Academic literature on the topic 'Defibrillation'

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Journal articles on the topic "Defibrillation"

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Ul Haq, Ehtesham, and Bassam Omar. "Traumatic Tension Pneumothorax as a Cause of ICD Failure: A Case Report and Review of the Literature." Case Reports in Cardiology 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/261705.

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Background. Tension pneumothorax can infrequently cause ventricular arrhythmias and increase the threshold of defibrillation. It should be suspected whenever there is difficulty in defibrillation for a ventricular arrhythmia.Purpose. To report a case of traumatic tension pneumothorax leading to ventricular tachycardia and causing defibrillator failure.Case. A 65-year-old African-American female was brought in to our emergency department complaining of dyspnea after being forced down by cops. She had history of mitral valve replacement for severe mitral regurgitation and biventricular implantable cardioverter defibrillator inserted for nonischemic cardiomyopathy. Shortly after arrival, she developed sustained ventricular tachycardia, causing repetitive unsuccessful ICD shocks. She was intubated and ventricular tachycardia resolved with amiodarone. Chest radiograph revealed large left sided tension pneumothorax which was promptly drained. The patient was treated for congestive heart failure; she was extubated on the third day of admission, and the chest tube was removed.Conclusion. Prompt recognition of tension pneumothorax is essential, by maintaining a high index of suspicion in patients with an increased defibrillation threshold causing ineffective defibrillations.
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Curnis, Antonio, Claudio Muneretto, Gianluigi Bisleri, Manuel Cerini, Lorenza Inama, Francesca Salghetti, Raffaella De Vito, et al. "Thoracoscopic Implantation of An Array Electrode in the Pericardium Transverse Sinus to Reduce Defibrillation Threshold." Innovations: Technology and Techniques in Cardiothoracic and Vascular Surgery 12, no. 4 (July 2017): e6-e9. http://dx.doi.org/10.1097/imi.0000000000000384.

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Among the implantable cardioverter defibrillator recipients, there is still a subgroup of patients in whom the defibrillation threshold is too high and the maximal shock output of the implantable cardioverter defibrillator can fail to terminate a ventricular arrhythmia. We report a new thoracoscopic minimally invasive approach to place a standard array electrode in the transverse pericardial sinus of a patient implanted with a cardiac resynchronization and defibrillation therapy device with persistent high defibrillation threshold. This approach was developed to achieve very low shock impedance with a consequent increase in the current flow and reduction of defibrillation threshold.
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Skyschally, Andreas, Georgios Amanakis, Markus Neuhäuser, Petra Kleinbongard, and Gerd Heusch. "Impact of electrical defibrillation on infarct size and no-reflow in pigs subjected to myocardial ischemia-reperfusion without and with ischemic conditioning." American Journal of Physiology-Heart and Circulatory Physiology 313, no. 5 (November 1, 2017): H871—H878. http://dx.doi.org/10.1152/ajpheart.00293.2017.

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Ventricular fibrillation (VF) occurs frequently during myocardial ischemia-reperfusion (I/R) and must then be terminated by electrical defibrillation. We have investigated the impact of VF/defibrillation on infarct size (IS) or area of no reflow (NR) without and with ischemic conditioning interventions. Anesthetized pigs were subjected to 60/180 min of coronary occlusion/reperfusion. VF, as identified from the ECG, was terminated by intrathoracic defibrillation. The area at risk (AAR), IS, and NR were determined by staining techniques (patent blue, triphenyltetrazolium chloride, and thioflavin-S). Four experimental protocols were analyzed: I/R ( n = 49), I/R with ischemic preconditioning (IPC; n = 22), I/R with ischemic postconditioning (POCO; n = 22), or I/R with remote IPC (RIPC; n = 34). The incidence of VF was not different between I/R (44%), IPC (45%), POCO (50%), and RIPC (33%). IS was reduced by IPC (23 ± 12% of AAR), POCO (31 ± 16%), and RIPC (22 ± 13%, all P < 0.05 vs. I/R: 41 ± 12%). NR was not different between protocols (I/R: 17 ± 15% of AAR, IPC: 15 ± 18%, POCO: 25 ± 16%, and RIPC: 18 ± 17%). In pigs with defibrillation, IS was 50% larger than in pigs without defibrillation but independent of the number of defibrillations. Analysis of covariance confirmed the established determinants of IS, i.e., AAR, residual blood flow during ischemia (RMBFi), and a conditioning protocol, and revealed VF/defibrillation as a novel covariate. VF/defibrillation in turn was associated with larger AAR and lower RMBFi. Lack of dose-response relation between IS and the number of defibrillations excluded direct electrical injury as the cause of increased IS. Obviously, AAR size and RMBFi account for both IS and the incidence of VF. IS and NR are mechanistically distinct phenomena. NEW & NOTEWORTHY Ventricular fibrillation/defibrillation is associated with increased infarct size. Electrical injury is unlikely the cause of such association, since there is no dose-response relation between infarct size and number of defibrillations. Ventricular fibrillation, in turn, is associated with a larger area at risk and lower residual blood flow.
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Ross, Linda, Brett Williams, and Malcolm Boyle. "Defibrillation safety: an examination of paramedic perceptions using eye-tracking technology." BMJ Simulation and Technology Enhanced Learning 1, no. 2 (September 3, 2015): 62–66. http://dx.doi.org/10.1136/bmjstel-2015-000033.

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ObjectiveThe importance of access to early defibrillation for patients in cardiac arrest has been emphasised as a critical part of the chain of survival by resuscitation bodies internationally; as such defibrillation has become a key procedure for many out-of-hospital emergency healthcare providers. However, little research has been undertaken specifically addressing students’ safety during defibrillation procedures. The objective of this study was to examine visual and verbal safety checks prior to defibrillation utilising eye-tracking technology.MethodsThis was an observational study of student safety during cardiac rhythm analysis, defibrillator charging and immediately prior to defibrillation during a resuscitation attempt using a medium fidelity mannequin. The participants completed two 10 min simulations each requiring three defibrillation attempts. The κ statistic was used to determine the agreement by the student of their perceived safety performance and that viewed in the video.ResultsIn both scenarios the student's level of agreement for their perceived defibrillation safety performance and what was observed in the video decreased from defibrillation one to three in both scenarios. However, there was agreement in their overall defibrillation safety performance for both scenarios.ConclusionsStudent perceptions of their actions during defibrillation are not always an accurate representation of their actual actions.
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Qu, Fujian, Fidel Zarubin, Brian Wollenzier, Vladimir P. Nikolski, and Igor R. Efimov. "The Gurvich waveform has lower defibrillation threshold than the rectilinear waveform and the truncated exponential waveform in the rabbit heart." Canadian Journal of Physiology and Pharmacology 83, no. 2 (February 1, 2005): 152–60. http://dx.doi.org/10.1139/y04-131.

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Implantable cardioverter defibrillator studies have established the superiority of biphasic waveforms over monophasic waveforms. However, external defibrillator studies of biphasic waveforms are not as widespread. Our objective was to compare the defibrillation efficacy of clinically used biphasic waveforms, i.e., truncated exponential, rectilinear, and quasi-sinusoidal (Gurvich) waveforms in a fibrillating heart model. Langendorff-perfused rabbit hearts (n = 10) were stained with a voltage-sensitive fluorescent dye, Di-4-ANEPPS. Transmembrane action potentials were optically mapped from the anterior epicardium. We found that the Gurvich waveform was significantly superior (p < 0.05) to the rectilinear and truncated exponential waveforms. The defibrillation thresholds (mean ± SE) were as follows: Gurvich, 0.25 ± 0.01 J; rectilinear-1, 0.34 ± 0.01 J; rectilinear-2, 0.33 ± 0.01 J; and truncated exponential, 0.32 ± 0.02 J. Using optically recorded transmembrane responses, we determined the shock-response transfer function, which allowed us to predict the cellular response to waveforms at high accuracy. The passive parallel resistor-capacitor model (RC-model) predicted polarization superiority of the Gurvich waveform in the myocardium with a membrane time constant (τm) of less than 2 ms. The finding of a lower defibrillation threshold with the Gurvich waveform in an in vitro model of external defibrillation suggests that the Gurvich waveform may be important for future external defibrillator designs.Key words: defibrillation, optical mapping, biphasic waveform, Gurvich waveform.
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Bänsch, Dietmar. "Defibrillation Testing During Defibrillator Implantation." Arrhythmia & Electrophysiology Review 1 (2012): 51. http://dx.doi.org/10.15420/aer.2012.1.51.

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Implantable cardioverter defibrillators (ICDs) terminate ventricular tachycardia (VT) and ventricular fibrillation (VF) with high efficacy. ICDs improve mortality in patients after survived sudden cardiac death (SCD) and in patients at high risk of dying suddenly. All trials which show a benefit of ICD therapy, have performed some kind of defibrillation testing in order to prove correct system function, sensing of VF and effective defibrillation. Current devices show a shock efficacy of 80–90 % for singular shocks and devices provide up to seven rescue shocks. The probability that a device does not terminate an episode of VT or VF should therefore be very low. However, it is difficult to abandon defibrillation testing because prospective data is lacking that demonstrate non-inferiority, if ICDs are implanted without some kind of test. Two prospective trials are on the way and will be finish by 2013/14: the SIMPLE and NORDIC trial, which will answer the question if defibrillation testing can be abandoned without any effect on the benefit of ICD therapy or if testing may even be harmful.
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Peters, W., S. Solingen, Y. Kobayashi, R. Scharf, W. J. Mandel, and E. S. Gang. "Transmyocardial impedance during single and multiple internal ventricular defibrillation shocks." American Journal of Physiology-Heart and Circulatory Physiology 267, no. 2 (August 1, 1994): H684—H693. http://dx.doi.org/10.1152/ajpheart.1994.267.2.h684.

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Little is known about the transmyocardial impedance during internal ventricular defibrillation. In a canine model, using high rate on-line digitization, random shock delivery, and titanium electrodes, we determined the relationship among voltage, current, and impedance, delivered energy, and defibrillation success within the individual and within successive defibrillation shocks. Impedance decreased with repeated defibrillation in 10 of 11 dogs. Impedance always increased during trapezoidal discharges, whereas voltage decreased. Impedance was lower with high energy-voltage shocks in all dogs. Visually, voltage and current waveform did not show a phase shift. There was no difference in the total energy delivered and the energy converted into heat by the resistive part of the impedance. With a formula valid only for resistive loads, the capacitance of the defibrillator was calculated to be within the measurement accuracy and tolerance of the factory-provided value of 132 microF. Polarization voltage was consistently observed. Thus the transmyocardial impedance during defibrillation is primarily resistive, nonlinear voltage dependent, and declines with successive shocks. Defibrillation success was not influenced by these phenomena.
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Prokopenko, A. V., and E. A. Ivanitskiy. "Experience of using the of subcutaneous cardioverter-defibrillators in the world practice: review." Journal of Arrhythmology 29, no. 4 (December 8, 2022): 42–46. http://dx.doi.org/10.35336/va-2022-4-06.

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The article provides a review of international clinical studies on the use of a subcutaneous implantable cardioverter-defibrillator (ICD) in comparison with classical intravenous defibrillation systems. Subcutaneous ICDs have shown themselves to be a worthy alternative to intravenous defibrillating systems for the primary prevention of sudden cardiac death, when the patient is not indicated for anti-tachy stimulation and anti-brady stimulation. World experience on the use of subcutaneous ICDs proves the safety and effectiveness of the functioning of the subcutaneous ICD system, excluding from the patient’s life the formidable risks associated with the implantation procedure and further functioning of the classical intravenous ICD system.
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Goodloe, J. M., L. D. Vinson, M. L. Cox, and B. D. Burns. "P059: Paramedic compliance with a novel defibrillation strategy in a large, urban EMS system in the United States." CJEM 19, S1 (May 2017): S98. http://dx.doi.org/10.1017/cem.2017.261.

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Introduction: Emergency Medical Services (EMS) care confers distinct impact upon survivability from sudden cardiac arrest. Many studies have been conducted regarding EMS interventions for cardiac arrest, though fewer studies have been published detailing specific analysis of paramedic compliance with standing orders, particularly those involving a novel energy strategy in defibrillation. Methods: Adults in sudden cardiac arrest with resuscitation initiated, including at least one defibrillation, between July 1, 2016 and December 1, 2016 were enrolled. Education on a novel defibrillation strategy, involving weight-based joule settings and double sequential external defibrillation (DSED) was delivered in classroom and internet-accessed settings. Paramedics then performed hands-on practice in DSED. All resuscitations were reviewed from electronic medical records (EMRs) completed by treating paramedics, alongside telemetry and defibrillation events recorded, transmitted, and analyzed in proprietary software (CODE-STAT™, Physio-Control Corporation, Redmond, WA). All ECGs and defibrillation events were reviewed by an emergency physician to determine energy settings used by paramedics for determining the accuracy of compliance with protocol-based standing orders. Results: During the 5 month study period, the paramedics involved treated 133 adults in sudden cardiac arrest involving perceived ventricular fibrillation that was treated with at least one defibrillation. 76/90 (84.4%) with estimated weight &lt;100 kg were treated with correct joule settings, though only 7/43 (16.3%) with estimated weight ≥100kg received all defibrillations at 360J as protocol-specified. 26/44 (59.1%) in refractory ventricular fibrillation, defined as requiring a fourth defibrillation, received DSED as protocol-specified. Conclusion: Paramedics, when specifically trained on a novel defibrillation strategy, involving both weight-based joule settings and use of DSED for refractory ventricular fibrillation, are inconsistently able to quickly and successfully incorporate that strategy in EMS resuscitation care. Further educational endeavours are warranted to achieve higher defibrillation strategy protocol compliance.
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Chiladakis, John, Fani Zagkli, and Dimitrios Alexopoulos. "External defibrillation on an implantable defibrillator." Journal of Anesthesia 28, no. 2 (October 6, 2013): 312. http://dx.doi.org/10.1007/s00540-013-1710-9.

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Dissertations / Theses on the topic "Defibrillation"

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Oeding, Matthew. "Defibrillation safety." Thesis, Oeding, Matthew (2012) Defibrillation safety. Other thesis, Murdoch University, 2012. https://researchrepository.murdoch.edu.au/id/eprint/13113/.

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In the past years, there has been a dramatic transition between the use of older monophasic defibrillators to newer, more sophisticated, biphasic types. As these biphasic defibrillators are more efficient, they require less energy and therefore create less of a risk to bystanders. Due to the lack of research around these new defibrillators, the current recommended procedures may not accurately reflect the safety of medical personnel. Because of this, the recommended “all clear” period may in fact become detrimental to the health of the patient as it causes the cessation of crucial activities of medical staff such as IV canalization and chest compressions. This thesis is aimed at assisting in a study to be performed by the Professor of Emergency Medicine at Royal Perth Hospital by designing a device capable of measuring, storing and analyzing the leakage voltages from a patient and their environment whilst undergoing defibrillation. The device that was designed consisted of a data acquisition system that would measure the voltages using standard ECG leads, and then wirelessly transmit that data to a laptop for further processing. Throughout the entire design process, the focus was aimed at ensuring the device would meet all the criteria specified in the required standards and cause no detrimental effect to the patient being monitored. At the end of the thesis period, a functional schematic was designed and tested, ready for manufacture as well as a solid framework of the software component of the project.
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McKeown, Paschal Patrick Joseph. "Transoesophageal cardioversion and defibrillation." Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334471.

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Darragh, K. M. "Optimisation of Defibrillation for Ventricular Fibrillation." Thesis, Queen's University Belfast, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527675.

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Harbinson, Mark Thomas. "Studies in atrial and ventricular defibrillation." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361286.

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Caldwell, Jane Cochrane. "Ventricular fibrillation in ischaemia and its defibrillation." Thesis, University of Glasgow, 2006. http://theses.gla.ac.uk/6196/.

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ECG signals were recorded from isolated, Langendorff-perfused rabbit hearts to establish the relationship between dominant frequency and myocardial perfusion during ventricular fibrillation. Lower perfusion rates produced faster rates of dominant frequency decline, to lower steady state values. Optically mapping the anterior epicardial surface demonstrated heterogeneity of dominant frequency in ventricular fibrillation. During low-flow ischaemia, the dominant frequency reduction was restricted to the left ventricle. Application of individual ischaemic components during ventricular fibrillation demonstrated that raised [K+]EC, but not hypoxia or acidic pHEC, reproduced the ischaemic reduction of dominant frequency in the ECG, pseudoECG and over the left ventricular epicardial surface. In contrast, minimum defibrillation energies were increased by hypoxia and acidic pHEC, and not by raised [K+]EC. The dominant frequency heterogeneity during ventricular fibrillation in low-flow ischaemia and raised [K+]EC was not due to differential prolongation of repolarisation or post-repolarisation refractoriness in the left ventricle. Monophasic action potential studies showed that APD90 was reduced to similar degrees in each ventricle by low-flow ischaemia and raised [K+]EC. Effective refractory period was not altered in either ventricle by either condition. Low-flow ischaemia decreased conduction velocity in the left, but not the right ventricle. Conduction velocities were unaltered by raised [K+]EC in either ventricle. The activation threshold of the left ventricle was increased in low-flow ischaemia and raised [K+]EC, whilst the threshold of the right ventricle was unchanged. The increased activation threshold was associated with decreased upstroke velocity and diastolic depolarisation.
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Morgan, Stuart William. "Low-Energy Defibrillation Using Resonant Drift Pacing." Thesis, University of Liverpool, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.507718.

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Huber, Claudia. "Konzeption und Evaluation eines Qualitätsmanagementsystems im Bereich der Frühdefibrillation." kostenfrei, 2008. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1217/.

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Santos, José Angel. "Transcutaneous pulsed mode power delivery to implants for the treatment of atrial fibrillation." Thesis, University of Ulster, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251911.

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Wilson, Carol Mildred. "Studies on cardiac defibrillation : waveform, threshold and damage." Thesis, Queen's University Belfast, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357514.

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Walsh, S. J. "Biphasic waveforms for internal and external atrial defibrillation." Thesis, Queen's University Belfast, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.401795.

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Books on the topic "Defibrillation"

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Early defibrillation. St. Louis: Mosby Year Book, 1991.

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K, Copass Michael, ed. EMT defibrillation. 3rd ed. Akron, Ohio: Emergency Training, 1989.

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Cross, American Red, ed. Automated external defibrillation. St. Louis, MO: Mosby Lifeline, 1998.

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Graves, Judith Reid. Rapidzap: Automated defibrillation. Englewood Cliffs, N.J: Prentice Hall, 1989.

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Council, National Safety, ed. AED: Automated external defibrillation. Sudbury, Mass: Jones and Bartlett Publishers, 1998.

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EMT-D: Prehospital defibrillation. Bowie, Md: Brady Communication Co., 1985.

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American Safety & Health Institute. Automated external defibrillation [AED]. Holiday, FL: NIRC, 2003.

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Hayes, David L., and Paul A. Friedman, eds. Cardiac Pacing, Defibrillation and Resynchronization. Oxford, UK: Wiley-Blackwell, 2008. http://dx.doi.org/10.1002/9781444300659.

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Hayes, David L., Samuel J. Asirvatham, and Paul A. Friedman, eds. Cardiac Pacing, Defibrillation and Resynchronization. Oxford, UK: Wiley-Blackwell, 2013. http://dx.doi.org/10.1002/9781118483923.

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John, Camm A., and Lindemans Frederic Willem, eds. Transvenous defibrillation and radiofrequency ablation. Armonk, NY: Futura Pub., 1995.

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Book chapters on the topic "Defibrillation"

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Ahmed, Hesham M., Christopher T. Aquina, Vicente H. Gracias, J. Javier Provencio, Mariano Alberto Pennisi, Giuseppe Bello, Massimo Antonelli, et al. "Defibrillation." In Encyclopedia of Intensive Care Medicine, 683–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_194.

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Mellema, Matthew S., Craig Cornell, and Casey J. Kohen. "Defibrillation." In Advanced Monitoring and Procedures for Small Animal Emergency and Critical Care, 235–44. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118997246.ch18.

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Schneider, T., and B. Wolcke. "Early Defibrillation." In Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E., 801–11. Milano: Springer Milan, 2001. http://dx.doi.org/10.1007/978-88-470-2903-3_79.

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Walcott, Gregory P., Cheryl R. Killingsworth, and Raymond E. Ideker. "External Defibrillation." In Cardiopulmonary Resuscitation, 211–27. Totowa, NJ: Humana Press, 2005. http://dx.doi.org/10.1385/1-59259-814-5:211.

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Molina, J. Ernesto. "The Infracardiac Defibrillator Lead Implant for Defibrillation Therapy." In Cardiothoracic Surgical Procedures and Techniques, 107–11. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-75892-3_22.

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Singer, Igor, and Douglas Lang. "The Defibrillation Threshold." In Implantable Cardioverter Defibrillator Therapy: The Engineering-Clinical Interface, 89–129. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-6345-7_5.

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Blanchard, Susan M., Raymond E. Ideker, Randolph A. S. Cooper, and J. Marcus Wharton. "The Defibrillation Waveform." In Implantable Cardioverter Defibrillator Therapy: The Engineering-Clinical Interface, 147–61. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4615-6345-7_7.

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Laske, Timothy G., Anna Legreid Dopp, and Paul A. Iaizzo. "Pacing and Defibrillation." In Handbook of Cardiac Anatomy, Physiology, and Devices, 443–73. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-372-5_27.

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Brice, Aaron E. "Cardioversion and Defibrillation." In Cardiology Procedures, 229–34. London: Springer London, 2016. http://dx.doi.org/10.1007/978-1-4471-7290-1_27.

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Chawla, Rajesh, Roseleen Kaur Bali, and Pradeep Jain. "Defibrillation and Cardioversion." In ICU Protocols, 423–31. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-0902-5_42.

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Conference papers on the topic "Defibrillation"

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Anderson, Croal, Mckeown, Cochrane, and Adgey. "Transoesophageal Defibrillation." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595775.

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Anderson, J. A., S. Croal, P. Mckeown, D. J. Cochrane, and Aaj Adgey. "Transoesophageal defibrillation." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761162.

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Dalzell, McKeown, Roberts, Anderson, and Adgey. "Cellular Transtelephonic Defibrillation." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595857.

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Dalzell, G. W. N., P. P. McKeown, M. J. D. Roberts, J. Anderson, and A. A. J. Adgey. "Cellular transtelephonic defibrillation." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761243.

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Gale, T. J. "Modeling Defibrillation Electrode Performance." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1615309.

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Ideker, R. E., J. M. Wharton, N. Shibata, P. S. Chen, P. D. Wolf, and W. M. Smith. "The mechanism of ventricular defibrillation." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94482.

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Allen, Stewart, Carlisle, Kernohan, and Adgey. "Fibrillation Frequency And Ventricular Defibrillation." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595767.

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Allen, J. D., A. J. Stewart, E. J. F. Carlisle, W. G. Kernohan, and A. A. J. Adgey. "Fibrillation frequency and ventricular defibrillation." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761156.

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Dillon, Stephen M. "Optical recording for investigating electrical defibrillation." In OE/LASE '94, edited by Leonard J. Cerullo, Kenneth S. Heiferman, Hong Liu, Halina Podbielska, Abund O. Wist, and Lucia J. Zamorano. SPIE, 1994. http://dx.doi.org/10.1117/12.176580.

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Gorbunov, Boris B., Vyacheslav A. Vostrikov, Andrey A. Galyastov, Igor V. Nesterenko, Dmitry V. Telyshev, and Maxim V. Denisov. "Guaranteed Defibrillation on a Cardiomyocyte Model." In 2020 Ural Symposium on Biomedical Engineering, Radioelectronics and Information Technology (USBEREIT). IEEE, 2020. http://dx.doi.org/10.1109/usbereit48449.2020.9117701.

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Reports on the topic "Defibrillation"

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Pshezhetskiy, Dmitry, Tanveer Alam, and Heba Alshaker. Unsynchronised Cardioversion as a Cause of Ventricular Tachycardia in a Patient with Atrial Fibrillation. Nature Library, November 2020. http://dx.doi.org/10.47496/nl.ccr.2020.01.02.

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Abstract:
Background: Synchronised cardioversion (SC) is used to terminate tachycardic arrhythmia by applying electric current to the thorax. SC is synchronised to the R wave of the cardiac cycle and ventricular tachycardia (VT) or ventricular fibrillation (VF) can occur if an electrical shock is provided in a nonsynchronised way. Case Presentation: Here we present a case of a 66-year-old man who had elective cardioversion for atrial fibrillation worsened by severe left ventricular impairment. A manual defibrillator was used for the cardioversion, which, after the first synchronised shock, reverted to defibrillator mode. An unsynchronised shock was administered and induced VT, which was reverted to sinus rhythm with a defibrillation shock. Conclusion: When using manual defibrillator for SC, the machine needs to be set to a synchronised mode. The synchronisation to the R wave needs to be checked before every shock.
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Tucker, Dana L. Possible Triggers and Temporal Patterns of Implantable Cardioverter Defibrillator Discharges: A Preliminary Study. Fort Belvoir, VA: Defense Technical Information Center, January 1999. http://dx.doi.org/10.21236/ad1012254.

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Sylvester, James C. Testing And Evaluation of the Physio-Control, Inc., Lifepak 500 Automated External Defibrillator. Fort Belvoir, VA: Defense Technical Information Center, June 1998. http://dx.doi.org/10.21236/ada357731.

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Sylvester, James C. Testing and Evaluation of the Heartstream, Inc., Model EM Semi-Automatic Defibrillator External. Fort Belvoir, VA: Defense Technical Information Center, December 1998. http://dx.doi.org/10.21236/ada359533.

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Hade, Edward W., and Jacqueline D. Hale. Test and Evaluation of the Zoll Medical Inc., PD2OOO Cardiac Monitor/Pacemaker/Defibrillator System. Fort Belvoir, VA: Defense Technical Information Center, September 1997. http://dx.doi.org/10.21236/ada329155.

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Hade, Edward, and Jacqueline D. Hale. Testing and Evaluation of the Medical Research Laboratories, Inc., 360SLX Cardiac Monitor/Pacemaker/Defibrillator System. Fort Belvoir, VA: Defense Technical Information Center, May 1998. http://dx.doi.org/10.21236/ada349535.

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Haun, Jeffrey D., Joseph R. Licina, Bill Olding, and Martin Quattlebaum. Test and Evaluation Report of the Physio Control Defibrillator/Monitor Model LIFEPAK (Trade Name) 10. Fort Belvoir, VA: Defense Technical Information Center, March 1991. http://dx.doi.org/10.21236/ada234593.

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Nedd, Steven. Can a Self-Diagnostic Digitally Controlled Pacemaker/Defibrillator Device be Used for Alerting Military Personnel When a Soldier Health Condition Becomes Compromised Out in the Field. Fort Belvoir, VA: Defense Technical Information Center, January 2007. http://dx.doi.org/10.21236/ada469014.

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Steven, Nedd. Can A Self-Diagnostic Digitally Controlled Pacemaker/Defibrillator Device be Used For Alerting Military Personnel When a Soldier Health Condition Becomes Compromised Out in the Field. Fort Belvoir, VA: Defense Technical Information Center, September 2006. http://dx.doi.org/10.21236/ada471920.

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Shalganov, Tchavdar, Milko Stoyanov, and Vassil Traykov. Outcomes following catheter ablation for ventricular tachycardia in adult patients with structural heart disease and implantable cardioverter-defibrillator: protocol for an updated systematic review and meta-analysis of randomized studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0080.

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Review question / Objective: Does catheter ablation for scar-related monomorphic ventricular tachycardia improve outcomes (defined as any appropriate ICD therapy, appropriate ICD shocks, all-cause mortality, VT storm, cardiovascular mortality, cardiovascular hospitalizations, complications) in adult patients with ischemic or non-ischemic cardiomyopathy and implantable cardioverter-defibrillator? Condition being studied: Ventricular tachycardia in patients with structural heart disease is usually an arrhythmia using the myocardial scar as a substrate for reentry. It poses a risk of syncope and sudden cardiac death, especially in patients with reduced ejection fraction. Most antiarrhythmic drugs are of little value and their use is restricted in patients with LV systolic dysfunction. Catheter ablation is a viable option for the treatment of ventricular tachycardia. In patients with previous myocardial infarction the arrhythmogenic scar is located most frequently subendocardially and is readily accessible using endocardial approach, while in non-ischemic cardiomyopathy the scar is frequently located in the midmyocardial or subepicardial layers. This is the reason endocardial catheter ablation to be less effective in those patients and to more often necessitate epicardial approach.
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