Relevant bibliographies by topics / Diaphragm electromyogram

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  3. Conference papers

Academic literature on the topic 'Diaphragm electromyogram'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Diaphragm electromyogram"

1

Supinski, G. S., A. F. DiMarco, F. Hussein, and M. D. Altose. "Alterations in respiratory muscle activation in the ischemic fatigued canine diaphragm." Journal of Applied Physiology 67, no. 2 (August 1, 1989): 720–29. http://dx.doi.org/10.1152/jappl.1989.67.2.720.

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The purpose of the present study was to examine the respiratory motor response to diaphragm fatigue. Studies were performed using in situ diaphragm muscle strips dissected from the left costal diaphragm in anesthetized dogs. The left inferior phrenic artery was isolated, and diaphragmatic strip fatigue was elicited by occluding this vessel. Strip tension, strip electromyographic activity, parasternal electromyographic activity, and the electromyogram of the right hemidiaphragm were recorded during spontaneous breathing efforts before, during, and after periods of phrenic arterial occlusion. In separate trials, we examined the neuromuscular responses to phrenic arterial occlusion at arterial PCO2 (PaCO2) of 40, 55, and 75 Torr. No fatigue and no alteration in electromyographic activities were observed in trials at PaCO2 of 40 Torr. During trials at PaCO2 of 55 and 75 Torr, however, diaphragm tension fell, the peak height of the diaphragm strip electromyogram decreased, and the peak heights of the parasternal and right hemidiaphragm electromyograms increased. Relief of phrenic arterial occlusion resulted in a return of strip tension and all electromyograms toward base-line values. In additional experiments, the left phrenic nerve was sectioned in the chest after producing fatigue. Phrenic section was followed by an increase in the peak height of the left phrenic neurogram (recorded above the site of section). This latter finding suggests that diaphragm strip motor drive may be reflexly inhibited during the development of fatigue by neural traffic carried along phrenic afferents.
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Kimata, H., A. Morita, S. Furuhata, H. Itakura, K. Ikenobu, and Y. Kimura. "Assessment of laughter by diaphragm electromyogram." European Journal of Clinical Investigation 39, no. 1 (January 2009): 78–79. http://dx.doi.org/10.1111/j.1365-2362.2008.02037.x.

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Luo, Y. M., L. C. Johnson, M. I. Polkey, M. L. Harris, R. A. Lyall, M. Green, and J. Moxham. "Diaphragm electromyogram measured with unilateral magnetic stimulation." European Respiratory Journal 13, no. 2 (February 1, 1999): 385–90. http://dx.doi.org/10.1183/09031936.99.13238599.

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Sinderby, C., S. Friberg, N. Comtois, and A. Grassino. "Chest wall muscle cross talk in canine costal diaphragm electromyogram." Journal of Applied Physiology 81, no. 5 (November 1, 1996): 2312–27. http://dx.doi.org/10.1152/jappl.1996.81.5.2312.

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Sinderby, C., S. Friberg, N. Comtois, and A. Grassino.Chest wall muscle cross talk in the canine costal diaphragm electromyogram. J. Appl. Physiol.81(5): 2312–2327, 1996.—The present paper describes the influence of cross talk from the abdominal and intercostal muscles on the canine diaphragm electromyogram (EMG). The diaphragm EMG was recorded with bipolar surface electrodes placed on the costal portion of the diaphragm (abdominal side), aligned in the fiber direction, and positioned in a region with a relatively low density of motor end plates. The results indicated that cross talk may occur in the diaphragm EMG, especially during conditions of loaded breathing and light general anesthesia. The cross-talk signals showed characteristics that were entirely different from the diaphragm EMG. Although the diaphragm EMG was typical for signals recorded with electrodes aligned in the fiber direction, the cross-talk signals were characteristic of those obtained with electrode pairs not aligned in the direction of the muscle fibers. Alterations in electrode positioning, interelectrode distance, and/or electrode surface area cannot guarantee the elimination of cross-talk signals, whereas spinal anesthesia at a high thoracic level will paralyze the sources of the cross talk and hence eliminate the cross-talk signals. By taking advantage of the differences in EMG signal characteristics for the diaphragm EMG and cross-talk signals, an index that has the capability to detect cross talk was developed.
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Eikermann, Matthias, Philipp Fassbender, Atul Malhotra, Masaya Takahashi, Shigeto Kubo, Amy S. Jordan, Shiva Gautam, David P. White, and Nancy L. Chamberlin. "Unwarranted Administration of Acetylcholinesterase Inhibitors Can Impair Genioglossus and Diaphragm Muscle Function." Anesthesiology 107, no. 4 (October 1, 2007): 621–29. http://dx.doi.org/10.1097/01.anes.0000281928.88997.95.

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Background It is standard practice to administer a cholinesterase inhibitor (e.g., neostigmine) at the end of a surgical case to reverse suspected effects of neuromuscular blocking agents regardless of whether such residual effects are present. The authors hypothesized that cholinesterase inhibition when given the in absence of neuromuscular blockade (NB) would decrease upper airway dilatory muscle activity and consequently upper airway volume. Methods The authors measured genioglossus and diaphragm electromyograms during spontaneous ventilation in anesthetized, tracheostomized rats before and after administration of neostigmine (0.03, 0.06, or 0.12 mg/kg), after recovery of the train-of-four ratio (quadriceps femoris muscle) to unity after NB (n = 18). For comparison, the authors made the same measurements in rats that had no previous NB (n = 27). In intact anesthetized rats, the authors measured upper airway volume and end-expiratory lung volume by magnetic resonance imaging before and after 0.12 mg/kg neostigmine (n = 9). Results Neostigmine treatment in rats that had fully recovered from NB based on the train-of-four ratio caused dose-dependent decreases in genioglossus electromyogram (to 70.3 +/- 7.6, 49.2 +/- 3.2, and 39.7 +/- 2.3% of control, respectively), decreases in diaphragm electromyogram (to 103.1 +/- 6.5, 83.1 +/- 4.7, and 68.7 +/- 7.3% of control), and decreases in minute ventilation to a nadir value of 79.6 +/- 6% of preneostigmine baseline. Genioglossus electromyogram effects were the same when neostigmine was given with no previous NB. Neostigmine caused a decrease in upper airway volume to 83 +/- 3% of control, whereas end-expiratory lung volume remained constant. Conclusions The cholinesterase inhibitor neostigmine markedly impairs upper airway dilator volume, genioglossus muscle function, diaphragmatic function, and breathing when given after recovery from vecuronium-induced neuromuscular block.
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Sinderby, C., L. Lindstrom, and A. E. Grassino. "Automatic assessment of electromyogram quality." Journal of Applied Physiology 79, no. 5 (November 1, 1995): 1803–15. http://dx.doi.org/10.1152/jappl.1995.79.5.1803.

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Power spectrum analysis of the diaphragm electromyogram (EMGdi) is time consuming, and no criteria have been developed to objectively quantify contamination of the signal. The present work describes a set of computer algorithms that automatically select EMGdi free of the electrocardiogram and numerically quantify the common artifacts that affect the EMGdi. The algorithms were tested 1) on human EMGdi (n = 5) obtained with esophageal electrodes positioned at the level of the gastroesophageal junction, 2) on EMGdi obtained in mongrel dogs (n = 5) with intramuscular electrodes in the costal diaphragm, and 3) on computer-simulated power spectra. For authentic and simulated power spectra, indexes were obtained by the algorithms and were able to quantify signal disturbances induced by noise, electrode motion, esophageal peristalsis (in humans), and non-QRS complex-related electrocardiogram activity. With the index inclusion thresholds set to levels that allowed for a high signal acceptance rate with relatively small artifact-induced fluctuations (10–15%) of the EMGdi center frequency, the computer algorithms were found to be as reliable as or more reliable than other methods, including careful visual selection of the time domain signals by experienced analysts. In conclusion, the frequency domain application of computer algorithms offers a reliable and reproducible means to objectively quantify the sources that contaminate the interference pattern EMG.
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7

Beck, J., C. Sinderby, J. Weinberg, and A. Grassino. "Effects of muscle-to-electrode distance on the human diaphragm electromyogram." Journal of Applied Physiology 79, no. 3 (September 1, 1995): 975–85. http://dx.doi.org/10.1152/jappl.1995.79.3.975.

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It has been suggested that esophageal recordings of the diaphragm electromyogram (EMGdi) are influenced by changes in chest wall configuration. Whether the changes are of physiological or artifactual origin is unclear. For example, the distance between the esophageal electrode and the diaphragm is likely to alter with chest wall configuration and may lead to misinterpretations of EMGdi. The aims of this study were 1) to evaluate and quantify the effect of the muscle-to-electrode (ME) distance filter on EMGdi, as obtained with a multiple-array esophageal electrode, 2) to take advantage of the ME distance filter to locate the position of the diaphragm with respect to the electrode, and 3) to evaluate the influence of lung volume and chest wall configuration on EMGdi center frequency (CF) while controlling for the ME distance filter and signal quality. Five subjects performed six static contractions of the diaphragm at each of seven chest wall configurations, as evaluated by the method of K. Konno and J. Mead (J. Appl. Physiol. 22: 407–422, 1967). EMGdi was measured with seven pairs of electrodes mounted on an esophageal catheter. The pair of electrodes whose EMGdi power spectra were the least filtered by the ME distance was assumed to be closest to the diaphragm. The results of the study indicated that 1) EMGdi power spectra were strongly affected by the distance between the diaphragm and the electrodes. CF decreased by approximately 1 Hz/mm displacement away from the electrode pair closest to the diaphragm; and 2) no systematic relationship was found between changes in chest wall configuration and CF, when CF was measured from the electrode pair closest to the diaphragm. We conclude that the EMGdi CF can be reliably measured with a multiple-array esophageal electrode that covers the span of diaphragmatic excursion and by selecting the pair of electrodes that is the closest to the diaphragm.
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LaFramboise, W. A., and D. E. Woodrum. "Elevated diaphragm electromyogram during neonatal hypoxic ventilatory depression." Journal of Applied Physiology 59, no. 4 (October 1, 1985): 1040–45. http://dx.doi.org/10.1152/jappl.1985.59.4.1040.

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Diaphragmatic electromyogram (EMG) was obtained in eight 48-h-old unanesthetized monkeys while breathing air and then either of two different hypoxic gas mixtures (12 or 8% O2 in N2) for 5 min. Minute ventilation (VI) rose significantly above control levels by 1 min of hypoxemia while animals were breathing either of the hypoxic gas mixtures as tidal volume (VT) and slope and rate moving average EMG increased. The relative gains in VI were associated with comparable increases in diaphragmatic neural activity per minute (EMG/min = peak EMG X frequency) during this early phase of hypoxemia. VI subsequently fell to control levels (inspired O2 fraction = 12%, arterial PO2 = 23 +/- 3 Torr) or significantly below (inspired O2 fraction = 8%, arterial PO2 = 18 +/- 0.4 Torr) by 5 min of hypoxemia, secondary to changes in VT. Despite the decline in VI, slope and rate moving average EMG, and EMG/min remained statistically above control values by 5 min of hypoxemia, although there was a trend for EMG/min to decrease slightly from the 1-min peak response. These findings demonstrate that hypoxic-induced depression of neural input to the diaphragm is not independently responsible for the biphasic nature of the newborn ventilatory response, although it cannot be ruled out as a contributor. The fall in inspiratory volumes despite constant elevated EMG activity suggests the presence of a change in respiratory mechanics and/or an impairment in diaphragmatic contractile function without offsetting neural compensatory activity.
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Burns, David P., Sarah E. Drummond, Dearbhla Bolger, Amélie Coiscaud, Kevin H. Murphy, Deirdre Edge, and Ken D. O’Halloran. "N-acetylcysteine Decreases Fibrosis and Increases Force-Generating Capacity of mdx Diaphragm." Antioxidants 8, no. 12 (November 24, 2019): 581. http://dx.doi.org/10.3390/antiox8120581.

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Respiratory muscle weakness occurs due to dystrophin deficiency in Duchenne muscular dystrophy (DMD). The mdx mouse model of DMD shows evidence of impaired respiratory muscle performance with attendant inflammation and oxidative stress. We examined the effects of N-acetylcysteine (NAC) supplementation on respiratory system performance in mdx mice. Eight-week-old male wild type (n = 10) and mdx (n = 20) mice were studied; a subset of mdx (n = 10) received 1% NAC in the drinking water for 14 days. We assessed breathing, diaphragm, and external intercostal electromyogram (EMG) activities and inspiratory pressure during ventilatory and non-ventilatory behaviours. Diaphragm muscle structure and function, cytokine concentrations, glutathione status, and mRNA expression were determined. Diaphragm force-generating capacity was impaired in mdx compared with wild type. Diaphragm muscle remodelling was observed in mdx, characterized by increased muscle fibrosis, immune cell infiltration, and central myonucleation. NAC supplementation rescued mdx diaphragm function. Collagen content and immune cell infiltration were decreased in mdx + NAC compared with mdx diaphragms. The cytokines IL-1β, IL-6 and KC/GRO were increased in mdx plasma and diaphragm compared with wild type; NAC decreased systemic IL-1β and KC/GRO concentrations in mdx mice. We reveal that NAC treatment improved mdx diaphragm force-generating capacity associated with beneficial anti-inflammatory and anti-fibrotic effects. These data support the potential use of NAC as an adjunctive therapy in human dystrophinopathies.
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Goldman, Ernesto, Christer Sinderby, Lars Lindstrom, and Alex Grassino. "Influence of Atracurium on the Diaphragm Mean Action Potential Conduction Velocity in Canines." Anesthesiology 90, no. 3 (March 1, 1999): 855–62. http://dx.doi.org/10.1097/00000542-199903000-00029.

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Background It has been shown that progressive neuromuscular blockade (NMB) affects the electromyogram power spectrum and compound muscle action potential duration in skeletal muscle. These measures are linked to the mean muscle action potential conduction velocity (APCV), but no studies have confirmed a relation between the mean APCV and NMB. The aim of this study was to determine whether diaphragm mean APCV is affected by NMB. Methods The effects of NMB on diaphragm mean APCV were evaluated in five mongrel dogs. Progressive NMB was induced by slow intravenous infusion of atracurium. During spontaneous breathing, the diaphragm mean APCV was determined by electromyogram signals, in the time and frequency domains. The magnitude of NMB was quantified by the amplitude of the compound muscle action potential and by changes in muscle shortening during supramaximal stimulation of the phrenic nerve. Results Progressive NMB was associated with a decrease in diaphragm mean APCV. At approximately 70% reduction in the compound muscle action potential amplitude, diaphragm mean APCV had decreased more than 20%. Recovery after NMB was characterized by a restoration of the mean APCV to control values. Conclusion This study shows that progressive NMB paralyzes motor units within the diaphragm in an orderly manner, and the blockade first affects muscle fibers with high APCV before it affects fibers with lower APCV.
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Dissertations / Theses on the topic "Diaphragm electromyogram"

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Singh, Bhajan. "The function of the human diaphragm as a volume pump and measurement of its efficiency." University of Western Australia. School of Biomedical and Chemical Sciences, 2004. http://theses.library.uwa.edu.au/adt-WU2004.0029.

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[Truncated abstract] The function of the diaphragm as a volume pump has not been adequately evaluated because there are no accurate methods to measure the volume displaced by diaphragm motion (ΔVdi). As a consequence, the work done, power output and efficiency of the diaphragm have not been measured. Efficiency of the diaphragm could be measured by relating the power output of the diaphragm to its neural activation. The aims of this thesis were to (a) develop a new biplanar radiographic method to measure ΔVdi and use this to evaluate the effect of costophrenic fibrosis and emphysema on ΔVdi, (b) develop a new fluoroscopic method to enable breath-by-breath measurements of ΔVdi, (c) evaluate a method for quantifying neural activation of the diaphragm, and (d) combine measurements of transdiaphragmatic pressure, ΔVdi, inspiratory duration and neural activation of the diaphragm to quantify the neuromechanical efficiency of the diaphragm
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Conference papers on the topic "Diaphragm electromyogram"

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Schaeffer, Michele, Lyndsey Pearsall, and Dennis Jensen. "Variability Of The Diaphragm Electromyogram (EMGdi) Response To Strenuous Exercise In Healthy Humans." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6390.

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Wong, V., R. Shah, W. Zhang, A. Mohindra, HV Fletcher, GF Rafferty, J. Moxham, SDR Harridge, NR Lazarus, and CJ Jolley. "S81 Use of the diaphragm electromyogram to investigate the effect of healthy ageing on neural respiratory drive." In British Thoracic Society Winter Meeting 2019, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 4 to 6 December 2019, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2019. http://dx.doi.org/10.1136/thorax-2019-btsabstracts2019.87.

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Uesawa, Hayato, Tomoya Takehara, and Akinori Ueno. "Non-contact measurements of diaphragm electromyogram, electrocardiogram and respiratory variations with sheet-type fabric electrodes for neonatal monitoring." In 2018 IEEE EMBS International Conference on Biomedical & Health Informatics (BHI). IEEE, 2018. http://dx.doi.org/10.1109/bhi.2018.8333361.

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Chuang, Sandra, Arthur Teng, Jane Butler, Simon Gandevia, Hiran Selvadurain, and Adam Jaffe. "CPAP can improve respiratory effort as measured by surface electromyogram of the diaphragm (sEMGdi) in children with sleep-disordered breathing." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa4351.

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