Relevant bibliographies by topics / Dementia risk

Academic literature on the topic 'Dementia risk'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Dementia risk"

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Johnson, Rebecca A., and Jason Karlawish. "A review of ethical issues in dementia." International Psychogeriatrics 27, no. 10 (June 10, 2015): 1635–47. http://dx.doi.org/10.1017/s1041610215000848.

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ABSTRACTDementia raises many ethical issues. The present review, taking note of the fact that the stages of dementia raise distinct ethical issues, focuses on three issues associated with stages of dementia's progression: (1) how the emergence of preclinical and asymptomatic but at-risk categories for dementia creates complex questions about preventive measures, risk disclosure, and protection from stigma and discrimination; (2) how despite efforts at dementia prevention, important research continues to investigate ways to alleviate clinical dementia's symptoms, and requires additional human subjects protections to ethically enroll persons with dementia; and (3) how in spite of research and prevention efforts, persons continue to need to live with dementia. This review highlights two major themes. First is how expanding the boundaries of dementias such as Alzheimer's to include asymptomatic but at-risk persons generate new ethical questions. One promising way to address these questions is to take an integrated approach to dementia ethics, which can include incorporating ethics-related data collection into the design of a dementia research study itself. Second is the interdisciplinary nature of ethical questions related to dementia, from health policy questions about insurance coverage for long-term care to political questions about voting, driving, and other civic rights and privileges to economic questions about balancing an employer's right to a safe and productive workforce with an employee's rights to avoid discrimination on the basis of their dementia risk. The review highlights these themes and emerging ethical issues in dementia.
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Cipriani, Gabriele, Sabrina Danti, Lucia Picchi, Angelo Nuti, and Mario Di Fiorino. "Daily functioning and dementia." Dementia & Neuropsychologia 14, no. 2 (June 2020): 93–102. http://dx.doi.org/10.1590/1980-57642020dn14-020001.

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Abstract. Dementia is characterized by a decline in memory, language, problem-solving and in other cognitive domains that affect a person’s ability to perform everyday activities and social functioning. It is consistently agreed that cognitive impairment is an important risk factor for developing functional disabilities in patients with dementia. Functional status can be conceptualized as the ability to perform self-care, self- maintenance and physical activity. A person with dementia usually requires help with more complex tasks, such as managing bills and finances, or simply maintaining a household. Good functional performance is fundamental for elderly people to maintain independency and avoid institutionalization. The purpose of this review is to describe functional changes in demented patients, evaluating the variability in subgroups of dementias.
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Alty, Jane, Maree Farrow, and Katherine Lawler. "Exercise and dementia prevention." Practical Neurology 20, no. 3 (January 21, 2020): 234–40. http://dx.doi.org/10.1136/practneurol-2019-002335.

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Ageing, genetic, medical and lifestyle factors contribute to the risk of Alzheimer’s disease and other dementias. Around a third of dementia cases are attributable to modifiable risk factors such as physical inactivity, smoking and hypertension. With the rising prevalence and lack of neuroprotective drugs, there is renewed focus on dementia prevention strategies across the lifespan. Neurologists encounter many people with risk factors for dementia and are frequently asked whether lifestyle changes may help. Exercise has emerged as a key intervention for influencing cognition positively, including reducing the risk of age-related cognitive decline and dementia. This article focuses on the current evidence for physical inactivity as a modifiable dementia risk factor and aims to support neurologists when discussing risk reduction.
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Copeland, J. R. M., Cherie McCracken, M. E. Dewey, K. C. M. Wilson, Mark Doran, Chris Gilmore, Anne Scott, and Bernie Larkin. "Undifferentiated dementia, Alzheimer's disease and vascular dementia: Age- and gender-related incidence in Liverpool." British Journal of Psychiatry 175, no. 5 (November 1999): 433–38. http://dx.doi.org/10.1192/bjp.175.5.433.

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BackgroundDoes incidence of dementia follow the age pattern of prevalence? Is gender a risk factor? Do patterns of incidence differ between dementias?AimsTo assess age-specific incidence rates of undifferentiated dementias, Alzheimer's disease and vascular dementia.Method5222 individuals aged $65 years, were interviewed using the Geriatric Mental State/History and Aetiology Schedule. The AGECAT package was used to identify cases at three interviewing waves at two-year intervals. Diagnoses were made using ICD −10 Research Criteria and validated against neurological and psychological examination, with imaging and neuropathology on unselected subsamples.ResultsIncidence rates of the dementias increase with age. Age patterns are similar between Alzheimer's disease and vascular dementia. Gender appears influential in Alzheimer's disease. In England and Wales, 39 437 new cases of Alzheimer's disease (4.9/1000 person-years at risk); 20 513 of vascular dementia (2.6/1000 person-years) and 155 169 of undifferentiated dementia (19/1000 person-years) can be expected each year.ConclusionsIncidence rates for Alzheimer's disease and vascular dementia appear to behave differently with an increased risk of Alzheimer's disease for women compared to vascular dementia.
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Kim, Seon-Jip, Sang Min Park, Hyun-Jae Cho, and Ji Woon Park. "Primary headaches increase the risk of dementias: An 8-year nationwide cohort study." PLOS ONE 17, no. 8 (August 18, 2022): e0273220. http://dx.doi.org/10.1371/journal.pone.0273220.

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Background Headache, a highly prevalent neurological disorder, has consistently been linked with an elevated risk of dementia. However, most studies are focused on the relationship with migraine in limited age groups. Therefore, the objective of this research was to look at the link between various type of headaches and dementias based on longitudinal population-based data. Methods and results Participants diagnosed with headache from 2002 to 2005 were selected and major covariates were collected. The diagnoses of Alzheimer’s disease, vascular dementia, and other dementias were observed from 2006 until 2013. The adjusted hazard ratios (aHRs) and 95% confidence intervals (CIs) of dementias according to headache type were calculated by Cox proportional hazards regression. A number of 470,652 participants were observed for a mean of 7.6 years (standard deviation: 1.2), for approximately 3.6 million person-years. Both tension type headache (TTH) and migraine elevated the risk of all-cause dementias (TTH, aHR 1.18, 95% CI 1.13–2.24; migraine, aHR 1.18, 95% CI 1.13–2.24). Headaches had a greater influence in females and non-smokers as a risk factor of dementias. Patients with migraine who consumed alcohol had a higher risk of dementia, however this was not true with TTH patients. Among participants without comorbidities, TTH patients were more susceptible to dementia than migraine patients. Headache patients had a higher proportion of females regardless of headache type and approximately 1.5 times more individuals had three or more comorbidities compared to those without headache. Conclusions Headache could be an independent predictor for subsequent dementia risk. Future studies should focus on clarifying pathogenic pathways and possible dementia-related preventive measures in headache populations.
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Nordestgaard, Liv Tybjærg, Mette Christoffersen, and Ruth Frikke-Schmidt. "Shared Risk Factors between Dementia and Atherosclerotic Cardiovascular Disease." International Journal of Molecular Sciences 23, no. 17 (August 29, 2022): 9777. http://dx.doi.org/10.3390/ijms23179777.

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Alzheimer’s disease is the most common form of dementia, and the prodromal phases of Alzheimer’s disease can last for decades. Vascular dementia is the second most common form of dementia and is distinguished from Alzheimer’s disease by evidence of previous stroke or hemorrhage and current cerebrovascular disease. A compiled group of vascular-related dementias (vascular dementia and unspecified dementia) is often referred to as non-Alzheimer dementia. Recent evidence indicates that preventing dementia by lifestyle interventions early in life with a focus on reducing cardiovascular risk factors is a promising strategy for reducing future risk. Approximately 40% of dementia cases is estimated to be preventable by targeting modifiable, primarily cardiovascular risk factors. The aim of this review is to describe the association between risk factors for atherosclerotic cardiovascular disease and the risk of Alzheimer’s disease and non-Alzheimer dementia by providing an overview of the current evidence and to shed light on possible shared pathogenic pathways between dementia and cardiovascular disease. The included risk factors are body mass index (BMI); plasma triglyceride-, high-density lipoprotein (HDL) cholesterol-, low-density lipoprotein (LDL) cholesterol-, and total cholesterol concentrations; hypertension; diabetes; non-alcoholic fatty liver disease (NAFLD); physical inactivity; smoking; diet; the gut microbiome; and genetics. Furthermore, we aim to disentangle the difference between associations of risk factors in midlife as compared with in late life.
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Mayeda, Elizabeth Rose. "Invited Commentary: Examining Sex/Gender Differences in Risk of Alzheimer Disease and Related Dementias—Challenges and Future Directions." American Journal of Epidemiology 188, no. 7 (March 2, 2019): 1224–27. http://dx.doi.org/10.1093/aje/kwz047.

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Abstract The majority of people living with Alzheimer disease (AD) and related dementias are women. Longer life expectancy is one factor thought to contribute to this observation, but possible sex-specific biological mechanisms have received considerable attention from the research community. In the current issue of the Journal, Buckley et al. (Am J Epidemiol. 2019;188(7):1213–1223) use death certificate information on all deaths occurring among adults aged ≥60 years in Australia between 2006 and 2014 to evaluate sex/gender differences in rates of death with dementia (all types), AD dementia, and vascular dementia listed on the death certificate. The paper by Buckley et al. highlights several important methodological challenges for research examining sex/gender differences in risk of AD and related dementias, including challenges in measurement, survival bias and competing risks, and selection bias arising from sample selection. The current evidence on possible sex-specific biological risk factors for AD is intriguing, but there are numerous alternative explanations for differences in AD dementia and AD biomarkers between women and men. Triangulation of evidence from study designs with different strengths and weaknesses and transdisciplinary collaboration will be vital to generating conclusive evidence about sex/gender differences in risk of AD and related dementias.
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Rifkind, Patrice. "Dementia Risk Factor." Brain & Life 16, no. 2 (2020): 8. http://dx.doi.org/10.1097/01.nnn.0000660736.25747.f1.

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Klimova, Blanka, Petra Maresova, and Kamil Kuca. "Combat Military Personnel and Selective Risk Factors for the Development of Dementias - A Review." Current Psychiatry Research and Reviews 15, no. 1 (May 2, 2019): 44–48. http://dx.doi.org/10.2174/1573400515666190114155451.

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: Due to the growth of life expectancies and the increasing number of elderly population all over the world, there is a risk of growth of aging diseases such as dementia. Recent research studies also indicate that there will be a growing number of military veterans who will be affected by dementia, already at the age of 55+ years. In the case of combat military personnel, the most common dementias are Alzheimer’s disease and vascular dementia. These two dementias are very similar because their main symptoms are the same. The purpose of this review is to explore two main risk factors influencing the development of the dementias. These include posttraumatic stress disorder (PTSD) and traumatic brain injury (TBI). Furthermore, the authors of this study focus on the exploration of the treatment of PTSD and TBI in order to delay the development of dementias among combat military personnel. : For the purpose of this study, a method of literature review of available sources exploring these two main risk factors of dementia among combat military personnel was used. Based on the evaluation of these literature sources, possibilities of pharmacological and non-pharmacological approaches to the treatment and care of these people were described.
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Gerritsen, Lotte, Emma L. Twait, Palmi V. Jonsson, Vilmundur Gudnason, Lenore J. Launer, and Mirjam I. Geerlings. "Depression and Dementia: The Role of Cortisol and Vascular Brain Lesions. AGES-Reykjavik Study." Journal of Alzheimer's Disease 85, no. 4 (February 15, 2022): 1677–87. http://dx.doi.org/10.3233/jad-215241.

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Background: Late-life depression (LLD) is related to an increased risk of developing dementia; however, the biological mechanisms explaining this relationship remain unclear. Objective: To determine whether the relationship between LLD and dementia can be best explained by the glucocorticoid cascade or vascular hypothesis. Methods: Data are from 4,354 persons (mean age 76±5 years) without dementia at baseline from the AGES-Reykjavik Study. LLD was assessed with the MINI diagnostic interview (current and remitted major depressive disorder [MDD]) and the Geriatric Depression Scale-15. Morning and evening salivary cortisol were collected (glucocorticoid cascade hypothesis). White matter hyperintensities (WMH; vascular hypothesis) volume was assessed using 1.5T brain MRI. Using Cox proportional hazard models, we estimated the associations of LLD, cortisol levels, and WMH volume with incident all-cause dementia, AD, and non-AD dementia. Results: During 8.8±3.2 years of follow-up, 843 persons developed dementia, including 397 with AD. Current MDD was associated with an increased risk of developing all-cause dementia (HR = 2.17; 95% CI 1.66–2.67), with risks similar for AD and non-AD, while remitted MDD was not (HR = 1.02; 95% CI 0.55–1.49). Depressive symptoms were also associated with increased risk of dementia, in particular non-AD dementias. Higher levels of evening cortisol increased risk of dementia, but this was independent of MDD. WMH partially explained the relation between current MDD and dementia risk but remained increased (HR = 1.71; 95% CI 1.34–2.08). Conclusion: The current study highlights the importance of LLD in developing dementia. However, neither the glucocorticoid cascade nor the vascular hypotheses fully explained the relation between depression and dementia.
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Dissertations / Theses on the topic "Dementia risk"

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Cornett, Patricia F. "Risk Factors for Vascular Dementia." Thesis, University of North Texas, 2005. https://digital.library.unt.edu/ark:/67531/metadc4781/.

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Dementia is a devastating disorder that commonly affects people over the age of 65. Alzheimer's disease and vascular dementia are the most common forms of dementias. A number of studies have implicated cardiovascular risks as important factors in the development of dementia. These risks include high-risk behaviors such as smoking and risks related at least partially to health behaviors such as diet and exercise. This study examines a group of cardiovascular risk factors, as defined by the Framingham study, to ascertain if they are predictors of dementia. A retrospective chart review of 481consecutive patients seen in a geriatric medicine clinic produced a sample of 177 individuals diagnosed with dementia and 304 individuals without a dementia diagnosis. Relative risk ratio (RRR) results indicate that a history of hypertension (RRR= 1.80, p = .009) and a history of hypercholesterolemia (RRR = 1.85, p = .016) are significant predictors of Alzheimer's disease. A history of tobacco use (RRR = 2.18, p = .01) is a significant predictor of vascular dementia. Stepwise regression analyses indicate that hypercholesterolemia is an independent predictor of dementia (b = -.113, p = .009) and hypercholesterolemia (b = -.104, p = .018) and hypertension (b = -.094, p = .031) clustered together have an additive risk factor effect. These results are discussed in terms of the importance of specific health behaviors in the development and possible prevention of dementia.
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Mawanda, Francis. "Emerging risk factors for dementia: associations between clinical infections, PTSD, psychotropic PTSD medication use, and the risk for dementia." Diss., University of Iowa, 2015. https://ir.uiowa.edu/etd/2117.

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Dementia is a major public health problem worldwide. Emerging research indicates that clinical infections and PTSD could be important risk factors for dementia. However, evidence for infections and the risk of dementia primarily examines central nervous system (CNS) infections. Extant epidemiological evidence for systemic bacterial infections and the risk for dementia is limited while that for PTSD and the risk for dementia did not account for psychotropic medications commonly used in management of PTSD and could affect cognitive function. The purpose of this study was to 1) review the evidence for CNS infections as possible causes of Alzheimer’s disease (AD) dementia, and 2) using nationwide Veterans Health Administration databases, conduct original retrospective cohort analyses in nationally representative samples of U.S. veterans aged 56 years and older to determine the associations between systemic bacterial infections, PTSD, and psychotropic PTSD medication use with the risk for developing dementia. Review of the research pertaining to an infectious AD etiology hypothesis including the various mechanisms through which different clinical and subclinical infections could cause or promote the progression of AD, and the concordance between putative infectious agents and the epidemiology of AD showed evidence linking AD to an infectious cause to be largely inconclusive; however, the amount of evidence suggestive of an association is too substantial to ignore. Analysis of the associations between systemic bacterial infections and the risk for dementia showed a significant association between exposure to any systemic bacterial infection and an increased risk for dementia (hazard ratio [HR] = 1.20; 95% confidence interval [CI] = 1.16-1.24) after adjustment for demographic characteristics, and medical and psychiatric comorbidity. In addition, septicemia (HR=1.39; 95%CI=1.16-1.66), bacteremia (HR=1.22; 95%CI=1.0-1.49), osteomyelitis (HR=1.20; 95%CI=1.06-1.37), pneumonia (HR=1.10; 95%CI=1.02-1.19), UTI (HR=1.13; 95%CI=1.08-1.18), and cellulitis (HR=1.14; 95%CI=1.09-1.20) were independently associated with significantly increased risk of developing dementia after adjustment for potential confounders. Analysis of the associations between PTSD and psychotropic PTSD medication use with the risk for dementia showed a significant association between PTSD and the risk for dementia (HR=1.35; 95%CI=1.27-1.43) after adjustment for demographic characteristics, medical and psychiatric comorbidity, and health care utilization. Analysis of the impact of psychotropic PTSD medications including selective serotonin reuptake inhibitors (SSRI), serotonin-norepinephrine reuptake inhibitors (SNRI), benzodiazepines (BZA), novel antidepressants (NA) and atypical antipsychotics (AA) on the association between PTSD and the risk for dementia showed significant interactions between PTSD and use of SSRIs (p<.0001), NAs (p=.0016), and AAs (p<.0001). Multivariate analysis showed a significant association between PTSD and an increased risk for dementia among individuals not using any psychotropic PTSD medications at baseline (HR=1.70; 95%CI=1.58-1.82). PTSD patients using SSRIs (HR=2.10; 95%CI=1.82-2.41), NAs (2.19; 95%CI=1.94-2.48) or AAs (4.56; 95%CI=4.04-5.15) were significantly more likely to develop dementia compared to those without PTSD and not using any psychotropic PTSD medications. PTSD patients using SSRIs (HR=1.24; 95%CI=1.08-1.42), NAs (HR=1.29; 95% CI=1.14-1.46) or AAs (HR=2.69; 95%CI=2.38-3.04) were also significantly more likely to develop dementia compared to those with PTSD and not using any psychotropic PTSD medications. SNRI (HR=1.35; 95%CI=1.26-1.46) and BZA drug use (HR=1.40; 95%CI=1.35-1.45) at baseline was associated with an increased risk for dementia regardless of PTSD diagnosis. These findings indicate; 1) evidence for an infectious AD etiology hypothesis in inconclusive, 2) both severe (e.g. sepsis), and less severe (e.g. cellulitis) systemic bacterial infections are collectively and independently associated with an increased risk of dementia among older U.S. veterans hence prevention of systemic bacterial infections could positively influence the risk for dementia among older adults, and 3) PTSD and psychotropic medication use are associated with an increased risk for dementia among U.S. veterans. Further epidemiologic, clinical, and basic science research is required to elucidate the mechanisms and the associations between infections and the risk for dementia and to determine if the independent and effect modifying impacts of psychotropic PTSD medication use on the risk for dementia are related to differences in PTSD severity, other psychiatric comorbidity, or whether psychotropic PTSD medication use is an independent risk factor for dementia.
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Jong, F. J. de. "Endocrine factors, retinal vessels, and risk of dementia." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2007. http://hdl.handle.net/1765/13953.

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Podewils, Laura Jean. "Physical activity and dementia risk a prospective study /." Available to US Hopkins community, 2003. http://wwwlib.umi.com/dissertations/dlnow/3080747.

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Billioti, de Gage Sophie. "Benzodiazepines and risk of dementia in the elderly." Thesis, Bordeaux, 2015. http://www.theses.fr/2015BORD0106/document.

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Ce travail porte sur l’étude du risque de démence chez les personnes âgées ayant consommé des benzodiazépines. Ces médicaments méritent une attention particulière du fait de (i) leur utilisation trop systématique et le plus souvent chronique contrairement aux recommandations préconisant des durées d’utilisation courtes (ii) leurs effets délétères sur la cognition demeurant mal évalués à long terme. La plupart des études conduites sur ce sujet ont conclu à une augmentation du risque de démence chez les sujets ayant utilisé des benzodiazépines. Un biais protopathique pouvait cependant, en partie du moins, avoir expliqué ces résultats : la prescription de benzodiazépines pouvait avoir été motivée par des prodromes souvent observés au cours des années précédant le diagnostic de la maladie. Afin de mieux prendre en considération ce biais, le projet BENZODEM a utilisé les ressources de la cohorte PAQUID (3777 sujets ≥ 65 ans tirés au sort sur les listes électorales de Dordogne et Gironde bénéficiant d’un suivi de plus de 20 ans). Ce projet, combinant deux études de cohorte et une étude cas-­‐témoins, a conclu à un risque de démence augmenté de 46 à 62% chez les utilisateurs de benzodiazépines et retardé de 5 à 15 ans par rapport à l’initiation du traitement. La seconde partie du programme (BENZODEM2) a consisté en une étude cas-­‐témoins conduite sur un large échantillon de sujets de plus de 65 ans enregistrés sur la base de données de la Régie de l’Assurance Maladie du Québec (RAMQ). Ce programme a permis (1) de valider les précédents résultats (risque augmenté de 30 à 80% en fonction de la dose, la durée du traitement et la nature des molécules) (2) d’identifier les profils de consommation associés à un excès de risque : consommateurs de plus de 3 mois avec une relation dose-­‐effet marquée et molécules à longue demi-­‐ vie d’élimination. Des explorations complémentaires ont permis de conclure que cet excès de risque n’était pas expliqué par une mortalité différentielle entre groupes comparés ni par la prescription d’autres médicaments psychotropes. Une autre étude menée sur PAQUID montrait une absence de différence entre consommateurs et non consommateurs de benzodiazépines vis-­‐à-­‐vis de l’évolution des scores mesurant les fonctions cognitives. Ces résultats ont permis d’émettre des hypothèses concernant le mécanisme de l’association entre utilisation de benzodiazépines et démence: (1) les benzodiazépines pourraient constituer des marqueurs précoces de la maladie ; (2) les benzodiazépines pourraient aussi diminuer les capacités de recours à la réserve cognitive en réponses aux lésions précoces de la maladie au stade préclinique ; (3) il est aussi possible que ces deux explications soient combinées
This work deals with the risk of dementia in elderly individuals who have used benzodiazepines. These drugs deserve particular attention because (i) their use appears to be too systematic and most often chronic despite good practice guidelines recommending short durations of use (ii) their deleterious effects on cognition remain underevaluated for the long-­‐term. Most of the studies conducted concluded that there was an increased risk of dementia among benzodiazepine users. In fact, a protopathic bias could, at least in part, have explained these results. Indeed, the prescription of benzodiazepines could have been motivated by the prodromes often observed several years before the clinical diagnosis of a dementia. With the aim of better controlling for this bias, the BENZODEM project used the resources of the PAQUID cohort (3777 subjects ≥65 years randomly sampled from electoral lists in South-­‐West France, with a 20-­‐ year follow-­‐up). This project combined two cohort studies and one case-­‐control. These studies concluded in a risk of dementia increased by 46 to 62% in benzodiazepine users and delayed by 5 to 15 years after treatment initiation. The second part of the programme (BENZODEM2) consisted of a case-­‐control study conducted in a large sample of subjects >65 years registered in the Quebec Health care database (Régie de l’Assurance Maladie du Québec, RAMQ). It was thus possible(1) to validate the previous results by using a different population (the risk was found to be increased by 30 to 80% depending on the patterns of use regarding dose, duration and type of molecule), (2) to identify the patterns of use which appeared to be at risk; excess risk was only apparent for uses of more than three months with a marked dose-­‐effect relationship, and was higher for molecules with a long elimination half-­‐life. Complementary explorations using the PAQUID cohort indicated that the excess risk in exposed was not explained by a differential mortality rate between the groups compared. Other studies suggested that the link found remained independently of the prescription of other psychotropics. Another analysis in the PAQUID cohort showed that, in the absence of dementia, no difference was observed between benzodiazepine users and non-­‐users with regards to the evolution of scores evaluating cognitive functions. These results led to several assumptions about the putative mechanism explaining the relationship found between benzodiazepine use and dementia: (1) benzodiazepines could be early markers of symptoms such as anxiety, depression or insomnia, which are potential prodromes or risk factors for this disease, (2) these drugs could also reduce the ability to use cognitive reserve in order to cope with early lesions of the disease during the preclinical stage, (3) the association found could also result from these two mechanisms
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Eriksson, Ulrika K. "Inflammation-associated risk factors for Alzheimer's disease and dementia." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-802-0/.

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Liolitsa, Danae. "Genetic risk factors in Alzheiner's disease : a hypothesis-based candidate gene approach." Thesis, King's College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.252104.

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Rönnemaa, Elina. "Predictors of Dementia : Insulin, Fatty Acids and Vascular Risk Factors." Doctoral thesis, Uppsala universitet, Medicinska och farmaceutiska vetenskapsområdet, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-164528.

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Identification of modifiable risk factors for Alzheimer’s disease (AD) is crucial in order to diminish suffering from this devastating disease. The aim of this thesis was to investigate if different aspects of glucose metabolism, insulin, fatty-acid composition or other vascular risk factors predict the future development of AD and dementia. This thesis is based on the Uppsala Longitudinal Study of Adult Men (ULSAM) cohort, which started in 1970. A total of 2322 men at age 50 were examined with focus on vascular risk factors. The cohort was re-examined at ages 60, 71, 77, 82 and 88. Incident diagnoses of AD, vascular dementia, other dementias and cognitive impairment were assessed in 2005–2010. The risk of AD was increased in subjects with lower early insulin response measured with both an intravenous glucose tolerance test at 50 years and an oral glucose tolerance test at 71 years of age. The presence of vascular risk factors such as hypertension, obesity, hypercholesterolemia and smoking increased the risk of future vascular dementia but not of AD. Furthermore, saturated fatty acids at midlife were inversely associated with risk of AD. No evidence of a protective effect of omega-3 fatty acids against dementia was found. The susceptibility allele, APOE ε4, was the strongest individual risk factor. APOE ε4 carriers with vascular risk factors had the greatest risk of developing dementia. Low insulin response was a risk factor for AD mainly in APOE ε4 non-carriers. Disturbances in insulin and glucose metabolism, vascular risk factors and fatty acids are linked differentially to the pathogenesis of AD and vascular dementia. These observations should be considered when future clinical approaches are planned to prevent and postpone the onset of dementia.
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Ding, Xiuhua. "MODELING DEMENTIA RISK, COGNITIVE CHANGE, PREDICTIVE RULES IN LONGITUDINAL STUDIES." UKnowledge, 2016. http://uknowledge.uky.edu/epb_etds/9.

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Dementia is increasing recognized as a major problem to public health worldwide. Prevention and treatment strategies are in critical need. Nowadays, research for dementia usually featured as complex longitudinal studies, which provide extensive information and also propose challenge to statistical methodology. The purpose of this dissertation research was to apply statistical methodology in the field of dementia to strengthen the understanding of dementia from three perspectives: 1) Application of statistical methodology to investigate the association between potential risk factors and incident dementia. 2) Application of statistical methodology to analyze changes over time, or trajectory, in cognitive tests and symptoms. 3) Application of statistical learning methods to predict development of dementia in the future. Prevention of Alzheimer’s disease with Vitamin E and Selenium (PREADViSE) (7547 subjects included) and Alzheimer’s disease Neuroimaging Initiative (ADNI) (591 participants included) were used in this dissertation. The first study, “Self-reported sleep apnea and dementia risk: Findings from the PREADViSE Alzheimer’s disease prevention trial ”, shows that self-reported baseline history of sleep apnea was borderline significantly associated with risk of dementia after adjustment for confounding. Stratified analysis by APOE ε4 carrier status showed that baseline history of sleep apnea was associated with significantly increased risk of dementia in APOE ε4 non-carriers. The second study, “comparison of trajectories of episodic memory for over 10 years between baseline normal and MCI ADNI subjects,” shows that estimated 30% normal subjects at baseline assigned to group 3 and 6 stay stable for over 9 years, and normal subjects at baseline assigned to Group 1 (18.18%) and Group 5 (16.67%) were more likely to develop into dementia. In contrast to groups identified for normal subjects, all trajectory groups for MCI subjects at baseline showed the tendency to decline. The third study, “comparison between neural network and logistic regression in PREADViSE trial,” demonstrates that neural network has slightly better predictive performance than logistic regression, and also it can reveal complex relationships among covariates. In third study, the effect of years of education on response variable depends on years of age, status of APOE ɛ4 allele and memory change.
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De, Ronchi Diana. "Education and dementing disorders : the role of schooling in dementia and cognitive impairment /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-349-3/.

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Books on the topic "Dementia risk"

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Clive, Ballard, and Lovestone Simon, eds. Management of dementia. 2nd ed. New York: Informa Healthcare USA, 2008.

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Jacobsen, Sarah R. Vascular dementia: Risk factors, diagnosis, and treatment. Hauppauge, N.Y: Nova Science, 2011.

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Gilmour, Helen. Living alone with dementia: Diagnosis, care planning, risk. [Enniskillen: The Author], 2002.

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Román, Gustavo C. Managing vascular dementia: Concepts, issues, and management. London: Science Press, 2003.

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Clarke, Charlotte L. Risk assessment and management for living well with dementia. London: Jessica Kingsley Publishers, 2011.

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Anderson, Nicole D. Living with mild cognitive impairment: A guide to maximizing brain health and reducing risk of dementia. Oxford: Oxford University Press, 2012.

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Rodolfo, Paoletti, and International Symposium on Women's Health in Menopause (3rd : 1998 : Florence, Italy), eds. Women's health and menopause: Risk reduction strategies--improved quality of health. Dordrecht: Kluwer Academic Publishers, 1999.

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American madness: The rise and fall of dementia praecox. Cambridge, Mass: Harvard University Press, 2011.

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Legal issues relating to football head injuries: Hearings before the Committee on the Judiciary, House of Representatives, One Hundred Eleventh Congress, first and second sessions, October 28, 2009 and January 4, 2010. Washington: U.S. G.P.O., 2010.

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Reducing Your Risk Of Dementia. Sheldon, 2011.

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Book chapters on the topic "Dementia risk"

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Amaducci, L., and A. Lippi. "Risk Factors for Dementia." In Dementia, 129–41. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-6805-6_9.

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Rylance, Rebecca, and James Kidd. "Risk management." In Dementia Care, 47–57. Boca Raton, FL : CRC Press, 2016.: Routledge, 2018. http://dx.doi.org/10.4324/9781315382074-5.

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Nakamura, Shigenobu, Hirofumi Maruyama, Hiromasa Toji, Hideshi Kawakami, Michiko Yamada, and Yasuyo Mimori. "Risk factors for dementia." In Neuroscientific Basis of Dementia, 279–86. Basel: Birkhäuser Basel, 2001. http://dx.doi.org/10.1007/978-3-0348-8225-5_34.

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Alon, Sara. "Cognitive Impairment and Dementia: A Risk Factor for Elder Abuse and Neglect." In Dementia Care, 269–82. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3864-0_15.

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Hollingworth, Paul, and Julie Williams. "Genetic Risk Factors for Dementia." In The Handbook of Alzheimer's Disease and Other Dementias, 195–234. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444344110.ch6.

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Jeelani, Humira, Jahangir Nabi, Yasmeena Akhter, Nahida Tabasum, Dil Afroze, Faheem Hyder Pottoo, and Fasil Ali. "ApoE: A Risk Factor for Dementia." In Current Thoughts on Dementia, 3–29. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-7606-2_1.

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Mad’ari, Aladár, Jana Farbakova, and Norbert Žilka. "Preventive and Risk Factors of Canine Dementia." In Canine and Feline Dementia, 145–54. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-53219-6_9.

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Mante, Priscilla Kolibea, and Nana Ofori Adomako. "Sex Hormones as Risk Factors for Dementia." In Current Thoughts on Dementia, 85–114. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-7606-2_4.

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Zelinski, Elizabeth M. "Does cognitive training reduce risk for dementia?" In APA handbook of dementia., 419–32. Washington: American Psychological Association, 2018. http://dx.doi.org/10.1037/0000076-022.

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Roberto, Karen A., and Kirby Deater-Deckard. "Risk for elder abuse among persons with dementia." In APA handbook of dementia., 599–614. Washington: American Psychological Association, 2018. http://dx.doi.org/10.1037/0000076-032.

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Conference papers on the topic "Dementia risk"

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O'Donoghue, Jim, Mark Roantree, and Andrew McCarren. "Variable interactions in risk factors for dementia." In 2016 IEEE Tenth International Conference on Research Challenges in Information Science (RCIS). IEEE, 2016. http://dx.doi.org/10.1109/rcis.2016.7549314.

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Weiner, Jochen, Claudia Frankenberg, Johannes Schroder, and Tanja Schultz. "Speech Reveals Future Risk of Developing Dementia: Predictive Dementia Screening from Biographic Interviews." In 2019 IEEE Automatic Speech Recognition and Understanding Workshop (ASRU). IEEE, 2019. http://dx.doi.org/10.1109/asru46091.2019.9003908.

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Myasoedova, Elena, and Susanne Kammerer. "Risk factors for dementia in RA patients discovered." In EULAR 2022 Congress, edited by Elena Myasoedova and Dennis McGonagle. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/bd04efaa.

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Innocencio, Giovanna de Camargo, Paulo Roberto Hernandes Júnior, Juliana de Souza Rosa, Patrick de Abreu Cunha Lopes, and Jhoney Francieis Feitosa. "Epidemiological profile of Dementia in the state of São Paulo in the last 5 years." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.175.

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Background: dementia is a syndrome characterized by the presence of a progressive deficit in cognitive function, with interference in social and occupational activities, with risk factors varying with genetic and environmental stressors. The differential diagnosis must identify potentially reversible conditions, of different etiologies, such as metabolic changes, intoxications, and nutritional deficiencies. In primary degenerative dementias and sequelae forms, the etiological diagnosis carries therapeutic and prognostic implications. Objectives: to analyse the current epidemiological profile of dementia in the state of São Paulo in the last 5 years. Methods: a literature review was carried out based on articles available in the Scielo and PubMed database and an observational, descriptive and cross- sectional collection of epidemiological data on dementias available in DATASUS – SUS Hospital Information System (SIH/SUS) – in the last 5 years – January 2016 to December 2020 – assessing the number of hospitalizations, the amount of public spending, mortality rate and permanence. Results: in the analyzed period, 3.105 hospitalizations were observed due to occurrences related to dementia, representing a total expenditure of R$37.847.961,13, with 2017 being the year with the highest number of hospitalizations and responsible for the highest amount spent. The total mortality rate in the 5 years studied was 5,57, corresponding to 173 deaths, with 2020 being the year with the highest rate while 2017 had the lowest rate. The average of the permanence in the hospital was 180 days. Conclusion: patients with dementia need early diagnosis and procedures to reduce the rate of hospitalizations and mortality, as well as public costs. For this, technological innovations, using structural and functional neuroimaging methods, as well as biology and molecular genetics techniques, have presented perspectives for the early diagnosis of dementia.
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Huang, Yo-Ping, and Samuele Zaza. "Evaluating dementia risk from MRI by fuzzy inference model." In 2014 IEEE 11th International Conference on Networking, Sensing and Control (ICNSC). IEEE, 2014. http://dx.doi.org/10.1109/icnsc.2014.6819705.

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Saidane, O., M. Sellami, I. Mahmoud, A. Ben Tekaya, H. Ajlani, R. Tekaya, and L. Abdelmoula. "AB0387 Is rheumatoid arthritis a risk factor for dementia ?" In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.4715.

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Eltelbany, Ahmed, and Karin Drooff. "Biologic treatment decreases dementia risk in senior IBD patients." In Digestive Disease Week 2022, edited by Rachel Giles. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/c9f10dda.

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Fitriana, Lisna Anisa, Slamet Rohaedi, Sri Sumartini, Linda Amalia, and Lilis Komariyah. "Risk Factors of Dementia in Elderly at Nursing Home." In Proceedings of the 3rd International Conference on Sport Science, Health, and Physical Education (ICSSHPE 2018). Paris, France: Atlantis Press, 2019. http://dx.doi.org/10.2991/icsshpe-18.2019.48.

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Scherrer, Jeffrey, Joanne Salas, Christine Jacobs, and Timothy Wiemken. "Lower dementia risk in patients vaccinated against Herpes Zoster." In NAPCRG 49th Annual Meeting — Abstracts of Completed Research 2021. American Academy of Family Physicians, 2022. http://dx.doi.org/10.1370/afm.20.s1.2590.

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Tseng, Hsiao-Ting, Hsiao-Chi Li, Chia-Lun Lo, Tai-Hsiang Shen, and Shu-Chiung Lin. "Predicting Dementia Risk to Depressive Disorder Patients: A classification Approach." In 2019 International Conference on Machine Learning and Cybernetics (ICMLC). IEEE, 2019. http://dx.doi.org/10.1109/icmlc48188.2019.8949191.

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Reports on the topic "Dementia risk"

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Fillipin, Federico, Pamela Seron, and Ruvistay Gutierrez-Arias. Effectiveness of antihypertensive drugs to prevent cognitive decline, mild cognitive impairment, and dementia. An overview of systematic reviews. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0057.

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Review question / Objective: To determine effectiveness of antihypertensive drugs to prevent different dementia subtypes such as Alzheimer’s disease and vascular dementia. Condition being studied: Dementia is a global health burden, with the number of affected individuals increasing. A recent meta-analysis reported that the prevalence of all-type dementia was 697 per 10,000 people and the prevalence of Alzheimer’s disease was 324 per 10,000 people. The SHEP and SYST-EUR were the two first randomized controlled trials to show that hypertension treatment reduces dementia risk.
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Wei, Huijie, Xin Mu, Yu Li, Hua Lei, De Yang, Tian Li, and Junwei Ren. Meta-analysis of the association between CHCHD10 Pro34Ser variant and the risk of frontotemporal dementia. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2021. http://dx.doi.org/10.37766/inplasy2021.5.0090.

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Chen, Hui, Yuhui Huang, Liyan Huang, and Changzheng Yuan. Mediterranean-Dietary Approaches to Stop Hypertension (MIND) diet and risk of dementia: Meta-analysis of cohort studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0127.

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Basis, Najwa, and Tamar Shochat. Associations between religion and sleep: A systematic review of observational studies in the adult population. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0057.

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Review question / Objective: The aim of this systematic review is to investigate observational studies on the association between religion and sleep in the adult population. To this end, the proposed systematic review will address the following question: What is the role religion plays in shaping an individual's sleep health? Condition being studied: Sleep is a fundamental biological process increasingly recognized as a critical indicator of development and overall health. Generally, insufficient sleep is associated with depressed mood, daytime fatigue, poor daytime functioning and daytime sleepiness, increased risk of cancer, cardiovascular problems, diabetes, and the cause of the higher risk of mortality. Furthermore, changes in sleep architecture and quality have been related to cognitive deterioration, including dementia and Alzheimer's disease. Here we will identify the role of religion in elements of sleep health, to include sleep duration and sleep quality, and associated health outcomes in the adult population.
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A less healthy lifestyle increases the risk of dementia. National Institute for Health Research, November 2019. http://dx.doi.org/10.3310/signal-000847.

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Being overweight is linked with an increased risk of dementia in new research. National Institute for Health Research, March 2021. http://dx.doi.org/10.3310/alert_45547.

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