Academic literature on the topic 'Drosophila'
Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles
Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Drosophila.'
Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.
You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.
Journal articles on the topic "Drosophila":
BAŞPINAR, Hüseyin, Tülin AKŞİT, Alper KESİCİ, Ferenc DEUTSCH, Balazs KİSS, and Laszlo PAPP. "Aydın İli (Türkiye) meyve bahçelerindeki Drosophilidae (Diptera) familyası türlerinin mevsimsel yoğunlukları ve tür çeşitliliği ve birlikte saptanan diğer Diptera türleri." Turkish Journal of Entomology 46, no. 3 (September 1, 2022): 289–98. http://dx.doi.org/10.16970/entoted.1088263.
Hotimah, Husnul, Purwatiningsih Purwatiningsih, and Kartika Senjarini. "Morphological Description of Drosophila melanogaster Wild Type (Diptera:Drosophilidae), Sepia and Plum Strain." Jurnal ILMU DASAR 18, no. 1 (February 1, 2017): 55. http://dx.doi.org/10.19184/jid.v18i1.3113.
VAN DER LINDE, KIM, DAVID HOULE, GREG S. SPICER, and SCOTT J. STEPPAN. "A supermatrix-based molecular phylogeny of the family Drosophilidae." Genetics Research 92, no. 1 (February 2010): 25–38. http://dx.doi.org/10.1017/s001667231000008x.
Boycheva Woltering, Svetlana, Jörg Romeis, and Jana Collatz. "Influence of the Rearing Host on Biological Parameters of Trichopria drosophilae, a Potential Biological Control Agent of Drosophila suzukii." Insects 10, no. 6 (June 25, 2019): 183. http://dx.doi.org/10.3390/insects10060183.
Karaningannavar, Shwetha, Rajat Hegde, and Ramesh Babu Yarajarla. "Simple and Rapid PCR-RFLP based species identification in Drosophila suzukii and Drosophila immigrans larvae." Research Journal of Biotechnology 19, no. 3 (January 31, 2024): 48–50. http://dx.doi.org/10.25303/1903rjbt048050.
Liu, Xuxiang, Yongbang Yang, Qingwen Fan, Qinyuan Zhang, and Qinge Ji. "Effect of Ultraviolet-B Radiating Drosophila melanogaster as Host on the Quality of Trichopria drosophilae, a Pupal Parasitoid of Drosophila suzukii." Insects 14, no. 5 (April 28, 2023): 423. http://dx.doi.org/10.3390/insects14050423.
Trivellone, Valeria, Michela Meier, Corrado Cara, Lucia Pollini Paltrinieri, Felix Gugerli, Marco Moretti, Sarah Wolf, and Jana Collatz. "Multiscale Determinants Drive Parasitization of Drosophilidae by Hymenopteran Parasitoids in Agricultural Landscapes." Insects 11, no. 6 (May 30, 2020): 334. http://dx.doi.org/10.3390/insects11060334.
Chassagnard, Μ. Τ., and L. Tsacas. "Drosophila schmidti Duda: redescription et iconographie des genitalia (Diptera, Drosophilidae)." ENTOMOLOGIA HELLENICA 5 (May 31, 2017): 69. http://dx.doi.org/10.12681/eh.13950.
Buonocore Biancheri, María Josefina, Segundo Ricardo Núñez-Campero, Lorena Suárez, Marcos Darío Ponssa, Daniel Santiago Kirschbaum, Flávio Roberto Mello Garcia, and Sergio Marcelo Ovruski. "Implications of the Niche Partitioning and Coexistence of Two Resident Parasitoids for Drosophila suzukii Management in Non-Crop Areas." Insects 14, no. 3 (February 23, 2023): 222. http://dx.doi.org/10.3390/insects14030222.
Wilson, Carolyn. "A review of the monitoring and management of Spotted-Wing Drosophila (Drosophila suzukii) in lowbush blueberrie." Proceedings of the Nova Scotian Institute of Science (NSIS) 49, no. 1 (March 30, 2017): 145. http://dx.doi.org/10.15273/pnsis.v49i1.6984.
Dissertations / Theses on the topic "Drosophila":
Le, Thomas Adrien. "Piwi function and piRNA cluster regulation : Drosophila melanogaster." Electronic Thesis or Diss., Paris 6, 2014. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2014PA066688.pdf.
PiRNAs are a diverse population of small RNA found in the animal germline to silence mobile genetic elements: loaded into Piwi proteins, they guide homology-dependent cleavage of active transposon mRNAs. In Drosophila, three Piwi proteins are expressed, from which two, AUB and AGO3, are known to destroy transposon transcripts in the cytoplasm. The third one, Piwi itself, is nuclear and the molecular mechanism of its function remains unknown. The main sources of piRNAs are discrete genomic loci called piRNA clusters, however it is not known what differentiate them from non-piRNA producing loci. During my PhD, I focused my work on two central questions:1) What is the role of Piwi in the nucleus? We showed that Piwi is responsible for transcriptional silencing by mediating installment of repressive marks, especially H3K9me3, over active transposons copies in a piRNA dependent manner.2) How are piRNA clusters defined, and what regulates their expression? Analyzing what features differentiate a piRNA producing loci from any non-producing loci in the genome, we were able to single out some specific characteristics: . We showed that maternally inherited piRNAs are responsible to define germline clusters at the next generation through two mechanisms: in the nucleus, by deposition of H3K9me3 onto complementary genomic sequence, and, in the cytoplasm, by initiating the ping-pong cycle using cluster transcripts as substrates, leading to their processing into mature piRNAs.. We found that cluster promoters are essential to mediate full cluster transcription, which is allowed thanks to a very specific chromatin signature necessary to ensure piRNA production
Le, Thomas Adrien. "Piwi function and piRNA cluster regulation : Drosophila melanogaster." Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066688/document.
PiRNAs are a diverse population of small RNA found in the animal germline to silence mobile genetic elements: loaded into Piwi proteins, they guide homology-dependent cleavage of active transposon mRNAs. In Drosophila, three Piwi proteins are expressed, from which two, AUB and AGO3, are known to destroy transposon transcripts in the cytoplasm. The third one, Piwi itself, is nuclear and the molecular mechanism of its function remains unknown. The main sources of piRNAs are discrete genomic loci called piRNA clusters, however it is not known what differentiate them from non-piRNA producing loci. During my PhD, I focused my work on two central questions:1) What is the role of Piwi in the nucleus? We showed that Piwi is responsible for transcriptional silencing by mediating installment of repressive marks, especially H3K9me3, over active transposons copies in a piRNA dependent manner.2) How are piRNA clusters defined, and what regulates their expression? Analyzing what features differentiate a piRNA producing loci from any non-producing loci in the genome, we were able to single out some specific characteristics: . We showed that maternally inherited piRNAs are responsible to define germline clusters at the next generation through two mechanisms: in the nucleus, by deposition of H3K9me3 onto complementary genomic sequence, and, in the cytoplasm, by initiating the ping-pong cycle using cluster transcripts as substrates, leading to their processing into mature piRNAs.. We found that cluster promoters are essential to mediate full cluster transcription, which is allowed thanks to a very specific chromatin signature necessary to ensure piRNA production
Entrevan, Marianne. "Caractérisation de la diversité des sites de fixation des protéines du groupe Polycomb chez la Drosophile." Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT029/document.
Polycomb group (PcG) complexes were initially discovered in Drosophila as transcriptionnal repressors of homeotic genes. To date, we know that they are involves in a large pleithora of biological processes including the maintenance of stem cells plasticity, differentiation, X chromosome inactivation and imprinting. PcG complexes are highly conserved from Drosophila to Humans and can be divided into two main complexes: PRC1 and PRC2 (Polycomb repressive complex 1 and 2). Both complexes have a histone modifying activity: PRC1 catalyses the mono-ubiquitination of the lysine 118 on histone H2A (H2AK118Ub) and PRC2 catalyses the tri-methylation of the lysine 27 on histone H3 (H3K27me3).In Drosophila, these complexes are recruited to cis regulatory elements named Polycomb Responsive Elements (PREs) that drive the epigenetic inheritance of silent chromatin states throughout development. Importantly, PcG complexes do not contain DNA-binding activity but are recruited to PREs via their interaction with Transcription Factors (TF) recognizing DNA motifs clustered at PREs. However the mechanism how PREs target PcG complexes is still not well understood due to the complexity of PcG recruitment, which is reflected at different levels: The DNA signature between PREs can differ significantly and several TF are implicated in PcG recruitment, but none of them is sufficient to recruit PcG complexes to PREs. Moreover PcG complexes can cooperate in different ways to stabilize each other’s binding. Finally, another layer of complexity is found at a more global level since PcG complexes do not only bind repressed sites, but they are also found at active regions.Therefore, our working hypothesis is that different classes of PREs exist in Drosophila. My PhD work was thus to define these different classes of PREs on a genome-wide scale and to functionally characterize them in order to get a complete molecular description of PRE function. Understanding how PcG complexes are recruited is of high importance, since deregulation of both, PcG complexes and their recruiting factors can led to cancer and diseases. My work led to the identification of six different classes of PREs that are characterized by different chromatin and genomic features. Interestingly the majority of PREs are associated with active genes that can be divided into housekeeping regulatory regions and developmental enhancers. In addition another class comprises bona fide chromatin domain boundaries. On the other hand PREs associated with repressed chromatin states shows features of previously described PREs and associate with repressed genes and PcG-associated histone marks. Finally another class comprises PREs that are likely in a poised chromatin state. We further demonstrated that PREs located at repressed and active regions differ in their combination of TF. In vivo analyses along with a transcriptomic analysis performed in cell lines mutated for a member of PcG complexes revealed that PcG complexes play a repressive role at both, active and repressed PREs.Taken together, our result suggest an unexpected heterogeneity of PREs and contributes to the better understanding of their characteristics and function
Barthez, Marine. "Functional characterisation of Drosophila M1BP." Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0298.
The transcription factor Motif 1 Binding protein (M1BP) is a zinc finger protein known to be involved in the pausing of RNA Polymerase II (Pol II) at the transcription start site of thousands of Drosophila genes. I was able to demonstrate direct protein interaction between M1BP and the Drosophila Hox proteins AbdA and Ubx, providing evidence that Hox-M1BP collaborate to regulate gene expression. While M1BP expression is maintained during all larval stages, loss of M1BP expression in the feeding stage induces premature autophagy. In characterising the diverse functions of M1BP, I determined that M1BP transcriptionally regulates 25% of all cellular metabolic pathways. Indeed, many severe mitochondrial defects and phenotypes are observed upon M1BP knock down in the Drosophila fat body and indirect flight muscle. One of the major consequences of M1BP knock down is that the respiratory chain is strongly impacted forcing the cell to switch to anaerobic respiration for the production of ATP. Together, these data provide evidence that M1BP is an essential transcriptional regulator of mitochondrial and cellular metabolic processes. In searching for a vertebrate homolog of Drosophila M1BP, I identified ZKSCAN3 as a vertebrate functional homolog of M1BP in autophagy repression in Drosophila fat body or in human cell lines. Additionally, transcriptomic studies demonstrate that ZKSCAN3 expression in the Drosophila fat body reverses the deregulation of the majority of genes observed upon M1BP knock down. Together with the identification that ZKSCAN3 binds to 90% of M1BP genomic targets, these data provide evidence that M1BP and ZKSCAN3 are functionally homologous proteins
Ducuing, Antoine. "Signalling and morphogenesis during Drosophila dorsal closure." Thesis, Lyon, 2016. http://www.theses.fr/2016LYSEN002/document.
Drosophila dorsal closure is a key embryonic process during which the dorsal-most epidermal cells called leading edge cells differentiate and act in a coordinated manner to close a transient dorsal hole covered by the amnioserosa in a process reminiscent of wound healing. I showed that JNK and DPP are wired in a network motif called ‘feed-forward loop’ (FFL) that controls leading edge cell specification and differentiation. The DPP branch of the FFL filters unwanted JNK activity that occurs during thermal stress. Next, I focused on the actin cable, a supra-cellular structure produced by the leading edge cells during dorsal closure or wound healing from fly to humans. My data suggest that the actin cable does not provide a major contractile force. Rather, the actin cable balances forces and stabilizes cell geometry so that closure resolves in a perfectly structured and scar-free tissue. The absence of the cable leads to cell shape irregularities as well as patterning and planar cell polarity defects that are reminiscent of scarring. We propose that the cable prevents scaring by acting as a mechanical freeze field that protects fine cellular structures from the major closure forces that operate at tissue level. Altogether, my work brings new insights on the signalling and morphogenesis during dorsal closure
Lepot, Frédérique. "Recherches sur l'apprentissage associatif chez la drosophile (drosophila melanogaster)." Toulouse 3, 1986. http://www.theses.fr/1986TOU30033.
Lepot, Frédérique. "Recherches sur l'apprentissage associatif chez la Drosophile, Drosophila melanogaster." Grenoble 2 : ANRT, 1986. http://catalogue.bnf.fr/ark:/12148/cb37599134t.
Ruby, Vincent. "Étude des évènements mitochondriaux impliqués dans le contrôle de l'apoptose par rbf1, l'homologue de drosophile du gène suppresseur de tumeur rb." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLV039/document.
The gene rb is the first tumor suppressor discovered in humans. Its prevents the appearance of tumors by regulating negatively the cell cycle. The role of pRb in apoptosis is more complex and the molecular mechanisms triggered by this transcription factor are not completely elucidated. There is a rb homologue in drosophila: rbf1. I participated in the characterization of mitochondrial events induced during activation of apoptosis by Rbf1 in a proliferating tissue of this model organism, the wing disc. In this apoptosis pathway, the Debcl protein, the only drosophila pro-apoptotic member of the Bcl-2 family, is activated and induces recruitment and oligomerization of Drp1, the main effector of mitochondrial fission. This triggers the mitochondrial fragmentation and the accumulation of mitochondrial reactive oxygen species (ROS). Both events participate to the transmission of the apoptotic signal. I have also been able to highlight the implication of factors involved in maintaining mitochondrial quality control which ensures the integrity of the mitochondria and, if necessary, triggers the degradation of damaged elements by mitophagy. Finally, I have contributed to the study of the links between translation and apoptosis induced by Rbf1. In this study, we show that the Poly-A Binding Protein (PABP) can suppress the Rbf1-induced notch phenotype in adults while cell death induced during larval stage was not inhibited but increased. These results prompted us to study the compensation mechanisms induced by the translational apparatus, which allowed us to show that a mRNA translation-related mechanism could counteract the loss of tissue resulting from Rbf1-induced apoptosis independently of apoptosis inhibition
Garrido, Damien. "Etude de l’homéostasie lipidique chez Drosophila melanogaster." Thesis, Université Paris-Saclay (ComUE), 2015. http://www.theses.fr/2015SACLS030.
Fatty acid (FA) metabolism is crucial in maintaining homeostasis, but also in a numerous of processes including signaling, energy storage, protection to temperature loss, regulation of behavior... In addition, FA metabolism is deregulated in several pathologies including diabetes, obesity, and cancers... Therefore, the enzymes that catalyze the reactions of the FA metabolic pathways constitute attractive targets to develop novel therapies. However the consequences of these deregulations in healthy organism are still poorly known, in particular at the level of each organ.The aim of my PhD was to estimate how FA metabolism participates in the regulation of homeostasis within a whole body organism. To address these issues, I used the genetic possibilities of the Drosophila model, whose metabolism is similar to that of mammals.I showed that FA synthesis contributes to neutralize the toxic effects of dietary sugar. This process operates in cooperation with the methylglyoxal detoxification pathway, which prevents the formation of compounds resulting from the non-enzymatic glycation. I also contributed to a project showing that the precursors of hydrocarbons and pheromones have a flexible origin, which depends on lipid homeostasis and may affect sexual recognition between individuals. Currently, I’m studying the consequences of FA synthesis inhibition in various deregulated growth models. Finally, in a preliminary work, I showed that the FA metabolism is essential in the digestive tract, possibly by disrupting water homeostasis in larvae. Taken together, these results will help to characterize the importance of FA metabolism in healthy organism as well as in deregulated processes
Lamiré, Laurie-Anne. "Identification and characterization of the mechanical role of germline growth in Drosophila melanogaster epithelial morphogenesis." Thesis, Lyon, 2019. http://www.theses.fr/2019LYSEN002.
Epithelial morphogenesis is essential for organ formation. I use the Drosophila ovarian follicle as a model for studying cell flattening. A follicle is composed of growing germ cells surrounded by a monolayer of cuboidal epithelial cells. At a specific stage of development, some of these cells flatten out following a regulated wave. This flattening is partly controlled by a pressure gradient from part of the germ cells (the nurse cells). All germ cells are connected via cytoplasmic bridges. This thesis studies the mechanisms leading to the generation of the gradient of pressure, and to the molecular modulation induced by this mechanical force to allow flattening. I have shown that the number and diameter of cytoplasmic bridges affect the pressure. Using three-dimensional reconstructions of follicles, I studied the role of differential growth of nurse cells by measuring the change in germ cell volume during epithelial cell flattening. Finally, I looked for the molecular mechanism leading to the flattening and influenced by a mechanical stimulus from the germinal pressure, supporting a role of the Hippo pathway in this process. In conclusion, we propose that germ cell growth mechanically and genetically influences epithelial cells to allow elongation, and thus the acquisition of the final form of the follicle
Books on the topic "Drosophila":
Dahmann, Christian, ed. Drosophila. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2541-5.
Dahmann, Christian, ed. Drosophila. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-583-1.
Dahmann, Christian, ed. Drosophila. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6371-3.
Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.
Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.
Ashburner, M. Drosophila. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1989.
1929-, Tobari Yoshiko N., ed. Drosophila ananassae: Genetical and biological aspects. Tokyo: Japan Scientific Societies Press, 1993.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. Drosophila Genetics. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7.
Bratu, Diana P., and Gerard P. McNeil, eds. Drosophila Oogenesis. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2851-4.
Arkhipova, Irina R. Drosophila retrotransposons. New York: Springer-Verlag, 1995.
Book chapters on the topic "Drosophila":
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "General." In Drosophila Genetics, 1–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_1.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Morphology of Drosophila Melanogaster." In Drosophila Genetics, 33–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_2.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Transmission Genetics." In Drosophila Genetics, 55–101. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_3.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Phenogenetics." In Drosophila Genetics, 103–34. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_4.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Mutation Genetics." In Drosophila Genetics, 135–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_5.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Population Genetics." In Drosophila Genetics, 163–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_6.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Cytology and Cytogenetics." In Drosophila Genetics, 177–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_7.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Molecular Biology." In Drosophila Genetics, 189–202. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_8.
Graf, Ulrich, Nancy van Schaik, and Friedrich E. Würgler. "Results and Answers." In Drosophila Genetics, 203–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-76805-7_9.
Romeo, Yves, and Bruno Lemaitre. "Drosophila Immunity." In Innate Immunity, 379–94. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-570-1_22.
Conference papers on the topic "Drosophila":
Zhang, Aijun. "Attractive blend for spotted wing drosophila,Drosophila suzukii(Matsumura)." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.111493.
Liang, Jingdong, Jixiang Sun, and Yuancheng Xie. "Research of drosophila detection and courtship drosophila differentiation against complicated background." In 2011 International Conference on Electrical and Control Engineering (ICECE). IEEE, 2011. http://dx.doi.org/10.1109/iceceng.2011.6057886.
Sward, Grace. "An exploration into pesticide resistance in spotted wing drosophila, Drosophila suzukii." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.114081.
Yi, Hoonbok. "Drosophila suzukiiin South Korea." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93351.
GÖPFERT, M. C., and D. ROBERT. "MICROMECHANICS OF DROSOPHILA AUDITION." In Proceedings of the International Symposium. WORLD SCIENTIFIC, 2003. http://dx.doi.org/10.1142/9789812704931_0042.
Becher, Paul G. "Detangling behavioral responses to semiochemicals in the spotted wing drosophila,Drosophila suzukii." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93348.
Guédot, Christelle. "Using CSIA to identify unexpected hosts for spotted wing drosophila,Drosophila suzukii." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94563.
Tang, Siew Bee. "Screening of RNAi targets and impacts on spotted wing drosophila,Drosophila suzukii." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.115004.
Kirkpatrick, Danielle M. "Improving the efficiency of monitoring tools for spotted wing drosophila,Drosophila suzukii." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.108543.
Wells, Simon, and Chris Reed. "A drosophila for computational dialectics." In the fourth international joint conference. New York, New York, USA: ACM Press, 2005. http://dx.doi.org/10.1145/1082473.1082724.
Reports on the topic "Drosophila":
Bernards, Andre. Functional Analysis of Drosophila NF1. Fort Belvoir, VA: Defense Technical Information Center, July 2005. http://dx.doi.org/10.21236/ada444270.
Walker, James A. Developing a Drosophila Model of Schwannomatosis. Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada575950.
Walker, James A. Developing a Drosophila Model of Schwannomatosis. Fort Belvoir, VA: Defense Technical Information Center, February 2013. http://dx.doi.org/10.21236/ada575951.
Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, December 2008. http://dx.doi.org/10.21236/ada488787.
Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, January 2009. http://dx.doi.org/10.21236/ada532314.
Zhong, Yi. Functional Analysis of Human NF1 in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ada465210.
Greenspan, Ralph J. Gene Networks Underlying Chronic Sleep Deprivation in Drosophila. Fort Belvoir, VA: Defense Technical Information Center, June 2014. http://dx.doi.org/10.21236/ada610340.
Gerald M. Rubin. Resources for Biological Annotation of the Drosophila Genome. Office of Scientific and Technical Information (OSTI), August 2005. http://dx.doi.org/10.2172/842216.
Zhong, Yi. NF-1 Dependent Gene Regulation in Drosophila Melanogaster. Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada471891.
Segal, Daniel, Lawrence Gilbert, and Shalom Applebaum. Molecular Genetic Dissection of Juvenile Hormone Synthesis in Drosophila. United States Department of Agriculture, June 1993. http://dx.doi.org/10.32747/1993.7604297.bard.