Academic literature on the topic 'Craniofacial morphogenesis'
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Journal articles on the topic "Craniofacial morphogenesis"
Klueber, Kathleen M. "Craniofacial Morphogenesis." Ear, Nose & Throat Journal 71, no. 10 (October 1992): 472–76. http://dx.doi.org/10.1177/014556139207101008.
Full textCobourne, M. "Review: Craniofacial Morphogenesis." European Journal of Orthodontics 25, no. 2 (April 1, 2003): 214–15. http://dx.doi.org/10.1093/ejo/25.2.214.
Full textSolursh, Michael, and Jeffrey Murray. "Craniofacial Morphogenesis Workshop Report." Cleft Palate-Craniofacial Journal 31, no. 3 (May 1994): 230–31. http://dx.doi.org/10.1597/1545-1569(1994)031<0230:cmwr>2.3.co;2.
Full textSolursh, Michael, and Jeffrey Murray. "Craniofacial Morphogenesis Workshop Report." Cleft Palate-Craniofacial Journal 31, no. 3 (May 1994): 230–31. http://dx.doi.org/10.1597/1545-1569_1994_031_0230_cmwr_2.3.co_2.
Full textRadlanski, RJ. "Prenatal craniofacial morphogenesis: four-dimensional visualization of morphogenetic processes." Orthodontics & Craniofacial Research 6 (August 2003): 89–94. http://dx.doi.org/10.1034/j.1600-0544.2003.240.x.
Full textMundhada, A., U. Kulkarni, V. Swami, S. Deshmukh, and A. Patil. "Craniofacial Muscles-differentiation and Morphogenesis." Annual Research & Review in Biology 9, no. 6 (January 10, 2016): 1–9. http://dx.doi.org/10.9734/arrb/2016/24329.
Full textHelms, J. A. "New insights into craniofacial morphogenesis." Development 132, no. 5 (February 2, 2005): 851–61. http://dx.doi.org/10.1242/dev.01705.
Full textSzabo-Rogers, Heather L., Lucy E. Smithers, Wardati Yakob, and Karen J. Liu. "New directions in craniofacial morphogenesis." Developmental Biology 341, no. 1 (May 2010): 84–94. http://dx.doi.org/10.1016/j.ydbio.2009.11.021.
Full textChai, Yang, and Robert E. Maxson. "Recent advances in craniofacial morphogenesis." Developmental Dynamics 235, no. 9 (2006): 2353–75. http://dx.doi.org/10.1002/dvdy.20833.
Full textLu, Chung-Ling, and Jinoh Kim. "Craniofacial Diseases Caused by Defects in Intracellular Trafficking." Genes 12, no. 5 (May 13, 2021): 726. http://dx.doi.org/10.3390/genes12050726.
Full textDissertations / Theses on the topic "Craniofacial morphogenesis"
Coussens, Anna Kathleen. "Molecular regulation of calvarial suture morphogenesis and human craniofacial diversity." Thesis, Queensland University of Technology, 2007. https://eprints.qut.edu.au/16481/1/Anna_Coussens_Thesis.pdf.
Full textCoussens, Anna Kathleen. "Molecular regulation of calvarial suture morphogenesis and human craniofacial diversity." Queensland University of Technology, 2007. http://eprints.qut.edu.au/16481/.
Full textLi, Wai-Yee. "TGF-B Signalling in Epithelial-Mesenchymal Interactions : Craniofacial Morphogenesis and Fetal Wound Healing." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503632.
Full textSun, Zhao. "New molecular mechanisms controlling dental epithelial stem cell maintenance, growth and craniofacial morphogenesis." Diss., University of Iowa, 2016. https://ir.uiowa.edu/etd/5652.
Full textKatsube, Motoki. "Critical Growth Processes for the Midfacial Morphogenesis in the Early Prenatal Period." Kyoto University, 2019. http://hdl.handle.net/2433/242383.
Full textAlrajeh, Moussab. "Embryologie de la neurofibromatose de type I : morphogenese craniofaciale et regulations du gene NF1 dans la crete neurale." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS471/document.
Full textThe neurofibromatosis-type 1 (NF1) (Von Recklinghausen disease) is an autosomal disorder, which stems from misrgulation of Neurofibromin (NF1), a gene encoding a tumour-suppressor protein which acts as a negative regulator of RAS proteins. Mutations of NF1 are causally linked to many types of tumours located in skin, nerves, but also in the brain (intra- cerebral tumours and gliomas). NF1 patients have a high risk of developing both benign and malignant tumours. The diversity of deficits and the nature of cellular lineages attribute all these tumoral manifestations to deregulation of neural crest cell (NC) derivatives. The NC is a multipotent stem cell population that contributes to a variety of cell types in vertebrate embryo, which include skeletogenic, glial, pigment cells as well as pericytes. In order to understand the pathologic process of this disease, it is essential to analyze the molecular mechanisms involved in the survival, proliferation and differentiation of NC.Our objectives are therefore to gain insights into the molecular cascade responsible for the diversity of NC derivatives at cephalic level. We opt for a drastic approach consisting in eradicating NF1 activity from NC at the beginning of their migration. In our experimental model, we can analyze developmental interactions of NC and the epigenetic regulation of the NF1 gene, at their level. Espically class1 Histone deacetylases (HDAC) family of molecules. So we have developed a system which allows complete inactivation of the NF1 gene in NC specifically using interfering RNA molecules (silencing) transfected by electroporation in the bilateral NC, during the early stage of neurulation, using the chick embryo as an experimental model.We show that HDAC8 inactivation can reproduce the alterations of vascular phenotypes observed in NF1 hypomorphic embryos. Suggesting an important role of HDAC8 in regulating vasculogenesis and differentiation of pericytes NC. That could be by ectopic activation of Sox9 gene supporting the pathological transdifférenciaton pericytes in gliomateux process or intracerebral calcifications
"Epithelial signals regulate pigpen expression during craniofacial morphogenesis." Tulane University, 2002.
Find full textacase@tulane.edu
Billmyre, Katherine Kretovich. "Multiple roles of epithelial signaling during craniofacial and foregut morphogenesis." Diss., 2015. http://hdl.handle.net/10161/9827.
Full textAbstract
During embryonic development many structures crucial for breathing and eating arise from the pharyngeal and anterior foregut epithelium (FGE), which contains the oral ectoderm and the foregut endoderm. Proper differentiation and signaling within and from this epithelial tissue is necessary for the development of the mandible, the esophagus, and the trachea. Many birth defects occur in these structures that greatly disrupt the ability of affected infants to breathe and eat. This dissertation investigates the importance of the pharyngeal and anterior FGE in mandible and foregut development.
The most rostral portion of the pharyngeal epithelium contributes to the development of the mandible. At embryonic day 10.5 the mandible is a bud structure, composed of neural crest-derived mesenchyme and core mesoderm surrounded by pharyngeal epithelium. The mesenchyme needs to receive Hedgehog signaling for mandible development, but the epithelial tissue that signals to the mesenchyme has not been identified in mammals. Data presented in Chapter 2 show that Sonic Hedgehog is necessary at two distinct stages of mandible development by using a tissue specific genetic ablation to remove Sonic Hedgehog from the pharyngeal endoderm. First, we show that Sonic Hedgehog promotes cell survival prior to cartilage differentiation through immunostaining for Caspase-9, an apoptosis marker. Second, a rescue of early cell death with the p53 inhibitor pifithrin-α shows that Sonic Hedgehog is necessary for cartilage condensation and differentiation later in development. Without cartilage differentiation the mandible is unable to elongate properly and hypoplasia occurs.
Caudal to the pharyngeal epithelium is the anterior FGE, which develops into the larynx, esophagus and trachea. The anterior FGE is a single endodermal tube at E9.5 and by E11.5 compartmentalizes into two distinct tubes: the esophagus and trachea. While the signaling pathways involved in proper compartmentalization of the foregut are well studied, nothing is known about the cellular behaviors that drive this complex event. One important event during foregut compartmentalization is the establishment of dorso-ventral patterning, which is necessary for separation to occur. To elucidate the importance of dorso-ventral patterning, we take advantage of two genetic mouse models with disrupted patterning, an activation of and a removal of β-catenin in the ventral foregut endoderm. Data presented in Chapter 3 show that β-catenin is important for epithelial pseudostratification and the establishment of a region of double-positive cells at the dorso-ventral midline through close examination of epithelial morphogenesis at E10.5 prior to compartmentalization. This data has established two mouse models for studying changes in epithelial morphology during foregut compartmentalization. In total, this body of work details how signals originating in the pharyngeal and anterior foregut epithelium regulate both mesenchymal and epithelial behaviors during mandible and foregut development.
Dissertation
Gruda, Agron [Verfasser]. "Herkömmliche, modifizierte und neue Messmethoden zur kephalometrischen Untersuchung der pränatalen craniofacialen Morphogenese des Menschen anhand von bilateralen und frontalen Darstellungen von 3D-Rekonstruktionen und von Aufhellungspräparaten menschlicher Embryonen und Föten von 19 mm SSL bis 145 mm SSL / von Agron Gruda." 2010. http://d-nb.info/1010554476/34.
Full textBooks on the topic "Craniofacial morphogenesis"
E, Moyers Robert, Vig Katherine W. L, Burdi Alphonse R, and Ferrara Andrea M, eds. Craniofacial morphogenesis and dysmorphogenesis. Ann Arbor, Mich: Center for Human Growth and Development, University of Michigan, 1988.
Find full textKawakami, Toshiyuki. Cell differentiation of neoplastic cells originating in the oral and craniofacial regions. New York: Nova Science, 2008.
Find full textE, Moyers Robert, Vig Katherine W. L, Burdi Alphonse R, and Ferrara Andrea M, eds. Craniofacial morphogenesis and dysmorphogenesis. Ann Arbor, Mich: Center for Human Growth and Development, University of Michigan, 1988.
Find full textNational Institute of Dental Research (U.S.), ed. Toward a Molecular Understanding of Craniofacial Morphogenesis. [S.l: s.n., 1998.
Find full textBook chapters on the topic "Craniofacial morphogenesis"
Rawlins, Joseph T., and Lynne A. Opperman. "Tgf-&Bg;; Regulation of Suture Morphogenesis and Growth." In Craniofacial Sutures, 178–96. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000115038.
Full textJussila, Maria, Emma Juuri, and Irma Thesleff. "Tooth Morphogenesis and Renewal." In Stem Cells in Craniofacial Development and Regeneration, 109–34. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118498026.ch6.
Full textTrainor, Paul A. "Molecular Blueprint for Craniofacial Morphogenesis and Development." In Stem Cells in Craniofacial Development and Regeneration, 1–29. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118498026.ch1.
Full textKatsube, Motoki. "Morphometric Analysis for the Morphogenesis of the Craniofacial Structures and the Evolution of the Nasal Protrusion in Humans." In Multidisciplinary Computational Anatomy, 247–52. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-4325-5_32.
Full textMishina, Yuji, and Nobuhiro Kamiya. "Embryonic Skeletogenesis and Craniofacial Development." In Bone Morphogenetic Proteins: Systems Biology Regulators, 39–72. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47507-3_3.
Full textTerheyden, Hendrik, and Søren Jepsen. "Craniofacial reconstruction with bone morphogenetic proteins." In Bone Morphogenetic Proteins: Regeneration of Bone and Beyond, 133–55. Basel: Birkhäuser Basel, 2004. http://dx.doi.org/10.1007/978-3-0348-7857-9_6.
Full textLavelle, Christopher L. B. "Craniofacial morphogenesis." In Applied Oral Physiology, 176–82. Elsevier, 1988. http://dx.doi.org/10.1016/b978-0-7236-0818-9.50023-6.
Full textMerlo, Giorgio R., Annemiek Beverdam, and Giovanni Levi. "Dlx genes in craniofacial and limb morphogenesis">Dlx genes in craniofacial and limb morphogenesis." In Murine Homeobox Gene Control of Embryonic Patterning and Organogenesis, 107–32. Elsevier, 2003. http://dx.doi.org/10.1016/s1569-1799(03)13004-3.
Full textDudas, Marek, and Vesa Kaartinen. "TGF-β Superfamily and Mouse Craniofacial Development: Interplay of Morphogenetic Proteins and Receptor Signaling Controls Normal Formation of the Face." In Current Topics in Developmental Biology, 65–133. Elsevier, 2005. http://dx.doi.org/10.1016/s0070-2153(05)66003-6.
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