Relevant bibliographies by topics / Disease programming

Academic literature on the topic 'Disease programming'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Disease programming"

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BARKER, D. J. P. "In utero programming of chronic disease." Clinical Science 95, no. 2 (August 1, 1998): 115–28. http://dx.doi.org/10.1042/cs0950115.

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1.Many human fetuses have to adapt to a limited supply of nutrients. In doing so they permanently change their structure and metabolism. 2.These ‘programmed' changes may be the origins of a number of diseases in later life, including coronary heart disease and the related disorders stroke, diabetes and hypertension. 3.This review examines the evidence linking these diseases to fetal undernutrition and provides an overview of previous studies in this area.
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김민형. "Fetal Programming and Adult Disease." JOURNAL OF THE KOREAN SOCIETY OF MATERNAL AND CHILD HEALTH 21, no. 1 (January 2017): 1–13. http://dx.doi.org/10.21896/jksmch.2017.21.1.1.

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Alexander, Barbara T. "Developmental Programming of Cardiovascular Disease." Colloquium Series on Integrated Systems Physiology: From Molecule to Function 5, no. 1 (June 27, 2013): 1–77. http://dx.doi.org/10.4199/c00084ed1v01y201305isp038.

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Falcão, Mário Cícero. "Fetal programming and future disease." Revista do Hospital das Clínicas 59, no. 6 (2004): 319–20. http://dx.doi.org/10.1590/s0041-87812004000600002.

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Bollinger, Lance M., Celsi E. Cowan, and Thomas P. LaFontaine. "Exercise Programming for Parkinsonʼs Disease." Strength and Conditioning Journal 34, no. 2 (April 2012): 55–59. http://dx.doi.org/10.1519/ssc.0b013e31824db335.

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Astorino, Todd A., and Matt M. Schubert. "Exercise Programming for Cardiovascular Disease." Strength and Conditioning Journal 34, no. 5 (October 2012): 60–64. http://dx.doi.org/10.1519/ssc.0b013e31825ab1aa.

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Vehaskari, V. Matti. "Prenatal programming of kidney disease." Current Opinion in Pediatrics 22, no. 2 (April 2010): 176–82. http://dx.doi.org/10.1097/mop.0b013e328336ebc9.

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Lau, C., J. M. Rogers, M. Desai, and M. G. Ross. "Fetal Programming of Adult Disease." Obstetric Anesthesia Digest 32, no. 2 (June 2012): 78–79. http://dx.doi.org/10.1097/01.aoa.0000414057.17428.db.

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Kratimenos, Panagiotis, and Anna A. Penn. "Placental programming of neuropsychiatric disease." Pediatric Research 86, no. 2 (April 19, 2019): 157–64. http://dx.doi.org/10.1038/s41390-019-0405-9.

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Thornburg, K. L. "The programming of cardiovascular disease." Journal of Developmental Origins of Health and Disease 6, no. 5 (July 15, 2015): 366–76. http://dx.doi.org/10.1017/s2040174415001300.

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In spite of improving life expectancy over the course of the previous century, the health of the U.S. population is now worsening. Recent increasing rates of type 2 diabetes, obesity and uncontrolled high blood pressure predict a growing incidence of cardiovascular disease and shortened average lifespan. The daily >$1billion current price tag for cardiovascular disease in the United States is expected to double within the next decade or two. Other countries are seeing similar trends. Current popular explanations for these trends are inadequate. Rather, increasingly poor diets in young people and in women during pregnancy are a likely cause of declining health in the U.S. population through a process known as programming. The fetal cardiovascular system is sensitive to poor maternal nutritional conditions during the periconceptional period, in the womb and in early postnatal life. Developmental plasticity accommodates changes in organ systems that lead to endothelial dysfunction, small coronary arteries, stiffer vascular tree, fewer nephrons, fewer cardiomyocytes, coagulopathies and atherogenic blood lipid profiles in fetuses born at the extremes of birthweight. Of equal importance are epigenetic modifications to genes driving important growth regulatory processes. Changes in microRNA, DNA methylation patterns and histone structure have all been implicated in the cardiovascular disease vulnerabilities that cross-generations. Recent experiments offer hope that detrimental epigenetic changes can be prevented or reversed. The large number of studies that provide the foundational concepts for the developmental origins of disease can be traced to the brilliant discoveries of David J.P. Barker.
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Dissertations / Theses on the topic "Disease programming"

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Erwig, Lars-Peter. "Macrophage programming in inflammatory disease." Thesis, University of Aberdeen, 2004. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU194083.

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Before embarking on the work presented here I showed that certain activating signals, such as IFN-gamma and TNF-alpha programmed macrophages to develop distinct sets of properties in vitro, which included unresponsiveness to other types of activation. This raised the question whether macrophage programming occurs in passive and active renal inflammation and whether the macrophage programme could be biased by systemic administration of cytokines. The data presented here shows that macrophages infiltrating acutely inflamed glomeruli of rats with nephrotoxic nephritis display programmed behaviour: operationally they behave as though programmed by IFN-gamma, and maintain these characteristics despite systematic administration of anti-inflammatory cytokines such as IL-4 or TGF-beta. This triggered further studies using a model of mesangioproliferative nephritis that can be adapted to induce resolving or progressive glomerular injury. These show that glomerular localisation does not always induce macrophage programming and that whether macrophages become programmed or not depends on the nature of the injury. Furthermore the data shows that macrophages become committed to a particular programme shortly after entering a programming environment. These observations raise question about the factors that induce macrophage programming at early stages of inflammatory disease and its consequences for its outcome. It provides an important mechanistic insight into how macrophage functional development is influenced by the underlying disease process.
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Hansell, J. A. "Oxidative stress and developmental programming of cardiovascular disease." Thesis, University of Cambridge, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603660.

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Building on a wealth of human and experimental data, these PhD studies present the hypothesis that oxidative stress underlies the molecular basis via which pregnancy complicated by prenatal hypoxia or prenatal under-nutrition contributes to the developmental programming of cardiovascular disease. An integrative approach was employed at the systems, isolated organ and molecular levels using a combination of fetal and adult rat offspring and chronically cathetised, unanaesthetised fetal sheep preparations. The data show that pregnancy complicated by hypoxia or undernutrition promoted asymmetric intrauterine growth restriction with subsequent postnatal catch-up growth. Maternal treatment with melatonin increased the placental expression of antioxidant enzymes and restored birth weight in undernourished but not in hypoxic pregnancy. Hypoxic but not undernourished pregnancy resulted in thickening of the fetal aortic wall with no alterations to the morphology of the fetal heart. In marked contrast, by adulthood offspring of hypoxic pregnancy showed dilated cardiomyopathy with increased vasoconstrictor reactivity in mesenteric arteries, but no changes in aortic structure. Pregnancy complicated by undernutrition did not affect the heart or the aorta in adult offspring but it did programme an increase in vascoconstrictor reactivity to ET-1 in mesenteric arteries. Maternal treatment with melatonin relieved all of the adverse cardiovascular consequences in adult offspring of hypoxic and undernourished pregnancy. Experiments in ovine pregnancy indicate that the xanthine oxidase pathway contributes to the increased generation of reactive oxygen species in hypoxic pregnancy. Combined, the work offers the potential for therapeutic targets for clinical intervention against a developmental origin of cardiovascular disease in complicated pregnancy.
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Martin-Gronert, Malgorzata Sylvia. "Mechanisms underlying the developmental programming of health and disease." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608293.

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Blackmore, Heather Louise. "Programming of cardiovascular disease by maternal diet-induced obesity." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648545.

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Rose, Catherine Margaret. "Programming of cardiovascular disease : an exploration of epigenetic mechanisms." Thesis, University of Edinburgh, 2015. http://hdl.handle.net/1842/21082.

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Fetal exposure to excess glucocorticoid is associated with low birth weight and increased cardiovascular disease risk in first generation offspring. Such phenotypes can be produced experimentally through the administration of the synthetic glucocorticoid dexamethasone (Dex) to pregnant rats during the last week of gestation. These ‘programmed effects’ can be transmitted to a second generation through both maternal and paternal lines. The overall hypothesis for this thesis was that the transmission of programmed effects through the male line may result from alterations in fetal germ cells, which form sperm in adulthood. Epigenetic reprogramming of germ cells is characterised by the genome-wide erasure and subsequent re-establishment of 5-methylcytosine (5mC), however this process has not previously been described for the rat. Furthermore, the involvement of more recently identified cytosine modifications; 5-hydroxymethylcytosine (5hmC), 5- formylcytosine (5fC) and 5-carboxylcytosine (5caC), has not been characterised during germ cell ontogeny. Using immunofluorescence to study DNA modifications during late gestation I identified that 5hmC, 5fC and 5caC were present between e14.5 and e16.5 but absent thereafter. In contrast, 5mC was absent during this time but remethylation was noted from e19.5 onwards. Prenatal Dex exposure was associated with the presence of significantly more 5mC-positive germ cells at e19.5 relative to controls. This difference did not persist at e20.5 suggesting that Dex exposure promotes premature global remethylation. The mechanisms for this are unclear since there were no differences between groups in the localisation of the DNA methyltransferases DNMT3a and 3b, or in markers of normal testis maturation. To enable the study of gene-specific changes in DNA methylation in the germline a colony of Germ Cell Specific-Enhanced Green Fluorescent Protein (GCS-EGFP) rats was established and characterised. GCS-EGFP rats had a transgenerational decrease in pup weight with Dex exposure, as in Wistar rats. The expression of both established and novel candidate genes was compared between strains. Multiple genes across different pathways had altered expression, with some affected in both Wistar and GCS-EGFP rats, whilst other differences were strain-specific. Enhanced Reduced Representation Bisulfite Sequencing was performed on liver and fetal germ cells from males exposed to Dex in utero to explore effects on DNA methylation. These studies confirm that epigenetic reprogramming occurs in the rat and that this process may be susceptible to modification by prenatal Dex exposure. GCS-EGFP rats also exhibited a Dex programming phenotype, with decreased pup weight and altered liver gene expression. The use of this unique strain of rats will permit dissection of the mechanisms for the transmission of programmed phenotypes across generations.
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Yuan, Ruoxi. "Dynamic Programming of Innate Immunity in Health and Disease." Diss., Virginia Tech, 2016. http://hdl.handle.net/10919/82925.

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Whether innate immune cells may be adapted into potential memory states has becoming an important question in the field of immunity. Although previous conceptual paradigm failed to acknowledge this important question, emerging clinical and basic observations have started to shed intriguing clues to shake the previous dogma regarding innate immunity of being "simple", "raw", "first-line defense with no memory". We have aimed to further address this fundamental issue in this dissertation work, under the close guidance of Dr. Liwu Li. We have chosen to use the model system of Toll-Like-Receptor (TLR) signaling networks within primary monocytes. TLRs play fundamental roles in sensing pathogen-associated molecular patterns (PAMPs) and modulation of innate immunity. Lipopolysaccharide (LPS), an endotoxin found on the cell membrane of gram-negative bacteria, is the ligand of TLR4 and induces a range of inflammatory as well as anti-inflammatory responses. Higher dosages of LPS were known to cause robust yet transient expression of pro-inflammatory mediators. On the other hand, the effects of super-low dose LPS, commonly manifested in humans with adverse health conditions, have been largely ignored in the basic research field. Super-low dose LPS may skew host immune environment into a mild non-resolving pro-inflammatory state, which is a risk factor for inflammatory diseases such as atherosclerosis, compromised wound healing, and elevated risks for sepsis. Our central hypothesize is that monocytes may be adapted by super-low dose LPS into a non-resolving low-grade inflammatory state conducive for the pathogenesis of inflammatory diseases. We have employed both in vitro cell culture system as well as in vivo disease models to test this hypothesis. For the in vitro system, we have cultured primary murine monocytes with increasing signal strength of LPS. Monocyte phenotypes such as the expression of key inflammatory mediators including cytokines, chemokines, and cellular surface markers were studied. Potential molecular and cellular mechanisms were examined. We revealed a novel low-grade inflammatory monocyte phenotype termed ML adapted by super-low dose LPS, mediated through IRF5. For the in vivo system, we have employed both acute and chronic models of inflammation. For the chronic model, we have tested the effects of super-low dose LPS on monocyte polarization in vivo, as well as its contribution to the pathogenesis of atherosclerosis. Furthermore, we have tested the effects of programmed monocytes on wound healing. For the acute model, we have tested the effects of pre-conditioning with super-low dose LPS on the subsequence risks of sepsis elicited by cecal ligation and puncture. We have demonstrated aggravated atherosclerosis, compromised wound healing, and increased sepsis mortality in mice pre-conditioned with super-low dose LPS. Taken together, our findings reveal that monocytes can be differentially programmed into distinct states, depending on the signal strength of LPS. The differential programming and adaptation of monocytes can occur both in vitro and in vivo, and may bear profound pathological consequences.
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Marchand, Michael C. "Fetal programming of renal morphology and function." Thesis, University of Northampton, 2004. http://nectar.northampton.ac.uk/2681/.

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Previous epidemiological evidence from a number of studies supports the hypothesis that the risk of essential hypertension, coronary heart disease and non-insulin dependent diabetes is, in part, programmed by intrauterine nutritional status. An increasing number of human studies indicate that the developing kidney is particularly vulnerable to the adverse effects of fetal growth retarding influences. In animals growth retarding diets or other insults, which have an impact on the development of cardiovascular functions, also appear to impact upon nephron number. In this study, the feeding of a 9% casein diet to pregnant rats, a mild protein restriction, reduced nephron number in the offspring, which progressively declined with age compared to those exposed to an 1 8% control diet. At weaning low-protein exposed offspring had hypertension and evedence of renal insufficiency. On natural death, the kidneys from aged male rats exposed to both low-protein and control maternal diets had a higher incidence glornerulosclerosis and renal disruption than females. Supplementing the maternal 9% casein diet with 3% glycine, 1.5% urea and 3% alanine in the rat normalised nephron number in the offspring. Only the addition of glycinc in the maternal low- protein diet prevented the appearance of high blood pressure in the offspring. In this study it has been demonstrated that in humans, those of a low birth weight or ponderal index, a marker of fetal undernutrition, had evidence of increased glomerular permeability, but not elevated blood pressure at age 10. This association was not evident at age 12 or in a separate cohort of young adults. It is possible that hypertension and a reduced nephron reserve are not causally associated. The evidence from this thesis suggest that prenatal undernutrition may programme renal structure in later life, but that renal programming is not one of the primary mechanisms leading to hypertension
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Blackman, Nicole. "Chronic Disease and Injury Prevention Programming for Canada's Indigenous Population." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/5165.

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Local public health units in the province of Ontario, Canada, are often the primary source of health promotion and health education resources, but many do not provide programming specific to the Indigenous population. As of January 2018, modernization of the Ontario Public Health Standards requires public health units to work with the Indigenous population in providing culturally appropriate programs and services. The practice question guiding the capstone project was to determine what chronic disease and injury prevention programs exist that are culturally appropriate for the Indigenous population. The purpose of this project was to do an environmental scan and compile an inventory of existing health promotion programming that is culturally appropriate to the Indigenous population. In total, 72 Indigenous-specific programs were identified from the 26 organizations that were included in the environmental scan. Of the 26 organizations, 3 were public health units, 7 were Aboriginal health access centers, 7 were Indigenous friendship centers, 5 were Indigenous health organizations, and 4 were non-Indigenous organizations with an Indigenous component. Results from the capstone project will inform public health units of available, culturally appropriate programs that can be adapted to their local context, thereby addressing a significant gap in the current public health system. This doctoral project aligns with the design of a new model of care in the Ontario public health system and has the potential to address a gap in practice at both the local and provincial level by providing culturally appropriate guidance in the effective delivery of CDIP programming specific to the Indigenous population. This positive social change would impact the health status of this underserved population.
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Warner, Matthew John. "Mechanisms of post-transcriptional gene regulation in the developmental programming of adulthood disease." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610291.

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Forsén, Tom. "Early growth and adult disease : programming of coronary heart disease, type 2 diabetes and hypertension by fetal and childhood growth." Helsinki : University of Helsinki, 2000. http://ethesis.helsinki.fi/julkaisut/laa/kansa/vk/forsen/.

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Books on the topic "Disease programming"

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Langley-Evans, S. C., ed. Fetal nutrition and adult disease: programming of chronic disease through fetal exposure to undernutrition. Wallingford: CABI, 2004. http://dx.doi.org/10.1079/9780851998213.0000.

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Ncgoncgo, N. Proceedings of MTP II Broad Programming Workshop for AIDS in the Workplace in Botswana. [Gaborone] Botswana: The Unit, 1996.

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Pinkston, Elsie M. Analysis of behavioral programming for Alzheimer's and other dementia clients: Final report. Chicago, Ill: University of Chicago, School of Social Service Administration, 1988.

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Pinkston, Elsie M. Analysis of behavioral programming for Alzheimer's and other dementia clients: Final report. Chicago, Ill: University of Chicago, School of Social Service Administration, 1988.

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Ncgoncgo, N. Proceedings of MTP II Broad Programming Workshop for Women and AIDS in Botswana. [Gaborone] Botswana: The Unit, 1996.

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Page, Sara. As men we care--: Male involvement in community home-based care programming in Zambia. Lusaka: Zambia Red Cross Society, 2008.

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Johnson, Cary Alan. Off the map: How HIV/AIDS programming is failing same-sex practicing people in Africa. New York, NY: International Gay and Lesbian Human Rights Commission, 2007.

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Malawi, UNICEF. Vulnerability & child protection in the face of HIV: Report of the United Nations Technical Review Team on programming for children affected by HIV and AIDS in Malawi. [Lilongwe, Malawi]: UNICEF, 2011.

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Analysis of experience: The role of public-private partnerships in HIV/AIDS prevention, control, and treatment programming. Lanham, MD: University Press of America, Inc., 2005.

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United Nations Population Fund. Zambia, ed. Rapid socio-cultural research as a methodology for informing sexual and reproductive health/HIV/AIDS programming in North-Western Province, Zambia. Zambia: Government of the Republic of Zambia and United Nations Population Fund, 2005.

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Book chapters on the topic "Disease programming"

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Franco, Maria. "Perinatal Programming of Cardiovascular Disease." In Arterial Disorders, 83–91. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-14556-3_6.

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Desai, Mina, Erin Keen-Rhinehart, and Michael G. Ross. "Perinatal Appetite Programming." In Early Life Origins of Human Health and Disease, 142–63. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000221161.

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Brøns, Charlotte, and Louise Justesen. "Fetal Programming of Fatty Liver Disease." In The Human Gut-Liver-Axis in Health and Disease, 65–80. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-98890-0_4.

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García-Jiménez, Beatriz, Agapito Ledezma, and Araceli Sanchis. "MMRF for Proteome Annotation Applied to Human Protein Disease Prediction." In Inductive Logic Programming, 67–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-21295-6_11.

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Gupta, Fiona, and Punit Agrawal. "Optimizing Deep Brain Stimulation Programming in Parkinson’s Disease." In Surgery for Parkinson's Disease, 81–90. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-23693-3_7.

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Nijland, Mark J., and Peter W. Nathanielsz. "Developmental Programming of the Kidney." In Early Life Origins of Human Health and Disease, 133–41. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000221160.

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Lee, Eva K., and Tsung-Lin Wu. "Classification and Disease Prediction Via Mathematical Programming." In Handbook of Optimization in Medicine, 1–50. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-09770-1_12.

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Gatford, Kathryn L., Miles J. De Blasio, Miodrag Dodic, Dane M. Horton, and Karen L. Kind. "Perinatal Programming of Adult Metabolic Homeostasis." In Early Life Origins of Health and Disease, 157–76. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/0-387-32632-4_13.

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Cripps, Roselle L., and Susan E. Ozanne. "Early-Life Programming of Adipogenesis and Adiposity." In Adipose Tissue in Health and Disease, 459–72. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2010. http://dx.doi.org/10.1002/9783527629527.ch24.

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Relton, Caroline L., George Davey-Smith, and Susan E. Ozanne. "Developmental Epigenetic Programming in Diabetes and Obesity." In Environmental Epigenomics in Health and Disease, 235–53. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-36827-1_11.

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Conference papers on the topic "Disease programming"

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Lee, Eva K., Tsung-Lin Wu, Onur Seref, O. Erhun Kundakcioglu, and Panos Pardalos. "Classification and disease prediction via mathematical programming." In DATA MINING, SYSTEMS ANALYSIS AND OPTIMIZATION IN BIOMEDICINE. AIP, 2007. http://dx.doi.org/10.1063/1.2817343.

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Zhang, Xinhong, Xiangyu Wang, and Fan Zhang. "Infectious Disease Dynamics Model Considering Suspected Population." In 2021 12th International Symposium on Parallel Architectures, Algorithms and Programming (PAAP). IEEE, 2021. http://dx.doi.org/10.1109/paap54281.2021.9720315.

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Wang, Chen-Shu, Chun-Jung Juan, Tung-Yao Lin, Chun-Chang Yeh, and Shang-Yu Chiang. "Prediction Model of Cervical Spine Disease Established by Genetic Programming." In the 4th Multidisciplinary International Social Networks Conference. New York, New York, USA: ACM Press, 2017. http://dx.doi.org/10.1145/3092090.3092097.

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Hirose, Hideo, and Liangliang Wang. "Prediction of Infectious Disease Spread Using Twitter: A Case of Influenza." In 2012 Fifth International Symposium on Parallel Architectures, Algorithms and Programming (PAAP). IEEE, 2012. http://dx.doi.org/10.1109/paap.2012.23.

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Senatore, Rosa, Antonio Della Cioppa, and Angelo Marcelli. "Automatic Diagnosis of Parkinson Disease through Handwriting Analysis: A Cartesian Genetic Programming Approach." In 2019 IEEE 32nd International Symposium on Computer-Based Medical Systems (CBMS). IEEE, 2019. http://dx.doi.org/10.1109/cbms.2019.00071.

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Alejandre Alcazar, Miguel A., Philipp Kasper, Eva Rother, Ruth Kuschewski, and Jörg Dötsch. "Maternal Obesity Induces Obstructive Lung Disease In The Offspring: Pulmonary Pulmonary Implications Of Metabolic Programming?" In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6751.

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Zulfadhilah, Muhammad. "Expert System for Eye Disease Diagnosis with Best First Search (BFS) Method Using Web-Based Programming." In Proceedings of the First National Seminar Universitas Sari Mulia, NS-UNISM 2019, 23rd November 2019, Banjarmasin, South Kalimantan, Indonesia. EAI, 2020. http://dx.doi.org/10.4108/eai.23-11-2019.2298406.

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Xia, Tian, Jeremy Cosgrove, Jane Alty, Stuart Jamieson, and Stephen Smith. "Application of classification for figure copying test in Parkinson's disease diagnosis by using cartesian genetic programming." In GECCO '19: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3319619.3326822.

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Yutaka SASAKI and Masato SUZUKI. "Construction of the Automatic Diagnosis System of Plant Disease Using Genetic Programming Which Paid Its Attention to Variety." In 2003, Las Vegas, NV July 27-30, 2003. St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2003. http://dx.doi.org/10.13031/2013.13718.

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Harrison, Shauna M., Katherine C. Smith, and Ann C. Klassen. "Abstract A12: Explicit and implicit relevance in the coverage of chronic disease prevention in Spanish-language morning news programming." In Abstracts: AACR International Conference on the Science of Cancer Health Disparities‐‐ Sep 18-Sep 21, 2011; Washington, DC. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1055-9965.disp-11-a12.

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Reports on the topic "Disease programming"

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Thompson, Joseph. How WASH Programming has Adapted to the COVID-19 Pandemic. Institute of Development Studies (IDS), December 2020. http://dx.doi.org/10.19088/slh.2021.001.

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Since first appearing at the end of 2019, the novel coronavirus disease (COVID-19) has spread at a pace and scale not seen before. On 11 March 2020, the World Health Organization (WHO) declared COVID-19 a pandemic. A rapid response was called for, and actors across the globe worked quickly to develop sets of preventative measures to contain the disease. One mode of transmission identified early on in the crisis was via surfaces and objects (fomites) (Howard et al. 2020). To combat this, hand hygiene was put forward as a key preventative measure and heralded as ‘the first line of defence against the disease’ (World Bank 2020). What followed was an unprecedented global focus on handwashing with soap. Health messages on how germs spread, the critical times at which hands should be washed, and methods for correct handwashing were shared (Centers for Disease Control and Prevention 2020). Political leaders around the world promoted handwashing and urged people to adopt the practice to protect against the coronavirus. The primary and secondary impacts of COVID-19 have affected people and industries in a variety of different ways. For the WASH sector, the centring of handwashing in the pandemic response has led to a sudden spike in hygiene activity. This SLH Rapid Topic Review takes stock of some of the cross-cutting challenges the sector has been facing during this period and explores the adaptations that have been made in response. It then looks forwards, thinking through what lies ahead for the sector, and considers the learning priorities for the next steps.
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Thompson, Joseph. How WASH Programming has Adapted to the COVID-19 Pandemic. The Sanitation Learning Hub, Institute of Development Studies, December 2020. http://dx.doi.org/10.19088/slh.2021.0015.

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Abstract:
Since first appearing at the end of 2019, the novel coronavirus disease (COVID-19) has spread at a pace and scale not seen before. On 11 March 2020, the World Health Organization (WHO) declared COVID-19 a pandemic. A rapid response was called for, and actors across the globe worked quickly to develop sets of preventative measures to contain the disease. One mode of transmission identified early on in the crisis was via surfaces and objects (fomites) (Howard et al. 2020). To combat this, hand hygiene was put forward as a key preventative measure and heralded as ‘the first line of defence against the disease’ (World Bank 2020). What followed was an unprecedented global focus on handwashing with soap. Health messages on how germs spread, the critical times at which hands should be washed, and methods for correct handwashing were shared (Centers for Disease Control and Prevention 2020). Political leaders around the world promoted handwashing and urged people to adopt the practice to protect against the coronavirus. The primary and secondary impacts of COVID-19 have affected people and industries in a variety of different ways. For the WASH sector, the centring of handwashing in the pandemic response has led to a sudden spike in hygiene activity. This SLH Rapid Topic Review takes stock of some of the cross-cutting challenges the sector has been facing during this period and explores the adaptations that have been made in response. It then looks forwards, thinking through what lies ahead for the sector, and considers the learning priorities for the next steps.
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