Books on the topic 'Cystic fibrosis'

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1

Silverstein, Alvin. Cystic fibrosis. New York: F. Watts, 1994.

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2

Davis, Stephanie Duggins, Margaret Rosenfeld, and James Chmiel, eds. Cystic Fibrosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7.

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3

Moss, Richard B., ed. Cystic Fibrosis. Totowa, NJ: Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-0475-6.

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4

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-117-8.

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5

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-120-8.

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6

Gold, Susan Dudley. Cystic fibrosis. New York: Crestwood House, 2000.

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7

B, Davis Pamela, ed. Cystic fibrosis. New York: M. Dekker, 1993.

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8

1951-, Goodfellow P., ed. Cystic fibrosis. Oxford: Oxford University Press, 1989.

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9

B, Fiel Stanley, ed. Cystic fibrosis. Philadelphia: Saunders, 1998.

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10

E, Hodson Margaret, Geddes Duncan M, and Bush, Andrew, 1954 Apr. 24-, eds. Cystic fibrosis. 3rd ed. London: Hodder Arnold, 2007.

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11

Jacqueline, Langwith, ed. Cystic fibrosis. Detroit: Greenhaven Press, 2009.

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12

Giddings, Sharon. Cystic fibrosis. New York: Chelsea House, 2009.

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13

Bjorklund, Ruth. Cystic fibrosis. New York: Marshall Cavendish Benchmark, 2009.

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14

Mandolfo, Anna. Cystic fibrosis. Southampton: Dulphar Laboratories, 1991.

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15

O, Warner J., ed. Cystic fibrosis. Edinburgh: Churchill Livingstone, 1992.

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16

M, Geddes Duncan, and Hodson Margaret E, eds. Cystic fibrosis. London: Chapman & Hall Medical, 1995.

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17

Rosaler, Maxine. Cystic fibrosis. New York: The Rosen Pub. Group, 2007.

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18

J, David T., Goeting Nicola L. M, and Duphar Medical Relations, eds. Cystic fibrosis. Southampton: Duphar Medical Relations, 1991.

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19

Capewell, Gillian. Cystic fibrosis. London: Office of Health Economics, 1986.

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20

Gillian, Capewell, and Association of the British Pharmaceutical Industry. Office of Health Economics., eds. Cystic fibrosis. London: Office of Health Economics, 1986.

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21

Dennis, Shale, ed. Cystic fibrosis. London: BMJ Publishing Group, 1996.

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22

Lewis, Allen Julian, Rubenstein Ronald, and Panitch Howard B, eds. Cystic fibrosis. New York, NY: Informa Healthcare USA, 2010.

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23

Harris, Ann. Cystic fibrosis: The facts. 3rd ed. Oxford: Oxford University Press, 2003.

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24

Filbrun, Amy G., Thomas Lahiri, and Clement L. Ren. Handbook of Cystic Fibrosis. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32504-0.

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25

Farinha, Carlos M. CFTR and Cystic Fibrosis. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65494-2.

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26

Yen, Elizabeth H., and Amanda Radmer Leonard, eds. Nutrition in Cystic Fibrosis. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16387-1.

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27

R, Yankaskas James, and Knowles Michael R, eds. Cystic fibrosis in adults. Philadelphia: Lippincott-Raven, 1999.

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28

Maurice, Super, ed. Cystic fibrosis: The facts. 2nd ed. Oxford [England]: Oxford University Press, 1991.

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29

National Heart, Lung, and Blood Institute, ed. Facts about cystic fibrosis. [Bethesda, Md: National Institutes of Health, National Heart, Lung, and Blood Institute, 1995.

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30

Jenni, Murray, Health Technology Assessment Programme, Great Britain. Standing Group on Health Technology., National Co-ordinating Centre for HTA (Great Britain), and HTA Commissioning Board, eds. Screening for cystic fibrosis. Alton: Core Research on behalf of NCCHTA, 1999.

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31

A, Frizzell Raymond, ed. Physiology of cystic fibrosis. Bethesda, MD: American Physiological Society, 1999.

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32

A, Frizzell Raymond, ed. Physiology of cystic fibrosis. Bethesda, MD: American Physiological Society, 1999.

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33

National Heart, Lung, and Blood Institute., ed. Facts about cystic fibrosis. [Bethesda, Md: National Institutes of Health, National Heart, Lung, and Blood Institute, 1995.

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34

E, Hodson Margaret, ed. Cystic fibrosis in colour. London: Gower, 1990.

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35

Maurice, Super, ed. Cystic fibrosis, the facts. 3rd ed. Oxford: Oxford University Press, 1995.

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36

A, Dodge J., Brock D. J. H, and Widdicombe J. W, eds. Cystic fibrosis-- current topics. Chichester: J. Wiley, 1993.

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37

Ann, Harris, ed. Cystic fibrosis: The facts. 2nd ed. Oxford, England: Oxford University Press, 2008.

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38

N, Leatte Paul, ed. Cystic fibrosis etiology, diagnosis, and treatments. Hauppauge, NY: Nova Science Publishers, 2009.

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39

Hodson, Margaret, Duncan Geddes, and Andrew Bush. Cystic Fibrosis. 3rd ed. A Hodder Arnold Publication, 2007.

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40

Hodson, Margaret, Duncan Geddes, and Andrew Bush. Cystic Fibrosis. Taylor & Francis Group, 2007.

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41

Webb, A. K. Cystic Fibrosis. European Respiratory Society, 2006.

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42

Spilsbury, Richard. Cystic Fibrosis. Rosen Publishing Group, 2018.

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43

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Cystic fibrosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0021.

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Abstract:
Gastrointestinal manifestations 156Management of gastrointestinal symptoms in children with CF 158Nutrition in CF 158Nutritional management 159Vitamins 160The incidence of cystic fibrosis (CF) is around 1 in 2500. Cases are diagnosed as a consequence of population screening or high-risk screening, or following presentation with clinical symptoms typical of the disorder. The basic defect is in the CFTR (cystic fibrosis transmembrane conductance regulator) protein which codes for a cyclic adenosine monophosphate-regulated chloride transporter in epithelial cells of exocrine organs. This is involved in salt and water balance across epithelial surfaces. The gene is on chromosome 7. There are multiple known mutations, the most common being ...
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44

Webb, A. K., and F. A. Ratjen, eds. Cystic Fibrosis. European Respiratory Society Journals Ltd, 2006. http://dx.doi.org/10.1183/1025448x.erm3506.

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45

Mall, Marcus A., and J. Stuart Elborn, eds. Cystic Fibrosis. European Respiratory Society, 2014. http://dx.doi.org/10.1183/1025448x.erm6414.

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46

Bright-Thomas, Rowland J., and Andrew M. Jones. Cystic fibrosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0132.

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Cystic fibrosis is the most common lethal autosomal recessive disorder in Caucasians. There is no known survival advantage of the heterozygote carrier state. Chronic progressive pulmonary infection and bronchiectasis are the major causes of morbidity and mortality. The disease affects all ductal systems where the basic defect is manifest, including the pancreas, gastrointestinal tract, sinuses, hepatobiliary system, and male reproductive system, and has significant effects on nutrition and growth.
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47

Horsley, Alex, Steve Cunningham, and Alistair Innes, eds. Cystic Fibrosis. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199582709.001.0001.

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48

Martin, David, and Junzheng Wu. Cystic Fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0021.

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Cystic fibrosis (CF) is an inherited chronic disease that affects about 30,000 children and adults in the United States and 70,000 worldwide. CF is the most common fatal inherited disorder affecting Caucasians in the United States. While its presentation can vary in severity, the most common clinical manifestations are progressive lung damage and chronic digestive problems due to exocrine gland dysfunction and the production of thick viscous mucus. Careful perioperative management is important to avoid respiratory complications.
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49

Tinch, Brian, David Martin, and Junzheng Wu. Cystic Fibrosis. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0018.

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Cystic fibrosis is an inherited disorder. The diagnosis should be suspected in an infant who has meconium ileus or infants presenting to the operating room with volvulus. Cystic fibrosis is characterized by frequent mucous plugging in the respiratory tract which may manifest as wheezing and frequent intermittent flare-ups of respiratory decompensation. Optimization of the affected child’s respiratory status prior to elective surgery is mandatory to prevent difficulty with intraoperative ventilation. While the laryngeal mask airway may be used for short procedures, the use of an endotracheal tube facilitates suctioning of the frequently inspissated secretions that accompany cystic fibrosis in order to optimize ventilation.
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50

Snell, Jamey, and Thomas J. Mancuso. Cystic Fibrosis. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0023.

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Cystic fibrosis (CF) is an inherited, autosomal recessive, multisystem disease. Dysfunction of the cystic fibrosis transmembrane conductance regulator protein (CFTR) in epithelial cells is the primary defect in CF. Defects in CFTR are the cause for lung disease, exocrine pancreatic insufficiency and failure, male infertility, and liver disease. CF can present with a variety of respiratory and gastrointestinal signs, including meconium ileus in the newborn period, hypernatremic dehydration, pulmonary insufficiency, nasal polyps, and insulin-dependent diabetes mellitus. As affected children grow, dysfunction in CFTR leads to chronic and progressive lung disease, characterized by suppurative infection and the development of bronchiectasis. CFTR dysfunction also affects exocrine function, leading to pancreatic insufficiency, malabsorption, and growth failure. In the past, history and physical exam with sweat chloride testing were the cornerstones of diagnosis. Diagnosis is now made with the newborn screening test for immunoreactive trypsinogen.
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