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Journal articles on the topic 'Disorder'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 June 2024

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1

Aleksandrowicz, Jerzy. "Neurotic “disorders” or “disorder”?" Psychiatria Polska 53, no. 2 (April 30, 2019): 293–312. http://dx.doi.org/10.12740/pp/onlinefirst/97374.

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2

Gordon, Robert P., Emma K. Brandish, and David S. Baldwin. "Anxiety disorders, post-traumatic stress disorder, and obsessive–compulsive disorder." Medicine 44, no. 11 (November 2016): 664–71. http://dx.doi.org/10.1016/j.mpmed.2016.08.010.

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3

Impey, Bethan, Robert P. Gordon, and David S. Baldwin. "Anxiety disorders, post-traumatic stress disorder, and obsessive–compulsive disorder." Medicine 48, no. 11 (November 2020): 717–23. http://dx.doi.org/10.1016/j.mpmed.2020.08.005.

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4

Palomo, José Luis, Francisco Arias, Néstor Szerman, Pablo Vega, Ignacio Basurte, and Beatriz Mesías. "Dual disorders in individuals under treatment for both alcohol and cocaine: Madrid study on the prevalence of dual disorders." Salud mental 40, no. 6 (December 12, 2017): 257–64. http://dx.doi.org/10.17711/sm.0185-3325.2017.033.

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Introduction. Descriptive data about co-occurrence of alcohol and cocaine consumption is scarce, despite its important prevalence. Dual disordes shows high prevalence in clinical samples, and patients report worse evolution and need more health services. Objective. To compare psychopathology in patients in treatment with lifetime alcohol and cocaine (Alc + Coc) substance use disorder (SUD) with subjects with alcohol but not cocaine (Alc) lifetime SUD and cocaine but not alcohol (Coc) SUD. Method. The sample consisted of 837 outpatients from Madrid, Spain, under treatment in substance misuse or mental health units. Two analyses were made: we compared subjects in the Alc + Coc (n = 366) to the Alc group (n = 162), and then to the Coc group (n = 122). Socio-demographic variables were addressed by interview. The Mini International Neuropsychiatric Interview (MINI) was used to evaluate Axis I disorders and the Personality Disorder Questionnaire (PDQ) to evaluate Personality Disorders (PD). Results. Compared to Alc group, patients in the Alc + Coc group were younger, had different socio-demographic characteristics, had more proportion of cannabis and opioid SUD, had less proportion of major depressive disorder, obsessive and depressive PD, more proportion of antisocial PD and lower suicide risk. Compared to the Coc group, they had more cannabis SUD and lower opioid SUD, showed higher prevalence of bipolar disorder, general anxiety disorder, paranoid, histrionic and dependent PD. Discussion and conclusion. We present a cross-sectional study describing comorbidity of dual disordes on treatment-seeking concurrent alcohol and cocaine problematic users. This concurrence showed different dual disordes prevalence profile than single users in some specific mental disorders.
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Casalini, F., N. Mosti, S. Belletti, V. Mastria, S. Rizzato, A. Del Carlo, M. Fornaro, L. Dell’Osso, and G. Perugi. "Bipolar disorder and disreactive disorders." International Clinical Psychopharmacology 28 (December 2012): e34. http://dx.doi.org/10.1097/01.yic.0000423296.62412.57.

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Karamuctafalioĝlu, K. O., and N. Karamuctafalioğlu. "DYSTHYMIC DISORDER AND PERSONALITY DISORDERS." Clinical Neuropharmacology 15 (1992): 518B. http://dx.doi.org/10.1097/00002826-199202001-01010.

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7

Hollander, E. "Obsessive-compulsive disorder related disorders." International Clinical Psychopharmacology 11 (December 1996): 75–88. http://dx.doi.org/10.1097/00004850-199612005-00007.

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8

Beavers, Christine M., and Theocharis Stamatatos. "Disorder! Disorder! Disorder!" Acta Crystallographica Section A Foundations and Advances 73, a1 (May 26, 2017): a301. http://dx.doi.org/10.1107/s0108767317097057.

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9

JA, Fernandez. "Dual Disorder: Substance Use Disorder in People with Severe Mental Disorders." Journal of Addiction & Addictive Disorders 9, no. 1 (January 27, 2022): 1–6. http://dx.doi.org/10.24966/aad-7276/100079.

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History of the concept: In the early 1980s, Pepper and Ryglewicz [1] used the acronym YACP (Young Adult Chronic Patient) to define a new profile of “new chronics” as opposed to the profile of the “old institutionalised chronic”.
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10

Mueser, K. T., S. D. Rosenberg, R. E. Drake, K. M. Miles, G. Wolford, R. Vidaver, and K. Carrieri. "Conduct disorder, antisocial personality disorder and substance use disorders in schizophrenia and major affective disorders." Journal of Studies on Alcohol 60, no. 2 (March 1999): 278–84. http://dx.doi.org/10.15288/jsa.1999.60.278.

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11

Bushnell, J. A., J. E. Wells, and M. A. Oakley-Browne. "Impulsivity in Disordered Eating, Affective Disorder and Substance use Disorder." British Journal of Psychiatry 169, no. 3 (September 1996): 329–33. http://dx.doi.org/10.1192/bjp.169.3.329.

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BackgroundFailure to control impulsive behaviour has been postulated as an underlying mechanism common to substance use disorder, sociopathy and to a substantial subgroup of women with bulimia nervosa.MethodThree hundred and one women recruited to a general population study were selected either at random or because they had lifetime substance use disorder, affective disorder or symptoms of bulimia. A subsequent interview determined the existence of problems with impulsivity. Behaviour that is an integral part of a DSM–III axis 1 disorder was excluded from the impulsivity measure.ResultsSimilar rates of impulsivity were found in all three of these types of disorder, and this was little different from the rate found in the women selected randomly from the general population. However, among those with comorbid disorder there was more impulsivity, and the more comorbid disorders found, the higher the proportion with problems of impulsivity.ConclusionsBecause those in treatment facilities are more likely to have other comorbid disorders (Berkson's bias), findings derived from observations of women with bulimia who are in treatment may be compromised by selection bias and may have limited applicability to those with the disorder who are not in treatment.
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12

Krogh Andersen, Erik, Inger G. Krogh Andersen, and Gudrun Ploug-Sørensen. "Disorder in natrolites: structure determinations of three disordered natrolites and one lithium-exchanged disordered natrolite." European Journal of Mineralogy 2, no. 6 (December 19, 1990): 799–808. http://dx.doi.org/10.1127/ejm/2/6/0799.

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13

FISTIKÇI, Nurhan, Münevver NACIOĞLU, Şakire EREK, Abdülkadir TABO, Evrim ERTEN, Ayşegül Selcen GÜLER, Murat KALKAN, and Ömer SAATÇİOĞLU. "Anksiyete Bozukluklarında Afektif Mizaç Farklılıkları; Panik Bozukluğu ve Obsesif Kompulsif Bozuklukta Karşılaştırılması." Nöro Psikiyatri Arşivi 50, no. 4 (2013): 337–43. http://dx.doi.org/10.4274/npa.y6464.

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14

Elia, Josephine, David M. Stoff, and Emil F. Coccaro. "Biological correlates of impulsive disruptive behavior disorders: Attention deficit hyperactivity disorder, conduct disorder, and borderline personality disorder." New Directions for Mental Health Services 1992, no. 54 (1992): 51–57. http://dx.doi.org/10.1002/yd.23319925411.

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15

Lense, Miriam D., Eniko Ladányi, Tal-Chen Rabinowitch, Laurel Trainor, and Reyna Gordon. "Rhythm and timing as vulnerabilities in neurodevelopmental disorders." Philosophical Transactions of the Royal Society B: Biological Sciences 376, no. 1835 (August 23, 2021): 20200327. http://dx.doi.org/10.1098/rstb.2020.0327.

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Millions of children are impacted by neurodevelopmental disorders (NDDs), which unfold early in life, have varying genetic etiologies and can involve a variety of specific or generalized impairments in social, cognitive and motor functioning requiring potentially lifelong specialized supports. While specific disorders vary in their domain of primary deficit (e.g. autism spectrum disorder (social), attention-deficit/hyperactivity disorder (attention), developmental coordination disorder (motor) and developmental language disorder (language)), comorbidities between NDDs are common. Intriguingly, many NDDs are associated with difficulties in skills related to rhythm, timing and synchrony though specific profiles of rhythm/timing impairments vary across disorders. Impairments in rhythm/timing may instantiate vulnerabilities for a variety of NDDs and may contribute to both the primary symptoms of each disorder as well as the high levels of comorbidities across disorders. Drawing upon genetic, neural, behavioural and interpersonal constructs across disorders, we consider how disrupted rhythm and timing skills early in life may contribute to atypical developmental cascades that involve overlapping symptoms within the context of a disorder's primary deficits. Consideration of the developmental context, as well as common and unique aspects of the phenotypes of different NDDs, will inform experimental designs to test this hypothesis including via potential mechanistic intervention approaches. This article is part of the theme issue ‘Synchrony and rhythm interaction: from the brain to behavioural ecology’.
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16

Thomas, Chris. "Hoarding Disorder." International Journal of Practical Nursing 4, no. 3 (2016): 133–37. http://dx.doi.org/10.21088/ijpn.2347.7083.4316.4.

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17

Dave, Vibha A. "Mood Disorder." Paripex - Indian Journal Of Research 2, no. 2 (January 15, 2012): 259–61. http://dx.doi.org/10.15373/22501991/feb2013/94.

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18

Ivanov, Hristo Y., Vili K. Stoyanova, Nikolay T. Popov, and Tihomir I. Vachev. "Autism Spectrum Disorder - A Complex Genetic Disorder." Folia Medica 57, no. 1 (March 1, 2015): 19–28. http://dx.doi.org/10.1515/folmed-2015-0015.

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Abstract Autism spectrum disorder is an entity that reflects a scientific consensus that several previously separated disorders are actually a single spectrum disorder with different levels of symptom severity in two core domains - deficits in social communication and interaction, and restricted repetitive behaviors. Autism spectrum disorder is diagnosed in all racial, ethnic and socioeconomic groups and because of its increased prevalence, reported worldwide through the last years, made it one of the most discussed child psychiatric disorders. In term of aetiology as several other complex diseases, Autism spectrum disorder is considered to have a strong genetic component.
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19

Friborg, Oddgeir, Monica Martinussen, Sabine Kaiser, Karl Tore Øvergård, Egil W. Martinsen, Phöbe Schmierer, and Jan Harald Rosenvinge. "Personality Disorders in Eating Disorder Not Otherwise Specified and Binge Eating Disorder." Journal of Nervous and Mental Disease 202, no. 2 (February 2014): 119–25. http://dx.doi.org/10.1097/nmd.0000000000000080.

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20

Konarski, Jakub Z., Roger S. McIntyre, Sidney H. Kennedy, Shahryar Rafi-Tari, Joanna K. Soczynska, and Terence A. Ketter. "Volumetric neuroimaging investigations in mood disorders: bipolar disorder versus major depressive disorder." Bipolar Disorders 10, no. 1 (January 10, 2008): 1–37. http://dx.doi.org/10.1111/j.1399-5618.2008.00435.x.

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21

Vandrey, Ryan, Kimberly A. Babson, Evan S. Herrmann, and Marcel O. Bonn-Miller. "Interactions between disordered sleep, post-traumatic stress disorder, and substance use disorders." International Review of Psychiatry 26, no. 2 (April 2014): 237–47. http://dx.doi.org/10.3109/09540261.2014.901300.

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22

Millichap, J. Gordon. "Sleep Disorders in Attention Deficit Disorder." Pediatric Neurology Briefs 13, no. 9 (September 1, 1999): 72. http://dx.doi.org/10.15844/pedneurbriefs-13-9-11.

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23

Cosci, Fiammetta, Koen R. J. Schruers, Kenneth Abrams, and Eric J. L. Griez. "Alcohol Use Disorders and Panic Disorder." Journal of Clinical Psychiatry 68, no. 06 (June 15, 2007): 874–80. http://dx.doi.org/10.4088/jcp.v68n0608.

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24

CUMELLA, EDWARD J. "Obsessive-Compulsive Disorder With Eating Disorders." American Journal of Psychiatry 156, no. 6 (June 1999): 982. http://dx.doi.org/10.1176/ajp.156.6.982.

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25

Lopez-Ibor, J. J. "Obsessive compulsive disorder and other disorders." International Clinical Psychopharmacology 7 (June 1992): 25–26. http://dx.doi.org/10.1097/00004850-199206001-00006.

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26

Rijkers, Cleo, Maartje Schoorl, Daphne van Hoeken, and Hans W. Hoek. "Eating disorders and posttraumatic stress disorder." Current Opinion in Psychiatry 32, no. 6 (November 2019): 510–17. http://dx.doi.org/10.1097/yco.0000000000000545.

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27

Verri, A., Re Nappi, E. Vallero, C. Galli, G. Sances, and E. Martignoni. "Premenstrual dysphoric disorder and eating disorders." Cephalalgia 17, no. 20_suppl (December 1997): 25–28. http://dx.doi.org/10.1177/0333102497017s2008.

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Premenstrual Dysphoric Disorder (PMDD) can be differentiated from Premenstrual Syndrome (PMS) by the use of the research criteria provided by the Diagnostic and Statistical Manual (DSM) IV. Indeed, PMS corresponds to mild clinical symptoms, such as breast tenderness, bloating, headache and concomitant minor mood changes, while premenstrual magnification occurs when physical and psychological symptoms of a concurrent axis I disorder get worse during the late luteal phase. Changes in appetite and eating behavior have been documented in women suffering from PMS, with an increased food intake occurring during the luteal phase. Moreover, in women with PMS, a major effect of the phase of the menstrual cycle on appetite has been documented and a high correlation with self-ratings of mood, particularly depression, has been described only in such disturbance. The aim of the present study was to analyse the clinical similarities between PMDD and Eating Disorders (in particular Bulimia Nervosa and Binge Eating Disorder). Thus, we compared the DSM III-R comorbidity, the personality dimensions and the eating attitudes in these patients, attempting to identify any relationship between groups. Twelve PMDD women (mean age 28 years), diagnosed using DSM IV criteria and premenstrual assessor form, were compared with 10 eating disorder (ED) women (6 Bulimia Nervosa, 4 Binge Eating Disorder) (mean age 25 years) and with 10 control women matched for age. The following instruments were used: (i) clinical interview with DSM III-R criteria (SCID); (ii) a psychometric study with TPQ for the evaluation of three personality dimensions (novelty seeking, harm avoidance and reward dependence); (iii) EAT/26 for the evaluation of eating attitudes. Results show that a high comorbidity for mood and anxiety disorders in PMDD and ED is well documented. Our PMDD patients share a 16.6% of comorbidity with ED, whereas such an association is present onlv in 2.3% of the general population. In addition, as a common clue, the personality dimension, harm avoidance, linked to a serotonin mediation is significantly more frequent in PMDD and ED than in normal controls. In conclusion: from the present study it seems clear that a certain degree of similarity exists between the PMDD and ED. However, whether or not these two disorders really share common ground from a physiopathological point of view still has to be clarified by more extensive studies.
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Liebowitz, Michael R. "Anxiety Disorders and Obsessive Compulsive Disorder." Neuropsychobiology 37, no. 2 (1998): 69–71. http://dx.doi.org/10.1159/000026480.

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29

Baer, Lee, and Michael A. Jenike. "Personality Disorders in Obsessive Compulsive Disorder." Psychiatric Clinics of North America 15, no. 4 (December 1992): 803–12. http://dx.doi.org/10.1016/s0193-953x(18)30210-7.

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López-Ibor, J. J. "Obsessive-compulsive disorder and other disorders." European Neuropsychopharmacology 1, no. 3 (September 1991): 275. http://dx.doi.org/10.1016/0924-977x(91)90523-w.

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31

Pallia, R. "Sleep disorder in autism spectrum disorders." Neuropsychiatrie de l'Enfance et de l'Adolescence 60, no. 5 (July 2012): S58—S59. http://dx.doi.org/10.1016/j.neurenf.2012.05.234.

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32

Gralton, Ernest, and Julie Crocombe. "Psychopathic disorder and autistic spectrum disorders." Psychiatric Bulletin 23, no. 11 (November 1999): 692. http://dx.doi.org/10.1192/pb.23.11.692.

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Winkley, Linda. "Psychopathic disorder and autistic spectrum disorders." Psychiatric Bulletin 24, no. 4 (April 2000): 155. http://dx.doi.org/10.1192/pb.24.4.155.

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Ding, Hui, Mengyuan Ouyang, Jinyi Wang, Minyao Xie, Yanyuan Huang, Fangzheng Yuan, Yunhan Jia, Jun Wang, Na Liu, and Ning Zhang. "Obsessive-Compulsive Disorder and Metabolic Disorders." Journal of Nervous & Mental Disease 210, no. 12 (December 2022): 951–59. http://dx.doi.org/10.1097/nmd.0000000000001594.

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35

Chilcoat, Howard D., and Naomi Breslau. "Posttraumatic Stress Disorder and Drug Disorders." Archives of General Psychiatry 55, no. 10 (October 1, 1998): 913. http://dx.doi.org/10.1001/archpsyc.55.10.913.

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36

Hepp, Urs, and Gabriella Milos. "Gender identity disorder and eating disorders." International Journal of Eating Disorders 32, no. 4 (October 17, 2002): 473–78. http://dx.doi.org/10.1002/eat.10090.

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37

Marcos Altable and Jesús Romero-Imbroda. "Psychiatric comorbidities in conduct disorders and neurobiological bases." World Journal of Advanced Research and Reviews 20, no. 1 (October 30, 2023): 873–81. http://dx.doi.org/10.30574/wjarr.2023.20.1.2016.

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Conduct disorder is rarely isolated, and the international literature shows a high and very diverse comorbidity. This article presents in particular the psychiatric comorbidity of conduct disorder with other externalized disorders such as attention deficit hyperactivity disorder (ADHD), oppositional defiant disorder (ODD), and internalized disorders such as mood disorder (depressive and bipolar disorder), anxiety disorders, including post-traumatic stress disorder and substance use disorders (abuse/dependence). These disorders seem to have a neurobiological substrate which implicates brain and hormonal changes, neurotransmitters alterations and environmental influences.
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Peleg, Tamar, and Arieh Y. Shalev. "Longitudinal Studies of PTSD: Overview of Findings and Methods." CNS Spectrums 11, no. 8 (August 2006): 589–602. http://dx.doi.org/10.1017/s109285290001364x.

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ABSTRACTPosttraumatic stress disorder (PTSD) has a discernible starting point and typical course, hence the particular appropriateness of longitudinal research in this disorder. This review outlines the salient findings of longitudinal studies published between 1988 and 2004. Studies have evaluated risk factors and risk indicators of PTSD, the disorder's trajectory, comorbid disorders and the predictive role of acute stress disorder. More recent studies used advanced data analytic methods to explore the sequence of causation that leads to chronic PTSD. Advantages and limitations of longitudinal methods are discussed.
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Pavlova, B., R. H. Perlis, O. Mantere, C. M. Sellgren, E. Isometsä, P. B. Mitchell, M. Alda, and R. Uher. "Prevalence of current anxiety disorders in people with bipolar disorder during euthymia: a meta-analysis." Psychological Medicine 47, no. 6 (December 20, 2016): 1107–15. http://dx.doi.org/10.1017/s0033291716003135.

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BackgroundAnxiety disorders are highly prevalent in people with bipolar disorder, but it is not clear how many have anxiety disorders even at times when they are free of major mood episodes. We aimed to establish what proportion of euthymic individuals with bipolar disorder meet diagnostic criteria for anxiety disorders.MethodWe performed a random-effects meta-analysis of prevalence rates of current DSM-III- and DSM-IV-defined anxiety disorders (panic disorder, agoraphobia, social anxiety disorder, generalized anxiety disorder, specific phobia, obsessive–compulsive disorder, post-traumatic stress disorder, and anxiety disorder not otherwise specified) in euthymic adults with bipolar disorder in studies published by 31 December 2015.ResultsAcross 10 samples with 2120 individuals with bipolar disorder, 34.7% met diagnostic criteria for one or more anxiety disorders during euthymia [95% confidence interval (CI) 23.9–45.5%]. Direct comparison of 189 euthymic individuals with bipolar disorder and 17 109 population controls across three studies showed a 4.6-fold increase (risk ratio 4.60, 95% CI 2.37–8.92, p < 0.001) in prevalence of anxiety disorders in those with bipolar disorder.ConclusionsThese findings suggest that anxiety disorders are common in people with bipolar disorder even when their mood is adequately controlled. Euthymic people with bipolar disorder should be routinely assessed for anxiety disorders and anxiety-focused treatment should be initiated if indicated.
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Quintero-Garzola, Gabriel C. "A Revision of the Relationship between Gambling Disorder, Attention Deficit Hyperactivity Disorder and Parkinson´s Disease." Pensando Psicología 13, no. 22 (October 26, 2017): 89–107. http://dx.doi.org/10.16925/pe.v13i22.1991.

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Purpose: For the present review, publications in the field of gambling disorder that deal with its relationships with others, mainly attention deficit hyperactivity disorder (ADHD) and Parkinson’s disease (PD) were consulted.Methods: The current revision includes a total of 63 references published between 1987 and 2017. It included human stud­ies and revisions regarding the comorbidity of gambling disorder with ADHD or PD. The search terms included: gambling disorder, gambling disorder comorbidity, gambling disorder and adhd, gambling disor­der and pd, gambling disorder and impulsivity. The present review fo­cused on the link among gambling disorder and ADHD or PD, because there were a large number of publications related to these disorders. For organization purpose the current work was split into two main parts: 1) Revision of previous scientific reviews about gambling dis­order, and 2) Overview and conclusions of experimental work about gambling disorder.Conclusions: The principal conclusions of the cur­rent review are: 1) subjects with a gambling disorder have a higher in­cidence of ADHD(and also of attention deficit disorder [ADD]), 2) the presence of ADHD in subjects that suffer of gambling disorder implies more challenges for the health care system, and 3) PD treatments that increase the agonism of dopamine type of receptor are related to an elevated probability for developing a gambling problem or an impulse control disorder.
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Lochner, Christine, Modise Mogotsi, Pieter L. du Toit, Debra Kaminer, Dana J. Niehaus, and Dan J. Stein. "Quality of Life in Anxiety Disorders: A Comparison of Obsessive-Compulsive Disorder, Social Anxiety Disorder, and Panic Disorder." Psychopathology 36, no. 5 (2003): 255–62. http://dx.doi.org/10.1159/000073451.

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Bankier, B., M. Aigner, U. Demal, and M. Bach. "P01.06 Alexithymia in DSM-IV disorder: Comparative evaluation in somatoform disorders, panic disorder, obsessive-compulsive disorder, and depression." European Psychiatry 15, S2 (October 2000): 322s. http://dx.doi.org/10.1016/s0924-9338(00)94417-1.

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Lemón, Linda, Fernando Fernández-Aranda, Susana Jiménez-Murcia, and Anders Håkansson. "Eating disorder in gambling disorder: A group with increased psychopathology." Journal of Behavioral Addictions 10, no. 3 (October 5, 2021): 540–45. http://dx.doi.org/10.1556/2006.2021.00060.

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Abstract Background and aims Theoretical background and previous data provide some similarities between problematic gambling and eating behaviors, and a theoretically increased clinical severity in individuals suffering from both conditions. However, large datasets are lacking, and therefore, the present study aimed to study, in a nationwide register material, psychiatric comorbidity, age and gender in gambling disorder (GD) patients with or without eating disorder (ED). Methods Diagnostic data from a nationwide register were used, including all individuals with a GD diagnosis in specialized health care in Sweden, in the years 2005–2016 (N = 2,099). Patients with GD and an ED diagnosis (n = 57) were compared to GD patients without ED. Results Patients with GD+ED were significantly more likely than other GD patients to also have a diagnosis of drug use disorder, depressive disorders, bipolar disorders, other mood disorder, anxiety disorders, personality disorders, and neuropsychiatric disorders, when controlling for gender. In logistic regression, a comorbid ED in GD was associated with female gender, younger age, depressive disorder and personality disorders. Discussion and conclusion In nationwide register data, despite the low number of GD+ED patients, GD patients with ED appear to have a more severe psychiatric comorbidity than GD patients without ED. The combined GD+ED conditions may require particular screening and clinical attention, as well as further research in larger and longitudinal studies.
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Park, Emma C., Glenn Waller, and Kenneth Gannon. "Early Improvement in Eating Attitudes during Cognitive Behavioural Therapy for Eating Disorders: The Impact of Personality Disorder Cognitions." Behavioural and Cognitive Psychotherapy 42, no. 2 (February 1, 2013): 224–37. http://dx.doi.org/10.1017/s1352465812001117.

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Background: The personality disorders are commonly comorbid with the eating disorders. Personality disorder pathology is often suggested to impair the treatment of axis 1 disorders, including the eating disorders. Aims: This study examined whether personality disorder cognitions reduce the impact of cognitive behavioural therapy (CBT) for eating disorders, in terms of treatment dropout and change in eating disorder attitudes in the early stages of treatment. Method: Participants were individuals with a diagnosed eating disorder, presenting for individual outpatient CBT. They completed measures of personality disorder cognitions and eating disorder attitudes at sessions one and six of CBT. Drop-out rates prior to session six were recorded. Results: CBT had a relatively rapid onset of action, with a significant reduction in eating disorder attitudes over the first six sessions. Eating disorder attitudes were most strongly associated with cognitions related to anxiety-based personality disorders (avoidant, obsessive-compulsive and dependent). Individuals who dropped out of treatment prematurely had significantly higher levels of dependent personality disorder cognitions than those who remained in treatment. For those who remained in treatment, higher levels of avoidant, histrionic and borderline personality disorder cognitions were associated with a greater change in global eating disorder attitudes. Conclusions: CBT's action and retention of patients might be improved by consideration of such personality disorder cognitions when formulating and treating the eating disorders.
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Fiani, Dimitri, Solangia Engler, Sherecce Fields, and Chadi Albert Calarge. "Iron Deficiency in Attention-Deficit Hyperactivity Disorder, Autism Spectrum Disorder, Internalizing and Externalizing Disorders, and Movement Disorders." Child and Adolescent Psychiatric Clinics of North America 32, no. 2 (April 2023): 451–67. http://dx.doi.org/10.1016/j.chc.2022.08.015.

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46

Gelder, M. G. "The Classification of Anxiety Disorders." British Journal of Psychiatry 154, S4 (May 1989): 28–32. http://dx.doi.org/10.1192/s0007125000295731.

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The classification of anxiety disorders is a controversial subject, and this controversy is reflected in the differences between the systems adopted in DSM-III-R and in the draft of ICD-10. The scheme in ICD-10 is the simpler: anxiety disorders are divided into phobic disorders and other anxiety disorders, and each is divided further into three subgroups. The three phobic disorders are: agoraphobia, social phobia, and specific phobia. The three subgroups of ‘other anxiety disorders’ are panic disorder, generalised anxiety disorder, and mixed anxiety and depressive disorder. The subdivisions of phobic disorder are those now generally adopted in most countries, and are uncontroversial. The subdivisions of generalised anxiety disorder, and mixed anxiety depressive disorder are also widely accepted, the latter group being particularly frequent among patients seen in general practice and not referred on to psychiatrists. Only the category of panic disorder is controversial. In addition to these categories which are specifically allocated to anxiety disorders, two others are relevant: ‘reactions to severe stress and adjustment disorders’, and anxious personality disorder.
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Atre-Vaidya, Nutan, and Syed M. Hussain. "Borderline Personality Disorder and Bipolar Mood Disorder: Two Distinct Disorders or a Continuum?" Journal of Nervous & Mental Disease 187, no. 5 (May 1999): 313–15. http://dx.doi.org/10.1097/00005053-199905000-00010.

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48

van Dijke, Annemiek, Julian D. Ford, Onno van der Hart, Maarten van Son, Peter van der Heijden, and Martina Buhring. "Complex posttraumatic stress disorder in patients with borderline personality disorder and somatoform disorders." Psychological Trauma: Theory, Research, Practice, and Policy 4, no. 2 (2012): 162–68. http://dx.doi.org/10.1037/a0025732.

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49

Phillips, Katharine A., and Walter H. Kaye. "The Relationship of Body Dysmorphic Disorder and Eating Disorders to Obsessive-Compulsive Disorder." CNS Spectrums 12, no. 5 (May 2007): 347–58. http://dx.doi.org/10.1017/s1092852900021155.

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ABSTRACTBody dysmorphic disorder (BDD) and eating disorders are body image disorders that have long been hypothesized to be related to obsessive-compulsive disorder (OCD). Available data suggest that BDD and eating disorders are often comorbid with OCD. Data from a variety of domains suggest that both BDD and eating disorders have many similarities with OCD and seem related to OCD. However, these disorders also differ from OCD in some ways. Additional research is needed on the relationship of BDD and eating disorders to OCD, including studies that directly compare them to OCD in a variety of domains, including phenomenology, family history, neurobiology, and etiology.
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Liao, Tzu-Chu, Yi-Ting Lien, Sabrina Wang, Song-Lih Huang, and Chuan-Yu Chen. "Comorbidity of Atopic Disorders with Autism Spectrum Disorder and Attention Deficit/Hyperactivity Disorder." Journal of Pediatrics 171 (April 2016): 248–55. http://dx.doi.org/10.1016/j.jpeds.2015.12.063.

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