Academic literature on the topic 'Cushing's'
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Journal articles on the topic "Cushing's"
Giugni, Aldo Schenone, Shylaja Mani, Subramanian Kannan, and Betul Hatipoglu. "Exophthalmos: A Forgotten Clinical Sign of Cushing's Syndrome." Case Reports in Endocrinology 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/205208.
Full textPascual, José María, and Ruth Prieto. "Harvey Cushing and pituitary Case Number 3 (Mary D.): the origin of this most baffling problem in neurosurgery." Neurosurgical Focus 41, no. 1 (July 2016): E6. http://dx.doi.org/10.3171/2016.2.focus1592.
Full textTubbs, R. Shane, Neal Patel, Brian Vala Nahed, Aaron A. Cohen-Gadol, and Robert J. Spinner. "Reflections on the contributions of Harvey Cushing to the surgery of peripheral nerves." Journal of Neurosurgery 114, no. 5 (May 2011): 1442–48. http://dx.doi.org/10.3171/2010.11.jns10804.
Full textEllis, Harold. "Harvey Cushing: Cushing's Disease." Journal of Perioperative Practice 22, no. 9 (September 2012): 298–99. http://dx.doi.org/10.1177/175045891202200906.
Full textHaubrich, William S. "Cushing of Cushing's ulcers." Gastroenterology 119, no. 1 (July 2000): 22. http://dx.doi.org/10.1016/s0016-5085(00)70076-x.
Full textSeymour, Zachary A., and Aaron A. Cohen-Gadol. "Cushing's lost cases of “radium bomb” brachytherapy for gliomas." Journal of Neurosurgery 113, no. 1 (July 2010): 141–48. http://dx.doi.org/10.3171/2009.11.jns091393.
Full textMehta, Vivek A., Olindi Wijesekera, Courtney Pendleton, Alfredo Quiñones-Hinojosa, George I. Jallo, and Edward S. Ahn. "Harvey Cushing and “birth hemorrhage”: early pediatric neurosurgery at The Johns Hopkins Hospital." Journal of Neurosurgery: Pediatrics 8, no. 6 (December 2011): 647–53. http://dx.doi.org/10.3171/2011.9.peds11198.
Full textCarey, Michael E. "Major Harvey Cushing's difficulties with the British and American armies during World War I." Journal of Neurosurgery 121, no. 2 (August 2014): 319–27. http://dx.doi.org/10.3171/2014.5.jns122285.
Full textWahl, Christopher J., R. Shane Tubbs, Dennis D. Spencer, and Aaron A. Cohen-Gadol. "Harvey Cushing as a book collector, bibliophile, and archivist: the precedence for the genesis of the Brain Tumor Registry." Journal of Neurosurgery 111, no. 5 (November 2009): 1091–95. http://dx.doi.org/10.3171/2008.7.jns08511.
Full textGreenblatt, Samuel H. "The image of the “brain surgeon” in American culture: the influence of Harvey Cushing." Journal of Neurosurgery 75, no. 5 (November 1991): 808–11. http://dx.doi.org/10.3171/jns.1991.75.5.0808.
Full textDissertations / Theses on the topic "Cushing's"
Ritchie, Catherine Marian. "Aspects of hypertension in endocrine disease." Thesis, Queen's University Belfast, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357488.
Full textMunir, Alia. "RNA interference as therapy in a model of Cushing's disease." Thesis, University of Sheffield, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.577520.
Full textGroenewald, Liechka. "The prevalence of sleep apnea in patients with Cushing's syndrome." Diss., University of Pretoria, 2020. http://hdl.handle.net/2263/77081.
Full textDissertation (MSc)--University of Pretoria, 2020.
Physiology
MSc
Restricted
McNicol, A. M. "Pathology of pituitary corticotrophs : Clinical and experimental studies." Thesis, University of Glasgow, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.372421.
Full textPires, Encuentra Patricia. "Anomalías de la sustancia blanca cerebral en el síndrome de cushing en relación a sintomatología psiquiátrica y rendimiento cognitivo." Doctoral thesis, Universitat Autònoma de Barcelona, 2017. http://hdl.handle.net/10803/457523.
Full textCushing’s syndrome (CS) is an endocrine disorder due to prolonged exposure to cortisol. This endogenous hypercortisolism determines endocrine, cognitive and psychiatric symptoms. A few imaging studies have investigated brain structures in patients with CS and have observed brain damage. The aims of the studies described in this thesis were to explore the microstructure of cerebral white matter (WM), a topic hardly investigated up to now in CS, and to evaluate neuropsychiatric symptoms (depression and anxiety) and cognitive impairment (information processing speed) and their relationship with the WM. Diffusion Tensor Imaging (DTI) is a Magnetic Resonance (MR) imaging technique that allows noninvasive, in vivo study of the brain by assessing the motion of water molecules along and across neural axons. Different DTI maps show WM architecture and can depict microstructural WM abnormalities. Fractional anisotropy (FA) reflects WM integrity, while increases in mean diffusivity (MD) may be caused by demyelination or edema. Decreases in axial diffusivity (AD) indicate axonal loss, while increased radial diffusivity (RD) is related to demyelination. Thirty-five patients with CS and 35 healthy control volunteers were included in these studies. In the first study, a DTI analysis was performed comparing patients with CS with healthy controls, followed by a second analysis in which patients were separated into active CS, remitted CS, and cured CS. Results revealed widespread reductions in whole brain FA, increases in MD and RD and partially also of AD in patients with CS compared to healthy controls. The subgroup analysis demonstrated this same pattern of WM damage in patients with active, remitted and cured CS, suggesting an early onset and persistent damage of brain WM microstructure. DTI alterations seemed to be independent of concomitant hypercortisolism and the presence or absence of other cardiovascular risk factors included into the analysis like hypertension, dyslipidemia, and obesity. Our aim in the second study was to investigate mood disturbances and cognitive impairment in CS patients, and their relationship to WM alterations on DTI. We hypothesized that mood disturbances, evaluated using the Beck Depression Inventory II (BDI-II) and State-Trait Anxiety Inventory (STAI), would be greater in CS patients than in controls, and related to widespread WM alterations on DTI. We additionally hypothesized that CS patients would show abnormalities in information processing speed, evaluated using the Symbol Digit Modalities Test (SDMT), possibly related to WM alterations, since WM state has been related to connectivity function. Results showed greater mood disturbances (depression and anxiety symptoms), but no differences in information processing speed scores, in CS patients compared to healthy controls. In the case-control DTI study, depressive symptoms correlated negatively with FA and positively with RD, a pattern which is linked to loss of myelin and axonal integrity. FA and AD correlated positively with information processing speed. These results indicate that WM integrity are associated with better information processing in CS patients and controls.
Luzi, Nicole. "An Enzymology and Inhibition Study of a cAMP-Dependent Protein Kinase Linked to ACTH-Independent Cushing's Syndrome." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/6058.
Full textBatista, Sergio Luchini. "Osteoporose na doença de Cushing: valor preditivo da quantificação de adiposidade visceral e óssea sobre a remodelação e densidade mineral óssea." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-30032017-085813/.
Full textThe anti-inflammatory and immunosuppressive properties of glucocorticoids (GC) justify the use of these steroids in various clinical conditions, despite its significant adverse effects. Osteoporosis induced by glucocorticoids (OIG) is considered the most important cause of secondary osteoporosis. It is a multifactorial disease involving systemic, tissue and bone cell signaling changes. Furthermore, hypercortisolism is also associated with obesity, redistribution of fat, insulin resistance and diabetes mellitus. Interestingly, these metabolic disorders in which bone mass is preserved, there is increased bone fragility. In recent years, evidence shows various complex interaction between the mineral and energy metabolism, in particular between adipose tissue and bone. In this scenario, Cushing\'s disease (CD) is a desirable clinical model to evaluate various mechanisms involved in the complex process of developing osteoporosis. The objective of this study was to evaluate, in a baseline study and a prospective study, various aspects of the interaction between the mineral and energy metabolism in women with DC and their possible impact on bone mass, as well as the association between bone mass and different types of adipose tissue. In the baseline study, we evaluated three groups of individuals, matched by sex and age: control group (C, n = 27), obese (O; n = 16) and Cushing\'s disease group (CD, n = 16). In the prospective study, we evaluated the CD group at three time points: preoperative (Pre-op; n = 11), 6 months postoperative (6th month PO; n = 10) and 12 months postoperatively (12th month PO; n = 10). In the baseline study, the O and CD groups differed in relation to C as the weight and BMI (p <0.05). The CD group showed significantly higher blood glucose, insulin, glycosylated hemoglobin (HbA1c), HOMA-IR and leptin in relation to the C and O groups (p <0.05). Additionally, the CD group showed lower levels of osteocalcin in relation to the C and O groups (p <0.05) as well as PTH, 25-OH vitamin D (25 (OH) D), and adiponectin in relation to the C group (P <0.05). There was no difference between the groups regarding dosages of IGF-I and preadipocyte factor 1 (Pref-1). The CD group had lower bone mass in the lumbar spine in relation to the C and O groups (p <0.05) and lower bone mass in the total body when compared to the O group (P <0.05). The Trabecular bone score (TBS) was able to show impaired bone quality in groups O and CD, showing greater involvement in CD group (p <0.05). Bone marrow adiposity (BMA) in L3 was significantly higher in the CD group compared to the C and O groups (p <0.05). The CD group showed increased subcutaneous fat content (SAT), visceral (VAT), VAT/SAT ratio and intrahepatic lipid (IHL) in relation to the C group (p<0.05). Additionally, the CD group had a higher content of VAT in relation to the O group (p<0.05). Osteocalcin correlated positively with TBS (r = 0.5, p <0.0001) and negatively with HOMA-IR (r = -0.4, p <0.01) and AMO of L3 (r = - 0.4, p <0.01). The TBS was negatively correlated with HOMA-IR index (r = -0.6, p <0.0001) and AMO of L3 (r = - 0.5, p <0.001). The AMO of L3 positively correlated with BMI (r = 0.4, p <0.01), HOMA-IR (r = 0.3, p <0.05), leptin (r = 0.3, p < 0.05), VAT/SAT ratio (r = 0.6, p <0.0001) and IHL (r = 0.5, p <0.05). In the prospective study, there was a reduction in weight and BMI and blood glucose, insulin, HOMA-IR, glycated hemoglobin and leptin (p <0.05). Additionally, there was increased levels of 25(OH)D, osteocalcin and deoxypyridinoline (p <0.05). There were no significant differences between the levels of adiponectin and Pref-1. The TBS was stable and there was no occurance of new fractures by vertebral fracture assessment (VFA). In body composition by dual-energy X-ray absorptiometry (DXA), threre was a reduction of total fat mass and improvement in apendicular lean body mass index by Foundation for the National Institutes of Health (FNIH) (p <0.05). The BMA of L3 significantly reduced in the 6th month PO, remaining stable on the 12th month PO (p <0.05). There was a significant reduction of VAT, VAT/SAT ratio and IHL in the prospective follow-up (p <0.05). This study confirms previous data showing that endogenous hypercortisolism has a profound negative effect on the skeleton, in particular on trabecular bone. Moreover, it is the first study to show that there is a negative correlation between TBS with HOMA-IR and BMA; it is possible that changes in energy metabolism are at least partly responsible for the increased risk of fracture in DC.
Alencar, Guilherme Asmar. "Aspectos clínicos e moleculares da hiperplasia adrenal macronodular independente de ACTH em sua forma familial." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-03122013-091817/.
Full textINTRODUCTION: ACTH-independent macronodular adrenal hyperplasia (AIMAH) is a rare disease characterized by functioning adrenal macronodules and increased, autonomous and sustained cortisol production. This condition is an uncommon cause of Cushing\'s syndrome (CS). While the sporadic form of the disease appears to be the most frequent, the true prevalence of its familial form is unknown. Despite being a known clinical entity for almost 50 years, the pathophysiological process that leads to AIMAH, the predisposing genetic alterations and important clinical, laboratory and radiological aspects of the disease have not been fully clarified. The recent identification of a large group of relatives with familial AIMAH allowed the accomplishment of the present study. OBJECTIVES: The following were the aims of this study: 1) characterize the development of familial AIMAH through correlations between clinical manifestations, laboratory data and radiological findings; 2) investigate the possible association between AIMAH and the occurrence of intracranial meningioma; 3) characterize the metabolic activity of the adrenal glands in this disease; 4) define the inheritance pattern of the disease in the family studied; and 5) map chromosomal regions and loci potentially related to the genetic etiology of familial AIMAH. METHODS: 96 members of the family studied were initially subjected to a detailed clinical and laboratory evaluation. Computed tomography (CT) scans were performed for the radiological characterization of the adrenal glands. Magnetic resonance imaging scans and 18F-fluorodeoxyglucose positron emission tomography/computed tomography (18F-FDG-PET/CT) scans were performed on patients with both forms of the disease (familial and sporadic) to investigate the presence of intracranial meningioma and characterize the metabolic activity of the adrenal glands, respectively. In vivo studies for aberrant hormone receptors were also conducted on those patients with familial AIMAH. In another phase of the study, different molecular biology techniques were employed to investigate the genetic etiology of familial AIMAH. For such, sequencing of the ACTH receptor gene (MC2R), a linkage study using specific microsatellite markers, a single nucleotide polymorphism (SNP)-based genome-wide linkage study and the sequencing of suspect genes were performed. RESULTS: The evaluation of the family revealed the diagnosis of 15 cases of the disease (7 women and 8 men) in three consecutive generations. AIMAH was transmitted to subsequent generations by both genders and half of the siblings were affected in some segments of the family. Mean age at diagnosis was 52.8 +-11.3 years (range: 32 to 74 years) and about 86% (12/14) of the patients exhibited subclinical CS. Both midnight salivary cortisol and 24-hour urinary cortisol demonstrated low sensitivity (21% and 14%, respectively) for the diagnosis of familial AIMAH. Plasma ACTH levels were low ( < 10 pg/ml) in 46% (5/11) of patients with the disease. In about 62% (8/13) of cases, serum dehydroepiandrosterone sulphate (DHEAS) levels were below the normal range. Simple logistic regression models revealed that the probability (odds ratio) of an individual having the disease in the family was greater in the presence of plethora, progressive weight gain or after the diagnosis of diabetes or prediabetes. Adrenal thickening associated with the presence of bilateral nodules was the most common radiological finding in familial AIMAH. However, radiological abnormalities were found in only one of the adrenal glands in one third of the patients (5/15). Throughout the in vivo studies for aberrant hormone receptors, distinct responses were frequently observed among the individuals with familial AIMAH. One third (5/15) of the patients who underwent magnetic resonance imaging scans had typical images of intracranial meningiomas. The 18F-FDG-PET/CT scan revealed increased metabolic activity of the hyperplastic adrenals in patients with both overt and subclinical CS. The molecular studies delimited genomic regions on chromosomes 16 and 11 potentially related to the genetic cause of familial AIMAH. Some suspected genes (GPR56, GPR97 and GPR114), located in these genomic regions, were sequenced, but no mutations were found. CONCLUSIONS: In the extended family studied, AIMAH followed an autosomal dominant pattern of inheritance and subclinical CS was the most common presentation of the disease. The 1 mg overnight dexamethasone suppression test proved to be the screening test of choice for the initial evaluation of patients suspected to have familial AIMAH, due mainly to the low sensitivity of midnight salivary cortisol and 24-hour urinary cortisol as screening tests. A normal level of plasma ACTH was a common laboratory finding in familial AIMAH. Low serum levels of DHEAS proved to be a relatively early finding associated with the subclinical CS determined by the disease. Adrenal CT scans revealed different radiological patterns among patients with familial AIMAH, with a fairly frequent rate of asymmetry between glands. The distinct responses observed throughout the in vivo studies for aberrant hormone receptors, among family members, favor the hypothesis that these receptors may be an epiphenomenon resulting from cell proliferation and dedifferentiation. An increased prevalence of intracranial meningioma was demonstrated in both the familial and sporadic forms of AIMAH. For the first time, it was shown that AIMAH may exhibit increased 18FFDG uptake on the PET/CT scan, similarly to adrenal carcinoma and metastasis. The main genomic regions potentially associated with familial AIMAH were delimited on chromosome 16 (16p12.1, 16p11.2, 16q12.1, 16q13 and 16q21) and chromosome 11 (11q23.1) (reference genome: NCBI36/hg18)
Crespo, Martín Iris. "Neuropsychological evaluation of patients with acromegaly and Cushing’s syndrome: Long-term effects." Doctoral thesis, Universitat Autònoma de Barcelona, 2016. http://hdl.handle.net/10803/392681.
Full textThe studies described in this thesis focus on the neuropsychological consequences in patients who have suffered Cushing’s syndrome (CS, due to glucocorticoid excess) and acromegaly (due to excess of growth hormone -GH-). It is known that prolonged exposure to glucocorticoids may have long-lasting adverse effects in CS patients who do not completely return to premorbid level of functioning and quality of life despite long-term cure. On the other hand, in acromegalic patients, potential long-lasting effects of GH on the brain affecting personality, cognition and behaviour have not been considered until recently. Making a choice is a common behavior in daily life and requires selecting one option among several alternative possibilities. The Iowa Gambling Task (IGT) is a measurement of decision making that mimics real-life risk taking situations since it involves uncertainty, reward and punishment. Frontal cortex has a key role in the IGT because it has the ability to coordinate processing among its millions of neurons in order to direct them towards future goals and advantageous decisions. The aims of these studies were to evaluate memory and decision making in patients with CS and acromegaly, and explore their relationship with frontal cortex and affective disorders. Thirty-five CS patients and thirty-five matched controls were evaluated using the IGT and 3Tesla magnetic resonance imaging (MRI) to assess frontal cortical thickness. Thirty-one patients with acromegaly and thirty-one healthy controls were evaluated using the IGT, Rey Auditory Verbal Learning Test (RAVLT), State-Trait Anxiety Inventory (STAI) and Beck Depression Inventory-II (BDI-II). In the first study, CS patients presented an altered decision-making strategy compared to controls, choosing a lower number of the safer cards and higher number of riskier cards. They showed more difficulties than controls to learn the correct profiles of wins and losses for each card group. In whole brain analysis, CS patients showed decreased cortical thickness in the left superior frontal cortex, left precentral cortex, left insular cortex, left and right rostral anterior cingulate cortex, and right caudal middle frontal cortex compared to controls. In the second study, acromegalic patients showed impairments in delayed verbal memory and more anxiety and depressive symptoms than controls. Regarding the IGT, acromegalic patients presented an altered decision-making strategy compared to controls, choosing a lower number of the safer cards and higher number of the riskier cards. Multiple correlations between anxiety and depressive symptoms and performance in memory and decision making were found. These studies show impaired decision making in both CS and acromegalic patients. They selected more disadvantageous choices than advantageous choices. Whereas healthy controls gradually learned to favor the advantageous decks, patients with acromegaly and CS continued to experience large punishment throughout training. We have not found a direct relationship between poor decision making and frontal cortical thinning in CS patients, opposite to previous studies on other clinical conditions. Decision deficits on the IGT were correlated with memory deficits in CS and acromegalic patients. Moreover, acromegalic patients show affective alterations (anxiety and depressive symptoms) that influenced delayed memory and decision making.
González, Ramírez Mar 1991. "Bioinformatic analysis of epigenetic regulatory mechanisms in development and disease." Doctoral thesis, TDX (Tesis Doctorals en Xarxa), 2021. http://hdl.handle.net/10803/671370.
Full textUna regulació apropiada de l’expressió gènica és necessària per a un correcte desenvolupament i homeòstasi dels organismes. Els mecanismes epigenètics representen una informació addicional, a més de la seqüència genètica, crucial per al correcte funcionament de cada cèl·lula. Les modificacions d’histones, que modulen i s’associen a activació o repressió transcripcionals, són una característica epigenètica important. Gràcies al modelatge predictiu, hem estudiat quines modificacions d’histones es relacionen millor amb la funció dels enhancers o promotors en cèl·lules mare embrionàries de ratolí, durant la diferenciació i en el desenvolupament animal. Hem trobat que modificacions d’histones diferents es relacionen millor amb enhancers o promotors, respectivament. Hem estudiat el rol dels poised enhancers durant la diferenciació i el desenvolupament. Hem vist que l’activació dels poised enhancers no és exclusiva del llinatge neural, sinó un mecanisme implicat en la diferenciació de tot tipus cel·lular. Hem caracteritzat el paisatge epigenètic de la síndrome de Cushing. Hem trobat alteracions epigenètiques i transcripcionals després d’una remissió de la malaltia a llarg termini, relacionades amb una profunda alteració del ritme circadiari. Aquestes troballes prometen ser rellevants per a futurs avenços terapèutics.
Books on the topic "Cushing's"
Cushing's crusade. London: Faber and Faber, 2013.
Find full textK, Biller Beverly M., and SpringerLink (Online service), eds. Cushing's Disease. Boston, MA: Springer Science+Business Media, LLC, 2011.
Find full textBronstein, Marcello D., ed. Cushing's Syndrome. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60327-449-4.
Full textSwearingen, Brooke, and Beverly M. K. Biller, eds. Cushing's Disease. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0011-0.
Full textT, Hanson Scott, ed. The architecture of Cushing's Island. Cushing's Island, Me: Cushing's Island Association, 2012.
Find full textservice), SpringerLink (Online, ed. Cushing's Syndrome: Pathophysiology, Diagnosis and Treatment. Totowa, NJ: Springer Science+Business Media, LLC, 2011.
Find full textParker, James N., and Philip M. Parker. The official patient's sourcebook on Cushing's syndrome. San Diego, Calif: Icon Health Publications, 2002.
Find full textM, Gonchar A., Kulikov L. K, and Poli͡a︡nskiĭ B. A, eds. Giperfunkt͡s︡ii͡a︡ nadpochechnikov: Print͡s︡ipy i metody korrekt͡s︡ii. Novosibirsk: "Nauka", Sibirskoe otd-nie, 1988.
Find full textOne More for the People. Portland, OR: Perfect Day Publishing, 2011.
Find full textBrickner, Colleen. Inside out: An autobiography. Spokane, Wash: A.H. Clark Co., 1992.
Find full textBook chapters on the topic "Cushing's"
Castro, Margaret, Lucila Leico Elias, Paula Conde Lamparelli Elias, and Ayrton Custodio Moreira. "Physiology and Pathophysiology of the HPA Axis." In Cushing's Syndrome, 1–20. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_1.
Full textDunn, Ian F., and Edward R. Laws. "The Surgical Management of Cushing’s Disease." In Cushing's Syndrome, 121–29. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_10.
Full textYoung, William F., and Geoffrey B. Thompson. "Adrenal-Directed Treatment Options for Cushing’s Disease." In Cushing's Syndrome, 131–37. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_11.
Full textJagannathan, Jay, Edward R. Laws, and Jason P. Sheehan. "Radiation Therapy and Stereotactic Radiosurgery for Cushing’s Disease." In Cushing's Syndrome, 139–49. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_12.
Full textDang, Cuong, and Peter J. Trainer. "Medical Management of Cushing’s Syndrome." In Cushing's Syndrome, 151–62. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_13.
Full textAlexandraki, Krystallenia I., Andrea M. Isidori, and Ashley B. Grossman. "ACTH-Dependent Cushing Syndrome: Clinical and Diagnostic Aspects, and Treatment Approaches for Ectopic Cushing’s Syndrome." In Cushing's Syndrome, 163–76. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_14.
Full textAssie, Guillaume, Laurence Guignat, Jérôme Bertherat, and Xavier Bertagna. "Revisiting the Nelson’s Syndrome: Corticotroph Tumor Progression After Bilateral Adrenalectomy in Cushing’s Disease." In Cushing's Syndrome, 177–87. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_15.
Full textFragoso, Maria Candida Barisson Villares, Sorahia Domenice, Ana Claudia Latronico, and Berenice Bilharinho Mendonca. "ACTH-Independent Cushing’s Syndrome: Adrenocortical Tumors." In Cushing's Syndrome, 189–208. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_16.
Full textBourdeau, Isabelle, Antoine Lampron, Tânia Longo Mazzuco, and André Lacroix. "ACTH-Independent Cushing’s Syndrome: Bilateral Macronodular Hyperplasia." In Cushing's Syndrome, 209–24. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_17.
Full textStratakis, Constantine. "ACTH-Independent Cushing’s Syndrome: Primary Pigmented Nodular Adrenal Disease in the Context of Carney’s Complex." In Cushing's Syndrome, 225–34. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-449-4_18.
Full textConference papers on the topic "Cushing's"
Kolia, Nadeem R., Paul A. Gardner, Carl H. Snyderman, and Eric W. Wang. "Sinonasal Outcomes after Pituitary Surgery in Patient's with Cushing's Disease." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702408.
Full textMalenke, Jordan A., Justin Morse Lola Chambless, and Justin Turner. "Medically Refractory Cushing's Syndrome as a Paraneoplastic Manifestation of Esthesioneuroblastoma." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702715.
Full textSabahi, Mohammadmahdi, Taha Shahbazi, Seyed Farzad Maroufi, Kevin Vidal, Pablo Recinos, Varun Kshettry, Florian Roser, Badih Adada, and Hamid Borghei-Razavi. "MRI-Negative Cushing's Disease: A Systematic Review and Meta-Analysis." In 31st Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1743760.
Full textAnania, Yury, Venteicher S. Andrew, Pearce M. Thomas, and Gardner A. Paul. "Cyclic Cushing's Disease in Mixed Gangliocytoma–Pituitary Adenoma Tumor: Case Report." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702700.
Full textFaucz, Fabio R., Felix Beuschlei, Martin Fassnacht, Guilaume Assie, Davide Calebiro, Constantine Stratakis, Andrea Osswald, et al. "Abstract LB-182: Constitutive activation of PRKACA in adrenal Cushing's syndrome." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-lb-182.
Full textHubbard, Michael R., Hekmat Nasiri, Sudhir Rajan, and Sameer Arora. "Metastatic Pituitary Carcinoma As A Cause Of Cushing's Syndrome Associated Pneumocystis Pneumonia." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6103.
Full textHendarto, Hari. "Iatrogenic Cushing's syndrome caused by treatment with traditional herbal medicine, a case report." In 1st International Integrative Conference on Health, Life and Social Sciences (ICHLaS 2017). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/ichlas-17.2017.9.
Full textMcCormick, Justin P., Matthew Z. Sun, Iram Shafqat, Anthony P. Heaney, Marvin Bergsneider, and Marilene B. Wang. "Review of Venous Thromboembolic Prophylaxis in Cushing's Syndrome Patients Undergoing Endoscopic Transsphenoidal Surgery." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725301.
Full textPeeters, Sophie, Maya Harary, Andrew Wang, Krystal Karunungan, Alex Valenzuela, Won Kim, Marilene Wang, Jeffrey Suh, Jivianne Lee, and Marvin Bergsneider. "Pseudocapsular Dissection Improves Residual, Recurrence Rates and Time to Recurrence in Cushing's Disease Patients." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725359.
Full textChakrabarti, Jayati, Jennifer Eschbacher, Saptarshi Mallick, Kelvin W. Pond, Kevin Yuen, Curtis Thorne, Andrew S. Little, and Yana Zavros. "A Novel Human Pituitary Adenoma Organoid Model to Accelerate Targeted Treatment of Cushing's Disease." In 31st Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2022. http://dx.doi.org/10.1055/s-0042-1743680.
Full textReports on the topic "Cushing's"
Zhao, Nairui, Xinxin Yang, Cuiliu Li, Jie Ma, and Xiuping Yin. Efficacy and safety of pasireotide for Cushing's disease: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0070.
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