Relevant bibliographies by topics / Diabetes – Complications

Academic literature on the topic 'Diabetes – Complications'

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Published: 4 June 2021
Last updated: 25 July 2024

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Journal articles on the topic "Diabetes – Complications"

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Mammen, Dr Sheena A. "Knowledge of Diabetes, its Treatment and Complications in Diabetic Patients." Journal of Medical Science And clinical Research 05, no. 05 (May 12, 2017): 21838–40. http://dx.doi.org/10.18535/jmscr/v5i5.99.

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Bloomgarden, Z. T. "Diabetes Complications." Diabetes Care 27, no. 6 (May 25, 2004): 1506–14. http://dx.doi.org/10.2337/diacare.27.6.1506.

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Fernandes, Tanya. "Diabetes complications." Nursing Standard 23, no. 25 (February 25, 2009): 58. http://dx.doi.org/10.7748/ns2009.02.23.25.58.c7174.

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Creaven, Matthew. "Diabetes complications." Nursing Standard 24, no. 5 (October 7, 2009): 59–60. http://dx.doi.org/10.7748/ns.24.5.59.s52.

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Creaven, Matthew. "Diabetes complications." Nursing Standard 24, no. 5 (October 7, 2009): 59. http://dx.doi.org/10.7748/ns2009.10.24.5.59.c7321.

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Bishop, Tina. "Diabetes complications." Primary Health Care 25, no. 2 (March 2, 2015): 14. http://dx.doi.org/10.7748/phc.25.2.14.s15.

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Irace, Concetta. "Awareness of Diabetes Complication in Subjects with Type 2 Diabetes." Diabetes & Obesity International Journal 7, no. 1 (2022): 1–5. http://dx.doi.org/10.23880/doij-16000251.

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Patients with diabetes are well-educated about the self-management of glycemic control; conversely, the education on the screening of complications are less structured and personalized. We designed our study to assess the awareness of complications in a sample of people with type 2 diabetes (T2D). This is an observational cross-sectional study. A questionnaire aimed to evaluate the knowledge of micro- and macro-vascular diabetic complications and of the tests used to detect them was provided to consecutive people with T2D. Three-hundred eleven participants with T2D were enrolled and competed the questionnaire. The majority of them were aware of retinopathy (98%), kidney disease (90%), cardiovascular diseases (57%), and leg sensitive abnormalities (83%), while few were aware of sexual (38%), bladder (45%), gastrointestinal (27%) and cardiovascular autonomic disorders (0.6%). Among those who were aware of sexual disorders, 33% defined the complication specific of male sex and 5% of both sexes. About one-third were aware of albuminuria, and 37% indicated electromyography as the standard test for peripheral neuropathy. An adequate level of awareness for most complications was observed. However, some complications linked to autonomic neuropathy and standard tests to detect diabetic nephropathy and peripheral neuropathy were poorly known. Furthermore, bladder, gastrointestinal disorders, and cardiac autonomic neuropathy were also less aware or unaware. A comprehensive education might be helpful to prevent the lesser-known complications and avoid inappropriate and expensive diagnostic tests.
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Susanto, Nugroho, and Yelli Yani Rusyani. "DIABETES COMPLICATIONS AS DIABETES PATIENTS PREDICTORS OF REFERRAL." Jurnal Berkala Epidemiologi 10, no. 1 (January 30, 2022): 68. http://dx.doi.org/10.20473/jbe.v10i12022.68-75.

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Background: Diabetes prevalence is growing faster in both developing and developed countries. Kidney failure, stroke, heart attack, leg amputation, and nerve damage are complications of diabetes caused by diabetes patients predictors of referral. Purpose: The study aims to describe diabetes complications as diabetes patients predictors of referral. Methods: The study design was used cross-sectional method. The population study is the participant with diabetes who have been treated in Public Health Care of Cangkringan from January 2018th until January 2019th. Samples were collected from total sampling who fulfilled the inclusion and exclusion criteria as complete medical records. The total sample was 414. Data of gender and age were collected from the medical record. Data of place was collected from GPS. Data of blood glucose level, complications and referral were collected from the medical record. Statistic test using chi-square and regression logistic. Results: Most diabetes patients were female, age > 55 years, diagnosis state long, distance ≤ 5 kilometres, no insulin-dependent, no complication, and no referral. Sex no significant differences PR = 0.91 (95% CI; 0.68-1.21), Age no significant different PR = 1.16 (95% CI; 0.88-1.51). Insulin significant different PR = 3.93 (95% CI: 3.17-4.88). Complication significant different PR = 3.53 (95% CI; 2.92-4.26). The main contributing to diabetes referral is a complication (β = 4.25; PR 71.20). Conclusion: The factors contributing to diabetes referral are insulin-dependent and complication, and the main factor contributing to diabetes referral is diabetes complication.
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Bohannon, Nancy J. V. "Complications of diabetes." Postgraduate Medicine 105, no. 2 (February 1999): 65. http://dx.doi.org/10.3810/pgm.1999.02.529.

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Sander, Ruth. "Complications with diabetes." Nursing Older People 21, no. 4 (May 21, 2009): 15. http://dx.doi.org/10.7748/nop.21.4.15.s29.

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Dissertations / Theses on the topic "Diabetes – Complications"

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Nordwall, Maria. "Long term complications in juvenile diabetes mellitus." Doctoral thesis, Linköping : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-6377.

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Choudhury, Maitrayee. "Complications in cystic fibrosis-related diabetes." Thesis, Cardiff University, 2017. http://orca.cf.ac.uk/100648/.

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Cystic fibrosis-related diabetes (CFRD) is a secondary form of diabetes, associated with increasing age in subjects with Cystic Fibrosis (CF). With improved life expectancy, CFRD is anticipated to increase in prevalence in addition to its complications. The aim of this study was to investigate the use of HbA1c as an early predictor of disease, as well as investigate microvascular and macrovascular complications in an adult CF cohort attending the All Wales Cystic Fibrosis Centre. The current method of using the conventional oral glucose tolerance test (OGTT) to diagnose CFRD was compared to using glycated haemoglobin (HbA1c). The findings demonstrated that a HbA1c value ≥ 5.5%/36mmol/mol was significantly predictive of the development of dysglycaemia over a 6-year period. The association between HbA1c and development of diabetic retinopathy (DR) was analysed. The study demonstrated 23% of CF patients with CFRD screened for DR had evidence of moderate to severe diabetic retinopathy. They had a higher HbAc1 and longer duration of CFRD compared to those without severe forms of DR. This suggests that microvascular complications are present in CFRD and to a similar extent as in type 1 diabetes mellitus. The prevalence of cardiac autonomic neuropathy (CAN) in CFRD was tested in 71 subjects with CF. CF subjects who were of an older age group demonstrated an inverse correlation with heart rate variability (HRV) during deep breathing (p < 0.05). CF dysglyaemic individuals with severe forms of diabetic retinopathy had reduced HRV during deep breathing compared to subjects with mild or no DR (p < 0.05). The presence of arterial stiffness in CFRD was examined in 65 CF subjects and 31 healthy volunteers. Age, gender and mean arterial pressure were significant predictors of increased augmentation index (AIx) and pulse wave velocity (PWV). Glycaemic control did not influence the arterial stiffness measurement outcomes. The CF group demonstrated a greater Aix than healthy volunteers (HV) (P < 0.05) when other variables were controlled in the analysis, suggesting possible increased inflammatory mechanism leading to increased Aix accounting for these findings. CF dysglycaemic subjects had greater PWV than CFNGT subjects which was only significant at the 10% level. The study findings demonstrate HbA1c has a predictive value in the diagnosis of CFRD based on a positive OGTT. Severe DR is prevalent in CFRD and is associated with a reduction in HRV during deep breathing. Glycaemic control is not predictive of arterial stiffness, in contrast to age, gender and MAP. Thus future consideration of the use of HbA1c may help to predict individuals with underlying dysglycaemia and reduce the risk of the development of associated microvascular complications.
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Wu, Di. "Discovery of glyco-biomarkers for diabetes and complications in diabetes." Thesis, University of Dundee, 2015. https://discovery.dundee.ac.uk/en/studentTheses/e2eee4c8-c74c-4fc6-8658-ab51ecd793de.

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Diabetes mellitus is a group of metabolic diseases characterized in disordered blood glucose levels with many altered genes, proteins and metabolites. The chronic hyperglycemia in diabetes damages organ systems and makes the diabetic patients vulnerable to one or more complications. The onset of diabetes and complications in diabetes are estimated to occur 5 to 10 years before the clinical diagnosis. The early diagnosis of diabetes prevents the damage and pathological progress of diabetes and complications in diabetes. The central hypothesis of this study was that the protein N-glycosylation pathway in diabetes might be influenced by elevated hexosamine flux in diabetes elevating sugar nucleotide levels. This, in turn, might affect the N-glycan branching pathway and result in detectable changes in plasma glycoprotein glycoforms. The aim of the study was to discover potential glyco-biomarkers for diabetes and complication in diabetes. Mass spectrometry based glycomic quantification was performed to look for alterations in N-glycan profiles between normal and diabetic plasma samples. Fucosylated N-glycans and intersected N-glycans were found to be up-regulated in diabetes with statistical significance. The elevated fucosylation in diabetes was verified at the glycosylation site level by quantitative Aleuria aurantia lectin (AAL) pull-down experiments. Fucosylated fetuin-A and α-1-acid glycoprotein were selected as candidate glyco-biomarkers. A lectin ELISA using F(abʹ)2 fragments as capture antibody was developed to validate the fucosylation changes by screening 20 normal and 20 diabetic patient plasma samples. We conclude that the fucosylation level of fetuin-A is a potential glyco-biomarker for diabetes. Additional work is necessary to see whether this glyco-biomarker correlates with any pathological complications of diabetes.
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Okafor, Eugene O. "Decreasing Acute Diabetes Complications Through Self-Management Education." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/5922.

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Diabetes mellitus is a chronic disease that affects millions of people in the United States. The purpose of this project was to develop a guideline to help clinical staff provide clear and concise diabetes self-management instructions to patients in a community setting. Orem's self-care deficit theory (SCD) and health belief model (HBM) provided a platform to assess how patients' self-care deficit contributes to illness and the effect of patients' perception of illness. SCD theory and the HBM provided the framework for the development of the guideline to decrease diabetes acute complications through self-management education. The practice-focused question was whether the diabetes treatment guideline would decrease diabetes complication, improve the quality of care received by the diabetic patients, and if the facility would adopt the developed guideline. AGREE II Tool was used to assess the quality of the guideline and the staffs' desire for the adoption of the guideline. Data were collected from questionnaires given to staff members at the practice site in 2 rounds. Six medical staff were asked to critique the initial guideline, and 5 medical professionals were asked to assess the final guideline. Most of the participants' scores indicated strong agreement that full consideration was met. The score in all 6 AGREE II domains was above 90%, and 100% of the participants recommended the guideline to be adopted in the facility. Data analysis indicated the diabetes practice guideline is valid, will enhance the treatment of diabetes, and the practice site employees were eager to adopt the treatment guideline. Findings may be used to increase population health and reduce acute complications from diabetes mellitus.
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Yu, Zhen. "Altered drug responses in diabetic and hypertensive-diabetic cardiomyopathy." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/29406.

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Diabetes mellitus has been associated with both clinical and experimental cardiac dysfunction. Diabetic cardiomyopathy which is characterized by depressed cardiac contractility is accompanied by a variety of biochemical changes in Ca⁺⁺ metabolism. This cardiomyopathy may occur in the presence of normal coronary arteries and normal blood pressure. However, some studies have shown that hypertension is more prevalent among diabetics and can aggravate the cardiovascular abnormalities associated with diabetes. To understand the mechanisms of diabetic cardiomyopathy and consequences of combined hypertension and diabetes, experiments were designed to measure cardiac tissue responses to various inotropic agents in experimental diabetes. Six weeks following streptozotocin (STZ) administration, Wistar, spontaneously hypertensive (SHR) and Wistar Kyoto (WKY) rats exhibited the 'classical signs' of diabetes which included: hyperglycemia, hypoinsulinemia, hyperlipidemia (except in WKY), and hypothyroidism. Decreased basal atrial rate and increased basal developed force (BDF) suggest a depressed SA node function and an alteration of Ca⁺⁺ utilization by diabetic ventricles. Decreased post quiescent potentiation (PQP) values (except in WKY) in ventricular tissues suggest a diminished amount of releasable Ca⁺⁺ from sarcoplasmic reticulum (SR). Decreased post stimulation potentiation (PSP) values in SHR papillary muscles (PM) are probably suggestive of a depressed sarcolemmal Na⁺-Ca⁺⁺ exchange function in this tissue. Diabetic rats show subsensitivity to β-adrenergic stimulation in ventricular tissues, supersensitivity and hyperresponsiveness to Ca⁺⁺ and α-adrenergic stimulation (except in WKY) in ventricular tissues and left atria (LA) and supersensitivity to BAY K 8644 in SHR LA and hyperresponsiveness to verapamil in ventricular strips. These alterations may be attributed to a change in receptor number and/or a post receptor alteration. Ryanodine decreased the PQP of Wistar and SHR PM and SHR LA in both controls and diabetics. It especially abolished PQP in SHR diabetic tissues, but had no effect on WKY tissues, which may suggest a difference in the SR function in these tissues. SR with impaired Ca⁺⁺ uptake may contribute to these phenomena in diabetic rats. Ryanodine also diminished (PQP + BDF) of SHR LA and (PQP/BDF) of Wistar and SHR PM, ˙but had no effects on control and other diabetic tissues. It appears that ryanodine has some influence on the Na⁺-Ca⁺⁺ exchange generated by sarcolemma (SL) of certain diabetic tissues. Further experiments are required to clarify this. SHR diabetic rats had greater changes in most of the measurements such as hyperlipidemia, depressed PQP and PSP values, and altered drug responses. This model exhibited very high mortality as compared to Wistar and WKY diabetic rats. As has been shown previously, the combination of hypertension and diabetes exerts a synergistic effect on the cardiac dysfunction in this model, and that altered lipid metabolism, SL and SR function are all involved in the development of cardiomyopathy. WKY diabetic rats, on the other hand, exhibited no significant changes in blood lipids, or in response to phenylephrine or to Ca⁺⁺ (LA) stimulation. Lack of change in these factors may explain the relatively normal cardiac function of this model as measured previously.
Pharmaceutical Sciences, Faculty of
Graduate
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Bunker, Richard David. "Enzymes associated with the complications of diabetes mellitus." Thesis, University of Auckland, 2010. http://hdl.handle.net/2292/7142.

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Diabetes mellitus (DM) is a metabolic disease resulting from failures in the production or response to the hormone insulin. Much of the pathogenesis and mortality attributed to DM are due to the long-term complications of hyperglycaemia, which is characteristic of the disease. This thesis presents structural and functional studies of two previously uncharacterised human enzymes, dihydrodipicolinate synthase-like protein (DHDPSL) and D-xylulokinase (XK). Both enzymes were revealed to have unexplored associations with DM. DHDPSL is distantly related (~25% sequence identity) to a family of Schiff base-dependent aldolases that include dihydrodipicolinate synthase and N-acetylneuraminate lyase. Despite these distant homologies the biological function of DHDPSL is unknown. It also does not map to any known metabolic pathway in humans, but is targeted to the mitochondrial compartment consistent with the presence of a mitochondrial targeting sequence. There are also strong associations between mutations in the Dhdpsl gene and primary hyperoxaluria type III a rare disorder of endogenous oxalate production. The DHDPSL crystal structure was determined by X-ray crystallography utilising in situ proteolysis of a fusion of DHDPSL with maltose-binding protein for crystallisation. Two apoforms and six Schiff base complexes with potential ligands were analysed at best to 2.0 A�� resolution and with an Rfree of 18.3%. DHDPSL is folded as (a/ss)���-barrel with a C-terminal subdomain and forms a tetramer in the crystal. The structural consequences of the diseaserelevant DHDPSL mutations were analysed and were found to largely affect the C-terminal subdomain. Findings also showed that DHDPSL acts as an oxaloacetate decarboxylase and is therefore likely to be a bifunctional oxaloacetate decarboxylase /4-hydroxy-2-ketoglutarate aldolase present in the liver and kidney mitochondria. Overall, these results revealed the presence of a potentially significant metabolic pathway in mitochondria whereby oxaloacetate can be converted to pyruvate. XK has a potential role in the regulation of de novo lipogenesis in the liver that has gained little previous attention. Excessive hepatic lipid accumulation is linked to impaired insulin response and the development of DM. XK was identified and produced recombinantly in Eschericia coli aided by molecular chaperones. Crystals suitable for structural analysis were obtained after five generations of repeated seeding. Five crystal structures were used to analyse substrate binding, the best of which was determined at 2.0 A�� resolution with an Rfree of 17.8%. XK assumes an actin-like two-domain FGGY sugar kinase fold. The most striking feature revealed by the XK molecular structure was a dramatic domain movement that must accompany catalysis. A competitive inhibitor of XK, 5-deoxy-5-fluoro-D-xylulose (Ki of 25.4 M) was also functionally validated and structurally analysed. A supplemental structural study of a major hypoxic response protein of unknown function, Rv1738, from the causative agent of tuberculosis, Mycobacterium tuberculosis is also described. This study presents the first novel protein structure to be determined by the racemic protein crystallography method. The Rv1738 structure exposes a relationship with a family of ribosome-inhibiting stress-response proteins that may be indicative of its function.
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Morgan, Eileen. "Type 1 diabetes - epidemiology, risk factors and complications." Thesis, Queen's University Belfast, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678213.

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This thesis examined the incidence and temporal trends of Type 1 diabetes diagnosed in Northern Ireland children using data from the Northern Ireland Childhood Diabetes Register (NICDR). Overall, there was evidence of a departure from linearity in incidence with indications that rates are levelling off in recent years. Further analyses also indicated that birth cohort effects were evident in the incidence rates suggesting that exposures in early life may play an aetiological role in this condition. A systematic review and meta-analyses was performed in this thesis to investigate the association of childhood vaccinations and subsequent risk of Type 1 diabetes. Twelve studies investigating a range of vaccinations were included. Results provided no evidence to suggest an association between childhood vaccinations and risk of Type 1 diabetes. A study using data from the Clinical Practice Research Datalink (CPRD) was included in this thesis to report findings on depression and other complications in young people diagnosed with Type 1 diabetes. This study found that rates of depression were significantly higher in cases with diabetes compared to controls without diabetes. Results also showed elevated rates of microvascular complications and significantly higher rates of cardiovascular disease compared to matched controls. Another focus of this thesis was on mortality in individuals with Type 1 diabetes. Population-based studies reporting relative mortality in Type 1 diabetes diagnosed in young people were systematically reviewed. In total, 23 independent studies were included. Associations between relative mortality and study/ country characteristics were explored. In addition to this review, a further two UK-based studies were performed to investigate mortality, one using data from the NICDR and the other using the CPRD. Both studies found excess mortality rates in individuals with Type 1 diabetes when compared, respectively, to the general population and to a group of controls without diabetes.
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Naito, Masaki. "Therapeutic Impact of Leptin on Diabetes, Diabetic Complications, and Longevity in Insulin-Deficient Diabetic Mice." Kyoto University, 2012. http://hdl.handle.net/2433/157453.

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Cross, Deborah F. "Genetic analysis of the microvascular complications of diabetes mellitus." Thesis, University of Plymouth, 2002. http://hdl.handle.net/10026.1/2316.

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There is increasing evidence to suggest that genetic factors are involved in the pathogenesis of microvascular complications in diabetes mellitus. Recent studies have suggested that genetic variations in the aldose reductase (ALR2) gene may contribute to the genetic susceptibility to microvascular complications. Aldose reductase is the first and rate-limiting enzyme of the polyol pathway and is implicated in the pathogenesis of diabetic microvascular disease (nephropathy, retinopathy and neuropathy). It has recently been shown that the three polymorphisms of the ALR2 gene are associated with susceptibility to microvascular complications in both TIDM and T2DM. The aim of this study was to investigate the CA dinucleotide repeat polymorphism (5'ALR2) that is located -2100bp and the C-106T substitution in the promoter region of the ALR2 gene, and also the A+ 11842C within intron 8 of the ALR2 gene itself. DNA from 285 Caucasoid patients with TIDM and well-defined microvascular disease and 120 normal healthy controls, as well as 60 Southern Indian patients with T2DM and 43 non diabetic controls were typed. The 5'ALR2 Z-2/X genotype was significantly increased in patients with nephropathy (n=92), retinopathy (n=160) and neuropathy (n=104) compared to those with no microvascular disease after 19 years duration of diabetes (uncomplicated, n=66) (46%, 41%, 42% vs. 24%, respectively). In contrast, the frequency of the Z+2/Y genotype (where Y is not Z-2) was significantly reduced in the patients with nephropathy, retinopathy and neuropathy compared to the uncomplicated (17%, 23%, 23% vs. 52%, respectively). Similar observations were made in the Southern Indian T2DM patients, however no significant differences were found. In the patients with TIDM the C-106 allele was associated with the Z-2 5'ALR2 allele. The C/Z-2 haplotype was present in 32% of the nephropaths, 32% of the retinopaths and 35% of the neuropaths compared to 11.5% of the uncomplicated. The A+ 11842 allele was also associated with the C-1 06 allele in TIDM patients with microvascular disease. The reported mitochondrial polymorphism (mt5178A/C) was not found in this. TIDM population, possibly due to differences in the background frequencies between ethnic groups. Family studies investigating the transmission of the 5'ALR2 and C-106T alleles from parents to offspring with diabetic nephropathy found preferential transmission of the Z-2 allele although this was not statistically significant. Functional studies of the activity of the ORE in TIDM patients with and without microvascular disease showed differences in the mean OREBP binding activity. OREB and OREC were found to have increased activity in response to hyperglycaemia in the complicated patients compared to the uncomplicated and normal controls. In conclusion, these results confirm the role of the aldose reductase gene in the genetic susceptibility to diabetic microvascular complications, and a possible role of the DI7S934 polymorphism in T2DM. These results also provide a novel insight into the role of the ORE of the ALR2 gene in the pathogenesis of diabetic microvascular complications. Further studies are now required to determine the molecular basis of these observations. Hopefully, in the future it will be possible to offer 'high risk' patients therapeutic intervention that will prevent the ravages of the long term complications of diabetes mellitus.
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Cho, Chi Shing. "Proteomic and medicinal approaches to diabetes and its complications." HKBU Institutional Repository, 2006. http://repository.hkbu.edu.hk/etd_ra/663.

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Books on the topic "Diabetes – Complications"

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K, Shaw, ed. Diabetic complications. Chichester: J. Wiley, 1996.

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Benson, William Edmunds. Diabetes and its ocular complications. Philadelphia: Saunders, 1988.

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C, Pickup John, and Williams Gareth MD, eds. Chronic complications of diabetes. Oxford: Blackwell Scientific Publications, 1994.

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Donnelly, Richard, and Edward Horton, eds. Vascular Complications of Diabetes. Malden, Massachusetts, USA: Blackwell Publishing, Inc., 2005. http://dx.doi.org/10.1002/9780470751503.

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Duvnjak, Marko, and Lea Smirčić-Duvnjak, eds. Gastrointestinal Complications of Diabetes. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-75856-5.

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G, McNally Paul, ed. Diabetes and cardiovascular complications. London: Science Press, 1999.

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Laurie, Coulston, and Australasian Association of Clinical Biochemists., eds. Diabetes, glycation and complications. Mt. Lawley, W.A: Australasian Association of Clinical Biochemists, 1996.

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Michael, Brownlee, and King George L, eds. Chronic complications of diabetes. Philadelphia: Saunders, 1996.

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1958-, Hirsch Irl B., and White N. H, eds. Acute complications of diabetes. Philadelphia: Saunders, 2000.

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Florida. Agency for Health Care Administration. and Florida. State Center for Health Statistics., eds. Health outcome series: Complications of diabetes study. [Tallahassee, Fla.]: The Agency, 1999.

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Book chapters on the topic "Diabetes – Complications"

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Wondisford, Fredric E. "Diabetes Complications." In Essentials of Endocrinology and Metabolism, 31–37. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39572-8_4.

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Harkless, Lawrence B., Jarrod Shapiro, and Lisa D. Breshars. "Foot Complications." In The Diabetes Textbook, 899–918. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11815-0_58.

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Harkless, Lawrence B., Jarrod Shapiro, and Joel Rodriguez-Saldana. "Foot Complications." In The Diabetes Textbook, 1021–39. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-25519-9_62.

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Christianakis, Stratos, Minh Chau Nguyen, and Richard S. Panush. "Musculoskeletal Complications of Diabetes Mellitus." In Diabetes, 239–51. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118405550.ch11.

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Caird, F. I. "Complications of Diabetes." In Diabetes in Elderly People, 83–90. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4899-3322-5_12.

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Fonseca, Vivian A., Merri Pendergrass, and Roberta Harrison McDuffie. "Complications of diabetes." In Diabetes in Clinical Practice, 41–57. London: Springer London, 2009. http://dx.doi.org/10.1007/978-1-84882-103-3_5.

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Choksi, Ishani, Shana Mencher, and Anisha Patel. "Acute Diabetes Complications." In Contemporary Endocrinology, 105–24. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-64133-7_10.

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Lansang, M. Cecilia, Richard David Leslie, Tahseen A. Chowdhury, and Keren Zhou. "Skin and musculoskeletal complications of diabetes." In Diabetes, 118–35. 2nd ed. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003240341-10.

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Shankarnarayan, Santosh. "Managing the Complications of Diabetes." In Managing Diabetes, 105–25. Tarporley: Springer Healthcare Ltd., 2012. http://dx.doi.org/10.1007/978-1-908517-81-4_6.

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Moini, Jahangir, Matthew Adams, and Anthony LoGalbo. "Pathophysiology of Diabetes." In Complications of Diabetes Mellitus, 2–10. Boca Raton: CRC Press, 2022. http://dx.doi.org/10.1201/9781003226727-1.

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Conference papers on the topic "Diabetes – Complications"

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Elcin, Huseyn. "EARLY IDENTIFICATION OF THE NEUROLOGICAL COMPLICATIONS OF DIABETES MELLITUS." In International Trends in Science and Technology. RS Global Sp. z O.O., 2021. http://dx.doi.org/10.31435/rsglobal_conf/30032021/7474.

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Diabetes mellitus is still a very common disease in the world and affects the daily lives of patients negatively. Diabetes is also known to be associated with neurological diseases such as peripheral nerve diseases, stroke and dementia. Among these, the most common disease is a peripheral nerve disease, and it has been reported that poor diabetic control increases the risk of development and can be prevented by education of the patients. Vascular dementia is more common in patients with diabetes than Alzheimer's disease, and it is thought that cerebrovascular diseases may berelated to cognitive impairment in diabetes. Although the mechanisms by which diabetes affects the brain are not clearly revealed, it is thought that changes in vascular structure, insulin resistance, glucose toxicity, oxidative stress, accumulation of glycation end products, hypoglycemic episodes and amyloid metabolism are effective.The aim of this article is to describe the neurological complications of diabetes and to emphasize the importance of patient education, good diabetes control and early diagnosis in preventing these complications.
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Vasil'ceva, O. YA, and K. N. Vitt. "Diabetes mellitus and thrombotic complications." In Scientific dialogue: Medical issues. ЦНК МОАН, 2019. http://dx.doi.org/10.18411/spc-15-05-2019-07.

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Kantawong, Krittika, Supan Tongphet, Panu Bhrommalee, Napa Rachata, and Sakkayaphop Pravesjit. "The Methodology for Diabetes Complications Prediction Model." In 2020 Joint International Conference on Digital Arts, Media and Technology with ECTI Northern Section Conference on Electrical, Electronics, Computer and Telecommunications Engineering (ECTI DAMT & NCON). IEEE, 2020. http://dx.doi.org/10.1109/ectidamtncon48261.2020.9090700.

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Jian, Yazan, Michel Pasquier, Assim Sagahyroon, and Fadi Aloul. "Using Machine Learning to Predict Diabetes Complications." In 2021 4th International Conference on Bio-Engineering for Smart Technologies (BioSMART). IEEE, 2021. http://dx.doi.org/10.1109/biosmart54244.2021.9677649.

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Elbarbary, Nancy. "8 How can we correlate time in range and complications?" In The 7th ASPED-ISPAD Diabetes Academy. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/bmjpo-2024-asped.8.

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Ayaka, Ono, Hironori Uchida, Yujie Li, and Yoshihisa Nakatoh. "Diabetes Diagnosis Using Plantar Thermogram Based on DenseNet." In 10th International Conference on Human Interaction and Emerging Technologies (IHIET 2023). AHFE International, 2023. http://dx.doi.org/10.54941/ahfe1004088.

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In Japan, the number of diabetics is rapidly increasing due to changes in lifestyle and social environment. In the early stages of diabetes, patients have few subjective symptoms and the disease may be left untreated for a long period of time. However, if metabolic abnormalities in diabetes persist over a long period of time, the likelihood of developing complications increases. Therefore, it is important to complete the diagnosis of diabetes as early as possible. The use of plantar thermography images is expanding as one way to determine diabetes. However, conventional techniques have not been evaluated to take into account the difficulties of acquiring images in actual use environments, such as out-of-focus or low-resolution cameras. This evaluation is essential in a practical diabetes detector. In this method, we created various simulated images assuming realistic usage environments and devices, and evaluated their impact on diabetes determination accuracy using Recall, Precision, and F-measure. The diabetes determination method uses DenseNet201, a convolutional neural network specifically designed for image classification. As a model, training is performed using only the source image, either single-foot images or both-foot images. The dataset consists of plantar thermogram images of 122 diabetic and 45 non-diabetic patients published by Hernandez et al. Due to the small amount of training data, the training was augmented with image processing such as rotation and reduction. For the original image, the Recall and F-measure for the single-foot image were 96.4% and 87.1% for the original image, and 100.0% and 78.9% for both-foot images, respectively. Considering the F-measure, the classification with a single foot as input data is relatively more accurate. Furthermore, even at 87.5% reduction, there was no effect of reduced resolution on the accuracy of diabetes determination, indicating that focusing has a significant effect on the accuracy of plantar thermography images.
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Mossel, DM, K. Moganti, H. Klüter, M. Harmsen, and J. Kzhyshkowska. "Hyperglycaemic control of histone code in metabolic inflammation and microvascular complications." In Diabetes Kongress 2018 – 53. Jahrestagung der DDG. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1641807.

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Zhu, Liang, and Robert Flower. "Role of Vasomotion in Control of Retina Edema in Diabetic Retinopathy: Quantification of Fluid Transport Through Retinal Capillaries." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-189507.

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Diabetic retinopathy refers to diabetes-related complications in the retina, It is a progressive disease and its symptoms in the eyes can vary from non-vision threatening to vision loss, and it can lead to permanent damage to the neuronal retinal tissue. The irreversible nature of the damage suggests that prevention of diabetes by eliminating risk factors and early screening are the cornerstone of relevant treatment to stop or limit visual damage in those patients.
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Custodi, P., G. P. Montecchio, C. Bendotti, G. Vandelli, M. T. Tenconi, and F. Piovella. "PLATELET FIBRONECTIN LEVELS AS A MARKER FOR THE PROGRESSION OF DIABETIC RETINOPATHY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643097.

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Aim of the study was to correlate fibronectin (Fn) and von Villebrand factor (vWf) levels measured in plasma and in platelets with the progression of diabetic retinopathy. Patients were classified in five groups reflecting the progression of this microvascular complication, on the basis of fluorangiographic findings (0 = no microangiopathy; 1= simple microangiopathy; 2= oedematous retinopathy; 3= ischaemic retinopathy; 4= ischaemic proliferative retinopathy). 43 patients were studied, 22 suffering from type I diabetes and 21 from type II diabetes, according to the classification of National Diabetes Data Group. Fn and vWf were measured in plasma and in platelet samples using an original double-sandwich microELISA method and expressed as micrograms/ml or as micrograms/10* platelets. Platelets were counted and solubilized with 0.5% Triton × 100. Bleeding time and platelet adhesion to glass beads were also evaluated on every patient. Intraplatelet Fn levels were reduced in retinopathies and correlate with the severity of the microvascular alteration, being the difference significant between the two extreme groups (p<0.05). vWf intraplatelet levels were also significantly lower in patients with severe microvascular complications (p<0.05). No significant differences were detected for plasma Fn and vWf levels in the 5 groups. Intraplatelet Fn and vWf levels may therefore be considered as markers of the severity of diabetic retinopathy. The leakage of Fn and vWf from activated platelet and the incorporation of these glycoproteins in the subendothelial matrix may be responsible for the worsening of this microvascular complication.
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Montani, N., S. B. Solerte, G. Gamba, M. Fioravanti, and E. Ferrari. "RELATIONSHIPS BETWEEN HAEMOSTATIC ENDOTHELIAL FUNCTIONS AND GLOMERULAR FILTRATION RATE IN SHORT-TERM TYPE I DIABETES MELLITUS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643101.

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It is known that the increase of glomerular filtration rate (GFR) represents an early sign of diabetic nephropathy. The changes of endothelial functions observed in diabetes might play a role in this respect. As F VIII vWF and fibronectin are synthetized by endothelial cells, we evaluated these components in 33 diabetic patients with short-term Type I (insulin dependent) diabetes mellitus, without retinopathy and macro-vascular complications. 15 pts. (mean age 29 ± 7 yrs; mean diabetes duration 2.9 ± 0.9 yrs) presented high GFR (154 ± 19 ml/min per 1.73 m2 ; albuminuria 7.2 ± 3.2 μg/min) and 18 pts. (mean age 30 ± 6 yrs; mean diabetes duration 3.0 ± 1 yrs) normal GFR (105 ± 11 ml/min per 1.73 m2 ; albuminuria 5 ± 2.8 μg/min).The following results were obtained:In conclusion the significant increase of FVIIIR:Ag and fibronectin levels in short-time type I diabetic patients with high GFR suggests an early endothelial cell function damage also related to the Door metabolic control.
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Reports on the topic "Diabetes – Complications"

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Trucco, Massimo. Towards a Possible Therapy for Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, October 2013. http://dx.doi.org/10.21236/ada606234.

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Trucco, Massimo. Towards a Possible Therapy for Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada606778.

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Trucco, Massimo. Towards a Possible Therapy for Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, October 2012. http://dx.doi.org/10.21236/ada613345.

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Trucco, Massimo. Towards A Possible Therapy for Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, December 2014. http://dx.doi.org/10.21236/ada619693.

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Jaganathan Geethalakshmi, Kruthica, Iyshwarya Bhaskar Kalarani, and Ramakrishnan Veerabathiran. Metabolic complications associated with lipodystrophic obesity and diabetes. Peeref, November 2022. http://dx.doi.org/10.54985/peeref.2211p4354869.

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Trucco, Massimo. Genetic and Environmental Pathways in Type 1 Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, September 2009. http://dx.doi.org/10.21236/ada544029.

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Trucco, Massimo. Genetic and Environmental Pathways in Type 1 Diabetes Complications. Fort Belvoir, VA: Defense Technical Information Center, September 2010. http://dx.doi.org/10.21236/ada544030.

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Ferdosian, Hengameh, Hadi Zamanian, Sayed Ali Emami, Elahe Sedighi, Mina Moridi, and Maryam Doustmehraban. Application of artificial intelligence in prediction of cardiovascular complications in patients with diabetes mellitus type 2: A protocol of systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0076.

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Review question / Objective: The aim of this systematic review is to evaluate AI-based models in identifying predictors of cardiovascular events and risk predtion in patients with diabetes mellitus type2. Condition being studied: T2DM patients have an increased risk of macrovascular and microvascular complications, lead to decreased quality of life and mortality. Considering the significance of cardiovascular complications in these patients, prediction of such events would be important. Different traditional statistical methods(such as regression) and new AI-besed algorithms are used to predict these complications in diabetic patients.
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Vakhlova, Irina, Irina Zaikova, Alexey Kiyaev, and Yulia Ibragimova. Electronic educational resource (EOR) "Module. Diabetes mellitus in children". SIB-Expertise, January 2024. http://dx.doi.org/10.12731/er0781.29012024.

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Diabetes mellitus occupies a leading place in the pathology of the endocrine system in children and adolescents and remains one of the most urgent health problems in most countries. In the last decade, the annual incidence of type 1 diabetes in children has shown a significant increase both in Russia and around the world. According to the International Diabetes Federation (IDF), it is increasing by 3% per year. In addition, in all European countries there is a "phenomenon of rejuvenation of diabetes" - an increase in the proportion of children who first fell ill with type 1 diabetes at a younger age: 25-30% up to 4 years of age; up to 80% - from 6 months to 9 years. The annual incidence of type 1 diabetes in children (0-14 years old) in the Sverdlovsk region over the past decade (2006-2017) has almost doubled: from 12.2 cases per 100 thousand children in 2006 to 23 ,7 in 2017 and occupies one of the leading places in the Russian Federation in this indicator. More than 200 children with type 1 diabetes are diagnosed per year, of which about 75% of children who become ill for the first time are under the age of 9 years. Type 1 diabetes is characterized by complete insulin dependence, severe course, early formation of specific complications that lead to a decrease in the quality and life expectancy. Unfortunately, in more than 70% of cases, DM is diagnosed at the stage of ketoacidosis, which requires urgent measures. The main reason for the late diagnosis of this disease is the lack of "diabetic alertness" among pediatricians and AFP physicians. The foregoing obliges a wide range of doctors, including pediatricians, to know the clinical and laboratory criteria for diagnosis, modern methods of monitoring and managing diabetes, possible complications and outcomes of the disease, and be able to provide emergency care. Timely diagnosis, self-monitoring, regular monitoring, prevention of complications is an opportunity to improve the quality of life of patients with diabetes.
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Sosa Munguía, Paulina del Carmen, Verónica Ajelet Vargaz Guadarrama, Marcial Sánchez Tecuatl, Mario Garcia Carrasco, Francesco Moccia, and Roberto Berra-Romani. Diabetes mellitus alters intracellular calcium homeostasis in vascular endothelial cells: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0104.

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Review question / Objective: What are the effects of diabetes mellitus on the calcium homeostasis in vascular endothelial cells? -To describe the effects of diabetes on the mechanisms that regulate intracellular calcium; -To describe other molecules/mechanisms that alters intracellular Ca2+ homeostasis. Condition being studied: Diabetes mellitus is a pathology with a high incidence in the population, characterized by an increase in blood glucose. People with diabetes are 2-4 times more likely to suffer from a cardiovascular complication, such as total or partial loss of sight, myocardial infarction, kidney failure, among others. Cardiovascular complications have been reported to derive from dysfunction of endothelial cells, which have important functions in blood vessels. In order to understand the etiology of this poor function of endothelial cells, it is necessary to study the molecular mechanisms involved in these functions, to identify the effects of diabetes and thus, develop new research that will mitigate the effects of this pathology.
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