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Journal articles on the topic 'Cushing's syndrome'

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Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2024

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1

Giugni, Aldo Schenone, Shylaja Mani, Subramanian Kannan, and Betul Hatipoglu. "Exophthalmos: A Forgotten Clinical Sign of Cushing's Syndrome." Case Reports in Endocrinology 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/205208.

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Exophthalmos is typically associated with Graves' ophthalmopathy. Although originally described by Harvey Cushing, exophthalmos is an underappreciated sign of Cushing's syndrome. We present a case of a 38-year-old female who presented with severe bilateral proptosis and was subsequently diagnosed with Cushings disease. We discuss the possible mechanisms causing exophthalmos in patients with either endogenous or exogenous hypercortisolemia.

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2

Arroyo, Regina G. G., Hannia I. P. Belmontes, Marlene de J. G. Torres, Brizza M. R. Mendoza, Begoña E. Gonzalez, Natalia C. Bonilla, Aran A. R. Ceja, and Gerardo G. Santiago. "Drug induced iatrogenic Cushing’s syndrome." International Journal of Research in Medical Sciences 12, no. 1 (December 28, 2023): 303–8. http://dx.doi.org/10.18203/2320-6012.ijrms20234026.

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Drug-induced (iatrogenic) Cushing's syndrome results from excessive or prolonged exposure to glucocorticoids, commonly prescribed for autoimmune, inflammatory, and hematological disorders due to their anti-inflammatory, immunosuppressive, and proapoptotic effects. Despite their therapeutic benefits, these medications can lead to a range of multisystemic symptoms mirroring those of endogenous Cushing’s syndrome. This review aims to elucidate the causes, clinical presentation, diagnosis, and management of iatrogenic Cushing's syndrome, emphasizing awareness of medications that can trigger its onset. The following review covers cortisol physiology, Cushing's syndrome etiology and subtypes, hypercortisolism complications and prognosis, and strategies for glucocorticoid withdrawal. This article synthesizes key findings and recommendations, highlighting challenges and controversies in the diagnosis and treatment of iatrogenic Cushing's syndrome.

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3

Kissane, Nicole A., and Juan C. Cendan. "Patients with Cushing's Syndrome are Care-Intensive Even in the Era of Laparoscopic Adrenalectomy." American Surgeon 75, no. 4 (April 2009): 279–83. http://dx.doi.org/10.1177/000313480907500402.

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We report outcomes from laparoscopic adrenalectomy (LA) comparing patients with Cushing's syndrome with those with other adrenal pathology with respect to length of stay (LOS), overall complications, and financial implications. We conducted a retrospective review of 80 continuous patients (103 glands) undergoing LA. The clinical diagnoses were: hypercortisolism (Cushing’ syndrome; n = 33), hyperaldosteronism (Conn's syndrome; n = 20), phaeochromocytoma (n = 16), and neoplasm (others; n = 11). Advanced care in the intermediate or intensive care unit was necessary in 27 patients, most frequently in our Cushing's population (16 of 33 [48%]). Six patients sustained major complications, including death in two patients; seven patients sustained minor complications. LOS was longer for patients with Cushing's syndrome (mean, 5.5 vs 3.3 days; P = 0.024). Financially, patients with Cushing's syndrome had statistically higher total hospital ( P = 0.009), advanced care ( P = 0.002), and anesthetic costs ( P = 0.005). LA in patients with Cushing's syndrome is associated with longer hospitalizations, more frequent major complications, and higher advanced care requirements, especially for patients undergoing bilateral adrenalectomy Minor complications were infrequent and median LOS was brief regardless of diagnosis. Patients with Cushing's syndrome had higher costs for overall hospital charges, advanced care, and anesthesia.

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4

Yanase, Toshihiko. "3) Cushing's Syndrome and Subclinical Cushing's Syndrome." Nihon Naika Gakkai Zasshi 107, Suppl (February 20, 2018): 115b—116a. http://dx.doi.org/10.2169/naika.107.115b.

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5

Yanase, Toshihiko. "3) Cushing's Syndrome and Subclinical Cushing's Syndrome." Nihon Naika Gakkai Zasshi 107, no. 9 (September 10, 2018): 1766–71. http://dx.doi.org/10.2169/naika.107.1766.

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6

Geçgel, Esra, Alper Alp, Emel Kılıçarslan Karpuzoğlu, Dilek Gibyeli Genek, and Bülent Huddam. "Uncommon presentation of small cell lung carcinoma with ectopic adrenocorticotropic hormone secretion and resistant hypokalemia: A case report." Ukrainian Journal of Nephrology and Dialysis, no. 1(81) (December 5, 2023): 3–9. http://dx.doi.org/10.31450/ukrjnd.1(81).2024.01.

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Paraneoplastic syndromes can serve as initial indicators of malignancy, with small cell lung cancer accounting for 13% of new lung cancer diagnoses. The most prevalent paraneoplastic syndrome associated with small cell lung cancer is inappropriate antidiuretic hormone syndrome, followed by ectopic adrenocorticotropic hormone-mediated Cushing’s syndrome. Cushing’s syndrome manifests as hypercortisolemia and presents with diverse symptoms, including central obesity, plethora, menstrual irregularities, hypertension/diabetes mellitus, ecchymoses, osteoporosis, muscle weakness, virilization/hirsutism, skin atrophy, decreased libido, and infertility. This case report details the uncommon presentation of small cell lung carcinoma manifesting with ectopic adrenocorticotropic hormone secretion (EAS), leading to resistant hypokalemia and rhabdomyolysis. This case emphasizes the importance of considering EAS in severe cases of Cushing's syndrome and highlights the diagnostic and therapeutic challenges associated with this condition.

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7

Pascual, José María, and Ruth Prieto. "Harvey Cushing and pituitary Case Number 3 (Mary D.): the origin of this most baffling problem in neurosurgery." Neurosurgical Focus 41, no. 1 (July 2016): E6. http://dx.doi.org/10.3171/2016.2.focus1592.

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From the very beginning of his career, Harvey Williams Cushing (1869–1939) harbored a deep interest in a complex group of neoplasms that usually developed at the infundibulum. These were initially known as “interpeduncular” or “suprasellar” cysts. Cushing introduced the term “craniopharyngioma” for these lesions, which he believed represented one of the most baffling problems faced by neurosurgeons. The patient who most influenced Cushing's thinking was a 16-year-old seamstress named “Mary D.,” whom he attended in December 1901, exactly the same month that Alfred Fröhlich published his seminal article describing an adiposogenital syndrome in a young boy with a pituitary cyst. Both Cushing's and Fröhlich's patients showed similar symptoms caused by the same type of tumor. Notably, Cushing and Fröhlich had met one another and became good friends in Liverpool the summer before these events took place. Their fortunate relationship led Cushing to realize that Fröhlich's syndrome represented a state of hypopituitarism and provided a useful method of diagnosing interpeduncular cysts. It is noteworthy that Cushing's very first neurosurgical procedure on a pituitary tumor was performed in the case of Mary D.'s “interpeduncular cyst,” on February 21, 1902. Cushing failed to remove this lesion, which was later found during the patient's autopsy. This case was documented as Pituitary Case Number 3 in Cushing's masterpiece, The Pituitary Body and Its Disorders, published in 1912. This tumor was considered “a teratoma”; however, multiple sources of evidence suggest that this lesion actually corresponded to an adamantinomatous craniopharyngioma. Unfortunately, the pathological specimens of this lesion were misplaced, and this prompted Cushing's decision to retain all specimens and documents of the cases he would operate on throughout his career. Accordingly, Mary D.'s case crystallized the genesis of the Cushing Brain Tumor Registry, one of Cushing's major legacies to neurosurgery. In this paper the authors analyze the case of Mary D. and the great influence it had on Cushing's conceptions of the pituitary gland and its afflictions, and on the history of pituitary surgery.

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8

Kelsall, Alan, and John Newell-Price. "Cushing's syndrome." Medicine 49, no. 8 (August 2021): 483–87. http://dx.doi.org/10.1016/j.mpmed.2021.05.006.

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9

Felicetta, James V. "Cushing's Syndrome." Postgraduate Medicine 86, no. 8 (December 1989): 79–90. http://dx.doi.org/10.1080/00325481.1989.11704497.

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10

Newell-Price, John. "Cushing's syndrome." Clinical Medicine 8, no. 2 (April 1, 2008): 204–8. http://dx.doi.org/10.7861/clinmedicine.8-2-204.

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11

Prague, J. K., S. May, and B. C. Whitelaw. "Cushing's syndrome." BMJ 346, mar27 3 (March 27, 2013): f945. http://dx.doi.org/10.1136/bmj.f945.

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12

Newell-Price, John. "Cushing's syndrome." Medicine 33, no. 11 (November 2005): 11–13. http://dx.doi.org/10.1383/medc.2005.33.11.11.

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13

Clayton, Laura H., and Kathy B. Dilley. "Cushing's Syndrome." American Journal of Nursing 98, no. 7 (July 1998): 40. http://dx.doi.org/10.1097/00000446-199807000-00040.

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14

Norton, Jeffrey A., Michelle Li, Jennifer Gillary, and Hop N. Le. "Cushing's syndrome." Current Problems in Surgery 38, no. 7 (July 2001): A3–545. http://dx.doi.org/10.1067/msg.2001.114088.

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15

Orth, David N. "Cushing's Syndrome." New England Journal of Medicine 332, no. 12 (March 23, 1995): 791–803. http://dx.doi.org/10.1056/nejm199503233321207.

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16

Newell-Price, John, Xavier Bertagna, Ashley B. Grossman, and Lynnette K. Nieman. "Cushing's syndrome." Lancet 367, no. 9522 (May 2006): 1605–17. http://dx.doi.org/10.1016/s0140-6736(06)68699-6.

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17

Munir, Alia, and John Newell-Price. "Cushing's syndrome." Medicine 37, no. 8 (August 2009): 403–6. http://dx.doi.org/10.1016/j.mpmed.2009.05.013.

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18

Daniel, Eleni, and John Newell-Price. "Cushing's syndrome." Medicine 41, no. 9 (September 2013): 508–11. http://dx.doi.org/10.1016/j.mpmed.2013.06.007.

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19

Daniel, Eleni, and John Newell-Price. "Cushing's syndrome." Medicine 45, no. 8 (August 2017): 475–79. http://dx.doi.org/10.1016/j.mpmed.2017.05.007.

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20

Boscaro, Marco, Luisa Barzon, Francesco Fallo, and Nicoletta Sonino. "Cushing's syndrome." Lancet 357, no. 9258 (March 2001): 783–91. http://dx.doi.org/10.1016/s0140-6736(00)04172-6.

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21

Giraldi, Francesca Pecori, Pietro Putignano, and Francesco Cavagnini. "Cushing's syndrome." Lancet 357, no. 9274 (June 2001): 2138. http://dx.doi.org/10.1016/s0140-6736(00)05212-0.

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22

Ioachimescu, Adriana G. "Cushing's Syndrome." Endocrinology and Metabolism Clinics of North America 47, no. 2 (June 2018): i. http://dx.doi.org/10.1016/s0889-8529(18)30505-x.

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23

Elliott, W. J. "Cushing's Syndrome." Yearbook of Cardiology 2007 (January 2007): 50–51. http://dx.doi.org/10.1016/s0145-4145(08)70028-5.

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24

Lacroix, André, Richard A. Feelders, Constantine A. Stratakis, and Lynnette K. Nieman. "Cushing's syndrome." Lancet 386, no. 9996 (August 2015): 913–27. http://dx.doi.org/10.1016/s0140-6736(14)61375-1.

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25

Howlett, T. A., L. H. Rees, and G. M. Besser. "Cushing's syndrome." Clinics in Endocrinology and Metabolism 14, no. 4 (November 1985): 911–45. http://dx.doi.org/10.1016/s0300-595x(85)80083-9.

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26

Barnett, Richard. "Cushing's syndrome." Lancet 388, no. 10045 (August 2016): 649. http://dx.doi.org/10.1016/s0140-6736(16)31280-6.

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27

Findling, James W., and Hershel Raff. "Cushing's Syndrome." Endocrinology and Metabolism Clinics of North America 34, no. 2 (June 2005): xiii—xv. http://dx.doi.org/10.1016/j.ecl.2005.01.015.

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28

Pivonello, Rosario, Maria Cristina De Martino, Monica De Leo, Gaetano Lombardi, and Annamaria Colao. "Cushing's Syndrome." Endocrinology and Metabolism Clinics of North America 37, no. 1 (March 2008): 135–49. http://dx.doi.org/10.1016/j.ecl.2007.10.010.

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29

Randeva, H. S., and P. M. G. Bouloux. "Cushing's syndrome." BMJ 320, Suppl S4 (April 1, 2000): 0004100. http://dx.doi.org/10.1136/sbmj.0004100.

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30

Shibli-Rahhal, Amal, Marta Van Beek, and Janet A. Schlechte. "Cushing's syndrome." Clinics in Dermatology 24, no. 4 (July 2006): 260–65. http://dx.doi.org/10.1016/j.clindermatol.2006.04.012.

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31

Hatipoglu, Betul A. "Cushing's syndrome." Journal of Surgical Oncology 106, no. 5 (June 27, 2012): 565–71. http://dx.doi.org/10.1002/jso.23197.

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32

Zopp, Stephanie, and Martin Reincke. "Cushing's syndrome." MMW - Fortschritte der Medizin 166, no. 6 (April 2024): 56–61. http://dx.doi.org/10.1007/s15006-024-3637-5.

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33

Chalaprawat, M. "Cushing's syndrome." Chulalongkorn Medical Journal 30, no. 11 (November 1986): 1137–53. http://dx.doi.org/10.58837/chula.cmj.30.11.9.

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34

Marova, E. I., and I. A. Voronkova. "Cyclic Cushing's syndrome." Problems of Endocrinology 56, no. 4 (August 15, 2010): 44–51. http://dx.doi.org/10.14341/probl201056444-51.

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The cyclic Cushing's syndrome is a rare disease characterized by multiple episodes of elevated cortisol levels alternating with periods of its normal secretion. The so-called hypercorticism cycles may be either regular or episodic with intercycle intervals as long as a few days to several years. Most researchers agree that the reliable diagnosis of cyclic Cushing's syndrome should be based on laboratory detection of 3 peaks and 2 falls of plasma cortisol level. Cyclic Cushing's syndrome may be either ACTH dependent or independent. A review of 65 verified cases indicates that this condition may be caused by pituitary corticotropinoma (54%), ectopic ACTH-producing tumour (26%), and adrenal tumour (roughly 11%). The cause of the disease remains uncertain in 9% of the patients. Pathophysiological mechanisms of cyclic Cushing's syndrome are poorly known. In certain cases of bilateral macronodular adrenal hyperplasia or adrenal corticosteroma, it may be associated with the presence of ectopic receptors or anomalous expression of normally located receptors. The majority of the patients presenting with cyclic Cushing's syndrome exhibit symptoms of classical hypercorticism that manifest themselves either on a permanent or cyclic basis. In a small number of patients, clinical signs of cyclic Cushing's syndrome are virtually absent. Variations of the clinical picture and conflicting results of hormonal assays taken together make cyclic Cushing's syndrome difficult to diagnose. Therefore, physicians must be aware of this condition and actively search for it in all patients believed to have an enhanced cortisol production despite normal results of laboratory analysis. Frequent changes of urinary or salivary free cortisol levels are reliable and convenient criteria for cyclic Cushing's syndrome in patients suspected to have this condition. Results of cortisol stimulation or suppression tests are likely to lead to a false conclusion due to spontaneous falls and rises in serum cortisol levels at the time of analysis. Given laboratory confirmation of cyclic Cushing's syndrome, subsequent studies should be focused on the elucidation of its cause. The average life expectancy of patients with cyclic Cushing's syndrome remains to be determined.

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35

O'Hare, J. P., J. A. Vale, S. Wood, and R. J. M. Corrall. "Factitious Cushing's syndrome." Acta Endocrinologica 111, no. 2 (February 1986): 165–67. http://dx.doi.org/10.1530/acta.0.1110165.

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Abstract. In this report we describe a patient who produced the clinical and laboratory features of Cushing's syndrome by injecting himself for many months with hydrocortisone. Factitious Cushing's syndrome is a variant of the Münchausen syndrome and should be considered if tests used to establish the diagnosis of Cushing's syndrome appear incongruous particularly in subjects with access to medical intelligence and drugs.

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36

Birtolo, Maria Francesca, Roberta Armignacco, Nesrine Benanteur, Bertrand Baussart, Chiara Villa, Daniel De Murat, Laurence Guignat, et al. "Whole blood transcriptomic signature of Cushing's syndrome." European Journal of Endocrinology 191, no. 1 (July 2024): 55–63. http://dx.doi.org/10.1093/ejendo/lvae083.

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Abstract Objective Cushing's syndrome is characterized by high morbidity and mortality with high interindividual variability. Easily measurable biomarkers, in addition to the hormone assays currently used for diagnosis, could reflect the individual biological impact of glucocorticoids. The aim of this study is to identify such biomarkers through the analysis of whole blood transcriptome. Design Whole blood transcriptome was evaluated in 57 samples from patients with overt Cushing's syndrome, mild Cushing's syndrome, eucortisolism, and adrenal insufficiency. Samples were randomly split into a training cohort to set up a Cushing's transcriptomic signature and a validation cohort to assess this signature. Methods Total RNA was obtained from whole blood samples and sequenced on a NovaSeq 6000 System (Illumina). Both unsupervised (principal component analysis) and supervised (Limma) methods were used to explore the transcriptome profile. Ridge regression was used to build a Cushing's transcriptome predictor. Results The transcriptomic profile discriminated samples with overt Cushing's syndrome. Genes mostly associated with overt Cushing's syndrome were enriched in pathways related to immunity, particularly neutrophil activation. A prediction model of 1500 genes built on the training cohort demonstrated its discriminating value in the validation cohort (accuracy .82) and remained significant in a multivariate model including the neutrophil proportion (P = .002). Expression of FKBP5, a single gene both overexpressed in Cushing's syndrome and implied in the glucocorticoid receptor signaling, could also predict Cushing's syndrome (accuracy .76). Conclusions Whole blood transcriptome reflects the circulating levels of glucocorticoids. FKBP5 expression could be a nonhormonal marker of Cushing's syndrome.

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37

SHEELER, L. R. "Cushing's syndrome--1988." Cleveland Clinic Journal of Medicine 55, no. 4 (July 1, 1988): 329–37. http://dx.doi.org/10.3949/ccjm.55.4.329.

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38

Terzolo, Massimo, Silvia Bovio, Anna Pia, Giangiacomo Osella, Giorgio Borretta, Alberto Angeli, and Giuseppe Reimondo. "Subclinical Cushing's syndrome." Arquivos Brasileiros de Endocrinologia & Metabologia 51, no. 8 (November 2007): 1272–79. http://dx.doi.org/10.1590/s0004-27302007000800013.

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Subclinical Cushing's syndrome (CS) is attracting increasing interest since the serendipitous discovery of an adrenal mass has become a rather frequent event owing to the routine use of sophisticated radiologic techniques. Cortical adenoma is the most frequent type of adrenal incidentaloma accounting for approximately 50% of cases in surgical series and even greater shares in medical series. Incidentally discovered adrenal adenomas may secrete cortisol in an autonomous manner that is not fully restrained by pituitary feedback, in 5 to 20% of cases depending on study protocols and diagnostic criteria. The criteria for qualifying subclinical cortisol excess are controversial and presently there is no consensus on a gold standard for the diagnosis of this condition. An increased frequency of hypertension, central obesity, impaired glucose tolerance, diabetes and hyperlipemia has been described in patients with subclinical CS; however, there is still no clear demonstration of the long-term complications of this condition whose management remains largely empirical. Either adrenalectomy or careful observation associated with treatment of the metabolic syndrome have been suggested as treatment options.

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39

COOK, DAVID M., and A. WAYNE MEIKLE. "Factitious Cushing's Syndrome." Journal of Clinical Endocrinology & Metabolism 61, no. 2 (August 1985): 385–87. http://dx.doi.org/10.1210/jcem-61-2-385.

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40

Rijnberk, A., P. J. Kinderen, and J. H. H. Thijssen. "Canine Cushing's Syndrome." Zentralblatt für Veterinärmedizin Reihe A 16, no. 1 (May 13, 2010): 13–28. http://dx.doi.org/10.1111/j.1439-0442.1969.tb01035.x.

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41

Ismail, A. A. A., and P. L. Walker. "Diagnosing Cushing's Syndrome." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 35, no. 2 (March 1998): 324–25. http://dx.doi.org/10.1177/000456329803500223.

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42

Reincke, Martin. "SUBCLINICAL CUSHING'S SYNDROME." Endocrinology and Metabolism Clinics of North America 29, no. 1 (March 2000): 43–56. http://dx.doi.org/10.1016/s0889-8529(05)70115-8.

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43

Flohr, Felix, and Helene Geddert. "Ectopic Cushing's Syndrome." New England Journal of Medicine 365, no. 25 (December 22, 2011): e46. http://dx.doi.org/10.1056/nejmicm1010540.

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44

Terzolo, Massimo, Giuseppe Reimondo, Silvia Bovio, and Alberto Angeli. "Subclinical Cushing's Syndrome." Pituitary 7, no. 4 (December 2004): 217–23. http://dx.doi.org/10.1007/s11102-005-4024-6.

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45

Ambrosi, Bruno, and Alessandro Sartorio. "Preclinical Cushing's syndrome." Trends in Endocrinology & Metabolism 7, no. 3 (April 1996): 113–14. http://dx.doi.org/10.1016/1043-2760(96)00031-8.

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46

Hodge, Beth O. "Familial Cushing's Syndrome." Archives of Internal Medicine 148, no. 5 (May 1, 1988): 1133. http://dx.doi.org/10.1001/archinte.1988.00380050137020.

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47

Tsinberg, Michael, Chienying Liu, and Quan-Yang Duh. "Subclinical Cushing's syndrome." Journal of Surgical Oncology 106, no. 5 (April 25, 2012): 572–74. http://dx.doi.org/10.1002/jso.23143.

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48

Thynne, Tilenka, Graham H. White, and Morton G. Burt. "Factitious Cushing's syndrome masquerading as Cushing's disease." Clinical Endocrinology 80, no. 3 (October 25, 2013): 328–32. http://dx.doi.org/10.1111/cen.12343.

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49

Rao, D. "Reversible Cardiomyopathy in Cushing Syndrome-A Rare Presentation." Indian Journal of Cardiovascular Disease in Women WINCARS 01, no. 03 (September 2016): 035–38. http://dx.doi.org/10.1055/s-0038-1656478.

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AbstractCushing syndrome (CS) is the result of extended exposure to excessive glucocorticoids from endogenous or exogenous sources. Cushing's syndrome (CS) is associated with reduced life quality and increased mortality, mostly due to cardiovascular disease. We report the case of a 26-year-old female patient who presented with heart failure who was found to have Cushing syndrome and recovered after supportive treatment.

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50

Chowdhury, Rezaul Haider, Sumon Rahman Chowdhury, Tasnuva Tanzil, and Md Minhazul Alam. "Ectopic Cushing’s Syndrome From Prostatic Adenocarcinoma: A Rare Clinical Entity." Journal of Chittagong Medical College Teachers' Association 31, no. 1 (June 1, 2020): 125–29. http://dx.doi.org/10.3329/jcmcta.v31i1.66752.

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A 61-year-old man known to have metastatic prostate adenocarcinoma was seen at Changi General Hospital, Singapore, because of severe hypokalaemia due to ACTH dependent Cushing’s syndrome. He underwent a Dotate PET CT which showed increased DOTA-NOC-avidity in the right side of the prostate gland. Subsequent immunohistochemical staining of prostate biopsy sample documented ACTH, synaptophysin and CD 56 positivity. He was suggested medical management for prostate cancer complicated by Cushing's syndrome. Unfortunately, Cushing’s syndrome was not controlled and the patient’s clinical condition progressively worsened. Subsequently, he developed fatal sepsis due to immunocompromised state. This case report describes a case of Cushing’s syndrome due to metastatic adenocarcinoma of the prostate, a tumour with very few therapeutic options and negative prognosis. JCMCTA 2020 ; 31 (1) : 125-129

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