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Journal articles on the topic 'Deafness'

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Author: Grafiati

Published: 4 June 2021

Last updated: 7 September 2024

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1

Szkutnik, Katarzyna, and Jurek Olszewski. "Analysis of the causes of the occurrence and treatment results of sudden hearing loss/deafness in the own material." Otolaryngologia Polska 76, no. 6 (December 14, 2022): 1–5. http://dx.doi.org/10.5604/01.3001.0016.0050.

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Introduction. The aim of the study was to analyze the causes of the occurrence and treatment results of sudden hearing loss / deafness in own material.Material and methods. The tests were performed in 64 women aged 10-89 years and 70 men aged 18-85 years, who were treated in the Department of Otolaryngology due to sudden hearing loss / deafness in 2011-2020. A retrospective analysis was carried out on the basis of the data contained in the medical documentation, taking into account: gender, age of patients, type of symptoms, establishing the circumstances and possible causes of the appearance of symptoms, audiological and imaging tests, the cause of the sudden hearing loss / deafness and applied treatment.Results. In the conducted research it was found that sudden hearing loss / deafness was the most common in women and men in the age range of 51-60 years and 61-70 years. Sudden hearing loss/ deafness was accompanied by the following symptoms in both women and men: tinnitus in 48,4% and 74,3%, tinnitus and vertigo in 28,1% and 14,3%, and vertigo in 6,3% and in 1,4%. In own research, the most common cause of sudden hearing loss/deafness in women and men was respectively: idiopathic cause in 43,8% and 40,0%, infectious in 18,8%, vascular in 12,4% and 12,9%, and neurovascular conflict (included separately) in 12,4% and 12,9%, while in men the acoustic trauma should also be mentioned in 18,5%. The best results in the treatment of sudden hearing loss / deafness on a three-point scale, which depends on the treatment method used, both in women and men, i.e. complete relief of symptoms obtained after the use of vascular drugs with steroid therapy and hyperbaric oxygen therapy - in 25,0% and 22,9%, partial relief of symptoms occurred after vascular treatment with steroid therapy - in 28,1% and 37,2%. Conclusions. Sudden hearing loss / deafness in both women and men was accompanied by tinnitus, tinnitus and vertigo and vertigo at the same time. In own research the most common cause of sudden hearing loss / deafness in both women and men was: idiopathic, infectious, vascular and neurovascular conflict. The best emergency treatment outcomes of sudden hearing loss / deafness according to a three-level scale, depending on the applied method of treatment, both in women and men, i.e. complete relief of symptoms was obtained after the use of vascular drugs with steroid therapy and hyperbaric oxygen therapyKey words: causes, occurrence, treatment results, sudden hearing loss / deafnes

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2

O'brien, Victoria. "Deafness." InnovAiT: Education and inspiration for general practice 6, no. 5 (May 2013): 283–90. http://dx.doi.org/10.1177/1755738012470442.

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Hearing loss is common. There are more than 10 million people in the UK with some form of hearing loss, making up one-in-six of the population. Of these, 3.7 million are of working age (16–64 years) and 6.3 million are of retirement age (65 years or greater). Therefore, it should be no surprise that the average GP will see at least four patients with hearing loss every day. However, many GPs still feel under-equipped to manage patients with hearing loss and many patients with hearing loss report receiving a reduced quality of consultation as a result.

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3

O’Brien, Vicky, and Ted Leverton. "Deafness." InnovAiT: Education and inspiration for general practice 11, no. 1 (January 2018): 6–13. http://dx.doi.org/10.1177/1755738017732364.

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Hearing loss is common. Over 10 000 000 people in the UK have a level of loss in their better ear of 25 dB or more; one-in-six of the population. Of these, 3 700 000 are of working age (16–64 years) and 6 300 000 million are 65 years or older. Although the average GP will see at least four patients with some form of hearing loss every day, many GPs feel under-equipped to manage patients with hearing loss, and many patients with hearing loss report a reduced quality of consultation.

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4

Levine, James A. "Deafness." Lancet 374, no. 9707 (December 2009): 2126–27. http://dx.doi.org/10.1016/s0140-6736(09)61995-4.

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5

TAGO, CHIAKI. "Mumps deafness--a comparison with sudden deafness." AUDIOLOGY JAPAN 32, no. 5 (1989): 557–58. http://dx.doi.org/10.4295/audiology.32.557.

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6

Zusho, Hiroyuki. "Perceptive Deafness and Presbycusis. Noise-Induced Deafness." AUDIOLOGY JAPAN 39, no. 2 (1996): 101–8. http://dx.doi.org/10.4295/audiology.39.101.

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7

Huang, Mengtian. "A Review of Genetic Diagnosis and Screening of Hereditary Deafness." Academic Journal of Science and Technology 2, no. 3 (September 8, 2022): 100–103. http://dx.doi.org/10.54097/ajst.v2i3.1534.

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The incidence of congenital hearing impairment in China is inching ever upwards. Deafness has always been a disabling disease that seriously affects the quality of human life. Molecular diagnostic technology is the main method to detect hereditary deafness. At present, there is no effective treatment for hereditary deafness, so the screening, early intervention and genetic diagnosis of hereditary deafness are particularly important. There are already genetic screening and genetic diagnosis methods for hereditary deafness. Through the analysis of the effectiveness of genetic screening and diagnosis of hereditary deafness, we can find an effective method to prevent hereditary deafness. The purpose of this review article is to explore effective methods to prevent hereditary deafness by analyzing the effectiveness of genetic screening and genetic diagnosis.

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8

Chen, Penghui, Longxia He, Xiuhong Pang, Xiaowen Wang, Tao Yang, and Hao Wu. "NLRP3 Is Expressed in the Spiral Ganglion Neurons and Associated with Both Syndromic and Nonsyndromic Sensorineural Deafness." Neural Plasticity 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/3018132.

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Nonsyndromic deafness is genetically heterogeneous but phenotypically similar among many cases. Though a variety of targeted next-generation sequencing (NGS) panels has been recently developed to facilitate genetic screening of nonsyndromic deafness, some syndromic deafness genes outside the panels may lead to clinical phenotypes similar to nonsyndromic deafness. In this study, we performed comprehensive genetic screening in a dominant family in which the proband was initially diagnosed with nonsyndromic deafness. No pathogenic mutation was identified by targeted NGS in 72 nonsyndromic and another 72 syndromic deafness genes. Whole exome sequencing, however, identified a p.E313K mutation in NLRP3, a gene reported to cause syndromic deafness Muckle-Wells Syndrome (MWS) but not included in any targeted NGS panels for deafness in previous reports. Follow-up clinical evaluation revealed only minor inflammatory symptoms in addition to deafness in six of the nine affected members, while the rest, three affected members, including the proband had no obvious MWS-related inflammatory symptoms. Immunostaining of the mouse cochlea showed a strong expression of NLRP3 in the spiral ganglion neurons. Our results suggested that NLRP3 may have specific function in the spiral ganglion neurons and can be associated with both syndromic and nonsyndromic sensorineural deafness.

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9

Siméon, Ludovic. "Dépistage et diagnostic de la surdité chez le chien." Bulletin de l'Académie Vétérinaire de France 176, no. 1 (2023): 21–28. http://dx.doi.org/10.3406/bavf.2023.18244.

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Screening and diagnosis of deafness in dogs is based on Brainstem Auditory Evoked Response or BAER recording. Deafness is usually classified according to five criteria : determinism, onset, severity, symmetry, and anatomical-functional stage reached. In breeds at risk for hereditary deafness, with white or piebald robes (Dalmatian Dogo argentino, Bull terrier, Australian cattle dog, etc.) or merle (Border collie, Australian shepherd, etc.), screening of breeding animals and newborns should be encouraged to reduce the prevalence of deafness in these breeds. In dogs with acute deafness, hearing impairment should be confirmed by BAERs recording and lesions should be in-vestigated by medical imaging (CT-scan and/or magnetic resonance imaging). Acquired causes of deafness are nu-merous : inflammatory (especially otitis media), toxic (or ototoxicity), traumatic, degenerative (or presbycusis). In acute deafness in dogs, total bilateral deafness is most often irreversible.

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10

Kalef M, Rahi, Raid Yaqoub Yousef, and Ali Abd-almer jwad. "Auditory brainstem evoked response in deaf children." AL-QADISIYAH MEDICAL JOURNAL 11, no. 19 (July 24, 2017): 102–6. http://dx.doi.org/10.28922/qmj.2015.11.19.102-106.

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This study is designed to determine the degree , type of deafness, and site of lesion in the auditory pathway of deaf children by using Brainstem evoked response audiometry.This study is prospective in nature. It considerd 56 patients with deafness which were assessed between July and December 2010 in E.N.T outpatient clinic, department of otolaryngology, in Al-Diwaniya teaching hospital, AL-diwaniya city, Iraq.The study shows that The degrees of deafness are: out of 56 patients, 12 patients (N=12, 21.4%) have mild deafness (20db- 40db loss), 16 patients (N-16, 28.6%) have moderate defness (40db- 60db loss), 28 patients (N=28, 50%) have sever or profound deafness (60db- 100db loss). The type and site of deafness are: out of 56 patients, 11 patients (N=11, 20% have conductive deafness, 26 patients (N=26, 46.4%) have cochlear (sensory) deafness, 17 patients (N=17, 30%) have retrocochlear (neural) deafness, 2 patients (N=2, 3.6%) have cortical deafness.the brainstem evoked response audiometry valuable audiological test to assess the auditory pathway in children.

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11

Inoue, Yasuhiro. "Mumps deafness." AUDIOLOGY JAPAN 51, no. 6 (2008): 617–23. http://dx.doi.org/10.4295/audiology.51.617.

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12

Asakuma, Shinichiro, and Kazuo Murai. "Sudden deafness." AUDIOLOGY JAPAN 53, no. 1 (2010): 46–53. http://dx.doi.org/10.4295/audiology.53.46.

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13

Yanagita, Noriyuki. "Sudden deafness." Practica Oto-Rhino-Laryngologica 78, no. 3 (1985): 299–311. http://dx.doi.org/10.5631/jibirin.78.299.

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14

Ham, Tae Young. "Congenital Deafness." Journal of Clinical Otolaryngology Head and Neck Surgery 1, no. 2 (December 1990): 34–38. http://dx.doi.org/10.35420/jcohns.1990.1.2.34.

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15

Jones, Cynthia M., and Shawn P. Saladin. "Fixing Deafness." Journal of Philosophy, Science & Law 14, no. 3 (2014): 16–32. http://dx.doi.org/10.5840/jpsl20141438.

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16

Brook, Ann. "Aiding deafness." Nursing Standard 2, no. 6 (November 7, 1987): 37. http://dx.doi.org/10.7748/ns.2.6.37.s84.

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17

Sutton-Spence, Rachel. "Constructing Deafness." Disability, Handicap & Society 7, no. 3 (January 1992): 290–91. http://dx.doi.org/10.1080/02674649266780321.

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18

McCabe, Brian F. "Beethoven's Deafness." Annals of Otology, Rhinology & Laryngology 113, no. 7 (July 2004): 511–25. http://dx.doi.org/10.1177/000348940411300702.

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19

Crunkhorn, Sarah. "Reversing deafness." Nature Reviews Drug Discovery 14, no. 9 (September 2015): 602. http://dx.doi.org/10.1038/nrd4723.

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20

Reardon, W. "Genetic deafness." Journal of Medical Genetics 29, no. 8 (August 1, 1992): 521–26. http://dx.doi.org/10.1136/jmg.29.8.521.

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21

Mort, Dominic J., and Adolfo M. Bronstein. "Sudden deafness." Current Opinion in Neurology 19, no. 1 (February 2006): 1–3. http://dx.doi.org/10.1097/01.wco.0000196155.92856.a1.

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22

Brody, Robert M., Brian D. Nicholas, Michael J. Wolf, Paula B. Marcinkevich, and Gregory J. Artz. "Cortical Deafness." Otology & Neurotology 34, no. 7 (September 2013): 1226–29. http://dx.doi.org/10.1097/mao.0b013e31829763c4.

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23

Fischel-Ghodsian, Nathan. "Mitochondrial Deafness." Ear and Hearing 24, no. 4 (August 2003): 303–13. http://dx.doi.org/10.1097/01.aud.0000079802.82344.b5.

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24

Wolf, Michael, Jossef Ben Shoshan, Moshe Birger, and Jona Kronenberg. "Conversion Deafness." Annals of Otology, Rhinology & Laryngology 102, no. 5 (May 1993): 349–52. http://dx.doi.org/10.1177/000348949310200505.

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Conversion deafness is very rarely encountered among adults. This report will illustrate two cases of this somatoform disorder following different traumatic experiences. It emphasizes the difficulties in establishing the diagnosis and reviews various aspects of treatment.

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25

Kanzaki, Jin. "Sudden Deafness." Oto-Rhino-Laryngologia Nova 9, no. 5 (1999): 198–202. http://dx.doi.org/10.1159/000027908.

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26

URBAN, ELIZABETH. "Childhood Deafness:." Journal of Analytical Psychology 34, no. 2 (April 1989): 143–57. http://dx.doi.org/10.1111/j.1465-5922.1989.00143.x.

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27

Mills, R. P. "Ballantyne's Deafness." Journal of the Royal Society of Medicine 95, no. 6 (June 2002): 321. http://dx.doi.org/10.1177/014107680209500621.

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28

Ryugo, David K., and Marilyn Menotti-Raymond. "Feline Deafness." Veterinary Clinics of North America: Small Animal Practice 42, no. 6 (November 2012): 1179–207. http://dx.doi.org/10.1016/j.cvsm.2012.08.008.

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29

Strain, George M. "Canine Deafness." Veterinary Clinics of North America: Small Animal Practice 42, no. 6 (November 2012): 1209–24. http://dx.doi.org/10.1016/j.cvsm.2012.08.010.

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30

Jacobs, Howard T. "Mitochondrial Deafness." Annals of Medicine 29, no. 6 (January 1997): 483–91. http://dx.doi.org/10.3109/07853899709007472.

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31

Kokotas, H., MB Petersen, and PJ Willems. "Mitochondrial deafness." Clinical Genetics 71, no. 5 (May 2, 2007): 379–91. http://dx.doi.org/10.1111/j.1399-0004.2007.00800.x.

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32

Douek, E. "Sensorineural deafness." BMJ 301, no. 6743 (July 14, 1990): 74–75. http://dx.doi.org/10.1136/bmj.301.6743.74.

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33

Fischel-Ghodsian, N. "Mitochondrial Deafness." Genetics in Medicine 2, no. 1 (January 2000): 51. http://dx.doi.org/10.1097/00125817-200001000-00027.

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34

Smith, P. E. M., C. N. Chitty, G. Williams, and D. Stephens. "Goya's deafness." Practical Neurology 8, no. 6 (December 1, 2008): 370–77. http://dx.doi.org/10.1136/jnnp.2008.161349.

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35

Kimberling, William J. "Hereditary deafness." American Journal of Medical Genetics 89, no. 3 (September 24, 1999): 121–22. http://dx.doi.org/10.1002/(sici)1096-8628(19990924)89:3<121::aid-ajmg1>3.0.co;2-u.

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36

ORIHARA, HIROMI. "Sensorineural deafness.4.Two cases of mumps deafness." AUDIOLOGY JAPAN 38, no. 5 (1995): 359–60. http://dx.doi.org/10.4295/audiology.38.359.

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37

HIBINO, HIROSHI. "Sensorineural deafness. VIII. A case of cortical deafness." AUDIOLOGY JAPAN 38, no. 5 (1995): 607–8. http://dx.doi.org/10.4295/audiology.38.607.

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38

SUGIO, YUICHIRO. "Functional deafness. Cases of exaggerated deafness in compensation." AUDIOLOGY JAPAN 41, no. 5 (1998): 387–88. http://dx.doi.org/10.4295/audiology.41.387.

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39

SATO, HITOSHI. "On infant functional deafness without consciousness of deafness." AUDIOLOGY JAPAN 41, no. 5 (1998): 397–98. http://dx.doi.org/10.4295/audiology.41.397.

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40

Fu, S., G. Chen, J. Dong, and L. Zhang. "<p>Prevalence and etiology of hearing loss in primary and middle school students in the Hubei Province of China</p>." Community Ear and Hearing Health 8, no. 11 (December 1, 2011): 5. http://dx.doi.org/10.56920/cehh.107.

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The purpose of this study was to investigate the prevalence and etiology of hearing loss in primary and middle school students in the Hubei province of China. During a 2-year period, 504,348 students were examined by a speech audiometry test, and 813 deaf students were detected. Among the 813 deaf students, 232 cases were diagnosed with congenital deafness and 560 cases had acquired deafness, among which 276 cases had aminoglycoside-antibiotic-induced deafness. The severity of deafness could be further ascertained in 804 other students, with 402 profound, 363 severe, 21 moderate and 18 mild deafness cases. The age at deafness onset of most students was under 3 years. The prevalence of congenital and acquired deafness was 0.046% (232/504,348) and 0.111% (560/504,348), respectively, much lower than previously reported in other regions of China. Furthermore, the genetic factor was identified as one of the principal causes of deafness by pedigree analysis. Published courtesy of: Audiol Neurootol. 2010; 15(6): 394-398

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41

Tona, Risa, Ivan A. Lopez, Cristina Fenollar-Ferrer, Rabia Faridi, Claudio Anselmi, Asma A. Khan, Mohsin Shahzad, et al. "Mouse Models of Human Pathogenic Variants of TBC1D24 Associated with Non-Syndromic Deafness DFNB86 and DFNA65 and Syndromes Involving Deafness." Genes 11, no. 10 (September 24, 2020): 1122. http://dx.doi.org/10.3390/genes11101122.

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Human pathogenic variants of TBC1D24 are associated with clinically heterogeneous phenotypes, including recessive nonsyndromic deafness DFNB86, dominant nonsyndromic deafness DFNA65, seizure accompanied by deafness, a variety of isolated seizure phenotypes and DOORS syndrome, characterized by deafness, onychodystrophy, osteodystrophy, intellectual disability and seizures. Thirty-five pathogenic variants of human TBC1D24 associated with deafness have been reported. However, functions of TBC1D24 in the inner ear and the pathophysiology of TBC1D24-related deafness are unknown. In this study, a novel splice-site variant of TBC1D24 c.965 + 1G > A in compound heterozygosity with c.641G > A p.(Arg214His) was found to be segregating in a Pakistani family. Affected individuals exhibited, either a deafness-seizure syndrome or nonsyndromic deafness. In human temporal bones, TBC1D24 immunolocalized in hair cells and spiral ganglion neurons, whereas in mouse cochlea, Tbc1d24 expression was detected only in spiral ganglion neurons. We engineered mouse models of DFNB86 p.(Asp70Tyr) and DFNA65 p.(Ser178Leu) nonsyndromic deafness and syndromic forms of deafness p.(His336Glnfs*12) that have the same pathogenic variants that were reported for human TBC1D24. Unexpectedly, no auditory dysfunction was detected in Tbc1d24 mutant mice, although homozygosity for some of the variants caused seizures or lethality. We provide some insightful supporting data to explain the phenotypic differences resulting from equivalent pathogenic variants of mouse Tbc1d24 and human TBC1D24.

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42

Takasaki, Tomohiko, Masahiko Higashikawa, Soichi Motoyama, Kunihiro Sugita, and Ichiro Kurane. "Serum antibodies to human herpesvirus 7, human herpesvirus 6 and cytomegalovirus in patients with idiopathic facial nerve palsy or sudden deafness." Journal of Laryngology & Otology 112, no. 7 (July 1998): 617–21. http://dx.doi.org/10.1017/s0022215100141271.

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AbstractThe aetiology of idiopathic facial nerve palsy (Bell's palsy) and sudden deafness are not known, although viruses have been suspected as a cause of them. We investigated the relationship between Bell's palsy or sudden deafness, and reactivation of cytomegalovirus, human herpesvirus 6 (HHV-6) and 7 (HHV-7).Paired sera were collected from 22 patients with Bell's palsy and 24 patients with sudden deafness. IgG antibody titres to HHV-7 were increased in one patient with Bell's palsy. IgG antibody titres to HHV-6 were increased in one patient with Bell's palsy and two with sudden deafness. IgG antibody titres to cytomegalovirus were increased in one patient with sudden deafness. Titres of the three viral antibodies were not increased simultaneously in any patients. These viruses may contribute to the development of Bell's palsy or sudden deafness in some cases. It is, however, unlikely that these viruses are the main cause of Bell's palsy and sudden deafness in the majority of patients.

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43

Nayoan, CR, IPFI White, Rahma Rahma, H. Harun, and Nur Syamsi. "An Overview: The Level of Knowledge about Congenital Deafness in Southern Tatura District Palu City Indonesia." EAS Journal of Medicine and Surgery 6, no. 04 (April 30, 2024): 145–49. http://dx.doi.org/10.36349/easjms.2024.v06i04.006.

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Background: Congenital deafness is caused by affecting factors that occur during pregnancy and at birth. The prevalence of congenital deafness in the world ranges from 1 to 3 events out of 1000 babies born alive. The level of public knowledge regarding congenital deafness–especially among the parents–is an important factor for management of children with congenital deafness starting from screening to final management. Objective: To describe level of knowledge about congenital deafness in Southern Tatura District. Methods: Descriptive observational study was used. Respondents were from Southern Tatura District, Palu city. The study was conducted in May 2018. Respondents who met the inclusion and exclusion criteria were interviewed using a questionnaire to assess the level of knowledge regarding congenital deafness. Results: Of 98 respondents, most of them were women (69.39%), were in the age group of 18-39 years (79.6%), were self-employed (51.03%) and had graduated from senior high school (53.07%). Among all respondents, we found that only 4.08% had good of knowledge about congenital deafness, 20.4% had moderate, and 7.51% had low knowledge. Conclusion: People living in the South Tatura District, Palu City had low knowledge of congenital deafness.

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44

Idola Pratiwi and I Made Wiranadha. "Characteristics of deafness in the elderly patients at Sanglah general hospital period march 2020-march 2021." GSC Biological and Pharmaceutical Sciences 21, no. 3 (December 30, 2022): 140–44. http://dx.doi.org/10.30574/gscbps.2022.21.3.0476.

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Introduction: Elderly is defined as an individual who has reached the age of 60 years and over. the number of elderly population in Indonesia currently reaches 28.7 million people, 10.6% of the total population of Indonesia. In 2020 Bali has entered the aging population, namely when the percentage aged 60 years and over reaches more than 10%. In line with the increasing life expectancy of people in Indonesia, health problems for the elderly will increase. One of the health problems of the elderly is hearing loss. Pathological changes in the hearing organ due to the degeneration process in the elderly can cause deafness. Objective: To determine the characteristics of deafness in elderly patients at Sanglah General Hospital during the period March 2020-March 2021. Methods: This study used a retrospective descriptive research design with cross sectional design, by taking secondary data from medical records of elderly patients with deafness who came to the ENT’s polyclinic at Sanglah General Hospital during the period March 2020-March 2021. Result: Characteristics deafness in elderly patients based on gender, the most found in male group compare to female group (53,3%: 46,5%), the largest age group was in the 60-70 years group as many as 33 people or 56.9%. Based on audiometric examination 39 elderly patients with deafness or 67.2% with sensorineural deafness. Characteristics based on the degree of deafness in elderly patients most found intensity in range between 56-70 dB, 20 people or 34.5% who experienced moderate deafness. The most common based on the side of the ear 53 people with deafness (91.4%) on both sides of the ear or bilateral. Conclusion: This study found that deafness in elderly patients is more commonly found in male, at age groups 60-70 years old with the most type of sensorineural deafness type, the most degree of deafness is moderate to severe degree, and deaf complaints on the both sides.

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45

Oliveira, Catarina, Marta Machado, Raquel Zenha, Luísa Azevedo, Luísa Monteiro, and Adelaide Bicho. "Surdez Congénita ou Precocemente Adquirida: Do Rastreio ao Seguimento, um Retrato de Portugal." Acta Médica Portuguesa 32, no. 12 (December 2, 2019): 767. http://dx.doi.org/10.20344/amp.11880.

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Introduction: Congenital deafness or early acquired deafness affects 1 to 3 out of 1000 newborns without risk factors and 20 to 40 out of 1000 newborns with risk factors. The universal newborn hearing screening enables its early identification. Children with congenital deafness/early acquired deafness have a higher prevalence of other conditions, especially ophthalmologic and neurodevelopmental ones, and at least 30% to 40% have at least one associated comorbidity.Material and Methods: We carried out a cross-sectional, multicenter study in which 83% (n = 30) of the hospitals/maternity hospitals of the National Health Service participated.Results: All surveyed hospitals/maternity hospitals routinely performed universal newborn hearing screening to all newborns before discharge; 63% referred children with risk factors for hearing loss to Otorhinolaryngology. All children with congenital deafness/early acquired deafness are referred to: Pediatrics in 23% hospitals/maternity hospitals. In 23 hospitals/maternity hospitals, all children with congenital deafness/early acquired deafness are referred to: Speech Therapy in 44% hospitals/ maternity hospitals; Ophthalmology in 17% hospitals/maternity hospitals; National System of Early Intervention in Childhood in 30% hospitals/maternity hospitals; 22% of hospitals/maternity hospitals refer all children with congenital deafness/early acquired deafness, with no identified cause, to Clinical Genetics clinics. The number of diagnoses of deafness in the years 2014 and 2015 was 2.5 and 1.5 per 1000 newborns, respectively, in 15 hospitals/maternity hospitals.Discussion: Awareness of universal newborn hearing screening seems to be widely spread in the National Health Service. The number of children with SC / SPA, as well as the percentage of different types of deafness diagnosed, were identical to those found in other studies and shows its importance. The assessment / follow-up of these children by specialties other than the otolaryngology was heterogeneous in different health entities and revealed that not all children with risk factors for deafness follow up advised by existing standards.Conclusion: Results show that Portugal made an important path in the screening and follow-up of children with SC / SPA. It is important, with the ultimate aim of continually improving the care of these children, to reflect on the involvement of specialties other than otolaryngology, such as the National Early Childhood Intervention System in the follow-up of these children.

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HIRAYAMA, MASATOSHI. "Sensorineural deafness.3.Examination of idiopathic bilateral sensorineural deafness." AUDIOLOGY JAPAN 38, no. 5 (1995): 347–48. http://dx.doi.org/10.4295/audiology.38.347.

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KOIDE, KAZUO. "Aging and deafness. Long term observation of sensorineural deafness." AUDIOLOGY JAPAN 38, no. 5 (1995): 385–86. http://dx.doi.org/10.4295/audiology.38.385.

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Kuzovkov, V. E., I. I. Chernushevich, S. B. Sugarova, A. S. Lilenko, D. D. Kalyapin, and D. S. Luppov. "Algorithm of diagnostics and surgery stage of cochlear implantation in patients with various congenital deafness etiology." Russian Otorhinolaryngology 21, no. 2 (2022): 45–50. http://dx.doi.org/10.18692/1810-4800-2022-2-45-50.

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Over the past few decades, in the world scientific literature devoted to the problems of auditory-speech rehabilitation, there has been an increased interest in studying the etiological features of congenital deafness. A deep understanding of the etiology and mechanisms of deafness development helped optimize the various stages of cochlear implantation and, thereby, increase its effectiveness. Objective. To develop an algorithm for obtaining the earliest possible information about the causative factor of congenital deafness for timely planning of rehabilitation measures, reducing the cost of excessive diagnostic search, and achieving the most effective results in the auditory and speech development of children. Patients and methods. From 2018 to 2021, a study of the etiological spectrum as well as the characteristics of cochlear implantation in patients with various etiologies of congenital deafness was conducted at the Saint Petersburg Research Institute for Ear, Throat, Nose, and Speech. We examined 100 patients under the age of 3 years (mean age 2,15 ± 0,2 years), of which 58 were female, and 42 were male. All patients had congenital bilateral deafness on newborn audiological screening and were candidates for cochlear implantation. Results and discussion. According to the data obtained, 37 patients (16 boys, 21 girls, mean age 2,3 ± 0,3 years) had congenital cytomegalovirus infection as an etiological factor in deafness. The presence of signs of congenital deafness of genetic etiology was detected in 58 patients (24 boys, 34 girls, mean age 1,9 ± 0,2 years): 43 of them with nonsyndromic deafness and 15 patients with syndromic deafness (4 boys, 11 girls, mean age 2,1 ± 0,4 years). In 5 patients, the etiological factor of congenital deafness could not be established even at the present stage of diagnostic search.

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Snapp, Hillary. "Nonsurgical Management of Single-Sided Deafness: Contralateral Routing of Signal." Journal of Neurological Surgery Part B: Skull Base 80, no. 02 (January 17, 2019): 132–38. http://dx.doi.org/10.1055/s-0039-1677687.

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AbstractIn recent years, an increasing research effort has been directed toward remediation of single-sided deafness. Contralateral routing of signal (CROS) is the longest standing rehabilitation solution for individuals with single-sided deafness. The primary goal of CROS technology is to transfer the signal received at the deaf ear to the better hearing ear, thereby reducing the impact of the acoustic head-shadow. This allows for individuals with single-sided deafness to regain access to sounds located at the deaf ear. The hearing deficits associated with single-sided deafness are often debilitating. While surgical management of single-sided deafness is on the rise, CROS hearing aids offer a nonsurgical option to compensate for some of the deficits that occur when a listener is limited to a single ear. Limitations of early CROS devices resulted in poor adoption and acceptance in those with single-sided deafness. Following significant advances in both design and technology, the acceptance of CROS devices has increased in recent years. This paper reviews relevant literature in CROS application for the management of single-sided deafness. Technological advances, benefits, limitations, and clinical considerations are also reviewed in this article.

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Wallis, Jacqueline Mae. "Is it ever morally permissible to select for deafness in one’s child?" Medicine, Health Care and Philosophy 23, no. 1 (September 21, 2019): 3–15. http://dx.doi.org/10.1007/s11019-019-09922-6.

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Abstract As reproductive genetic technologies advance, families have more options to choose what sort of child they want to have. Using preimplantation genetic diagnosis (PGD), for example, allows parents to evaluate several existing embryos before selecting which to implant via in vitro fertilization (IVF). One of the traits PGD can identify is genetic deafness, and hearing embryos are now preferentially selected around the globe using this method. Importantly, some Deaf families desire a deaf child, and PGD–IVF is also an option for them. Selection for genetic deafness, however, encounters widespread disapproval in the hearing community, including mainstream philosophy and bioethics. In this paper I apply Elizabeth Barnes’ value-neutral model of disability as mere-difference to the case of selecting for deafness. I draw on evidence from Deaf Studies and Disability Studies to build an understanding of deafness, the Deaf community, and the circumstances relevant to reproductive choices that may obtain for some Deaf families. Selection for deafness, with deafness understood as mere-difference and valued for its cultural identity, need not necessitate impermissible moral harms. I thus advocate that it is sometimes morally permissible to select for deafness in one’s child.

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