Relevant bibliographies by topics / Desensitisation

Academic literature on the topic 'Desensitisation'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Desensitisation"

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Ramanathan, C., and G. J. Smelt. "Desensitisation." BMJ 302, no. 6778 (March 23, 1991): 726–27. http://dx.doi.org/10.1136/bmj.302.6778.726-c.

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Blaszczynski, Alex, and Lia Nower. "Imaginal Desensitisation." Journal of Clinical Activities, Assignments & Handouts in Psychotherapy Practice 2, no. 4 (February 9, 2003): 1–14. http://dx.doi.org/10.1300/j182v02n04_01.

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Eiser, N. "Desensitisation today." BMJ 300, no. 6737 (June 2, 1990): 1412–13. http://dx.doi.org/10.1136/bmj.300.6737.1412.

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Ganderton, M. A. "Desensitisation today." BMJ 301, no. 6746 (August 4, 1990): 293. http://dx.doi.org/10.1136/bmj.301.6746.293-a.

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Pottle, A., and M. Barbir. "Statin desensitisation." Atherosclerosis Supplements 28 (September 2017): e11. http://dx.doi.org/10.1016/j.atherosclerosissup.2017.08.020.

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&NA;. "Cotrimoxazole desensitisation successful." Reactions Weekly &NA;, no. 653 (May 1997): 4. http://dx.doi.org/10.2165/00128415-199706530-00008.

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&NA;. "Cotrimoxazole desensitisation successful." Inpharma Weekly &NA;, no. 1089 (May 1997): 21. http://dx.doi.org/10.2165/00128413-199710890-00048.

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Kelsey, S. M., G. R. Struthers, T. Beswick, and D. R. Blake. "Desensitisation to allopurinol." Annals of the Rheumatic Diseases 46, no. 1 (January 1, 1987): 84. http://dx.doi.org/10.1136/ard.46.1.84.

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Ridley, M. G., and J. A. Mathews. "Desensitisation to allopurinol." Annals of the Rheumatic Diseases 46, no. 11 (November 1, 1987): 875. http://dx.doi.org/10.1136/ard.46.11.875-a.

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Vaughan, K., M. Wiese, R. Gold, and N. Tarrier. "Eye-Movement Desensitisation." British Journal of Psychiatry 164, no. 4 (April 1994): 533–41. http://dx.doi.org/10.1192/bjp.164.4.533.

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A novel approach is described for the treatment of post-traumatic stress disorder (PTSD). Eye-movement desensitisation (EMD) requires the patient to generate images of the trauma in the mind and define physiological and emotional arousal states. While concentrating on these states, lateral multisaccardic eye movements are induced. Ten consecutive cases are reported who presented with symptoms originating from a range of traumas. The effectiveness of EMD in reducing symptoms outlined by DSM–III–R is described. An independent rater indicated that eight of the ten cases showed considerable improvement in PTSD symptoms following EMD, which was maintained at follow-up. Particular reference is given to the ‘specificity’ of EMD in treating symptoms and the changing pattern of effect at follow-up.
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Dissertations / Theses on the topic "Desensitisation"

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Batuwangala, Madura Suharshana. "Human urotensin-II receptor desensitisation." Thesis, University of Leicester, 2009. http://hdl.handle.net/2381/7846.

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Human Urotensin-II (U-II) is a cyclic undecapeptide that binds to the U-II receptor UT. The desensitisation mechanisms of the UT receptor (G_{q/11} coupled GPCR) are not well defined and hampered by (1) lack of native (in-vitro) models; (2) paucity of ligands, especially non-peptides and (3) irreversible binding of U-II. There are some limited studies using rat aorta, where a U-II induced primary contractile response was reduced upon a secondary re-challenge after 5-hours. Studies were undertaken to characterise cell lines expressing native (SJCRH30) and recombinant human hUT (HEK293 and CHO) for their suitability in binding and functional assays (PI and Ca^2+). SAR studies were carried out to characterise novel analogues modified at Tyr^9 of the U-II(4-11) template. This led to the identification of [3,5-diiodoTyr^9]U-II(4-11) a partial agonist in aorta and Ca^2+ assays at rat UT. Full agonism was demonstrated at hUT in PI and Ca^2+ assays. Efforts were made to delineate functional and genomic desensitisation of hUT. There was no functional desensitisation in SJCRH30. In HEK293hUT functional heterologous desensitisation of hUT was observed, this was not so in CHOhUT; instead P_2YR was functionally attenuated. In SJCRH30 6-hr U-II treatments led to UT mRNA reduction. Genomic desensitisation was also studied in Peripheral blood mononuclear cells (PBMCs). U-II treatments alone did not affect UT mRNA. Lipolysaccharide treatment of PBMCs led to UT mRNA upregulation which was desensitised with U-II treatments. In recombinant systems UT mRNA was upregulated at 6-hr U-II treatments. In conclusion modification of the U-II(4-11) template at Tyr^9 is useful for reducing efficacy. There is a difference in desensitisation profiles of native and recombinant hUT, where native receptors are not prone to functional desensitisation while receptor mRNA is reduced. In recombinant systems, hUT undergoes desensitisation (HEK293hUT only) while receptor mRNA is increased in both systems.
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Baig, Asma Hamid. "Desensitisation and downregulation of the ACTH-receptor." Thesis, Queen Mary, University of London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271428.

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De, Silva Mahappuque Anushika Sumali. "Ca²⁺ sensitisation and desensitisation mechanisms in pulmonary artery." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499978.

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Modulation of Ca²⁺sensitivity of the smooth muscle contractile apparatus plays a fundamental role in the regulation of force. Increased CA²⁺ sensitivity may contribute towards various disease conditions such as pulmonary hypertension. Ca²⁺ sensitivity is regulated by the balance between myosin light chain kinase (MLCK)-dependent phosphorylation and myosin phosphatase (MP)-dependent dephosphorylation of myosin light chain (MLC-20). Several protein kinases have been implicated in modulating this process. In particular RhoA and its effector Rho kinase (Rhok) inhibits MP activity via the MYPTl subunit; whereas protein kinase C (PKC) activates MP inhibitory protein CPI-17. Additionally the mitogen activated protein kinases (MAPK) and heat shock protein 27 (HSP27) may effectively modulate Ca²⁺ sensitivity via the cytoskeleton.
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McNicol, A. "Desensitisation and potentiation of platelet inositol phospholipid hydrolysis." Thesis, University of Glasgow, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234872.

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Beaumont, Vahri. "Desensitisation of somatostatin receptors in NG108-15 cells." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246243.

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Fowler, Catherine. "Desensitisation of somatostatin receptors in NG108-15 cells." Thesis, University of Bristol, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391197.

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Howard, Helen Clare. "The pharmacological characterisation of oxytocin receptors in the rat brain." Thesis, University of Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299594.

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Mustafa, Sanam. "Regulation and desensitisation studies on the nicotinic acid receptors." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/510/.

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The lipid-modifying qualities of nicotinic acid, a B3 vitamin, have been exploited for many years to prevent cardiovascular disease and its associated mortality. Despite its widespread availability and clinical use, the clinical target and precise molecular mechanism by which nicotinic acid acts remains elusive. In order to maximise nicotinic acid’s full potential as a lipid-modulating drug, overcome its related side effects and further develop novel and more potent drugs it is vital to understand its molecular mechanism of action. Fifty years on from its arrival on the market, receptors which this drug acts upon have recently been identified as the G protein-coupled ‘nicotinic acid receptors’. A family of three highly homologous receptors, HM74, HM74A and GPR81 are characterised by their differing affinities for nicotinic acid. Comparison of these homologues revealed a high degree of similarity between them, with the greatest similarity between HM74 and HM74A. The main structural difference between these receptors is the longer C-terminal tail of HM74. As the C-terminal tail of GPCRs is often implicated in receptor regulation it was hypothesised that the differences in this region may result in differential regulation of HM74 and HM74A. This hypothesis was tested by examining the regulation and desensitisation characteristics of each receptor in a heterologous expression system. Both HM74 and HM74A failed to interact with β-arrestin 2 or internalise in response to nicotinic acid. However, both receptors were phosphorylated in an agonist-dependent manner. HM74 but not HM74A was demonstrated to desensitise as result of prolonged nicotinic acid exposure. The importance of the C-terminal region was further analysed by the use of chimeric nicotinic acid receptors, in which the C-terminal tail of HM74 and HM74A were exchanged. It was shown that the C-terminal tail of HM74 may be implicated in the desensitisation characteristics of this receptor. Furthermore, it was shown that HM74 and HM74A display differential characteristics with respect to ERK1/2 phosphorylation.
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Braksator, Ellen. "A Study on μ-opioid receptor desensitisation and internalisation." Thesis, University of Bristol, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520290.

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McPherson, Jamie Lorcan. "Functional selectivity and desensitisation of G protein-coupled receptors." Thesis, University of Bristol, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555868.

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The existence of functional selectivity at the mu-opioid receptor was examined by determining the efficacy of a range of opioid agonists for promoting G protein activation and arrestin-3 translocation. In general, there is a good correlation between the efficacy of an opioid agonist at promoting G protein signaling, and the efficacy at recruiting arrestin-3 to the receptor. Endomorphin 2 appears to be an example of a biased ligand with significantly higher efficacy for arrestin-3 translocation, while morphine does not appear to be biased. The kinetics of binding for DAMGO, morphine and endomorphin 2 were determined by competition kinetic assay as a potential explanation for the apparent bias of endomorphin 2. Mean occupancy times of DAMGO, endomorphin 2 and morphine at MOPr are similar to the time required for GRK2-mediated phosphorylation, indicating that kinetics of binding may be a determinant of their ability to promote arrestin-3 signaling at MOPr. The abilities of DAMGO, morphine and endomorphin 2 to induce desensitization of GIRK currents in AtT-20 cells expressing wild type mu-opioid receptor, mu-opioid receptor containing S261/363A substitutions, and mu-opioid receptor with a C terminal truncation from amino acid 354-398 were examined. From the results, it appears that agonist-induced acute desensitization in AtT-20 cells has multiple components. C terminal truncation of MOPr resulted in a slight inhibition of endomorphin 2-induced desensitization. Alanine substitution at serine 261 and 363 inhibited desensitization induced by endomorphin 2. Treatment with the GRK2 inhibitor 5-[2-(5-nitro-2- furyl)vinyl]-2-furoate slightly but significantly inhibited desensitization induced by DAMGO, and to a very small extent morphine, but not endomorph in 2, which may indicate that biased ligands trigger receptor regulation in a different manner to unbiased ligands.
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Books on the topic "Desensitisation"

1

Robyns, Benoit. Vector Control of Induction Machines: Desensitisation and Optimisation Through Fuzzy Logic. London: Springer London, 2012.

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Eye movement desensitization and reprocessing: Basic principles, protocols, and procedures. New York: Guilford Press, 1995.

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Fear, Rhona M. Systematic Desensitisation for Panic and Phobia. Routledge, 2018. http://dx.doi.org/10.4324/9780429480645.

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Degobert, Philippe, Benoit Robyns, and Bruno Francois. Vector Control of Induction Machines: Desensitisation and Optimisation Through Fuzzy Logic. Springer, 2012.

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Vector Control Of Induction Machines Desensitisation And Optimisation Through Fuzzy Logic. Springer, 2012.

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Fear, Rhona M. Systemic Desensitisation for Panic and Phobia: An Introduction for Health Professionals. Taylor & Francis Group, 2017.

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Book chapters on the topic "Desensitisation"

1

Çakmak Karaer, Işıl, Nuray Bayar Muluk, and Glenis K. Scadding. "Does Aspirin Desensitisation Work in N-ERD?" In Challenges in Rhinology, 99–108. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-50899-9_12.

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Trincavelli, Maria Letizia, Osele Ciampi, and Claudia Martini. "The Desensitisation as A3 Adenosine Receptor Regulation: Physiopathological Implications." In A3 Adenosine Receptors from Cell Biology to Pharmacology and Therapeutics, 75–90. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3144-0_5.

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Hui, Ada. "Institutional and Emotion Work in Forensic Psychiatry: Detachment and Desensitisation." In Mental Health Uncertainty and Inevitability, 137–65. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43970-9_6.

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Campbell, Caroline. "Techniques Which Help Children Cope with Local Anaesthesia (Including Systematic Needle Desensitisation)." In Dental Fear and Anxiety in Pediatric Patients, 197–225. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-48729-8_12.

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Baruteau, J., A. Broomfield, V. Crook, N. Finnegan, K. Harvey, D. Burke, M. Burch, G. Shepherd, and A. Vellodi. "Successful Desensitisation in a Patient with CRIM-Positive Infantile-Onset Pompe Disease." In JIMD Reports, 99–102. Cham: Springer International Publishing, 2013. http://dx.doi.org/10.1007/8904_2013_250.

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Kingerlee, Roger. "Practising Eye Movement Desensitisation and Reprocessing (EMDR) with Male Civilians and Male Veterans." In The Palgrave Handbook of Male Psychology and Mental Health, 461–82. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-04384-1_23.

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Dobie, Thomas G. "Cognitive-Behavioural Desensitisation Training—The Principles of My Original Programme Using a Rotating/Tilting Chair." In Motion Sickness, 247–73. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-97493-4_12.

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Balfour, D. J. K., and M. E. M. Benwell. "Desensitisation of the Stimulatory Effects of Nicotine on Dopamine Secretion in the Mesolimbic System of the Rat." In Effects of Nicotine on Biological Systems II, 211–17. Basel: Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7445-8_27.

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Mann, Dave. "Desensitisation." In Gestalt Therapy, 73–74. Routledge, 2020. http://dx.doi.org/10.4324/9781315158495-24.

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"Desensitisation." In The Dysfluency Resource Book, 117–28. Routledge, 2017. http://dx.doi.org/10.4324/9781315168913-15.

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Conference papers on the topic "Desensitisation"

1

Whitworth, N. J., and J. R. Maw. "Modelling shock desensitisation of heterogeneous explosives." In Proceedings of the conference of the American Physical Society topical group on shock compression of condensed matter. AIP, 1996. http://dx.doi.org/10.1063/1.50674.

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Rodríguez Esquíroz, A., J. Polo García, L. Ulacia Epelde, B. Larrayoz Sola, D. Tejada Marín, P. Aldave Cobos, R. De La Riva Bohigas, et al. "5PSQ-050 Successful desensitisation in a patient with dasatinib hypersensitivity." In 25th EAHP Congress, 25th–27th March 2020, Gothenburg, Sweden. British Medical Journal Publishing Group, 2020. http://dx.doi.org/10.1136/ejhpharm-2020-eahpconf.367.

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Baptista, M., R. Oliveira, P. Barreto, J. Fagundes, J. Cardoso, J. Guerra, H. Pité, M. Bernardo, M. Morais-Almeida, and M. Chambel. "CP-174 Successful desensitisation in a patient with lenalidomide hypersensitivity." In 22nd EAHP Congress 22–24 March 2017 Cannes, France. British Medical Journal Publishing Group, 2017. http://dx.doi.org/10.1136/ejhpharm-2017-000640.172.

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Sanchez, R. Vazquez, J. Sánchez-Rubio Ferrández, D. Córdoba Diaz, M. Córdoba Díaz, and T. Molina Garcia. "5PSQ-061 Stability of carboplatin infusion solutions used in desensitisation protocol." In Abstract Book, 23rd EAHP Congress, 21st–23rd March 2018, Gothenburg, Sweden. British Medical Journal Publishing Group, 2018. http://dx.doi.org/10.1136/ejhpharm-2018-eahpconf.415.

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Molinier, A. "Implementation and Test of a Load Desensitisation Algorithm in Distance Protection." In 15th International Conference on Developments in Power System Protection (DPSP 2020). Institution of Engineering and Technology, 2020. http://dx.doi.org/10.1049/cp.2020.0039.

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Jackson, Matthew, Sarah Callaghan, David Austin, Douglas Muir, Andrew Sutton, Robert Wright, Paul Williams, et al. "9 A rapid protocol for aspirin desensitisation following hypersensitivity reactions: our single-centre experience in patients with stable and unstable coronary artery disease." In British Cardiovascular Society Annual Conference ‘High Performing Teams’, 4–6 June 2018, Manchester, UK. BMJ Publishing Group Ltd and British Cardiovascular Society, 2018. http://dx.doi.org/10.1136/heartjnl-2018-bcs.9.

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