Books on the topic 'Death – Causes – Econometric models'

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1

Bhattacharya, Jay. Cause-specific mortality among medicare enrollees. Cambridge, Mass: National Bureau of Economic Research, 1996.

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2

Bhattacharya, Jay. Cause-specific mortality among medicare enrollees. Cambridge, MA: National Bureau of Economic Research, 1996.

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3

Robin, Sickles, and Taubman Paul 1939-, eds. Causes, correlates and consequences of death among older adults: Some methodological approaches and substantive analyses. Boston, Mass: Kluwer Academic Publishers, 1998.

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4

Breedon, F. J. M0: Causes and consequences. London: Bank of England, 1993.

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5

Engel, Charles. Deviations from purchasing power parity: Causes and welfare costs. Washington, D.C: Federal Reserve Board, 2000.

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6

R, Link Charles, and Staten Michael E, eds. Causes of litigation in workers' compensation. Kalamazoo, Mich: W.E. Upjohn Institute for Employment Research, 1995.

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7

Globalization of international financial markets: Causes and consequences. Aldershot, Hants, England: Ashgate, 1999.

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8

Temin, Peter. The causes of American business cycles: An essay in economic historiography. Cambridge, MA: National Bureau of Economic Research, 1998.

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9

Tremblay, Pierre. Crime and destiny: Hazard models for offenders. Montréal: Université de Montréal, 2000.

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10

Abel, Andrew B. Birth, death and taxes. Cambridge, MA: National Bureau of Economic Research, 1989.

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11

Donohue, John J. Uses and abuses of empirical evidence in the death penalty debate. Cambridge, Mass: National Bureau of Economic Research, 2006.

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12

Ellison, Glenn. What causes industry agglomeration?: Evidence from coagglomeration patterns. Cambridge, Mass: National Bureau of Economic Research, 2007.

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13

Dabla-Norris, Era. The distributional bias of public education: Causes and consequences. [Washington, D.C]: International Monetary Fund, IMF Institute, 2004.

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14

Buchmueller, Thomas C. Did community rating induce an adverse selection death spiral?: Evidence from New York, Pennsylvania, and Connecticut. Cambridge, MA: National Bureau of Economic Research, 1999.

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15

Statistics Canada. Analytical Studies Branch., ed. Death in the industrial world: Plant closures and capital retirement. Ottawa: Statistics Canada, 2005.

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16

Comin, Diego. The rise in firm-level volatility: Causes and consequences. Cambridge, Mass: National Bureau of Economic Research, 2005.

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17

Comin, Diego. The rise in firm-level volatility: Causes and consequences. Cambridge, MA: National Bureau of Economic Research, 2005.

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18

Blank, Rebecca M. What causes public assistance caseloads to grow? Cambridge, MA: National Bureau of Economic Research, 1997.

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19

Jaffee, Dwight M. The causes and consequences of rate regulation in the auto insurance industry. Cambridge, MA: National Bureau of Economic Research, 1995.

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20

Anyanwu, J. C. Economic and political causes of civil wars in Africa: Some econometric results. Abidjan, Côte d'Ivoire: African Development Bank, 2002.

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21

Dabla-Norris, Era. What causes firms to hide output?: The determinants of informality. Washington, D.C: International Monetary Fund, IMF Institute, 2005.

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22

Carroll, Chris. Death to the log-linearized consumption Euler equation!: (and very poor health to the second-order approximation). Cambridge, MA: National Bureau of Economic Research, 1997.

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23

Wagstaff, Adam. On decomposing the causes of health sector inequalities with an application to malnutrition in Vietnam. Washington, D.C: World Bank, Development Research Group, Public Services for Human Development, and, Development Data Group, 2001.

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24

Markowitz, Sara. The effectiveness of cigarette regulations in reducing cases of sudden infant death syndrome. Cambridge, MA: National Bureau of Economic Research, 2006.

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25

Markowitz, Sara. The effectiveness of cigarette regulations in reducing cases of sudden infant death syndrome. Cambridge, Mass: National Bureau of Economic Research, 2006.

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26

Campbell, Thomas. The causes and effects of liability reform: Some empirical evidence. Cambridge, MA: National Bureau of Economic Research, 1995.

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27

Economic Policy Conference of the Federal Reserve Bank of St. Louis (12th 1987 Federal Reserve Bank of St. Louis). U.S. trade deficit: Causes, consequences, and cures : the proceedings of the twelth Annual Economic Policy Conference of the Federal Reserve Bank of St. Louis. Boston: Kluwer Academic Publishers, 1989.

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28

Behrman, Jere R., Paul Taubman, and Robin C. Sickles. Causes, Correlates and Consequences of Death among Older Adults: Some Methodological Approaches and Substantive Analyses. Springer London, Limited, 2012.

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29

Behrman, Jere R., Paul Taubman, and Robin C. Sickles. Causes, Correlates and Consequences of Death Among Older Adults: Some Methodological Approaches and Substantive Analyses. Springer, 2012.

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30

Inflation in the SEACEN countries: Its causes and management. Kuala Lumpur, Malaysia: South East Asian Central Banks, Research and Training Centre, 1996.

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31

Elizabeth, Godfrey A., and National Center for Health Statistics (U.S.), eds. Mortality surveillance system: Models from the second year. Hyattsville, Md. (6525 Belcrest Rd., Hyattsville 20782): U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Center for Health Statistics, 1994.

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32

M, Chevarley Frances, and National Center for Health Statistics (U.S.), eds. Mortality surveillance system: Models from the first year. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Center for Health Statistics, 1992.

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33

Elizabeth, Godfrey A., and National Center for Health Statistics (U.S.), eds. Mortality surveillance system: Models from the second year. Hyattsville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Center for Health Statistics, 1994.

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34

Mortality surveillance system: Models from the first year (Vital and health statistics). For sale by the U.S. G.P.O., Supt. of Docs, 1993.

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35

Rau, Roland. Seasonality in Human Mortality: A Demographic Approach. Springer London, Limited, 2006.

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36

Rau, Roland. Seasonality in Human Mortality: A Demographic Approach (Demographic Research Monographs). Springer, 2006.

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37

Rau, Roland. Seasonality in Human Mortality: A Demographic Approach. Springer Berlin / Heidelberg, 2010.

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38

Michel, Jean-Baptiste. Biology of vascular wall dilation and rupture. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0016.

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Arterial pathologies, important causes of death and morbidity in humans, are closely related to modifications in the circulatory system during evolution. With increasing intraluminal pressure and arterial bifurcation density, the arterial wall becomes the target of interactions with blood components and outward convection of plasma solutes and particles, including plasma zymogens and leukocyte proteases. Abdominal aortic aneurysms of atherothrombotic origin are characterized by the presence of an intraluminal thrombus (ILT), a major source of proteases, including plasmin, MMP-9, and elastase. Saccular cerebral aneurysms are characterized by the interaction of haemodynamics and arterial bifurcation defects, of either genetic or congenital origin. They also develop an intrasaccular thrombus, implicated in rupture. Aneurysms of the ascending aorta (TAAs) are not linked to atherothrombotic disease, and do not develop an ILT. The most common denominator of TAAs, whatever their aetiology, is the presence of areas of mucoid degeneration, and increased convection and vSMC-dependent activation of plasma zymogens within the wall, causing extracellular matrix proteolysis. TAA development is also associated with an epigenetic phenomenon of SMAD2 overexpression and nuclear translocation, potentially linked to chronic changes in mechanotransduction. Aortic dissections share common aetiologies and pathology (areas of mucoid degeneration) with TAAs, but differ by the absence of any compensatory epigenetic response. There are main experimental animal models of aneurysms, all characterized by the cessation of aneurysmal progression after interruption of the exogenous stimuli used to induce it. These new pathophysiological approaches to aneurysms in humans pave the way for new diagnostic and therapeutic tools.
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