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Relevant bibliographies by topics / Diabetes – Etiology

Academic literature on the topic 'Diabetes – Etiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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Journal articles on the topic "Diabetes – Etiology"

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Cinek, Ondřej, and Zdeněk Šumník. "Type 1 diabetes: etiology and epidemiology." Vnitřní lékařství 65, no. 4 (April 1, 2019): 235–47. http://dx.doi.org/10.36290/vnl.2019.041.

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Bratchikova, N. A., A. I. Zhelnina, D. V. Lopatin, and L. V. Sivakova. "DIABETES MELLITUS: ETIOLOGY, PATHOGENESIS, TREATMENT." Международный студенческий научный вестник (International Student Scientific Herald), no. 1 2020 (2020): 13. http://dx.doi.org/10.17513/msnv.19902.

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Todd, John A. "Etiology of Type 1 Diabetes." Immunity 32, no. 4 (April 2010): 457–67. http://dx.doi.org/10.1016/j.immuni.2010.04.001.

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Rossini, A. A., J. P. Mordes, and E. S. Handler. "Speculations on etiology of diabetes mellitus. Tumbler hypothesis." Diabetes 37, no. 3 (March 1, 1988): 257–61. http://dx.doi.org/10.2337/diabetes.37.3.257.

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Schade, D. S., D. A. Drumm, W. C. Duckworth, and R. Philip Eaton. "The Etiology of Incapacitating, Brittle Diabetes." Diabetes Care 8, no. 1 (January 1, 1985): 12–20. http://dx.doi.org/10.2337/diacare.8.1.12.

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Taylor, R. "Type 2 Diabetes: Etiology and reversibility." Diabetes Care 36, no. 4 (March 21, 2013): 1047–55. http://dx.doi.org/10.2337/dc12-1805.

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Ambler, GR. "Type 1 Diabetes: Etiology And Treatment." Journal of Paediatrics and Child Health 40, no. 4 (April 2004): 243–44. http://dx.doi.org/10.1111/j.1440-1754.2004.00351.x.

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Rodger, N. Wilson. "Type 1 Diabetes: Etiology and Treatment." Annals of Internal Medicine 139, no. 10 (November 18, 2003): 874. http://dx.doi.org/10.7326/0003-4819-139-10-200311180-00036.

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Tolstoi, Linda G., and John B. Josimovich. "Gestational Diabetes Mellitus: Etiology and Management." Nutrition Today 34, no. 5 (September 1999): 178–88. http://dx.doi.org/10.1097/00017285-199909000-00003.

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Tolstoi, Linda G., and John B. Josimovich. "Gestational Diabetes Mellitus: Etiology and Management." Nutrition Today 34, no. 5 (September 1999): 178–88. http://dx.doi.org/10.1097/00017285-199934050-00003.

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Dissertations / Theses on the topic "Diabetes – Etiology"

1

Topp, Brian G. "The etiology and natural history of type 2 diabetes /." Burnaby B.C. : Simon Fraser University, 2005. http://ir.lib.sfu.ca/handle/1892/2423.

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Ferreira, Filipa Cristina Costa. "A Diabetes - Principais Parâmetros para o Controlo da Diabetes." Master's thesis, Faculdade de Ciências Médicas. UNL, 2013. http://hdl.handle.net/10362/10001.

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RESUMO: A Diabetes Mellitus é uma doença metabólica crónica, com deficiência a nível do metabolismo dos hidratos de carbono, lípidos e proteínas, resultante de deficiências na secreção ou ação da insulina, ou de ambas, que quando não tratada antecipadamente e de modo conveniente, pode ter consequências muito graves. Dado a incidência a nível mundial da Diabetes Mellitus, torna-se de elevada importância avaliar toda a sua envolvência e estudar bem quais os critérios a ter em consideração. Este trabalho propõe-se estudar para além dos parâmetros bioquímicos relacionados com a doença - Glicose e Hemoglobina Glicada A1c (HbA1c), analisar os resultados dos últimos cinco anos (2008-2012) dos ensaios interlaboratoriais do PNAEQ, do Departamento de Epidemiologia, do Instituto Nacional de Saúde Dr. Ricardo Jorge. Foram também analisadas as metodologias utilizadas e as variações interlaboratoriais, de forma a entender qual ou quais são os parâmetros mais adequados para o seu diagnóstico e controlo. Este estudo utilizou a população de laboratórios portugueses, públicos e privados, de Portugal Continental e Ilhas, um laboratório de Angola e outro de Macau que se inscreveram no PNAEQ nestes cinco anos, sendo a amostra composta pelo n.º de participações. No programa de Química Clinica foram distribuídas 38 amostras e no programa de HbA1c foram distribuídas 22 amostras. Para a glicose, o nível de desempenho nos ensaios é na globalidade das amostras de Excelente, no entanto verifica-se que sempre que a concentração da amostra é de nível patológico, que a maioria dos ensaios o desempenho foi inferior – Bom. O método de eleição e com CV% mais baixos foi o método da hexoquinase. Para a HbA1c, o nível de desempenho nos ensaios é na globalidade das amostras de Excelente. O método de eleição e com CV% mais baixos foi o método de HPLC. O CV% para a glicose ronda desde 2010 a 2012, os 3% e para a HbA1c foi de aproximadamente 4,0% em 2012. A HbA1c tem mostrado ser uma ferramenta muito útil, importante e robusta na monitorização da Diabetes, sendo hoje em dia quase sempre requisitada em análises de rotina a diabéticos de modo a prevenir complicações que possam vir a acorrer. No futuro poderá ser um importante, senão o parâmetro de futuro, para o diagnóstico da Diabetes, no entanto, mesmo já tendo sido muito trabalhada a sua padronização, ainda existem questões por responder como quais são na realidade todos os seus interferentes, qual a verdadeira relação da HbA1c com a glicose média estimada, em todas as populações e com estudos epidemiológicos. Também a própria educação do diabético e clínico deve ser aprimorada, pelo que neste momento as PTGO e os doseamentos de glicose em jejum devem ser utilizados e encontrando-se a Norma da DGS N.º 033/2011 de acordo com as necessidades e com o estado da arte deste parâmetro. A implementação da glicose média estimada será uma mais-valia na monitorização dos diabéticos pelo que deverá ser uma das prioridades a ter em conta no futuro desta padronização, uniformizando a decisão clinica baseada nela e minimizando a dificuldade de interpretação de resultados de laboratório para laboratório. --------------ABSTRACT: Diabetes Mellitus is a chronic metabolic disease, with a deficit in the metabolism of carbohydrates, lipids and proteins, resulting from deficiencies in insulin secretion or action, or both, which if, when not early treated in a proper way, may result in very serious consequences. Given the worldwide incidence of diabetes mellitus, it is highly important to evaluate all its background and study specifically all the criteria to take into consideration. The aim of this thesis is to study and evaluate beyond the biochemical parameters related to the disease - Glucose and Glycated Haemoglobin A1c (HbA1c), analyze the results of the last five years (2008-2012) of the PNAEQ interlaboratorial tests, in the Department of Epidemiology of National Institute of Health Dr. Ricardo Jorge. It is also intended to analyze the methodologies used and the interlaboratorial variations, in order to understand the most suitable parameters for the diagnosis and control. This study was based in a population of Portuguese laboratories, public and private, of Portugal mainland and islands, a laboratory of Angola and other from Macau, who enrolled in PNAEQ in these five years, and the sample was composed by the n. º of holdings. In the Clinical Chemistry Program there were distributed 38 samples and in the program HbA1c were distributed 22 samples. For glucose, the level of performance in the total nº of the samples was Excellent; however, it was found that when the concentration level of the sample was pathological, in most of the tests the performance was Good. The most preferred method with the lowest CV% is the hexokinase method. For the HbA1c, as a whole, the samples’ tests were Excellent, at the level of performance. The method of election with the lower CV% was the HPLC. The CV% for glucose was around 3%, from 2010 to 2012 and the HbA1c was approximately 4.0% in 2012. The HbA1c method has demonstrated to be a very useful tool, important and robust for monitoring diabetes, being nowadays, almost always required in routine analysis to prevent future complications. In the future it may be an important parameter, if not the most important, for the diagnosis of diabetes. However, despite it has already been standardized, there are still some questions that need to be answered, such as, which are in fact all their interferences, which is the true connection of HbA1c, when compared with the estimated average glucose, in all populations and epidemiological studies. Moreover, the education of the patient and the doctor concerning diabetes should be improved. Nowadays, the Oral Glucose Tolerance Test (OGTT) and fasting glucose determinations should be used and, the needs and the state of the art of this parameter, should be in accordance with the Standard DGS N. º 033/2011. The Implementation of the estimated average glucose will be an added value in monitoring diabetics and, therefore, should be a priority to consider in its future standardization and clinical decision based on it, will be uniform and the difficulty of interpreting results from laboratory to laboratory will be minimal.

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Orr, Neil John. "Patterns of care for diabetes: risk factors for vision-threatening retinopathy." Thesis, The University of Sydney, 2005. http://hdl.handle.net/2123/1421.

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OBJECTIVES: In Australia, diabetes causes significant morbidity and mortality. Whilst the need to prevent diabetes and its complications has been widely recognised, the capacity of health care systems - which organise diabetes care - to facilitate prevention has not been fully established. METHODS: A series of seven population-based case-control studies were used to examine the effectiveness of the Australian health care system and its capacity to manage diabetes. Six of the studies compared the patterns of care of patients who had developed advanced diabetes complications in 2000 (cases), to similar patients who remained free of the condition (controls) across Australia and for various risk groups. A secondary study investigated the role of treating GPs in the development of the outcome. RESULTS: A strong relationship between the patterns of care and the development of advanced diabetes complications was found and is described in Chapter 4. In Chapter 5, this same relationship was investigated for each Australian state and territory, and similar findings were made. The study in Chapter 6 investigated whether late diagnosis or the patterns of care was the stronger risk factor for advanced diabetes complications, finding that the greatest risk was associated with the latter. In Chapter 7 the influence of medical care during the pre-diagnosis period was explored, and a strong relationship between care obtained in this period and the development of advanced complications was found. In Chapter 8, which investigated the role of socio-economic status in the development of advanced complications, found that the risk of advanced diabetes complications was higher in low socio-economic groups. Chapter 9 investigated geographic isolation and the development of advanced diabetes complications and found that the risk of advanced complications was higher in geographically isolated populations. Finally, Chapter 10, which utilised a provider database, found that some GP characteristics were associated with the development of advanced diabetes complications in patients. CONCLUSION: A number of major risk factors for the development of advanced complications in Australia was found. These related to poorer diabetes management, later diagnosis, low socioeconomic status and geographic isolation. Strategies must be devised to promote effective diabetes management and the early diagnosis of diabetes across the Australian population.

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Orr, Neil John. "Patterns of care for diabetes: risk factors for vision-threatening retinopathy." University of Sydney, 2005. http://hdl.handle.net/2123/1421.

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Master of Public Health
OBJECTIVES: In Australia, diabetes causes significant morbidity and mortality. Whilst the need to prevent diabetes and its complications has been widely recognised, the capacity of health care systems - which organise diabetes care - to facilitate prevention has not been fully established. METHODS: A series of seven population-based case-control studies were used to examine the effectiveness of the Australian health care system and its capacity to manage diabetes. Six of the studies compared the patterns of care of patients who had developed advanced diabetes complications in 2000 (cases), to similar patients who remained free of the condition (controls) across Australia and for various risk groups. A secondary study investigated the role of treating GPs in the development of the outcome. RESULTS: A strong relationship between the patterns of care and the development of advanced diabetes complications was found and is described in Chapter 4. In Chapter 5, this same relationship was investigated for each Australian state and territory, and similar findings were made. The study in Chapter 6 investigated whether late diagnosis or the patterns of care was the stronger risk factor for advanced diabetes complications, finding that the greatest risk was associated with the latter. In Chapter 7 the influence of medical care during the pre-diagnosis period was explored, and a strong relationship between care obtained in this period and the development of advanced complications was found. In Chapter 8, which investigated the role of socio-economic status in the development of advanced complications, found that the risk of advanced diabetes complications was higher in low socio-economic groups. Chapter 9 investigated geographic isolation and the development of advanced diabetes complications and found that the risk of advanced complications was higher in geographically isolated populations. Finally, Chapter 10, which utilised a provider database, found that some GP characteristics were associated with the development of advanced diabetes complications in patients. CONCLUSION: A number of major risk factors for the development of advanced complications in Australia was found. These related to poorer diabetes management, later diagnosis, low socioeconomic status and geographic isolation. Strategies must be devised to promote effective diabetes management and the early diagnosis of diabetes across the Australian population.

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Marshall, Julie Ann. "The role of dietary fiber in the etiology of noninsulin-dependent diabetes mellitus /." Thesis, Connect to this title online; UW restricted, 1987. http://hdl.handle.net/1773/10952.

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Turatti, Luiz Alberto Andreotti. "Papel dos componentes do sistema GH-IGF-IGFBP nos mecanismos envolvidos na resposta imunológica do Diabetes Melito tipo 1." Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-10102014-114845/.

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Com o objetivo de verificar se as proteínas do sistema GH-IGF-IGFBP e o receptor do fator de crescimento insulina símile tipo I (IGF-IR) estão envolvidos na etiopatogenia do Diabetes Melito tipo 1 (DM1) , foram estudados 23 pacientes prépúberes portadores de DM1 em diferentes fases do diagnóstico (Grupo A: tempo de diagnóstico <= 6 meses; Grupo B: tempo de diagnóstico > 6 meses) e 10 indivíduos pré-púberes sadios como grupo controle (Grupo C). A expressão do mRNA do IGFIR realizada através do ensaio molecular de RT-PCR nos linfócitos periféricos T e B não demonstrou diferenças estatisticamente significantes nos linfócitos T quando comparados indivíduos diabéticos e controles, sugerindo que a ativação imunológica destas células seja independente da ação do IGF-IR. Observou-se uma maior expressão do mRNA do IGF-IR dos linfócitos B de pacientes diabéticos em relação ao grupo controle (p < 0,05). A avaliação das proteínas do sistema GH-IGF-IGFBP não demonstrou diferença estatística significante entre os grupos. Estes achados, associados à presença de auto-anticorpos para o DM1 (ICA, anti-GAD e anti-IA2) sugerem fortemente o papel do IGF-IR na ativação dos linfócitos B envolvidos na etiopatogenia do DM1
Aiming to verify if GH-IGF-IGFBP proteins system and insulin-like growth factor type I receptor (IGF-IR) are implicated on pathofisiology of type 1 Diabetes Mellitus (DM1), we studied 23 prepubertal patients with DM1 on different stages of diagnosis (Group A: time of diagnosis <= 6 months; Group B: time of diagnosis > 6 months) and 10 prepubertal healthy subjects as control group (Group C). The RT-PCR molecular assay for IGF-IR mRNA on peripheral T and B lymphocytes didn\'t show statistical differences between the groups when T cells were analyzed. We found an increase of IGF-IR mRNA expression on B cells from diabetic patients when compared to healthy subjects (p< 0,05). There were no differences in the GH-IGF-IGFBP proteins system levels between the groups. Our study suggest that IGF-IR in association with diabetes-related autoantibodies (ICA, anti-GAD and anti-IA2) presence could activate B cells involved on pathofisiology of DM1

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Agardh, Emilie. "The influence of psychosocial stress, socioeconomic differences and coffee consumption in the etiology of type 2 diabetes /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-180-6/.

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Lewandowski, Paul, and mikewood@deakin edu au. "Liver fat metabolism, obesity and diabetes in Psammomys Obesus." Deakin University. School of Health Sciences, 1999. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20050825.111432.

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Defects in fat metabolism are central to the aetiology and pathogenesis of obesity and type II diabetes. The liver plays a central role in these disease states via its regulation of glucose and fat metabolism. In addition, accumulation of fat within the liver has been associated with changes in key pathways of carbohydrate and fat metabolism. However a number of questions remain. It is hypothesised that fat accumulation within the liver is a primary defect in the aetiology and pathogenesis of obesity and type II diabetes. Fat accumulating in the liver is the result of changes in the gene expression of key enzymes and proteins involved with fat uptake, fat transport, fat oxidation, fat re-esterification or storage and export of fat from the liver and these changes are regulated by key lipid responsive transcription factors. To study these questions Psammomys obesus was utilised. This polygenic rodent model of obesity and type II diabetes develops obesity and diabetes in a similar pattern to susceptible human populations. In addition dietary and environmental changes to Psammomys obesus were employed to create different states of energy balance, which allowed the regulation of liver fat gene expression to be examined. These investigations include: 1) Measurement of fat accumulation and fatty acid binding proteins in lean, obese and diabetic Psammomys obesus. 2) Characterisation of hepatic lipid enzymes, transport protein and lipid responsive transcription factor gene expression in lean, obese and diabetic Paammomys obesus. 3) The effect of acute and chronic energy restriction on hepatic lipid metabolism in Psammomys obesus. 4) The effect of sucrose feeding on the development of obesity and type II diabetes in Psammomys obesus. 5) The effect of nicotine treatment in lean and obese Psammomys obesus, 6) The effect of high dose leptin administration on hepatic fat metabolism in Psammomys obesus. The results of these studies demonstrated that fat accumulation within the liver was not a primary defect in the aetiology and pathogenesis of obesity and type II diabetes. Fat accumulating in the liver was not the result of changes in the gene expression of key enzymes and proteins involved in hepatic fat metabolism. However changes in the mRNA level of the transcription factors PPAR∝ and SREBP-1C was associated with the development of diabetes and the gene expression of these two transcription factors was associated with changes in diabetic status.

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Rodrigues, Brian Baltzar. "Hypertension and diabetic cardiomyopathy." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24906.

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The isolated perfused working heart was used to study hypertensive- diabetes induced alterations in cardiac function at 6 and 12 weeks after the induction of diabetes. There was no difference in cardiac function between normotensive Wistar and spontaneously hypertensive (SHR) diabetic rats at 6 weeks after diabetes induction. Wistar-Kyoto (WKY) rats were also included as normotensive controls in our 12-week study. Successful induction of diabetes was confirmed by the presence of hyperglycemia, hypoinsulinemia, glycosuria and increased haemoglobin glycosylation in all three diabetic groups. However, quantitation of various parameters of heart function revealed highly significant differences between SHR diabetic animals and all other groups, associated with an increased mortality. Serum lipids were elevated in SHR and Wistar and unaffected in WKY diabetic rats. Furthermore, thyroid hormone levels were not depressed in WKY diabetic rats and could explain the lack of cardiac dysfunction in these animals. The data provide further evidence that the combination of hypertension and diabetes mellitus produces greater myocardial dysfunction than is seen with either disease alone and is associated with a significant mortality. The effects of hydralazine on blood lipids, systolic pressure and cardiac performance were assessed in male Wistar rats, 6 weeks after they were made diabetic with streptozotocin (STZ). When hydralazine was administered for a 6-week period to the diabetic rats, their blood lipids were not significantly different from that of non-diabetic rats despite a low serum insulin. In contrast, blood lipids were elevated in the diabetic rats that were not treated with hydralazine; these animals also had low insulin levels. Cardiac performance was depressed in the untreated diabetic animals, but the cardiac performance of the hydralazine-treated diabetic animals showed a definite improvement which could be partly explained by their normal thyroid status in contrast to the untreated diabetic animals which were slightly hypothyroid. Blood pressure was elevated only in the untreated diabetic animals. Thus hydralazine controlled the high serum lipids and blood pressure and improved cardiac performance in STZ diabetic rats. To examine the influence of sex differences in the STZ model of diabetes, we studied left ventricular function in hearts from 6 week male and female rats. Significantly lower values for +dP/dt occurred in male diabetic rats compared with their own controls or female diabetics at most left atrial filling pressures. Decreases in this value for female diabetic rats compared to their own controls occurred only at high left atrial pressures. It appears that diabetes mellitus produces greater myocardial dysfunction in male diabetic rats.
Pharmaceutical Sciences, Faculty of
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Stevens, Joseph. "Coxsackievirus Infection of B Cells: Towards a Better Understanding of the Etiology of Type 1 Diabetes." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1522418340151761.

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Books on the topic "Diabetes – Etiology"

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M, Sperling, ed. Type 1 diabetes: Etiology and treatment. Totowa, N.J: Humana Press, 2003.

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Léon, Aucoin, and Prideux Tristan, eds. Handbook of type 1 diabetes mellitus: Etiology, diagnosis, and treatment. Hauppauge, N.Y: Nova Science Publishers, 2009.

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Prabhakar, Sharma S. Advances in pathogenesis of diabetic nephropathy. Hauppauge, N.Y: Nova Science Publishers, 2010.

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Comparison of Type I and Type II Diabetes, Similarities, and Dissimilarities in Etiology, Pathogenesis and Complications (Conference) (1984 Toronto). Comparison of type I and type II diabetes: Similarities and dissimilarities in etiology, pathogenesis and complications. New York: Plenum, 1985.

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C, Levy-Marchal, and Czernichow P, eds. Epidemiology and etiology of insulin-dependent diabetes in the young. Basel: Karger, 1992.

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G, Leslie R. D., ed. Molecular pathogenesis of diabetes mellitus. Basel: Karger, 1997.

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Bluestone, Jeffrey A., Mark Atkinson, and Peter Arvan. Type 1 diabetes. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory Press, 2012.

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S, Feman Stephen, ed. Ocular problems in diabetes mellitus. Boston: Blackwell Scientific Publications, 1991.

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1951-, Barnett A. H., and Kumar Sudhesh, eds. Obesity and diabetes. 2nd ed. Chichester, West Sussex, UK: Wiley-Blackwell, 2009.

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Pierce, Grant N. Heart dysfunction in diabetes. Boca Raton, Fla: CRC Press, 1988.

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Book chapters on the topic "Diabetes – Etiology"

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Rambaran, Varma H., and Nalini K. Singh. "Etiology." In Alternative Medicines for Diabetes Management, 1–6. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003322429-1.

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Ramasamy, Ravichandran, and Ann Marie Schmidt. "Etiology of diabetes mellitus." In Diabetes Mellitus and Oral Health, 1–26. Hoboken, NJ, USA: John Wiley & Sons, Inc, 2014. http://dx.doi.org/10.1002/9781118887837.ch1.

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Olafiranye, Oladipupo, Sidrah Mahmud, Ferdinand Zizi, Samy I. McFarlane, Girardin Jean-louis, and Gbenga Ogedegbe. "Resistant Hypertension: Etiology, Evaluation and Management." In Diabetes and Hypertension, 65–73. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-60327-357-2_6.

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Andersen, Dana K. "Pancreatogenic diabetes: etiology, implications, and management." In Pancreatitis, 211–24. Chichester, UK: John Wiley & Sons, Ltd, 2017. http://dx.doi.org/10.1002/9781118924907.ch14c.

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Yasmin, Iqra, Wahab Ali Khan, Saima Naz, Muhammad Waheed Iqbal, Chinaza G. Awuchi, Chukwuebuka Egbuna, Sadia Hassan, Kingsley C. Patrick-Iwuanyanwu, and Chukwuemelie Zedech Uche. "Etiology of Obesity, Cancer, and Diabetes." In Dietary Phytochemicals, 1–27. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-72999-8_1.

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Flynn, Colleen, and George Bakris. "Arterial hypertension in diabetes: etiology and treatment." In International Textbook of Diabetes Mellitus, 1079–90. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118387658.ch74.

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Kruszynska, Yolanta T. "Type 2 Diabetes Mellitus: Etiology, Pathogenesis and Clinical Manifestations." In Principles of Diabetes Mellitus, 179–99. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-1-4757-6260-0_10.

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Almutlaq, Nourah, and Erica A. Eugster. "Central Diabetes Insipidus (Etiology, Epidemiology, and Management)." In Endocrine Conditions in Pediatrics, 327–30. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52215-5_55.

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Burns, Carrie, and Imali Sirisena. "Type 2 Diabetes: Etiology, Epidemiology, Pathogenesis, Treatment." In Metabolic Syndrome, 601–17. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-11251-0_34.

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Burns, Carrie, and Imali Sirisena. "Type 2 Diabetes-Etiology, Epidemiology, Pathogenesis, Treatment." In Metabolic Syndrome, 1–19. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-12125-3_34-1.

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Conference papers on the topic "Diabetes – Etiology"

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Tessaro, Camila Lorenzini, Renata Dal-Prá Ducci, Cláudia Suemi Kamoi Kay, Otto Jesus Hernandez Fustes, Lineu César Werneck, Paulo José Lorenzoni, and Rosana Hermínia Scola. "Clinical and electrophysiological characteristics of the peripheral polyneuropathies from a single specialized center in the Southern Brazil." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.359.

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Background: The peripheral polyneuropathies have etiological heterogeneity, with more than a hundred known causes. In addition, they have a lack of information related to their epidemiology. Objectives: The aim of this study is to determine the prevalence of each etiology of polyneuropathy in a single specialized center from Southern Brazil and to correlate main clinical manifestations and electrophysiological aspects. Design and setting: Observational cross-sectional study. Neuromuscular disorder center from a tertiary service. Methods: This study comprised individuals with electrodiagnostic tests compatible with polyneuropathy from a neuromuscular disorder center. Selected patients were those who underwent nerve conduction studies between 2008 and 2017. Through analysis of medical records, polyneuropathies were classified according to etiology and neurophysiological aspect. Results: The sample population consisted of 380 patients who has a male predominance (59.5%), with a median age of 43 years (26-57). The main etiologies were inflammatory (23.7%), hereditary (18.9%), idiopathic (13.7%), multifactorial (11.1%) and diabetes (10.8%). The main electrophysiological patterns were Axonal Sensorimotor Polyneuropathy (36.1%) and Demyelinating- Axonal Sensorimotor Polyneuropathy (27.9%). Idiopathic, diabetic, and multifactorial polyneuropathies had predominantly axonal pattern, while inflammatory and hereditary polyneuropathies had mainly demyelinating pattern. Conclusions: The frequency of inflammatory etiology was higher than previously described and frequency of diabetic polyneuropathy was lower. There may be a change in epidemiology of polyneuropathy in specialized centers, with a tendency to decrease idiopathic polyneuropathy. Electrodiagnostic testing is useful for etiological diagnosis since electrophysiological patterns correlate with specific etiologies.

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Marques, Marina Trombin, Leonardo de Sousa Bernardes, Rafael Zini Moreira da Silva, Matheus Gonçalves Maia, Edson Junior Gonçalves Bechara, Eduardo dos Santos Sousa, Juliana Rodrigues Dias Primo, Vivian Dias Baptista Gagliardi, and Rubens José Gagliardi. "Trousseau Syndrome in a patient on Direct Oral Anticoagulant use: A Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.508.

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Background: The Three Territory Sign (TTS) is a radiologic marker of ischemic stroke associated with malignant neoplastic diseases (Trousseau Syndrome) and corresponds to a rare stroke etiology. Case Report: Female, 62-year-old patient, with comorbidities of smoking, hypertension and diabetes, presented with a sudden faciobrachial-predominant left hemiparesis settled in the day before the admission. Diagnosed with a metastatic rectal adenocarcinoma seven months before, she underwent a rectosigmoidectomy three months ago and developed deep vein thrombosis, starting anticoagulant therapy with rivaroxaban 20mg daily. A Magnetic Resonance Imaging (MRI) revealed several lesions with restricted diffusion in multiple vascular territories, bilaterally, corresponding to ischemic stroke. Etiologic investigation did not detect signs of cardioembolism, nor significant vessel stenosis or unstable atherosclerotic plaques. In admission, she had a D-dimer level of 11,43μg (0- 0,5μg/mL). Conclusion: The evidence of TTS is about six times more frequent in stroke related to malignancies compared to cardioembolic etiology. The D-dimer is a marker of malignancies in cryptogenic stroke, elevated in 75% of cases. The most common associated neoplasms are pulmonary (40%) and gastrointestinal (33,3%). In the MRI, the lesions can be isolated or gathered, generally small and peripheral. There is no evidence regarding the ideal preventive therapy. It is necessary to reinforce the importance of investigating malignancies in patients presenting with cryptogenic stroke and TTS, a syndrome that is still poorly recognized.

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Heyne, E., G. Färber, O. Walther, H. Kirov, S. Freiburger, T. Doenst, and M. Schwarzer. "Mitochondrial Function in Human Heart Failure is Dependent on Etiology, Severity, and the Presence of Diabetes." In 49th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1705354.

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Silva, Paula Baleeiro Rodrigues, Paulo Eduardo Lahoz Fernandez, Victória Veiga Ribeiro Gonçalves, Pedro Vinicius Brito Alves, Jorge Fernando de Miranda Pereira, Isabela Fonseca Risso, Ingrid Caroline Freitas Barboza, et al. "Acute glaucoma mimicking trigeminal autonomic cephalalgias: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.367.

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Context: Trigeminal autonomic cephalalgias (TAC) are unilateral and recurrent headache with ipsilateral cranial autonomic symptoms such as lacrimation, conjunctival injection, ptosis and changes on pupillary diameter. Acute glaucoma is a rare etiology of headache in the emergency room and can be confused with TAC because it also presents with similar symptoms in the affected eyes. Case report: We present a case report of a 50-year-old insulin-dependent diabetic woman with severe headache that started the day before admission. The headache was left hemicranial, pulsatile and associated with ipsilateral lacrimation. On physical examination we observed mild conjunctival hyperemia, ptosis, visual loss of 20/800 and fixed mydriasis in left eye. Because of the visual loss the patient was referred for urgent ophthalmological evaluation that revealed high intraocular pressure (41 mmHg) and neovascularization of the iris, suggesting the diagnosis of acute glaucoma associated with the neovascularization related to diabetes mellitus. Conclusions: Acute glaucoma should be distinguished from TAC given the overlap of severe headache and ipsilateral eye symptom. However, the presence of fixed mydriasis and progressive visual loss suggested the diagnosis of acute glaucoma, since in TAC miosis is the usual pupillary alteration, as a consequence of parasympathetic overactivation, and visual loss is uncommon.

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Guarino, Maria, Marlene Lages, Ipek Suluova, Rui Fonseca Pinto, and Nuno Lopes. "The CBmeter: designing innovative strategies for early diagnosis of metabolic diseases." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1001410.

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Diabetes is a disease with high prevalence worldwide, however, about 44% of patients are asymptomatic, which leads to a later diagnosis of the disease and, consequently, increases the risk of developing complications. The development of new approaches for early diagnosis is imperative to allow proper adoption of preventive measures. From a motivational point of view, it is easier for patients to adopt healthy eating habits and lifestyles when there is an altered marker that indicates subclinical disease, particularly in a pathology that remains asymptomatic until advanced stages. Thus, timely diagnosis based on a measurable and monitorable indicator is extremely important so that such behaviors are implemented as early as possible, increasing effective health gains and reducing the costs related to this pathology. Pre-clinical studies in animal models have shown that the etiology of type 2 diabetes mellitus (T2DM) is related to alterations in the carotid bodies (CB), chemosensory organs located in the bifurcation of the carotid arteries. In animals with T2DM it has been observed that the CBs are overactivated causing an increased heart rate, respiratory rate, and blood glucose levels. In humans, this mechanism has been confirmed but is not yet well-characterized. This paper highlights the importance of developing a device that allows early detection of changes in CB activity correlating it with emerging diabetes. The design strategies to prototype the CBmeter were to model and characterize the features of interest for the diagnosis- respiratory rate, heart rate, peripheral oxygen saturation and glucose - in healthy people and people with diabetes using a combination of set commercial sensors pre-existent in the market that were integrated to collect real-time data. After determining health and disease patterns, the CBmeter development pipeline includes a co-design approach in which physiologists, endocrinologists, nurses, computer and electrical engineers, designers and patients are collaborating to develop an easy-to-use, portable, and minimally invasive medical device that associates CB function with endocrine dysregulation, with very small discomfort and risk for users. The definition and specification of the most appropriate architecture for the CBmeter, in order to allow its modularity, signal acquisition and consequently the communication between the sensor/device and the receiver/backend in the most efficient way is being allied to the selection of materials, tools and steps to create an innovative product, that will fill a technical gap in the market, designed for the early diagnosis of metabolic diseases, in a subclinical phase, with the potential to contribute with significant gains for public health in the medium/long term.

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Laden Hammoud, Shema El, Gabrielle Gruppelli Good, André Luiz Cristani Bizetto, and Anderson Matsubara. "Dissecção espontânea da artéria carótida em jovem: Um artigo de revisão." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.211.

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Background: Spontaneous dissection of the carotid artery is a significant cause of stroke in young people, and may be the etiology of up to 25% of ischemic strokes in this age group. Understanding the causes and clinical parameters about the disease is essential, since the perception of signs and symptoms that anticipate a cerebrovascular accident is an important prognostic factor. Objectives: Expand knowledge about the clinical and etiological mechanisms of spontaneous carotid dissection, in addition to the possibilities of diagnostic tools, providing an early approach to the disease. Methods: The study was carried out through the selection of scientific articles of systematic review on pathology, published in journals stored in the database of the Scientific Electronic Library Online (SCIELO), PUBMED and Google Scholar website. Results: The pathology is associated with several factors, including systemic arterial hypertension, type II diabetes mellitus, antiphospholipid antibody syndromes, mild cervical trauma and genetic alterations. Acute ipsilateral pain to the neck, associated with unilateral throbbing headache, was the most reported symptom. Conclusions: The perception of symptoms anticipating a cerebrovascular accident is essential to prevent secondary injuries. Although digital angiography is the gold standard test, MRI angiography and color Doppler ultrasound have been increasingly used during the acute phase of spontaneous carotid dissections.

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Siqueira, Fernando, Vanessa Siqueira, Lucas Falcão, Arthur Bezerra, and Carlos Silva. "THE INFLUENCE BETWEEN ALZHEIMER’S DISEASE AND HEALTHY EATING: A SYSTEMATIC REVIEW." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda062.

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Background: Alzheimer’s Disease (AD) is a neurodegenerative disease responsible for neuronal losses that affect mainly the cortex and hippocampus, which begin to shrink in size, damaging cognitive functions. This process affects cholinergic neurons, influencing acetylcholine (ACH) levels, a memoryrelated neurotransmitter. Glucose metabolism and low thiamine levels appear to be affected by AD. Consequently, diabetes becomes a disease associated with AD and the thiamine deficiency levels depress the use of glucose by the brain. Thus, nutrition may have a role in preventing dementia through the treatment and prevention. Objective: To summarize the knowledge about this topic by reviewing articles and analyzing if healthy eating influences the development of AD. Methods: Selection of articles from the Scielo database. Results: Inflammation contributes to the pathogenesis of AD. The effect observed in patients with adherence to the Mediterranean diet translates into a decrease in inflammatory markers at the plasma level. One of the symptoms, memory loss, can be prevented by a micronutrient named thiamine, a precursor of ACH, it is found in the leguminous. The Mediterranean diet has been shown to attribute a neuroprotective activity which goes with its anti-inflammatory effect. Conclusion: AD starts by its multifactorial etiology that consists of genotype and phenotype. Nutrition would be efficient as a preventive and a therapeutic alternative among other.

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Souza, Felipe Fanine de, Ana Luiza da Silva Wendhausen, Felipe Reinert Avilla Machado, Gustavo Figueiredo da Silva, Maria Eduarda Angelo de Mendonça Fileti, Raddib Eduardo Noleto da Nóbrega de Oliveira, Rafael Pereira Guimarães, et al. "Chorea in a Non-Ketotic Hyperglycemic State: Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.099.

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Context: Non-ketotic hyperglycemia is a rare cause of chorea. Clinical findings are usually unilateral and potentially reversible after treatment for hyperglycemia. Hyperglycemia leads to asymmetric multifocal petechial hemorrhages of the basal ganglia, leading to a dysfunction of neuronal networks that connect the basal ganglia and the motor cortical areas, mainly affecting the subthalamic nucleus and contralateral striatum, which is highlighted by typical hyperdense lesions of the basal ganglia in computed tomography (CT) of the brain. This study aimed to report a case of a patient with choreiform movements due to a rare etiology of hyperglycemia nonketotic in a Hospital Public of Joinville, SC. The study was carried out through the collection and analysis of a patient’s medical record. Case report: Female patient, 54 years old, who presented for 6 days choreiform movements in the face, left upper limb and, discreetly, in the left lower limb. Snake tongue sign and milkmaid’s grip positive, without dysarthria. In the laboratory exam, glucose of 600 mg / dL; without further changes. Cranial tomography showed hyperdensity in the putamen region on the right. The treatment was started to obtain better glycemic control and Risperidone 3 mg / day. Conclusions: It is concluded, then, that non-ketotic hyperglycemia is an uncommon, but reversible cause of chorea, and may manifest itself due to an uncontrolled non-ketotic diabetes mellitus. Its pathogenic mechanism remains to be clarified. In addition, clinical, epidemiological, imaging and laboratory findings, together, corroborate for early diagnosis and proper management.

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Reis, Ana Luísa Lopes Espínola da Costa, Leonardo Henrique Gandolfi de Souza, and Vitor Roberto Pugliesi Marques. "Ischemic stroke with agraphestesia signal focus." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.470.

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Introduction: The ischemic stroke is one of the main causes of death and disability in Brazil. Among the main risk factors are age, atrial fibrillation (AF), diabetes, dyslipidemia and physical inactivity. The main etiology of stroke is cardioembolic, resulting in obstruction of the cerebral arteries by a thrombus of cardiac origin. The artery most affected in ischemic strokes is the middle cerebral artery. The stroke has main characteristics, with emphasis on the sudden onset of symptoms, involvement of a focal area, ischemia caused by obstruction of a vessel and neurological deficits depending on the affected area. Graphesthesia is defined as a cutaneous sensory ability to recognize letters or numbers traced on the skin. The loss of this sensory ability is known as agraphesthesia. Case Report: M.A.F.O. female, 78a, arrived at the UPA complaining of mental confusion. Patient denies previous stroke. Personal history of systemic arterial hypertension. Upon physical examination, the patient was conscious, self and disoriented and inattentive. He was able to repeat and evoke words, without measurable motor déficits. Left upper limb with agraphestesia. Computed tomography was requested, which showed an extensive hypodense area in the right parietoccipital region, which leads to the erasure of the furrows between the adjacent gyres, which may correspond to a recent ischemic event. Magnetic resonance imaging, diffusion-restricted area with correspondence on the ADC map, located in the right temporoparietal region inferring an acute ischemic event. An electrocardiogram was also requested, which showed an irregular rhythm, characteristic of atrial fibrillation, resulting in a diagnostic hypothesis of cardioembolic ischemic stroke. Discussion: The involvement of post-central ischemic gyrus lesions may correspond to paresthesia, anesthesia, hypoesthesia; the involvement of secondary and terciary areas of sensitivity in the upper parietal lobe, especially in the active movements of the hand and in the modalities of integrated sensitivity, their lesions may be clinically affected by: apraxias, dysgraphias, hemineglect, agraphestesia, stereoagnosia and spacial disorientation.

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Gomes Mussi, Isis, Sthéfanny Figueiredo Mozzer, Marina Tosi Torres, and Valdebrando Mendonça Lemos. "Estudo da prevalência de etiologias da doença renal crônica em Campos dos Goytacazes." In Semana Científica da Faculdade de Medicina de Campos. Faculdade de Medicina de Campos, 2022. http://dx.doi.org/10.29184/anaisscfmc.v12022p9.

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Introdução: A Doença Renal Crônica (DRC) é caracterizada por redução da taxa de filtração glomerular por três meses ou mais, podendo ter ou não presença de lesão renal e constitui problema de saúde pública mundial, tendo como principais causas a hipertensão e o diabetes mellitus. O controle rigoroso destas permite prevenir e preservar a função renal. Após o diagnóstico de DRC torna-se necessário dar início a um programa de terapia renal substitutiva. Objetivos: Analisar, a partir dos prontuários dos acometidos com DRC em Campos dos Goytacazes, as causas prevalentes responsáveis por levar os pacientes ao programa de diálise. Métodos: Estudo transversal realizado a partir da coleta de informações dos prontuários no período janeiro de 2022 a dezembro de 2022. O projeto foi submetido à aprovação do diretor técnico dos dois serviços de tratamento dialítico existentes em Campos, bem como do Comitê de Ética em Pesquisa. Resultados e Discussão: A etiologia mais prevalente, até o momento, foi o diabetes (35,44%), seguido da hipertensão arterial (30,12%), o que soma a 140 e 119 pacientes, respectivamente, em um N=395. Conclusão: O diabetes e a hipertensão foram encontradas em mais de 65% dos pacientes. Os resultados foram compatíveis com o esperado.

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Reports on the topic "Diabetes – Etiology"

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Sosa Munguía, Paulina del Carmen, Verónica Ajelet Vargaz Guadarrama, Marcial Sánchez Tecuatl, Mario Garcia Carrasco, Francesco Moccia, and Roberto Berra-Romani. Diabetes mellitus alters intracellular calcium homeostasis in vascular endothelial cells: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0104.

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Review question / Objective: What are the effects of diabetes mellitus on the calcium homeostasis in vascular endothelial cells? -To describe the effects of diabetes on the mechanisms that regulate intracellular calcium; -To describe other molecules/mechanisms that alters intracellular Ca2+ homeostasis. Condition being studied: Diabetes mellitus is a pathology with a high incidence in the population, characterized by an increase in blood glucose. People with diabetes are 2-4 times more likely to suffer from a cardiovascular complication, such as total or partial loss of sight, myocardial infarction, kidney failure, among others. Cardiovascular complications have been reported to derive from dysfunction of endothelial cells, which have important functions in blood vessels. In order to understand the etiology of this poor function of endothelial cells, it is necessary to study the molecular mechanisms involved in these functions, to identify the effects of diabetes and thus, develop new research that will mitigate the effects of this pathology.

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Pu, Fenglan, Tianli Li, Yingqiao Wang, Chunmei Tang, Chen Shen, and Jianping Liu. Cordyceps preparations for preventing contrast-induced nephropathy: A protocol of systematic review of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0098.

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Review question / Objective: To systematically evaluate the efficacy and safety of cordyceps preparations as a complementary preventive therapy for Contrast-induced nephropathy (CIN). Condition being studied: At present, contrast agents are widely used in diagnostic and interventional radiology examinations worldwide. However, they can affect kidney function and cause a risk of renal impairment. Contrast-induced nephropathy (CIN) is defined as a rise in serum creatinine (SCr) levels by ≥ 25% of baseline or 44 µmol/l from the pre-contrast value within 72 h of intravascular administration of a contrast agent in the absence of an alternative etiology. The incidence of CIN varies widely among studies depending on study population and baseline risk factors, as for high-risk groups such as pre-existing renal insufficiency, diabetes, advanced age, or receiving nephrotoxic agents, the incidence is up to 30–50%. To date, CIN has been the third most common cause of hospital-acquired renal failure, after impaired renal perfusion and nephrotoxic medications, which can lead to longer hospital stay, increased costs and higher mortality.

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