Relevant bibliographies by topics / Dual-factor

Academic literature on the topic 'Dual-factor'

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Published: 10 December 2022
Last updated: 29 January 2023

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Journal articles on the topic "Dual-factor"

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Lê, Sébastien, and Jérôme Pagès. "DMFA: Dual Multiple Factor Analysis." Communications in Statistics - Theory and Methods 39, no. 3 (January 13, 2010): 483–92. http://dx.doi.org/10.1080/03610920903140114.

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Thachil, Jecko. "Dual origins and dual roles for von Willebrand factor." Journal of Thrombosis and Haemostasis 19, no. 1 (January 6, 2021): 308–9. http://dx.doi.org/10.1111/jth.15152.

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Thachil, Jecko. "Dual origins and dual roles for von Willebrand factor." Journal of Thrombosis and Haemostasis 19, no. 1 (January 2021): 308–9. http://dx.doi.org/10.1111/jth.15152.

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REN, Zhong, Ying LIU, Guodong LIU, and Zhen HUANG. "Improved wavelet denoising with dual-threshold and dual-factor function." Journal of Computer Applications 33, no. 9 (November 6, 2013): 2595–98. http://dx.doi.org/10.3724/sp.j.1087.2013.02595.

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Yoo, Myeong Han, and Seong Eun Park. "The Influence of Dual-Factor Concepts of Street Performance Audience on Place Attachment." Journal of Arts and Cultural Management 11, no. 2 (December 31, 2018): 99–37. http://dx.doi.org/10.15333/acm.2018.12.30.99.

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Zhang, Ruqing, Jiafeng Guo, Yixing Fan, Yanyan Lan, and Xueqi Cheng. "Dual-factor Generation Model for Conversation." ACM Transactions on Information Systems 38, no. 3 (June 26, 2020): 1–31. http://dx.doi.org/10.1145/3394052.

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Farahat, Asmaa, and Khalid Hussein. "Dual-Band BSF with Enhanced Quality Factor." Applied Computational Electromagnetics Society 36, no. 1 (February 27, 2021): 89–98. http://dx.doi.org/10.47037/2020.aces.j.360113.

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In the present work, a U-shaped CPW resonator (CPWR) with, generally, unequal arms is proposed to produce high Q-factor bandstop filter (BSF) based on broadside-coupling between the CPWR and a CPW through-line (CPWTL), which are printed on opposite faces of a thin substrate. The unequal arms of the U-shape and the finite width of the ground strips of the CPWR are shown to produce much higher Q-factor than that of equal arms and infinitely extending side ground planes. The dimensions of the CPWTL are optimized for impedance matching while the dimensions of the CPWR are optimized to obtain the highest Q-factor. The effect of the loss tangent of the dielectric substrate material on the Q-factor is investigated. It is shown that the difference between the lengths of the unequal arms of the U-shaped resonator can be used to control the Q-factor. Thanks to the computational efficiency of the employed electromagnetic simulator, enough number of trials has been successfully performed in reasonable time to arrive at the final design of the BSF. A prototype of the proposed BSF is fabricated for experimental investigation of its performance. The experimental measurements show good agreement when compared with the corresponding simulation results.
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Pejhan, Shervin, Stan VanUum, Neil Duggal, Brian Rotenberg, Michael Mayich, Robert Hammond, and Qi Zhang. "Somatotroph Adenoma with Dual Transcription Factor Expression." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 48, s2 (July 2021): S10. http://dx.doi.org/10.1017/cjn.2021.170.

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A 20-year-old male presented with evidence of gigantism/acromegaly. Endocrinological investigations identified elevated growth hormone levels and a failed glucose tolerance test. Imaging revealed a macroadenoma expanding the sella with encroachment on the optic chiasm and cavernous sinuses. Trans-sphenoidal resection was undertaken and a gross total removal was achieved. Histopathological features were typical of a densely granulated somatotroph adenoma with abundant growth hormone expression, scattered prolactin expression and sparse examples of fibrous bodies. Unexpectedly, the adenoma not only expressed PIT-1 but also SF-1 transcription factors. This finding suggests that the adenoma may have been pluripotent. The prognostic significance of this finding is uncertain although the patient is stable from an endocrinological and imaging perspective approximately one-year post-op. A pituitary adenoma of this nature has not been previously reported. The recent literature on atypical transcription factor expression patterns and revisions to the classification of pituitary adenomas will be reviewed.Learning ObjectivesAppreciate the rarity of dual transcription factor expression in pituitary adenomasRationalize the use of transcription factor characterization in the revised WHO classification of pituitary adenomas
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Lyons, Michael D., E. Scott Huebner, Kimberly J. Hills, and Svetlana V. Shinkareva. "The Dual-Factor Model of Mental Health." Canadian Journal of School Psychology 27, no. 2 (May 18, 2012): 183–96. http://dx.doi.org/10.1177/0829573512443669.

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More, R. S., and D. D. Ahire. "Power Factor Improvement using Dual Boost Converter." International Journal of Engineering Trends and Technology 42, no. 3 (December 25, 2016): 153–58. http://dx.doi.org/10.14445/22315381/ijett-v42p230.

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Dissertations / Theses on the topic "Dual-factor"

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Bourke, Patrick Anthony. "A general factor in dual task performance decrement." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359419.

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Slobodchikoff, Tatyana G. "The Slavic Dual: Morphosyntactic Feature Economy as a Factor in Language Change." Diss., The University of Arizona, 2013. http://hdl.handle.net/10150/297021.

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Dual number marked on personal pronouns and verbal agreement suffixes disappeared in the majority of Slavic languages except for three - Slovenian, Upper Sorbian, and Lower Sorbian. Previous studies do not provide a principled account why (i) the monomorphemic dual was reanalyzed as bimorphemic in Slovenian, Upper, and Lower Sorbian, and why (ii) it was replaced by the plural in the majority of Slavic languages including Russian and Kashubian. In this dissertation, I investigate diachronic changes in the morphosyntactic category of the Slavic dual which occurred in the 11th-15th centuries. In this dissertation, I present new diachronic data obtained through digital corpora of Old Slavic manuscripts. Within the framework of Distributed Morphology, I propose a new principle of Morphosyntactic Feature Economy which explains these two different patterns of diachronic change in the Slavic dual pronouns and verbal agreement. I argue that the principle of Morphosyntactic Feature Economy is one of the factors which drives diachronic change in the category of number in Slavic languages. Morphosyntactic Feature Economy is a principle of efficient computation. It plays an essential role in restructuring of the morphosyntactic category of the Slavic dual. Morphosyntactic restructuring of the Slavic dual, triggered by its morphosyntactic and semantic markedness, results in a morphosyntactically 'simpler' category of number. As a consequence of the application of the principle of Morphosyntactic Feature Economy, the language learner acquires a featurally restructuctured category of number, which is simpler and more computationally efficient.
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McKay, Brock L. (Brock Lindsay). "Dual Factor Socially Desirable Responding and Contrasts in Intrinsic and Extrinsic Religious Motivation." Thesis, University of North Texas, 1994. https://digital.library.unt.edu/ark:/67531/metadc278762/.

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A follow-up was done to Leak and Fish's (1989) study of intrinsically and extrinsically religious individuals using Paulhus' (1984) Balanced Inventory of Desirable Responding, a two factor scale of socially desirable responding measuring self-deceptive enhancement (SDE) and impression management (IM). 275 introduction to psychology students were group tested and categorized by gender and by religious orientation with Allport and Ross's (1967) fourfold Religious Orientation Scale (ROS). Differences between the four types were hypothesized on the religious relevance of the SDE and IM scale items. A difference score was also computed by contrasting two instructional sets on the BIDR as a measure of variation across situations. Measures of private and public self-consciousness, social anxiety, fear of negative evaluation, and self esteem were included.
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Oest, Megan Elizabeth. "Dual Osteogenic and Angiogenic Growth Factor Delivery as a Treatment for Segmental Bone Defects." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16264.

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A new model of a critically-sized segmental femoral bone defect in rats was developed to enable in vivo imaging and facilitate post-mortem mechanical testing of samples. The critically-sized nature of the model was assessed and confirmed. The efficacy of sustained co-delivery of osteogenic (BMP-2 and TGF- Ò3) and angiogenic (VEGF) growth factors in promoting functional bone repair was assessed. Effects of scaffold modification in terms of geometry and composition were evaluated. The results indicated that co-delivery of BMP-2 and TGF- Ò3 resulted in a dose-dependent improvement in functional bone repair. Modification of the polylactide scaffold to include an absorbable ceramic component and a cored out geometry enhanced rate of union. Addition of VEGF to the scaffold treatment did not significantly impact revascularization of the defect site or functional repair of the bone defect. These data demonstrate that the complex environment of an acute bone defect requires different treatment strategies than simple ectopic models would suggest. A positive predictive correlation between bone repair parameters measured in vivo and mechanical functionality was established. The novel defect model demonstrated robustness and reproducibility. Implications for further research are discussed.
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Hu, Jing. "Targeting Transcription Factor NF-kappa B by Dual Functional Oligodeoxynucleotide Complex for Inhibition of Neuroinflammation." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439301618.

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Sarnoff, Tamar Jill. "METAPHOR, COGNITIVE ELABORATION AND PERSUASION." Diss., The University of Arizona, 2009. http://hdl.handle.net/10150/194626.

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Metaphors have long been a subject of interest to philosophers, scholars and researchers. Recent insights into the nature and function of metaphor have spurred new interest in the persuasive effects of metaphor. To date, research on the relation between metaphors and attitudes has produced mixed findings. This paper argues that there are several limitations in previous models and designs and this work attempted to resolve several of them. The rationale for the study is based on the Elaboration Likelihood Model (ELM) of persuasion, which argues that cognitive elaboration is a strong predictor of attitudes. Researchers have posited that metaphors should evoke more cognitive elaboration than literal counterparts. This paper reports the results of a study that tested the relationship between metaphors, cognitive elaboration, and attitudes. Participants were exposed to one of 72 message conditions and responded to a set of psychological and attitude scales. Many of the hypotheses were not supported, including tests of the amount of cognitive effort that subjects reported and results related to attitude change by metaphor type. Results indicated that attitudes were stable across time, which is consistent with the ELM.
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Ogino, Shuichi. "Efficacy of the dual controlled release of HGF and bFGF impregnated with a collagen/gelatin scaffold." Kyoto University, 2018. http://hdl.handle.net/2433/231010.

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Thalji, Amanda Lynn. "A Dual-Factor Model of Mental Health in High School Students: Group Characteristics and Social Functioning." Scholar Commons, 2012. http://scholarcommons.usf.edu/etd/4239.

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A dual-factor model of psychological functioning examines the presence of wellness (i.e., subjective well-being; SWB) and psychopathology (i.e., internalizing and externalizing behavior problems) in explaining youth mental health functioning. Using a dual-factor model, previous research has yielded four unique groups of elementary and middle school youth as well as college-age adults with distinct levels of wellness and psychopathology. The present empirical investigation included valid data from 500 adolescents from two high schools (grades 9 to 11). This exploratory study produced four groups of students with unique mental health profiles aligned with previous studies investigating the dual-factor model. Tukey-Kramer comparisons determined that among groups classified as having elevated symptoms of psychopathology, those that also report high levels of SWB (i.e., symptomatic but content youth) are more likely to be rated as having externalizing problems, and those with low levels of SWB (i.e., troubled youth) are more likely to report symptoms of internalizing problems. Evaluation of group differences on positive mental health indicators suggest that differences between groups with elevated SWB versus low SWB were due to differences in life satisfaction and negative affect. Tukey-Kramer comparisons indicated that youth with complete mental health reported optimal functioning in terms SWB. Youth identified as having low levels of SWB, appeared to report similarly low levels of life satisfaction and positive affect, but those that also reported elevated levels of psychopathology, particularly internalizing problems, had greater levels of negative affect. Additional findings from this study demonstrate the utility of classifying high school students' mental health according to a dual-factor model. Results of a MANCOVA suggest a significant effect for mental health group membership as yielded from a dual-factor model on students' social-functioning. Follow up ANCOVAs and Tukey-Kramer comparisons suggest that high SWB in tandem with low levels of psychopathology (i.e., complete mental health) is associated with a host of optimal functioning in terms of teacher-rated social skills, perceptions of interpersonal relationships, receipt of social support, reduced likelihood of victimization, and high quality romantic experiences. For youth with psychopathology, average to high levels of SWB (i.e., as in symptomatic but content students), may buffer them from experiencing poor social functioning, particularly in terms of perceived social support, peer victimization, general interpersonal relations, and satisfaction with romantic experiences. Overall results from this study support the presence of dual-factor model in high school students and the importance of assessment of positive and negative indicators in order to effectively gain a comprehensive understanding of adolescents' social functioning.
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Ebisine, Kimimuepigha. "Dual regulation of transcription factor Nrf2 by Keap1 and the beta-TrCP/GSK-3 in cancer." Thesis, University of Dundee, 2019. https://discovery.dundee.ac.uk/en/studentTheses/20338051-4e92-417e-aeb1-4cd3dc86eda4.

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Cancer is one of the foremost causes of death worldwide with about 14.1 million new incidences and 8.2 cancer related deaths occurring globally. NF-E2 p45-related factor 2 (Nrf2), a cap-'n'-collar basic leucine zipper (CNC-bZIP) transcription factor, prevents carcinogenesis through expression of genes that ensure the excretion, enzymatic modification, and repair of oxidative damage in cells containing the antioxidant response element (ARE) in their promoter region. Beyond providing cytoprotection against oxidative stress and xenobiotics, Nrf2 pays a role in maintaining basic physiological processes such as energy metabolism and cell cycle regulation. Whilst Nrf2 plays a pivotal role in preventing degenerative and inflammatory disease, upregulation of Nrf2 promotes tumourigenesis in cancerous cells. Therefore, understanding the mechanisms controlling Nrf2 activity is important in translational medicine. Nrf2 is regulated by proteasomal degradation by Kelch-like ECH-associated protein 1 (Keap1) an E3 ubiquitin ligase substrate adaptor protein that recruits of cullin-3 (Cul3) to Nrf2 via its Neh2 domain. Nrf2 is also negatively regulated by phosphorylation by glycogen synthase kinase-3 (GSK-3) causing β-transducin repeat-containing protein (β-TrCP) to ubiquitinate Nrf2 by Skp1-Cul1-F-box (SCF) ubiquitin ligase through the Neh6 domain of Nrf2. Several research groups have shown that induction of ARE-driven genes can be regulated by phosphoinositide 3- kinase- protein kinase B (PI3K-Akt/PKB) signalling pathway. The ability of tert-butylhydroquinone (tBHQ), 1-[2-cyano-3,12-dioxooleana-1,9(11)-diene-28-oyl]imidazole (CDDO-Im), diethyl maleate (DEM), curcumin, carnosol, ferulic acid and sulforaphane (SFN) to activate Nrf2-target genes in a Keap1-dependent or Keap1-independent manner was tested. It was discovered that all compounds, except for SFN, activate Nrf2-target genes in a Keap1-independent manner, inhibiting GSK-3 and functioning through the Neh6 domain of Nrf2. Analysis of the involvement of PI3K-Akt/PKB pathway in Nrf2 activation revealed that regulation of Nrf2 through the PI3K-Akt/PKB pathway is independent of Keap1 but dependent on GSK-3. Also, it was shown that tBHQ, DEM, CDDO-Im, curcumin, ferulic acid directly decreased phosphatase and tensin homolog (PTEN) activity, thereby preventing formation of the phosphodegron in the Neh6 domain of Nrf2. With increased Nrf2 levels reported in various cancers including lung cancer, leading to the progression of these cancers, Nrf2 can be seen as a double-edged sword. Loss-of-function somatic mutations in KEAP1 as well as somatic mutation in NFE2L2 has been reported in several human cancers playing a role in the development of such cancer. Using short hairpin RNA (shRNA) and the CRISPR/Cas9 system to generate stable Nrf2 knockdown A549 and H460 cells, the second part of this thesis investigated biochemical and physiological changes that occur, when the Nrf2 is genetically downregulated, and further on to determine what mechanism(s) is responsible for decreased cell proliferation in tumours. The findings obtained confirm that downregulation of Nrf2 from the human non-small lung adenocarcinoma epithelial cell line A549 and H460, in which Nrf2 is upregulated though somatic mutations in KEAP1, results in decreased cell proliferation. Analysis of the genes involved in NADPH generation and pentose phosphate pathway (PPP) show that decrease in Nrf2 caused a decrease in the expression of genes involved in PPP. Although knockdown of Nrf2 resulted in a decrease in cell proliferation, it was shown that this decrease was not as a result of cell death. Nrf2 is able to control cell proliferation by induction of metabolic reprogramming geared towards favoring anabolic pathways and influencing the PPP as well as provide energy source required for cell proliferation.
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Yehya, Alaa. "New insights into Brain-derived Neurotrophic Factor Dual Signaling : imbalance implications in mechanisms of neuroprotection and neurotoxicity." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONTS058.

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Le « Brain-Derived Neurotrophic Factor » (BDNF) est la neurotrophine la plus abondante et la plus répandue dans le cerveau humain. De nombreuses études se sont intéressées à son rôle dans la survie neuronale, la croissance et la plasticité synaptique. La signalisation BDNF est dépendante de deux récepteurs, le récepteur tyrosine kinase (TrkB) et le récepteur neurotrophine p75 (p75NTR). Il est bien établi que le rôle trophique du BDNF est assuré via son récepteur de haute-affinité TrkB, alors que la forme précurseur proBDNF active p75NTR vers la voie d'apoptose. Cette double signalisation est physiologiquement contrôlée par un équilibre entre les différentes voies. Les résultats obtenus à partir des études cliniques et des modèles animaux suggèrent un rôle de la signalisation BDNF dans les tauopathies, caractérisées par l'existence de dépôts intracérébraux de protéine tau, une caractéristique commune à certaines maladies neurodégénératives, notamment la maladie d'Alzheimer (MA). Cependant, aucune investigation n'a été menée jusqu'à présent sur les modifications que pouvaient induire les tauopathies dans la signalisation BDNF et si une dérégulation de l'expression du BDNF pouvait affecter ses propres récepteurs TrkB et p75NTR.Dans ce travail de thèse, nous avons utilisé une lignée de poisson-zèbre transgénique portant la mutation humaine TAUP301L retrouvée notamment dans le démence fronto-temporale. Nous avons mesuré l'expression de BDNF et de ses deux récepteurs au niveau transcriptionnel et protéique. Nous n'avons observé aucune modification des taux d'expression de BDNF et de TrkB, en revanche, nous avons noté une augmentation significative de p75NTR. A l'aide de la même lignée transgénique, nous avons induit une baisse d'expression de BDNF via la micro-injection de morpholinos. De manière remarquable, la baisse d'expression de BDNF affecte de façon différentielle TrkB et p75NTR. En effet, nous avons observé une diminution de l'expression de TrkB et parallèlement une augmentation de p75NTR. De plus, la baisse d'expression de BDNF aggrave la neurotoxicité associée au développement de la tauopathie ce qui se traduit par une augmentation de la mort neuronale et de l'hyperphosphorylation de tau, cette dernière étant concommittante à une activation de la Glycogen Synthétase Kinase 3 beta (GSK3beta).Une diminution de l'effet neuroprotecteur de BDNF à travers un déséquilibre de ces récepteurs de signalisation a été également montré en étudiant le rôle de BDNF au cours du développement de la ligne latérale postérieure (PLL). Ce système est considéré comme un modèle d'étude particulièrement pertinent pour évaluer différents processus biologiques comme la migration cellulaire collective ou la régénération cellulaire. Nous avons détecté l'expression de BDNF dans plusieurs structures de la PLL. La diminution d'expression de BDNF conduit à un défaut de migration du primordium de la PLL, associé à une augmentation de la mort cellulaire. De plus, nous avons observé une réduction de la prolifération cellulaire et un défaut de repousse axonale du nerf, ce qui conduit à des anomalies de régénération à la fois du nerf de la PLL et des cellules ciliées. Nos résultats suggèrent que le BDNF joue un rôle essentiel au cours du développement de la PLL et démontrent la pertinence du système de la ligne latérale en tant que modèle d'étude des fonctions de BDNF.En conclusion, notre étude représente la première analyse du rôle in vivo de BDNF et de ses 2 récepteurs de signalisation. Nous avons ainsi montré les répercussions d'une dérégulation des voies de signalisation du BDNF. Un équilibre entre ces deux voies est essentiel pour le développement et la survie cellulaire, ce qui fait de BDNF non seulement une cible thérapeutique potentielle, mais également une neurotrophine clé pouvant activer plusieurs circuits de signalisation, potentialisant ainsi son rôle protecteur
Brain-derived neurotrophic factor (BDNF) is the most abundant secreted and widely distributed neurotrophin in human brain. It has been extensively studied for its role in neuronal survival, growth and synaptic plasticity. BDNF signaling mediated through tryosine receptor kinase B (TrkB) and p75NTR neurotrophin receptor (p75NTR). It is well established that BDNF beneficial actions are mediated by it is high-affinity TrkB, whereas pro-BDNF activates p75NTR towards apoptosis. This diverse dual signaling is normally under a tight balance regulation. Based on clinical and animal studies, it has been suggested that BDNF signaling is involved in tauopathy, which is a pathological hallmark in several neurodegenerative diseases, including Alzheimer's disease (AD). However, what changes tauopathy may induce on BDNF signaling, and whether BDNF deregulation could affect its two signaling receptors (TrkB, p75NTR), and eventually tauopathy pathogenesis, have not been investigated. In this study we used a transgenic zebrafish line for human Tau-P301L tauopathy, and measured transcriptional and protein levels of BDNF and of its two signaling receptors. We found no modification of BDNF and TrkB expression levels, but a significant up-regulation of p75NTR. We then used the same transgenic line to generate BDNF knockdown using morpholino microinjection technique. Interestingly, BDNF knockdown differentially affects TrkB and p75NTR; we observed a reduction of TrkB expression and an increase in p75NTR expression. In addition, BDNF knockdown aggravates tauopathy-associated toxicity; we found an increase in neuronal cell death and tau hyperphosphorylation, the latter was accompanied by an activation of tau glycogen synthase kinase 3beta (GSK3beta). Attenuation of BDNF neuroprotective effects through imbalance of its signaling receptors was further highlighted through studying BDNF role in the development of zebrafish posterior lateral line system (PLL). This system has recently emerged as a powerful tool to study several dynamic biological processes, including collective cell migration and nerve/hair cells regeneration. We detected BDNF expression in different PLL components. BDNF knockdown led to an impairment of the PLL primordium migration due to concomitant increase in cell death rate. In addition, reduced cell proliferation and defect in axonal re-growth were observed , which led to major defects of PLL nerve/hair cells regeneration, respectively. These findings suggest that BDNF has an essential role in PLL development, but more important they introduce PLL as research model to study BDNF functions. This is the first study to provide a detailed in vivo analysis of BDNF and its two signaling receptors. Our findings highlight several implications of BDNF signaling deregulation. Balanced signaling clearly has essential roles in survival and development, in addition to being a therapeutic target, BDNF can itself activate diverse molecular pathways, thus setting up a potential circuitry that could enhance its protective role
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Books on the topic "Dual-factor"

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Shipley, David D. Industrial salesforce motivation and Herzberg's dual factor theory. Stoke-on-Trent: North Staffordshire Polytechnic, 1986.

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Otruba, Kathy. LAN9303M/LAN9303Mi Small Form Factor Three Port 10/100 Managed Ethernet Switch with Dual MII/RMII/Turbo MII - Data Sheet. Microchip Technology Incorporated, 2016.

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Hirschtritt, Matthew E., Marc N. Potenza, and Linda C. Mayes. Impulsivity and Co-occurring Psychiatric Disorders. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0033.

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Dual diagnosis, the co-occurrence of substance abuse and another psychiatric disorder, is common. There is evidence to suggest that impulsivity may serve as a common substrate for these comorbidities, despite behavioral and biochemical differences between disorders. This chapter describes common neurobiological and behavioral findings between individuals with high impulsivity and those with dual diagnosis. Specifically, we focus on the co-occurrence of substance abuse with schizophrenia, eating disorders, attention-deficit/hyperactivity disorder, antisocial and borderline personality disorders, and bipolar disorder. For each type of dual diagnosis, we review literature that provides empirical evidence for the presence of impulsivity and treatment recommendations. In this context, we propose a “bottom-up” conceptualization, in which clusters of co-occurring phenotypes are used to formulate diagnostic and clinical plans; such an approach may produce more homogeneous diagnostic groups than exist in the current system. Further, a bottom-up approach may reveal that dual-diagnosis disorders represent distinct groups that share a common factor of impulsivity.
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Publications, Brotherhood Of Iron. Fast and the Furious - the Worlds Only Dual Factor System Which Builds Muscle and Shreds Fat at the Same Time! Created in the Lab - Proven in the Gym! Lulu Press, Inc., 2013.

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Chong, Ji Y., and Michael P. Lerario. Can I Go Home Now? Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0027.

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Transient ischemic attack (TIA) is a risk factor for subsequent stroke. A TIA diagnosis should incorporate findings on brain MRI and noninvasive angiography, if available. Some patients with TIA are at high risk of early recurrence. Rapid evaluation and treatment reduce that risk. Some new data suggest that short-term dual antiplatelet therapy may reduce stroke recurrence in patients with TIA or minor stroke.
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Lazarus, Philip J., Shannon Suldo, and Beth Doll, eds. Fostering the Emotional Well-Being of our Youth. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780190918873.001.0001.

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Fostering the Emotional Well-Being of Our Youth: A School-Based Approach is an edited work that details best practices in comprehensive school mental health services based upon a dual-factor model of mental health that considers both psychological wellness and mental illness. In the introduction, the editors respond to the question: Are our students all right? Then, each of the text’s 24 chapters (five sections) describes empirically sound and practical ways that professionals can foster supportive school climates and implement evidence-based universal interventions to promote well-being and prevent and reduce mental health problems in young people. Topics include conceptualizing and framing youth mental health through a dual-factor model; building culturally responsive schools; implementing positive behavior interventions and supports; inculcating social-emotional learning within schools impacted by trauma; creating a multidisciplinary approach to foster a positive school culture and promote students’ mental health; preventing school violence and advancing school safety; cultivating student engagement and connectedness; creating resilient classrooms and schools; strengthening preschool, childcare and parenting practices; building family–school partnerships; promoting physical activity, nutrition, and sleep; teaching emotional self-regulation; promoting students’ positive emotions, character, and purpose; building a foundation for trauma-informed schools; preventing bullying; supporting highly mobile students; enfranchising socially marginalized students; preventing school failure and school dropout; providing evidence-based supports in the aftermath of a crisis; raising the emotional well-being of students with anxiety and depression; implementing state-wide practices that promote student wellness and resilience; screening for academic, behavioral, and emotional health; and accessing targeted and intensive mental health services.
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Kenworthy, Lane. America’s Great Decoupling. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198807032.003.0012.

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This chapter sets out the significant extent to which the fruits of economic growth since the late 1970s have gone to the very top of the income distribution in the case of the United States. There has been little growth in real wages and incomes for most households, with dual earning being the main source of any such increase. The chapter identifies the growth in incomes at the very top as the main factor accounting for stagnation across the rest of the distribution, and discussed the range of channels through which this relationship operates, more and less directly. It also discusses broader debates about the extent to which real incomes are likely to reflect changes in living standards, and concludes that, while not telling the whole story, they remain central.
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Janssen, Ted, Gervais Chapuis, and Marc de Boissieu. Structure. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824442.003.0004.

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This chapter discusses the X-ray and neutron diffraction methods used to study the atomic structures of aperiodic crystals, addressing indexing diffraction patterns, superspace, ab initio methods, the structure factor of incommensurate structures; and diffuse scattering. The structure solution methods based on the dual space refinements are described, as they are very often applied for the resolution of aperiodic crystal structures. Modulation functions which are used for the refinement of modulated structures and composite structures are presented and illustrated with examples of structure models covering a large spectrum of structures from organic to inorganic compounds, including metals, alloys, and minerals. For a better understanding of the concept of quasicrystalline structures, one-dimensional structure examples are presented first. Further examples of quasicrystals, including decagonal quasicrystals and icosahedral quasicrystals, are analysed in terms of increasing shells of a selected number of polyhedra. The notion of the approximant is compared with classical forms of structures.
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Book chapters on the topic "Dual-factor"

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Coppi, R., and P. D’Urso. "The Dual Dynamic Factor Analysis Models." In Studies in Classification, Data Analysis, and Knowledge Organization, 47–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-55991-4_5.

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Sun, Ke, Shikui Tu, David Yang Gao, and Lei Xu. "Canonical Dual Approach to Binary Factor Analysis." In Independent Component Analysis and Signal Separation, 346–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-00599-2_44.

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Zhang, Wei, A. Thomas Look, Toshiya Inaba, and Stephen D. Nimer. "IL3BP1, A Transcription Factor with Dual Function." In Molecular Biology of Hematopoiesis 5, 397–402. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0391-6_48.

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Frey, Bruno S., and Margit Osterloh. "Motivation — A Dual-Edged Factor of Production." In Successful Management by Motivation, 3–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-662-10132-2_1.

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Gao, Hang, and Yi Liu. "FedDFA: Dual-Factor Aggregation for Federated Driver Distraction Detection." In Big Data and Social Computing, 237–50. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7532-5_15.

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Redei, Eva, and Christopher J. Evans. "Dual Control of Corticotropin Secretion: Isolation of Corticotropin-Inhibiting Factor." In Neuropeptides and Stress, 61–72. New York, NY: Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3514-9_6.

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Ho, Purdy, and John Armington. "A Dual-Factor Authentication System Featuring Speaker Verification and Token Technology." In Lecture Notes in Computer Science, 128–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-44887-x_16.

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Furlong, Michael J., Erin Dowdy, Stephanie Moore, and Eui Kyung Kim. "Adapting the Dual-Factor Model for Universal School-Based Mental Health Screening." In Handbook of Positive Psychology in Schools, 56–88. 3rd ed. New York: Routledge, 2022. http://dx.doi.org/10.4324/9781003013778-6.

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Kumar, Aashish, Preeti Chandrakar, and Rifaqat Ali. "An Improved Two Factor Authentication Scheme for TMIS Based on Dual Biometrics." In Cloud Security, 161–74. First edition. | Boca Raton : CRC Press, 2021.: CRC Press, 2021. http://dx.doi.org/10.1201/9780367821555-12.

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Wassem, R., F. O. Pedrosa, M. Buck, M. G. Yates, L. U. Rigo, and E. M. Souza. "Dual Role for the Integration Host Factor at an Enhancer-Dependent nifA Promoter." In Nitrogen Fixation: From Molecules to Crop Productivity, 135. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/0-306-47615-0_68.

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Conference papers on the topic "Dual-factor"

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Yang, Zigang, Hardik Gandhi, Lei Ding, and Lars Jorgensen. "Crest factor reduction for dual-band systems." In 2013 IEEE/MTT-S International Microwave Symposium - MTT 2013. IEEE, 2013. http://dx.doi.org/10.1109/mwsym.2013.6697564.

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Abdul, Arif Mohammad, Sudarson Jena, and M. Balraju. "Dual factor authentication to procure cloud services." In 2016 Fourth International Conference on Parallel, Distributed and Grid Computing (PDGC). IEEE, 2016. http://dx.doi.org/10.1109/pdgc.2016.7913252.

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Maji, Saurav, Utsav Banerjee, Samuel H. Fuller, Rabia Tugce Yazicigil, and Anantha P. Chandrakasan. "Securing Embedded Medical Devices using Dual-Factor Authentication." In 2021 IEEE 34th International Symposium on Computer-Based Medical Systems (CBMS). IEEE, 2021. http://dx.doi.org/10.1109/cbms52027.2021.00040.

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Tseng, Wen-Hung, and Kai-Ming Feng. "A quality factor enhanced dual-loop optoelectronic oscillator." In 2016 IEEE International Frequency Control Symposium (IFCS). IEEE, 2016. http://dx.doi.org/10.1109/fcs.2016.7546806.

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Pipiska, M., M. Frivaldsky, and P. Hudak. "Dual interleaved power factor correction for welding inverter." In 2018 ELEKTRO. IEEE, 2018. http://dx.doi.org/10.1109/elektro.2018.8398262.

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Attaianese, C., V. Nardi, F. Parillo, and G. Tomasso. "Dual Boost High performances Power Factor Correction (PFC)." In 2008 IEEE Applied Power Electronics Conference and Exposition - APEC 2008. IEEE, 2008. http://dx.doi.org/10.1109/apec.2008.4522848.

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Li, Lei, Quan Liu, and Xuemei Jiang. "USB Key-Based Dual-Factor Dynamic Authentication Scheme." In 2010 International Conference on Computational Intelligence and Security (CIS). IEEE, 2010. http://dx.doi.org/10.1109/cis.2010.103.

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Darly, S. S., P. Vanaja Ranjan, K. V. Bindu, and B. Justus Rabi. "A novel dual boost rectifier for power factor improvement." In 2011 1st International Conference on Electrical Energy Systems (ICEES). IEEE, 2011. http://dx.doi.org/10.1109/icees.2011.5725314.

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Asadian, Mohammad H., Sina Askari, Ian B. Flader, Yunhan Chen, Dustin D. Gerrard, Dongsuk D. Shin, Hyun-Keun Kwon, Thomas W. Kenny, and Andrei M. Shkel. "High Quality Factor Mode Ordered Dual Foucault Pendulum Gyroscope." In 2018 IEEE Sensors. IEEE, 2018. http://dx.doi.org/10.1109/icsens.2018.8589896.

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Mahmood, Mohammed, Huang-Jen Chiu, Yu-Kang Lo, Quang Trong Nha, Phu Hieu Pham, and Irwan Purnama. "Isolated dual boost bridgeless power factor correction AC-DC converter." In 2013 1st International Future Energy Electronics Conference (IFEEC). IEEE, 2013. http://dx.doi.org/10.1109/ifeec.2013.6687551.

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Reports on the topic "Dual-factor"

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Gafni, Yedidya, Moshe Lapidot, and Vitaly Citovsky. Dual role of the TYLCV protein V2 in suppressing the host plant defense. United States Department of Agriculture, January 2013. http://dx.doi.org/10.32747/2013.7597935.bard.

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TYLCV-Is is a major tomato pathogen, causing extensive crop losses in Israel and the U.S. We have identified a TYLCV-Is protein, V2, which acts as a suppressor of RNA silencing. Intriguingly, the counter-defense function of V2 may not be limited to silencing suppression. Our recent data suggest that V2 interacts with the tomato CYP1 protease. CYP1 belongs to the family of papain-like cysteine proteases which participate in programmed cell death (PCD) involved in plant defense against pathogens. Based on these data we proposed a model for dual action of V2 in suppressing the host antiviral defense: V2 targets SGS3 for degradation and V2 inhibits CYP1 activity. To study this we proposed to tackle three specific objectives. I. Characterize the role of V2 in SGS3 proteasomal degradation ubiquitination, II. Study the effects of V2 on CYP1 maturation, enzymatic activity, and accumulation and, III. Analyze the effects of the CYP1-V2 interaction on TYLCV-Is infection. Here we describe results from our study that support our hypothesis: the involvement of the host's innate immune system—in this case, PCD—in plant defense against TYLCV-Is. Also, we use TYLCV-Is to discover the molecular pathway(s) by which this plant virus counters this defense. Towards the end of our study we discovered an interesting involvement of the C2 protein encoded by TYLCV-Is in inducing Hypersensitive Response in N. benthamianaplants which is not the case when the whole viral genome is introduced. This might lead to a better understanding of the multiple processes involved in the way TYLCV is overcoming the defense mechanisms of the host plant cell. In a parallel research supporting the main goal described, we also investigated Agrobacteriumtumefaciens-encoded F-box protein VirF. It has been proposed that VirF targets a host protein for the UPS-mediated degradation, very much the way TYLCV V2 does. In our study, we identified one such interactor, an Arabidopsistrihelix-domain transcription factor VFP3, and further show that its very close homolog VFP5 also interacted with VirF. Interestingly, interactions of VirF with either VFP3 or VFP5 did not activate the host UPS, suggesting that VirF might play other UPS-independent roles in bacterial infection. Another target for VirF is VFP4, a transcription factor that both VirF and its plant functional homolog VBF target to degradation by UPS. Using RNA-seqtranscriptome analysis we showed that VFP4 regulates numerous plant genes involved in disease response, including responses to viral and bacterial infections. Detailed analyses of some of these genes indicated their involvement in plant protection against Agrobacterium infection. Thus, Agrobacterium may facilitate its infection by utilizing the host cell UPS to destabilize transcriptional regulators of the host disease response machinery that limits the infection.
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Hicks, Jacqueline. Export of Digital Surveillance Technologies From China to Developing Countries. Institute of Development Studies, August 2022. http://dx.doi.org/10.19088/k4d.2022.123.

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There is evidence to show that Chinese companies, with some state credit backing, are selling digital surveillance technologies to developing countries, which are then sometimes used in authoritarian practices. However, there is little direct evidence to show that surveillance technologies sold by Chinese companies have more authoritarian potential than the technologies sold by non-Chinese companies. Some researchers define “surveillance technologies” as including any form of digital infrastructure. There is data to show that developing country governments are contracting Chinese companies to build digital infrastructures. Other researchers define “surveillance technologies” as smart city projects. It is estimated that in 2019, Chinese smart city technologies have been purchased in over 100 countries worldwide. Other researchers look at more specific elements of smart cities: There are estimates that the “AI surveillance” components of smart cities have been purchased in 47-65 countries worldwide, and the “data integration” security platforms in at least 80 countries. None of these figures imply anything about how these technologies are used. The “dual use” nature of these technologies means that they can have both legitimate civilian and public safety uses as well as authoritarian control uses. There is evidence of some governments in Africa using Chinese surveillance technologies to spy on political opponents and arrest protesters. Some authors say that some Chinese smart city projects are actually not very effective, but still provide governments with a “security aesthetic”. Research also shows that Chinese smart city technologies have been sold mostly to illiberal regimes. However, in the wider context, there is also ample evidence of non-Chinese surveillance technologies contributing to authoritarian control in developing countries. There is also evidence that UK companies sell surveillance technologies to mostly illiberal regimes. Some reports consulted for this rapid review imply that Chinese surveillance technologies are more likely to be used for authoritarian control than those sold by non-Chinese companies. This analysis is largely based on circumstantial rather than direct evidence. They rely on prior judgements, which are themselves subject to ongoing enquiry in the literature: Almost all of the reports consulted for this rapid review say that the most important factor determining whether governments in developing countries will deploy a particular technology for repressive purposes is the quality of governance in the country. No reports were found in the literature reviewed of Chinese state pressure on developing countries to adopt surveillance technologies, and there were some anecdotal reports of officials in developing countries saying they did not come under any pressure to buy from Chinese companies.
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