Academic literature on the topic 'Cryptococcus neoformans'
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Journal articles on the topic "Cryptococcus neoformans"
Mukherjee, Jean, Matthew D. Scharff, and Arturo Casadevall. "Cryptococcus neoformans infection can elicit protective antibodies in mice." Canadian Journal of Microbiology 40, no. 10 (October 1, 1994): 888–92. http://dx.doi.org/10.1139/m94-141.
Full textGaylord, Elizabeth A., Hau Lam Choy, and Tamara L. Doering. "Dangerous Liaisons: Interactions of Cryptococcus neoformans with Host Phagocytes." Pathogens 9, no. 11 (October 27, 2020): 891. http://dx.doi.org/10.3390/pathogens9110891.
Full textLev, Sophie, Cecilia Li, Desmarini Desmarini, David Liuwantara, Tania C. Sorrell, Wayne J. Hawthorne, and Julianne T. Djordjevic. "Monitoring Glycolysis and Respiration Highlights Metabolic Inflexibility of Cryptococcus neoformans." Pathogens 9, no. 9 (August 21, 2020): 684. http://dx.doi.org/10.3390/pathogens9090684.
Full textSilva, Vanessa K. A., Robin C. May, and Marcio L. Rodrigues. "Pyrifenox, an ergosterol inhibitor, differentially affects Cryptococcus neoformans and Cryptococcus gattii." Medical Mycology 58, no. 7 (January 8, 2020): 928–37. http://dx.doi.org/10.1093/mmy/myz132.
Full textMylonakis, Eleftherios, Roberto Moreno, Joseph B. El Khoury, Alexander Idnurm, Joseph Heitman, Stephen B. Calderwood, Frederick M. Ausubel, and Andrew Diener. "Galleria mellonella as a Model System To Study Cryptococcus neoformans Pathogenesis." Infection and Immunity 73, no. 7 (July 2005): 3842–50. http://dx.doi.org/10.1128/iai.73.7.3842-3850.2005.
Full textSong, Ying, Yufang Qiu, Weiyou Liu, and Xiaoliang Yuan. "Research progress in immunological mechanisms of Cryptococcus." Trends in Immunotherapy 5, no. 2.1 (October 22, 2021): 72. http://dx.doi.org/10.24294/ti.v5.i2.1.1370.
Full textKing, John W. "Cryptococcus neoformans." Emerging Infectious Diseases 14, no. 5 (May 2008): 762. http://dx.doi.org/10.3201/eid1405.0508.
Full textWarnock, David W. "Cryptococcus neoformans." Journal of Antimicrobial Chemotherapy 44, no. 1 (July 1999): 139. http://dx.doi.org/10.1093/jac/44.1.139.
Full textMayer, François L., and James W. Kronstad. "Cryptococcus neoformans." Trends in Microbiology 28, no. 2 (February 2020): 163–64. http://dx.doi.org/10.1016/j.tim.2019.10.003.
Full textRocco, Mark La. "Cryptococcus neoformans." Clinical Microbiology Newsletter 14, no. 23 (December 1992): 177–81. http://dx.doi.org/10.1016/0196-4399(92)90044-a.
Full textDissertations / Theses on the topic "Cryptococcus neoformans"
Mathoulin-Pelissier, Simone. "Les sérotypes de cryptococcus néoformans en pathologie humaine et dans l'environnement." Bordeaux 2, 1994. http://www.theses.fr/1994BOR23005.
Full textLaurenson, Ian F. "A study of Cryptococcus neoformans varieties gattii and neoformans." Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/22395.
Full textSyme, Rachel Mona. "Antigen processing and presentation of Cryptococcus neoformans." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0014/NQ34704.pdf.
Full textScofield, Melanie. "Heme utilization and storage by Cryptococcus neoformans." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/17425.
Full textAlanio, Alexandre. "Dynamique de l'adaptation de Cryptococcus neoformans à l'hôte." Paris 7, 2013. http://www.theses.fr/2013PA077182.
Full textCryptococcosis is an opportunistic infection due to the ubiquitous yeast Cryptococcus neoformans. This pathogen is a facultative intracellular organism. Interaction with immune cells including monocytes/macrophages lineage and dendritic cells are of major importance in the natural history of the infection. In humans, the pathophysiology of the infection evolves in three steps (i) primoinfection in childhood, (ii) dormancy, demonstrated from epidemiological and genotypic data in the lab few years ago (Garcia-Hermoso et al. 1999), (iii) reactivation upon immunosuppression. In terms of disease, clinical presentation and outcome of cryptococcosis are known to be diverse among individuals even those sharing the same underlying diseases. To address the question of the impact of fungal diversity on the natural course of the infection at the macrophage-level (standardized model of yeasts/ j774 macrophages in vitro interaction), murine model-level (murine model of cryptococcosis in OF1 outbred mice) and human-level (CryptoA/D database), we studied (i) the diversity of C. Neoformans/macrophages interactions using well characterized clinical isolates (ii) the correlation between in vitro phenotype of the isolate and clinical outcome in humans (iii) the diversity of adaptation to the host. We developed new assays and new tools using flow cytometry I (quantitative flow cytometry, multispectral imaging flow cytometry, sorting), microscopy (dynamic imaging), gene expression analysis (single-cell quantitative real time PCR) to overcome technical issues. We found high variation in phagocytic, 2 hours-, 48hours-intracellular proliferation indexes among the 54 ClinCn compared to H99. No correlation with the genotype was observed. The lack of sterilization at week 2 despite active antifungal therapy was significantly associated with a lower phagocytic index, whereas treatment failure at month 3 and death from cryptococcosis were significantly related to a higher 2 hours-intracellular proliferation. Among 9 selected clinical isolates compared to H99, (i) the virulence in mice was significantly different, intracellular expression of some virulence factors correlated with (ii) intracellular proliferation and (iii) phagocytic indexes. With a focus on multiplication and stress response and considering the H99 reference strain, we observed the appearance of various populations of yeasts during mice and macrophage infections. After sorting yeasts populations, we observed that a specific one was less prone and dependent of serum to grow compared to the other p'opulations. Gene expression analysis revealed that this population had specific metabolic characteristics that could reflect dormancy. We found a high diversity of C. Neoformans upon interaction with macrophages considering 54 clinical isolates in correlation with clinical outcome in humans, but also a considerable adaptation to host in our two models considering the reference strains H99. We observed also even more diversity of fungal adaptation to host when clinical isolates were considered. Ail together, these data suggest that cryptococcosis and fungal disease in general could be more complex diseases since diversity, plasticity and adaptation of the fungal organism to hosts is high and heterogeneous
Denoyelle, Patricia. "Evaluation comparative du fungitest R avec une méthode de microdilution pour l'étude de la sensibilité aux antifongiques d'isolats de Cryptococcus neoformans." Paris 5, 1999. http://www.theses.fr/1999PA05P056.
Full textVan, de Moer Ariane. "Production et caractérisation d'anticorps monoclonaux anti-Cryptococcus neoformans dirigés contre le galactoxylomannane de la paroi : application au diagnostic et au pronostic des cryptococcoses chez les patients atteints du syndrome de l'immunodéficience acquise." Montpellier 1, 1990. http://www.theses.fr/1990MON13504.
Full textHarrison, Thomas Stephen. "Interactions between Human Immunodeficiency Virus and Cryptococcus neoformans." Thesis, St George's, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299059.
Full textOliveira, Fabiana Freire Mendes de. "Análise funcional do gene ERG6 em Cryptococcus neoformans." reponame:Repositório Institucional da UnB, 2013. http://repositorio.unb.br/handle/10482/14252.
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O advento de pacientes portadores do HIV e de pacientes imunocomprometidos provocou um aumento na incidência de micoses fúngicas, como a criptococcose. Existem várias classes de antifúngicos disponíveis comercialmente para o tratamento dessas infecções que são utilizadas de acordo com a patologia e o patógeno. No entanto, são relatados casos de resistência a essas drogas e existe uma grande preocupação em relação aos efeitos adversos que elas podem causar nos pacientes. Nesse contexto, o desenvolvimento de novas drogas antifúngicas poderia ser uma solução para a problemática atual das drogas disponíveis e isso requer o estudo de novos alvos moleculares. O gene ERG6 codifica a enzima esterol C-24 metiltransferase que atua na conversão de zimosterol em fecosterol. Constitui uma etapa que ocorre em fungos para a biossíntese do ergosterol, mas não ocorre nos hospedeiros animais, que possuem enzimas específicas para a conversão do zimosterol até colesterol. No presente trabalho, a função do gene ERG6 foi investigada e caracterizada no fungo Cryptococcus neoformans demonstrando que a ausência desse gene gera diversas alterações fenotípicas, tais como sensibilidade ao estresse osmótico, ao estresse oxidativo e maior susceptibilidade a diferentes drogas antifúngicas. Além disso, foi ainda observado que a capacidade de crescer em meios contento diferentes estressores de parede se mostra alterada. Em relação aos fatores de virulência conhecidos para C. neoformans, o fungo foi incapaz de crescer a temperatura de 37°C, porém não teve sua produção de cápsula ou melanina afetadas. No entanto, em testes para observação da sua capacidade de virulência in vitro utilizando macrófagos e in vivo com lagartas da espécie Galleria mellonella mostraram uma redução na virulência em mutantes de ERG6. Por fim a análise dos esteróis de membrana mostrou que ocorre uma alteração em toda a composição de esteróis da membrana celular. Dessa forma, por ser uma enzima encontrada principalmente em fungos, a Erg6 pode ser um alvo molecular potencial para drogas antifúngicas. ______________________________________________________________________________ ABSTRACT
The advent of HIV patients and immunocompromised patients caused an increase in the incidence of fungal mycoses, such as cryptococcosis. There are several antifungal agents commercially available for the treatment of such infections that are chosen based on the disease and the pathogen. However, cases of resistance are reported to these drugs and there is great concern about the adverse effects that they can cause on patients. In this context, the development of new antifungal drugs could be a solution to the current problem of the available drugs and it requires the study of potential molecular targets. The gene ERG6 encodes the sterol C-24 methyltransferase, an enzyme that acts on the conversion zimosterol in fecosterol. This is a step that occurs in fungi in the ergosterol biosynthesis, but does not occur in the animal hosts which have specific enzymes for the conversion of zymosterol to cholesterol. In this study, gene function of ERG6 was investigated and characterized in the Cryptococcus neoformans demonstrating the absence of this gene generates several phenotypic changes, such as sensitivity to osmotic stress, oxidative stress and increased susceptibility to different antifungal drugs. Furthermore, it was also observed that the ability to grow on media with different cell wall stressors was also altered. It was observed that the lack of ERG6 greatly affects the permeabillity of the membrane resulting in osmotic and oxidative stress sensitivity and changing antifungal drugs susceptibility. Furthermore, it was also observed that the ability to grow in medium with cell wall stressor was altered. About the virulence factor, C. neoformans was unable to grow at 37°C, but it had not affected the production of melanin or capsule. However, virulence tests in vitro with macrophages and in vivo with Galleria mellonella caterpillars showed a decrease in the virulence of ERG6 mutants. Finally, the membrane sterols analysis demonstrated a change in the membrane sterol composition. Thus, being an enzyme found especially fungi, the Erg6 may be a potential molecular target for antifungal drugs.
Barros, Amanda Lira Nogueira. "Análise funcional do gene VELB de Cryptococcus neoformans." reponame:Repositório Institucional da UnB, 2014. http://repositorio.unb.br/handle/10482/16480.
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As proteínas Velvet compreendem quatro membros altamente conservados entre ascomicetos e basidiomicetos, os quais compartilham o domínio Velvet. Eles regulam diferentes vias de sinalização em resposta a estímulos ambientais e também coordenam o metabolismo secundário e diferenciação assexual e sexual em diferentes espécies de fungos. Recentemente, as proteínas Velvet foram implicadas na virulência de alguns agentes patogênicos de plantas. Buscou-se avaliar o papel das proteínas Velvet no fungo patogênico humano Cryptococcus neoformans, causador da criptococose, importante micose sistêmica e a terceira doença mais prevalente em indivíduos portadores do vírus HIV. Entre as micoses sistêmicas, ela é classificada como tendo a maior incidência em pacientes imunocomprometidos. A infecção por C.neoformans ocorre pela inalação de células infecciosas e é considerada uma infecção pulmonar primária, o que pode levar a uma infecção disseminada e meningoencefalite. O objetivo geral do trabalho foi avaliar o papel do gene VELB na patogenidade e morfogênese de C. neoformans. O primeiro passo realizado no estudo foi a deleção do gene VELB em Cryptococcus neoformans. O cassete foi transformado em células leveduriformes haploides de C. neoformans diretamente através de biobalística. Confirmou-se a deleção por PCR e Southern Blot. Após confirmação, os transformantes foram submetidos a diferentes condições de crescimento para determinação de seus fenótipos e virulência. Foram realizados testes de estresse osmótico (NaCl), estresse na parede celular (CongoRed, SDS, cafeína) e estresse oxidativo com peróxido de hidrogênio. Foram avaliados os fatores de virulência de C. neoformans como fosfolipase, urease, melanina e cápsula. Os testes fenotípicos não apresentaram alterações entre a linhagem e mutante indicando que o gene VELB parece não ter nenhum envolvimento em virulência e resposta a agentes estressores. Para avaliação da capacidade de acasalamento foram realizados co-cultivos no escuro em diferentes meios de cultura (Filament, SLAD e MS), além disto foi testada a capacidade de fusão dos mutantes. Estes testes demonstraram que a deleção de VELB bloqueia a produção de hifas no acasalamento entre mutantes e que estes apresentam também defeito no processo de fusão celular. Estes resultados gerados neste trabalho sobre o papel de VELB corroboram os dados anteriormente obtidos pelo nosso grupo de pesquisa de que os membros da família velvet estão diretamente envolvidos na regulação do ciclo sexual de C. neoformans. _________________________________________________________________________________ ABSTRACT
The Velvet proteins consist of four highly conserved members in ascomycetes and basidiomycetes, sharing the Velvet domain. They regulate different signaling ways in response to environmental stimulation and also coordinate secondary metabolism and asexual to sexual differentiation in different species of fungi. Recently, Velvet proteins have been implicated in the virulence of some pathogenic agents of plants. It was evaluated the role of Velvet proteins in the human pathogenic fungus Cryptococcus neoformans that causes cryptococcosis, an important systemic mycosis and the third most prevalent disease in HIV positive individuals. Compared to other systemic mycoses, it is classified as having the highest incidence in immunocompromised patients. C.neoformans infection is by inhalation of infectious cells and is considered a primary pulmonary infection, which can lead to widespread infection and meningoencephalitis. The overall objective of this study was to evaluate the role of the VELB gene in morphogenesis and pathogenicity of C. neoformans. The first step was performed at study deletion of the gene VELB in Cryptococcus neoformans. The cassette was transformed into haploid yeast cells of C. neoformans directly through biolistic. The deletion was confirmed by PCR and Southern blot. After confirmation, the transformants were submitted to different growth conditions to determine their phenotypes and virulence. Tests of osmotic stress (KCl, NaCl, sorbitol), stress in the cell wall (Congo Red, Calcofluor, SDS, caffeine) and oxidative stress with hydrogen peroxide were performed. Virulence factors of C. neoformans as phospholipase, urease, melanin and capsule were evaluated. Phenotypic tests showed no change between the strain and mutant indicating that the VELB gene seems to have no involvement in virulence and response to stressors. To evaluate the ability of mating co-cultures were performed in the dark in different culture media (Filament, SLAD and MS), in addition it was tested the ability of fusion of mutants. These tests showed that the deletion of VELB blocks the production of hyphae in mating and that these mutants also exhibit defective cell fusion process. These results generated in this paper about the VELB function corroborate data previously 13 obtained by our research group that the members of the velvet family are directly involved in the regulation of the sexual cycle of C. neoformans.
Books on the topic "Cryptococcus neoformans"
Casadevall, Arturo, and John R. Perfect. Cryptococcus neoformans. Washington, DC, USA: ASM Press, 1998. http://dx.doi.org/10.1128/9781555818241.
Full text1949-, Perfect John R., ed. Cryptococcus neoformans. Washington, D.C: ASM Press, 1998.
Find full textEsther, Segal, and Baum Gerald L, eds. Pathogenic yeasts and yeast infections. Boca Raton: CRC Press, 1994.
Find full textCasadevall, Arturo, and John R. Perfect. Cryptococcus Neoformans. Wiley & Sons, Limited, John, 2014.
Find full textHalliday, Catriona L., and Sarah E. Kidd. Cryptococcus species. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0012.
Full textGómez, Luis Guardiola. Actividad y Farmacodinamia de Cinco Antifungicos Frente a Cryptococcus Neoformans. Universidad Complutense de Madrid, Servicio de Publicaciones, 2006.
Find full textSANTOS, P. S., and M. F. C. PIRES. Atividade antifúngica dos óleos essenciais e principais compostos químicos sobre Cryptococcus neoformans. Dialética, 2022. http://dx.doi.org/10.48021/978-65-252-3019-1.
Full textWilson, John W., and Lynn L. Estes. Fungal Infections. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199797783.003.0127.
Full textBicanic, Tihana, and Thomas S. Harrison. Fungal central nervous system infections. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0022.
Full textBook chapters on the topic "Cryptococcus neoformans"
Stechenberg, Barbara. "Cryptococcus neoformans." In The Neurological Manifestations of Pediatric Infectious Diseases and Immunodeficiency Syndromes, 303–5. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-391-2_31.
Full textCoia, John, and Heather Cubie. "Cryptococcus neoformans." In The Immunoassay Kit Directory, 691–94. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-009-0359-3_10.
Full textLamps, Laura W. "Cryptococcus neoformans." In Surgical Pathology of the Gastrointestinal System: Bacterial, Fungal, Viral, and Parasitic Infections, 115–17. New York, NY: Springer US, 2009. http://dx.doi.org/10.1007/978-1-4419-0861-2_18.
Full textKretschmar, Marianne. "Cryptococcus neoformans." In Lexikon der Infektionskrankheiten des Menschen, 206–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-39026-8_226.
Full textLodge, Jennifer K., and John R. Perfect. "Cryptococcus neoformans." In Pathogen Genomics, 197–214. Totowa, NJ: Humana Press, 2002. http://dx.doi.org/10.1007/978-1-59259-172-5_13.
Full textDromer, Françoise, Arturo Casadevall, John Perfect, and Tania Sorrell. "Cryptococcus neoformans: Latency and Disease." In Cryptococcus, 429–39. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816858.ch31.
Full textIdnurm, Alexander, Yong-Sun Bahn, Wei-Chiang Shen, Julian C. Rutherford, and Fritz A. Mühlschlegel. "Sensing Extracellular Signals in Cryptococcus neoformans." In Cryptococcus, 175–87. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816858.ch14.
Full textPirofski, Liise-Anne, and Arturo Casadevall. "Acquired Humoral Immunity to Cryptococcus neoformans." In Cryptococcus, 397–408. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816858.ch28.
Full textMitchell, Thomas G., Elizabeth Castañeda, Kirsten Nielsen, Bodo Wanke, and Marcia S. Lazéra. "Environmental Niches for Cryptococcus neoformans and Cryptococcus gattii." In Cryptococcus, 235–59. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816858.ch18.
Full textColeman, Jeffrey J., Cara J. Chrisman, Arturo Casadevall, and Eleftherios Mylonakis. "Cryptococcus neoformans: Nonvertebrate Hosts and the Emergence of Virulence." In Cryptococcus, 261–67. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555816858.ch19.
Full textConference papers on the topic "Cryptococcus neoformans"
Liu, J., X. Chen, X. Putten, F. Hagen, T. Boekhout, and F. J. Verbeek. "Image mining for pathogen yeast Cryptococcus neoformans." In IET International Conference on Visual Information Engineering (VIE 2006). IEE, 2006. http://dx.doi.org/10.1049/cp:20060507.
Full textFurtado, Lucas Cruz, Alexandre Almeida da Silva, and Júlio César Claudino dos Santos. "Cerebral cryptococcoma successfully treated by isavuconazole in an immunocompetent patient: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.723.
Full textLiu, Jinshuo, Dengyi Zhang, Yu Yao, Shubo Liu, and Farry Hagen. "Automated Feature Selection for Pathogen Yeast Cryptococcus Neoformans." In 2007 IEEE International Symposium on Industrial Electronics. IEEE, 2007. http://dx.doi.org/10.1109/isie.2007.4374839.
Full textMcHale, L., and K. Smith. "A Rare Case of Nosocomial Pulmonary Cryptococcus Neoformans." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3951.
Full textSaleem, T., H. Qurashi, S. Sendil, C. Qian, and V. Nookala. "Cryptococcus Neoformans Esoteric Lung Mass, a Rare Presentation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a6954.
Full textJinshuo Liu, P. van der Putten, F. Hagen, Xinmeng Chen, and T. Boekhout. "Detecting Virulent Cells of Cryptococcus Neoformans Yeast: Clustering Experiments." In 18th International Conference on Pattern Recognition (ICPR'06). IEEE, 2006. http://dx.doi.org/10.1109/icpr.2006.437.
Full textSuleiman, H. A., K. Chapman, V. Badami, and E. Rojas. "Disseminated Cryptococcus Neoformans Following Treatment for PR-3 ANCA Vasculitis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7101.
Full textKirincich, J., R. Purpura, and M. Dettmer. "A Parkinsonism Mimicker: Subacute Cerebral Vasculitis Secondary to Cryptococcus Neoformans." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1615.
Full textTeixeira, Lilian Eckstein, and Bruna Kuhn De Freitas Silva. "CRIPTOCOCOSE CUTÂNEA PRIMÁRIA EM PACIENTE IMUNOSSUPRIMIDOS." In I Congresso Brasileiro de Doenças Infectocontagiosas On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/2227.
Full textLin, Hao, He Zhang, Jiaming Chen, Qinke Zhang, Zihui Fan, and Shuo Chen. "Identifying pulmonary Cryptococcus neoformans infection by serum surface-enhanced Raman spectroscopy." In Optics in Health Care and Biomedical Optics IX, edited by Qingming Luo, Xingde Li, Yuguo Tang, Ying Gu, and Dan Zhu. SPIE, 2019. http://dx.doi.org/10.1117/12.2537660.
Full textReports on the topic "Cryptococcus neoformans"
Persisting Cryptococcus yeast species Vishniacozyma victoriae and Cryptococcus neoformans elicit unique airway inflammation in mice following repeated exposure (dataset). U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, March 2023. http://dx.doi.org/10.26616/nioshrd-1061-2023-0.
Full text