Relevant bibliographies by topics / Cystic Fibrosis in adulthood

Academic literature on the topic 'Cystic Fibrosis in adulthood'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Cystic Fibrosis in adulthood"

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Simmonds, Nicholas J. "Cystic Fibrosis Presenting in Adulthood." Clinical Pulmonary Medicine 20, no. 1 (January 2013): 1–5. http://dx.doi.org/10.1097/cpm.0b013e31827a292e.

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Hota, Partha, and Rachna Madan. "Cystic Fibrosis from Childhood to Adulthood." Radiologic Clinics of North America 58, no. 3 (May 2020): 475–86. http://dx.doi.org/10.1016/j.rcl.2019.12.003.

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Palmer, Marykay Lannon, and Laura S. Boisen. "Cystic Fibrosis and the Transition to Adulthood." Social Work in Health Care 36, no. 1 (November 12, 2002): 45–58. http://dx.doi.org/10.1300/j010v36n01_04.

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Tješić Drinković, Dorian, and Duška Tješić-Drinković. "Clinical presentations of cystic fibrosis in adulthood." Paediatria Croatica 59, no. 2 (June 26, 2015): 74–80. http://dx.doi.org/10.13112/pc.2015.12.

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Baran, E., N. Granero, B. Hendriksen, F. Butti, V. D'Ascenzo, S. Ibarra, V. Pistorio, L. Volta, and G. García. "22 Misdiagnosis of cystic fibrosis in adulthood." Journal of Cystic Fibrosis 12 (June 2013): S54. http://dx.doi.org/10.1016/s1569-1993(13)60165-4.

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Bourgani, E., C. Kosti, E. Stagaki, and F. Diamantea. "P027 Diagnosis of cystic fibrosis in adulthood." Journal of Cystic Fibrosis 19 (June 2020): S62. http://dx.doi.org/10.1016/s1569-1993(20)30364-7.

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Nick, Jerry A., and David M. Rodman. "Manifestations of cystic fibrosis diagnosed in adulthood." Current Opinion in Internal Medicine 5, no. 1 (February 2006): 68–73. http://dx.doi.org/10.1097/01.mcp.0000183052.56728.76.

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Radlovic, Nedeljko. "Cystic fibrosis." Srpski arhiv za celokupno lekarstvo 140, no. 3-4 (2012): 244–49. http://dx.doi.org/10.2298/sarh1204244r.

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Cystic fibrosis (CF) is a multisystemic autosomal recessive disease caused by a defect in the expression of CFTR protein, i.e. chloride channel present in the apical membrane of respiratory, digestive, reproductive and sweat glands epithelium. It primarily occurs in the Caucasians, while being considerably or exceptionally rare in persons of other races. Absence, deficit or structural and functional abnormalities of CFTR protein lead to mucosal hyperconcentration in the respiratory, digestive and reproductive systems and malabsorption of chloride and sodium in the sweat glands. Thus, the clinical features of patients? with CF are predominated by respiratory, digestive and reproductive disorders, as well as the tendency to dehydration in the condition of increased sweating. Beside genotype variations, the degree of disease manifestation is also essentially influenced by various exogenous factors, such as the frequency and severity of respiratory infections, the level of aero-pollution, quality of immunoprophylaxis, patients? nutritional condition and other. Chloride concentration of over 60 mmol/L in sweat, a high level of immunoreactive chymotrypsinogen in blood and the verification of homozygous mutation of CFTR gene are the basic methods in the diagnostics of the disease. CF belongs to the group of severe and complex chronic diseases, and therefore requires multidisciplinary therapeutic approach. Owing to the improvement of healthcare provision, most patients with CF now survive into adulthood. In addition, their quality of life is also considerably improved.
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Harriss, Patrick B., Anusha Jogimahanti, Vincent Herlevic, Hrishikesh Samant, James D. Morris, and Paul Jordan. "2439 Cystic Fibrosis Liver Disease: A Curious Case of Atypical Cystic Fibrosis Diagnosed in Adulthood." American Journal of Gastroenterology 114, no. 1 (October 2019): S1350—S1351. http://dx.doi.org/10.14309/01.ajg.0000599288.13732.20.

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Phillipson, Greg. "Cystic fibrosis and reproduction." Reproduction, Fertility and Development 10, no. 1 (1998): 113. http://dx.doi.org/10.1071/r98044.

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Cystic fibrosis (CF) is the most common autosomal recessive disease. CF and congenital bilateral absence of the vas deferens (CBAVD) share a genetic and embryological background. Since the 1960s, medical therapy to reduce the progressive obstructive lung disease and nutritional deficiencies has resulted in most CF patients reaching adulthood. With the improved life expectancy of CF patients, new issues in reproductive health and pregnancy management have arisen. Puberty is delayed, with menarche often occurring eighteen months later than the average. Almost all men with CF are azoospermic. In both CF and CBAVD, the vas deferens is absent and the seminal vesicles are often hypoplastic. Many women with CF are subfertile, and if pregnancy is achieved there is an observed increase in maternal morbidity and mortality. The understanding of the molecular basis of CF and CBAVD has evolved, with the identification of hundreds of CF gene mutations and discovery of an associated intron polymorphism of the CF gene. The concept of severe and mild mutations has been introduced to explain the severe and mild phenotype variations such as the pancreatic insufficient and pancreatic sufficient patient. This paper reviews the above issues to assist with the management of infertile couples with CF or CBAVD.
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Dissertations / Theses on the topic "Cystic Fibrosis in adulthood"

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Hogan, Joanne V. "The transition to adulthood for young people with cystic fibrosis." Thesis, Liverpool John Moores University, 2008. http://researchonline.ljmu.ac.uk/5865/.

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Chadwick, Helen Kay. "Cognitive function in cystic fibrosis and cystic fibrosis related diabetes (CFRD)." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/16912/.

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Cystic fibrosis (CF) is a complex multisystem disease caused by a gene mutation of a protein called the CF Transmembrane Conductance Regulator (CFTR). Glucose tolerance abnormalities are common in CF and the prevalence of CF related diabetes (CFRD), which shares clinical characteristics with type 1 (T1DM) and type 2 diabetes (T2DM), increases with age. Impaired glucose tolerance (IGT), T1DM and T2DM are associated with cognitive impairment relative to healthy controls. The overall aim of this thesis was to examine cognitive function in CF. Study 1 investigated the impact of CF on cognitive function, in people with CFRD (n=49), people with CF who are not diabetic (CFND, n=49) and healthy controls (n=49). Memory, attention and processing speed, and cognitive flexibility was impaired in CFRD, and to a lesser degree in CFND, relative to healthy controls. Study 2 assessed cognitive function over a 1-3 year period in people with CFRD (N=36) and found no evidence of cognitive decline despite a decline in lung function. Study 3 compared cognitive function in people with CFRD who were post transplant (CFRDTx, n=18), people with CFRD (who were not post transplant, n=18), and healthy controls (n=18). CFRD was associated with impairment in attention and processing speed, spatial working memory, cognitive flexibility and to a lesser extent verbal memory. Cognitive function did not improve post transplantation in people with CFRD. Study 4 followed up people with CFRDTx (N=8) over an 18±6 month period and found no decline in cognitive function. Taken together, the evidence presented in this thesis suggests that diabetes in CF is associated with cognitive impairment, and that maintaining glycaemic control protects against cognitive decline. The cognitive impairment observed in people with CF is of clinical significance and has implications for self care and disease management. The recent discovery that CFTR is present in the pancreas and the brain has important implications for the effects of the new CFTR potentiator and corrector therapies on cognitive function in CF.
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Dwyer, Tiffany Jane. "Exercise in cystic fibrosis." Discipline of Physiotherapy, Faculty of Health Sciences, University of Sydney, 2010. http://hdl.handle.net/2123/6349.

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Doctor of Philosophy (PhD)
Exercise and physical activity have many benefits for adults with cystic fibrosis (CF), including the potential to aid mucus clearance, improve lung function, exercise capacity and quality of life. Despite the recommendations from consensus documents for CF adults to engage in regular physical activity, exercise participation amongst this population is often very low. No in-depth study has been undertaken to explore the determinants of exercise participation for this group and no studies have examined the benefits of habitual physical activity on the health status and quality of life of CF adults. As well, the current methods to quantify physical activity are problematic. The series of studies, involving CF adults, in this thesis was therefore undertaken in order to examine the physiological rationale for the use of exercise as an airway clearance technique, to understand the decision making process to engage in exercise, to determine if health status and quality of life were affected by exercise participation, and to establish the accuracy of a recently-developed objective measure of physical activity. The study in Chapter 2 provided some physiological rationale for choosing treadmill exercise to aid airway clearance in CF. The main findings were that a single bout of moderate intensity exercise increased the subjective ease of expectoration, most likely due to the increased ventilation with exercise, and that sputum viscoelasticity was favourably decreased immediately following treadmill exercise compared to cycle exercise and control. The studies in Chapters 3 and 4 identified the main beliefs regarding exercise for CF adults and highlighted that the main predictors of exercise intention and participation for this group were aspects of perceived and actual control to exercise, namely self-efficacy or confidence to exercise, feeling healthy, receiving encouragement to exercise, and rating exercise as a high priority treatment. Positive ratings of these aspects of control either increased exercise participation directly, indirectly by increasing intention, or strengthened the conversion of exercise intention to participation. Strategies aimed at targeting these aspects of control are therefore likely to be effective in increasing exercise participation for CF adults. The study in Chapter 5 demonstrated that CF adults, who reportedly performed at least 90 minutes of moderate to strenuous exercise per week, had significantly higher quality of life and fewer days in hospital over the following year than their peers, who exercised less. The difference in hospitalisation between the CF adults, who reportedly exercised more than 90 minutes per week and those who did not, was independent of baseline lung function, and the females who reportedly performed less than 90 minutes of exercise per week had three times as many days in hospital than their high-activity peers. The study in Chapter 6 showed that the SenseWear Pro3 Armband activity monitor provided a reasonable estimate of energy expenditure and step count. Also, its accuracy was not affected by diagnosis with CF, despite the potential for the high salt content in the sweat to interfere with the device’s physiological sensors placed on the skin. Overall, this series of studies adds to the growing evidence of the physical and psychological benefits from exercise participation for CF adults, as well as providing some empirical evidence upon which to base strategies to improve exercise participation for this group and support for an objective measure of physical activity.
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Kahre, Tiina. "Cystic fibrosis in Estonia /." Online version, 2004. http://dspace.utlib.ee/dspace/bitstream/10062/577/5/KahrePhD.pdf.

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Utley, Courtney, and Kristen L. McHenry. "Advances in Cystic Fibrosis." Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etsu-works/2546.

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The purpose of this review was to identify the history of and advances in cystic fibrosis (CF). New treatment plans, medication developments, and a historical perspective of airway clearance therapy (ACT) will be presented. The importance of treatment compliance and time management in the care of cystic fibrosis patients will also be discussed. Furthermore, the development of cystic fibrosis clinics and the pivotal role they play in the treatment of the disease will be addressed. Lastly, a brief discussion concerning the need for and process of lung transplantation will be reported.
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Dobson, Lee. "Glucose tolerance in cystic fibrosis." Thesis, University of Exeter, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.403679.

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Hurley, Matthew. "Lung infection in cystic fibrosis." Thesis, University of Nottingham, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.716679.

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Cystic fibrosis (CF) is characterised by viscid secretions that, in the lungs, pre-dispose to infection. Many people with CF experience 'pulmonary exacerbations' accompanied by intermittent deteriorations in lung function. Pulmonary exacerbations are associated with a more rapid decline in lung health over time and are associated with infection. Early infection managed aggressively may be successfully eradicated. With successive infections and cumulative lung damage, chronic infection is established. Chronic infection leads to progressive decline in lung function with associated reductions in quality of life and increased treatment burden, morbidity and mortality. Naturally antibiotic-tolerant organisms that evolve resistance to commonly used antibiotics are problematic, in particular Pseudomonas aeruginosa. As is the case with other organisms that cause lung infection in those with CF, P. aeruginosa's antibiotic tolerance and ability to establish chronic infection is thought to be conferred through the biofilm mode of growth. The adaptability of the organism to pressures exerted by antibiotics and competition within the lung environment promote antibiotic resistance. New strategies effective in preventing and managing chronic infection are likely to yield improvement in survival and quality of life, however the development of such agents is yet to materialise.
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Downey, D. G. "Airways inflammation in cystic fibrosis." Thesis, Queen's University Belfast, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269047.

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Evans, Katharine Sarah Emily. "Cystic Fibrosis and the eye." Thesis, Cardiff University, 2009. http://orca.cf.ac.uk/54848/.

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Cystic Fibrosis (CF) results from the defective function of CF Transmembrane Conductance Regulator (CFTR), an ion channel which facilitates epithelial chloride secretion. Previous observations of dry eye and abnormal visual function in CF subjects have been considered secondary manifestations due to associated vitamin A deficiency (VAD) and CF-related diabetes (CFRD). However, CFTR is fundamentally present in the corneal, conjunctival and retinal pigment epithelium and the corneal endothelium. The hypothesis for this thesis was that abnormal chloride secretion in CF causes reduced basal tear secretion and abnormal photoreceptor function: these investigations aimed to identify primary and secondary ocular manifestations of CF. Fluorescein tear break-up time was significantly reduced in adult CF subjects compared to healthy controls. Increased signs of ocular surface inflammation and higher tear feming grades were recorded in CF subjects, although differences failed to reach significance. Tear film stability was further reduced in CF adults with VAD suggesting the aetiology of dry eye appears to be a combination of primary and secondary manifestations of the disease. Visual function was essentially normal in CF juveniles but was adversely affected in CF adults compared to controls. Impaired distance and near visual acuity (DVA and NVA), contrast sensitivity (CS), dark adaptation (DA) and colour vision (CV) appeared to be a primary manifestation as differences were exaggerated in subjects with predicted increased levels of CFTR disruption and disease severity. These results provide support for the hypothesis and suggest normal rod and cone photoreceptor function are compromised by abnormal CFTR action. DVA, NVA, CS and DA were significantly affected by CFRD status and DA and CV were similarly reduced in VAD subjects. Therefore, abnormal visual function in CF is further modulated by secondary disease characteristics. These findings present the distinction between primary and secondary ocular manifestations of CF, which is novel to this investigation.
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Wright, Adam. "The macrophage in cystic fibrosis." Thesis, University of Leicester, 2007. http://hdl.handle.net/2381/8783.

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Background: Cystic fibrosis (CF) is caused by absent/defective CF transmembrane conductance regulator protein (CFTR). CF is characterized by thick airway mucus, chronic infection and neutrophil inflammation leading to respiratory failure. I analysed airway macrophages (MΦs) and their expression of pattern recognition receptors (PRRs) in CF, since these cells are crucial to airway immune defence and they can orchestrate inflammation. I also performed transcript analysis of CF monocyte-derived MΦs (MDMs). Methods: Sputum was induced in CF paediatric and adult cohorts. Phenotype and function of CF MΦ were determined by flow cytometry and compared to controls. Monocytes (>92% purity) were grown in vitro to generate MDMs (n=15). Transcripts encoding the entire human genome were analysed (n=5) and expression of individual genes were confirmed by RT-PCR. Results: In classical CF (n=10) there was an increase in the proportion of monocyte-like small MΦs (of total MΦ) compared to controls (n=10) (73 ± 18% and 16 ± 8%, respectively, p< 0.0001). In non-classical CF (n=4), with milder lung disease, small MΦs increased to 31 ± 20% (p>0.05). PRRs were absent on small MΦs from CF and control. In contrast, clear expression could be detected on large MΦs from control but not CF. In line with this, CF small MΦs showed a strongly reduced uptake of particles compared to controls. Microarray analysis of MDMs revealed α- and β-tryptase as being significantly higher under constitutive and stimulated conditions in CF compared to control. However, using RT-PCR, expression of α- and β-tryptase was similar between groups. Conclusions: The phenotype of small MΦs in CF suggests that these cells are newly recruited monocytes from blood. Low expression of PRRs on these cells in CF and their reduced uptake indicates a reduced capacity to clear inhaled particles, which may contribute to further damage in CF. Further to this I was unable to confirm any transcript differences between CF and control MDMs due to mutant CFTR.
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Books on the topic "Cystic Fibrosis in adulthood"

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Cystic fibrosis. Oxford: Oxford University Press, 2010.

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Cystic fibrosis. Detriot: Lucent Books, 2013.

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Giddings, Sharon. Cystic fibrosis. New York: Chelsea House, 2009.

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Rosaler, Maxine. Cystic fibrosis. New York: The Rosen Pub. Group, 2007.

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Bjorklund, Ruth. Cystic fibrosis. New York: Marshall Cavendish Benchmark, 2009.

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E, Hodson Margaret, Geddes Duncan M, and Bush, Andrew, 1954 Apr. 24-, eds. Cystic fibrosis. 3rd ed. London: Hodder Arnold, 2007.

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Capewell, Gillian. Cystic fibrosis. London: Office of Health Economics, 1986.

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Silverstein, Alvin. Cystic fibrosis. New York: F. Watts, 1994.

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Mandolfo, Anna. Cystic fibrosis. Southampton: Dulphar Laboratories, 1991.

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Davis, Stephanie Duggins, Margaret Rosenfeld, and James Chmiel, eds. Cystic Fibrosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42382-7.

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Book chapters on the topic "Cystic Fibrosis in adulthood"

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Akong, Kathryn, and Meerana Lim. "Cystic Fibrosis." In Textbook of Clinical Pediatrics, 2209–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-02202-9_237.

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Cantin, André M. "Cystic Fibrosis." In Molecular Basis of Pulmonary Disease, 339–68. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-59745-384-4_16.

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Griesenbach, Uta, Duncan M. Geddes, and Eric W. F. W. Alton. "Cystic Fibrosis." In Molecular Biology of the Lung, 181–98. Basel: Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8831-8_11.

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Brown, Sarah, and Ian M. Balfour-Lynn. "Cystic Fibrosis." In Pediatric Thoracic Surgery, 427–43. London: Springer London, 2009. http://dx.doi.org/10.1007/b136543_35.

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Dodge, John A. "Cystic Fibrosis." In Clinical Nutrition, 477–91. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119211945.ch24.

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Heim, Ruth A. "Cystic Fibrosis." In Diagnostic Molecular Pathology in Practice, 3–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19677-5_1.

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Gilbert, Patricia. "Cystic fibrosis." In The A-Z Reference Book of Syndromes and Inherited Disorders, 84–88. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-6918-7_22.

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Dawson, R. L. "Cystic Fibrosis." In Teacher Information Pack 4: Physical, 37–44. London: Macmillan Education UK, 1985. http://dx.doi.org/10.1007/978-1-349-09003-7_5.

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Oates, Gabriela R., and Michael S. Schechter. "Cystic Fibrosis." In Respiratory Medicine, 97–112. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-43447-6_9.

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Dinwiddie, R. "Cystic Fibrosis." In Pancreatic Disease, 323–31. London: Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-3356-8_28.

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Conference papers on the topic "Cystic Fibrosis in adulthood"

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Gartner, Silvia, Antonio Alvarez, Sandra Rovira, Javier De Gracia, Antonio Moreno, Teresa Casals, Paula Fernadez, et al. "Cystic fibrosis diagnosis in adulthood as a result of study of relatives within the newborn screening programme." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.oa1443.

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Low, TB, CM Greene, SJ O'Neill, and NG McElvaney. "Autoantibodies in Cystic Fibrosis." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2875.

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Modarezi, Mohammad Reza, Safura Navaie, Rouhollah Shirzadi, and Majd Keyvanfar. "Cystic Fibrosis in Iran." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4525.

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Degrugillier, Fanny, Xavier Decrouy, Virginie Prulière-Escabasse, Chong Jiang, Daniela Rotin, Pascale Fanen, and Stéphanie Simon. "HspB5 in Cystic Fibrosis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa3421.

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Barker, Billie J., and Mark K. Robbins. "Cystic Fibrosis Self-Diagnosis." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5896.

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Bar-Yoseph, Ronen, Anat Ilivitzki, Michal Gur, Gur Mainzer, Fahed Hakim, Galit Livnat, Zeev Schnapp, et al. "Exercise capacity in patients with cystic fibrosis vs non-cystic fibrosis bronchiectasis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa484.

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Purdy, C., J. Kanyinda, A. Hahn, B. Harkness, A. C. Koumbourlis, and I. R. Sami-Zakhari. "Uncommon Organisms in Cystic Fibrosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1916.

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Cook, D. P., C. Thomas, A. Wu, A. Norlander, D. A. Stoltz, and R. S. Peebles. "Th2 Dysfunction in Cystic Fibrosis." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1993.

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Murugesan, V., S. N. Mahmood, and E. Oweis. "Cystic Fibrosis: A National Study of Adult Patients Admitted for Cystic Fibrosis in 2014." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4636.

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Svetlana, Sciuca, Gatcan Stefan, Balanetchi Ludmila, and Cospormac Viorica. "P118 Cystic fibrosis influence on the fetus, newborn and infant child with cystic fibrosis." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.206.

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Reports on the topic "Cystic Fibrosis in adulthood"

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Garber, Alan, and Joseph Fenerty. Costs and Benefits of Prenatal Screening For Cystic Fibrosis. Cambridge, MA: National Bureau of Economic Research, October 1988. http://dx.doi.org/10.3386/w2749.

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Taub, Daniel, and Joshua Page. Cystic Fibrosis: Exploration of Evolutionary Explanations for the High Frequency of a Common Genetic Disorder. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), October 2013. http://dx.doi.org/10.1534/gsaprep.2013.004.

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Borisova, Dayana, Tanya Strateva, Tsvetelina Paunova-Krasteva, Ivan Mitov, and Stoyanka Stoitsova. Phenotypic Investigation of Paired Pseudomonas aeruginosa Strains Isolated from Cystic Fibrosis Patients Prior- and Post-tobramycin Treatment. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, August 2018. http://dx.doi.org/10.7546/crabs.2018.08.05.

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Barros-Poblete, Marisol, Rodrigo Torres-Castro, Mauricio Henríquez, Anita Guequen, Isabel Blanco, and Carlos Flores. Dysbiosis as a prognostic factor for clinical worsening in chronic respiratory disease: A systematic review and metanalysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0089.

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Review question / Objective: Is dysbiosis a prognostic factor for clinical worsening in patients with chronic respiratory diseases?. Condition being studied: Dysbiosis, defined as changes in the quantitative and qualitative composition of the microbiota. Eligibility criteria: Over 18 years old adult patients with chronic respiratory diseases clinical diagnosis (cystic fibrosis, chronic obstructive pulmonary disease, asthma, idiopathic pulmonary fibrosis, interstitial lung disease, sarcoidosis, bronchiectasis, non-CF bronchiectasis, pulmonary hypertension) according to the International Statistical Classification of Diseases and Related Health Problems (ICD) from OMS) and international guidelines of each disease.
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Journal articles Dissertations / Theses Books
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