure." In Update in Intensive Care and Emergency Medicine, 192–98. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-83042-6_22.
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Raghavendra, Srinivas, and Petri T. Piiroinen. "Expectation Dynamics and Hyperinflation." In An Introduction to Economic Dynamics, 91–108. London: Routledge, 2023. http://dx.doi.org/10.4324/9780429324406-7.
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Ort, Václav, and Lukáš Konupka. "The Effects of Expiratory Flow Limitation and Different Inspiratory and Expiratory Airway Resistances on Dynamic Hyperinflation of the Lungs: A Bench Study." In IFMBE Proceedings, 823–27. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-9035-6_152.
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"Dynamic Hyperinflation." In Encyclopedia of Trauma Care, 505. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_100521.
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Gladwin, Mark T. "Dynamic hyperinflation and intrinsic positive end-expiratory pressure." In Mechanical Ventilation, 83–94. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199773947.003.0007.
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"Thoracics." In Emergencies in Anaesthesia, edited by Keith G. Allman, Andrew K. McIndoe, and Iain H. Wilson, 209–34. Oxford University Press, 2009. http://dx.doi.org/10.1093/med/9780199560820.003.0007.
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Intrathoracic tracheal/bronchial obstruction 210 Inhaled foreign body 212 Tracheal injury or laceration 214 Bronchopleural fistula (BPF) 216 Hypoxia during one-lung ventilation (OLV) 218 Sudden high airway pressure during one-lung ventilation 220 Pneumothorax of dependent lung during one-lung ventilation 222 Dynamic hyperinflation (DHI) 224 Cardiac herniation postpneumonectomy ...
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Bennett, James, and Gerard Gould. "Thoracics." In Emergencies in Anaesthesia, 215–46. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198758143.003.0008.
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In this chapter on life-threatening thoracic problems in anaesthesia, the authors offer their guidance and expertise on the best-practice methods of dealing with each of these. These problems include tracheal/bronchial obstruction, inhaled foreign body, tracheal injury or laceration, bronchopleural fistula, hypoxia/pneumothorax/high airway pressure during one-lung ventilation, dynamic hyperinflation, cardiac herniation postpneumonectomy, major airway bleeding, and bleeding during mediastinoscopy. In addition, the definitions, presentation, management strategies, investigations, risk factors, exclusions and causes, and any special considerations for each potential thoracically related situation are given and elaborated upon. Lists of up-to-date online resources and further reading are also provided here, offering invaluable know-how to encourage the reader to broaden their knowledge.
Conference papers on the topic "Dynamic hyperinflation"
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Cheyne, William Spencer, Jinelle Gelinas, Laura Harp, and Neil D. Eves. "Hemodynamic effects of incremental dynamic hyperinflation." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.oa1963.
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Feron-Lahaije, Anke J., Laura Willems, P. N. R. Dekhuijzen, Hanneke A. van Helvoort, and Yvonne F. Heijdra. "MPH And CPET Induced Dynamic Hyperinflation." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4590.
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van der Meer, A. N. C., K. de Jong, E. H. Bel, and A. ten Brinke. "Dynamic Hyperinflation: A Treatable Trait in Asthma." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6112.
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van der Meer, Akke-Nynke A. N., Aranka Hoekstra-Kuik, Anneke ten Brinke, and E. H. D. Bel. "Dynamic hyperinflation: an important target for treatment in asthma." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa3985.
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Peeters, Anneleen, Audrey Herpeux, Inge Muylle, Maria Gabrovska, and Vincent Ninane. "Dynamic hyperinflation during maximal voluntary ventilation in healthy subjects." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa2014.
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Urban, Matthias, Anna Katharina Mayr, Ingrid Schmidt, Otto Chris Burghuber, and Georg-Christian Funk. "Expiratory stenosis breathing – a novel model of dynamic hyperinflation." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2466.
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van der Meer, A. N., K. De Jong, E. H. Bel, and A. Ten Brinke. "Dynamic hyperinflation predicts impaired daily life activity in asthma." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4001.
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Hernandez Gonzalez, Cristina, Marta Iscar Urrutia, Marta García Clemente, Liliana Perez Martinez, Angelica Orellana Gonzalez, Ana Isabel Enríquez Rodríguez, Gemma Rubinos Cuadrado, María José Vázquez López, Ramón Fernández Álvarez, and Pere Casan Clara. "Dynamic hyperinflation causes exercise limitation in patients with cystic fibrosis." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4866.
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Pini, Laura, Alberto Valsecchi, Enrico Boni, Michele Guerini, and Claudio Tantucci. "Acute Dynamic Hyperinflation And Systemic Inflammation In Stable COPD Patients." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2907.
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Dorey-Stein, Z., M. Patel, A. J. Gangemi, M. Zantah, E. Dominguez Castillo, A. Pandya, J. Thomas, et al. "Static and Dynamic Hyperinflation in Patients Undergoing Evaluation for Bronchoscopic Lung Volume Reduction with Severe Hyperinflation and Advanced Emphysema." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6862.
Full textReports on the topic "Dynamic hyperinflation"
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Lopez, Jose, and Kris James Mitchener. Uncertainty and Hyperinflation: European Inflation Dynamics after World War I. Cambridge, MA: National Bureau of Economic Research, May 2018. http://dx.doi.org/10.3386/w24624.
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