Relevant bibliographies by topics / Disease control

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Academic literature on the topic 'Disease control'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 June 2025

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Journal articles on the topic "Disease control"

UB, Muhammad Iqbal. "Aedes Mosquito: Disease and Control." Pharmaceutical Drug Regulatory Affairs Journal 7, no. 1 (2024): 1–2. http://dx.doi.org/10.23880/pdraj-16000152.

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Rajagopalan, P. K. "Aspects of Vector Borne Disease Control." Journal of Communicable Diseases 50, no. 01 (2018): 28–31. http://dx.doi.org/10.24321/0019.5138.201806.

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Sutthisa, W. "Biological Control Properties of Cyathus spp. to Control Plant Disease Pathogens." Journal of Pure and Applied Microbiology 12, no. 4 (2018): 1755–60. http://dx.doi.org/10.22207/jpam.12.4.08.

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Murodulloyev, Xolmuhammad. "Disease Control of Apricot Trees." INTERNATIONAL JOURNAL OF LIFE SCIENCE AND AGRICULTURE RESEARCH 01, no. 01 (2022): 14–16. https://doi.org/10.5281/zenodo.10444146.

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ABSTRACT The article provides information about apricot diseases and their types. Wilsonomyces carpophilus is the fungus that causes apricot shot holes. It spends the winter on diseased buds as well as twigs. During winter and spring showers, as well as when water splashes up from the ground, the spores on these sections of the tree might be transported. Because those spores require 24 hours of moisture to develop in and induce infection, damp and humid circumstances are conducive to disease propagation. Apricot tree illnesses may wreck years of labor in a matter of days, yet the majority of apricot tree diseases are easily identified, treated, and sometimes prevented. Here's all you need to know about apricot tree diseases.

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MILNER, FABIO AUGUSTO. "Disease Control." Mathematical Population Studies 17, no. 2 (2010): 67–68. http://dx.doi.org/10.1080/08898481003689403.

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Sharma, SN. "Kyasanur Forest Disease: Vector Surveillance and its Control." Journal of Communicable Diseases 51, no. 02 (2019): 38–44. http://dx.doi.org/10.24321/0019.5138.201915.

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Faderin, Emmanuel. "Genomic Technologies in Vector-Borne Disease Surveillance and Control." International Journal of Research Publication and Reviews 5, no. 10 (2024): 270–82. http://dx.doi.org/10.55248/gengpi.5.1024.2709.

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Marriott, Gill. "Communicable disease control." Primary Health Care 11, no. 1 (2001): 17. http://dx.doi.org/10.7748/phc.11.1.17.s16.

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Woodhouse, S. "Communicable disease control." BMJ 321, no. 7254 (2000): 2. http://dx.doi.org/10.1136/bmj.321.7254.s2-7254.

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Sutton, Turner B. "Plant disease control." Crop Protection 14, no. 3 (1995): 261. http://dx.doi.org/10.1016/0261-2194(95)90006-3.

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Dissertations / Theses on the topic "Disease control"

Franco, Iborra Sandra. "Mitochondrial quality control in neurodegenerative diseases: focus on Parkinson’s disease and Huntington’s disease." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/565668.

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Darrerament s’han produït avanços importants que han contribuït al coneixement dels mecanismes de disfunció cel·lular i mort en la malaltia de Parkinson (MP) i en la malaltia de Huntington (MH). Ambdues malalties són trastorns del moviment que es caracteritzen per la pèrdua específica de neurones dels ganglis basals, les neurones dopaminèrgiques de la substància nigra (SN), en el cas de la MP i les neurones espinoses de l’estriat, en el cas de la MH. Malgrat les diferències, ambdues comparteixen processos patològics comuns com la presència de proteïnes malplegades, l’estrés oxidatiu i disfunció mitocondrial. La mitocòndria és la font d’energia principal en les cèl·lules eucariotes, però també és un orgànul dinàmic relacionat amb una gran quantitat de processos cel·lulars. La disrupció de la homeòstasis mitocondrial i la subseqüent disfunció mitocondrial juguen un paper important en la patofisiologia de les malalties neurodegeneratives. El manteniment de la integritat mitocondrial a través de diferents mecanismes de control és crític per a la superviviència neuronal. Aquesta tesi es centra en l’estudi dels mecanismes de control de qualitat mitocondrial en la MP i la MH, per tal d’entendre millor els mecanismes que duen a la mort cel·lular. En el primer capítol, he estudiat el transport de proteïnes a la mitocòndria en models in vitro i in vivo de la MP. In vitro, la inhibició del complexe I produeix una alteració del transport de proteïnes a la mitocòndria així com una disminució dels nivells de proteïnes OXPHOS, acumulació de proteïnes agregades i disminució dels nivells de chaperones mitocondrials. Per tal de restablir el transport de proteïnes mitocondrials es van sobreexpressar dos components clau del sistema de translocases: la translocasa de la membrana externa 20 (TOM20) i la translocasa de la membrana interna 23 (TIM23). La sobreexpressió in vitro de TOM20 i TIM23 va restaurar el transport de proteïnes mitocondrials i va alleugerar la disfunció mitocondrial i la mort cel·lular. La inhibició del complexe I en ratolins també dóna lloc a una alteració del transport de proteïnes mitocondrials i produeix neurodegeneració del sistema dopaminèrgic. La sobreexpressió de TIM23 va restaurar parcialment el transport de proteïnes i va protegir lleugerament les neurones dopaminèrgiques de la SN. En canvi, la sobreexpressió de TOM20 va ser incapaç de millorar el transport de proteïnes mitocondrials i, fins i tot, va exacerbar la mort cel·lular. Aquests resultats posen de relleu el paper de la disfunció del transport de proteïnes mitocondrials, en particular de dos dels seus components, en la patogènesis de la MP i suggereixen la necessitat de futurs estudis es centrin en altres elements d’aquest sistema. En el segon capítol, he estudiat el paper de la proteïna huntingtina en la mitofàgia i com la seva mutació, que dóna lloc a una expansió de glutamines, pot afectar a aquesta funció. Per a tal fi, he treballat en un model in vitro de cèl·lules estriatals ST-Q7 (control) i ST-Q111 (mutant). En condicions fisiològiques, la mitofàgia induïda no es troba mitjançada pel reclutament de parkin als mitocondris despolaritzats. La huntingtina mutada afecta la mitofàgia induïda a través de l’alteració de la seva funció de scaffold en diferents passos del procés de mitofàgia: (i) activació d’ULK1 a través de l’alliberament de mTORC1, (ii) formació del complexe Beclin 1-Vps15,(iii) interacció dels adaptadors de mitofàgia OPTN i NDP52 amb huntingtina i, (iv) amb LC3. Com a resultat, els mitocondris de les cèl·lules ST-Q111 estan més danyats i tenen una respiració mitocondrial deficient. Aquests resultats demostren la presència d’una alteració en la mitofàgia com un mecanisme lligat a la MH. En conclusió, el descobriment de noves dianes mitocondrials en la MP i MH emfatitza el paper important que juga el control de qualitat mitocondrial en la neurodegeneració. In the past years, several important advances have expanded our understanding of the pathways that lead to cell dysfunction and death in Parkinson’s disease (PD) and Huntington’s disease (HD). Both diseases are movement disorders characterized by the loss of a specific subset of neurons within the basal ganglia, dopaminergic neurons in the substantia nigra pars compacta (SNpc), in the case of PD, and medium spiny neurons in the striatum, in the case of HD,. Despite distinct clinical and pathological features, these two neurodegenerative disorders share critical underlying pathogenic mechanisms such as the presence of misfolded and/or aggregated proteins, oxidative stress and mitochondrial anomalies. Mitochondria are the prime energy source in most eukaryotic cells, but these highly dynamic organelles are also involved in a multitude of cellular events. Disruption of mitochondrial homeostasis and the subsequent mitochondrial dysfunction plays a key role in the pathophysiology of neurodegenerative diseases. Therefore, maintenance of mitochondrial integrity through different surveillance mechanisms is critical for neuronal survival. In this thesis I have studied in depth some mitochondrial quality control mechanisms in the context of PD and HD, in order to broaden the knowledge about the pathomechanisms leading to cell death. In the first chapter I have studied mitochondrial protein import in in vitro and in vivo models of PD. In vitro, complex I inhibition, a characteristic pathological hallmark in PD, impaired mitochondrial protein import. This was associated with OXPHOS protein downregulation, accumulation of aggregated proteins inside mitochondria and downregulation of mitochondrial chaperones. Therefore, we aimed to reestablish the mitochondrial protein import by overexpressing two key components of the system: translocase of the outer membrane 20 (TOM20) and translocase of the inner membrane 23 (TIM23). Overexpression of TOM20 and TIM23 in vitro restored protein import into mitochondria and ameliorated mitochondrial dysfunction and cell death. Complex I inhibition also impaired mitochondrial protein import and led to dopaminergic neurodegeneration in vivo. Overexpression of TIM23 partially rescued protein import into mitochondria and slightly protected dopaminergic neurons in the SNpc. On the contrary, TOM20 overexpression did not rescue protein import into mitochondria and exacerbated neurodegeneration in both SNpc and striatum. These results highlight mitochondrial protein import dysfunction and the distinct role of two of their components in the pathogenesis of PD and suggest the need for future studies to target other elements in the system. In the second chapter, I have studied the role of huntingtin in mitophagy and how the polyglutamine expansion present in mutant huntingtin can affect its function. For such, I worked with differentiated striatal ST-Q7 (as control) and ST-Q111 (as mutant) cells, expressing full length huntingtin. In these conditions, induced mitophagy was not mediated by Parkin recruitment into depolarized mitochondria. Mutant huntingtin impaired induced mitophagy by altering wildtype huntingtin scaffolding activity at different steps of mitophagy process: (i) ULK1 activation through its release from the mTORC1, (ii) Beclin1-Vps15 complex formation, (iii) interaction of the mitophagy adapters OPTN and NDP52 with huntingtin and (iv) with LC3. As a result, mitochondria from ST-Q111 cells exhibited increased damage and altered mitochondrial respiration. These results uncover impaired mitophagy as a potential pathological mechanism linked with HD. In conclusion, we have discovered new mitochondrial targets for PD and HD emphasizing the important role that mitochondrial quality control plays in neurodegeneration

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Du, Toit Eben Francois. "Pinning control of disease networks." Thesis, University of Pretoria, 2015. http://hdl.handle.net/2263/56133.

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The modelling of contagion spread on contact networks provide valuable insights to epidemiologists and policymakers trying to control and eradicate diseases. This thesis proposes, implements and analyses a methodology for inserting disease contact networks of HIV into feedback control loops and applying open-loop pinning control to their nodes. Pinning control aims to medicate only a portion of an entire network in order to achieve the same outcomes that would be seen when all nodes are controlled. The control loops are simulated using networks ranging from size N = 100 nodes to N = 10000 nodes. Simulations aim to control the average maximum incidence in the networks by first estimating the reference average transmissibility from the statistical physics technique known as bond percolation. Once the average transmissibility is known, node-, network- and population mass-action models can be measured for incidence. Two selective pinning control strategies, namely proportional feedback and nonlinear model predictive control (NMPC), are compared with one another and also with a random pinning strategy. The budget, measured in quality-adjusted life years (QALYs), is added to the cost-function for NMPC control. It is shown that budget can indeed be controlled while incidence varies, while incidence may be controlled as budget varies. Pinning control of disease networks is a feasible methodology to analyse the future and steady-state outcomes of interventions in fast-spreading (high-risk) disease contact networks. Modellering van die verspreiding van siektes oor kontak-netwerke verskaf waardevolle inligting aan beleidmakers en epidemioloë wat besluit op maatreëls vir voorkoming teen die siekte. Hierdie proefskrif hou n metode voor wat gebruik word om siekteverspreidings-netwerke te simuleer en te analiseer. Dit word gedoen op netwerke met nodusse wat varieer tussen N = 100 en N = 10000. Netwerke waarin HIV versprei word gebruik. Penbeheer word in n oopluskonfigurasie op elke nodus toegepas binne n geslote terugvoerlus op netwerkvlak. Penbeheer se doel is om slegs sekere nodusse te beheer om dieselfde uitkomste vir die voorkoms van HIV tydens n epidemie te meet. Die doel is om die gemiddelde waarskynlikheid vir oordrag van die siekte tussen nodusse te beheer en sodoende, deur middel van die tegniek genaamd bond percolation , te bepaal hoe groot die finale epidemie gaan wees. Sodra die gemiddelde waarskynlikheid bekend is, kan nodus-, netwerk- en populasiemodelle saamgestel word. Twee selektiewe penbeheer-strategieë (proporsioneel, en NMPC) word met mekaar en met n derde willekeurige tegniek vergelyk. Die beheer van begrotings, gemeet in quality-adjusted life years (QALYs), word deur die NMPC strategie hanteer. Siektes binne kontaknetwerke kan dus beheer word met selektiewe penbeheer. Penbeheer-strategieë word ook vergelyk op grond van die dosisse wat hulle benodig, asook die akkuraatheid van die bestendigde-toestand resultate. Penbeheer van siekteverspreidings-netwerke is n werkbare metode om toekomstige en bestendigde-toestand uitkomste van mediese ingrepe op netwerke mee te analiseer. Thesis (PhD)--University of Pretoria, 2015. tm2016 Electrical, Electronic and Computer Engineering PhD Unrestricted

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Hillman, Anne M. "Perceived control in the everyday occupational roles of people with Parkinson's disease and their partners." Connect to full text, 2006. http://hdl.handle.net/2123/1621.

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Thesis (Ph. D.)--University of Sydney, 2006. Title from title screen (viewed May 1, 2007). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Occupation and Leisure Sciences. Includes bibliographical references. Also issued in print.

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Bennett, Richard. "The economics of livestock disease control." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316155.

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Cox, Mark. "Ecological economics of wildlife disease control." Thesis, University of York, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310898.

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Cattaneo, Gabriele 1984. "Bilingual language control : Evidence from Parkinson's disease." Doctoral thesis, Universitat Pompeu Fabra, 2017. http://hdl.handle.net/10803/482046.

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How can bilinguals easily and flexibly switch between languages without errors? The aim of this thesis is to advance our knowledge about the control mechanisms involved in bilingual language production. I investigated this issue in two ways. Firstly, by exploring similarities between bilingual language control (bLC) and domain-general executive control (EC). Secondly, by investigating the specificity of bLC mechanisms within the linguistic domain. To do so, I measured behavioural responses of Catalan/Spanish bilinguals affected by Parkinson’s disease and healthy controls in tasks tapping bLC, EC and language control, when only one language was involved. I explored Parkinson’s disease individuals because this neurodegenerative disorder affects brain areas involved in both domain-general EC and the bLC. The findings of this dissertation suggest that some bLC mechanisms are highly specific and are not involved in other linguistic or non-linguistic control contexts. However, some other mechanisms of bLC are shared across different domains of control and implemented by EC mechanisms. These results extend previous knowledge about the nature of mechanisms involved in bLC and are useful for future models of bilingual language production. Com ho fan els bilingües per passar d’una llengua a una altra amb facilitat, flexibilitat i sense cometre errors? L’objectiu d’aquesta tesi és avançar el nostre coneixement sobre els mecanismes de control involucrats en la producció de llenguatge en bilingües. He investigat aquest tema de dues maneres: Primer: he explorat les similituds entre el control bilingüe del llenguatge i el control executiu de domini general. Segon: he investigat l’especificitat dels mecanismes de control bilingüe dins del domini lingüístic. Per aquest supòsit, he avaluat les respostes conductuals de bilingües Català/Castellà afectats per la malaltia del Parkinson i controls bilingües (no malalts) en tasques de control de llenguatge bilingüe, control executiu i control de llenguatge quan una sola llengua hi està involucrada. He estudiat aquesta malaltia degenerativa perquè afecta zones del cervell involucrades tant en control executiu de domini general, com en control bilingüe del llenguatge. Els descobriments d’aquesta dissertació suggereixen que alguns dels mecanismes de control bilingüe del llenguatge són altament específics, i no estan involucrats en altres contextos de control lingüístic o no lingüístic. D’altra banda, altres mecanismes de control estan compartits a través de diferents dominis i estan implementats per mecanismes de control executiu.

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Chochó, Karen S. "Hispanic migrants and cross-border disease control of Arizona's vaccine preventable diseases." restricted, 2008. http://etd.gsu.edu/theses/available/etd-04222008-151047/.

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Thesis (M.Ph.)--Georgia State University, 2008. Title from file title page. Richard Rothenberg, committee chair; Russ Toal, Karen E. Gieseker, committee members. Electronic text (135 p. : col. ill.) : digital, PDF file. Description based on contents viewed August 12, 2008. Includes bibliographical references (p. 127-135).

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Chocho, Karen. "Hispanic Migrants and Cross-border Disease Control of Arizona's Vaccine Preventable Diseases." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/iph_theses/35.

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BACKGROUND: According to the Centers for Disease Control and Prevention and the National Immunization Program, there is an increase in the re-emergence of past diseases. Even with mandatory vaccination practices in the United States, there are still a number of cases of vaccine-preventable diseases (VPDs) reported yearly. It is speculated that the re-emergence of VPDs is in part due to the increase in international travel as well as the influx of immigrants. One particular group of interest includes the Hispanic migrants coming from Central and South America where some of these diseases are endemic. OBJECTIVE: The purpose of this paper is to determine the extent of VPD cases in the border state of Arizona that may be attributed to Hispanic migrant influx using data from the MMWR: Summary of Notifiable Diseases reports for the United States and the ADHS data from all Arizona counties. RESULTS: Since 1995, rates of hepatitis B and pertussis have been increasing in Arizona and have become higher for non-Hispanics than Hispanics. In 2005, hepatitis B rates were 1.53* for the United States and 7.31* for Arizona; pertussis rates were 8.72* for the United States and 21.60* for Arizona. CONCLUSION: The results of this study's analysis show the need to improve immunization efforts within the non-Hispanic populations in all Arizona counties. (*Per 100,000 population)

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Yoak, Andrew James. "Disease Control through Fertility Control: Explorations in Two Urban Systems." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1430989186.

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Shipton, Deborah. "Autoimmune disease in rodents : control and specificity." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326005.

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Books on the topic "Disease control"

Gray, Susan Heinrichs. Disease control. Cherry Lake Pub., 2009.

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Strange, Richard N. Plant Disease Control. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-4632-4.

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Behr, Marcel A., Karen Stevenson, and Vivek Kapur, eds. Paratuberculosis: organism, disease, control. CABI, 2020. http://dx.doi.org/10.1079/9781789243413.0000.

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Behr, M. A., and D. M. Collins, eds. Paratuberculosis: organism, disease, control. CABI, 2010. http://dx.doi.org/10.1079/9781845936136.0000.

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Hawker, Jeremy, Norman Begg, Iain Blair, Ralf Reintjes, and Julius Weinberg, eds. Communicable Disease Control Handbook. Blackwell Publishing Ltd, 2005. http://dx.doi.org/10.1002/9780470755129.

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Wallace, Rodrick, Luis Fernando Chaves, Luke R. Bergmann, et al. Clear-Cutting Disease Control. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72850-6.

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Walters, Dale, ed. Disease Control in Crops. Wiley-Blackwell, 2009. http://dx.doi.org/10.1002/9781444312157.

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A, Hadidi, Khetarpal R. K, and Koganezawa H, eds. Plant virus disease control. APS Press, 1998.

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Elwell, H. A. Natural pest & disease control. Natural Farming Network with assistance from the Plant Protection Improvement Programme, 1995.

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1943-, Howe-Grant Mary, ed. Chemotherapeutics and disease control. Wiley, 1993.

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Book chapters on the topic "Disease control"

Walsh, Peter D. "Disease control." In Key Topics in Conservation Biology 2. John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118520178.ch24.

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Viret, Olivier, and Katia Gindro. "Disease Control." In Science of Fungi in Grapevine. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-68663-4_8.

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AbstractFungal disease control in grapevine is essential for the production of high-quality grapes. Regardless of the strategies and modes of production implemented (organic, biodynamic, integrated) and whatever the climatic conditions, growing sensitive Vitis vinifera cultivars depends on the success of disease control. Since the arrival of downy and powdery mildew in Europe over 150 years ago, no alternative to spraying the vines with fungicide at regular intervals has been found. Active ingredients in the fungicides were based on copper and sulphur until the development of newly synthesised molecules in the late 1950s, followed by the release onto the market of several plant protection products. Their regular use raises environmental and human health issues for scientists and practitioners, still searching for more natural and effective alternatives. Despite major research efforts on this front, even naturally occurring molecules used as plant protection products must be applied precisely on the leaves and grape surfaces, requiring the right product, the right dosage and the right timing of the application depending on the biological development of the pathogens and sensitivity of the vine. Success also ultimately depends on the technical performance of the sprayer and its calibration to guarantee efficacy, as well as the protection of the environment and the user.

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Deveaux, Tim. "Disease control." In Bassett’s Environmental Health Procedures. Routledge, 2019. http://dx.doi.org/10.1201/9780429060847-19.

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Lin, Vivian, James Smith, and Sally Fawkes. "Disease control." In Public Health Practice in Australia, 3rd ed. Routledge, 2025. https://doi.org/10.4324/9781003611912-18.

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Meulemans, G. "Control by Vaccination." In Newcastle Disease. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_17.

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Berger, Michael. "Disease risks of obesity." In Weight Control. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0583-5_1.

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Bennejean, G. "Newcastle Disease : Control Policies." In Newcastle Disease. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_16.

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Thornton, Denise H. "Quality Control of Vaccines." In Newcastle Disease. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_19.

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Hahr, Allison J., and Mark E. Molitch. "Glycemic Control." In Diabetes and Kidney Disease. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0793-9_16.

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Krikorian, Armand A., and Angela Pauline P. Calimag. "Glycemic Control." In Diabetes and Kidney Disease. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-86020-2_21.

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Conference papers on the topic "Disease control"

Azaševac, Tijana. "Arterial hypertension and chronic kidney disease - is everything under control?" In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.321a.

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Hypertension is one of the most common diseases that affects the heart and blood vessels. The number of people suffering from chronic kidney disease is increasing, but this disease often remains unrecognized until advanced stages. Kidney diseases can be the causes of arterial hypertension, but this connection is bidirectional and hypertension often leads to the development of chronic kidney disease. Of the non-pharmacological measures, dietary Na+ restriction is particularly effective, while pharmacological treatment of arterial hypertension in these patients should be started with renin–angiotensin system blockers. Second-line therapy may include either a long-acting dihydropyridine calcium channel blocker or diuretics, while third-line therapy is a combination of all three classes of drugs. The thiazide-like diuretic chlorthalidone is effective and can also be used in patients with advanced renal disease and uncontrolled hypertension, as well as in the treatment of resistant hypertension. Sodium-glucose cotransporter type 2 inhibitors and finerenone, although not registered as antihypertensive drugs, have this effect, and also improve renal and cardiovascular function, as well as outcomes of patients with albuminuria. Renal denervation is one of the treatment options for uncontrolled hypertension, but its effect on patients with advanced kidney disease has not yet been sufficiently studied.

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Udalova, Zh V., N. N. Butorina, and S. V. Zinovieva. "STUDY OF THE EFFECT OF AN INSECTICIDAL PREPARATION BASED ON PAECILOMYCES FUMOSOROSEA AGAINST ROOT-KNOT NEMATODE MELOIDOGYNE ARENARIA." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.480-486.

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The efficacy of a commercial insecticidal preparation based on Paecilomyces fumosorosea, Pecilomycin PM116, against root-knot nematode (Meloidogyne arenaria) was evaluated on tomatoes. A comparison was made with biological (Nematophagin) and chemical (Vydate 5G) nematicides. Pecilomycin and Nematophagin did not suppress the development of plants, their weight and size were at the level of healthy controls. The analysis of photosynthetic pigments showed that the chlorophyll b level, the sum of chlorophylls and carotenoids when the root system was treated with Pecilomycin was comparable to healthy controls. Despite the fact that the plants treated with Pecilomycin were completely infected, their infection rate was 1.27 times lower than the controls, and single galls were only present on the roots. The treatment of the roots with Pecilomycin did not significantly affect the number and size of galls; the females in them were at the control level; however, the number of eggs in oothecae was 1.5 times less. Thus, Pecilomycin has an inhibitory effect on the root-knot nematode and a beneficial effect on the tomato plant. Further research and search for new strains of P. fumosorosea with high virulence against one or more pests opens up opportunities for the creation of new bionematicides.

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Sharapova, N. R., F. I. Vasilevich, and V. M. Bachinskaya. "DETERMINATION OF THE EFFICACY OF A PLANT-BASED FEED ADDITIVE AGAINST THE CAUSATIVE AGENT OF CRYPTOSPORIDIOSIS IN CALVES." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2025. https://doi.org/10.31016/978-5-6053355-1-1.2025.26.346-351.

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This article presents a study to identify the efficacy of a plant-based feed additive against the causative agent of cryptosporidiosis in calves, Cryptosporidium parvum and Cryptosporidium muris. Currently, there is a tendency to exacerbate the epizootic situation on cryptosporidiosis, which is associated with intensive cattle breeding technology, animal crowding, lack of timely diagnosis and treatment, as well as high resistance of sporulated oocysts in the external environment. Despite the fact that the main aspects of the cryptosporidiosis issue have been deeply studied, the issues of effective treatment and prevention still remain relevant. Therefore, the search for effective and safe drugs against the pathogens of cryptosporidiosis is of interest not only for fundamental parasitology but also for modern livestock enterprises. The experiment involved 3 groups of animals: positive and negative controls and an experimental group where the tested feed additive was used. The animals in the negative control group and the experimental group were infected on day 5 after the start of the experiment. The experimental group had higher average daily and gross weight gain compared to the negative control. The plant-based feed additive showed its efficacy against the causative agent of cryptosporidiosis in calves.

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"Protection of citrus production areas by regional control in Okinawa Island." In Epidemiology and disease management of citrus HLB disease for sustainable citrus production in ASPAC. Food and Fertilizer Technology Center for the Asian and Pacific Region, 2012. https://doi.org/10.56669/loqz7142.

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Jiarui, Wu. "Systems Biology and Complex Disease." In 2007 Chinese Control Conference. IEEE, 2006. http://dx.doi.org/10.1109/chicc.2006.4346759.

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Vasilevich, F. I., S. A. Shemyakova, and N. V. Esaulova. "VETERINARY AND MEDICAL SIGNIFICANCE OF HORSEFLY (TABANIDAE). REVIEW." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.133-137.

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The article provides information on the harmful effect of horseflies (Diptera, Tabanidae) as a midge component and a vector (carrier) transmitting pathogens of infectious and parasitic diseases including zoonosis. Horseflies are harmful to animals and humans in places of their abundance. Horseflies cause significant economic losses to livestock. With an intense attack of horseflies, individual areas of the skin of animals represent a continuous bleeding surface. Horsefly saliva inserted into a wound at the time of the bite is very toxic and allergenic causing a local inflammatory process and general intoxication of the body. The insects are of particular danger as vectors transmitting pathogens of animals and humans. The role of horseflies in the transmission of tularemia in natural foci of this infection has been proven. The sources of horsefly infection are primarily various small mammals including water rats. Horseflies are equally important as carriers of the anthrax pathogen. Causative agents of Coxiella burnetti infection, blackleg, pasteurellosis, and other infections have been isolated from horseflies. Horseflies are involved in the transmission of Trypanosoma evansi in horses and camels, anaplasmosis in cattle, Theileria cervi in reindeer, and equine infectious anemia virus.

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Ivanov, V. A., and T. N. Sivkova. "EFFECT OF DIROFILARIA REPENS EXTRACT ON HEMATOLOGICAL PARAMETERS OF MICE IN A SINGLE INJECTION." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.189-193.

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Dirofilaria repens infection is widespread in animals in the Russian Federation; however, morphological changes in this disease are not described sufficiently, and carcinogenic effects of helminths to the host organism are not established. We have studied blood changes in mice after a single subcutaneous injection of somatic extract from mature Dirofilaria. The mice were euthanized after 12 hours; blood samples were taken for а complete blood count (CBC) by an automated hematology analyzer and for slides preparation to count a white blood cell differential. In the control group, all the studied parameters were within the reference values with the exception of increased hematocrit. According to the results of the leukogram analysis in mice of the control group, slight lymphocytopenia, and an increase in the number of immature and stab neutrophils were recorded. After a single injection of the D. repens extract, two specimens from the experimental group showed only increased hematocrit, while other animals had leukocytopenia. The count of white blood cells in the experimental group detected neutrophilic leukocytosis with an increase in the number of immature, stab and segmented neutrophils. The detected changes indicate initiated nonspecific immune response.

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Vagin, N. A., N. S. Malysheva, and N. A. Samofalova. "INFECTION OF RODENTS AND INSECTIVORES WITH TRICHINELLA IN THE KURSK REGION." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.117-122.

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The study of the Trichinella distribution in representatives of different systematic taxa is quite relevant as it allows us to establish the role of infected animals in the functioning of the parasitic system of the causative agent of trichinellosis. Scientific works contain contradictory information on the presence of the Trichinella infection in rodents and insectivores. Many scientists publish data on the detection of Trichinella in species of these orders of mammals. There is a large number of works showing the absence of the Trichinella invasion in different species of rodents and insectivores. The purpose of our research was to study the Trichinella infection rate in rodents and insectivores in the Kursk Region. Trichinella larvae were detected by compressor trichinelloscopy and by the method of digesting muscle tissue in artificial gastric juice. Capsule-forming Trichinella (Trichinella spp.) were found in the muscles of infected animals. In total, 7 species of rodents and 5 species of insectivores were studied. The Trichinella invasion was detected in the common vole (1.8%) and in Erinaceus roumanicus (4.2%). The results obtained show that rodents and insectivores are involved in the circulation of Trichinella in the Kursk Region. We believe that representatives of these orders of animals serve as one of the sources of infection for many species of predatory mammals.

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Klimova, E. S., M. E. Mkrtchyan, and T. V. Babintseva. "EFFICIENCY OF SOIL TREATMENT AGAINST CONTAMINATION BY EXOGENOUS PARASITE STAGES." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.209-214.

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The results of many years of research revealed that in the conditions of the Udmurt Republic, in the soil on the territory of the ground runs of livestock enterprises, eggs of nematodes of the suborders Trichocephalata, Strongylata and Rhabdidata, and Eimeria oocysts were found. At the same time, Eimeria oocysts were stationary dominant in terms of quantity per 1 gram of soil (on average, from 204.4±13.9 to 267.4±15.7 pieces). Of the eggs of helminths, exogenous stages of representatives of the suborder Strongylata spp. were most often found, on average 166.1±9.2. In order to assess the effectiveness of soil disinfection on the ground runs, a hot solution of sodium hydroxide (caustic soda) was studied at a 5% concentration at a rate of 1 L/m2 with a 6-hour exposure and a Ligrocid at a 0.25% concentration at a rate of 0.25 L/m2 with 20-min. exposure. The results of our studies showed that when using Ligrocide, the intense-effectiveness of treatment against exogenous stages of helminths from the suborders Strongylata and Rhabdidata was 100%. However, despite its high efficiency, a single treatment does not completely destroy whipworm eggs and Eimeria oocysts. The degree of soil contamination with these parasites remains at the level of 5.6–16.8%. It was found that the soil disinfection on the ground runs of livestock enterprises with 5% sodium hydroxide was ineffective both against protozoan oocysts (intense-effectiveness, 65.6%) and nematode eggs (intense-effectiveness from 20 to 77.8%).

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Bagaeva, U. V., and A. R. Salbieva. "SIGNIFICANCE OF TERRESTRIAL MOLLUSKS IN THE FORMATION OF DICROCELIASIS FOCI IN THE FOOTHILL ZONE OF THE REPUBLIC OF NORTH OSSETIA-ALANIA." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. All-Russian Scientific Research Institute for Fundamental and Applied Parasitology of Animals and Plant – a branch of the Federal State Budget Scientific Institution “Federal Scientific Centre VIEV”, 2023. http://dx.doi.org/10.31016/978-5-6048555-6-0.2023.24.62-66.

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The Republic of North Ossetia Alania remains contaminated by a number of trematode infections including dicroceliasis. The infection rate of cattle is on average 53% in the Republic. Five hundred twenty-six specimens of terrestrial mollusks were collected by the route method in various biocenoses of the foothill zone located at an altitude of 530 – 850 m above sea level. Fourteen species were identified. The dissections revealed that dicroceliasis foci were maintained and function with the involvement of two species of terrestrial mollusks in stock routes and pastures of the studied zone. According to the laboratory studies, infection of the mollusk Helicella derbentina (Kryn.) with the trematode larvae was 5.1%. Five of 98 dissected specimens were infected. The infection rate in Chondrula tridens (Mull.) was 4.6%. During the helminthological study of 87 specimens of this species, dicrocelia larvae were found in 4. According to preliminary data, such species of the studied terrestrial mollusks as Succinea pfeifferi (Rossm.), Cochlicopa lubrica (Műll.), Euomphalia ravergieri (Fer.), Euomphalia selecta (Klika), and Fruticocampylaea narzanensis (Kryn.) identified in stock routes and pastures of the foothill zone do not participate in the development cycle of the trematode Dicrocoelium lanceatum. Further research is required.

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Reports on the topic "Disease control"

Denton-Schneider, Jon, and Eduardo Montero. Disease, Disparities, and Development: Evidence from Chagas Disease Control in Brazil. National Bureau of Economic Research, 2025. https://doi.org/10.3386/w33518.

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Davis, Robert E., Edna Tanne, James P. Prince, and Meir Klein. Yellow Disease of Grapevines: Impact, Pathogen Molecular Detection and Identification, Epidemiology, and Potential for Control. United States Department of Agriculture, 1994. http://dx.doi.org/10.32747/1994.7568792.bard.

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Grapevine yellows diseases characterized by similar symptoms have been reported in several countries including Israel, the United States, France, Italy, Spain, Germany and Australia. These diseases are among the most serious known in grapevine, but precise knowledge of the pathogens' identities and modes of their spread is needed to devise effective control stratgegies. The overall goals of this project were to develop improved molecular diagnostic procedures for detection and identification of the presumed mycoplasmalike organism (MLO) pathogens, now termed phytoplasmas, and to apply these procedures to investigate impact and spread and potential for controlling grapevine yellows diseases. In the course of this research project, increased incidence of grapevine yellows was found in Israel and the United States; the major grapevine yellows phytoplasma in Israel was identified and tis 16S rRNA gene characterized; leafhopper vectors of this grapevine yellows phytoplasma in Israel were identified; a second phytoplasma was discovered in diseased grapevines in Israel; the grapevine yellows disease in the U.S. was found to be distinct from that in Israel; grapevine yellows in Virginia, USA, was found to be caused by two different phytoplasmas; both phytoplasmas in Virginia grapevines were molecularly characterized and classified; commercial grapevines in Europe were discovered to host a phytoplasma associated with aster yellow disease in the USA, but this phytoplasma has not been found in grapevine in the USA; the Australian grapevine yellows phytoplasma was found to be distinct from the grapevine phytoplasmas in Israel, the United States and Europe and was described and named "Candidatus phytoplasma australiense", and weed host plants acting as potential reservoirs of the grapevine phytoplasmas were discovered. These and other findings from the project should aid in the design and development of strategies for managing the grapevine yellows disease problem.

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Kamp, J. A. L. M., A. van der Meer, G. Polder, M. Afonso, and J. van der Wolf. AGROS disease control seed potatoes : report 2020-2023. Wageningen Plant Research, 2024. https://doi.org/10.18174/656676.

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Salerno, Reynolds Mathewson, and William Kessler. Infectious Disease Detection and Control in the Developing World. Office of Scientific and Technical Information (OSTI), 2014. http://dx.doi.org/10.2172/1163498.

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Asquith, Brian J. Rent Control - Is the Cure Worse Than the Disease? W.E. Upjohn Institute, 2019. http://dx.doi.org/10.17848/pb2019-15.

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McMillan, Margaret, William Masters, and Harounan Kazianga. Disease Control, Demographic Change and Institutional Development in Africa. National Bureau of Economic Research, 2013. http://dx.doi.org/10.3386/w19245.

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Doorenbos, Amanda. Understanding Malaria and Host Cell Interactions for Disease Control. Iowa State University, 2023. http://dx.doi.org/10.31274/cc-20240624-1530.

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Morck, Randall, David Stangeland, and Bernard Yeung. Inherited Wealth, Corporate Control and Economic Growth: The Canadian Disease. National Bureau of Economic Research, 1998. http://dx.doi.org/10.3386/w6814.

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Khawar, Laila. Video: Infectious disease control, elimination and eradication: what's the difference? Edited by Michael Joiner and Sam Hendricks. Monash University, 2024. http://dx.doi.org/10.54377/2915-3b09.

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Kumar, Aishani, Thendral Yalini, and Sunil Kumar C. Unlocking Cellular Control: The Promise of PROTACs in Disease Intervention. Science Reviews - Biology, 2024. http://dx.doi.org/10.57098/scirevs.biology.3.2.1.

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The discovery of proteolysis-targeting chimeras (PROTACs) is among the most exciting and promising avenues in cancer therapy. These fascinating compounds signify a paradigm shift from traditional approaches to medication development, offering a new idea that leverages the complexities of biological mechanisms to accomplish highly focused degradation of particular proteins implicated in pathological processes. This novel strategy has the potential to address a number of drawbacks with conventional therapy techniques, such as the development of drug resistance and unexpected adverse effects resulting from interactions that are not intended. The fundamental attraction of PROTACs is their distinct mode of action, which is based on controlling the cell's own machinery for protein degradation. This orchestrated degradation translates to a substantial reduction in the levels of disease-driving proteins, often leading to the disruption of critical pathways involved in cancer growth and progression. The in-depth principles underlying PROTAC technology are thoroughly explored in this review study, which also provides insight into the complex chemical mechanisms that enable these chimeric molecules to specifically degrade certain proteins while leaving others intact. Showcasing the potential of PROTACs as a revolutionary force in targeted cancer therapy, and focusing on its application in prostate and breast cancer especially, the article draws from a comprehensive compilation of preclinical and clinical studies, advancements, and breakthroughs in the field. The methods used to create and refine PROTACs for various cancer types will be examined throughout the review, along with the subtleties of the ligand and linker choices that are crucial to their effectiveness and selectivity. The difficulties and possibilities of transferring this ground-breaking technology from the lab to clinical practice will also be thoroughly examined, with an emphasis on issues like bioavailability, administration strategies, and potential resistance mechanisms. Through the integration of perspectives from various studies, the objective is to present a thorough but succinct review of the state of ongoing PROTAC research, emphasizing both, noteworthy advancements and the important issues that still need to be resolved. In the end, our investigation into PROTACs aims to shed light on how they can change the face of cancer therapy by providing a preview of a day when targeted protein degradation of disease-causing proteins would lead the way in novel therapeutic approaches.

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