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Books on the topic 'Dopaminergic neurons'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Umberto, Di Porzio, Pernas-Alonso Roberto, and Perrone-Capano Carla, eds. Development of dopaminergic neurons. R.G. Landes Co., 1999.

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2

Beart, P. M., G. N. Woodruff, and D. M. Jackson, eds. Pharmacology and Functional Regulation of Dopaminergic Neurons. Palgrave Macmillan UK, 1988. http://dx.doi.org/10.1007/978-1-349-10047-7.

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3

Giovanni, Giuseppe. Birth, Life and Death of Dopaminergic Neurons in the Substantia Nigra. Springer-Verlag Vienna, 2009.

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4

Giovanni, Giuseppe, Vincenzo Di Matteo, and Ennio Esposito, eds. Birth, Life and Death of Dopaminergic Neurons in the Substantia Nigra. Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-92660-4.

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5

Kjell, Fuxe, and Wenner-Grenska samfundet, eds. Trophic regulation of the basal ganglia: Focus on dopamine neurons. Pergamon, 1994.

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6

M, Beart P., Woodruff G. N, Jackson D. M, and International Congress of Pharmacology, (10th : 1987 : Sydney, N.S.W.), eds. Pharmacology and functional regulation of dopaminergic neurons: Proceedings of a satellite symposium of the IUPHAR 10th International Congress of pharmacology, 31 Aug.- 2 Sep. 1987. Macmillan, 1988.

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7

M, Beart P., Woodruff Geoffrey N, Jackson D. M, International Union of Pharmacology, and International Congress of Pharmacology (10th : 1987 : Cessnock, N.S.W.), eds. Pharmacology and functional regulation of dopaminergic neurons: Proceedings of a satellite symposium of the IUPHAR 10th International Congress of Pharmacology, 31 August-2 September 1987. Macmillan Press, Scientific & Medical, 1988.

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8

M, Beart P., Woodruff Geoffrey N, Jackson D. M, International Union of Pharmacology, and International Congress of Pharmacology (10th : 1987 : Cessnock, N.S.W.).)., eds. Pharmacology and functional regulation of dopaminergic neurons: Proceedings of a satellite symposium of the IUPHAR 10th International Congress of Pharmacology, 31 August-2 September 1987. PSG Pub. Co., 1988.

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9

W, Stone T., ed. CNS neurotransmitters and neuromodulators: Glutamate. CRC Press, 1995.

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10

Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson’s Disease. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0699-7.

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11

Dong, Jing-fei. Morphological and biochemical characterization of human second trimester foetal dopaminergic neurones and identification of factors influencing their survival and preservation in vitro: A study related to clinical neural transplantation for Parkinson's disease. University of Wolverhampton, 1993.

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12

Beart, P. Dopaminergic Neurons. Palgrave Macmillan, 1988.

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13

(Editor), D. M. Jackson, ed. Pharmacology and Functional Regulation of Dopaminergic Neurons. Mosby-Year Book, 1989.

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14

Esposito, Ennio, Vincenzo Di Matteo, and Giuseppe di Giovanni. Birth, Life and Death of Dopaminergic Neurons in the Substantia Nigra. Springer, 2012.

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15

Esposito, Ennio, Vincenzo Di Matteo, and Giuseppe di Giovanni. Birth, Life and Death of Dopaminergic Neurons in the Substantia Nigra. Springer, 2011.

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16

Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson's Disease. Springer London, Limited, 2012.

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17

Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson's Disease. Springer, 2012.

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18

Beninger, Richard J. Neuroanatomy and dopamine systems. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0011.

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Neuroanatomy and dopamine systems explains how sensory signals ascend the central nervous system via a series of nuclei; axons detecting specific elements converge onto higher-order neurons that respond to particular stimulus features. Assemblies of feature-detection cells in the cerebral cortex detect complex stimuli such as faces. These cell assemblies project to motor nuclei of the dorsal and ventral striatum where they terminate on dendritic spines of efferent medium spiny neurons. Dopaminergic projections from ventral mesencephalic nuclei terminate on the same spines. Individual corticost

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19

Beninger, Richard J. Dopamine and the elements of incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0003.

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Dopamine and the elements of incentive learning explains how, in lever pressing for food tasks, incentive learning produces a gradient of attractiveness of environment stimuli: during magazine training, food activates dopaminergic neurons and the click and food cup become conditioned incentive stimuli, acquiring the ability to elicit approach and other responses; during lever-press training, the click activates dopaminergic neurons and the lever and lever-related stimuli become conditioned incentive stimuli. In conditioned place preference, amphetamine enhances dopaminergic neurotransmission a

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20

Beninger, Richard J. Schizophrenia, Parkinson’s disease, and attention deficit hyperactivity disorder. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0009.

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Schizophrenia, Parkinson’s disease, and attention deficit hyperactivity disorder (ADHD) discusses how hyperactive dopaminergic neurotransmission appears to underlie schizophrenia’s positive symptoms, loss of dopaminergic neurons in adulthood leads to Parkinson’s disease, and dopamine neuron hypofunction in childhood and adolescence may underlie ADHD. Positive schizophrenia symptoms may arise from excessive incentive learning that is gradually lost with antipsychotic treatment. Declarative learning and memory may contribute to delusions based on excessive incentive learning. Loss of responsiven

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21

Chiara, Gaetano Di. Dopamine in the CNS I. Springer London, Limited, 2012.

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22

Chiara, Gaetano Di. Dopamine in the CNS I. Springer Berlin / Heidelberg, 2012.

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23

Dopamine in the CNS I. Springer, 2002.

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24

Dopamine in the CNS I. Springer, 2011.

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25

Triarhou, Lazaros C. Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson's Disease (Advances in Experimental Medicine and Biology, 517). Kluwer Academic/Plenum Publishers, 2002.

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26

Rosemary Eleonor Anne.* Craig. Dopamine metabolism in pregnancy: impact of amino acid supplementation on regional dopaminergic neurons in dams and fetuses. 1989.

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27

Beninger, Richard J. Drug abuse and incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0010.

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Drug abuse and incentive learning explains how abused drugs, including nicotine, ethanol, marijuana, amphetamine, cocaine, morphine, and heroin, produce conditioned place preference and are self-administered; dopamine receptor antagonists block these effects. Stimuli that become reliable predictors of drug reward produce burst firing in dopaminergic neurons, but the drug retains its ability to activate dopaminergic neurons. Thus, repeated drug users experience two activations of dopaminergic neurotransmission, one upon exposure to the conditioned stimuli signaling the drug and another upon tak

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28

Walsh, Richard A. Smoothing out the Ups and Downs. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0001.

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The progressive loss of nigrostriatal dopaminergic neurons with advancing disease exposes the trough between every dose of levodopa in Parkinson’s disease. This is due to the combination of a loss of native dopamine production to fill in this interdose interval and a reduction in dopaminergic terminals to take up and release dopamine long beyond its short plasma half-life. The clinical result is wearing off—an awareness in patients of returning symptomatology while waiting for their next dose. Where consistent and impacting negatively on function on any level, there are a number of initial ora

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29

Beninger, Richard J. Life's rewards. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.001.0001.

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Life’s Rewards: Linking Dopamine, Incentive Learning, Schizophrenia, and the Mind explains how increased brain dopamine produces reward-related incentive learning, the acquisition by neutral stimuli of increased ability to elicit approach and other responses. Dopamine decreases may produce inverse incentive learning, the loss by stimuli of the ability to elicit approach and other responses. Incentive learning is gradually lost when dopamine receptors are blocked. The brain has multiple memory systems defined as “declarative” and “non-declarative;” incentive learning produces one form of non-de

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30

Beninger, Richard J. Dopamine as the dependent variable. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0005.

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Dopamine as the dependent variable discusses how postmortem biochemistry, intracerebral microdialysis, electrophysiological recording, in vivo electrochemistry, and positron emission tomography studies provide compelling evidence that dopaminergic neurons are activated by primary rewarding stimuli including food and water and by numerous conditioned incentives, including money. Early in training, primary rewarding stimuli activate dopaminergic neurons. When a cue is reliably paired with a primary rewarding stimulus over trials, the dopamine response begins to be seen upon presentation of the c

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31

Beninger, Richard J. Introduction. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0001.

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The Introduction provides a brief overview of the book. The central theme is dopamine-mediated reward-related incentive learning—the acquisition by neutral stimuli of an increased ability to elicit approach and other responses. The brain has multiple memory systems defined as “declarative” and “non-declarative”; incentive learning produces one form of non-declarative memory. Once incentive learning is established it is gradually lost when the rewarding stimulus is no longer available or when dopamine function is reduced. Decreases in dopaminergic neurotransmission may produce inverse incentive

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32

Walsh, Richard A. “I Am Not Sure If I Should Do DaT”. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190607555.003.0008.

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Nuclear medicine-based imaging techniques can provide an estimation of nigrostriatal tract denervation based on radionucleotide uptake in the distal presynaptic terminals of dopaminergic neurons. Although unhelpful in differentiating between differing etiologies of denervation in varied neurodegenerative disorders associated with parkinsonism, this imaging is justified in situations in which parkinsonism is believed to be drug-induced or functional or in cases in which subclinical parkinsonism is suspected. The most common clinical situation in which dopamine transporter imaging is helpful is

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33

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Acetylcholine. Taylor & Francis Group, 2020.

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34

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Acetylcholine. Taylor & Francis Group, 2020.

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35

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Neuroactive Steroids (CNS Neurotransmitters & Neuromodulators). CRC, 1996.

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36

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Dopamine. CRC-Press, 1996.

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37

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Acetylcholine. Taylor & Francis Group, 2020.

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38

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Acetylcholine. CRC, 1994.

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39

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Glutamate. CRC, 1995.

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40

Stone, Trevor W. CNS Neurotransmitters and Neuromodulators: Acetylcholine. Taylor & Francis Group, 2020.

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41

Dopaminergic Neuron Transplantation in the Weaver Mouse Model of Parkinson's Disease. Springer, 2003.

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42

Mayer, Adelheid. Transmitterfreisetzung aus zerebralen noradrenergen, dopaminergen und cholinergen Neuronen bei Einzelpulsen und kurzen Pulsfolgen: Modulation durch Autorezeptoren und pharmakologische Autorezeptor-Charakterisierung. 1989.

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