Relevant bibliographies by topics / Disease control

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Disease control'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Disease control"

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Rajagopalan, P. K. "Aspects of Vector Borne Disease Control." Journal of Communicable Diseases 50, no. 01 (March 29, 2018): 28–31. http://dx.doi.org/10.24321/0019.5138.201806.

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Sutthisa, W. "Biological Control Properties of Cyathus spp. to Control Plant Disease Pathogens." Journal of Pure and Applied Microbiology 12, no. 4 (December 30, 2018): 1755–60. http://dx.doi.org/10.22207/jpam.12.4.08.

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Sharma, SN. "Kyasanur Forest Disease: Vector Surveillance and its Control." Journal of Communicable Diseases 51, no. 02 (August 28, 2019): 38–44. http://dx.doi.org/10.24321/0019.5138.201915.

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MILNER, FABIO AUGUSTO. "Disease Control." Mathematical Population Studies 17, no. 2 (April 26, 2010): 67–68. http://dx.doi.org/10.1080/08898481003689403.

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Marriott, Gill. "Communicable disease control." Primary Health Care 11, no. 1 (February 2001): 17. http://dx.doi.org/10.7748/phc.11.1.17.s16.

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Woodhouse, S. "Communicable disease control." BMJ 321, no. 7254 (July 15, 2000): 2. http://dx.doi.org/10.1136/bmj.321.7254.s2-7254.

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Sutton, Turner B. "Plant disease control." Crop Protection 14, no. 3 (May 1995): 261. http://dx.doi.org/10.1016/0261-2194(95)90006-3.

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Williams, Jane. "Infectious disease control." Equine Health 2012, no. 3 (January 26, 2012): 32–33. http://dx.doi.org/10.12968/eqhe.2012.1.3.32.

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Galbraith, N. S. "Infectious disease control." BMJ 300, no. 6738 (June 9, 1990): 1477–78. http://dx.doi.org/10.1136/bmj.300.6738.1477.

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Schofield, C. J. "Appropriate disease control." Parasitology Today 1, no. 6 (December 1985): 181–82. http://dx.doi.org/10.1016/0169-4758(85)90181-4.

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Dissertations / Theses on the topic "Disease control"

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Franco, Iborra Sandra. "Mitochondrial quality control in neurodegenerative diseases: focus on Parkinson’s disease and Huntington’s disease." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/565668.

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Darrerament s’han produït avanços importants que han contribuït al coneixement dels mecanismes de disfunció cel·lular i mort en la malaltia de Parkinson (MP) i en la malaltia de Huntington (MH). Ambdues malalties són trastorns del moviment que es caracteritzen per la pèrdua específica de neurones dels ganglis basals, les neurones dopaminèrgiques de la substància nigra (SN), en el cas de la MP i les neurones espinoses de l’estriat, en el cas de la MH. Malgrat les diferències, ambdues comparteixen processos patològics comuns com la presència de proteïnes malplegades, l’estrés oxidatiu i disfunció mitocondrial. La mitocòndria és la font d’energia principal en les cèl·lules eucariotes, però també és un orgànul dinàmic relacionat amb una gran quantitat de processos cel·lulars. La disrupció de la homeòstasis mitocondrial i la subseqüent disfunció mitocondrial juguen un paper important en la patofisiologia de les malalties neurodegeneratives. El manteniment de la integritat mitocondrial a través de diferents mecanismes de control és crític per a la superviviència neuronal. Aquesta tesi es centra en l’estudi dels mecanismes de control de qualitat mitocondrial en la MP i la MH, per tal d’entendre millor els mecanismes que duen a la mort cel·lular. En el primer capítol, he estudiat el transport de proteïnes a la mitocòndria en models in vitro i in vivo de la MP. In vitro, la inhibició del complexe I produeix una alteració del transport de proteïnes a la mitocòndria així com una disminució dels nivells de proteïnes OXPHOS, acumulació de proteïnes agregades i disminució dels nivells de chaperones mitocondrials. Per tal de restablir el transport de proteïnes mitocondrials es van sobreexpressar dos components clau del sistema de translocases: la translocasa de la membrana externa 20 (TOM20) i la translocasa de la membrana interna 23 (TIM23). La sobreexpressió in vitro de TOM20 i TIM23 va restaurar el transport de proteïnes mitocondrials i va alleugerar la disfunció mitocondrial i la mort cel·lular. La inhibició del complexe I en ratolins també dóna lloc a una alteració del transport de proteïnes mitocondrials i produeix neurodegeneració del sistema dopaminèrgic. La sobreexpressió de TIM23 va restaurar parcialment el transport de proteïnes i va protegir lleugerament les neurones dopaminèrgiques de la SN. En canvi, la sobreexpressió de TOM20 va ser incapaç de millorar el transport de proteïnes mitocondrials i, fins i tot, va exacerbar la mort cel·lular. Aquests resultats posen de relleu el paper de la disfunció del transport de proteïnes mitocondrials, en particular de dos dels seus components, en la patogènesis de la MP i suggereixen la necessitat de futurs estudis es centrin en altres elements d’aquest sistema. En el segon capítol, he estudiat el paper de la proteïna huntingtina en la mitofàgia i com la seva mutació, que dóna lloc a una expansió de glutamines, pot afectar a aquesta funció. Per a tal fi, he treballat en un model in vitro de cèl·lules estriatals ST-Q7 (control) i ST-Q111 (mutant). En condicions fisiològiques, la mitofàgia induïda no es troba mitjançada pel reclutament de parkin als mitocondris despolaritzats. La huntingtina mutada afecta la mitofàgia induïda a través de l’alteració de la seva funció de scaffold en diferents passos del procés de mitofàgia: (i) activació d’ULK1 a través de l’alliberament de mTORC1, (ii) formació del complexe Beclin 1-Vps15,(iii) interacció dels adaptadors de mitofàgia OPTN i NDP52 amb huntingtina i, (iv) amb LC3. Com a resultat, els mitocondris de les cèl·lules ST-Q111 estan més danyats i tenen una respiració mitocondrial deficient. Aquests resultats demostren la presència d’una alteració en la mitofàgia com un mecanisme lligat a la MH. En conclusió, el descobriment de noves dianes mitocondrials en la MP i MH emfatitza el paper important que juga el control de qualitat mitocondrial en la neurodegeneració.
In the past years, several important advances have expanded our understanding of the pathways that lead to cell dysfunction and death in Parkinson’s disease (PD) and Huntington’s disease (HD). Both diseases are movement disorders characterized by the loss of a specific subset of neurons within the basal ganglia, dopaminergic neurons in the substantia nigra pars compacta (SNpc), in the case of PD, and medium spiny neurons in the striatum, in the case of HD,. Despite distinct clinical and pathological features, these two neurodegenerative disorders share critical underlying pathogenic mechanisms such as the presence of misfolded and/or aggregated proteins, oxidative stress and mitochondrial anomalies. Mitochondria are the prime energy source in most eukaryotic cells, but these highly dynamic organelles are also involved in a multitude of cellular events. Disruption of mitochondrial homeostasis and the subsequent mitochondrial dysfunction plays a key role in the pathophysiology of neurodegenerative diseases. Therefore, maintenance of mitochondrial integrity through different surveillance mechanisms is critical for neuronal survival. In this thesis I have studied in depth some mitochondrial quality control mechanisms in the context of PD and HD, in order to broaden the knowledge about the pathomechanisms leading to cell death. In the first chapter I have studied mitochondrial protein import in in vitro and in vivo models of PD. In vitro, complex I inhibition, a characteristic pathological hallmark in PD, impaired mitochondrial protein import. This was associated with OXPHOS protein downregulation, accumulation of aggregated proteins inside mitochondria and downregulation of mitochondrial chaperones. Therefore, we aimed to reestablish the mitochondrial protein import by overexpressing two key components of the system: translocase of the outer membrane 20 (TOM20) and translocase of the inner membrane 23 (TIM23). Overexpression of TOM20 and TIM23 in vitro restored protein import into mitochondria and ameliorated mitochondrial dysfunction and cell death. Complex I inhibition also impaired mitochondrial protein import and led to dopaminergic neurodegeneration in vivo. Overexpression of TIM23 partially rescued protein import into mitochondria and slightly protected dopaminergic neurons in the SNpc. On the contrary, TOM20 overexpression did not rescue protein import into mitochondria and exacerbated neurodegeneration in both SNpc and striatum. These results highlight mitochondrial protein import dysfunction and the distinct role of two of their components in the pathogenesis of PD and suggest the need for future studies to target other elements in the system. In the second chapter, I have studied the role of huntingtin in mitophagy and how the polyglutamine expansion present in mutant huntingtin can affect its function. For such, I worked with differentiated striatal ST-Q7 (as control) and ST-Q111 (as mutant) cells, expressing full length huntingtin. In these conditions, induced mitophagy was not mediated by Parkin recruitment into depolarized mitochondria. Mutant huntingtin impaired induced mitophagy by altering wildtype huntingtin scaffolding activity at different steps of mitophagy process: (i) ULK1 activation through its release from the mTORC1, (ii) Beclin1-Vps15 complex formation, (iii) interaction of the mitophagy adapters OPTN and NDP52 with huntingtin and (iv) with LC3. As a result, mitochondria from ST-Q111 cells exhibited increased damage and altered mitochondrial respiration. These results uncover impaired mitophagy as a potential pathological mechanism linked with HD. In conclusion, we have discovered new mitochondrial targets for PD and HD emphasizing the important role that mitochondrial quality control plays in neurodegeneration
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Du, Toit Eben Francois. "Pinning control of disease networks." Thesis, University of Pretoria, 2015. http://hdl.handle.net/2263/56133.

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The modelling of contagion spread on contact networks provide valuable insights to epidemiologists and policymakers trying to control and eradicate diseases. This thesis proposes, implements and analyses a methodology for inserting disease contact networks of HIV into feedback control loops and applying open-loop pinning control to their nodes. Pinning control aims to medicate only a portion of an entire network in order to achieve the same outcomes that would be seen when all nodes are controlled. The control loops are simulated using networks ranging from size N = 100 nodes to N = 10000 nodes. Simulations aim to control the average maximum incidence in the networks by first estimating the reference average transmissibility from the statistical physics technique known as bond percolation. Once the average transmissibility is known, node-, network- and population mass-action models can be measured for incidence. Two selective pinning control strategies, namely proportional feedback and nonlinear model predictive control (NMPC), are compared with one another and also with a random pinning strategy. The budget, measured in quality-adjusted life years (QALYs), is added to the cost-function for NMPC control. It is shown that budget can indeed be controlled while incidence varies, while incidence may be controlled as budget varies. Pinning control of disease networks is a feasible methodology to analyse the future and steady-state outcomes of interventions in fast-spreading (high-risk) disease contact networks.
Modellering van die verspreiding van siektes oor kontak-netwerke verskaf waardevolle inligting aan beleidmakers en epidemioloë wat besluit op maatreëls vir voorkoming teen die siekte. Hierdie proefskrif hou n metode voor wat gebruik word om siekteverspreidings-netwerke te simuleer en te analiseer. Dit word gedoen op netwerke met nodusse wat varieer tussen N = 100 en N = 10000. Netwerke waarin HIV versprei word gebruik. Penbeheer word in n oopluskonfigurasie op elke nodus toegepas binne n geslote terugvoerlus op netwerkvlak. Penbeheer se doel is om slegs sekere nodusse te beheer om dieselfde uitkomste vir die voorkoms van HIV tydens n epidemie te meet. Die doel is om die gemiddelde waarskynlikheid vir oordrag van die siekte tussen nodusse te beheer en sodoende, deur middel van die tegniek genaamd bond percolation , te bepaal hoe groot die finale epidemie gaan wees. Sodra die gemiddelde waarskynlikheid bekend is, kan nodus-, netwerk- en populasiemodelle saamgestel word. Twee selektiewe penbeheer-strategieë (proporsioneel, en NMPC) word met mekaar en met n derde willekeurige tegniek vergelyk. Die beheer van begrotings, gemeet in quality-adjusted life years (QALYs), word deur die NMPC strategie hanteer. Siektes binne kontaknetwerke kan dus beheer word met selektiewe penbeheer. Penbeheer-strategieë word ook vergelyk op grond van die dosisse wat hulle benodig, asook die akkuraatheid van die bestendigde-toestand resultate. Penbeheer van siekteverspreidings-netwerke is n werkbare metode om toekomstige en bestendigde-toestand uitkomste van mediese ingrepe op netwerke mee te analiseer.
Thesis (PhD)--University of Pretoria, 2015.
tm2016
Electrical, Electronic and Computer Engineering
PhD
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Hillman, Anne M. "Perceived control in the everyday occupational roles of people with Parkinson's disease and their partners." Connect to full text, 2006. http://hdl.handle.net/2123/1621.

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Thesis (Ph. D.)--University of Sydney, 2006.
Title from title screen (viewed May 1, 2007). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Occupation and Leisure Sciences. Includes bibliographical references. Also issued in print.
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Bennett, Richard. "The economics of livestock disease control." Thesis, University of Reading, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.316155.

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Cox, Mark. "Ecological economics of wildlife disease control." Thesis, University of York, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.310898.

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Chochó, Karen S. "Hispanic migrants and cross-border disease control of Arizona's vaccine preventable diseases." restricted, 2008. http://etd.gsu.edu/theses/available/etd-04222008-151047/.

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Thesis (M.Ph.)--Georgia State University, 2008.
Title from file title page. Richard Rothenberg, committee chair; Russ Toal, Karen E. Gieseker, committee members. Electronic text (135 p. : col. ill.) : digital, PDF file. Description based on contents viewed August 12, 2008. Includes bibliographical references (p. 127-135).
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Chocho, Karen. "Hispanic Migrants and Cross-border Disease Control of Arizona's Vaccine Preventable Diseases." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/iph_theses/35.

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BACKGROUND: According to the Centers for Disease Control and Prevention and the National Immunization Program, there is an increase in the re-emergence of past diseases. Even with mandatory vaccination practices in the United States, there are still a number of cases of vaccine-preventable diseases (VPDs) reported yearly. It is speculated that the re-emergence of VPDs is in part due to the increase in international travel as well as the influx of immigrants. One particular group of interest includes the Hispanic migrants coming from Central and South America where some of these diseases are endemic. OBJECTIVE: The purpose of this paper is to determine the extent of VPD cases in the border state of Arizona that may be attributed to Hispanic migrant influx using data from the MMWR: Summary of Notifiable Diseases reports for the United States and the ADHS data from all Arizona counties. RESULTS: Since 1995, rates of hepatitis B and pertussis have been increasing in Arizona and have become higher for non-Hispanics than Hispanics. In 2005, hepatitis B rates were 1.53* for the United States and 7.31* for Arizona; pertussis rates were 8.72* for the United States and 21.60* for Arizona. CONCLUSION: The results of this study's analysis show the need to improve immunization efforts within the non-Hispanic populations in all Arizona counties. (*Per 100,000 population)
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Cattaneo, Gabriele 1984. "Bilingual language control : Evidence from Parkinson's disease." Doctoral thesis, Universitat Pompeu Fabra, 2017. http://hdl.handle.net/10803/482046.

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How can bilinguals easily and flexibly switch between languages without errors? The aim of this thesis is to advance our knowledge about the control mechanisms involved in bilingual language production. I investigated this issue in two ways. Firstly, by exploring similarities between bilingual language control (bLC) and domain-general executive control (EC). Secondly, by investigating the specificity of bLC mechanisms within the linguistic domain. To do so, I measured behavioural responses of Catalan/Spanish bilinguals affected by Parkinson’s disease and healthy controls in tasks tapping bLC, EC and language control, when only one language was involved. I explored Parkinson’s disease individuals because this neurodegenerative disorder affects brain areas involved in both domain-general EC and the bLC. The findings of this dissertation suggest that some bLC mechanisms are highly specific and are not involved in other linguistic or non-linguistic control contexts. However, some other mechanisms of bLC are shared across different domains of control and implemented by EC mechanisms. These results extend previous knowledge about the nature of mechanisms involved in bLC and are useful for future models of bilingual language production.
Com ho fan els bilingües per passar d’una llengua a una altra amb facilitat, flexibilitat i sense cometre errors? L’objectiu d’aquesta tesi és avançar el nostre coneixement sobre els mecanismes de control involucrats en la producció de llenguatge en bilingües. He investigat aquest tema de dues maneres: Primer: he explorat les similituds entre el control bilingüe del llenguatge i el control executiu de domini general. Segon: he investigat l’especificitat dels mecanismes de control bilingüe dins del domini lingüístic. Per aquest supòsit, he avaluat les respostes conductuals de bilingües Català/Castellà afectats per la malaltia del Parkinson i controls bilingües (no malalts) en tasques de control de llenguatge bilingüe, control executiu i control de llenguatge quan una sola llengua hi està involucrada. He estudiat aquesta malaltia degenerativa perquè afecta zones del cervell involucrades tant en control executiu de domini general, com en control bilingüe del llenguatge. Els descobriments d’aquesta dissertació suggereixen que alguns dels mecanismes de control bilingüe del llenguatge són altament específics, i no estan involucrats en altres contextos de control lingüístic o no lingüístic. D’altra banda, altres mecanismes de control estan compartits a través de diferents dominis i estan implementats per mecanismes de control executiu.
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Yoak, Andrew James. "Disease Control through Fertility Control: Explorations in Two Urban Systems." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1430989186.

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Shipton, Deborah. "Autoimmune disease in rodents : control and specificity." Thesis, University of Oxford, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.326005.

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Books on the topic "Disease control"

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Gray, Susan Heinrichs. Disease control. Ann Arbor, MI: Cherry Lake Pub., 2009.

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Strange, Richard N. Plant Disease Control. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-4632-4.

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Behr, Marcel A. Paratuberculosis: Organism, disease, control. Wallingford, UK: CABI, 2010.

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Elwell, H. A. Natural pest & disease control. Harare, Zimbabwe: Natural Farming Network with assistance from the Plant Protection Improvement Programme, 1995.

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1943-, Howe-Grant Mary, ed. Chemotherapeutics and disease control. New York: Wiley, 1993.

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Behr, Marcel A., Karen Stevenson, and Vivek Kapur, eds. Paratuberculosis: organism, disease, control. Wallingford: CABI, 2020. http://dx.doi.org/10.1079/9781789243413.0000.

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Behr, M. A., and D. M. Collins, eds. Paratuberculosis: organism, disease, control. Wallingford: CABI, 2010. http://dx.doi.org/10.1079/9781845936136.0000.

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Hawker, Jeremy, Norman Begg, Iain Blair, Ralf Reintjes, and Julius Weinberg, eds. Communicable Disease Control Handbook. Malden, Massachusetts, USA: Blackwell Publishing Ltd, 2005. http://dx.doi.org/10.1002/9780470755129.

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Wallace, Rodrick, Luis Fernando Chaves, Luke R. Bergmann, Constância Ayres, Lenny Hogerwerf, Richard Kock, and Robert G. Wallace. Clear-Cutting Disease Control. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-72850-6.

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Walters, Dale, ed. Disease Control in Crops. Oxford, UK: Wiley-Blackwell, 2009. http://dx.doi.org/10.1002/9781444312157.

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Book chapters on the topic "Disease control"

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Walsh, Peter D. "Disease control." In Key Topics in Conservation Biology 2, 452–66. Oxford: John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118520178.ch24.

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Deveaux, Tim. "Disease control." In Bassett’s Environmental Health Procedures, 475–507. Ninth edition. | Abingdon, Oxon; New York, NY : Routledge, [2020]: Routledge, 2019. http://dx.doi.org/10.1201/9780429060847-19.

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Meulemans, G. "Control by Vaccination." In Newcastle Disease, 318–32. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_17.

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Berger, Michael. "Disease risks of obesity." In Weight Control, 1–7. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0583-5_1.

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Bennejean, G. "Newcastle Disease : Control Policies." In Newcastle Disease, 303–17. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_16.

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Thornton, Denise H. "Quality Control of Vaccines." In Newcastle Disease, 347–65. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-1759-3_19.

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Hahr, Allison J., and Mark E. Molitch. "Glycemic Control." In Diabetes and Kidney Disease, 205–14. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0793-9_16.

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Krikorian, Armand A., and Angela Pauline P. Calimag. "Glycemic Control." In Diabetes and Kidney Disease, 443–67. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-86020-2_21.

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Liu, Xinzhi, and Peter Stechlinski. "Switching Control Strategies." In Infectious Disease Modeling, 135–78. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53208-0_5.

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Liu, Xinzhi, and Peter Stechlinski. "Pulse Control Strategies." In Infectious Disease Modeling, 179–226. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53208-0_6.

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Conference papers on the topic "Disease control"

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Jiarui, Wu. "Systems Biology and Complex Disease." In 2007 Chinese Control Conference. IEEE, 2006. http://dx.doi.org/10.1109/chicc.2006.4346759.

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Aness, Ansari Fatima, Renita Fernandes, Bilal Shaikh, Kamran Shaikh, and Nida Shaikh. "Leaf Disease Detection." In 2022 6th International Conference On Computing, Communication, Control And Automation (ICCUBEA). IEEE, 2022. http://dx.doi.org/10.1109/iccubea54992.2022.10010873.

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Marsden, PA, B. Ibrahim, AA Woodcock, SJ Fowler, and JA Smith. "Cough and Disease Control in Asthma." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5757.

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Stengel, R. F., R. Ghigliazza, N. Kulkarni, and O. Laplace. "Optimal control of a viral disease." In Proceedings of American Control Conference. IEEE, 2001. http://dx.doi.org/10.1109/acc.2001.946229.

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Pare, Philip E., Ji Liu, Carolyn L. Beck, and Tamer Basar. "Networked Infectious Disease–Contaminated Water Model." In 2019 18th European Control Conference (ECC). IEEE, 2019. http://dx.doi.org/10.23919/ecc.2019.8795741.

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Kim, Sehjeong, and Dong Eui Chang. "Border screening vs. community level disease control for infectious diseases: Timing and effectiveness." In APPLIED MATHEMATICS AND COMPUTER SCIENCE: Proceedings of the 1st International Conference on Applied Mathematics and Computer Science. Author(s), 2017. http://dx.doi.org/10.1063/1.4981958.

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Ashamiss, Adel A., Mahmoud A. Hussien, and Roger E. Salters. "Blood flow and brain cell region models for Alzheimer's disease." In Control (MSC). IEEE, 2011. http://dx.doi.org/10.1109/cca.2011.6044365.

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Elwefati, Shada E., Ahmed J. Abougarair, and Moftah M. Bakush. "Control of Epidemic Disease Based Optimization Technique." In 2021 IEEE 1st International Maghreb Meeting of the Conference on Sciences and Techniques of Automatic Control and Computer Engineering MI-STA. IEEE, 2021. http://dx.doi.org/10.1109/mi-sta52233.2021.9464453.

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Vijayshankar, Arun, and Sandip Roy. "Cost of fairness in disease spread control." In 2012 IEEE 51st Annual Conference on Decision and Control (CDC). IEEE, 2012. http://dx.doi.org/10.1109/cdc.2012.6426109.

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Meng, Jiao, Xin Huo, Hui Zhao, Liming Zhang, Xun Wang, and Yang Wang. "Image-based Handwriting Analysis for Disease Diagnosis." In 2022 41st Chinese Control Conference (CCC). IEEE, 2022. http://dx.doi.org/10.23919/ccc55666.2022.9902136.

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Reports on the topic "Disease control"

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Salerno, Reynolds Mathewson, and William Kessler. Infectious Disease Detection and Control in the Developing World. Office of Scientific and Technical Information (OSTI), October 2014. http://dx.doi.org/10.2172/1163498.

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Asquith, Brian J. Rent Control - Is the Cure Worse Than the Disease? W.E. Upjohn Institute, August 2019. http://dx.doi.org/10.17848/pb2019-15.

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McMillan, Margaret, William Masters, and Harounan Kazianga. Disease Control, Demographic Change and Institutional Development in Africa. Cambridge, MA: National Bureau of Economic Research, July 2013. http://dx.doi.org/10.3386/w19245.

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Davis, Robert E., Edna Tanne, James P. Prince, and Meir Klein. Yellow Disease of Grapevines: Impact, Pathogen Molecular Detection and Identification, Epidemiology, and Potential for Control. United States Department of Agriculture, September 1994. http://dx.doi.org/10.32747/1994.7568792.bard.

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Grapevine yellows diseases characterized by similar symptoms have been reported in several countries including Israel, the United States, France, Italy, Spain, Germany and Australia. These diseases are among the most serious known in grapevine, but precise knowledge of the pathogens' identities and modes of their spread is needed to devise effective control stratgegies. The overall goals of this project were to develop improved molecular diagnostic procedures for detection and identification of the presumed mycoplasmalike organism (MLO) pathogens, now termed phytoplasmas, and to apply these procedures to investigate impact and spread and potential for controlling grapevine yellows diseases. In the course of this research project, increased incidence of grapevine yellows was found in Israel and the United States; the major grapevine yellows phytoplasma in Israel was identified and tis 16S rRNA gene characterized; leafhopper vectors of this grapevine yellows phytoplasma in Israel were identified; a second phytoplasma was discovered in diseased grapevines in Israel; the grapevine yellows disease in the U.S. was found to be distinct from that in Israel; grapevine yellows in Virginia, USA, was found to be caused by two different phytoplasmas; both phytoplasmas in Virginia grapevines were molecularly characterized and classified; commercial grapevines in Europe were discovered to host a phytoplasma associated with aster yellow disease in the USA, but this phytoplasma has not been found in grapevine in the USA; the Australian grapevine yellows phytoplasma was found to be distinct from the grapevine phytoplasmas in Israel, the United States and Europe and was described and named "Candidatus phytoplasma australiense", and weed host plants acting as potential reservoirs of the grapevine phytoplasmas were discovered. These and other findings from the project should aid in the design and development of strategies for managing the grapevine yellows disease problem.
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Morck, Randall, David Stangeland, and Bernard Yeung. Inherited Wealth, Corporate Control and Economic Growth: The Canadian Disease. Cambridge, MA: National Bureau of Economic Research, November 1998. http://dx.doi.org/10.3386/w6814.

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Haider, Huma. Malaria, HIV and TB in Mozambique: Epidemiology, Disease Control and Interventions. Institute of Development Studies, January 2022. http://dx.doi.org/10.19088/k4d.2022.035.

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Malaria, HIV and tuberculosis (TB) are significant public health concerns in Mozambique. Malaria was the fourth leading cause of death in the country in 2019, accounting for 42% of deaths among children under 5 years of age (Mugabe et al., 2021; USAID, 2018). Mozambique is among the top eight countries with the highest HIV prevalence; with the second highest mother-to-child transmission (MTCT) rate in the world (Fuente-Soro et al., 2021; Nacarapa et al., 2021). The incidence of TB is rising, with pediatric TB cases almost tripling in recent years (WHO, 2020b; Nguenha et al., 2018; Orlando et al., 2018). Mozambique has one of the highest global incidence of malaria-HIV and TB-HIV co-infection, which raises the likelihood of poor clinical outcomes (Moon et al., 2019; USAID, 2018). This rapid literature review highlights key aspects of the epidemiology of malaria, HIV and TB in Mozambique and challenges in prevention, detection and treatment; and surveys select interventions that seek to address these challenges. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.
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Haider, Huma. Malaria, HIV and TB in Mozambique: Epidemiology, Disease Control and Interventions. Institute of Development Studies, January 2022. http://dx.doi.org/10.19088/k4d.2022.035.

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Malaria, HIV and tuberculosis (TB) are significant public health concerns in Mozambique. Malaria was the fourth leading cause of death in the country in 2019, accounting for 42% of deaths among children under 5 years of age (Mugabe et al., 2021; USAID, 2018). Mozambique is among the top eight countries with the highest HIV prevalence; with the second highest mother-to-child transmission (MTCT) rate in the world (Fuente-Soro et al., 2021; Nacarapa et al., 2021). The incidence of TB is rising, with pediatric TB cases almost tripling in recent years (WHO, 2020b; Nguenha et al., 2018; Orlando et al., 2018). Mozambique has one of the highest global incidence of malaria-HIV and TB-HIV co-infection, which raises the likelihood of poor clinical outcomes (Moon et al., 2019; USAID, 2018). This rapid literature review highlights key aspects of the epidemiology of malaria, HIV and TB in Mozambique and challenges in prevention, detection and treatment; and surveys select interventions that seek to address these challenges. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.
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Haider, Huma. Malaria, HIV and TB in Mozambique: Epidemiology, Disease Control and Interventions. Institute of Development Studies, January 2022. http://dx.doi.org/10.19088/k4d.2022.035.

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Malaria, HIV and tuberculosis (TB) are significant public health concerns in Mozambique. Malaria was the fourth leading cause of death in the country in 2019, accounting for 42% of deaths among children under 5 years of age (Mugabe et al., 2021; USAID, 2018). Mozambique is among the top eight countries with the highest HIV prevalence; with the second highest mother-to-child transmission (MTCT) rate in the world (Fuente-Soro et al., 2021; Nacarapa et al., 2021). The incidence of TB is rising, with pediatric TB cases almost tripling in recent years (WHO, 2020b; Nguenha et al., 2018; Orlando et al., 2018). Mozambique has one of the highest global incidence of malaria-HIV and TB-HIV co-infection, which raises the likelihood of poor clinical outcomes (Moon et al., 2019; USAID, 2018). This rapid literature review highlights key aspects of the epidemiology of malaria, HIV and TB in Mozambique and challenges in prevention, detection and treatment; and surveys select interventions that seek to address these challenges. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.
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Haider, Huma. Malaria, HIV and TB in Nigeria: Epidemiology and Disease Control Challenges. Institute of Development Studies (IDS), December 2021. http://dx.doi.org/10.19088/k4d.2022.040.

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Nigeria has the world’s highest number of people affected by malaria and the world’s second largest human immunodeficiency virus (HIV)/AIDS burden. There is a high occurrence of co-infection of malaria in HIV patients (Gumel et al., 2021). Nigeria is also ranked as one of the thirty high tuberculosis (TB) and TB-HIV co-infection burden countries in the world (Odume et al., 2020, 8). Co-infection can make each disease more severe and potentially more infectious (Gumel et al., 2021; Jemikalajah et al., 2021; Chukwuocha et al., 2019). This rapid literature review highlights key aspects of the epidemiology of malaria, HIV and TB in Nigeria, in addition to challenges in controlling the three diseases, in terms of prevention, detection and treatment. This is part of a series of reports looking into Epidemiology of Malaria, human immune deficiency virus (HIV) and tuberculosis (TB) across a set of African Nations.
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Anders, Katie. New tools needed to control the spread of mosquito-borne disease. Monash University, April 2022. http://dx.doi.org/10.54377/8fc0-e89d.

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