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Books on the topic 'Dementia risk'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Clive, Ballard, and Lovestone Simon, eds. Management of dementia. 2nd ed. New York: Informa Healthcare USA, 2008.

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2

Jacobsen, Sarah R. Vascular dementia: Risk factors, diagnosis, and treatment. Hauppauge, N.Y: Nova Science, 2011.

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3

Gilmour, Helen. Living alone with dementia: Diagnosis, care planning, risk. [Enniskillen: The Author], 2002.

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4

Román, Gustavo C. Managing vascular dementia: Concepts, issues, and management. London: Science Press, 2003.

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5

Clarke, Charlotte L. Risk assessment and management for living well with dementia. London: Jessica Kingsley Publishers, 2011.

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6

Anderson, Nicole D. Living with mild cognitive impairment: A guide to maximizing brain health and reducing risk of dementia. Oxford: Oxford University Press, 2012.

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7

Rodolfo, Paoletti, and International Symposium on Women's Health in Menopause (3rd : 1998 : Florence, Italy), eds. Women's health and menopause: Risk reduction strategies--improved quality of health. Dordrecht: Kluwer Academic Publishers, 1999.

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8

American madness: The rise and fall of dementia praecox. Cambridge, Mass: Harvard University Press, 2011.

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9

Legal issues relating to football head injuries: Hearings before the Committee on the Judiciary, House of Representatives, One Hundred Eleventh Congress, first and second sessions, October 28, 2009 and January 4, 2010. Washington: U.S. G.P.O., 2010.

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10

Reducing Your Risk Of Dementia. Sheldon, 2011.

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11

Tom, Smith. Reducing Your Risk of Dementia. SPCK Publishing, 2011.

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12

Piggott, Margaret Ann. Neurochemical pathology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0007.

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This chapter considers the neurodegenerative disorders Alzheimer’s disease (AD), Lewy body dementias (dementia with Lewy bodies (DLB) and Parkinson’s disease dementia(PDD)), frontotemporal dementia (FTD); and also vascular dementia (VaD) which results from cerebrovascular disease. These different conditions, which give rise to dementia syndromes, each have distinct neurochemical pathologies, with important implications for treatment. As increased age is the common risk factor generally associated with dementing illnesses, neurochemical changes are set in the context of the changes which occur during ageing. A detailed understanding of the neurotransmitter function in each condition can lead to rational drug design and treatment strategies appropriate for each group of patients. Neurochemical pathology in transmitter systems underlying clinical features of these disorders are reviewed.
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13

Beller, Jerry, and Beller Health. Dementia Overview: 15 Dementia Types, Causes, Risk Factors, and Symptoms. Independently Published, 2019.

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14

Tanner, Pamela. Dementia: Prevalence, Risk Factors and Management Strategies. Nova Science Publishers, Incorporated, 2014.

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15

Briggs, John, Jerry Beller, and Beller Health. DEMENTIA Types, Symptoms, & Risk Factors: Dementia Guide for Patients, Families, Caregivers, & Medical Professionals. Independently published, 2020.

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16

Rayman, Margaret. Healthy Eating to Reduce the Risk of Dementia. Kyle Books, 2019.

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17

Healthy Eating to Reduce the Risk of Dementia. Octopus Publishing Group, 2019.

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18

Mecca, Adam P., and Rajesh R. Tampi. Risk of Death with Atypical Antipsychotic Medications for Dementia. Edited by Ish P. Bhalla, Rajesh R. Tampi, Vinod H. Srihari, and Michael E. Hochman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190625085.003.0016.

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This chapter provides a summary of a landmark meta-analysis that investigated the risks of atypical antipsychotic use to treat psychosis, aggression, or agitation in patients with dementia. The chapter briefly reviews the study design, as well as implications and limitations. A relevant clinical case concludes the chapter. In summary, atypical antipsychotic use for 6 to 26 weeks was associated with increased risk of death (Odds Ratio of 1.54 for antipsychotic vs placebo). There were no differences in risk between individual medications, disease severity, indication for antipsychotic, or treatment setting. In patients with psychosis, agitation, or aggression due to dementia, the efficacy of atypical antipsychotics is questionable and their use comes with considerable risks of side effects and adverse events.
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19

Clarke, Charlotte L., John Keady, Catherine E. Gibb, and Catherine Gibb. Risk Assessment and Management for Living Well with Dementia. Kingsley Publishers, Jessica, 2011.

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20

Alessi, Charles, Larry W. Chambers, and Muir Gray. Increase your Brainability—and Reduce your Risk of Dementia. Oxford University Press, 2021. http://dx.doi.org/10.1093/oso/9780198860341.001.0001.

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Our experience is that many people including many members of the medical profession are confused about what happens to our brains and minds as we live longer. Consistently, they overestimate the effects of ageing and underestimate the potential for improving the ability of the brain and the mind no matter what age you may be. Linked to this is the fact that, although Alzheimer’s disease cannot be prevented, the risk of dementia can be reduced by at least a third because there are many other causes of dementia than Alzheimer’s disease having to do with healthy brain tissue, blood supply to the brain and ability to interact with people and ideas. With this knowledge, we outline steps individuals can take themselves, with family and friends, their community and the health service to improve brain health.
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21

Increase Your Brainability--And Reduce Your Risk of Dementia. Oxford University Press, 2021.

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22

Briggs, John, Jerry Beller, Beller Health, and Brain Research. DEMENTIA Types, Risk Factors and Symptoms: Dementia Guide for Doctors, Caregivers, Patients, and Loved Ones. Independently Published, 2020.

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23

Fratiglioni, Laura, and Chengxuan Qiu. Epidemiology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0031.

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This chapter deals with the occurrence, determinants and primary prevention of dementia. Dementia is one of the major causes of functional dependence, poor quality of life, institutionalisation and mortality among elderly people. The risk of dementia increases almost exponentially with advancing age. As the population ages, dementia poses a serious threat to public health and social welfare system of our society. Accumulating evidence suggests that cardiovascular risk factors significantly contribute to the development and expression of dementia. Thus, adequate management of vascular risk factors and related disorders can be one of the preventative strategies against cognitive ageing and dementia. In addition, psychosocial factors such as educational achievement, socially-integrated and mentally-stimulating lifestyles are critical for delaying the onset of dementia by increasing cognitive reserve. Taken together, maintaining vascular health and adopting a healthy cognitive lifestyle from a life-course perspective may be the most promising strategy to achieve late-life cognitive health.
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24

Renton, Alan E., and Alison M. Goate. Genetics of Dementia. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0051.

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The genetic architecture of dementia is polygenic and complex, with risk alleles spanning frequency–effect size space. Despite significant progress, most genes influencing these disorders await discovery. Known risk loci implicate perturbed pathways that coalesce around recurring mechanistic themes, notably the autophagosome-lysosome system, the cytoskeleton, endocytosis, innate immunity, lipid metabolism, mitochondria, and the ubiquitin-proteasome system. Phenotypic and pathophysiological pleiotropy suggests some conditions form continuous clinicopathogenetic disease spectra blurring classical diagnoses. Future large-scale genome sequencing of global populations will significantly elucidate etiopathogenesis and is likely to reframe nosology. Furthermore integrative prospective cohort studies have the potential to revolutionize our understanding of dementia.
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25

Uddin, Sahab, and Ghulam Ashraf. Current Thoughts on Dementia: From Risk Factors to Therapeutic Interventions. Springer Singapore Pte. Limited, 2022.

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26

Dresser, Rebecca. Dementia, Dignity, and Physician-Assisted Death. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190675967.003.0007.

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As more people live into their later years, more of them become susceptible to Alzheimer’s disease and other age-related dementias. Many people fear dementia, and some argue that physician-assisted death should be available to those seeking to avoid the indignity of life with dementia. Distinct issues surround the relationship of dignity and assisted death for potential or actual dementia patients. This chapter examines the role of dignity concerns in addressing (1) requests for assisted death by at-risk individuals to avoid a possible future with dementia, (2) requests for assisted death by individuals with mild or moderate dementia, and (3) advance directives requesting assisted death in the event of a later dementia diagnosis or appearance of specific behavioral manifestations of dementia (e.g., apparent inability to recognize family and friends).
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27

Brommelhoff, Jessica A. Depression in Dementia Syndromes. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.007.

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Depressive symptoms are common in many dementia syndromes, and depressive disorders are much more common in older adults with dementia than in cognitively intact older adults. Depression may be a risk factor for, or a prodromal feature of, subsequent dementia. Several neuropathological mechanisms have been suggested to explain these relationships, including the role of underlying cerebrovascular risk factors for depression and cognitive impairment. Depression also may be present in dementia as an emotional reaction to cognitive decline, or as a recurrence of early and midlife depression. Differential diagnosis between depression and dementia is essential, but complicated by problems in assessment, overlapping symptoms between the two conditions, and other medical co-morbidities. Pharmacological treatment of depression in dementia may also be complicated by medical co-morbidity, and can run the risk for adverse reactions or interactions between medications. Psychotherapy and psychosocial interventions, however, hold some promise for effective reduction of depressive symptoms.
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28

Taylor, Kathleen. Dementia: A Very Short Introduction. Oxford University Press, 2020. http://dx.doi.org/10.1093/actrade/9780198825784.001.0001.

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Dementia: A Very Short Introduction explains how dementia is diagnosed, its different types and symptoms, and its effects on sufferers and their families. Why is dementia resistant to treatment? Why has the most successful scientific hypothesis not led to a cure? Are there variations between different countries, and given the rise in the ageing population, are there more or fewer cases than we think? This VSI looks at the history of dementia research and examines the genetic, physiological, and environmental risk factors and how individuals might reduce them. It also investigates developments in diagnosis and symptom management, and the economic and political context of dementia care.
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29

Dementia (Epidemiology of Mental Disorders and Psychosocial Problems). World Health Organization, 1994.

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30

Beller, Jerry, and Beller Health. 16 Dementia Types, Symptoms, and Risk Factors: Alzheimer's LBD PDD DLB FTD PPA BvFTD LATE Vascular Dementia Huntington's, Etc. Independently Published, 2019.

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31

Kumar and Pradeep Mishra. Dementia Math Workbook Volume One: Large Print Math Problems and Solutions to Reduce the Risk of Parkinson, Alzheimer, Dementia. Independently Published, 2022.

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32

The mindspan diet: Reduce Alzheimer's risk, minimize memory loss, and keep your brain young. Ballantine Books, 2016.

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33

Beller, Jerry, and Beller Health. Dementia Types, Risk Factors, and Symptoms: Alzheimer's Disease Vascular Lewy Body Frontotemporal Huntington's Normal Pressure Hydrocephalus Wernicke Korsakoff Dementias. Independently Published, 2019.

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34

Dening, Karen Harrison. Dementia, multimorbidity, and frailty. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198806677.003.0006.

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Increasing age is the strongest risk factor for developing dementia, frailty, and other co-morbid conditions. Dementia is not a disease in itself but a syndrome; a collection of symptoms: such as a decline in memory, reasoning, and communication skills, and a gradual loss of skills needed to carry out daily living activities. Whereas frailty is a distinctive health state in which multiple body systems gradually lose their in-built reserves. However, often ‘layered’ on these two may also be several other conditions that coexist within an individual. There is a growing body of evidence to support the psychosocial care for people with dementia as the disease advances but little of the effect and outcomes where there is a combination of these conditions. This chapter explores some of the issues for families affected by these conditions and the care and support that may be of benefit through case management.
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35

Mindspan Diet: Reduce Alzheimer's Risk, and Keep Your Brain Young. Oneworld Publications, 2017.

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36

Estep, Preston W. Mindspan Diet: Reduce Alzheimer's Risk, and Keep Your Brain Young. Oneworld Publications, 2016.

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37

Mindspan Diet: Reduce Alzheimer's Risk, and Keep Your Brain Young. Oneworld Publications, 2016.

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38

Kosik, K. S. Outsmarting Alzheimer's: What you can do to reduce your risk. 2015.

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39

Beller, Jerry, and Beller Health. Dementia Types, Risk Factors, and Symptoms 2019: Alzheimer's Disease , Vascular , Lewy Body , Frontotemporal , Huntington's , Normal Pressure Hydrocephalus , Wernicke Korsakoff Dementias. Independently Published, 2019.

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40

Harold, Denise, and Julie Williams. Molecular genetics and biology of dementia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199644957.003.0008.

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Considerable progress has been made in our understanding of the genetics and molecular biology of dementia. In this chapter we focus predominantly on the most common form of dementia, Alzheimer’s disease (AD), but also discuss vascular dementia and frontotemporal dementia. Genetic mutations have been identified that cause Mendelian subtypes of each disorder, and in recent years genome-wide association studies have greatly aided the identification of risk genes for more common forms of disease. For example, 9 susceptibility genes have been identified in AD in the past 3 years as a result of genome-wide association studies, the first robust risk loci to be identified since APOE in 1993. This progress in genetic research is having a dramatic effect on our understanding of disease pathogenesis, by refining previous ideas and defining new primary disease mechanisms.
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41

Coping with Mild Cognitive Impairment: A Guide to Managing Memory Loss, Effective Brain Training and Reducing the Risk of Dementia. Kingsley Publishers, Jessica, 2020.

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42

Jordan, Mary. Coping with Mild Cognitive Impairment: A Guide to Managing Memory Loss, Effective Brain Training and Reducing the Risk of Dementia. Kingsley Publishers, Jessica, 2020.

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43

Estep, Preston W. The mindspan diet: Reduce Alzheimer's risk, minimize memory loss, and keep your brain young. 2016.

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44

Jotterand, Fabrice, Marcello Ienca, Tenzin Wangmo, and Bernice Elger, eds. Intelligent Assistive Technologies for Dementia. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780190459802.001.0001.

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The development and implementation of intelligent assistive technologies (IATs) to compensate for the specific physical and cognitive deficits of older adults with dementia have been recognized by many as one of the most promising approaches to this emerging financial and caregiving burden. In the past 15 years, advancements in artificial intelligence (AI), pervasive and ubiquitous computing (PUC), and other advanced trends in software and hardware technology have led to the development and design of a wide range of IATs to help older people compensate for the physical and sensory deficits that may accompany dementia and age-related cognitive decline. These technologies are designed to support impaired older adults in the completion of activities of daily living, assist them in the prevention or management of risk, and/or maintain their recreational and social environment. The widespread implementation and use of assistive technologies is a very rapid process, which is reshaping dementia care and producing constantly changing strategies. This volume aims at providing an up-to-date overview of the current state of the art of assistive technologies for dementia care and an examination of their implications at the medical level, including psychological and clinical issues and their ethical and regulatory challenges. The overall goal of this book is to raise societal awareness on the use of IATs for dementia care and take a first step into developing an international regulatory and policy framework.
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45

Hobbins, Ngaire. Brain, Body, Food: The Ultimate Guide to Thriving into Later Life and Reducing Dementia Risk. Ngaire Hobbins, 2020.

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46

Rayman, Margaret. Healthy eating to reduce the risk of dementia: 100 fantastic recipes based on extensive, in-depth research. 2015.

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47

Kumar. Activities for Alzheimer and Dementia Patients : Multi-Digit Addition and Subtraction #1: Large Print Math Problems and Solutions for Reducing Risk of Parkinson, Alzheimer, Dementia. Independently Published, 2022.

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48

Steinberg, Martin, and Paul B. Rosenberg. Cognitive Impairment and Depression in Older Patients. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199959549.001.0001.

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Depression, mild cognitive impairment (MCI) and dementia in the elderly can present with similar features such as cognitive complaints, loss of initiative, and difficulties with psychosocial functioning. These can be difficult to distinguish in the office setting, especially when mild in severity. The relationships between the three syndromes remains incompletely understood. Patients with MCI are at high risk for conversion to dementia. Depression may be either a risk factor for or early manifestation of MCI. Depression in late life is associated with Alzheimer’s disease (AD) and other dementias, but the causal relationship remains controversial. Depression may also increase the risk of conversion from MCI to dementia and be more strongly associated with conversion to Vascular dementia (VaD) than to AD. This book will provide guidance to clinicians in the diagnosis and management of these complex conditions in the office setting.
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49

Steinberg, Martin, and Paul B. Rosenberg. Cognitive Impairment and Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199959549.003.0001.

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Depression, mild cognitive impairment (MCI) and dementia in the elderly can present with similar features such as cognitive complaints, loss of initiative, and difficulties with psychosocial functioning. These can be difficult to distinguish in the office setting, especially when mild in severity. The relationships between the three syndromes remains incompletely understood. Patients with MCI are at high risk for conversion to dementia. Depression may be either a risk factor for or early manifestation of MCI. Depression in late life is associated with Alzheimer’s disease (AD) and other dementias, but the causal relationship remains controversial. Depression may also increase the risk of conversion from MCI to dementia and be more strongly associated with conversion to Vascular dementia (VaD) than to AD. This book will provide guidance to clinicians in the diagnosis and management of these complex conditions in the office setting.
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50

Colbert, Don. Dr. Colbert's Healthy Brain Zone: Reverse Memory Loss and Reduce Your Risk of Dementia and Alzheimer's. Charisma Media, 2023.

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