Relevant bibliographies by topics / DoS Resistance

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'DoS Resistance'

Author: Grafiati
Published: 28 June 2021
Last updated: 13 February 2022

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Journal articles on the topic "DoS Resistance"

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Pink, D. A. C., and P. Hand. "Plant resistance and strategies for breeding resistant varieties." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (January 1, 2002): S9—S14. http://dx.doi.org/10.17221/10310-pps.

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An explanation of the ‘boom-bust’ cycle of resistance breeding was provided by the gene-for-gene relationship between a pathogen and its host. Despite this understanding, most R genes continued to be deployed singly and resistance has been ephemeral. The reasons for breeding ‘single R gene’ varieties are discussed. Alternative strategies for the deployment of R genes and the use of quantitative race non-specific resistance have been advocated in order to obtain durable resistance. The feasibility of both of these approaches is discussed taking into account the impact of technologies such as plant transformation and marker-assisted selection. A change in focus from durability of the plant phenotype to that of the crop phenotype is advocated.
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Çelik, Cem. "Increasing antimicrobial resistance in nosocomial pathogens; multidrug-resistant extensively drug-resistant and pandrug-resistant Acinetobacter baumannii." Journal of Microbiology and Infectious Diseases 4, no. 1 (March 1, 2014): 7–12. http://dx.doi.org/10.5799/ahinjs.02.2014.01.0116.

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Андреевская, С. Н., Т. Г. Смирнова, Е. Е. Ларионова, И. Ю. Андриевская, Л. Н. Черноусова, and А. Эргешов. "Изониазид-резистентные Mycobacterium tuberculosis: частота выявления, спектры резистентности и генетические детерминанты устойчивости." Phylogenetic Analysis, no. 1;2020 (2020): 22–28. http://dx.doi.org/10.24075/vrgmu.2020.001.

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Отсутствие ускоренной диагностики туберкулеза с устойчивостью возбудителя к изониазиду с сохраненной чувствительностью к рифампицину (ИР-ТБ) может быть причиной низкой эффективности терапии и приводить к амплификации лекарственной резистентности, в том числе к формированию множественной лекарственной устойчивости. Целью работы было определить частоту встречаемости ИР-ТБ в современной популяции, охарактеризовать фенотипическую чувствительность и генетические детерминанты устойчивости к изониазиду представителей этой группы M. tuberculosis на репрезентативном материале. Анализировали результаты определения лекарственной чувствительности, полученные при исследовании молекулярно- генетическими и/или культуральными методами изолятов M. tuberculosis / ДНК M. tuberculosis, выделенных от больных туберкулезом легких из клинических отделений Центрального научно-исследовательского институа туберкулеза за период 2011–2018 гг. Частота ИР-ТБ составила 12% от всех выявленных случаев туберкулеза. M. tuberculosis с ИР были как монорезистентными к изониазиду (45%), так и полирезистентными (устойчивыми к 2–6 противотуберкулезным препаратам), а устойчивость к изониазиду была обусловлена мутациями в гене katG, приводящими к высокому уровню резистентности. На основании анализа литературных данных и собственных наблюдений подчеркивается важность разработки и внедрения новых простых молекулярных тестов для определения устойчивости одновременно к рифампицину и изониазиду.
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Gokalp, Deniz, Alpaslan Tuzcu, Ster Irmak, Mithat Bahceci, and Ozlem Demirpence. "FREQUENCY OF ASPIRIN RESISTANCE IN PATIENTS WITH TYPE 1 AND 2 DIABETES MELLITUS AND ITS ASSOCIATION WITH METABOLIC PARAMETERS." International Journal of Surgery and Medicine 2, no. 3 (2016): 122. http://dx.doi.org/10.5455/ijsm.aspirin-resistance-diabetes.

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Singh, Amresh Kumar. "Resistance patterns and trends of extensively drug-resistant tuberculosis: 5-year experience." Journal of Microbiology and Infectious Diseases 03, no. 04 (December 1, 2013): 169–75. http://dx.doi.org/10.5799/ahinjs.02.2013.04.0103.

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Kumar, Sinha Ritesh, and Chandra Satish. "Insulin resistance." Asian Pacific Journal of Health Sciences, Supplimentary 2014 (2014): 71–78. http://dx.doi.org/10.21276/apjhs.2014.1.1s.15.

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Shahina, Sk jasmine, Sheeba Ali Siddiqui, and Dr Padma Krishnan. "Nasal Carriage of Methicillin Resistant Staphylococci with Inducible Clindamycin Resistance and Pvl Gene." International Journal of Scientific Research 3, no. 6 (June 1, 2012): 392–94. http://dx.doi.org/10.15373/22778179/june2014/132.

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Wang, S. C., and P. S. Wei. "Modeling Dynamic Electrical Resistance During Resistance Spot Welding." Journal of Heat Transfer 123, no. 3 (November 28, 2000): 576–85. http://dx.doi.org/10.1115/1.1370502.

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Dynamic electrical resistance during resistance spot welding has been quantitatively modeled and analyzed in this work. A determination of dynamic resistance is necessary for predicting the transport processes and monitoring the weld quality during resistance spot welding. In this study, dynamic resistance is obtained by taking the sum of temperature-dependent bulk resistance of the workpieces and contact resistances at the faying surface and electrode-workpiece interface within an effective area corresponding to the electrode tip where welding current primarily flows. A contact resistance is composed of constriction and film resistances, which are functions of hardness, temperature, electrode force, and surface conditions. The temperature is determined from the previous study in predicting unsteady, axisymmetric mass, momentum, heat, species transport, and magnetic field intensity with a mushy-zone phase change in workpieces, and temperature and magnetic fields in the electrodes of different geometries. The predicted nugget thickness and dynamic resistance versus time show quite good agreement with available experimental data. Excluding expulsion, the dynamic resistance curve can be divided into four stages. A rapid decrease of dynamic resistance in stage 1 is attributed to decreases in contact resistances at the faying surface and electrode-workpiece interface. In stage 2, the increase in dynamic resistance results from the primary increase of bulk resistance in the workpieces and an increase of the sum of contact resistances at the faying surface and electrode-workpiece interface. Dynamic resistance in stage 3 decreases, because increasing rate of bulk resistance in the workpieces and contact resistances decrease. In stage 4 the decrease of dynamic resistance is mainly due to the formation of the molten nugget at the faying surface. The molten nugget is found to occur in stage 4 rather than stage 2 or 3 as qualitatively proposed in the literature. The effects of different parameters on the dynamic resistance curve are also presented.
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Cohen, Y. "Systemic induced resistance." Plant Protection Science 38, SI 1 - 6th Conf EFPP 2002 (January 1, 2002): S122—S125. http://dx.doi.org/10.17221/10334-pps.

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Biotic and abiotic agents may induce resistance in plants against pathogens. Abiotic agents may be synthetic or natural. The natural, non-protein amino acid BABA (DL-β-aminobutyric acid) induces systemic resistance in crop plants against pathogens. Dry, killed mycelia of Penicillium chrysogenum (DM) induces local resistance in plants against soil-borne pathogens. The activity of BABA and DM are described here in detail. Both products were shown to effectively control plant disease in nature.
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Manninger, K. "Effective Resistance Genes as Sources of Resistance against Hungarian Eheat Rusts." Czech Journal of Genetics and Plant Breeding 38, No. 3-4 (August 1, 2012): 153–54. http://dx.doi.org/10.17221/6257-cjgpb.

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Dissertations / Theses on the topic "DoS Resistance"

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Börjesson, Stefan. "Antibiotic Resistance in Wastewater : Methicillin-resistant Staphylococcus aureus (MRSA)and antibiotic resistance genes." Doctoral thesis, Linköpings universitet, Medicinsk mikrobiologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-17709.

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A large part of the antibiotics consumed ends up in wastewater, and in the wastewater the antibiotics may exert selective pressure for or maintain resistance among microorganisms. Antibiotic resistant bacteria and genes encoding antibiotic resistance are commonly detected in wastewater, often at higher rates and concentrations compared to surface water. Wastewater can also provide favourable conditions for the growth of a diverse bacterial community, which constitutes a basis for the selection and spread of antibiotic resistance. Therefore, wastewater treatment plants have been suggested to play a role in the dissemination and development of antibiotic resistant bacteria. Methicillin-resistant Staphylococcus aureus (MRSA) is a large problem worldwide as a nosocomial pathogen, but knowledge is limited about occurrence in non-clinical environments, such as wastewater, and what role wastewater plays in dissemination and development of MRSA.   In this thesis we investigated the occurrence of MRSA in a full-scale wastewater treatment plant (WWTP). We also investigated the concentration of genes encoding resistance to aminoglycosides (aac(6’)-Ie+aph(2’’)), β-lactam antibiotics (mecA) and tetracyclines (tetA and tetB) in three wastewater-associated environments: (1) soil from an overland flow area treating landfill leachates, (2) biofilm from a municipal wastewater treatment plant, and (3) sludge from a hospital wastewater pipeline. In addition, concentrations of mecA, tetA and tetB were investigated over the treatment process in the WWTP. These investigations were performed to determine how the prevalence and concentration of MRSA and the antibiotic resistence genes are affected in wastewater and wastewater treatment processes over time. The occurrence of MRSA was investigated by cultivation and a commercially available real-time PCR assay. In order to determine concentrations of the genes aac(6’)-Ie+aph(2’’), mecA, tetA and tetB in wastewater we developed a LUXTM real-time PCR assay for each gene.   Using cultivation and real-time PCR we could for the first time describe the occurrence of MRSA in wastewater and show that it had a stable occurrence over time in a WWTP. MRSA could mainly be detected in the early treatment steps in the WWTP, and the wastewater treatment process reduced the number and diversity of cultivated MRSA. However, our results also indicate that the treatment process selects for strains with more extensive resistance and possibly higher virulence. The isolated wastewater MRSA strains were shown to have a close genetic relationship to clinical isolates, and no specific wastewater lineages could be detected, indicating that they are a reflection of carriage in the community. Taken together, these data indicate that wastewater may be a potential reservoir for MRSA and that MRSA are more prevalent in wastewater than was previously thought.   The real-time PCR assays, for aac(6’)-Ie+aph(2’’), mecA, tetA, and tetB that we developed, were shown to be sensitive, fast, and reproducible methods for detection and quantification of these genes in wastewater environments. The highest concentrations of all genes were observed in the hospital pipeline, and the lowest in the overland flow system, with tetA and aac(6´)-Ie+aph(2´´) detected in all three environments. In the full-scale WWTP, we continuously detected mecA, tetA and tetB over the treatment process and over time. In addition, it was shown that the treatment process reduces concentrations of all three genes. The data presented in this thesis also indicate that the reduction for all three genes may be connected to the removal of biomass, and in the reduction of tetA and tetB, sedimentation and precipitation appear to play an important role.
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OUNNOUGHENE, MARC Kahn Jean-Pierre. "LA DEPRESSION RESISTANTE QUELS TRAITEMENTS POUR QUELLE RESISTANCE ? /." [S.l.] : [s.n.], 2000. http://www.scd.uhp-nancy.fr/docnum/SCDMED_T_2000_OUNNOUGHENE_MARC.pdf.

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Pfeltz, Richard F. Wilkinson Brian J. "The effects of vancomycin resistance selection and magnesium on resistance expression in methicillin-resistant Staphylococcus aureus." Normal, Ill. Illinois State University, 1999. http://wwwlib.umi.com/cr/ilstu/fullcit?p9927774.

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Thesis (Ph. D.)--Illinois State University, 1999.
Title from title page screen, viewed July 20, 2006. Dissertation Committee: Brian J. Wilkinson (chair), Radheshyam K. Jayaswal, Alan J. Katz, Anthony J. Otsuka, David L. Williams. Includes bibliographical references and abstract. Also available in print.
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Butler, C. A. "Mechanisms of steroid resistance in therapy resistant asthma." Thesis, Queen's University Belfast, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.546017.

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Maffeis, Laura <1981&gt. "Correlation between insulin resistance and treatment-resistant acne." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2013. http://amsdottorato.unibo.it/5654/.

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Physiologically during puberty and adolescence, when juvenile acne usually appears, the response to a glucose load is increased if compared to the one observed in adult and at pre-pubertal age, while insulin sensitivity is reduced. Insulin is a hormone that acts at different levels along the axis which controls the sex hormones. It increases the release of LH and FSH by pituitary gland, stimulates the synthesis of androgens in the gonads and stimulates the synthesis of androgenic precursors in adrenal glands. Finally, it acts in the liver by inhibiting the synthesis of Sex Hormone Binding Globulin (SHBG). Insulin is also able to act directly on the production of sebum and amplify the effects of Iinsulin Growth Factor-1 in the skin, inhibiting the synthesis of its binding protein (IGF Binding Protein-1). In female subjects with acne and Polycystic Ovary Syndrome (PCOS) insulin resistance is a well known pathogenetic factor, while the relationship between acne and insulin resistance has been poorly investigated in males so far. The purpose of this study is to investigate the correlation between insulin resistance and acne in young males who do not respond to common therapies. Clinical and biochemical parameters of glucose, lipid metabolism, androgens and IGF-1 were evaluated. Insulin resistance was estimated by Homeostasis Model assessment (HOMA-IR) and Oral Glucose Tolerance Test was also performed. We found that subjects with acne had higher Sistolic and Diastolic Blood Pressure, Waist/Hip Ratio, Waist Circumference, 120' OGTT serum insulin and serum IGF-1 and lower HDL-cholesterol than subjects of comparable age and gender without acne. The results thus obtained confirmed what other authors have recently reported about a metabolic imbalance in young males with acne. Furthermore, these results support the hypothesis that insulin resistance might play an important role in the pathogenesis of treatment-resistant acne in males.
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Barnawi, Ibrahim. "Mechanisms of apoptosis resistance in chemotherapy-resistant cancer cells." Thesis, University of Reading, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627931.

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Most chemotherapy drugs work through the induction of apoptosis in tumour cells. Mutations in apoptotic pathways, notably p53 and Bcl-2, are common in cancer and associated with increased resistance to apoptosis and therefore to chemotherapy. The sensitivity of two types of cells have been compared, A 172 human glioblastoma cells and MDA-MB-23l human breast cancer cells, for their sensitivity to the chemotherapy drugs cisplatin, doxorubicin, gemcitabine, vincristine and temozolomide. Data shows that there were differences in the sensitivity of breast cancer and glioma cells to a variety of chemotherapy drugs. There were differences in nitric oxide synthase (NOS) expression between the two different types of cancer cells and yet a role was not observed for nitric oxide in changing the expression of the Bcl-2 family of proteins (both pro- or antiapoptotic). Additionally, NOS expression changed following treatment with chemotherapy drugs, suggesting that this may be the mechanism by which the cells become resistant.
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Pelfrey, Clara Mackenzie. "Genetic resistance to experimental autoimmune encephalomyelitis : analysis of the resistance mechanisms in the Lewis resistant or LeR rat /." The Ohio State University, 1988. http://rave.ohiolink.edu/etdc/view?acc_num=osu148759742413614.

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Chung, Whasun Oh. "Macrolide resistance and its linkage to tetracycline resistance /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/9279.

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Cornick, Jen. "Roadmap to resistance : antimicrobial resistance in Malawian pneumococci." Thesis, University of Liverpool, 2012. http://livrepository.liverpool.ac.uk/11173/.

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Multi-drug resistant (MDR) Streptococcus pneumoniae are a major public health concern worldwide. In Malawi a resource poor country, even the simplest antimicrobials remain a precious commodity. Given the limited number of antimicrobials available for the management of MDR invasive pneumococcal disease (IPD) measures need to be implemented to limit the spread of resistance. In order to design such measures it is essential that we gain a better understanding of the evolution of antimicrobial resistance in this setting. The purpose of this thesis was to assess the molecular basis and mode of dissemination of antimicrobial resistance in S. pneumoniae with the aim of identifying a biomarker of antimicrobial resistance that could be used to design a diagnostic PCR to assist epidemiological surveillance of antimicrobial resistance and inform treatment policy. Malawi introduced the 13-valent pneumococcal conjugate vaccine (PCV13) in 2011. To provide baseline data to assess the impact of PCV13 all invasive pneumococci isolated from children admitted to Queen Elizabeth Central Hospital, Malawi 2004-2006 were serotyped and subjected to antimicrobial susceptibility testing. The data suggested PCV13 will not provide protection against 61% of penicillin resistant pneumococci and if serotype replacement occurs following the introduction of PCV13, the incidence of penicillin resistant IPD could therefore increase Over 130 resistant and susceptible pneumococcal isolates from carriage and invasive disease were subjected to whole genome sequencing. The employment of an in vitro and in silco analytical approach established that S. pneumoniae employs a diverse array of antimicrobial resistance mechanisms, the dissemination of which is likely to be driven by high antimicrobial consumption. A relatively high incidence of antimicrobial resistant was observed in serotype 1 pneumococci, the most common cause of IPD in Malawi. This serotype is not usually associated with resistance in other geographic locations, the short duration of serotype 1 carriage is assumed to limit the chance it has to acquire resistance mechanisms via recombination. Interestingly the resistance mechanisms employed by serotype 1 had been acquired through multiple recombination events. Recombination was evidenced to contribute to >90% of the variation in the serotype 1 genomes. To allow the identification of resistance biomarkers free from any preconceptions about which genes are involved in resistance, multiple antimicrobial resistant lineages were generated in vitro. Isolates were sequenced at several time points as resistance developed. Comparison of the resistant isolates to the wild type isolates identified single nucleotide polymorphisms in 46 genes, 40 of these genes have not previously been implicated in antimicrobial resistance. The role of these genes in resistance warrants further investigation. The analysis suggests that rather than a single biomarker future research needs to identify multiple biomarkers; the dynamic nature of this organism means that it can adopt one of many routes to antimicrobial resistance.
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Erkal, Hakan, and Sinan Kebapci. "Resistance to Change : A Constructive Approach for Managing Resistant Behaviors." Thesis, University of Kalmar, Baltic Business School, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:hik:diva-1813.

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This study aims to understand, describe, and analyze the factors that lead employees resist organizational change efforts. More specifically, by locating various types of roots and symptoms of resistance, we have developed a framework which managers or individuals, who plan to initiate a change program, can use to manage resistance and to benefit, if exist, from the constructive value of resistant behaviors of employees. Findings are drawn from the reinterpretation of two case studies which were conducted on the area. While the first one involves introduction of activity-based costing system in a Portuguese telecommunications company, second one analyzes implementation of a new management program, called BATON, in a university funded research organization. By relying on these case studies, existing models and concepts related to resistance were tested, reinterpreted and an alternative framework to manage resistance is developed. As a result of the study, it is found that despite the amount of theoretical concepts and tools, there is still an important deficiency in terms of resistance management, and managers usually tend to employ pre-set methods to overcome resistance in change management. Findings of the thesis provide those who plan to start and implement change programs with a comprehensive framework to locate, understand and analyze resistance and to take appropriate managerial actions in organizational change efforts.

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Books on the topic "DoS Resistance"

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Resistance. London: Little, Brown and Company, 1995.

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manual, National AIDS. Resistance. London: NAM Publications, 1999.

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Dillard, J. M. Resistance: Star Trek: The Next Generation. New York: Pocket Books, 2007.

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Resistance. Dakar?]: Les Éditions Madiba, 2013.

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1973-, Pérez Ramón, Smith C. P, and Chamberlain Kody, eds. Resistance. La Jolla, CA: Wildstorm, 2010.

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Whyte, Jack. Resistance. London: Sphere, 2014.

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Nielsen, Jennifer A. Resistance. New York: Scholastic, Incorporated, 2018.

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ill, Purvis Leland, ed. Resistance. New York: First Second, 2010.

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Resistance. London: Titan Books, 2015.

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Resistance. New York: Little, Brown and Company, 2008.

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Book chapters on the topic "DoS Resistance"

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Harinasuta, K. T., and D. Bunnag. "Resistance." In Parasitology in Focus, 867–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-662-09200-2_21.

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Weik, Martin H. "resistance." In Computer Science and Communications Dictionary, 1478. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_16205.

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Watson, Keith L. "Resistance." In Foundation Science for Engineers, 232–46. London: Macmillan Education UK, 1993. http://dx.doi.org/10.1007/978-1-349-12450-3_25.

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Floresco, Stan, Robert Kessler, Ronald L. Cowan, Robert Kessler, Ronald L. Cowan, Mark Slifstein, Andrea Cipriani, et al. "Resistance." In Encyclopedia of Psychopharmacology, 1154. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_4511.

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Basu, Dipak, and Victoria Miroshnik. "Resistance." In India as an Organization: Volume One, 85–92. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53372-8_3.

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Kimpara, Satoko, Samarea Lenore, and Larry E. Beutler. "Resistance." In Encyclopedia of Personality and Individual Differences, 4437–41. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_849.

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Neenan, Michael, and Windy Dryden. "Resistance." In Cognitive Behaviour Therapy, 262–65. Third edition. | London; New York: Routledge, Taylor & Francis Group, 2021. | Series: 100 key points and techniques: Routledge, 2020. http://dx.doi.org/10.4324/9781003134053-112.

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Weller, Robert P. "Resistance." In Resistance, Chaos and Control in China, 3–15. London: Palgrave Macmillan UK, 1994. http://dx.doi.org/10.1007/978-1-349-13203-4_1.

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Schmiechen-Ackermann, Detlef. "Resistance." In A Companion to Nazi Germany, 129–46. Chichester, UK: John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781118936894.ch8.

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Waygood, Adrian. "Resistance." In An Introduction to Electrical Science, 43–50. Second edition. | Abingdon, Oxon; New York, NY: Routledge,: Routledge, 2018. http://dx.doi.org/10.1201/9781351190435-6.

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Conference papers on the topic "DoS Resistance"

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Grosjean, Luce. "Resistance." In SA '17: SIGGRAPH Asia 2017. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3145631.3145660.

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Roush, Rick. "Resistance management in insect resistant genetically-engineered crops." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93214.

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Chiang, K. W. "Resistance extraction and resistance calculation in GOALIE?" In the 1989 26th ACM/IEEE conference. New York, New York, USA: ACM Press, 1989. http://dx.doi.org/10.1145/74382.74503.

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Owen, Micheal D. K. "Herbicide Resistance." In Proceedings of the First Annual Crop Production and Protection Conference. Iowa State University, Digital Press, 1991. http://dx.doi.org/10.31274/icm-180809-359.

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Sen, P., and J. A. Cocks. "Collision Resistance." In The Safety of RO-RO Passenger Ships. RINA, 1990. http://dx.doi.org/10.3940/rina.roro.1990.03.

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Wu, T. H., J. E. Ho, and P. S. Wei. "Dynamic electrical resistance effects in resistance spot welding." In 2010 5th International Microsystems, Packaging, Assembly and Circuits Technology Conference (IMPACT). IEEE, 2010. http://dx.doi.org/10.1109/impact.2010.5699534.

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Thevanayagam, S., and N. Ecemis. "Effects of Permeability on Liquefaction Resistance and Cone Resistance." In Geotechnical Earthquake Engineering and Soil Dynamics Congress IV. Reston, VA: American Society of Civil Engineers, 2008. http://dx.doi.org/10.1061/40975(318)92.

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Ćurcic, Radmila, Dragana Vukovic, Irena Zivanovic, Ivana Dakic, and Branislava Savic. "Resistance genotypes of multidrug-resistant Mycobacterium tuberculosis isolates in Serbia." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4632.

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Abuelma'atti, Muhammad Taher, and Sagar Kumar Dhar. "CFOA-based floating negative inductance, positive frequency dependent resistance and resistance-controlled capacitance and resistance emulator." In 2016 International Conference on Electronics, Information, and Communications (ICEIC). IEEE, 2016. http://dx.doi.org/10.1109/elinfocom.2016.7562980.

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Hartzler, Bob. "Herbicide Resistance Update." In Proceedings of the 1995 Integrated Crop Management Conference. Iowa State University, Digital Press, 1998. http://dx.doi.org/10.31274/icm-180809-598.

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Reports on the topic "DoS Resistance"

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Soupir, Michelle, Matt Helmers, and Thomas Moorman. Antibiotic Resistant Bacteria and Resistance Genes in Crop Fields. Ames: Iowa State University, Digital Repository, 2015. http://dx.doi.org/10.31274/farmprogressreports-180814-802.

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Soupir, Michelle L., Matthew J. Helmers, and Thomas B. Moorman. Antibiotic Resistant Bacteria and Resistance Genes in Crop Fields. Ames: Iowa State University, Digital Repository, 2013. http://dx.doi.org/10.31274/farmprogressreports-180814-640.

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Ramirez, Abelardo L., William D. Daily, and Andrew M. Binley. ELECTRICAL RESISTANCE TOMOGRAPHY. Office of Scientific and Technical Information (OSTI), June 2000. http://dx.doi.org/10.2172/15010154.

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Oettinger, Frank F., and David L. Blackburn. Thermal resistance measurements. Gaithersburg, MD: National Institute of Standards and Technology, 1990. http://dx.doi.org/10.6028/nist.sp.400-86.

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Dhuley, R. C. Thermal contact resistance. Office of Scientific and Technical Information (OSTI), July 2019. http://dx.doi.org/10.2172/1556950.

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Al-Merjan, Sami M. The Kuwaiti Resistance. Fort Belvoir, VA: Defense Technical Information Center, April 1996. http://dx.doi.org/10.21236/ada309263.

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Albers, John, and Harry L. Berkowitz. A collection of computer programs for two-probe resistance (spreading resistance) and four-probe resistance calculations, RESPAC. Gaithersburg, MD: National Institute of Standards and Technology, 1993. http://dx.doi.org/10.6028/nist.sp.400-91.

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Friedlander, Terence W. Investigating Genomic Mechanisms of Treatment Resistance in Castration Resistant Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, May 2014. http://dx.doi.org/10.21236/ada606110.

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Friedlander, Terence W. Investigating Genomic Mechanisms of Treatment Resistance in Castration Resistant Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, May 2013. http://dx.doi.org/10.21236/ada581161.

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Friedlander, Terence W. Investigating Genomic Mechanisms of Treatment Resistance in Castration Resistant Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, May 2015. http://dx.doi.org/10.21236/ada622720.

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