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Journal articles on the topic 'Drug therapy'

To see the other types of publications on this topic, follow the link: Drug therapy.

Author: Grafiati

Published: 4 June 2021

Last updated: 29 July 2024

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1

Santos, Zélia Maria de Sousa Araújo, Helder de Pádua Lima, Flávia Braga de Oliveira, Jamilly Silva Vieira, Natasha Marques Frota, and Jennara Candido do Nascimento. "User's adherence to hypertensive drug therapy." Rev Rene 14, no. 1 (2013): 11–22. http://dx.doi.org/10.15253/2175-6783.2013000100003.

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The low treatment adherence is one of the major factors for the lack of blood pressure control and risk for cardiovascular diseases. We aimed to analyze the hypertensive user's adherence to drug therapy. A descriptive study carried out with 400 hypertensive users, from May to August 2010, through interviews. Most interviewees were female (67.2%), aged over 60 (54.3%) and with brown skin (57.4%). 326 (81.5%) users were making regular use of medicine with a predominance of those with over 10 years of diagnosis (33.5%), those with up to 5 years of treatment (31.0%), and those who received the complete medication provided by SUS (39.2%). The knowledge on the types of antihypertensive drugs and the daily frequency of medication prevailed regardless the regularity of medication, the same happened regarding the lack of knowledge on side effects, to the discomforts caused and the complexity of drug therapy. We concluded that the treatment adherence prevailed in most users.

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2

Borba, Anna Karla de Oliveira Tito, Ana Paula de Oliveira Marques, Márcia Carrera Campos Leal, Roberta de Souza Pereira da Silva Ramos, Ana Clara Carvalho Gonçalves Guerra, and Thaysa Melo Caldas. "Adherence to drug therapy in diabetic elderly." Rev Rene 14, no. 2 (2013): 394–404. http://dx.doi.org/10.15253/2175-6783.20130002000018.

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Sectional and observational study, which investigates access to medicine, socioeconomic and demographic profiles and their association with drug adherence to drug therapy in diabetic elderly assisted in a gerontogeriatric public service. Data were collected from 126 participants through structured interviews between February and September 2011. The results reveal that 47.6% of the elderly received their drug from the public health system. The elderly women prevailed, in more than 9 years of studies, retired and income of 1-2 minimum wages. As for drug adherence, 93.7% reported using their medication regularly, but only 52.4% were considered compliant under the Test of Batalha. There was no association between socioeconomic and demographic variables and self-reported adherence. It is necessary to invest in educational initiatives targeted to the elderly patients to promote adherence to therapy.

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3

MRPhamiS, Ian Tritschler. "Drug therapy." Nursing Standard 8, no. 8 (November 10, 1993): 52. http://dx.doi.org/10.7748/ns.8.8.52.s65.

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4

&NA;. "DRUG THERAPY." American Journal of Nursing 97, no. 4 (April 1997): 10. http://dx.doi.org/10.1097/00000446-199704000-00006.

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5

Iseman, M. D. "DRUG THERAPY." Pediatric Infectious Disease Journal 13, no. 5 (May 1994): 426. http://dx.doi.org/10.1097/00006454-199405000-00026.

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6

Rochon, P. A., and J. H. Gurwitz. "Drug therapy." Lancet 346, no. 8966 (July 1995): 32–36. http://dx.doi.org/10.1016/s0140-6736(95)92656-9.

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7

Tank, D. "Drug therapy." International Journal of Gynecology & Obstetrics 70 (2000): D14—D15. http://dx.doi.org/10.1016/s0020-7292(00)82534-5.

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8

Buskila, Dan. "Drug therapy." Best Practice & Research Clinical Rheumatology 13, no. 3 (September 1999): 479–85. http://dx.doi.org/10.1053/berh.1999.0038.

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9

OʼDONNELL, JAMES. "DRUG THERAPY." Nursing 24, no. 3 (March 1994): 46–48. http://dx.doi.org/10.1097/00152193-199403000-00020.

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10

Davis, George A., and Mary H. H. Chandler. "DRUG THERAPY AND DRUG INTERACTIONS." Oral and Maxillofacial Surgery Clinics of North America 8, no. 2 (May 1996): 245–63. http://dx.doi.org/10.1016/s1042-3699(20)30897-9.

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11

Borchardt, J. K. "Beginnings of drug therapy: Drug therapy in ancient India." Drug News & Perspectives 16, no. 6 (2003): 403. http://dx.doi.org/10.1358/dnp.2003.16.6.829312.

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12

Aronson, J. K., and M. Hardman. "ABC of monitoring drug therapy. Why monitor drug therapy?" BMJ 305, no. 6859 (October 17, 1992): 947–48. http://dx.doi.org/10.1136/bmj.305.6859.947.

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13

Focsa, Alin, Alexandru Sava, Andra Ababei, and Maria Apotrosoaei. "Drug interactions in gastrointestinal disorders therapy." Romanian Journal of Pharmaceutical Practice 14, S (July 30, 2021): 25–29. http://dx.doi.org/10.37897/rjphp.2021.s.5.

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Drug-drug interactions are a major cause of adverse reactions in polypharmacy, the incidence being directly proportional to the number of associated drugs. In this work, we highlighted the main drug-drug interactions that occurred in the treatment of gastrointestinal symptoms with proton pump inhibitors and histamine H2-receptor antagonists. The two groups of agents, used in gastroesophageal reflux and ulcer disease, can be released and without a prescription on the recommendation of the pharmacist, contributing to the risk of emergence of drug-drug interactions. Drug-drug interactions can occur due to the reduction of the gastric acidity, altering the biotransformation or excretion reducing of the drug co-administrated. pH increased may affect the absorption of some drugs decreasing (e.g. ketoconazole, itraconazole, atazanavir, vitamin B12, magnesium) or increasing the absorption of others (e.g. triazolam, midazolam). Furthermore, an attention to the interaction with narrow therapeutic index drugs (e.g. warfarin, phenytoin, theophylline) is required because of the risk of the toxic or side effects by the accumulation of these drugs.

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14

Kubanov, A. A., A. E. Karamova, A. A. Vorontsova, and P. A. Kalinina. "Drug therapy of leprosy." Vestnik dermatologii i venerologii 92, no. 4 (August 24, 2016): 12–19. http://dx.doi.org/10.25208/0042-4609-2016-92-4-12-19.

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Leprosy (Hansen’s disease) is a chronic granulomatous bacterial infection mainly affecting the skin and peripheral nervous system yet also involving other organs and systems as a result of a pathological process. The causative agent of leprosy - Mycobacterium leprae - is an obligate intracellular microorganism. Despite the removal of a threat of a leprosy epidemic, European countries still record outbreaks of the disease mainly among migrants coming from endemic areas. A golden standard of the treatment of leprosy is a WHO-recommended combined drug therapy comprising drugs such as dapsone, clofazimine and rifampicin. The article provides current data on the mechanisms of action, efficacy and safety of these drugs and their combined scheme of treatment obtained as a result of clinical trials. Moreover, it also reviews new regimens of the drug therapy of leprosy including those with the use of drugs from the group of fluoroquinols as well as immunotherapy of the disease.

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15

Becker, W. J. "Evidence Based Migraine Prophylactic Drug Therapy." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 26, no. 3 (November 1999): 27–32. http://dx.doi.org/10.1017/s0317167100000160.

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Prophylactic drug therapy is a major component of overall migraine management. However, because we do not know how currently used prophylactic drugs exert their beneficial effects in migraine, their use is based primarily on clinical trials. In general, prophylactic drugs are indicated when patients have three or more attacks a month and symptomatic medication use alone is not satisfactory. The choice of drug must be individualized, and is influenced by contraindications, potential side effects, the need to treat associated symptoms like tension-type headache and insomnia, and drug cost. Whether an individual patient will respond to a given drug cannot be predicted, but there are varying degrees of scientific evidence supporting the use of each prophylactic drug in migraine. This evidence is best for metoprolol, divalproex, amitriptyline, atenolol, flunarizine and naproxen. Based on placebo-controlled crossover studies, it would appear that at least some prophylactic drugs exert the greater part of their prophylactic effects very quickly, and that these also disappear very quickly once the drug is stopped. This may not apply to all prophylactic drugs and more research is needed. More well designed clinical trials are needed to guide our use of migraine prophylactic drugs. Although clinical experience is useful, placebo responses and variations in the migraine tendency over time can make interpretation of this experience difficult. Major advances will likely only occur once the pathogenesis of migraine and the mode of action of the prophylactic drugs is better understood.

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16

Wtorek, Karol, Angelika Długosz, and Anna Janecka. "Drug resistance in topoisomerase-targeting therapy." Postępy Higieny i Medycyny Doświadczalnej 72 (December 21, 2018): 1073–83. http://dx.doi.org/10.5604/01.3001.0012.8131.

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Drug resistance is a well-known phenomenon that occurs when initially responsive to chemotherapy cancer cells become tolerant and elude further effectiveness of anticancer drugs. Based on their mechanism of action, anticancer drugs can be divided into cytotoxic-based agents and target-based agents. An important role among the therapeutics of the second group is played by drugs targeting topoisomerases, nuclear enzymes critical to DNA function and cell survival. These enzymes are cellular targets of several groups of anticancer agents which generate DNA damage in rapidly proliferating cancer cells. Drugs targeting topoisomerase I are mostly analogs of camtothecin, a natural compound isolated from the bark of a tree growing in China. Drugs targeting topoisomerase II are divided into poisons, such as anthracycline antibiotics, whose action is based on intercalation between DNA bases, and catalytic inhibitors that block topoisomerase II at different stages of the catalytic cycle. Unfortunately, chemotherapy is often limited by the induction of drug resistance. Identifying mechanisms that promote drug resistance is critical for the improvement of patient prognosis. Cancer drug resistance is a complex phenomenon that may be influenced by many factors. Here we discuss various mechanisms by which cancer cells can develop resistance to topoisomerase-directed drugs, which include enhanced drug efflux, mutations in topoisomerase genes, hypophosphorylation of topoisomerase II catalytic domain, activation of NF-κB transcription factor and drug inactivation. All these events may lead to the ineffective induction of cancer cell death. Attempts at circumventing drug resistance through the inhibition of cellular efflux pumps, use of silencing RNAs or inhibition of some important mechanisms, which can allow cancer cells to survive therapy, are also presented.

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17

Buckel, P. "Recombinant Protein Drugs.( Milestones in Drug Therapy)." Molecules 6, no. 12 (December 31, 2001): 1063. http://dx.doi.org/10.3390/61201063.

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18

JACOBS, Paul H., and Lexie NALL. "Antifungal drug therapy." Revista do Instituto de Medicina Tropical de São Paulo 33, no. 3 (June 1991): 174. http://dx.doi.org/10.1590/s0036-46651991000300015.

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19

Rinaldi, Michael G., Paul H. Jacobs, and Lexie Hall. "Antifungal Drug Therapy." Mycologia 84, no. 4 (July 1992): 604. http://dx.doi.org/10.2307/3760336.

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20

Teramoto, Tamio, Jun Sasaki, Hirotsugu Ueshima, Genshi Egusa, Makoto Kinoshita, Kazuaki Shimamoto, Hiroyuki Daida, et al. "Treatment - Drug Therapy." Journal of Atherosclerosis and Thrombosis 15, no. 4 (2008): 167–78. http://dx.doi.org/10.5551/jat.e619.

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21

Paice, Judith A., and Janet M. Magolan. "Intraspinal Drug Therapy." Nursing Clinics of North America 26, no. 2 (June 1991): 477–98. http://dx.doi.org/10.1016/s0029-6465(22)00259-6.

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22

Bennett, Jeffrey D. "EMERGENCY DRUG THERAPY." Dental Clinics of North America 39, no. 3 (July 1995): 501–21. http://dx.doi.org/10.1016/s0011-8532(22)00927-2.

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23

Bennett, Jeffrey D. "EMERGENCY DRUG THERAPY." Dental Clinics of North America 39, no. 3 (July 1995): 501–21. http://dx.doi.org/10.1016/s0011-8532(22)00927-2.

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24

Durkin, Walter James. "CANCER DRUG THERAPY." Primary Care: Clinics in Office Practice 19, no. 4 (December 1992): 759–78. http://dx.doi.org/10.1016/s0095-4543(21)00615-1.

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25

Modarressi, Taher. "Antiobesity drug therapy." Cleveland Clinic Journal of Medicine 88, no. 12 (December 2021): 657.1–657. http://dx.doi.org/10.3949/ccjm.88c.12001.

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26

Ro, Young Moo. "Antihypertensive Drug Therapy." Journal of the Korean Medical Association 46, no. 8 (2003): 753. http://dx.doi.org/10.5124/jkma.2003.46.8.753.

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27

Casey, Petra M. "Gynecological Drug Therapy." Mayo Clinic Proceedings 82, no. 8 (August 2007): 1021. http://dx.doi.org/10.4065/82.8.1018-b.

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28

Hartono, C., T. Muthukumar, and M. Suthanthiran. "Immunosuppressive Drug Therapy." Cold Spring Harbor Perspectives in Medicine 3, no. 9 (September 1, 2013): a015487. http://dx.doi.org/10.1101/cshperspect.a015487.

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29

Cowan, Campbell. "Antianginal drug therapy." Current Opinion in CARDIOLOGY 5, no. 4 (August 1990): 453–60. http://dx.doi.org/10.1097/00001573-199008000-00014.

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30

Zipes, D. P. "Targeted drug therapy." Circulation 81, no. 3 (March 1990): 1139–41. http://dx.doi.org/10.1161/01.cir.81.3.1139.

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31

McCune, Joseph W., and Garry E. Bayliss. "Immunosuppressive drug therapy." Current Opinion in Rheumatology 1, no. 1 (June 1989): 80–88. http://dx.doi.org/10.1097/00002281-198901010-00016.

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32

McCune, Joseph W., and Garry E. Bayliss. "Immunosuppressive drug therapy." Current Opinion in Rheumatology 2, no. 3 (June 1990): 497–505. http://dx.doi.org/10.1097/00002281-199002030-00014.

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33

Oak, Makarand K. "Gynaecological Drug Therapy." Journal of Obstetrics and Gynaecology 27, no. 8 (January 2007): 874. http://dx.doi.org/10.1080/01443610701789918.

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34

RAYBURN, WILLIAM F. "Fetal Drug Therapy." Obstetrical & Gynecological Survey 47, no. 1 (January 1992): 1–9. http://dx.doi.org/10.1097/00006254-199201000-00001.

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35

Martin, Stephen. "Schizophrenia: Drug Therapy." Practice Nursing 9, no. 16 (October 6, 1998): 34–37. http://dx.doi.org/10.12968/pnur.1998.9.16.34.

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36

Hitchings, Andrew W. "Monitoring drug therapy." Medicine 40, no. 7 (July 2012): 376–81. http://dx.doi.org/10.1016/j.mpmed.2012.04.001.

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37

Hitchings, Andrew W. "Monitoring drug therapy." Medicine 44, no. 7 (July 2016): 427–32. http://dx.doi.org/10.1016/j.mpmed.2016.04.004.

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38

Hitchings, Andrew W. "Monitoring drug therapy." Medicine 48, no. 7 (July 2020): 456–62. http://dx.doi.org/10.1016/j.mpmed.2020.04.008.

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39

Julian, Desmond G. "CARDIOVASCULAR DRUG THERAPY." Chest 92, no. 4 (October 1987): 30. http://dx.doi.org/10.1016/s0012-3692(16)31280-6.

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40

Rosenberg, Adam A., and Henry L. Galan. "FETAL DRUG THERAPY." Pediatric Clinics of North America 44, no. 1 (February 1997): 113–35. http://dx.doi.org/10.1016/s0031-3955(05)70466-1.

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41

Britton, Jeffrey W. "ANTIEPILEPTIC DRUG THERAPY." CONTINUUM: Lifelong Learning in Neurology 16 (June 2010): 105–20. http://dx.doi.org/10.1212/01.con.0000368234.64857.86.

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42

Somberg, John. "Antiarrhythmic Drug Therapy." Cardiology 72, no. 5-6 (1985): 329–48. http://dx.doi.org/10.1159/000173888.

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43

Polizzi, Maureen Goodhue. "Drug therapy 101." Nursing 45, no. 1 (January 2015): 44–46. http://dx.doi.org/10.1097/01.nurse.0000458920.95597.77.

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44

Podell, Michael. "Antiepileptic drug therapy." Clinical Techniques in Small Animal Practice 13, no. 3 (August 1998): 185–92. http://dx.doi.org/10.1016/s1096-2867(98)80040-6.

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45

Nance, Patricia, and Jay Meythaler. "Intrathecal Drug Therapy." Physical Medicine and Rehabilitation Clinics of North America 10, no. 2 (May 1999): 385–401. http://dx.doi.org/10.1016/s1047-9651(18)30202-x.

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46

Wilson, Richard. "Cardiac drug therapy." Journal of Vascular Surgery 24, no. 6 (December 1996): 1070–71. http://dx.doi.org/10.1016/s0741-5214(96)70063-0.

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47

Robson, S. C. "Fetal drug therapy." Current Obstetrics & Gynaecology 5, no. 4 (December 1995): 230–38. http://dx.doi.org/10.1016/s0957-5847(95)80009-3.

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48

Sobel, Michael. "Cardiovascular drug therapy." Journal of Vascular Surgery 13, no. 4 (April 1991): 567–68. http://dx.doi.org/10.1016/0741-5214(91)90329-s.

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49

Handler, Clive E. "Cardiovascular drug therapy." International Journal of Cardiology 19, no. 2 (May 1988): 275. http://dx.doi.org/10.1016/0167-5273(88)90092-7.

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50

Papich, Mark G. "Antibacterial Drug Therapy." Veterinary Clinics of North America: Small Animal Practice 28, no. 2 (March 1998): 215–31. http://dx.doi.org/10.1016/s0195-5616(98)82002-0.

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