Relevant bibliographies by topics / D-aspartate

Academic literature on the topic 'D-aspartate'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "D-aspartate"

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Yamada, Ryohei, Hisae Nagasaki, Yoko Nagata, Yasuo Wakabayashi, and Akio Iwashima. "Administration of D-aspartate increases D-aspartate oxidase activity in mouse liver." Biochimica et Biophysica Acta (BBA) - General Subjects 990, no. 3 (March 1989): 325–28. http://dx.doi.org/10.1016/s0304-4165(89)80053-4.

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Kim, P. M., X. Duan, A. S. Huang, C. Y. Liu, G. l. Ming, H. Song, and S. H. Snyder. "Aspartate racemase, generating neuronal D-aspartate, regulates adult neurogenesis." Proceedings of the National Academy of Sciences 107, no. 7 (January 26, 2010): 3175–79. http://dx.doi.org/10.1073/pnas.0914706107.

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Man, Eugene H., George H. Fisher, Iris L. Payan, Rodolfo Cadilla-Perezrios, Nancy M. Garcia, Radhika Chemburkar, Georgine Arends, and William H. Frey. "d-Aspartate in Human Brain." Journal of Neurochemistry 48, no. 2 (February 1987): 510–15. http://dx.doi.org/10.1111/j.1471-4159.1987.tb04122.x.

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Furuchi, Takemitsu, and Hiroshi Homma. "Free D-Aspartate in Mammals." Biological & Pharmaceutical Bulletin 28, no. 9 (2005): 1566–70. http://dx.doi.org/10.1248/bpb.28.1566.

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Katane, Masumi, and Hiroshi Homma. "D-Aspartate Oxidase: The Sole Catabolic Enzyme Acting on Free D-Aspartate in Mammals." Chemistry & Biodiversity 7, no. 6 (June 16, 2010): 1435–49. http://dx.doi.org/10.1002/cbdv.200900250.

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Kera, Yoshio, Hideaki Aoyama, Nobuyoshi Watanabe, and Ryo-hei Yamada. "Distribution of d-aspartate oxidase and free d-glutamate and d-aspartate in chicken and pigeon tissues." Comparative Biochemistry and Physiology Part B: Biochemistry and Molecular Biology 115, no. 1 (September 1996): 121–26. http://dx.doi.org/10.1016/0305-0491(96)00089-2.

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Huang, Yanhua H., Sukumaran Muralidharan, Saurabh R. Sinha, Joseph P. Y. Kao, and Dwight E. Bergles. "Ncm-d-aspartate: A novel caged d-aspartate suitable for activation of glutamate transporters and N-methyl-d-aspartate (NMDA) receptors in brain tissue." Neuropharmacology 49, no. 6 (November 2005): 831–42. http://dx.doi.org/10.1016/j.neuropharm.2005.07.018.

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Nagasaki, H., R. Yamada, R. Konno, Y. Yasumura, and A. Iwashima. "D-Aspartate oxidase activity and D-aspartate content in a mutant mouse strain lacking D-amino acid oxidase." Experientia 46, no. 5 (May 1990): 468–70. http://dx.doi.org/10.1007/bf01954233.

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Huang, Alex S., Dan A. Lee, and Seth Blackshaw. "d-Aspartate and d-aspartate oxidase show selective and developmentally dynamic localization in mouse retina." Experimental Eye Research 86, no. 4 (April 2008): 704–9. http://dx.doi.org/10.1016/j.exer.2008.01.015.

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Huang, A. S. "D-Aspartate Regulates Melanocortin Formation and Function: Behavioral Alterations in D-Aspartate Oxidase-Deficient Mice." Journal of Neuroscience 26, no. 10 (March 8, 2006): 2814–19. http://dx.doi.org/10.1523/jneurosci.5060-05.2006.

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Dissertations / Theses on the topic "D-aspartate"

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Carlson, Stephen Lee. "Characterization of D-Aspartate Receptor Currents in Aplysia californica." Scholarly Repository, 2010. http://scholarlyrepository.miami.edu/oa_dissertations/478.

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D-Aspartate (D-Asp) is an endogenous compound found in the central nervous system (CNS) of a variety of organisms. Despite its prevalence, however, relatively little understood of its physiological role. The prevailing theory is that D-Asp is an alternate agonist of N-methyl-D-aspartate receptor (NMDAR) channels. The goal of this work was to characterize the currents activated by D-Asp in neurons Aplysia californica, focusing on cells of the buccal S cluster (BSC). First, a general electrophysiological characterization was carried out, examining ion permeability, agonist dose-response, and the kinetics of activation, inactivation, and desensitization. D-Asp activated non-specific cation currents characterized by permeability to Na+ and K+. D-Asp-induced currents shared similar current-voltage relationships and time courses of activation and inactivation with L-glutamate (L-Glu)-induced currents. D-Asp currents, however, were subject to prolonged desensitization. Additionally, D-Asp activated currents independently of L-Glu, the known agonist of NMDAR channels, suggesting a non-NMDAR-dependent role of D-Asp. Next, select antagonists were used in an effort to pharmacologically characterize D-Asp receptor channels. These experiments suggested that D-Asp whole cell currents may be characterized by activation of multiple receptor sites, including NMDARS, excitatory amino acid transporters (EAATs), and a putative non-L-Glu D-Asp receptor. Furthermore, bath-applied D-Asp attenuated L-Glu-activated currents. Finally, D-Asp currents were compared to those evoked by acetylcholine (ACh) and serotonin (5-HT) in BSC cells. Results suggested that D-Asp activated receptor channels independently of ACh and 5-HT. Ten minute bath application of 5-HT was found to potentiate D-Asp current responses, likely through activation of a protein kinase C (PKC)-dependent mechanism, suggesting that D-Asp induced currents may be subject to synaptic plasticity associated with learning. While the identity of the putative D-Asp receptor remains elusive, the current work has advanced our understanding of the role D-Asp may play in the nervous system. These results should provide the groundwork for future studies aimed at identifying this unknown receptor channel, as well as investigation of the potential relationship of D-Asp receptor modulation to learning and memory in Aplysia, which may have relevance in higher organisms.
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Sim, Neil. "Molecular imaging probes for N-methyl-D-aspartate receptors." Thesis, Durham University, 2014. http://etheses.dur.ac.uk/10816/.

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The non-invasive detection of neuronal transmission is of prime importance in order to understand brain function better. This will aid cognitive neuroscience, as well as medical science, in the early detection of diseased states. Herein, approaches to molecular imaging of the NMDA receptor, a receptor subtype of the excitatory neurotransmitter glutamate, through the use of targeted contrast agents, is described. Initially, a series of NMDA receptor-targeted MRI contrast agents was developed based upon a known competitive NMDA receptor antagonist, appended to an N-linked ‘Gd-DOTA’ core that possesses a fast-exchanging water molecule. Their use as responsive MR imaging probes was evaluated in vitro using a neuronal cell line model, and three contrast agents showed large enhancements in cellular relaxation rates. In order to confirm NMDA receptor localisation, derivatives of the lead compounds were also prepared. The derivatives contained a biotin moiety, which allowed direct visualisation of the cell-surface receptors, after addition of an AvidinAlexaFluor®-488 conjugate. Using these derivatives, the specificity and reversibility (in the presence of glutamate) of binding at the NMDA receptor was demonstrated in living cells using laser scanning confocal microscopy. In an attempt to generate a single-component NMDA receptor-targeted optical imaging agent, a very bright europium complex conjugated to an NMDA receptor-binding moiety was synthesised. Unfortunately, upon incubation with a neuronal cell line model, complex localisation appeared to be dictated by the ligand structure and not by the receptor-binding moiety. One emerging imaging technique with potential applications in neuronal imaging is photoacoustic imaging. Two NMDA receptor-targeted photoacoustic imaging agents were synthesised and their ability to label NMDA receptors assessed in vitro. Finally, preliminary in vivo evaluation of the most promising photoacoustic imaging agent is described.
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Bera, Katarzyna D. "Autoantibodies to N-methyl D-aspartate receptors in autoimmune encephalitis." Thesis, University of Oxford, 2011. http://ora.ox.ac.uk/objects/uuid:6bbda982-ab5c-4982-b23a-0478c689869c.

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N-methyl-D-aspartate receptor (NMDAR) antibody encephalitis is a recently described autoimmune encephalopathy defined by the presence of serum antibodies that bind NMDARs (NMDAR-Abs). NMDAR-Ab encephalitis is a severe, but treatmentresponsive encephalitis with subacute onset. It can be associated with tumours and affects mainly young adults. Patients present with cognitive dysfunction, seizures, psychiatric and sleep disorders and most develop dyskinesias, autonomic instability and reduced consciousness. To explore further the NMDAR-Abs and their potential pathogenicity, a series of in vitro investigations were performed and preliminary attempts at passive transfer of disease. Human embryonic kidney (HEK) cells transfected with the NR1 and NR2B subunits, and live cultured neurons, were used first to detect NMDAR-Ab binding. Immunocytochemistry and ow cytometry demonstrated that binding to transfected HEK cells could be improved when NMDAR were presented in clusters by cotransfection with the postsynaptic density protein PSD-95. The NR1 subunit was identified as the target of NMDAR-Abs, and a novel quantitative assay based on immunoprecipitation of NR1 tagged by fusion with green uorescent protein was developed. Measurement of NMDAR-Ab levels showed that antibody levels corresponded to the clinical disease score within individual patients. Although the purification of full length NR1 was not successful, a secreted N-terminal construct was created and expressed in HEK cells. The binding of NMDAR-Abs was confirmed and this construct will be used for active immunisation in future. To explore pathogenic mechanisms in vitro, the main antibody subclasses were shown to be IgG1 and IgG3. Moreover the patients' autoantibodies, but not healthy control antibodies, were able to activate the complement cascade in vitro in cell lines and primary cultures. Finally, the NMDAR-Abs were shown to bind to primary microglial cultures and to cause morphological changes corresponding to early activation processes after prolonged exposure. The research has developed new assays that could be used for diagnosis and serial studies and revealed new potential mechanisms in NMDAR-Ab encephalitis.
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Yassin, Maged M. I. "N-methyl-D-aspartate, anoxia and glutamate antagonists in mammalian brain." Thesis, Queen's University Belfast, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.241524.

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Wu, Y. "Extrasynaptic signalling and plasticity mediated by N-Methyl-D-aspartate receptors." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1369568/.

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Synaptic N-Methyl-D-aspartate receptors (NMDARs) are crucial for neural coding and plasticity. However, little is known about the adaptive function of extrasynaptic NMDARs located on the dendritic shaft. Here we find that in CA1 pyramidal neurons backpropagating action potentials (bAPs) recruit shaft NMDARs exposed to ambient glutamate of non-vesicular origin. In contrast, spine NMDARs are "protected" under baseline conditions from such glutamate by perisynaptic transporters: bAP-evoked Ca2+ entry through these receptors can be detected upon synaptic glutamate release or local glutamate uncaging. During theta-burst firing, NMDAR-dependent Ca2+ entry either upregulates or downregulates an h-channel conductance (Gh) of the cell depending on whether synaptic glutamate release is intact or blocked. Gh plasticity in turn regulates dendritic input probed by local glutamate uncaging. Thus, the balance between activation of synaptic and extrasynaptic NMDARs can determine the sign of Gh-dependent plasticity. These results uncover a novel meta-plasticity mechanism potentially important for neural coding and memory formation.
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Lui, Pik Wa. "Modulation of N-methyl-D-aspartate receptor expression in neuronal cell culture." HKBU Institutional Repository, 2002. http://repository.hkbu.edu.hk/etd_ra/419.

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LeMaistre, Jillian. "Regulation of brain blood flow by astrocyte D-serine and N-methyl-D-aspartate receptors." Journal of Cerebral Blood Flow and Metabolism, 2012. http://hdl.handle.net/1993/8585.

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Functional hyperemia is an endogenous regulatory process coupling synaptic activity and elevated neuronal energy demand with increased local blood flow. This involves signalling between neurons, astrocytes and blood vessels, comprising the neurovascular unit. Astrocyte processes ensheath both synapses and blood vessels, permitting multi-modal responses to synaptic activity, where astrocyte cytoplasmic Ca2+ is elevated, triggering endfeet processes to release vasoactive molecules, such as arachidonic acid (AA) metabolites and gliotransmitters, such as D-serine. D-Serine is a co-agonist of the glycine regulatory site at N-methyl-D-aspartate (NMDA)-type glutamate receptors, and NMDA receptors play a role in functional hyperemia in vivo. Thus, our aim was to examine the role of astrocyte D-serine in NMDA receptor-mediated vasodilation. Using isolated pressurized mouse middle cerebral arteries (MCAs), we determined that co-application of glutamate and D-serine induced dose-dependent dilation which was mediated by NMDA receptors and endothelial nitric oxide synthase (eNOS) in an endothelial-dependent mechanism. This is the first evidence of direct vascular effects of D-serine and glutamate and suggests a possible role for endothelial NMDA receptor activation. Several studies indicate vascular endothelial cells express NMDA receptor subunits. However, expression in mouse endothelial cells has not been well characterized, so we identified NR1 and NR2C/2D subunit expression in primary brain endothelial cultures by PCR and immunocytochemistry, and further confirmed endothelial NR2C/2D expression in situ by immunohistochemistry. To further investigate astrocyte D-serine release and NMDA receptor-mediated functional hyperemia within the neurovascular unit, we used an acute cortical brain slice model where stimulation of astrocyte cytoplasmic Ca2+ induced vasodilation of nearby arterioles. Pharmacologically, D-serine release and NMDA receptor activation were implicated in this vasodilation. Endothelial-derived nitric oxide was also determined to induce dilation by inhibiting the production of an AA metabolite, 20-hydroxyeicostetranoic acid (20-HETE), a vasoconstrictor. This suggests an interaction between astrocyte vasoactive molecules, nitric oxide and D-serine, which warrants further investigation. Overall, our results provide evidence of modulation of NMDA receptor-mediated neurovascular coupling by astrocytic D-serine.
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Rutter, Anthony Richard. "Biochemical and pharmacological characterisation of the interaction between NMDA receptors and the scaffolding protein PSD-95." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.248043.

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Kotecha, Suhas Ashok. "G-protein coupled receptor modulation of N-methyl-D-aspartate channel activity." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ63766.pdf.

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Tofighy, Azita. "N-methyl-d-aspartate receptor desensitisation and anoxia in rat olfactory cortex." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361309.

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Books on the topic "D-aspartate"

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Raouf, Ramin K. Functional regulation of N-methyl-D-aspartate receptors by serine/threonine protein kinases. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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1962-, Li Min, ed. NMDA receptor protocols. Totowa, N.J: Humana Press, 1999.

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Bartlett, Mary Claire. Modulation of N-methyl-D-aspartate currents in cultured hippocampal neurones by protein kinase C. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1992.

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Malenfant, Sylvie A. The N-methyl-D-aspartate receptor system mediates spatial learning but not maternal experience effects. Ottawa: National Library of Canada, 1990.

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Konarski, Jakub Z. Molecular mechanism of protein kinase C enhancement of N-methyl-D-aspartate receptor calcium-dependent inactivation. Ottawa: National Library of Canada, 2002.

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P, Kozikowski Alan, and Barrionuevo German, eds. Neurobiology of the NMDA receptor: From chemistry to the clinic. New York, N.Y: VCH, 1991.

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Wrong, Andrew D. Bimodal modulation of N-methyl-D-aspartate-induced currents in rat CA1 hippocampal neurons by kainate application. Ottawa: National Library of Canada, 2002.

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Moussa, Raffy Cesario. The effect of seizure activity on tyrosine phosphorylation of the N-methyl-D-aspartate receptor in the hippocampus. Ottawa: National Library of Canada, 2002.

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Vij, Shilpa. Differential phosphorylation of the NR1 subunit of the N-methyl-D-aspartate receptor following hypoxia-ischemia in 7-and 21-day old rat brains. Ottawa: National Library of Canada, 2003.

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Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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Book chapters on the topic "D-aspartate"

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Schomburg, Dietmar, Margit Salzmann, and Dörte Stephan. "D-Aspartate oxidase." In Enzyme Handbook, 801–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-58051-2_166.

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Schomburg, Dietmar, Margit Salzmann, and Dörte Stephan. "D-Glutamate(D-aspartate) oxidase." In Enzyme Handbook, 875–77. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-58051-2_179.

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Fisher, George H., and Mara Tsesarskaia. "HPLC Methods for Determination of d-Aspartate and N-methyl-d-Aspartate." In Methods in Molecular Biology, 253–64. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-331-8_16.

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, Ben J. Harrison, Christos Pantelis, Christof Baltes, Thomas Mueggler, et al. "N-Methyl-D-Aspartate Receptor." In Encyclopedia of Psychopharmacology, 757–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1294.

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Kato, Yusuke, Diem Hong Tran, Huong Thi Thanh Trinh, and Kiyoshi Fukui. "d-Amino Acid Oxidase and d-Aspartate Oxidase." In D-Amino Acids, 293–309. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56077-7_19.

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Schomburg, Dietmar, and Ida Schomburg. "3-hydroxy-d-aspartate aldolase 4.1.3.41." In Class 3.4–6 Hydrolases, Lyases, Isomerases, Ligases, 467–69. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-36260-6_37.

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Rey, Jose A. "N-Methyl-D-Aspartate (NMDA) Receptors." In Encyclopedia of Clinical Neuropsychology, 1778. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1682.

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Rey, Jose A. "N-Methyl-D-Aspartate (NMDA) Receptors." In Encyclopedia of Clinical Neuropsychology, 1. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1682-2.

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Rey, Jose A. "N-Methyl-D-Aspartate (NMDA) Receptors." In Encyclopedia of Clinical Neuropsychology, 2458–59. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1682.

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Errico, Francesco, and Alessandro Usiello. "Neuromodulatory Activity of d-Aspartate in Mammals." In D-Amino Acids, 219–37. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56077-7_14.

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Conference papers on the topic "D-aspartate"

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Trajano, Maria Lourdes M., Pediatric Neurologist, and Karenne N. Somera. "N-methyl-d-aspartate Receptor Encephalitis in a Tertiary Hospital: A Case Report." In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.496.

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Ohgami, Masatsugu, Nobuhiko Takai, Masahiko Watanabe, Koichi Ando, Akiko Uzawa, and Ryoichi Hirayama. "EFFECT OF N-METHYL-D-ASPARTATE RECEPTOR ANTAGONIST ON RADIATION-INDUCED GUT INJURIES IN MICE." In RAD Conference. RAD Association, 2017. http://dx.doi.org/10.21175/radproc.2017.02.

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Fang, Yuqiang, King W. C. Lai, Catherine Y. Y. Iu, Cathy N. P. Lui, Carmen K. M. Fung, Hung-Wing Li, Ken K. L. Yung, and Ning Xi. "Investigation of N-methyl-D-aspartate induced mechanical behavior of neuroblastoma cells using atomic force microscopy." In 2013 IEEE 13th International Conference on Nanotechnology (IEEE-NANO). IEEE, 2013. http://dx.doi.org/10.1109/nano.2013.6720884.

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Young, Emma C., Helen Sumner, Samantha Decalmer, Lesley Houghton, Ashley A. Woodcock, and Jaclyn Smith. "Does Central Up-regulation Of The N-Methyl-D-Aspartate Receptor Contribute To Cough Reflex Hypersensitivity?" In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5906.

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Golovynska, Iuliia, Tatiana V. Beregova, Tatiana M. Falalyeyeva, Sergii Golovynskyi, Junle Qu, and Tymish Y. Ohulchanskyy. "Combining optical imaging and pharmacological methods to localize N-methyl-D-aspartate glutamate receptors in a stomach wall." In International Conference on Photonics and Imaging in Biology and Medicine. Washington, D.C.: OSA, 2017. http://dx.doi.org/10.1364/pibm.2017.w3a.107.

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Sareen, Nishtha, and Seth Koenig. "Limbic Encephalitis Including Anti-N-Methyl D-Aspartate Encephalitis May Be Under-Recognized In The Medical Intensive Care Unit." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5942.

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Manole, Corina, Cristina Serban, and Alexandru Bogdan Ciubara. "KETAMINE IN PERIOPERATIVE DEPRESSIVE SYMPTOMS (PDS) IMPROVEMENT - REVIEW." In The European Conference of Psychiatry and Mental Health "Galatia". Archiv Euromedica, 2023. http://dx.doi.org/10.35630/2022/12/psy.ro.9.

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Anxiety and depression are the most frequent psychiatric disorders associated with organic diseases. PDS (Perioperative Depression Symptoms) represent a depressive episode which occurs mostly in the early postoperative phase. It was observed that the patients presenting PSD have a higher risk of postoperative complications, an increased length of hospitalization and a more reserved prognosis. A series of recent studies have shown that the usage of ketamine in small doses significantly reduces major depression symptoms in the short time after its administration. The ketamine blocks the NMDA (N-methyl-D-aspartate) receptors, leading to the presynaptic release of glutamate and increasing the activity of dopaminergic neurons with antidepressant role.
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Haaf, Moritz, Stjepan Curic, Saskia Steinmann, Jonas Rauh, Christoph Mulert, and Gregor Leicht. "Glycine and the ketamine model of schizophrenia: Modulating the N-methyl-D-aspartate receptor (NMDAR) offers new insights into the glutamate hypothesis of schizophrenia." In Abstracts of the 3rd Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0042-1757664.

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Ramos-Bello, D., AN Rangel-Botello, G. Barragan-Pickens Aguilera, TA Luna-Zúñiga, AJ Pedro-Martínez, G. Martínez-Flores, A. Bravo-Oro, and C. Abud-Mendoza. "THU0501 Efficacy and safety of methotrexate as maintenance therapy for children with anti–n-methyl-d-aspartate receptor (NMDAR) encephalitis: experience of a single center." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2726.

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Akoumia, F. K. K. "Transcriptomic and Proteomic Analysis of the Platelet-Derived Growth Factor (PDGF) Response in Pulmonary Vascular Smooth Muscle Cells from Patients with Pulmonary Arterial Hypertension: Implication of the N-Methyl-D-Aspartate Receptor (NMDAR)." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a4608.

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Reports on the topic "D-aspartate"

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Patel, Tapan P., Scott C. Ventre, Pallab K. Singh, and David F. Meaney. NR2B-N-Methyl-D-Aspartate Receptors Contribute to Network Asynchrony and Loss of Long-Term Potentiation Following Mild Mechanical Injury In Vitro. Fort Belvoir, VA: Defense Technical Information Center, August 2012. http://dx.doi.org/10.21236/ada586145.

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