Log in

Relevant bibliographies by topics / Critical illness

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers
Reports

Academic literature on the topic 'Critical illness'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Critical illness.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Critical illness"

1

Economidou, Foteini, Evangelia Douka, Marinella Tzanela, Serafeim Nanas, and Anastasia Kotanidou. "Thyroid function during critical illness." HORMONES 10, no. 2 (April 15, 2011): 117–24. http://dx.doi.org/10.14310/horm.2002.1301.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Grimm, A., A. Günther, O. W. Witte, and H. Axer. "Critical-Illness-Polyneuropathie und Critical-Illness-Myopathie." Medizinische Klinik - Intensivmedizin und Notfallmedizin 107, no. 8 (October 28, 2012): 649–60. http://dx.doi.org/10.1007/s00063-012-0186-y.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Visser, Leo H. "Critical illness." Tijdschrift voor VerpleeghuisGeneeskunde 32, no. 2 (April 2007): 47–53. http://dx.doi.org/10.1007/bf03075232.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Pohl, Marcus, and Jan Mehrholz. "Begriffsbestimmung: Critical-Illness-Polyneuropathie und Critical-Illness-Myopathie." neuroreha 5, no. 01 (March 5, 2013): 10–16. http://dx.doi.org/10.1055/s-0033-1337343.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Collins, Timothy. "Critical Care Focus – Cardiology in Critical Illness Critical Care Focus – Cardiology in Critical Illness." Nursing Standard 16, no. 10 (November 21, 2001): 29. http://dx.doi.org/10.7748/ns2001.11.16.10.29.b142.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Maslove, David M., Benjamin Tang, Manu Shankar-Hari, Patrick R. Lawler, Derek C. Angus, J. Kenneth Baillie, Rebecca M. Baron, et al. "Redefining critical illness." Nature Medicine 28, no. 6 (June 2022): 1141–48. http://dx.doi.org/10.1038/s41591-022-01843-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Park, Soo-Hyun, Yoon-Jung Jeong, and Nam-Hee Kim. "Critical illness neuromyopathy." Annals of Clinical Neurophysiology 22, no. 2 (October 30, 2020): 61–66. http://dx.doi.org/10.14253/acn.2020.22.2.61.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Millichap, J. Gordon. "Critical-Illness Polyneuropathy." Pediatric Neurology Briefs 7, no. 2 (February 1, 1993): 14. http://dx.doi.org/10.15844/pedneurbriefs-7-2-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Confer, Jennifer, Janet Wolcott, and Robert Hayes. "Critical illness polyneuromyopathy." American Journal of Health-System Pharmacy 69, no. 14 (July 15, 2012): 1199–205. http://dx.doi.org/10.2146/ajhp110343.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

De Jonghe, Bernard, Tarek Sharshar, Jean-Pascal Lefaucheur, and Herv?? Outin. "Critical Illness Neuromyopathy." Clinical Pulmonary Medicine 12, no. 2 (March 2005): 90–96. http://dx.doi.org/10.1097/01.cpm.0000156639.67261.19.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Critical illness"

1

Trinder, Thomas John. "Splanchnic perfusion in critical illness." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295361.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Brealey, David Andrew. "Mitochondrial dysfunction in critical illness." Thesis, University College London (University of London), 2004. http://discovery.ucl.ac.uk/1446708/.

Full text

Abstract:

The mortality from septic shock is approximately 50%. Most patients die from the ensuing multi-organ dysfunction syndrome rather than the acute septic inflammatory process per se. The aetiology of the organ dysfunction is unknown. A characteristic phenomenon of an increasing severity of sepsis is a decrease in tissue oxygen extraction with a decrease (relative and/or absolute) in tissue oxygen consumption. Two theories have been advanced to explain this observed decrease in oxygen extraction. Traditionally, this has been ascribed to micro vascular shunting of blood away from nutrient capillaries. However, findings in both patients and animal models have demonstrated a raised tissue PO2, suggesting that the oxygen is available to cells but cannot be metabolised, i.e. a state of dysoxia. As mitochondria account for over 90% of total oxygen consumption, in the process of oxidative phosphorylation, it has been hypothesised that sepsis results in an inhibition of the mitochondrial enzymes involved in this process. If severe, this would be expected to lead to energy failure in the organs and, possibly, to initiation of apoptotic or necrotic cell death. Marked over-production of the intercellular messenger nitric oxide is a characteristic feature of sepsis; the mitochondrial damage theory has been given additional credence by the discovery that nitric oxide and its derivative, peroxynitrite, can inhibit or permanently damage mitochondrial enzymes involved in the oxidative phosphorylation pathway. The work leading to this thesis has demonstrated that sepsis is associated with an increase in nitric oxide production, a reduction in antioxidant protection, respiratory chain enzyme inhibition, and a depletion in tissue ATP levels. These changes were shown in both skeletal muscle biopsies obtained from critically ill patients in septic shock, and in skeletal muscle and liver biopsies obtained from a long-term septic rat model. These changes correlated with the severity of disease and eventual outcome. These findings thus demonstrate a mechanism that is present in both 'non-vital' and 'vital' organs, and across species. These findings may be epiphenomenal and causation needs to be definitively demonstrated. However, this work does suggest that mitochondrial dysfunction could be an important pathophysiological mechanism underlying sepsis-induced organ failure.

APA, Harvard, Vancouver, ISO, and other styles

3

Schoeman, Johan P. "Endocrine changes in canine critical illness." Thesis, University of Cambridge, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.611343.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Trubody, Victoria. "Reversible myocardial depression in critical illness." Thesis, University of Oxford, 2015. https://ora.ox.ac.uk/objects/uuid:d11d5bfe-ef0e-45f7-a8c1-ebdc9c58bc1b.

Full text

Abstract:

Introduction: Reversible myocardial depression (RMD) is a transient impairment of left ventricular systolic function. This has been described in sub populations of patients experiencing critical illness, but it is unclear if the epidemiological features are generalisable to the broader populations of critically ill adults. Previous work has identified that RMD is not benign and has been associated with a range of adverse sequelae such as arrhythmia, left ventricular outflow tract obstruction, and intra-ventricular thrombus. Consequently, studies were designed to determine the incidence, time course, and associated risk factors for the development of myocardial depression in the general population of adults experiencing critical illness. Methods: Myocardial depression was defined as a decrease in left ventricular ejection fraction =5% from AICU baseline and was assessed using serial transthoracic echocardiography. Three studies were conducted - two prospective observational cohort studies and a retrospective analysis. The incidence, absolute decrease in ejection fraction, and time course of myocardial depression was described. Routinely available demographic and clinical variables were collected. These were then rationalised and trialled as candidate explanatory variables in a logistic regression model to predict the development of myocardial depression. Results: The incidence of myocardial depression was between 16.3 - 34%, which occurred around day four of AICU admission. The median decline in LVEF at the onset of myocardial depression was between 6.5 - 14.7%, which progressed to between 10 - 17.5% at the nadir. Myocardial depression was not entirely reversible, with between 43.7 - 71.4% of participants demonstrating some degree of recovery, with LVEF improving between 13.3 - 20.2% across the studies. The probability of development of myocardial depression can be determined using five routinely collected variables, expressed as a factor; heart rate, systolic blood pressure, the presence of severe sepsis, cardiovascular organ dysfunction and sinus rhythm. Increasing systolic blood pressure, the presence of severe sepsis, and cardiovascular organ dysfunction were associated with increased risk. Increasing heart rate and the presence of sinus rhythm were associated with decreased risk. Model diagnostics indicated that the model was a good fit of the data. The model had a modest discriminating ability, with the area under the receiver operating characteristic curve = 0.69. Conclusion: The incidence of myocardial depression was between 16.3 - 34% of participants, and usually developed around day four of AICU admission. The decreases in left ventricular ejection fraction were considerable and were not always reversible. Myocardial depression can be predicted using routinely collected haemodynamic and clinical variables that are available within the first 24 hours of AICU admission.

APA, Harvard, Vancouver, ISO, and other styles

5

Conway, Morris Andrew. "Complement-mediated neutrophil dysfunction in critical illness." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/27824.

Full text

Abstract:

Critical illness, constituting an acute illness or injury resulting in organ dysfunction and failure, is associated with a profound, systemic activation of the immune system and inflammation-mediated organ damage. However, critically ill patients suffer a high rate of nosocomial infection with secondary sepsis being a common cause of death. This high prevalence of secondary infections argues for the influence of an immune suppression that may, at first glance, appear paradoxical in light of the pro-inflammatory nature of critical illness. Although immune cell hypo-function has been noted in clinical and experimental critical illness, the mediators of these effects remain poorly defined. In this thesis, neutrophil function was examined in the context of clinically suspected ventilator-associated pneumonia, a common and lethal intensive care acquired infection (ICU-AI), This demonstrated impaired bactericidal functions (phagocytosis and reactive oxygen species production) in neutrophils from both the peripheral and pulmonary compartments; however there was ample evidence of coexistent neutrophil activation (both cell surface markers and soluble mediators) and inflammation. An investigation of possible mediators of neutrophil dysfunction revealed a major role for C5a, the pro-inflammatory anaphylotoxin derived from complement C5. Recombinant C5a applied to healthy donor neutrophils was able to drive the defect in phagocytosis by phospho-inositol 3 kinase delta-mediated inhibition of RhoA and subsequent down regulation of actin polymerisation. The defects in RhoA and actin function were reversible with granulocyte-macrophage colony stimulating factor (GM-CSF) applied ex vivo, restoring phagocytic function to normal. Similar defects in RhoA and actin, and effective treatment with GMCSF, were found in neutrophils from critically ill patients. In a second cohort of critically ill adults recruited prior to developing any ICU-AI, C5a-mediated neutrophil dysfunction was an independent predictor of acquiring nosocomial infection, being associated with a 5.4 fold increased risk (95% Confidence interval 1.4-21.0). The same cohort of patients also displayed two other features of immune suppression, namely monocyte deactivation and elevated proportions of regulatory T-cells that were also associated with increased risk of infection (relative risk of infection 3 (95%CI1.3-6.9) and 2.4 (95%CI1.3-4.2) respectively). These measures acted additively with C5a-mediated dysfunction, those with no immune impairment having a zero rate of nosocomial infection with cumulative increases in impairments being associated with a progressive increase in risk of infection (p=0.0004 by Chi squared for trend). In conclusion, critical illness is characterised by a complex inflammatory state with features of simultaneous hyper- and hypoactivation. This remarkable duality is illustrated by the ability of a proinflammatory molecule, C5a, to drive neutrophil dysfunction, with this dysfunction being associated with a serious adverse event-nosocomial infection.

APA, Harvard, Vancouver, ISO, and other styles

6

Willard, Terence B. "Variability of biological signals in critical illness." Thesis, University of Manchester, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.595297.

Full text

Abstract:

Inhealthy physiological systems all organs act to maintain relative constancy or homeostasis through a highly complex and integrated communication control network of feedback loops. This constant interaction results in a fluctuation in the beat to beat value of the nominal heart rate. Heart rate variability (HRV) is used to describe the variation in the intervals between consecutive heartbeats or the intervals between consecutive R peaks of the QRS complex obtained from an electrocardiogram (ECG). The hypothesis that illness and injury is associated with a reduction in heart rate variability has been tested in various clinical settings and continues to be evaluated. However standard measures of heart rate variability using power spectral analysis have not always been conclusive and the interpretation of results is still being debated. Newer methods based on ideas from nonlinear mathematics are controversial. Inparticular, the method of approximate entropy (ApEn) may give misleading results. In this study a novel normalized entropy measure L was developed for assessing HRV, using theoretical methods and data from an acute hypovolaemic shock model for validation. Electrocardiogram data from the shock model experiments was processed to . obtain the R-R intervals for analysis and used to validate the measure L. The results were statistically significant in showing the differences between the baseline state and the post shock state and between the baseline and the post resuscitation state after allowance was made for the effects of changes in heart rate. From theoretical analysis and from experimental data the method was shown to be valid in both the time and frequency domain. Theoretical predictions of the inconsistencies of approximate entropy were confirmed by experimental results and in particular the method did not give statistically significant results for the experimental data using the accepted range of tolerance values and number of elements being compared. The standard measures of power spectral analysis were not statistically significant. However, by modifying the analysis to that of amplitude rather than power a statistically significant reduction was shown in the total amplitude and high frequency amplitude, after allowance was made for changes in heart rate, at the same states as the measure L.

APA, Harvard, Vancouver, ISO, and other styles

7

Jones, Christina H. "Rehabilitation following critical illness : support for patients." Thesis, University of Liverpool, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343717.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Ozkok, Erengul. "A stochastic model for critical illness insurance." Thesis, Heriot-Watt University, 2011. http://hdl.handle.net/10399/2449.

Full text

Abstract:

In this thesis, we present methods and results for the estimation of diagnosis inception rates for Critical Illness Insurance (CII) claims in the UK by cause. This is the first study which provides a stochastic model for the diagnosis inception rates for CII. The data are supplied by the UK Continuous Mortality Investigation and relate to claims settled in the years 1999 - 2005. First, we develop a model for the delay between dates of diagnosis and settlement of claims in CII using a generalised-lineartype model with Burr errors under both Bayesian and maximum likelihood approach. Variable selection using Bayesian methodology to obtain the best model with different prior distribution setups for the parameters is applied. For comparison purposes, a lognormal model and frequency-based model selection techniques are also considered. The non-recorded dates of diagnosis and settlement have been included in the analysis as missing values using their posterior predictive distribution and Markov Chain Monte Carlo methodology. Missing dates of diagnosis are estimated using the parsimonious claim delay distribution. With this complete data set, diagnosis inception rates for all causes (combined) and for specific causes are estimated using an appropriate claim delay distribution where the observed numbers of claim counts are assumed to have a Poisson distribution. To model the crude rates, a generalised linear model with Poisson errors and log-link function is used.

APA, Harvard, Vancouver, ISO, and other styles

9

Ismaeil, Taha. "Quantifying the severity of respiratory critical illness." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/42993/.

Full text

Abstract:

Clinicians use several oxygen-based indices in intensive care units as surrogates to determine the condition of the patient’s lung and verify monitoring progress. Examples of these oxygen indices include the ratio of arterial oxygen tension to inspired oxygen fraction (PaO2/FiO2 ratio); arterial/alveolar oxygen tension ratio (PaO2/PAO2); alveolar–arterial oxygen tension difference (PA-aO2); respiratory index (RI= (PA-aO2)/PaO2), and content-based venous admixture (Qs/Qt). One of the issues with this approach is that these indices fail to take into consideration several additional external pulmonary physiological factors and, as such, these indices could potentially mislead clinicians. This thesis explores the nature of the oxygen-tension-based indices response and examined the effect that varying certain external pulmonary factors, such as FiO2, PaCO2, Hb, respiratory rate, oxygen consumption, cardiac output, and respiratory quotient, had on PaO2 using virtual subjects and patients’ data to quantify oxygenation defect through a combination of mathematics, different diseases, and pathophysiology. There were one or two approaches that could lead us to the answer, and many dead end routes. Eventually, the research produced a new index that was compared and validated using two approaches. First, on virtual subjects with lung pathologies that were commonly seen in the intensive care unit and then on real clinical data that was obtained from the intensive care unit. The results of these validation investigations indicated that the proposed index is more robust and resistant to variations in certain external pulmonary factors than the PaO2/FiO2 ratio. As such, there is a strong indication that it may help to improve the quality of patient care provided. The feasibility of manually calculating and applying this newly proposed index in the ICU is an issue that merits further exploration. Theoretically, if the newly proposed index was found to be practicable, it could improve the healthcare provided; reduce the cost of unnecessary blood work, and save time and effort. However, due to the time it takes to calculate crPaO2 manually, the use of medical technology and computer applications is desirable.

APA, Harvard, Vancouver, ISO, and other styles

10

Rothwell, Peter Malcolm. "Thyroid and adrenocortical function during critical illness." Thesis, University of Edinburgh, 1995. http://hdl.handle.net/1842/21504.

Full text

Abstract:

The main aims of the thesis were as follows: (1) To determine the ranges of total thyroxine, triiodothyrone, TSH and cortisol concentrations at different levels of illness severity, and to relate these to mortality; (2) To determine the range of responses to standard dynamic tests of adrenocortical and thyroid function, and relate them to severity of illness and mortality; (3) To determine the relationship between the extent of the changes in total thyroid hormone concentrations during illness and metabolic rate; (4) To accurately determine the prognostic value of measurement of thyroxine, triiodothyrone, TSH and cortisol on admission to an ICU. The majority of critically ill patients had plasma cortisol concentrations above the upper limit of normal in health. In keeping with Seyle's General Adaption Theory, plasma cortisol concentration correlated with severity of illness. The normal ranges of plasma cortisol concentration, defined as population mean +/- 2SD, differed according to the severity of illness. A plasma cortisol concentration above 200 nmol/L would be within the 95% range for a moderately ill patient (APACHE II score <16) whereas a concentration of under 400 nmol/L would be below the expected range in a severely ill patient (APACHE II score >24). A prognostic index based on admission measurements of cortisol, thyroxine and thyrotropin concentrations was developed using multiple logistic regression analysis. The model obtained predicted outcome of illness with significantly greater accuracy than APACHE II scores.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Critical illness"

1

Weinhouse, Gerald L., and John W. Devlin, eds. Sleep in Critical Illness. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-06447-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

C, Parsons Lynn, Lehman Cheryl, and Prevost Suzanne S, eds. Rehabilitation after critical illness. Philadelphia: W.B. Saunders, 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Galley, Helen F. Cardiology in critical illness. Edited by NetLibrary Inc and Intensive Care Society of the United Kingdom. London: BMJ Books, 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

W, Ryan David, ed. Current practice in critical illness. London: Chapman & Hall, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Rombeau, J. L., and J. Takala, eds. Gut Dysfunction in Critical Illness. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80224-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Stevens, Robert D., Tarek Sharshar, and E. Wesley Ely, eds. Brain Disorders in Critical Illness. Cambridge: Cambridge University Press, 2013. http://dx.doi.org/10.1017/cbo9781139248822.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Wijdicks, Eelco F. M., 1954-, ed. Neurologic complications of critical illness. 2nd ed. Oxford: Oxford University Press, 2002.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

L, Rombeau John, and Takala J. 1953-, eds. Gut dysfunction in critical illness. Berlin: Springer, 1996.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

A, Cynober Luc, and Moore Frederick A. 1953-, eds. Nutrition and critical care. Basel: Karger, 2003.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

S, Wheeler Derek, Wong Hector R. 1963-, and SpringerLink (Online service), eds. Cardiovascular Pediatric Critical Illness and Injury. London: Springer-Verlag London, 2009.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Book chapters on the topic "Critical illness"

1

Serrano-Munuera, C., and I. Illa. "Critical Illness Polyneuropathy and Critical Illness Myopathy." In Mechanical Ventilation and Weaning, 118–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-56112-2_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Grimm, A., A. Günther, O. W. Witte, and H. Axer. "Critical-Illness-Polyneuropathie und Critical-Illness-Myopathie." In Weiterbildung Intensivmedizin und Notfallmedizin, 93–102. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-40738-3_8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Billah, Mohd Ma’Sum. "Critical Illness Takaful." In Islamic Insurance Products, 295–304. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-17681-5_23.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Al-Lozi, Muhammad, and Alan Pestronk. "Critical Illness Myopathy." In International Neurology, 447–48. Oxford, UK: Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444317008.ch117.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Camhi, S. L., and J. E. Nelson. "Chronic Critical Illness." In Yearbook of Intensive Care and Emergency Medicine, 908–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-49433-1_82.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Bolton, Charles F., and Daniel F. Hanley. "Critical Illness Neuropathy." In Neurocritical Care, 801–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-87602-8_70.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Goodman, Brent P., and Andrea J. Boon. "Critical illness neuromyopathy." In Evidence-Based Neurology: Management of Neurological Disorders, 243–48. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781119067344.ch23.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Benatar, Michael. "Critical Illness Weakness." In Neuromuscular Disease, 421–31. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1007/978-1-59745-106-2_23.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Abbasi, Adeel, Francis DeRoos, José Artur Paiva, J. M. Pereira, Brian G. Harbrecht, Donald P. Levine, Patricia D. Brown, et al. "Critical Illness Myopathy." In Encyclopedia of Intensive Care Medicine, 637. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1420.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Marik, Paul Ellis. "Chronic Critical Illness." In Handbook of Evidence-Based Critical Care, 43–54. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5923-2_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Critical illness"

1

Brummel, N. E., M. F. Mart, P. Pandhariapande, C. Wang, S. Yilmaz, J. Peng, and E. W. Ely. "Physical Activity During Critical Illness and Subsequent Disability in Survivors of Critical Illness." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4505.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Buchman, T. "Control theories in critical illness and critical care." In 2004 43rd IEEE Conference on Decision and Control (CDC) (IEEE Cat. No.04CH37601). IEEE, 2004. http://dx.doi.org/10.1109/cdc.2004.1428582.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Buchman, T. "Control theories in critical illness and critical care." In 2004 43rd IEEE Conference on Decision and Control (CDC) (IEEE Cat. No.04CH37601). IEEE, 2004. http://dx.doi.org/10.1109/cdc.2004.1429197.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Buchman, T. "Control theories in critical illness and critical care." In 2004 43rd IEEE Conference on Decision and Control (CDC) (IEEE Cat. No.04CH37601). IEEE, 2004. http://dx.doi.org/10.1109/cdc.2004.1429480.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Buchman, T. "Control theories in critical illness and critical care." In 2004 43rd IEEE Conference on Decision and Control (CDC). IEEE, 2004. http://dx.doi.org/10.1109/cdc.2004.1428571.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Buchman, T. "Control theories in critical illness and critical care." In 2004 43rd IEEE Conference on Decision and Control (CDC) (IEEE Cat. No.04CH37601). IEEE, 2004. http://dx.doi.org/10.1109/cdc.2004.1430178.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Retnani, Hani Dwi, Neva Satyahadewi, Hendra Perdana, and Ray Tamtama. "The calculation of critical illness insurance premiums with terminal illness condition." In INTERNATIONAL CONFERENCE ON ENGINEERING AND COMPUTER SCIENCE (ICECS) 2022: The Use of Innovative Technology in Accelerating Problems Sustainable Development. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0204804.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Calkins, K., and J. L. Guttormson. "Critical Illness Survivors' Perceptions of Recovery." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4146.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Brummel, N. E., K. Rengel, J. Wilson, E. Hollingsworth, P. Pandharipande, and E. W. Ely. "Association of Pre-Critical Illness Muscle Mass with Disability and Physical Function in Survivors of Critical Illness." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4328.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Ibarra, G., M. Velasquez, J. Cosico, M. Ruiz, and N. Bergasa. "Copper Deficiency Masked by Critical Illness Neuropathy." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5164.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Critical illness"

1

Vaughan, George M., Basil A. Pruitt, and Jr. Thyroid Function in Critical Illness and Burn Injury,. Fort Belvoir, VA: Defense Technical Information Center, July 1993. http://dx.doi.org/10.21236/ada277947.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Cox, Christopher, Catherine Hough, Shannon Carson, Douglas White, Jeremy Kahn, Maren Olsen, Derek Jones, Tamara Somers, Sarah Kelleher, and Laura Porter. Improving Psychological Distress Among Critical Illness Survivors and Their Informal Caregivers. Patient-Centered Outcomes Research Institute (PCORI), September 2018. http://dx.doi.org/10.25302/9.2018.cer.195.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Li, Liru, and Jie Huang. Nasogastric tube versus postpyloric tube feeding for critical illness: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2023. http://dx.doi.org/10.37766/inplasy2023.8.0104.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Villa, Michele, Massimo Le Pera, and Michela Bottega. Quality of Abstracts in Randomized Controlled Trials Published in Leading Critical Care Nursing Journals. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0039.

Full text

Abstract:

Review question / Objective: This review aims to evaluate the methodological quality of RCT-abstracts in leading critical care nursing journals. A methodological quality review with the Consolidated Standards of Reporting Trials (CONSORT) criteria will be performed in RCT-abstracts published between 2011-2021 in the first Scopus-ranking (2021) nursing journals. Eligibility criteria: Abstracts of scientific articles will be included if they fulfil the following inclusion criteria: 1) they report the results of parallel and/or cross-over group RCTs, 2) they are written in English, 3) they refer to the care of adult patients with acute/critical illness or conducted in adult ICUs.Manuscripts reporting results of pilot or feasibility studies, cluster trials, observational or cohort studies, interim analyses, economic analyses of RCTs, post-trial follow-up studies, subgroup and secondary analyses of previously published RCTs, editorials and RCTs without an abstract such as RCTs published as letters to the editor, single-subject clinical trials will be excluded.

APA, Harvard, Vancouver, ISO, and other styles

5

Yang, Hui, Xi-Xi Wan, Hui Ma, Zhen LI, Li Weng, Ying Xia, and Xiao-Ming Zhang. Prevalence and mortality risk of low skeletal muscle mass in critically ill patients: an updated systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0132.

Full text

Abstract:

Review question / Objective: The PICOS principle was adopted when we confirmed the study eligibility. The inclusion criteria were as follows: (1) patients were critically ill, which was defined as adult patients who were from the ICU department; (2) exposure: patients had a clear definition of LSMM based on CT scans, anthropometric methods and ultrasound; (3) presented the prevalence of LSMM or could be calculated by the available data from the article; and (4) study design: observational study (cohort study or cross-sectional study). Articles that were reviews, case reports, comments, correspondences, letters or only abstracts were excluded. Condition being studied: Critical illness often results in low skeletal muscle mass for multiple reasons. Multiple studies have explored the association between low skeletal muscle mass and mortality. The prevalence of low skeletal muscle mass and its association with mortality are unclear. This systematic review and meta-analysis aim to identify the prevalence and mortality risk of low skeletal muscle mass among critically ill patients.

APA, Harvard, Vancouver, ISO, and other styles

6

Brandl, Maria T., Shlomo Sela, Craig T. Parker, and Victor Rodov. Salmonella enterica Interactions with Fresh Produce. United States Department of Agriculture, September 2010. http://dx.doi.org/10.32747/2010.7592642.bard.

Full text

Abstract:

The emergence of food-borne illness outbreaks linked to the contamination of fruits and vegetables is a great concern in industrialized countries. The current lack of control measures and effective sanitization methods prompt the need for new strategies to reduce contamination of produce. Our ability to assess the risk associated with produce contamination and to devise innovative control strategies depends on the identification of critical determinants that affect the growth and the persistence of human pathogens on plants. Salmonella enterica, a common causal agent of illness linked to produce, has the ability to colonize and persist on plants. Thus, our main objective was to identify plant-inducible genes that have a role in the growth and/or persistence of S. enterica on postharvest lettuce. Our findings suggest that in-vitro biofilm formation tests may provide a suitable model to predict the initial attachment of Salmonella to cut-romaine lettuce leaves and confirm that Salmonella could persist on lettuce during shelf-life storage. Importantly, we found that Salmonella association with lettuce increases its acid-tolerance, a trait which might be correlated with an enhanced ability of the pathogen to pass through the acidic barrier of the stomach. We have demonstrated that Salmonella can internalize leaves of iceberg lettuce through open stomata. We found for the first time that internalization is an active bacterial process mediated by chemotaxis and motility toward nutrient produced in the leaf by photosynthesis. These findings may provide a partial explanation for the failure of sanitizers to efficiently eradicate foodborne pathogens in leafy greens and may point to a novel mechanism utilized by foodborne and perhaps plant pathogens to colonize leaves. Using resolvase in vivo expression technology (RIVET) we have managed to identify multiple Salmonella genes, some of which with no assigned function, which are involved in attachment to and persistence of Salmonella on lettuce leaves. The precise function of these genes in Salmonella-leaf interactions is yet to be elucidated. Taken together, our findings have advanced the understanding of how Salmonella persist in the plant environment, as well as the potential consequences upon ingestion by human. The emerging knowledge opens new research directions which should ultimately be useful in developing new strategies and approaches to reduce leaf contamination and enhance the safety of fresh produce.

APA, Harvard, Vancouver, ISO, and other styles

7

Viswanathan, Meera, Jennifer Cook Middleton, Alison Stuebe, Nancy Berkman, Alison N. Goulding, Skyler McLaurin-Jiang, Andrea B. Dotson, et al. Maternal, Fetal, and Child Outcomes of Mental Health Treatments in Women: A Systematic Review of Perinatal Pharmacologic Interventions. Agency for Healthcare Research and Quality (AHRQ), April 2021. http://dx.doi.org/10.23970/ahrqepccer236.

Full text

Abstract:

Background. Untreated maternal mental health disorders can have devastating sequelae for the mother and child. For women who are currently or planning to become pregnant or are breastfeeding, a critical question is whether the benefits of treating psychiatric illness with pharmacologic interventions outweigh the harms for mother and child. Methods. We conducted a systematic review to assess the benefits and harms of pharmacologic interventions compared with placebo, no treatment, or other pharmacologic interventions for pregnant and postpartum women with mental health disorders. We searched four databases and other sources for evidence available from inception through June 5, 2020 and surveilled the literature through March 2, 2021; dually screened the results; and analyzed eligible studies. We included studies of pregnant, postpartum, or reproductive-age women with a new or preexisting diagnosis of a mental health disorder treated with pharmacotherapy; we excluded psychotherapy. Eligible comparators included women with the disorder but no pharmacotherapy or women who discontinued the pharmacotherapy before pregnancy. Results. A total of 164 studies (168 articles) met eligibility criteria. Brexanolone for depression onset in the third trimester or in the postpartum period probably improves depressive symptoms at 30 days (least square mean difference in the Hamilton Rating Scale for Depression, -2.6; p=0.02; N=209) when compared with placebo. Sertraline for postpartum depression may improve response (calculated relative risk [RR], 2.24; 95% confidence interval [CI], 0.95 to 5.24; N=36), remission (calculated RR, 2.51; 95% CI, 0.94 to 6.70; N=36), and depressive symptoms (p-values ranging from 0.01 to 0.05) when compared with placebo. Discontinuing use of mood stabilizers during pregnancy may increase recurrence (adjusted hazard ratio [AHR], 2.2; 95% CI, 1.2 to 4.2; N=89) and reduce time to recurrence of mood disorders (2 vs. 28 weeks, AHR, 12.1; 95% CI, 1.6 to 91; N=26) for bipolar disorder when compared with continued use. Brexanolone for depression onset in the third trimester or in the postpartum period may increase the risk of sedation or somnolence, leading to dose interruption or reduction when compared with placebo (5% vs. 0%). More than 95 percent of studies reporting on harms were observational in design and unable to fully account for confounding. These studies suggested some associations between benzodiazepine exposure before conception and ectopic pregnancy; between specific antidepressants during pregnancy and adverse maternal outcomes such as postpartum hemorrhage, preeclampsia, and spontaneous abortion, and child outcomes such as respiratory issues, low Apgar scores, persistent pulmonary hypertension of the newborn, depression in children, and autism spectrum disorder; between quetiapine or olanzapine and gestational diabetes; and between benzodiazepine and neonatal intensive care admissions. Causality cannot be inferred from these studies. We found insufficient evidence on benefits and harms from comparative effectiveness studies, with one exception: one study suggested a higher risk of overall congenital anomalies (adjusted RR [ARR], 1.85; 95% CI, 1.23 to 2.78; N=2,608) and cardiac anomalies (ARR, 2.25; 95% CI, 1.17 to 4.34; N=2,608) for lithium compared with lamotrigine during first- trimester exposure. Conclusions. Few studies have been conducted in pregnant and postpartum women on the benefits of pharmacotherapy; many studies report on harms but are of low quality. The limited evidence available is consistent with some benefit, and some studies suggested increased adverse events. However, because these studies could not rule out underlying disease severity as the cause of the association, the causal link between the exposure and adverse events is unclear. Patients and clinicians need to make an informed, collaborative decision on treatment choices.

APA, Harvard, Vancouver, ISO, and other styles

8

Kuzmin, Vyacheslav, Alebai Sabitov, Andrei Reutov, Vladimir Amosov, Lidiia Neupokeva, and Igor Chernikov. Electronic training manual "Providing first aid to the population". SIB-Expertise, January 2024. http://dx.doi.org/10.12731/er0774.29012024.

Full text

Abstract:

First aid represents the simplest urgent measures necessary to save the lives of victims of injuries, accidents and sudden illnesses. Providing first aid greatly increases the chances of salvation in case of bleeding, injury, cardiac and respiratory arrest, and prevents complications such as shock, massive blood loss, additional displacement of bone fragments and injury to large nerve trunks and blood vessels. This electronic educational resourse consists of four theoretical educational modules: legal aspects of providing first aid to victims and work safety when providing first aid; providing first aid in critical conditions of the body; providing first aid for injuries of various origins; providing first aid in case of extreme exposures, accidents and poisonings. The electronic educational resource materials include 8 emergency conditions and 11 life-saving measures. The theoretical block of modules is presented by presentations, the text of lectures with illustrations, a video film and video lectures. Control classes in the form of test control accompany each theoretical module. After studying all modules, the student passes the final test control. Mastering the electronic manual will ensure a high level of readiness to provide first aid to persons without medical education.

APA, Harvard, Vancouver, ISO, and other styles

9

Understanding Eating Disorders. ACAMH, January 2022. http://dx.doi.org/10.13056/acamh.18865.

Full text

Abstract:

Around 1.25 million people in the UK suffer from eating disorders. These disorders can cause serious harm, both physically and emotionally, and they have the highest mortality rate of any mental illness. Early diagnosis, intervention and treatment is critical.

APA, Harvard, Vancouver, ISO, and other styles

10

Repositioning post partum care in Kenya. Population Council, 2005. http://dx.doi.org/10.31899/rh16.1013.

Full text

Abstract:

In Kenya, although 45 percent of maternal deaths occur within the first 24 hours after childbirth and 65 percent of maternal deaths occur during the first week postpartum, health-care providers continue to advise on a first check-up six weeks after childbirth. The early postpartum period is also critical to newborn survival, with 50–70 percent of life-threatening newborn illnesses occurring in the first week. Yet most strategies to reduce maternal and perinatal morbidity and mortality have focused on pregnancy and birth. In addition to the heavy workload of providers who do not assess the mother post-delivery when she may bring her infant for immunization, lack of knowledge, poverty, cultural beliefs and practices perpetuate the problem. The only register that exists for mothers post-delivery is for family planning, thus perpetuating the lack of emphasis on the early postpartum period with no standardized register to record care given. To address this gap in service delivery, the Population Council defined the minimal services a mother and baby should receive from a skilled attendant after birth. As stated in this brief, the development of a standardized postpartum register is one step toward advocating for providing early postpartum care among health-service providers.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!