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Books on the topic 'Cystic Fibrosis in adulthood'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Cystic fibrosis. Oxford: Oxford University Press, 2010.

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2

Cystic fibrosis. Detriot: Lucent Books, 2013.

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3

Giddings, Sharon. Cystic fibrosis. New York: Chelsea House, 2009.

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4

Rosaler, Maxine. Cystic fibrosis. New York: The Rosen Pub. Group, 2007.

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5

Bjorklund, Ruth. Cystic fibrosis. New York: Marshall Cavendish Benchmark, 2009.

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6

E, Hodson Margaret, Geddes Duncan M, and Bush, Andrew, 1954 Apr. 24-, eds. Cystic fibrosis. 3rd ed. London: Hodder Arnold, 2007.

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7

Capewell, Gillian. Cystic fibrosis. London: Office of Health Economics, 1986.

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8

Silverstein, Alvin. Cystic fibrosis. New York: F. Watts, 1994.

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9

Mandolfo, Anna. Cystic fibrosis. Southampton: Dulphar Laboratories, 1991.

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10

Davis, Stephanie Duggins, Margaret Rosenfeld, and James Chmiel, eds. Cystic Fibrosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7.

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11

Moss, Richard B., ed. Cystic Fibrosis. Totowa, NJ: Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-0475-6.

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12

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-117-8.

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13

Amaral, Margarida D., and Karl Kunzelmann, eds. Cystic Fibrosis. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-120-8.

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14

Gold, Susan Dudley. Cystic fibrosis. New York: Crestwood House, 2000.

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15

Dennis, Shale, ed. Cystic fibrosis. London: BMJ Publishing Group, 1996.

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16

Understanding cystic fibrosis. Jackson: University Press of Mississippi, 1998.

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17

Explaining -- cystic fibrosis. London: Franklin Watts, 2009.

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18

Explaining cystic fibrosis. Mankato, Minn: Smart Apple Media, 2010.

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19

A, Dodge J., Brock D. J. H, and Widdicombe J. W, eds. Cystic fibrosis-- current topics. Chichester: J. Wiley, 1993.

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20

Maurice, Super, ed. Cystic fibrosis, the facts. 3rd ed. Oxford: Oxford University Press, 1995.

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21

Harris, Ann. Cystic fibrosis: The facts. 3rd ed. Oxford: Oxford University Press, 2003.

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22

Ann, Harris, ed. Cystic fibrosis: The facts. 2nd ed. Oxford, England: Oxford University Press, 2008.

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23

Filbrun, Amy G., Thomas Lahiri, and Clement L. Ren. Handbook of Cystic Fibrosis. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32504-0.

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24

Farinha, Carlos M. CFTR and Cystic Fibrosis. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-65494-2.

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25

Yen, Elizabeth H., and Amanda Radmer Leonard, eds. Nutrition in Cystic Fibrosis. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16387-1.

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26

Maurice, Super, ed. Cystic fibrosis: The facts. 2nd ed. Oxford [England]: Oxford University Press, 1991.

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27

N, Leatte Paul, ed. Cystic fibrosis etiology, diagnosis, and treatments. Hauppauge, NY: Nova Science Publishers, 2009.

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28

Stableforth, David. Growing up with cystic fibrosis. Bromley: Cystic Fibrosis Research Trust, 1991.

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29

Cystic fibrosis: Diagnosis and protocols. New York: Humana Press, 2011.

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30

R, Yankaskas James, and Knowles Michael R, eds. Cystic fibrosis in adults. Philadelphia: Lippincott-Raven, 1999.

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31

Kriemler, Susi. Exercise, physical activity, and cystic fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0033.

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Cystic fibrosis (CF) is the most common genetic autosomal recessive disease of the Caucasian race, generally leading to death in early adulthood.1 The frequency of the gene carrier (heterozygote) is 1:20–25 in Caucasian populations, 1:2000 in African-Americans, and practically non-existent in Asian populations. The disease occurs in about 1 in every 2500 life births of the white population. Mean survival has risen from 8.4 years in 1969 to 32 years in 2000 due to improvements in treatment. The genetic defect causes a pathological electrolyte transport through the cell membranes by a defective chloride channel membrane transport protein [cystic fibrosis transmembrane conductance regulator (CFTR)]. With respect to the function, this affects mainly the exocrine glands of secretory cells, sinuses, lungs, pancreas, liver, and the reproductive tract of the human body leading to a highly viscous, water-depleted secretion. The secretion cannot leave the glands and in consequence causes local inflammation and destruction of various organs. The main symptoms include chronic inflammatory pulmonary disease with a progressive loss of lung function, exocrine and sometimes endocrine pancreas insufficiency, and an excessive salt loss through the sweat glands.1 A summary of the signs and symptoms of CF will be given with a special emphasis on the effect of exercise performance and capacity.

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32

Hill, Uta, Jane Ashbrook, and Charles Haworth. Metabolic and musculoskeletal effects of cystic fibrosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198702948.003.0009.

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This chapter provides a comprehensive update on the prevention, recognition, and treatment of low bone mineral density in people with CF. As life expectancy improves, the extra-pulmonary complications of CF are becoming increasingly important to quality of life. Up to 25 per cent of CF patients have reduced bone mineral density in adulthood, leading to the development of fragility fractures which cause pain, thereby interfering with airway clearance and predisposing to pulmonary infection. Osteoporosis can be a relative contraindication for lung transplantation. Other important musculoskeletal issues including CF arthropathy, growth, and urinary incontinence are covered. CF arthropathy is a non-erosive episodic sero-negative arthritis, often difficult to treat and which may require specialist input. Urinary incontinence is common girls and women with CF and has a negative impact on quality of life and ability to complete therapies. The pathophysiology and management of urinary incontinence are discussed.

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33

Hernell, O. Cystic Fibrosis. S Karger Pub, 2006.

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34

Allen, Julian L., Howard B. Panitch, and Ronald C. Rubenstein. Cystic Fibrosis. Taylor & Francis Group, 2017.

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35

Cystic fibrosis. London: Chapman & Hall Medical, 1995.

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36

Cystic Fibrosis. Oxford University Press, 2015.

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37

Miller, Petra. Cystic Fibrosis. Cavendish Square Publishing LLC, 2015.

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38

Spilsbury, Richard. Cystic Fibrosis. Rosen Publishing Group, 2018.

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39

Allen, Julian, Howard Panitch, and Ronald Rubenstein. Cystic Fibrosis. Taylor & Francis Group, 2016.

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40

Cystic Fibrosis. Cavendish Square, 2016.

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41

Allen, Julian, Howard Panitch, and Ronald Rubenstein. Cystic Fibrosis. Taylor & Francis Group, 2016.

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42

Abramovitz, Melissa. Cystic Fibrosis. Greenhaven Publishing LLC, 2013.

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43

Lee, Justin. Cystic Fibrosis. Rosen Publishing Group, 2001.

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44

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Cystic fibrosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0021.

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Gastrointestinal manifestations 156Management of gastrointestinal symptoms in children with CF 158Nutrition in CF 158Nutritional management 159Vitamins 160The incidence of cystic fibrosis (CF) is around 1 in 2500. Cases are diagnosed as a consequence of population screening or high-risk screening, or following presentation with clinical symptoms typical of the disorder. The basic defect is in the CFTR (cystic fibrosis transmembrane conductance regulator) protein which codes for a cyclic adenosine monophosphate-regulated chloride transporter in epithelial cells of exocrine organs. This is involved in salt and water balance across epithelial surfaces. The gene is on chromosome 7. There are multiple known mutations, the most common being ...

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45

Webb, A. K., and F. A. Ratjen, eds. Cystic Fibrosis. European Respiratory Society Journals Ltd, 2006. http://dx.doi.org/10.1183/1025448x.erm3506.

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46

Mall, Marcus A., and J. Stuart Elborn, eds. Cystic Fibrosis. European Respiratory Society, 2014. http://dx.doi.org/10.1183/1025448x.erm6414.

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47

Bright-Thomas, Rowland J., and Andrew M. Jones. Cystic fibrosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0132.

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Cystic fibrosis is the most common lethal autosomal recessive disorder in Caucasians. There is no known survival advantage of the heterozygote carrier state. Chronic progressive pulmonary infection and bronchiectasis are the major causes of morbidity and mortality. The disease affects all ductal systems where the basic defect is manifest, including the pancreas, gastrointestinal tract, sinuses, hepatobiliary system, and male reproductive system, and has significant effects on nutrition and growth.

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48

Horsley, Alex, Steve Cunningham, and Alistair Innes, eds. Cystic Fibrosis. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199582709.001.0001.

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49

Martin, David, and Junzheng Wu. Cystic Fibrosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0021.

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Cystic fibrosis (CF) is an inherited chronic disease that affects about 30,000 children and adults in the United States and 70,000 worldwide. CF is the most common fatal inherited disorder affecting Caucasians in the United States. While its presentation can vary in severity, the most common clinical manifestations are progressive lung damage and chronic digestive problems due to exocrine gland dysfunction and the production of thick viscous mucus. Careful perioperative management is important to avoid respiratory complications.

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50

Tinch, Brian, David Martin, and Junzheng Wu. Cystic Fibrosis. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0018.

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Cystic fibrosis is an inherited disorder. The diagnosis should be suspected in an infant who has meconium ileus or infants presenting to the operating room with volvulus. Cystic fibrosis is characterized by frequent mucous plugging in the respiratory tract which may manifest as wheezing and frequent intermittent flare-ups of respiratory decompensation. Optimization of the affected child’s respiratory status prior to elective surgery is mandatory to prevent difficulty with intraoperative ventilation. While the laryngeal mask airway may be used for short procedures, the use of an endotracheal tube facilitates suctioning of the frequently inspissated secretions that accompany cystic fibrosis in order to optimize ventilation.

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