Relevant bibliographies by topics / DC subsets

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'DC subsets'

Author: Grafiati
Published: 14 September 2022

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Journal articles on the topic "DC subsets"

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Alonso, Michael N., Michael T. Wong, Angela L. Zhang, Daniel Winer, Megan M. Suhoski, Lorna L. Tolentino, Juliana Gaitan, et al. "TH1, TH2, and TH17 cells instruct monocytes to differentiate into specialized dendritic cell subsets." Blood 118, no. 12 (September 22, 2011): 3311–20. http://dx.doi.org/10.1182/blood-2011-03-341065.

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Abstract Monocytes and T helper (TH) cells rapidly infiltrate inflamed tissues where monocytes differentiate into inflammatory dendritic cells (DCs) through undefined mechanisms. Our studies indicate that TH cells frequently interact with monocytes in inflamed skin and elicit the differentiation of specialized DC subsets characteristic of these lesions. In psoriasis lesions, TH1 and TH17 cells interact with monocytes and instruct these cells to differentiate into TH1- and TH17-promoting DCs, respectively. Correspondingly, in acute atopic dermatitis, TH2 cells interact with monocytes and elicit the formation of TH2-promoting DCs. DC formation requires GM-CSF and cell contact, whereas TH subset specific cytokines dictate DC function and the expression of DC subset specific surface molecules. Moreover, the phenotypes of T cell–induced DC subsets are maintained after subsequent stimulation with a panel of TLR agonists, suggesting that TH-derived signals outweigh downstream TLR signals in their influence on DC function. These findings indicate that TH cells govern the formation and function of specialized DC subsets.
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Ito, Tomoki, Ryuichi Amakawa, Tsuneyasu Kaisho, Hiroaki Hemmi, Kenichirou Tajima, Kazutaka Uehira, Yoshio Ozaki, Hideyuki Tomizawa, Shizuo Akira, and Shirou Fukuhara. "Interferon-α and Interleukin-12 Are Induced Differentially by Toll-like Receptor 7 Ligands in Human Blood Dendritic Cell Subsets." Journal of Experimental Medicine 195, no. 11 (May 28, 2002): 1507–12. http://dx.doi.org/10.1084/jem.20020207.

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Dendritic cells (DCs) play a crucial role in the immune responses against infections by sensing microbial invasion through toll-like receptors (TLRs). In humans, two distinct DC subsets, CD11c− plasmacytoid DCs (PDCs) and CD11c+ myeloid DCs (MDCs), have been identified and can respond to different TLR ligands, depending on the differential expression of cognate TLRs. In this study, we have examined the effect of TLR-7 ligands on human DC subsets. Both subsets expressed TLR-7 and could respond to TLR-7 ligands, which enhanced the survival of the subsets and upregulated the surface expression of costimulatory molecules such as CD40, CD80, and CD86. However, the cytokine induction pattern was distinct in that PDCs and MDCs produced interferon (IFN)-α and interleukin (IL)-12, respectively. In response to TLR-7 ligands, the Th1 cell supporting ability of both DC subsets was enhanced, depending on the cytokines the respective subsets produced. This study demonstrates that TLR-7 exerts its biological effect in a DC subset-specific manner.
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Ahmad, Sharon. "DC subsets do it differently." Nature Reviews Immunology 7, no. 2 (February 2007): 89. http://dx.doi.org/10.1038/nri2027.

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Girard, Melanie, Jaclyn C. Law, Maria I. Edilova, and Tania H. Watts. "Type I interferons drive the maturation of human DC3s with a distinct costimulatory profile characterized by high GITRL." Science Immunology 5, no. 53 (November 13, 2020): eabe0347. http://dx.doi.org/10.1126/sciimmunol.abe0347.

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Human mononuclear phagocytes comprise specialized subsets of dendritic cells (DCs) and monocytes, but how these subsets individually regulate expression of the molecular signals involved in T cell costimulation is incompletely understood. Here, we used multiparameter flow cytometry and CITE-sequencing to investigate the cell type–specific responses of human peripheral blood DC and monocyte subsets to type I interferons (IFN-I), focusing on differential regulation of costimulatory molecules. We report that IFN-β drives the maturation of the recently identified human CD1c+ CD5− DC3 subset into cells with higher GITRL and lower CD86 expression compared with other conventional DC subsets. Transcriptomic analysis confirmed that DC3s have an intermediate phenotype between that of CD1c+ CD5+ DC2s and CD14+ monocytes, characterized by high expression of MHCII, Fc receptors, and components of the phagocyte NADPH oxidase. IFN-β induced a shared core response in human DC and monocyte subsets as well as subset-specific responses, including differential expression of costimulatory molecules. Gene regulatory network analysis suggests that upon IFN-β stimulation NFKB1 drives DC3s to acquire a maturation program shared with DC2s. Accordingly, inhibition of NF-κB activation prevented the acquisition of a mature phenotype by DC3s upon IFN-β exposure. Collectively, this study provides insight into the cell type–specific response of human DC and monocyte subsets to IFN-I and highlights the distinct costimulatory potential of DC3s.
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Coates, P. Toby H., Simon M. Barratt-Boyes, Linyou Zhang, Vera S. Donnenberg, Peta J. O'Connell, Alison J. Logar, F. Jason Duncan, et al. "Dendritic cell subsets in blood and lymphoid tissue of rhesus monkeys and their mobilization with Flt3 ligand." Blood 102, no. 7 (October 1, 2003): 2513–21. http://dx.doi.org/10.1182/blood-2002-09-2929.

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Abstract We provide phenotypic and functional evidence of premonocytoid dendritic cells (DCs) and preplasmacytoid DCs in blood and of corresponding DC subsets in secondary lymphoid tissue of rhesus monkeys. Subsets were identified and sorted by 4-color flow cytometry using antihuman monoclonal antibodies cross-reactive with rhesus monkey. To mobilize pre-DC subsets, fms-like tyrosine 3 kinase ligand (Flt3L; 100 μg/kg subcutaneously) was administered for 10 days. Presumptive pre-DC subsets were identified within the lineage- (Lin-) major histocompatibility complex (MHC) class II+ fraction of blood mononuclear cells. Premonocytoid DCs were CD11c+CD123- (interleukin-3Rα- [IL-3Rα-]). Preplasmacytoid DCs were characterized as CD11c-CD123++ Flt3L increased the CD11c+ pre-DC (7-fold) and CD123++ pre-DC subsets (3-fold) in blood. The freshly isolated CD11c+ pre-DC subset induced modest proliferation of naive allogeneic T cells. After overnight culture with granulocyte macro-phage-colony-stimulating factor (GMCSF) and CD40L, both subsets up-regulated surface costimulatory molecules, and CD11c+ pre-DCs became potent allostimulators. Freshly isolated CD123++ pre-DCs showed typical plasmacytoid morphology and, when cultured with IL-3 and CD40L for 72 hours, developed mature DC morphology. Following stimulation with CD40L, CD11c+ pre-DCs secreted increased levels of IL-12p40. Importantly, herpes simplex virus-stimulated CD123++ pre-DCs, but not CD11c+ pre-DCs, secreted interferon-α (IFN-α). Corresponding DC subsets were identified by flow analysis and immunohistochemistry in lymph nodes wherein both populations were increased 2- to 3-fold by Flt3L administration. CD123+ pre-DCs produced IFN-α in response to in vivo viral infection. Thus, rhesus monkeys exhibit 2 distinct DC precursor populations that closely resemble those of humans. Both are mobilized into blood and lymphoid tissue by Flt3L, offering potential for their further characterization and possible therapeutic application. (Blood. 2003;102:2513-2521)
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Klebanoff, Christopher A., Sean P. Spencer, Parizad Torabi-Parizi, John R. Grainger, Rahul Roychoudhuri, Yun Ji, Madhusudhanan Sukumar, et al. "Retinoic acid controls the homeostasis of pre-cDC–derived splenic and intestinal dendritic cells." Journal of Experimental Medicine 210, no. 10 (September 2, 2013): 1961–76. http://dx.doi.org/10.1084/jem.20122508.

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Dendritic cells (DCs) comprise distinct populations with specialized immune-regulatory functions. However, the environmental factors that determine the differentiation of these subsets remain poorly defined. Here, we report that retinoic acid (RA), a vitamin A derivative, controls the homeostasis of pre-DC (precursor of DC)–derived splenic CD11b+CD8α−Esamhigh DCs and the developmentally related CD11b+CD103+ subset within the gut. Whereas mice deprived of RA signaling significantly lost both of these populations, neither pre-DC–derived CD11b−CD8α+ and CD11b−CD103+ nor monocyte-derived CD11b+CD8α−Esamlow or CD11b+CD103− DC populations were deficient. In fate-tracking experiments, transfer of pre-DCs into RA-supplemented hosts resulted in near complete conversion of these cells into the CD11b+CD8α− subset, whereas transfer into vitamin A–deficient (VAD) hosts caused diversion to the CD11b−CD8α+ lineage. As vitamin A is an essential nutrient, we evaluated retinoid levels in mice and humans after radiation-induced mucosal injury and found this conditioning led to an acute VAD state. Consequently, radiation led to a selective loss of both RA-dependent DC subsets and impaired class II–restricted auto and antitumor immunity that could be rescued by supplemental RA. These findings establish a critical role for RA in regulating the homeostasis of pre-DC–derived DC subsets and have implications for the management of patients with immune deficiencies resulting from malnutrition and irradiation.
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Yin, Xiangyun, Shuting Chen, and Stephanie C. Eisenbarth. "Dendritic Cell Regulation of T Helper Cells." Annual Review of Immunology 39, no. 1 (April 26, 2021): 759–90. http://dx.doi.org/10.1146/annurev-immunol-101819-025146.

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As the professional antigen-presenting cells of the immune system, dendritic cells (DCs) sense the microenvironment and shape the ensuing adaptive immune response. DCs can induce both immune activation and immune tolerance according to the peripheral cues. Recent work has established that DCs comprise several phenotypically and functionally heterogeneous subsets that differentially regulate T lymphocyte differentiation. This review summarizes both mouse and human DC subset phenotypes, development, diversification, and function. We focus on advances in our understanding of how different DC subsets regulate distinct CD4+ T helper (Th) cell differentiation outcomes, including Th1, Th2, Th17, T follicular helper, and T regulatory cells. We review DC subset intrinsic properties, local tissue microenvironments, and other immune cells that together determine Th cell differentiation during homeostasis and inflammation.
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Segura, Elodie, Jenny Valladeau-Guilemond, Marie-Hélène Donnadieu, Xavier Sastre-Garau, Vassili Soumelis, and Sebastian Amigorena. "Characterization of resident and migratory dendritic cells in human lymph nodes." Journal of Experimental Medicine 209, no. 4 (March 19, 2012): 653–60. http://dx.doi.org/10.1084/jem.20111457.

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Dendritic cells (DCs) initiate adaptive immune responses in lymph nodes (LNs). In mice, LN DCs can be divided into resident and tissue-derived populations, the latter of which migrate from the peripheral tissues. In humans, different subsets of DCs have been identified in the blood, spleen, and skin, but less is known about populations of resident and migratory tissue-derived DCs in LNs. We have analyzed DCs in human LNs and identified two populations of resident DCs that are present in all LNs analyzed, as well as in the spleen and tonsil, and correspond to the two known blood DC subtypes. We also identify three main populations of skin-derived migratory DCs that are present only in skin-draining LNs and correspond to the DC subsets found in the skin. Resident DCs subsets induce both Th1 and Th2 cytokines in naive allogeneic T lymphocytes, whereas the corresponding blood subsets failed to induce efficient Th2 polarization. LN-resident DCs also cross-present antigen without in vitro activation, whereas blood DCs fail to do so. Among migratory DCs, one subset was poor at both CD4+ and CD8+ T cell activation, whereas the other subsets induced only Th2 polarization. We conclude that in humans, skin-draining LNs host both resident and migratory DC subsets with distinct functional abilities.
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Kerkhoff, Nathalie, Shahram Kordasti, Thomas Seidl, Arjan A. Van de Loosdrecht, and Ghulam J. Mufti. "Expansion of CD141int Dendritic Cells in Myelodysplastic Syndrome (MDS)." Blood 120, no. 21 (November 16, 2012): 3856. http://dx.doi.org/10.1182/blood.v120.21.3856.3856.

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Abstract Abstract 3856 Introduction Immunological responses play an important role in the pathogenesis and progression of myelodysplastic syndromes (MDS). Several studies have confirmed that immune dysregulation in MDS may play a critical role in the initiation and progression of the dysplastic clone. We have already shown the importance of expanded Tregs in high risk MDS and its reverse correlation with the number of Th17 cells in low risk disease. However, the potential role of dendritic cells (DCs) in this immune dysregulation and in the expansion of Tregs in MDS is not fully understood. DCs are professional antigen presenting cells (APCs) and potent stimulators of T cells through (cross-)presentation of antigens via MHC class I and II molecules to CD8+ cytotoxic T lymphocytes (CTL) and CD4+ T helper (Th) cells, respectively. DCs are also important in the development of specific anti-tumor T-cell responses. Some subtypes of DCs also play an important role in the expansion of regulatory T cells (Tregs) and induce immune-suppression and editing. There are 3 main subpopulations of DCs in human: plasmacytoid DCs (CD303+ cells) and two types of myeloid DCs (CD1c+ and CD141+ cells). The aim of this study was to investigate the frequency of different DC subsets in the bone marrow and peripheral blood of MDS patients. Patients and methods Twelve peripheral blood and 26 bone marrow samples from MDS patients and 11 peripheral blood and 4 bone marrow samples from healthy donors (HDs) were studied. Three different DC subsets were investigated by flow-cytometry: plasmacytoid DC (Lineage−, HLA-DR+, CD303+), myeloid DC 1 (Lineage−, HLA-DR+, CD1c+) and myeloid DC 2 (Lineage−, HLA-DR+, CD141hi). Results Decreased frequency of DCs in peripheral blood compared to BM Frequencies of all DC subsets were significantly lower in patients' peripheral blood compared to patients' bone marrow (CD1c+ DC 2.56×102 v 7.02×102 and 0.06% v 0.33%, p=0.001; CD141+ DC 4.18×103 v 6.04×103 and 0.44% v 2.44%, p=0.005; CD303+ DC 0.06×102 v 1.07×102 and 0.01% v 0.13%, p=0.045). DC subsets The percentage of the CD1c+ DC subset was significantly reduced in the peripheral blood of MDS patients compared to HD (1.46×103 v 0.26×103, 0.18% v 0.05%, p=0.001) (figure 1). Interestingly, we have noticed an additional subset of DCs, which was not described before. The definition of this subset was based on intermediate expression of CD141 compared to previously described myeloid DCs. The CD141int subset was the only DC subset that was increased in peripheral blood of MDS patients compared to HD (0.41×102 v 2.30×102, 0.01% v 0.13%, p=0.019). Both CD1c+ and CD141int DC subsets were increased in the bone marrow of MDS patients compared to healthy donors. However, these differences were not statistically significant. Discussion Although the decreased number of circulating DCs is reported in MDS, our data suggest that this decrease is more profound in the peripheral blood compared to BM. We have also shown that this decrease is mainly in CD1c+ myeloid DCs. In this study, we describe two subsets of CD141 DCs, CD141hi and CD141int. The CD141int subpopulation is the only DC subset that is increased in MDS patients compared to HD. Our data suggest that DCs from peripheral blood may migrate to the bone marrow in response to an aberrant BM microenvironment in MDS. Nevertheless, the pattern of decrease in DC numbers is not uniform and CD141int DCs are in fact increased. It is already known that CD141+ DCs, play an important role in the induction of Tregs in an IL-10 dependent manner. We have also reported an increased number of Tregs in high-risk MDS, which correlates, with higher risk of progression toward AML. Our data suggest that CD141+ DCs may play a role in the expansion of Tregs in MDS. However, the exact function of this subset of DCs in MDS and their effect on CD4+ T-cells polarization needs to be investigated in more details. Disclosures: No relevant conflicts of interest to declare.
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Dalod, Marc, Tanya Hamilton, Rachelle Salomon, Thais P. Salazar-Mather, Stanley C. Henry, John D. Hamilton, and Christine A. Biron. "Dendritic Cell Responses to Early Murine Cytomegalovirus Infection." Journal of Experimental Medicine 197, no. 7 (April 7, 2003): 885–98. http://dx.doi.org/10.1084/jem.20021522.

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Differentiation of dendritic cells (DCs) into particular subsets may act to shape innate and adaptive immune responses, but little is known about how this occurs during infections. Plasmacytoid dendritic cells (PDCs) are major producers of interferon (IFN)-α/β in response to many viruses. Here, the functions of these and other splenic DC subsets are further analyzed after in vivo infection with murine cytomegalovirus (MCMV). Viral challenge induced PDC maturation, their production of high levels of innate cytokines, and their ability to activate natural killer (NK) cells. The conditions also licensed PDCs to efficiently activate CD8 T cells in vitro. Non-plasmacytoid DCs induced T lymphocyte activation in vitro. As MCMV preferentially infected CD8α+ DCs, however, restricted access to antigens may limit plasmacytoid and CD11b+ DC contribution to CD8 T cell activation. IFN-α/β regulated multiple DC responses, limiting viral replication in all DC and IL-12 production especially in the CD11b+ subset but promoting PDC accumulation and CD8α+ DC maturation. Thus, during defense against a viral infection, PDCs appear specialized for initiation of innate, and as a result of their production of IFN-α/β, regulate other DCs for induction of adaptive immunity. Therefore, they may orchestrate the DC subsets to shape endogenous immune responses to viruses.
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Dissertations / Theses on the topic "DC subsets"

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Kakwata-Nkor, Deluce Nora. "Induction de sous-populations de cellules dendritiques humaines pro-tolérogènes par des fragments d’anticorps bispécifiques." Thesis, Tours, 2019. http://www.theses.fr/2019TOUR3805.

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L’induction de tolérance immune reste un challenge important dans le domaine de la transplantation d’organe, des maladies auto-immunes et inflammatoires. Les cellules Dendritiques (DCs), piliers de la réponse immunitaire, jouent un rôle crucial aussi bien dans l’induction d’une immunité spécifique que dans celle d’une tolérance immune. Chez l’homme, il existe au moins quatre types de DCs effectrices majeures, les DCs conventionnelles (cDC), les DCs Plasmacytoïdes (pDCs), les DCs inflammatoires (MoDCS) et les cellules de Langerhans (LCs). L'objectif du projet est de préparer différents sous-types de DCs (cDC, moDCs, pDC, moLCs) afin d’analyser leurs capacités à synthétiser de l’IL-10 et à s’orienter vers un profil pro tolérogène grâce au ciblage des PRRs avec des fragments d'anticorps. La différenciation des moDCs et moLCs est faite à partir de monocytes CD14+ isolés des PBMCs, en présence des cytokines spécifiques. La purification des pDC est faite avec des billes anti-BDCA-2 à partir des PBMC. Pour chacun des types de DCs, un anticorps bispécifique (BsAb) ciblant les PRRs est construit. L'effet des BsAb sur les DCs est analysé au niveau phénotypique par l'expression des marqueurs de co-stimulation et de maturation (CD83, CD86, HLA-DR, CD25) et la sécrétion des cytokines pro ou anti-inflammatoires (IL-10, TGF-ß, Il-12p70, IFN-/). L’induction de lymphocytes T-régulateur est étudiée par co-culture in vitro des DCs traitées par BsAb et des lymphocytes T naïfs allogéniques
Dendritic cells (DCs) have a central role in immunity and induce both specific immunity and immune tolerance thanks to their surface pathogen receptors (PPRs). The immune tolerance induced by tolerant DCs (Tol-DCs) appears as an interesting way to explore in order to improve the long-term transplantation outcome. Four DC subsets, at least, have been identified including conventional DCs (BDCA-1; BDCA-3), plasmacytoid DCs (pDC), Inflammatory DCs(MoDC) and Langerhans cells (LC). For each DC subset, an array of pathogen recognition receptors (PRRs) have been identified on their surface. The PRRs profile differs between DC subsets providing an individual responsiveness to target specific pathogens as well as to trigger and modulate immunological responses. The aim is to target DC subset PRRs by bispecific antibodies (BsAb) in order to induce physiological tolerance. Monocyte derived DC (moDC) and monocyte derived Langerhans DC (moLC) were obtained from CD14+ cells. The plasmacytoïd DC (pDC) were purified from an enriched DC cells fraction obtained by Percoll® gradient of PBMCs. The moDC, pDC and moLC subsets were analyzed by phenotype labelling and FACS. A Bispecific Ab (tandem scFv) were built to target PRR on DC subsets. The tandem is made of 2 scFv of 55KDa. The BsAb were produced using insect S2 (BIC05) or CHO cell (BIC15 or BIC25) and purified by protein L column. Each scFV recognize a PRR on DC. Each BsAb have been evaluated on its DC target and on PBMC at the phenotypic and functional levels by evaluating the maturation markers (CD83, CD86, CD25 and HLA-DR), cytokine secretions (IL-10, IL-12p70 and IFN- ) and the capacity to activate naïve T-cell as well as to induce regulatory T-cell (Treg)
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Mbabazi, Shoan. "Investigation into power distribution-grid interfacing for subsea tidal generation : rectification to a common DC-bus." Thesis, University of Sheffield, 2015. http://etheses.whiterose.ac.uk/11744/.

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The research objective was to investigate the generation and collection of power generated in a distributed tidal generation site, and realise the most economic grid connection methodology for a practical installation. Current tidal steam prototype designs utilise variable speed gear boxes, power frequency converters and variable pitch blades in the nacelle, thus requiring complicated, heavy and expensive equipment in the nacelle. Moreover the variable blade pitch mechanism has a large power demand, and has to operate day after day submerged in water at depths between 30m – 80m. The approach taken in this research is to replace major cost components with much cheaper ones, as well as locating as many components as possible onshore where costs are less. This thesis investigates a novel tidal stream power generation system referred to as “passive rectification to a common DC-bus” where an array of 3-phase synchronous generators operating at diverse speeds are connected to a common DC bus via passive diode rectifiers. The proposed tidal stream topology shows that variable pitched blades, variable speed gearboxes and frequency converters in the nacelle can be replaced by a much simpler system that uses fixed pitch blades, a fixed ratio gearbox, generator excitation control system, and a diode bridge rectifier. These components are generally cheaper, lighter, have a lower power demand and are more reliable than the components they replace. Also some of the voltage and frequency conversion is carried out ashore where costs are less, in addition to the overall reduction in the number of components required as some of the components are shared by multiple devices. As a result system efficiency is maximised, in addition to facilitating a reduction in capital, installation and operational costs. Power conversion from the tidal generation power system to shore is achieved via passive rectification to a common DC-bus, thus system power regulation is achieved by individual generator field current control, to attain optimum system operation including, maximum power extraction, power limitation and stall control, in turn improving system reliability and controllability. Furthermore passive stall control via generator field current regulation eliminates the need for mechanical brakes which reduces cost and improves reliability. The feasibility of the proposed power generation system was carried out via computer simulations and later validated via small scale laboratory hardware simulations.
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Book chapters on the topic "DC subsets"

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Lornell, Kip. "Back Then It Was Called Hillbilly Music (1946–1957)." In Capital Bluegrass, 20–84. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780199863112.003.0002.

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Bluegrass began to emerge as a subset of hillbilly music shortly after the close of World War II. However, both the terms “country” music and “bluegrass” were not commonly used until the late 1950s. Throughout the 1950s WARL radio highlighted this music, mostly notably lead by DJ Don Owens. Mandolinist Buzz Busby was perhaps the most influential pioneering bluegrass music in the area and participated in the first local television show (1954) featuring this music. By the late 1940s DC Records became the first local label to record hillbilly music talent around Washington, DC, and the Happy Melody Boys were the first hillbilly/bluegrass band to appear on national television (1955). The chapter closes with the formation of the Country Gentlemen, arguably the most important local bluegrass band.
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Conference papers on the topic "DC subsets"

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Heaster, Tiffany M., David L. Elion, Rebecca S. Cook, and Melissa C. Skala. "Abstract 3702: Quantitative imaging of metabolic changes in macrophage subsets." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3702.

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Talmadge, James E., Holly Britton, Alicia Dafferner, Phyllis Warkentin, and Kathryn Cole. "Abstract LB-192: Phenotyping human myeloid derived suppressor cells (MDSC) subsets." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-lb-192.

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Weiss, Vivian L., Timothy H. Lee, Todd D. Armstrong, and Elizabeth M. Jaffee. "Abstract 1923: Regulatory T-cell subsets suppress high avidity CD8 T-cell activation and trafficking." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1923.

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O’Donnell, JL, KL Summers, and A. Rothwell. "THU0079 Dendritic cell (dc) subsets in rheumatoid arthritis (ra) analysed by 4 colour flow cytometry." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.956.

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Kannan, Nagarajan, Maisam Makarem, Long Nguyen, Jeff Dong, Peter Eirew, and Connie Eaves. "Abstract 2919: Different ROS control mechanisms and mutagenic consequences in primitive subsets of normal human mammary cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2919.

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Obidi, Joyce O., Patricia Burke, Serguei Soukharev, Laura Richman, Dirk Mendel, Theresa LaVallee, and Haifeng Bao. "Abstract 5582: Interrogation of signaling networks in DLBCL to improve classification of patient subsets for targeted therapy." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5582.

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Shahin, Lauren, Elena Cubedo, Ebony Coats, Jeff Boyd, and Diana Azzam. "Abstract 1906: The role of immunogenic SPANX antigens in distinct cancer stem cell subsets within triple negative breast cancers." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1906.

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Gara, Sudheer Kumar, Yanli Pang, Christine Hollander, and Li Yang. "Abstract 4988: FOXP3, a critical mediator of regulatory T-cell function, is expressed in subsets of myeloid cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4988.

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Almamun, Md, and Kristen H. Taylor. "Abstract 5365: Genome-wide DNA methylation analysis in subsets of precursor B-cells isolated from umbilical cord blood." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5365.

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Thayer, Matt, Alden Wong, David Draper, Dan Saims, and Scott C. Wise. "Abstract 5624: Characterization of proliferation in multiple T-cell subsets in the CT26 murine colon carcinoma model by multi-color flow cytometry." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-5624.

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