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Books on the topic 'Deep vessels'

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Author: Grafiati

Published: 10 March 2023

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1

R, Bass B., Oak Ridge National Laboratory, and U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Engineering., eds. A comparison of analysis methodologies for predicting cleavage arrest of a deep crack in a reactor pressure vessel subjected to pressurized-thermal-shock loading conditions. Washington, DC: Division of Engineering, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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2

Müzesi, Bodrum Sualtı Arkeoloji, ed. Sparkles from the deep: Glass vessels of the Bodrum Museum of Underwater Archaeology. Çayırova, Gebze, İstanbul: Bericap, 2000.

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3

Pelletier, James Laurence. Deep-draft vessel owners, U.S.A. Augusta, Me: Marine Techniques, 1997.

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4

Pelletier, James Laurence. Deep-draft vessel owners, U.S.A. Augusta, Me: Marine Techniques, 1996.

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5

Pelletier, James Laurence. Deep-draft vessel owners, foreign. Augusta, Me: Marine Techniques, 1996.

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6

(Firm), Odyssey Marine Exploration, ed. Oceans Odyssey 3: The deep-sea Tortugas shipwreck, Straits of Florida : a merchant vessel from Spain's 1622 Tierra Firme fleet. Oxford, UK: Oxbow Books, 2013.

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7

Canada. Agreement amending treaty with Canada concerning Pacific Coast albacore tuna vessels and port privileges: Message from the President of the United States transmitting agreement amending treaty between the government of the United States of America and the government of Canada on Pacific Coast albacore tuna vessels and port privileges done at Washington, D.C., May 26, 1981 (The "Treaty"), effected by an exchange of diplomatic notes at Washington on July 17, 2002, and August 13, 2002 (The "Agreement"). Washington: U.S. G.P.O., 2003.

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8

Canada. Agreement amending treaty with Canada concerning Pacific Coast albacore tuna vessels and port privileges: Message from the President of the United States transmitting agreement amending treaty between the government of the United States of America and the government of Canada on Pacific Coast albacore tuna vessels and port privileges done at Washington, D.C., May 26, 1981 (The "Treaty"), effected by an exchange of diplomatic notes at Washington on July 17, 2002, and August 13, 2002 (The "Agreement"). Washington: U.S. G.P.O., 2003.

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9

Heaton, P. M. The "Redbrook": A deep-sea tramp : an account of the management and operation of a South Wales owned vessel in the 1960s. Abergavenny: P. M. Heaton, 1995.

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10

2002), Antarktis-Expedition mit FS "Polarstern" (19th. The expeditions ANTARKTIS-XIX/3-4 of the research vessel Polarstern in 2002: ANDEEP I and II : Antartic benthic deep-sea biodiversity--colonization history and recent community patterns. Bremerhaven: Alfred-Wegener-Institut für Polar- und Meeresforschung, 2003.

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11

United States. Congress. House. Committee on Merchant Marine and Fisheries. Subcommittee on Merchant Marine. Surplus Vessel Act of 1990: Hearing before the Subcommittee on Merchant Marine of the Committee on Merchant Marine and Fisheries, House of Representatives, One Hundred First Congress, second session on H.R. 5118, a bill to deem certain national defense, reserve vessels surplus property, to direct the administrator of General Services to dispose of those vessels by selling them for scrapping in the United States, and for other purposes, September 10, 1990. Washington: U.S. G.P.O., 1991.

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12

Oceanography, Scripps Institution of. Cumulative index to the Initial reports of the Deep Sea Drilling Project: Covering volumes 1 through 96, reporting the results of Legs 1 through 96 of the cruises of the Drilling Vessel Glomar Challenger August 1968-November 1983. Washington, D.C: National Science Foundation, 1991.

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13

Deep-Draft Vessel Owners, Operators and Managers, Fishing Vessels - Shallow and Deep-Draft, Yachts Sail & Motor (Mariner's Directory & Guide , Vol 5). Marine Techniques, 2000.

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14

Singer, Mervyn. Pathophysiology and causes of pulmonary embolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0170.

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Pulmonary embolus is predominantly due to thrombus breaking off from deep veins or from within the right heart, lodging within large or small vessels within the pulmonary vasculature, causing a variable degree of clinical features ranging from asymptomatic through to shock and cardiac arrest. Non-thrombotic causes include air or fat embolism. Outcome is predicated by the degree of right ventricular dysfunction. There are multiple risk factors including surgery, arrhythmias, prolonged immobility, venous stasis, pregnancy and an underlying pro-thrombotic tendency, either congenital or acquired. Numerous risk stratification scores have been developed derived from clinical features, imaging findings and biochemical markers of right ventricular strain and myocardial damage.

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15

Deep-Draft Vessel Owners, Operators and Managers (Mariner's Directory & Guide , Vol 7). Marine Techniques, Inc., 1998.

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16

O'Connell, Martha, Martha O'Connell, and J&B Press LLC. Within This Vessel: You Are the Lungs Outside and I'm Breathing So Deep. J&B Press LLC, 2019.

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17

Tull, Malcolm. The Shipping of Fremantle. Liverpool University Press, 2018. http://dx.doi.org/10.5949/liverpool/9780968128824.003.0005.

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This chapter discusses the development of port activity to suit the movement of ships. Over the course of the twentieth century the average vessel size grew from 5200 to 19,000 gross registered tons. The development in ship technology changed the nature, shape and weight of cargo and passenger ships significantly enough to require a vast restructuring of the port to include deep-water channels and berths - a costly endeavour.

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18

Katsuragawa, Mario, Luiz Vianna Nonato, and Francisco Luiz Vicentini Neto. Guia do oceanógrafo: Manuseio de cabos e acessórios. Brazil Publishing, 2021. http://dx.doi.org/10.31012/978-65-5861-334-3.

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Oceanography is a multidisciplinary science that deals with the physical, chemical, geological e biological aspects of the marine environment. As most of the sciences, oceanographic research relies heavily on sampling procedures, which can be rather simple, as obtaining a water sample at sea surface, or very complex, as bringing an uncontaminated sediment sample from hadal regions (up to 11,000 m deep) to the surface. Despite the sampling operation complexity, it is of primary importance the use of the adequate instrumentation, as well the expertise of the instrument operator. Ideally, the operator should have a good knowledge of the technical characteristics of the instruments themselves, as well as the correct procedures for their operation. In addition, he should be well acquainted to the ship equipment employed in the deployment, operation and retrieval of those instruments, including cables, winches and cranes. Only if these aspects are taken in account a reliable and safe operation can be attained. In this way, this guide was conceived as a basic reference for researchers in their on-board operations, although certainly being useful for a wide range of outdoor activities. It was based on the authors years of experience in field work onboard research and fishing vessels. It includes notions on nautical cables, onboard load handling equipment and hardware, basic ropework, basic net weaving and repairing and a nautical glossary.

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19

Kingsley, Sean A., and Greg Stemm. Oceans Odyssey 3 : The Deep-Sea Tortugas Shipwreck, Straits of Florida: A Merchant Vessel from Spain's 1622 Tierra Firme Fleet. Oxbow Books, Limited, 2013.

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20

Kingsley, Sean A., and Greg Stemm. Oceans Odyssey 3 : The Deep-Sea Tortugas Shipwreck, Straits of Florida: A Merchant Vessel from Spain's 1622 Tierra Firme Fleet. Oxbow Books, Limited, 2013.

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21

Kingsley, Sean A., and Greg Stemm. Oceans Odyssey 3 : The Deep-Sea Tortugas Shipwreck, Straits of Florida: A Merchant Vessel from Spain's 1622 Tierra Firme Fleet. Oxbow Books, Limited, 2013.

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22

Cumulative index to the Initial reports of the Deep Sea Drilling Project: Covering volumes 1 through 96 : reporting the results of Legs 1 through 96 of the cruises of the drilling vessel Glomar Challenger, August 1968-November 1983. [College Station, Tex.]: Ocean Drilling Program, Texas A&M University, 1991.

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23

Sunderkötter, Cord, and Luis Requena. Panniculitides. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0165.

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Panniculitis is an inflammation that originates primarily in the subcutaneous fatty tissue (panniculus adiposus). It is associated with rheumatological diseases and with adverse events to rheumatological therapies (e.g. poststeroid panniculitis, erythema nodosum, infective panniculitis). The panniculitides are classified histopathologically into mostly septal panniculitis and mostly lobular panniculitis, according to the major or denser localization of the infiltrate, and also into those with or without vasculitis. Additional criteria involve the composition of the inflammatory infiltrate, the cause, and an underlying or associated disease. The clinical hallmarks of panniculitis are subcutaneous nodules or plaques, often located on the lower limb. A deep excisional biopsy is often required for a more precise diagnosis, given the often sparse and monotonous clinical symptoms. Erythema nodosum is the most common form and a typical example of septal panniculitis. It occurs in response to many different provoking factors, the most common trigger in children being a 'strep throat', in adults sarcoidosis. Clinically, it presents with a sudden symmetrical appearance of painful, tender, warm, erythematous nodes or plaques, usually on the shins, which resemble bruises. Classical and cutaneous polyarteriitis nodosa present a mostly septal panniculitis associated with vasculitis. Here subcutaneous, partially ulcerating nodules are surrounded by livedo racemosa. The mostly lobular panniculitides not associated with vasculitis include lupus panniculitis (lupus erythematosus profundus, typically with ensuing lipoatrophy and predilection for the upper part of the body), panniculitis in dermatomyositis (often calcifiying), cold panniculitis, pancreatic panniculitis, panniculitis due toα‎-antitrypsin deficiency, poststeroid panniculitis (in children after rapid withdrawal of corticosteroids), calciphylaxis (with and without renal failure), and factitious panniculitis (after mechanical, physical, or chemical injuries to the subcutaneous tissue, often self-inflicted). Nodular vasculitis (formerly erythema induratum Bazin) is a lobular panniculitis with vasculitis involving mostly the small blood vessels of the fat lobule. It appears to present a (hyper)reactive response to certain infections (tuberculosis, streptococci, candida) or to cold exposure or chronic venous insufficiency in susceptible females. In conclusion, the panniculitides are a heterogenous group of diseases requiring a systematic work-up and knowledge of certain histological or clinical criteria.

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24

Alchi, Bassam, and David Jayne. The patient with antiphospholipid syndrome with or without lupus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0164.

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Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by recurrent arterial or venous thrombosis and/or pregnancy loss, accompanied by laboratory evidence of antiphospholipid antibodies (aPL), namely anticardiolipin antibodies (aCL), lupus anticoagulant (LA), and antibodies directed against beta-2 glycoprotein 1 (β‎‎‎2GP1). APS may occur as a ‘primary’ form, ‘antiphospholipid syndrome,’ without any known systemic disease or may occur in the context of systemic lupus erythematosus (SLE), ‘SLE-related APS’. APS may affect any organ system and displays a broad spectrum of thrombotic manifestations, ranging from isolated lower extremity deep vein thrombosis to the ‘thrombotic storm’ observed in catastrophic antiphospholipid syndrome. Less frequently, patients present with non-thrombotic manifestations (e.g. thrombocytopaenia, livedo reticularis, pulmonary hypertension, valvular heart disease, chorea, and recurrent fetal loss).The kidney is a major target organ in both primary and SLE-related APS. Renal involvement is typically caused by thrombosis occurring at any location within the renal vasculature, leading to diverse effects, depending on the size, type, and site of vessel involved. The renal manifestations of APS include renal artery stenosis and/or renovascular hypertension, renal infarction, APS nephropathy (APSN), renal vein thrombosis, allograft vasculopathy and vascular thrombosis, and thrombosis of dialysis access.Typical vascular lesions of APSN may be acute, the so-called thrombotic microangiopathy, and/or chronic, such as arteriosclerosis, fibrous intimal hyperplasia, tubular thyroidization, and focal cortical atrophy. The spectrum of renal lesions includes non-thrombotic conditions, such as glomerulonephritis. Furthermore, renal manifestations of APS may coexist with other pathologies, especially proliferative lupus nephritis.Early diagnosis of APS requires a high degree of clinical suspicion. The diagnosis requires one clinical (vascular thrombosis or pregnancy morbidity) and at least one laboratory (LA, aCL, and/or anti-β‎‎‎2GP1) criterion, positive on repeated testing.The aetiology of APS is not known. Although aPL are diagnostic of, and pathogenic in, APS, a ‘second hit’ (usually an inflammatory event) may trigger thrombosis in APS. The pathogenesis of the thrombotic tendency in APS remains to be elucidated, but may involve a combination of autoantibody-mediated dysregulation of coagulation, platelet activation, and endothelial injury.Treatment of APS remains centred on anticoagulation; however, it has also included the use of corticosteroids and other immunosuppressive therapy. The prognosis of patients with primary APS is variable and unpredictable. The presence of APS increases morbidity (renal and cerebral) and mortality of SLE patients.

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