Relevant bibliographies by topics / Digallic acid

Academic literature on the topic 'Digallic acid'

Author: Grafiati
Published: 26 July 2024
Last updated: 27 July 2024

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Journal articles on the topic "Digallic acid"

1

Verzele, M., P. Delahaye, and J. van Dijck. "Digallic Acid." Bulletin des Sociétés Chimiques Belges 92, no. 2 (September 1, 2010): 181–86. http://dx.doi.org/10.1002/bscb.19830920212.

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2

Куркин (Kurkin), Владимир (Vladimir) Александрович (Аleksandrovich), Татьяна (Tat'yana) Константиновна (Konstantinovna) Рязанова (Ryazanova), Александр (Аleksandr) Викторович (Viktorovich) Жестков (Zhestkov), Артем (Аrtem) Викторович (Viktorovich) Лямин (Lyamin), Елена (Elena) Владимировна (Vladimirovna) Авдеева (Avdeeva), Анна (Аnna) Владимировна (Vladimirovna) Куркина (Kurkina), Ольга (Ol'ga) Евгеньевна (Evgen'evna) Правдивцева (Pravdivtseva), and Альберт (Аl'bert) Иванович (Ivanovich) Агапов (Agapov). "THE ANTIBACTERIAL ACTIVITY OF COMPOUNDS FROM LEAVES OF ARCTOSTAPHYLOS UVA-URS." chemistry of plant raw material, no. 3 (February 7, 2018): 53–60. http://dx.doi.org/10.14258/jcprm.2018033542.

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The aim of this paper is the isolation of individual compounds, which are caused the antibacterial activity of the leaves of the bearberry [Arctostaphylos uva-ursi (L.) Spreng.]. The leaves of Arctostaphylos uva-ursi, collected in Perm region, there were extracted with 70% ethanol, the obtained water-alcoholic infusion there was evaporated in vacuum.By means of the chromatographic methods with the using of silica gel 40/100 and eluent systems (chloroform and ethanol in several ratio) from the evaporated water-alcoholic extract of the leaves of Arctostaphylos uva-ursi, a substance with antibacterial activity, ethyl ester of p-digallic acid, which is a new natural compound, was isolated along with arbutin ((1-О-b-D-glucopyranoside of hydroquinone) from the leaves of this plant. The chemical structures of the ethyl ester of p-digallic acid and arbutin were established with the using of data of 1H-NMR-spectroscopy, UV-spectroscopy and mass-spectrometry..The antibacterial activity of ethyl ester of p-digallic acid against test cultures of gram-positive bacteria Bacillus cereus and Staphylococcus aureus, gram-negative bacteria Escherichia coli and Pseudomonas aeruginosa for the first time was determined. By antibacterial activity, arbutin was inferior not only to ethyl ester of p-digallic acid, but also to decoction from the leaves of the bearberry. Consequently, the ethyl ester of p-digallic acid is one in main component, which is take the contribution in the antibacterial activity of the decoction and other preparations of the leaves of Arctostaphylos uva-ursi.
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3

Bhouri, Wissem, Ines Skandrani, Mohamed ben Sghair, Marie-Geneviève Djoux Franca, Kamel Ghedira, and Leila Chekir Ghedira. "Digallic acid from Pistascia lentiscus fruits induces apoptosis and enhances antioxidant activities." Phytotherapy Research 26, no. 3 (July 21, 2011): 387–91. http://dx.doi.org/10.1002/ptr.3540.

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4

Bhouri, Wissem, Jihed Boubaker, Ines Skandrani, Kamel Ghedira, and Leila Chekir Ghedira. "Investigation of the apoptotic way induced by digallic acid in human lymphoblastoid TK6 cells." Cancer Cell International 12, no. 1 (2012): 26. http://dx.doi.org/10.1186/1475-2867-12-26.

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5

Victoroff, P. P. "Investigation of the Identity of Digallic Acid with Tannin as Mordants for Basic Dyes." Journal of the Society of Dyers and Colourists 43, no. 1 (October 22, 2008): 12–17. http://dx.doi.org/10.1111/j.1478-4408.1927.tb01402.x.

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6

Grassino, Susana B., and Miriam C. Strumia. "Novel UV-autocurable methacrylo-urethane polymeric films derived fromm-digallic acid: Preparation and characterization." Journal of Applied Polymer Science 70, no. 13 (December 26, 1998): 2575–83. http://dx.doi.org/10.1002/(sici)1097-4628(19981226)70:13<2575::aid-app3>3.0.co;2-3.

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7

Nakane, Hideo, Masanori Fukushima, and Katsuhiko Ono. "Differential Inhibition of Reverse Transcriptase and Various DNA Polymerases by Digallic Acid and Its Derivatives." Journal of Natural Products 53, no. 5 (September 1990): 1234–40. http://dx.doi.org/10.1021/np50071a015.

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8

Zheng, Fengxin, Suiqing Mai, Xiaolin Cen, Pei Zhao, Wenjie Ye, Jiale Ke, Shiqin Lin, et al. "Discovery of digallic acid as XOD/URAT1 dual target inhibitor for the treatment of hyperuricemia." Bioorganic Chemistry 147 (June 2024): 107381. http://dx.doi.org/10.1016/j.bioorg.2024.107381.

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9

Bhouri, Wissem, Safa Derbel, Ines Skandrani, Jihed Boubaker, Ines Bouhlel, Mohamed B. Sghaier, Soumaya Kilani, et al. "Study of genotoxic, antigenotoxic and antioxidant activities of the digallic acid isolated from Pistacia lentiscus fruits." Toxicology in Vitro 24, no. 2 (March 2010): 509–15. http://dx.doi.org/10.1016/j.tiv.2009.06.024.

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10

Yu, Shasha, Zhouwei Duan, Peng Li, Shiping Wang, Lijun Guo, Guanghua Xia, and Hui Xie. "Protective Effect of Polyphenols Purified from Mallotus oblongfolius on Ethanol-Induced Gastric Mucosal Injury by Regulating Nrf2 and MAPKs Pathways." Antioxidants 11, no. 12 (December 12, 2022): 2452. http://dx.doi.org/10.3390/antiox11122452.

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Mallotus oblongifolius (MO), which is rich in polyphenols, is a characteristic tea resource with medicinal value. In this study, a total of 45 polyphenolic components of MO, including narirutin, isoquercitrin, rutin and digallic acid, were identified by UPLC-Q-TOF/MS analysis. In addition, the gastroprotective effect of Mallotus oblongifolius polyphenols (MOP) on ethanol-induced gastric mucosal injury in rats was investigated. The rats received anhydrous ethanol after continuous gavage of MOP or lansoprazole for one week. In addition, the macro- and micro-damage induced by ethanol in the gastric tissue was significantly reduced after MOP pretreatment for one week. Further analysis showed that MOP prevented ethanol-induced acute gastric mucosal injury by increasing the expression of antioxidant enzymes (SOD, CAT, GSH-Px) and decreasing the expression of reactive oxygen species (ROS), lipid oxidation product (MDA) and myeloperoxidase (MPO). Meanwhile, MOP inhibited the phosphorylation of p38/ERK/JNK and promoted the activation of the Nrf2 pathway. These results suggested that MOP may be a promising therapeutic target for the prevention of ethanol-induced gastric mucosal injury by improving oxidative stress, inhibiting the p38/ERK/JNK signaling pathways and activating Nrf2 expression.
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