Relevant bibliographies by topics / Detoxification

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Detoxification'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 May 2024

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Journal articles on the topic "Detoxification"

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Agyapong, Vincent, Jackie Benbow, and Roy Browne. "Effectiveness of daily outpatient alcohol detoxification by an Irish public psychiatric hospital – A pilot study." Irish Journal of Psychological Medicine 24, no. 1 (March 2007): 23–26. http://dx.doi.org/10.1017/s0790966700010119.

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AbstractObjective: To assess the effectiveness of daily outpatient alcohol detoxification in an Irish public psychiatric hospital.Method: The outpatient records of patients presenting to the Assessment Unit of St Brendan's Hospital in one year (August 2004-July 2005) with symptoms of Alcohol Dependence Syndrome (ADS) and commencing daily outpatient detoxification were examined retrospectively for parameters relevant to the objectives of the study. The results were compiled and analysed using descriptive statistics.Results: Forty patients underwent outpatient alcohol detoxification in one year and complete records were available for 32 patients (80%). Twenty patients had fixed addresses in the hospital catchment areas, eight patients had no fixed addresses and the remaining four patients had addresses outside the catchment areas. Seven patients (22%) presented with a co-morbid psychiatric condition including depression (four patients), anxiety disorder (two patients) and personality disorder (one patient). All seven patients were known to psychiatric sector services. Of the 32 patients commencing detoxification, 28 patients (87.5%) attended on the second day whilst 22 patients (69%) attended their third day's appointment. Only 17 patients (53%) completed the outpatient detoxification. Thirteen patients (40.6%) received at least two outpatient detoxifications during the year; of whom seven patients (58%) received their second detoxification within two months of the first one. The record of 20 patients (62.5%) showed that they had received advice regarding selfreferral to counselling services.Conclusion: A high proportion of patients (47%) presenting with symptoms of ADS did not complete daily outpatient detoxifications. A high proportion of all patients (40.6%) also underwent multiple outpatient detoxifications during the year. It is possible that the separation between alcohol detoxification and alcohol counselling services in Ireland contributed to these disappointing results.
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Hakansson, Anders, and Emma Hallén. "Predictors of Dropout from Inpatient Opioid Detoxification with Buprenorphine: A Chart Review." Journal of Addiction 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/965267.

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Inpatient withdrawal treatment (detoxification) is common in opioid dependence, although dropout against medical advice often limits its outcome. This study aimed to assess baseline predictors of dropout from inpatient opioid detoxification with buprenorphine, including age, gender, current substance use, and type of postdetoxification planning. A retrospective hospital chart review was carried out for inpatient standard opioid detoxifications using buprenorphine taper, in a detoxification ward in Malmö, Sweden(N=122). Thirty-four percent of patients(n=42)dropped out against medical advice. In multivariate logistic regression, dropout was significantly associated with younger age (OR 0.93 [0.89–0.97]) and negatively predicted by inpatient postdetoxification plan (OR 0.41 [0.18–0.94]), thus favouring an inpatient plan as opposed to outpatient treatment while residing at home. Dropout was unrelated to baseline urine toxicology. In opioid detoxification, patients may benefit from a higher degree of postdetoxification planning, including transition to residential treatment, in order to increase the likelihood of a successful detoxification and treatment entry. Young opioid-dependent patients may need particular attention in the planning of detoxification.
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Balston, Alfred, Kuljit Hunjan, and Michael J. Kelleher. "The use of chlordiazepoxide for outpatient gamma-butyrolactone (GBL) detoxification: An observational study." Drug Science, Policy and Law 9 (January 2023): 205032452311675. http://dx.doi.org/10.1177/20503245231167544.

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Background Various detoxification regimens are used for gamma-hydroxybutyrate (GHB) and gamma-butyrolactone (GBL), including diazepam, barbiturates, baclofen and GHB itself. However, these regimens are primarily derived from inpatient units, and literature on outpatient GBL detoxification is sparse with no previous reports on chlordiazepoxide. We describe the characteristics of outpatient GBL detoxification using chlordiazepoxide. Methods Observational study of all patients who attended a community outpatient addiction service in South London between August 2015 and November 2017 seeking detoxification from GBL. The outpatient caseload is predominantly patients with alcohol, opioid and stimulant dependence. Routine clinical data including patient demographics, GBL usage, daily chlordiazepoxide dose and Clinical Institute Withdrawal Assessment for Alcohol-revised (CIWA-Ar) score were recorded. Results In the study period there were 17 attendances for GBL detoxification, 14 of which were undertaken in the outpatient setting. Twelve (86%) patients who had an outpatient detoxification were male, all of whom were men who have sex with men. Of 14 outpatient GBL detoxifications managed with chlordiazepoxide, 10 were successfully completed. One of the four patients that did not complete detoxification required inpatient treatment in an acute hospital. The average successful detoxification took 10 ± 3.1 days. For patients who completed a detoxification, the median maximum CIWA-Ar score on day one of the detoxification was 11 (range 2–17), with the mean dose of chlordiazepoxide used on day one being 140 mg (range 80–225 mg). Conclusions Chlordiazepoxide can be used for outpatient GBL detoxification in combination with a provision for crisis admission to hospital. CIWA-Ar score can be applied to GBL withdrawal to measure severity and inform a reducing regimen of chlordiazepoxide.
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Dell, Esther Y. "Detoxification." Journal of Consumer Health On the Internet 10, no. 3 (September 5, 2006): 105–9. http://dx.doi.org/10.1300/j381v10n03_10.

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Mason, Russ. "Detoxification." Alternative and Complementary Therapies 7, no. 4 (August 2001): 254–55. http://dx.doi.org/10.1089/107628001750424625.

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Stellato-Kabat, Douglas. "Acupuncture Detoxification." Social Work 39, no. 5 (September 1994): 623–24. http://dx.doi.org/10.1093/sw/39.5.623-a.

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Bolton, James R. "Solar detoxification." Solar Energy 56, no. 5 (May 1996): 375. http://dx.doi.org/10.1016/0038-092x(96)81766-x.

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Raushel, Frank M. "Catalytic detoxification." Nature 469, no. 7330 (January 2011): 310–11. http://dx.doi.org/10.1038/469310a.

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Palmer, Sue. "Detoxification school." Early Years Educator 8, no. 10 (January 2007): 32–34. http://dx.doi.org/10.12968/eyed.2007.8.10.22616.

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Bischof, Gary P., Judith A. Booker, Teresa L. Dyck, Elizabeth A. Graney, Irene P. Hamblen, Cheryl L. B. Hittinger, Mary K. Holzinger, and Christopher Smith. "Outpatient Detoxification." Alcoholism Treatment Quarterly 8, no. 2 (September 24, 1991): 119–29. http://dx.doi.org/10.1300/j020v08n02_11.

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Dissertations / Theses on the topic "Detoxification"

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Belford, Paul S. "Detoxification of organotin biocides." Thesis, Imperial College London, 1987. http://hdl.handle.net/10044/1/38234.

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Черниш, Єлізавета Юріївна, Елизавета Юрьевна Черныш, and Yelyzaveta Yuriivna Chernysh. "The sewage sludge detoxification." Thesis, Сумський державний університет, 2013. http://essuir.sumdu.edu.ua/handle/123456789/33561.

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Recycling of organic sewage sludge today is an urgent problem in the whole territory of Ukraine, which requires a solution. At the most of the municipal wastewater treatment plants, unfortunately, current removal, treatment and recycling of sewage sludge are not properly resolved. At present, the general part of sewage sludge is not carried out. Because it contains toxic chemicals, mainly heavy metals (HM) within the industrial waste water coming into the city sewer system after insufficient treatment or without treatment. As a result, sewage sludge is sent to the sludge pit and storage sites, which are assigned to the large land area. Therefore the process of HM removing from sewage sludge is of paramount importance to allow using sewage sludge as an organic fertilizer in agriculture. When you are citing the document, use the following link http://essuir.sumdu.edu.ua/handle/123456789/33561
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Ezzi, Mufaddal I. "Cyanide detoxification by soil microorganisms." Thesis, University of Surrey, 2001. http://epubs.surrey.ac.uk/842816/.

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Cyanides enter the environment through both natural and man-made sources. Natural sources include cyanogenesis by bacteria, fungi and plants. A number of cyanide catabolising microorganisms have also been reported in literature. This is the first reported instance of cyanide catabolism in Trichoderma harzianum. Four strains of T. harzianum, one of T. pseudokoningii were evaluated. An investigation was made into the occurrence and distribution of the cyanide catabolising enzymes. Three enzymes, cyanide hydratase, beta-cyanoalanine synthase and rhodanese, were studied. All the strains showed a high capacity to degrade cyanide via both the cyanide hydratase and rhodanese pathways, beta-cyanoalanine synthase activity, however, was not detected in any of the selected strains. In the studies on the kinetic characterization of the rhodanese enzyme, a broad pH optimum of 8.5 - 10.5 was obtained for all the strains and a broad temperature optimum of 35 - 55 °C was also observed. The KmCN and Vmax values ranged from 7-16 mM and from 0.069 - 0.093 betamoles. Min-1. mg protein-1, respectively, between the selected strains of Trichoderma. Strong evidence of cyanide biodegradation and co-metabolism emerged from studies with flask cultures where glucose was provided as a co-substrate. The rate of degradation of 2000 ppm CIST was enhanced almost three times in the presence of glucose. Plant microcosm studies carried out using pea and wheat seeds too gave further corroboration of the cyanide degrading and plant growth promotion capabilities of Trichoderma. Microcosms set-up with cyanide at 50 or 100 ppm CN, in the presence of Trichoderma, showed germination of both pea and wheat seeds. There was no seed germination in any of the controls in the absence of Trichoderma inoculation.
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MacLean, Morag. "Methylglyoxal detoxification in Escherichia coli." Thesis, University of Aberdeen, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.287604.

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Methylglyoxal is a naturally occurring toxic electrophile which, in E. coli, can be detoxified by several routes. This study has focused on the glutathione-dependent glyoxalase system. The genes encoding glyoxalase I (gloA) and glyoxalase II (gloB) were identified in the E. coli genome and cloned into the multi-copy plasmid pHG165. A null mutant in glyoxalase I (ΔgloA::KanR) was also created. Cells overexpressing glyoxalase I exhibit elevated rates of detoxification and enhanced tolerance of methylglyoxal. Analysis of the ΔgloA mutant has revealed that growth and viability are quite normal, unless the cell is challenged with methylglyoxal either added exogenously or synthesized by the cells. The mutant strain has a low rate of detoxification of methylglyoxal; only 20 -30 % of the level of the detoxification compared to the parent strain. Thus, the glutathione-dependent glyoxalase system was shown to be the dominant pathway for the methylglyoxal detoxification in E. coli. Glyoxalase I mutant cells rapidly lose viability when exposed to methylglyoxal as cell viability parallels the rate of detoxification. Glyoxalase I is the rate determining step in this pathway as overexpression of glyoxalase II does not affect the overall rate of metabolism of methylglyoxal. Methylglyoxal-elicited potassium efflux via the E. coli potassium channel KefB is enhanced in strains overexpressing glyoxalase I. Activation of KefB is diminished in the absence of functional glyoxalase I or in a strain overexpressing glyoxalase II. S-lactoylglutathione is the product of glyoxalase I and the substrate for glyoxalase II. This glutathione adduct is the major activator of the KefB and KefC systems. The glyoxalase I substrate, hemithiolacetal, could partially activate KefB and KefC when they were overexpressed on a multi-copy plasmid.
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Pillai, Jitesh Kannan. "Mechanisms of Arsenic Detoxification and Resistance." FIU Digital Commons, 2014. http://digitalcommons.fiu.edu/etd/1699.

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Arsenic is a ubiquitous environmental toxic substance. As a consequence of continual exposure to arsenic, nearly every organism, from Escherichia coli to humans have evolved arsenic detoxification pathways. One of the pathways is extrusion of arsenic from inside the cells, thereby conferring resistance. The R773 arsRDABC operon in E. coli encodes an ArsAB efflux pump that confers resistance to arsenite. ArsA is the catalytic subunit of the pump, while ArsB forms the oxyanion conducting pathway. ArsD is an arsenite metallochaperone that binds arsenite and transfers it to ArsA. The interaction of ArsA and ArsD allows for resistance to As(III) at environmental concentrations. The interaction between ArsA ATPase and ArsD metallochaperone was examined. A quadruple mutant in the arsD gene encoding a K2A/K37A/K62A/K104A ArsD is unable to interact with ArsA. An error-prone mutagenesis approach was used to generate random mutations in the arsA gene that restored interaction with the quadruple arsD mutant in yeast two-hybrid assays. Three such mutants encoding Q56R, F120I and D137V ArsA were able to restore interaction with the quadruple ArsD mutant. Structural models generated by in silico docking suggest that an electrostatic interface favors reversible interaction between ArsA and ArsD. Mutations in ArsA that propagate changes in hydrogen bonding and salt bridges to the ArsA-ArsD interface also affect their interactions. The second objective was to examine the mechanism of arsenite resistance through methylation and subsequent volatilization. Microbial ArsM (As(III) S-adenosylmethyltransferase) catalyzes the formation of trimethylarsine as the volatile end product. The net result is loss of arsenic from cells. The gene for CrArsM from the eukaryotic green alga Chlamydomonas reinhardtii was chemically synthesized and expressed in E. coli. The purified protein catalyzed the methylation of arsenite into methyl-, dimethyl- and trimethyl products. Synthetic purified CrArsM was crystallized in an unliganded form. Biochemical and biophysical studies conducted on CrArsM sheds new light on the pathways of biomethylation. While in microbes ArsM detoxifies arsenic, the human homolog, hAS3MT, converts inorganic arsenic into more toxic and carcinogenic forms. An understanding of the enzymatic mechanism of ArsM will be critical in deciphering its parallel roles in arsenic detoxification and carcinogenesis.
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Hamel, Robert D. "Aluminum detoxification mechanisms in Pseudomonas fluorescens." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0025/MQ31433.pdf.

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Hou, Shurong. "HUMAN BUTYRYLCHOLINESTERASE MUTANTS FOR COCAINE DETOXIFICATION." UKnowledge, 2014. http://uknowledge.uky.edu/pharmacy_etds/38.

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Cocaine is one of the most reinforcing drugs of abuse and has caused serious medical and social problems. There is no FDA-approved medication specific for cocaine. It is of a high priority to develop an effective therapeutic treatment for cocaine abuse. Human butyrylcholinesterase (BChE) has been recognized as a promising candidate of enzyme therapy to metabolize cocaine into biologically inactive metabolites and prevent it from reaching central nervous system (CNS). However, the catalytic activity of wide-type human BChE against cocaine is not sufficiently high for treatment of cocaine abuse. Dr. Zhan’s lab has successfully designed and discovered a series of high-activity mutants of human BChE specific for cocaine metabolism. This dissertation is mainly focused to address the possible concerns in further development of promising human BChE mutants for cocaine detoxification, including whether the administration of this exogenous enzyme will affect the cholinergic system, whether it can efficiently hydrolyze cocaine’s toxic metabolites, and whether the commonly used therapeutic agents will significantly affect the catalytic activity of the BChE mutants against cocaine when they are co-administered. According to the results obtained, all of the examined BChE mutants have a considerably improved catalytic efficiency against (-)-cocaine, without significantly improving the catalytic efficiency against any of the other examined substrates, including neurotransmitter acetylcholine. Two representative mutants (including E12-7) also have a considerably improved catalytic activity against cocaethylene (formed from combined use of cocaine and alcohol) compared to wild-type BChE, and E12-7 can rapidly metabolize cocaethylene, in addition to cocaine, in rats. Further evaluation of possible drug-drug interactions between E12-7 and some other commonly used therapeutic agents revealed that all of the examined agents, except some tricyclic antidepressants, do not significantly inhibit E12-7. In addition, an effort to discover new mutants with further improved activity against cocaine led to the discovery of a new BChE mutant, denoted as E20-7, according to both the in vitro and in vivo assays. The encouraging outcomes of the present investigation suggest that it is possible to develop a more effective enzyme therapy for cocaine abuse treatment using one of the most promising BChE mutants, such as E12-7 or E20-7.
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Brown, Stanley. "Heavy metal detoxification of sewage sludge." Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.302700.

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Zhang, Shiying. "Biological Detoxification of Mercury Contaminated Soil." DigitalCommons@USU, 1991. https://digitalcommons.usu.edu/etd/5384.

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This study examined biological mercury removal from soil using mercury-resistant bacteria in soil microcosms. Mercuric chloride was used to artificially contaminate Kidman soil to mercury concentrations of 5 ppm and 10 ppm. Soil moisture content was maintained at three levels, 20%, 30% and 50%. Mercury resistant-bacteria were added to soil samples and the mercury removal rate was compared to control samples without added bacteria. Mercury removal rate was initially enhanced by the addition of bacteria. After 30 days, no difference was observed between samples and controls with initial mercury concentration of 5 ppm when soil moisture content was 20%. At an initial mercury concentration of 10 ppm, soil samples had less mercury remaining than controls after 30 days. Autoclaved soil had a decreased mercury removal rate compared to soil not autoclaved. Addition of nutrient (sucrose) did not increase the mercury removal rate. A slurry-type bioreactor was found to be more efficient than a non-stir type. After 30 days of continuous stirring, 85-90% of the added mercury (10 ppm) was removed, while under the same conditions except no stirring, only around 60% of the mercury was removed. Overall, biological detoxification of mercury from contaminated soil can be achieved by using a slurry-type bioreactor with additon of mercury-resistant bacteria.
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Lawson, Kathryn René. "Catabolism as a mechanism of polyamine detoxification." Diss., The University of Arizona, 1998. http://hdl.handle.net/10150/288919.

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Maintenance of optimal polyamine pool levels is critical for cell survival. Intracellular polyamine depletion is usually cytostatic, whereas unregulated polyamine accumulation can result in cytotoxicity. The purpose of this work was to examine the importance of polyamine depletion in cell survival, either through increased polyamine catabolism or decreased polyamine synthesis. The polyamine analogue CHENSpm, which induces apoptosis in several cell lines, was used to examine the role of polyamine catabolism in cell survival. The susceptibility of Chinese hamster ovary cells and HCT 116 human colon cells to CHENSpm-mediated toxicity was inversely correlated with the level of polyamine oxidase (PAO) activity present in each cell type. Chinese hamster ovary cells (CHO), which contained high levels of PAO, were not growth inhibited by CBENSpm, however concomitant PAO inhibition led to a moderate growth suppression. The inhibition of the diamine exporter (DAX) in addition to PAO led to a CHENSpm-mediated cytotoxic response that was manifested as apoptosis induction. HPLC analysis of CHENSpm- treated CHO cell extracts revealed the presence of an unidentified amine that was not present when PAO was inhibited. This suggests that PAO is able to utilize CHENSpm as a substrate, and that this metabolism protects cells from CHENSpm-mediated cytotoxicity. The effect of polyamine depletion in apoptosis induced by the non-steroidal anti-inflammatory drug (NSAID) sulindac was examined in cells harboring an activated Ki-ras. Cells overexpressing Ki-ras underwent an accelerated apoptosis induction with either metabolite of sulindac, however overall toxicity was unaffected in long-term survival assays. DFMO did not affect apoptosis induction by sulindac sulfone, nor did it increase sulindac sulfone toxicity in long-term survival studies. DFMO alone was selectively cytotoxic to Ki-ras transfected clones in a dose-dependent manner. Ki-ras transfection increased c-myc expression, but had no effect on ODC steady-state mRNA levels. The downregulation of N1-spermidine/spermine acetyltransferase (SSAT) seen in Ki-ras transfected cells suggests polyamine, catabolism may protect cells from DFMO-induced cytotoxicity. These studies demonstrate that polyamine, catabolism may play an important role in cell survival under conditions of suboptimal polyamine levels.
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Books on the topic "Detoxification"

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Huggins, Hal A. Detoxification. Colorado Springs, CO: H.A. Huggins, 1990.

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Sixto, Malato Rodríguez, ed. Solar detoxification. Paris: UNESCO Pub., 2003.

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Problem drinking: Experiments in detoxification : report of the Detoxification Evaluation Project. London: Bedford Square, 1985.

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Medicine, Institute of Functional. Detoxification: A clinical monograph. Gig Harbor, WA: The Institute for Functional Medicine, 1999.

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Gan, Jay J., Peter C. Zhu, Steven D. Aust, and Ann T. Lemley, eds. Pesticide Decontamination and Detoxification. Washington, DC: American Chemical Society, 2003. http://dx.doi.org/10.1021/bk-2004-0863.

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Sherameti, Irena, and Ajit Varma, eds. Detoxification of Heavy Metals. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-21408-0.

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Chen, Chang-Hwei. Activation and Detoxification Enzymes. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1049-2.

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1963-, Gan Jay J., American Chemical Society. Division of Environmental Chemistry, and American Chemical Society Meeting, eds. Pesticide decontamination and detoxification. Washington, DC: American Chemical Society, 2004.

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Varma, A., and Irena Sherameti. Detoxification of heavy metals. Heidelberg: Springer, 2011.

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Chen, Chang-Hwei. Activation and Detoxification Enzymes. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-55287-8.

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Book chapters on the topic "Detoxification"

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Hayes, John D. "Detoxification." In Encyclopedia of Cancer, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_1590-2.

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Simola, Nicola, Micaela Morelli, Tooru Mizuno, Suzanne H. Mitchell, Harriet de Wit, H. Valerie Curran, Celia J. A. Morgan, et al. "Detoxification." In Encyclopedia of Psychopharmacology, 402. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1454.

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Hayes, John D. "Detoxification." In Encyclopedia of Cancer, 1354–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_1590.

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Hayes, John D. "Detoxification." In Encyclopedia of Cancer, 1101–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_1590.

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Scanlan, Nancy. "Detoxification." In Complementary Medicine for Veterinary Technicians and Nurses, 79–101. Ames, Iowa, USA: Blackwell Publishing Ltd., 2013. http://dx.doi.org/10.1002/9781118788950.ch7.

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Korn, Leslie E. "Detoxification." In Rhythms of Recovery, 231–44. New York: Routledge, 2021. http://dx.doi.org/10.4324/9781003171515-10.

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Kljajic, Jennifer, and Ahmed Zaafran. "Detoxification Strategies." In Advanced Therapeutics in Pain Medicine, 359–64. First edition. | Boca Raton : CRC Press, 2021.: CRC Press, 2020. http://dx.doi.org/10.1201/9780429504891-23.

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Cook, Tim. "Detoxification Centres." In Vagrant Alcoholics, 107–14. London: Routledge, 2023. http://dx.doi.org/10.4324/9781032616414-6.

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Martinetz, D. "Detoxification and Decomposition." In Chemical Waste, 113–265. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-69625-1_9.

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Cabrera, Alejandro, Sara Miralles, and Lucas Santos-Juanes. "Solar Water Detoxification." In Solar Resources Mapping, 341–51. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-97484-2_15.

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Conference papers on the topic "Detoxification"

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Rozhko, T. V., and N. S. Kudryasheva. "Bioluminescent monitoring of detoxification processes." In Fifth International Conference of CIS IHSS on Humic Innovative Technologies «Humic substances and living systems». CLUB PRINT ltd., 2019. http://dx.doi.org/10.36291/hit.2019.rozhko.076.

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Konodyuk, Nikita. "Prompt Tuning for Text Detoxification." In Dialogue. RSUH, 2022. http://dx.doi.org/10.28995/2075-7182-2022-21-1089-1096.

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Text detoxification is a challenging style transfer task, that implies paraphrasing into a neutral form while preserving the meaning as closely as possible. In this paper, we present a lightweight approach based on a recently proposed prompt tuning technique. Using RuGPT3-XL (Generative Pretrained Transformer-3 for Russian) as a frozen backbone, we train only a sequence of continuous embeddings inserted before and after an input text. Even though the number of trainable parameters is less than 0.025% of their total number, our approach achieves competitive performance compared to the methods involving full model tuning and ranks 4th on the leaderboard of shared RUSSE Detox task.
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Logacheva, Varvara, Daryna Dementieva, Sergey Ustyantsev, Daniil Moskovskiy, David Dale, Irina Krotova, Nikita Semenov, and Alexander Panchenko. "ParaDetox: Detoxification with Parallel Data." In Proceedings of the 60th Annual Meeting of the Association for Computational Linguistics (Volume 1: Long Papers). Stroudsburg, PA, USA: Association for Computational Linguistics, 2022. http://dx.doi.org/10.18653/v1/2022.acl-long.469.

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Gusev, Ilya. "Russian Texts Detoxification with Levenshtein Editing." In Dialogue. RSUH, 2022. http://dx.doi.org/10.28995/2075-7182-2022-21-264-272.

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Text detoxification is a style transfer task of creating neutral versions of toxic t exts. In this paper, we use the concept of text editing to build a two-step tagging-based detoxification model using a parallel corpus of Russian texts. With this model, we achieved the best style transfer accuracy among all models in the RUSSE Detox shared task, surpassing larger sequence-to-sequence models.
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Pletenev, Sergey. "Between Denoising and Translation: Experiments in Text Detoxification." In Dialogue. RSUH, 2022. http://dx.doi.org/10.28995/2075-7182-2022-21-447-455.

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This paper describes a solution for the RUSSE Detoxification competition held as part of the Dialogue 2022 conference. The paper presents experiments based on autoregressive and non-autoregressive models. The following approaches are described in this paper: 1) Detoxification as a special case of the text style-transfer problem and the use of modern approaches to solve this task in Russian. 2) Using the Automatic Post-Editing algorithm as a task of translation from toxic to normative Russian text. The article provides an analysis of the listed models, their results in detoxification of sentences, as well an analysis of errors and reasons why the models gave such a diverse result.
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Shishkin, D. A., A. A. Kolpakova, P. V. Udovik, I. N. Melnikov, and S. Ia Pichkhidze. "Development of a SO2 detoxification respirator." In ТЕНДЕНЦИИ РАЗВИТИЯ НАУКИ И ОБРАЗОВАНИЯ. НИЦ «Л-Журнал», 2015. http://dx.doi.org/10.18411/lj2015-12-23.

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Kriz, P., P. J. Kadlec, M. Dienstbier, R. Mikulíková, J. Cerman, J. Hladik, and P. Spatenka. "Detoxification of Seeds by Plasma Treatment." In Society of Vacuum Coaters Annual Technical Conference. Society of Vacuum Coaters, 2013. http://dx.doi.org/10.14332/svc13.proc.1117.

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Clothiaux, E., and W. Neely. "Detoxification of hazardous compounds using plasma discharges." In 33rd Aerospace Sciences Meeting and Exhibit. Reston, Virigina: American Institute of Aeronautics and Astronautics, 1995. http://dx.doi.org/10.2514/6.1995-246.

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Shao, Han, and Zhencai Chen. "Application of Artificial Intelligence in Drug Detoxification." In ISAIMS 2022: 2022 3rd International Symposium on Artificial Intelligence for Medicine Sciences. New York, NY, USA: ACM, 2022. http://dx.doi.org/10.1145/3570773.3570860.

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Kwak, Jin Myung, Minseon Kim, and Sung Ju Hwang. "Language Detoxification with Attribute-Discriminative Latent Space." In Proceedings of the 61st Annual Meeting of the Association for Computational Linguistics (Volume 1: Long Papers). Stroudsburg, PA, USA: Association for Computational Linguistics, 2023. http://dx.doi.org/10.18653/v1/2023.acl-long.565.

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Reports on the topic "Detoxification"

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Wild, James R. Detoxification of Acetylcholinesterase Inhibitors. Fort Belvoir, VA: Defense Technical Information Center, February 1987. http://dx.doi.org/10.21236/ada184892.

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Darcy, Philip. Waste Acid Detoxification and Reclamation. Fort Belvoir, VA: Defense Technical Information Center, April 1999. http://dx.doi.org/10.21236/ada607453.

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Raushel, Frank M. Enzymatic Detoxification of Chemical Warfare Agents. Fort Belvoir, VA: Defense Technical Information Center, December 2003. http://dx.doi.org/10.21236/ada421843.

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Majumder, S., M. Prairie, J. Shelnutt, and S. Khan. Engineered photocatalysts for detoxification of waste water. Office of Scientific and Technical Information (OSTI), December 1996. http://dx.doi.org/10.2172/420402.

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Varma, R. Microwave fluidized-bed detoxification of hazardous waste. Office of Scientific and Technical Information (OSTI), November 1991. http://dx.doi.org/10.2172/6022896.

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Doty, S., N. Widmer, K. Beninga, and J. Cole. Fabrication and Testing for Solar Detoxification Project. Fort Belvoir, VA: Defense Technical Information Center, December 1997. http://dx.doi.org/10.21236/ada337946.

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Kaar, Joel L., Richard Koepsel, and Alan J. Russell. Biocatalytic Buffering System for Detoxification of Nerve Agents. Fort Belvoir, VA: Defense Technical Information Center, November 2005. http://dx.doi.org/10.21236/ada440989.

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Ow, David W. ow@pgec ams usda gov. Molecular Genetics of Metal Detoxification: Prospects for Phytoremediation. Office of Scientific and Technical Information (OSTI), September 2000. http://dx.doi.org/10.2172/781718.

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Ow, David W. Molecular Genetics of Metal Detoxification: Prospects for Phytoremediation. Office of Scientific and Technical Information (OSTI), June 1999. http://dx.doi.org/10.2172/828166.

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Mehos, M., T. Williams, and C. Turchi. Overview of solar detoxification activities in the United States. Office of Scientific and Technical Information (OSTI), October 1994. http://dx.doi.org/10.2172/10108567.

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