Relevant bibliographies by topics / DNA replication

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'DNA replication'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "DNA replication"

1

Avemann, K., R. Knippers, T. Koller, and J. M. Sogo. "Camptothecin, a specific inhibitor of type I DNA topoisomerase, induces DNA breakage at replication forks." Molecular and Cellular Biology 8, no. 8 (1988): 3026–34. http://dx.doi.org/10.1128/mcb.8.8.3026-3034.1988.

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The structure of replicating simian virus 40 minichromosomes, extracted from camptothecin-treated infected cells, was investigated by biochemical and electron microscopic methods. We found that camptothecin frequently induced breaks at replication forks close to the replicative growth points. Replication branches were disrupted at about equal frequencies at the leading and the lagging strand sides of the fork. Since camptothecin is known to be a specific inhibitor of type I DNA topoisomerase, we suggest that this enzyme is acting very near the replication forks. This conclusion was supported b
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Avemann, K., R. Knippers, T. Koller, and J. M. Sogo. "Camptothecin, a specific inhibitor of type I DNA topoisomerase, induces DNA breakage at replication forks." Molecular and Cellular Biology 8, no. 8 (1988): 3026–34. http://dx.doi.org/10.1128/mcb.8.8.3026.

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The structure of replicating simian virus 40 minichromosomes, extracted from camptothecin-treated infected cells, was investigated by biochemical and electron microscopic methods. We found that camptothecin frequently induced breaks at replication forks close to the replicative growth points. Replication branches were disrupted at about equal frequencies at the leading and the lagging strand sides of the fork. Since camptothecin is known to be a specific inhibitor of type I DNA topoisomerase, we suggest that this enzyme is acting very near the replication forks. This conclusion was supported b
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Swindle, C. Scott, Nianxiang Zou, Brian A. Van Tine, George M. Shaw, Jeffrey A. Engler, and Louise T. Chow. "Human Papillomavirus DNA Replication Compartments in a Transient DNA Replication System." Journal of Virology 73, no. 2 (1999): 1001–9. http://dx.doi.org/10.1128/jvi.73.2.1001-1009.1999.

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ABSTRACT Many DNA viruses replicate their genomes at nuclear foci in infected cells. Using indirect immunofluorescence in combination with fluorescence in situ hybridization, we colocalized the human papillomavirus (HPV) replicating proteins E1 and E2 and the replicating origin-containing plasmid to nuclear foci in transiently transfected cells. The host replication protein A (RP-A) was also colocalized to these foci. These nuclear structures were identified as active sites of viral DNA synthesis by bromodeoxyuridine (BrdU) pulse-labeling. Unexpectedly, the great majority of RP-A and BrdU inco
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Liu, Guoqi, Michelle Malott, and Michael Leffak. "Multiple Functional Elements Comprise a Mammalian Chromosomal Replicator." Molecular and Cellular Biology 23, no. 5 (2003): 1832–42. http://dx.doi.org/10.1128/mcb.23.5.1832-1842.2003.

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ABSTRACT The structure of replication origins in metazoans is only nominally similar to that in model organisms, such as Saccharomyces cerevisiae. By contrast to the compact origins of budding yeast, in metazoans multiple elements act as replication start sites or control replication efficiency. We first reported that replication forks diverge from an origin 5′ to the human c-myc gene and that a 2.4-kb core fragment of the origin displays autonomous replicating sequence activity in plasmids and replicator activity at an ectopic chromosomal site. Here we have used clonal HeLa cell lines contain
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Laskey, Ronald. "The Croonian Lecture 2001 Hunting the antisocial cancer cell: MCM proteins and their exploitation." Philosophical Transactions of the Royal Society B: Biological Sciences 360, no. 1458 (2005): 1119–32. http://dx.doi.org/10.1098/rstb.2005.1656.

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Replicating large eukaryotic genomes presents the challenge of distinguishing replicated regions of DNA from unreplicated DNA. A heterohexamer of minichromosome maintenance (MCM) proteins is essential for the initiation of DNA replication. MCM proteins are loaded on to unreplicated DNA before replication begins and displaced progressively during replication. Thus, bound MCM proteins license DNA for one, and only one, round of replication and this licence is reissued each time a cell divides. MCM proteins are also the best candidates for the replicative helicases that unwind DNA during replicat
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Moiseeva, Tatiana N., Yandong Yin, Michael J. Calderon, et al. "An ATR and CHK1 kinase signaling mechanism that limits origin firing during unperturbed DNA replication." Proceedings of the National Academy of Sciences 116, no. 27 (2019): 13374–83. http://dx.doi.org/10.1073/pnas.1903418116.

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DNA damage-induced signaling by ATR and CHK1 inhibits DNA replication, stabilizes stalled and collapsed replication forks, and mediates the repair of multiple classes of DNA lesions. We and others have shown that ATR kinase inhibitors, three of which are currently undergoing clinical trials, induce excessive origin firing during unperturbed DNA replication, indicating that ATR kinase activity limits replication initiation in the absence of damage. However, the origins impacted and the underlying mechanism(s) have not been described. Here, we show that unperturbed DNA replication is associated
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Takebayashi, Shin-ichiro, Tyrone Ryba, Kelsey Wimbish, et al. "The Temporal Order of DNA Replication Shaped by Mammalian DNA Methyltransferases." Cells 10, no. 2 (2021): 266. http://dx.doi.org/10.3390/cells10020266.

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Multiple epigenetic pathways underlie the temporal order of DNA replication (replication timing) in the contexts of development and disease. DNA methylation by DNA methyltransferases (Dnmts) and downstream chromatin reorganization and transcriptional changes are thought to impact DNA replication, yet this remains to be comprehensively tested. Using cell-based and genome-wide approaches to measure replication timing, we identified a number of genomic regions undergoing subtle but reproducible replication timing changes in various Dnmt-mutant mouse embryonic stem (ES) cell lines that included a
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Thomas, David C., John D. Roberts, Ralph D. Sabatino, et al. "Fidelity of mammalian DNA replication and replicative DNA polymerases." Biochemistry 30, no. 51 (1991): 11751–59. http://dx.doi.org/10.1021/bi00115a003.

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Greci, Mark D., and Stephen D. Bell. "Archaeal DNA Replication." Annual Review of Microbiology 74, no. 1 (2020): 65–80. http://dx.doi.org/10.1146/annurev-micro-020518-115443.

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It is now well recognized that the information processing machineries of archaea are far more closely related to those of eukaryotes than to those of their prokaryotic cousins, the bacteria. Extensive studies have been performed on the structure and function of the archaeal DNA replication origins, the proteins that define them, and the macromolecular assemblies that drive DNA unwinding and nascent strand synthesis. The results from various archaeal organisms across the archaeal domain of life show surprising levels of diversity at many levels—ranging from cell cycle organization to chromosome
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Hernández-Tamayo, Rogelio, Luis M. Oviedo-Bocanegra, Georg Fritz, and Peter L. Graumann. "Symmetric activity of DNA polymerases at and recruitment of exonuclease ExoR and of PolA to the Bacillus subtilis replication forks." Nucleic Acids Research 47, no. 16 (2019): 8521–36. http://dx.doi.org/10.1093/nar/gkz554.

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AbstractDNA replication forks are intrinsically asymmetric and may arrest during the cell cycle upon encountering modifications in the DNA. We have studied real time dynamics of three DNA polymerases and an exonuclease at a single molecule level in the bacterium Bacillus subtilis. PolC and DnaE work in a symmetric manner and show similar dwell times. After addition of DNA damage, their static fractions and dwell times decreased, in agreement with increased re-establishment of replication forks. Only a minor fraction of replication forks showed a loss of active polymerases, indicating relativel
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Dissertations / Theses on the topic "DNA replication"

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Carrington, James T. "Post-replicative resolution of under-replication." Thesis, University of Dundee, 2017. https://discovery.dundee.ac.uk/en/studentTheses/f0a89d2a-6ee2-4175-ba65-d58aaaee4e24.

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The evolutionary pressure to prevent re-replication by inactivating licensing during S phase leaves higher-eukaryotes with large genomes, such as human cells, vulnerable to replication stresses. Origins licensed in G1 must be sufficient to complete replication as new origins cannot be licensed in response to irreversible replication fork stalling. Interdisciplinary approaches between cellular biology and biophysics predict that replication of the genome is routinely incomplete in G2, even in the absence of external stressors. The frequency of converging replication forks that never terminate d
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Rytkönen, A. (Anna). "The role of human replicative DNA polymerases in DNA repair and replication." Doctoral thesis, University of Oulu, 2006. http://urn.fi/urn:isbn:9514281381.

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Abstract The maintenance of integrity of the genome is essential for a cell. DNA repair and faithful DNA replication ensure the stability of the genome. DNA polymerases (pols) are the enzymes that synthesise DNA, a process important both in DNA replication and repair. In DNA replication DNA polymerases duplicate the genome during S phase prior to cell division. Pols α, δ, and ε are implicated in chromosomal DNA replication, but their exact function in replication is not yet completely clear. The mechanisms of different repair pathways and proteins involved are not yet completely characterised
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Chen, Shuhua. "Multiple mechanisms regulate the human replication factors : replication protein A and DNA polymerase alpha-during DNA replication and DNA repair /." [S.l. : s.n.], 2003.

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Anderson, Mary E. Ph D. (Mary Elizabeth)Massachusetts Institute of Technology. "Regulation of DNA replication and the replication initiator, DnaA, in Bacillus subtilis." Thesis, Massachusetts Institute of Technology, 2019. https://hdl.handle.net/1721.1/121876.

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Thesis: Ph. D., Massachusetts Institute of Technology, Department of Biology, 2019<br>Cataloged from PDF version of thesis. "February 2019."<br>Includes bibliographical references (pages 118-128).<br>DNA replication is a highly regulated process across all organisms. Improper regulation of DNA replication can be detrimental. I identified an overinitiating, conditional synthetic lethal mutant of Bacillus subtilis. I isolated suppressors of this mutant and uncovered novel genes associated with DNA replication. These suppressors acted both at the steps of initiation and elongation to overcome the
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Tavares, de Araujo Felipe. "DNA replication and methylation." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=37847.

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One of the main questions of modern biology is how our cells interpret our genetic and epigenetic information. DNA methylation is a covalent modification of the genome that is essential for mammalian development and plays an important role in the control of gene expression, genomic imprinting and X-chromosome inactivation (Bird and Wolffe, 1999; Szyf et al., 2000). Furthermore, changes in DNA methylation and DNA methyltransferase 1 (DNMT1) activity have been widely documented in a number of human cancers (Szyf, 1998a; Szyf et al., 2000).<br>In Escherichia coli, timing and frequency of initiati
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Upton, Amy Louise. "Replication of damaged DNA." Thesis, University of Nottingham, 2009. http://eprints.nottingham.ac.uk/11332/.

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DNA is under constant attack from numerous damaging agents and our cells deal with thousands of lesions every day. With such constant damage it is inevitable that the template will not be completely cleared of lesions before the replication complex arrives. The consequences of the replisome meeting an obstacle will depend upon the nature of the obstacle. I have focussed upon replication in Escherichia coli and the effect of UV-induced lesions, which would block synthesis by the replicative polymerases. It is accepted that a UV lesion in the lagging strand template can be bypassed by the replis
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Borazjani, Gholami Farimah. "Role of replicative primase in lesion bypass during DNA replication." Thesis, University of Sussex, 2017. http://sro.sussex.ac.uk/id/eprint/68762/.

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Maintenance of genome integrity and stability is fundamental for any form of life. This is complicated as DNA is highly reactive and always under attack from a wide range of endogenous and exogenous sources which can lead to different damages in the DNA. To preserve the integrity of DNA replication, cells hav evolved a variety of DNA repair pathways. DNA damage tolerance mechanisms serve as the last line of defence to rescue the stalled replications forks. TLS, error-prone type of DNA damage tolerance, acts to bypass DNA lesions and allows continuation of DNA replication. Surprisingly majority
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Pearson, Christopher Edmund. "DNA cruciforms and mammalian origins of DNA replication." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=28503.

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The objective of the research in this thesis is to investigate, at the molecular level, the sequences and/or structures involved in the initiation of mammalian DNA replication and to investigate the protein interactions with DNA cruciforms which have been implicated in the process of replication initiation. Four plasmids containing monkey (CV-1) early replicating nascent origin enriched sequences (ors), which had been shown previously to replicate autonomously in transfected CV-1, COS-7 and HeLa cells, were used in the establishment of an in vitro DNA replication system that uses HeLa cell ext
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Schorr, Stephanie. "Replication of Bulky DNA Adducts." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-125267.

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Bennett, Ellen R. (Ellen Ruth). "Activation of papovavirus DNA replication." Thesis, McGill University, 1991. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=70232.

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To define the viral target sites of cellular permissive factors, simian virus 40-polyomavirus hybrid origins for DNA replication were formed by joining the auxiliary domain of one viral origin to the origin core of the other, and vice versa. The replicative capacity of these constructs were assessed in a number of mouse and monkey cell lines which express the large T antigen of polyomavirus or SV40. The results of this analysis showed that the auxiliary domains of the viral replication origins could substitute for one another in DNA replication, provided that the viral origin core and its cogn
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Books on the topic "DNA replication"

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Adams, R. L. P. DNA replication. IRL Press, 1991.

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Halazonetis, Thanos D. DNA Replication & DNA Replication Stress. American Chemical Society, 2022. http://dx.doi.org/10.1021/acsinfocus.7e5022.

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A, Baker Tania, ed. DNA replication. 2nd ed. W.H. Freeman, 1992.

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L, Campbell Judith, ed. DNA replication. Academic, 1995.

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Vengrova, Sonya, and Jacob Dalgaard, eds. DNA Replication. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2596-4.

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Vengrova, Sonya, and Jacob Z. Dalgaard, eds. DNA Replication. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-815-7.

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Masai, Hisao, and Marco Foiani, eds. DNA Replication. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-6955-0.

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1941-, Kelly Thomas J., and Stillman Bruce, eds. Eukaryotic DNA replication. Cold Spring Harbor Laboratory, 1988.

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Julian, Blow J., ed. Eukaryotic DNA replication. IRL Press, 1996.

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Alan, Cann, ed. DNA virus replication. Oxford University Press, 2000.

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Book chapters on the topic "DNA replication"

1

Zhang, Huidong. "DNA Replication." In DNA Replication - Damage from Environmental Carcinogens. Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-7212-9_1.

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Kaguni, Jon M. "DNA Replication." In Molecular Life Sciences. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_53-2.

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Lygerou, Zoi, K. K. Koutroumpas, and John Lygeros. "DNA Replication." In Encyclopedia of Systems Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_40.

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Gooch, Jan W. "DNA Replication." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13591.

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Kaguni, Jon M. "DNA Replication." In Molecular Life Sciences. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_53.

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McHenry, Charles S. "DNA Replication." In Emerging Targets in Antibacterial and Antifungal Chemotherapy. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3274-3_3.

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Brown, Terry. "DNA Replication." In Introduction to Genetics, 2nd ed. Garland Science, 2025. https://doi.org/10.1201/9781003473862-11.

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Liu, H., J. H. Naismith, and R. T. Hay. "Adenovirus DNA Replication." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-662-05597-7_5.

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Adams, Roger L. P., John T. Knowler, and David P. Leader. "Replication of DNA." In The Biochemistry of the Nucleic Acids. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2290-0_6.

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Keshav, Kylie F., and Shonen Yoshida. "Mitochondrial DNA Replication." In Mitochondrial DNA Mutations in Aging, Disease and Cancer. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-12509-0_5.

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Conference papers on the topic "DNA replication"

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Temple, Mark D. "DNA Sonification Using 8-Channel Audio for Data Analyses and Music Composition." In ICAD 2024: The 29th International Conference on Auditory Display. International Community for Auditory Display, 2024. http://dx.doi.org/10.21785/icad2024.016.

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DNA sequences contain vast amounts of biological data and computer algorithms play an important role in processing these data for human inspection. Here we describe an updated computer-generated auditory display tool to be used as stand-alone audio or as a complement to a visual display for DNA sequence inspection. The auditory display uses musical notes to represent the data in relation to the process of gene expression or DNA replication. Given the use of musical notes in the auditory display raises the possibility these may be considered algorithmic music. To pursue this notion further the
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Brieba, Luis G., Mauricio Carbajal, Luis Manuel Montaño, Oscar Rosas-Ortiz, Sergio A. Tomas Velazquez, and Omar Miranda. "Conformational Dynamics in DNA Replication Selectivity." In Advanced Summer School in Physics 2007. AIP, 2007. http://dx.doi.org/10.1063/1.2825116.

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Manturov, Alexey O., and Anton V. Grigoryev. "Synchronization of DNA array replication kinetics." In Saratov Fall Meeting 2015, edited by Elina A. Genina, Valery V. Tuchin, Vladimir L. Derbov, et al. SPIE, 2016. http://dx.doi.org/10.1117/12.2229623.

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Mindek, Peter, Tobias Klein, and Alfredo De Biasio. "DNA replication of the lagging strand." In SIGGRAPH '23 Electronic Theater: Special Interest Group on Computer Graphics and Interactive Techniques Conference: Electronic Theater. ACM, 2023. http://dx.doi.org/10.1145/3577024.3588981.

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Obeid, Samra, Nina Blatter, and Andreas Marx. "Lost in replication: DNA polymerases encountering non-instructive DNA lesions." In XVth Symposium on Chemistry of Nucleic Acid Components. Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2011. http://dx.doi.org/10.1135/css201112027.

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Scholl, Bruno B., Ligia C. Palma, Victor S. Hariki, Maria Carolina Elias, and Marcelo S. Reis. "Tuning a predictive DNA replication programming computational model for Trypanosomatids." In Simpósio Brasileiro de Bioinformática. Sociedade Brasileira de Computação, 2024. https://doi.org/10.5753/bsb.2024.245608.

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In this paper, we report the tuning of a predictive DNA replication programming computational model for both Trypanosoma brucei and Trypanosoma cruzi, unicellular protozoan endoparasites that cause African sleeping sickness and Chagas disease, respectively. This is a stochastic dynamic model for simulating the DNA replication process with concomitant constitutive transcription, enabling the analysis of the interactions between replication and transcription in these organisms. Using Optuna, an open-source hyperparameter optimizer, we explored almost 5,000 parameter combinations across both trai
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Koutroumpas, K., Z. Lygerou, and J. Lygeros. "Parameter Identification for a DNA replication model." In 2008 8th IEEE International Conference on Bioinformatics and BioEngineering (BIBE). IEEE, 2008. http://dx.doi.org/10.1109/bibe.2008.4696726.

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Brückner, L., R. Xu, A. Herrmann, and A. G. Henssen. "Replication stress associated micronucleation of extrachromosomal DNA." In 35. Jahrestagung der Kind-Philipp-Stiftung für pädiatrisch onkologische Forschung. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1786559.

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LI, JUNTAO, MAJID ESHAGHI, JIANHUA LIU, and RADHA KRISHNA MURTHY KARUTURI. "NEAR-SIGMOID MODELING TO SIMULTANEOUSLY PROFILE GENOME-WIDE DNA REPLICATION TIMING AND EFFICIENCY IN SINGLE DNA REPLICATION MICROARRAY STUDIES." In The 6th Asia-Pacific Bioinformatics Conference. PUBLISHED BY IMPERIAL COLLEGE PRESS AND DISTRIBUTED BY WORLD SCIENTIFIC PUBLISHING CO., 2007. http://dx.doi.org/10.1142/9781848161092_0039.

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Lee, C. H., H. Teng, and J. S. Chen. "Atomistic to Continuum Modeling of DNA Molecules." In ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13157.

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The mechanical properties of DNA has very important biological implication. For example, the bending and twisting rigidities of DNA affect how it wraps around histones to form chromosomes, bends upon interactions with proteins, supercoils during replication process, and packs into the confined space within a virus. Many biologically important processes involving DNA are accompanied by the deformations of double helical structure of DNA.
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Reports on the topic "DNA replication"

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Meyer, Richard. DNA Replication During Conjugal Transfer of R1162. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada415172.

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Heimer, Brandon W., Kevin K. Crown, and George David Bachand. Assembling semiconductor nanocomposites using DNA replication technologies. Office of Scientific and Technical Information (OSTI), 2005. http://dx.doi.org/10.2172/875607.

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Green, Brian M. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-Replication. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada436928.

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Green, Brian M., and Joachim J. Li. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-replication. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada467931.

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Green, Brian. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-replication. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada482750.

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Crown, Kevin K., and George David Bachand. Patterning quantum dot arrays using DNA replication principles. Office of Scientific and Technical Information (OSTI), 2004. http://dx.doi.org/10.2172/919201.

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Lieberman, Paul. Function of BRCA1 at a DNA Replication Origin. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada429715.

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Chuang, Chen-Hua. Role of DNA Replication Defects in Breast Cancer. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada541310.

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Chuang, Chen-Hua. Role of DNA Replication Defects in Breast Cancer. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada523952.

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Kuo, Shue-Ru, and Thomas Melendy. DNA Replication Arrest and DNA Damage Responses Induced by Alkylating Minor Groove Binders. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada395141.

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