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Books on the topic 'Disease propagation'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 February 2022

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1

De Mello, Walmor C., and Michiel J. Janse. Heart Cell Coupling and Impulse Propagation in Health and Disease. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-1155-7.

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2

Grape and Wine Research and Development Corporation (Australia), ed. Using grapevine rootstocks: The Australian perspective. Adelaide: Winetitles, 1994.

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3

Mitra, Sisir, ed. Guava: botany, production and uses. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789247022.0000.

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Abstract This publication presents the current state of knowledge concerning the origin, history, culture and trade of guava throughout the world. The fruit composition and processing, as well as fruit set, development and maturation are described. The propagation, biotechnology, nutrition, irrigation, orchard management, flowering, physiological disorders, photosynthesis, productivity, pests (including nematodes), diseases, postharvest physiology and storage of guava are also discussed. The book is mainly targeted at guava researchers, teachers and academics, students, advisors and industry support personnel.
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4

McDonald, G. I. Armillaria in the northern Rockies: Delineation of isolates into clones. [Ogden, Utah]: U.S. Dept. of Agriculture, Forest Service, Intermountain Forest and Range Experiment Station, 1988.

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5

McDonald, G. I. Armillaria in the northern Rockies: Delineation of isolates into clones. Ogden, UT: U.S. Dept. of Agriculture, Forest Service, Intermountain Research Station, 1988.

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6

McDonald, G. I. Armillaria in the northern Rockies: Delineation of isolates into clones. Ogden, UT: U.S. Dept. of Agriculture, Forest Service, Intermountain Research Station, 1988.

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7

Roth, Lewis F. Response of dwarf mistletoe-infested ponderosa pine to thinning. [Portland, Or.]: U.S. Dept. of Agriculture, Forest Service, Pacific Northwest Forest and Range Experiment Station, 1985.

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8

McDonald, G. I. Armillaria in the northern Rockies: Pathogenicity and host susceptibility on pristine and disturbed sites. Ogden, UT: U.S. Dept. of Agriculture, Forest Service, Intermountain Research Station, 1987.

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9

McDonald, G. I. Armillaria in the northern Rockies: Pathogenicity and host susceptibility on pristine and disturbed sites. Ogden, UT: U.S. Dept. of Agriculture, Forest Service, Intermountain Research Station, 1987.

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10

McDonald, G. I. Armillaria in the northern Rockies: Pathogenicity and host susceptibility on pristine and disturbed sites. Ogden, UT: U.S. Dept. of Agriculture, Forest Service, Intermountain Research Station, 1987.

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11

Comité d'étude sur les risques de propagation à la faune de maladies de cervidés d'élevage (Québec). Les risques de propagation à la faune de maladies de cervidés d'élevage: Rapport soumis au Ministre du loisir, de la chasse et de la pêche et au Ministre de l'agriculture, des pêcheries et de l'alimentation en juillet 1993. Québec: Gouvernement du Québec, Ministère de l'environnement et de la faune, Direction de la faune et des habitats, 1995.

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12

Ming you li, ping guo gao chan zai pei ji shu. [Beijing]: Zhongguo ren shi chu ban she, 1996.

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13

Mello, Walmor C. Heart Cell Coupling and Impulse Propagation in Health and Disease. Springer, 2012.

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14

Fabbri, Andrea, Giorgio Bartolini, Maurizio Lambardi, and Stan Kailis. Olive Propagation Manual. CSIRO Publishing, 2004. http://dx.doi.org/10.1071/9780643091016.

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This practical manual is an authoritative guide to olive propagation, providing extended information on seed germination, rooting of cuttings, grafting and micropropagation. The authors describe each topic in detail and discuss the relative advantages and disadvantages of each procedure. The Olive Propagation Manual has been developed to take into account the future demand for olive oil, which is expected to increase to three million tonnes annually over the next 10 years. Such volumes will require active farming programs and olive trees for new orchards and the replacement of olive trees in existing orchards. As the olive industry moves from traditional manual methods to mechanised operations, planting stock will need to be developed to meet future challenges. Varietal selection will need to be directed to clones that are early bearing, disease resistant, able to be mechanically harvested, and produce quality fruit and oil. Each of these issues are addressed throughout this book. The Olive Propagation Manual explores historical perspectives, traditional methods and state-of-the-art olive propagation including theoretical explanations and all practical aspects.
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15

Walmor C. De Mello (Editor) and M. J. Janse (Editor), eds. Heart Cell Coupling and Impulse Propagation in Health and Disease (Basic Science for the Cardiologist). Springer, 2002.

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16

Ruttum, Joanne C. Development of in vitro lily scale budlets as related to virus elimination. 1991.

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17

Safar, Jiri G. Prion Paradigm of Human Neurodegenerative Diseases Caused by Protein Misfolding. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0005.

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Data accumulated from different laboratories argue that a growing number of proteins causing neurodegeneration share certain characteristics with prions. Prion-like particles were produced from synthetic amyloid beta (Aβ‎) peptides of Alzheimer’s disease (AD), from recombinant α‎-synuclein linked to Parkinson’s disease (PD), and from recombinant tau associated with frontotemporal dementias (FTD). Evidence from human prions reveals that variable disease phenotypes, rates of propagation, and targeting of different brain structures are determined by distinct conformers (strains) of pathogenic prion protein. Recent progress in the development of advanced biophysical tools identified the structural characteristics of Aβ‎ in the brain cortex of phenotypically diverse AD patients and thus allowed an investigation of the prion paradigm of AD. The findings of distinctly structured strains of human brain Aβ‎, forming a unique spectrum of oligomeric particles in the cortex of rapidly progressive cases, implicates these structures in variable rates of propagation in the brain.
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18

Krake, LR, N. Steele Scott, MA Rezaian, and RH Taylor. Graft-transmitted Diseases of Grapevines. CSIRO Publishing, 1999. http://dx.doi.org/10.1071/9780643101067.

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For almost 40 years, Australian researchers have been part of an international group of scientists who have studied graft-transmitted disorders of the grapevine. The Australian wine and grape industries are undergoing significant expansion as is the case in some other countries. Preventing the spread of pathogens, by producing clean propagation material, and minimising the disease load on new vines, is essential for the continuing success of the industry. This book covers the characteristics of each class of graft-transmitted pathogen, their effect on vines, how they spread and strategies for their control. Eleven of the most important diseases are illustrated and described comprehensively, including information about occurrence, symptoms, detection, transmission and effect on yield and quality. Finally there is a discussion of quarantine issues and disease management. This book will be an invaluable teaching tool and is intended for vineyard managers, grape growers, consultants, extension offers and students. While it provides a basic understanding of the nature of pathogens, it will aid in field assessment and identification of the often confusing disease symptoms.
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19

Barker, Thomas Herbert. Illustrations of the Origin and Propagation of Certain Epidemic Diseases. HardPress, 2020.

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20

Jha, Vivekanand. Acute kidney injury in the tropics. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0241.

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The spectrum of acute kidney injury (AKI) encountered in the hospitals of the tropical zone countries is different from that seen in the non-tropical climate countries, most of which are high-income countries. The difference is explained in large part by the influence of environment on the epidemiology of human disease. The key features of geographic regions falling in the tropical zones are climatic, that is, high temperatures and absence of winter frost, and economic, that is, lower levels of income. The causes and presentation of tropical AKI reflect these prevailing cultural, socioeconomic, climatic, and eco-biological characteristics.Peculiarities of tropical climate support the propagation of several infectious organisms that can cause AKI and the disease-transmitting vectors. In contrast to the developed world, where AKI usually develops in already hospitalized patients with multiorgan problems and iatrogenic factors play a major role, tropical AKI is acquired in the community due to issues of public health importance such as safe water, sanitation, infection control, and good obstetric practices. Infections such as malaria, leptospirosis, typhus, HIV, and diarrhoeal diseases; envenomation by animals or insects; ingestion of toxic herbs or chemicals; intravascular haemolysis; poisoning; and obstetric complications form the bulk of AKI in the tropics. Poor access to modern medical facilities and practices such as seeking treatment from traditional faith-healers contribute to poor outcomes.AKI extracts macro- and microeconomic costs from the affected population and reduces productivity. Improvement in the outcomes of tropical AKI requires improvement in basic public health through effective interventions, and accessibility to effective medical care.
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21

Bianconi, Ginestra. Classical Percolation, Generalized Percolation and Cascades. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198753919.003.0012.

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This chapter characterizes the robustness of multiplex and multilayer networks using classical percolation, directed percolation and antagonistic percolation. Classical percolation determines whether a finite fraction of nodes of the multilayer networks are connected by any type of connection. Classical percolation can be affected by multiplexity since the degree correlations among different layers can modulate the robustness of the entire multilayer network. Directed percolation describes the propagation of a disease requiring cooperative infection from different layers of the multiplex network. It displays a rich phase diagram including both continuous and discontinuous phase transitions. Antagonist percolation on a duplex network describes the competition between two layers and can give rise to hysteresis loops corresponding to phases that either one layer or the other can percolate Avalanches generated by the generalized Sandpile Model and Watts–Strogatz Model are also discussed, emphasizing their relevance for studying the stability of power grids and financial systems.
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22

Bianconi, Ginestra. Epidemic Spreading. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198753919.003.0013.

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Epidemic processes are relevant to studying the propagation of infectious diseases, but their current use extends also to the study of propagation of ideas in the society or memes and news in online social media. In most of the relevant applications epidemic spreading does not actually take place on a single network but propagates in a multilayer network where different types of interaction play different roles. This chapter provides a comprehensive view of the effect that multilayer network structures have on epidemic processes. The Susceptible–Infected–Susceptible (SIS) Model and the Susceptible–Infected–Removed (SIR) Model are characterized on multilayer networks. Additionally, it is shown that the multilayer networks framework can also allow us to study interacting Awareness and epidemic spreading, competing networks and epidemics in temporal networks.
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23

Mason, John. Nursery Management. CSIRO Publishing, 2004. http://dx.doi.org/10.1071/9780643092136.

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Nursery Management second edition is an introduction to setting up and running profitable and efficient nursery businesses covering production, wholesale and retail nurseries at various scales of operation. The book discusses all the practical aspects of nursery management from site selection, production systems, and plant propagation through to materials and equipment. Conventional as well as non-chemical biological control measures for pests, weeds and disease control are included. Management issues for production nurseries, wholesalers and retail nurseries are treated separately for each operation and cover products and services, budgeting, production management, work scheduling and staffing. The chapter on marketing looks at the major changes that have taken place in the nursery industry, particularly the roles that landscapers, garden centres and hardware stores now play in retailing. It covers market research, promotions, distribution and consumer laws and shows the operator how to define the scope of their operation to fit their resources and how to sell their product for maximum return. This accessible guide is essential reading for anyone considering entering the nursery industry, and for those already in the business.
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24

Childers, Norman Franklin, and Wayne B. Sherman. The Peach: Culture, Cultivars, Breeding, Propagation, Nutrition, Training and Pruning, Diseases and Insects, Harvesting, Storage and Marketing. Dr Norman F Childers Phd, 1988.

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25

Galderisi, Maurizio, and Sergio Mondillo. Assessment of diastolic function. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0009.

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Modern assessment of left ventricular (LV) diastolic function should be based on the estimation of degree of LV filling pressure (LVFP), which is the true determinant of symptoms/signs and prognosis in heart failure.In order to achieve this goal, standard Doppler assessment of mitral inflow pattern (E/A ratio, deceleration time, isovolumic relaxation time) should be combined with additional manoeuvres and/or ultrasound tools such as: ◆ Valsalva manoeuvre applied to mitral inflow pattern. ◆ Pulmonary venous flow pattern. ◆ Velocity flow propagation by colour M-mode. ◆ Pulsed wave tissue Doppler of mitral annuls (average of septal and lateral E′ velocity).In intermediate doubtful situations, the two-dimensional determination of left atrial (LA) volume can be diagnostic, since LA enlargement is associated with a chronic increase of LVFP in the absence of mitral valve disease and atrial fibrillation.Some new echocardiographic technologies, such as the speckle tracking-derived LV longitudinal strain and LV torsion, LA strain, and even the three-dimensional determination of LA volumes can be potentially useful to add further information. In particular, the reduction of LV longitudinal strain in patients with LV diastolic dysfunction and normal ejection fraction demonstrates that a subclinical impairment of LV systolic function already exists under these circumstances.
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26

Kerouedan, Dominique. African and global health care prospects: the importance of the use of knowledge. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0019.

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The fight against infectious diseases necessarily involves medical entomology and biology, medicine and public health. Other dimensions are influential in the field of international health care cooperation because of pressures inflicted on national policies, particularly in Africa. Based on a historical review of cooperation and the analysis of its evolutions over the past 15 years, we highlight the paradox of world health safety: owing to targeting almost exclusively the AIDS pandemic over the course of decades, disequilibria can be observed in health care systems, especially with regard to epidemiological alert and surveillance and health care personnel, two essential pillars in the control of infectious diseases. We suggest that the Ebola epidemic and its rapid propagation in several West African countries might be a result of over a decade security policies relating to AIDS. The private sector cannot take the place of this democratic governance, neither in rich nor in poor countries. It is time to restore the standards for national and world governance in this field.
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27

Badimon, Lina, Felix C. Tanner, Giovanni G. Camici, and Gemma Vilahur. Pathophysiology of thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0018.

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Ischaemic heart disease and stroke are major causes of death and morbidity worldwide. Coronary and cerebrovascular events are mainly a consequence of a sudden thrombotic occlusion of the vessel lumen. Arterial thrombosis usually develops on top of a disrupted atherosclerotic plaque because of the exposure of thrombogenic material, such as collagen fibrils and tissue factor (TF), to the flowing blood. TF, either expressed by subendothelial cells, macrophage- and/or vascular smooth muscle-derived foam-cells in atherosclerotic plaques, is a key element in the initiation of thrombosis due to its ability to induce thrombin formation (a potent platelet agonist) and subsequent fibrin deposition at sites of vascular injury. Adhered platelets at the site of injury also play a crucial role in the pathophysiology of atherothrombosis. Platelet surface receptors (mainly glycoproteins) interact with vascular structures and/or Von Willebrand factor triggering platelet activation signalling events, including an increase in intracellular free Ca2+, exposure of a pro-coagulant surface, and secretion of platelet granule content. On top of this, interaction between soluble agonists and platelet G-coupled protein receptors further amplifies the platelet activation response favouring integrin alpha(IIb)beta(3) activation, an essential step for platelet aggregation. Blood-borne TF and microparticles have also been shown to contribute to thrombus formation and propagation. As thrombus evolves different circulating cells (red-blood cells and leukocytes, along with occasional undifferentiated cells) get recruited in a timely dependent manner to the growing thrombus and further entrapped by the formation of a fibrin mesh.
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28

Webster, Jessica Lynn, and Marco Vignuzzi. Viral evolution and impact for public health strategies in low-income countries. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0007.

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Viruses, the simplest organisms, obligate parasites that encode structural proteins and replicative functions requiring the cellular machinery of the host to ensure their propagation. Viruses are masters of evolution. An analysis of infectious diseases emerging since the 1980s revealed that most were caused by viruses, especially those with RNA genomes. New viral emergences are generally the result of intrinsic changes in the genetics of the virus to increase transmission, virulence or host range, and environmental or ecological changes that favor contacts between viruses and humans or other hosts. We describe the molecular mechanisms of viruses that ensure their rapid adaptation and evolution. We describe instances where viral evolution appears partially responsible for recent outbreaks and discuss the challenges in identifying the cause or consequence of viral evolution in the context of resource-rich versus -limited countries.
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29

Badimon, Lina, and Gemma Vilahur. Atherosclerosis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0040.

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Atherosclerosis is the main underlying cause of heart disease. The continuous exposure to cardiovascular risk factors induces endothelial activation/dysfunction which enhances the permeability of the endothelial layer and the expression of cytokines/chemokines and adhesion molecules. This results in the accumulation of lipids (low-density lipoprotein particles) in the extracellular matrix and the triggering of an inflammatory response. Accumulated low-density lipoprotein particles suffer modifications and become pro-atherogenic, enhancing leucocyte recruitment and further transmigration across the endothelium into the intima. Infiltrated monocytes differentiate into macrophages which acquire a specialized phenotypic polarization (protective or harmful), depending on the stage of the atherosclerosis progression. Once differentiated, macrophages upregulate pattern recognition receptors capable of engulfing modified low-density lipoprotein, leading to foam cell formation. Foam cells release growth factors and cytokines that promote vascular smooth muscle cell migration into the intima, which then internalize low-density lipoprotein via low-density lipoprotein receptor-related protein-1 receptors. As the plaque evolves, the number of vascular smooth muscle cells decline, whereas the presence of fragile/haemorrhagic neovessels increases, promoting plaque destabilization. Disruption of this atherosclerotic lesion exposes thrombogenic surfaces that initiate platelet adhesion, activation, and aggregation, as well as thrombin generation. Both lipid-laden vascular smooth muscle cells and macrophages release the procoagulant tissue factor, contributing to thrombus propagation. Platelets also participate in progenitor cell recruitment and drive the inflammatory response mediating the atherosclerosis progression. Recent data attribute to microparticles a potential modulatory effect in the overall atherothrombotic process. This chapter reviews our current understanding of the pathophysiological mechanisms involved in atherogenesis, highlights platelet contribution to thrombosis and atherosclerosis progression, and provides new insights into how atherothrombosis may be modulated.
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30

Meech, William Witler. Quince Culture. An Illustrated Hand-book for the Propagation and Cultivation of the Quince With Descriptions of its Varieties, Insect Enemies, Diseases, and Their Remedies. Franklin Classics, 2018.

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31

Hadidi, Ahmed, Ricardo Flores, John Randles, and Joseph Semancik. Viroids. CSIRO Publishing, 2003. http://dx.doi.org/10.1071/9780643069855.

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This comprehensive volume presents indispensable and up-to-date information on viroids and viroid diseases. It provides a single source of information on the properties of viroids, the economic impact of viroid diseases, and methods for their detection and control. It examines the diseases associated with different plant species, the geographic distribution and epidemiology of viroids, diseases of possible viroid etiology, and the future applications of viroids. Viroids examines the biology of viroids, molecular characteristics, localization and movement, replication, pathogenesis, viroids and gene silencing, classification, viroid-like satellite RNAs, detection of viroids using bioamplification hosts, biological indexing, polyacrylamide gel electrophoresis, molecular hybridisation and polymerase chain reaction. The book looks at the geographical distribution and epidemiology of viroids in North America, Australasia, China, Japan, Europe, the Middle East, Africa, South America, and at the global level. It covers the control of viroids including quarantine of imported germplasm, availability of viroid-tested propagation materials, thermotherapy, tissue culture, and other conventional strategies as well as biotechnological control approaches. Special topics such as ribozyme reaction of viroids and economic advantages of viroid infection are also included. Other chapters summarise the current state of knowledge concerning viroid diseases of the crop in question and aspects of the natural history of viroids in horticulture. Among the crops covered are potato, tomato, tobacco, cucumber, pome fruits, stone fruits, avocado, citrus, grapevines, hop, chrysanthemum, coleus, columnea, and coconut palm. The four eminent editors of this watershed volume have assembled an international group of more than 70 scientists who have substantial experience with viroids and viroid diseases. They have produced a cohesive and comprehensive work that can be used by students, researchers, extension agents, and regulators. It may also be of a great value to science managers, policy makers, and industries in formulating policies and products to obtain viroid-free plants and control viroid diseases. The information on plant quarantine and certification programs will help anyone concerned with the safe movement of plant material across international boundaries or within a single country.
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32

Mounsey, K. E., and S. F. Walton. Scabies and other mite infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0073.

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Acariasis in humans and animals is caused by a diversity of parasitic mites taxonomically grouped into the class Arachnida, subclass Acari. The zoonotic species that can transfer from birds and animals to man (e.g. Cheyletiella spp; Dermanyssus spp and Ornithonyssus spp) are important in that they often cause major skin irritation or a hypersensitivity reactions or alternatively act as vectors of diseases such as scrub typhus. Like ticks the lifecycle of mites involves four life stages of development. The female mite lays eggs on the host or in the environment; the eggs hatch into larvae and pass through two nymphal stages. All stages have eight legs except the six-legged larva. Transmission is predominantly via direct contact between hosts; however fomites have been recognised as a potential source of infestation although the importance of this is variable and dependent on the ability of the mite to survive in the environment. The geographic range of most zoonotic species is worldwide although some varieties may be rare or non-existent in some countries. No developmental change or propagation of the organism occurs during the transmission.
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33

The trees of America: Native and foreign, pictorially and botanically delineated and scientifically and popularly described, being considered principally with reference to their geography and history, soil and situation, propagation and culture, accidents and diseases ... New York: Harper, 1986.

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