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1

Teracine, Edson Baptista, Renata Cristina Bigaram Della Volpe, and Fernando Luis de Castro Miquelino. CPqD 30 anos. Campinas, SP: CPqD, 2006.

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2

plc, Barbour Index, ed. CPD briefing. Windsor: Barbour Index plc, 1995.

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plc, Barbour Index, ed. CPD briefing. Windsor: Barbour Index plc, 1999.

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plc, Barbour Index, ed. CPD briefing. Windsor: Barbour Index plc, 1996.

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5

British Library. Dept. of Printed Books. CPM plus. [London]: Bowker-Saur, 1993.

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6

plc, Barbour Index, ed. CPD briefing. Windsor: Barbour Index plc, 1997.

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7

Azambuja, Jocelin. CPMs & APMs: Uma revolução comunitária. Porto Alegre: EdiPUCRS, 2008.

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8

United States. Administrative Office of the United States Courts. Human Resources Division., ed. Court personnel management systems: CPMS. Washington, DC: The Division, 1993.

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9

United States. Administrative Office of the United States Courts. Human Resources Division, ed. CPMS: Court Personnel Management System. Washington, DC (1 Columbus Circle, NE, Room 5-500B, Washington 20544): Administrative Office of the U.S. Courts, Human Resources Division, 1993.

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10

Physiotherapy, Chartered Society of, ed. The CPD process. London: Chartered Society of Physiotherapy, 2000.

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11

Janet, Grant, Chambers Ellie, Jackson Gordon, Open University, and Joint Centre for Education in Medicine., eds. The Good CPD guide: A practical guide to managed CPD. Sutton, Surrey: Reed Healthcare Publishing, 1999.

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12

FPGA/CPLD ying yong ji shu. 2nd ed. Bei jing: Dian zi gong ye chu ban she, 2014.

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13

O'Brien, James Jerome. CPM in construction management. 4th ed. New York: McGraw-Hill, 1993.

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14

Development, United States Office of Community Planning and. CPD administrated housing rehabilitation. Washington, D.C: U.S. Dept. of Housing and Urban Development, Office of Community Planning and Development, 1989.

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15

L, Plotnick Fredric, ed. CPM in construction management. 7th ed. New York: McGraw-Hill, 2010.

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16

Assurance, Institute of Quality, ed. CPD: Continuing professional development. London: Institute of Quality Assurance, 1995.

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17

O'Brien, James Jerome. CPM in construction management. 7th ed. New York: McGraw-Hill, 2010.

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18

O'Brien, James Jerome. CPM in construction management. 7th ed. New York: McGraw-Hill, 2010.

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19

O'Brien, James Jerome. CPM in construction management. 5th ed. New York: McGraw-Hill, 1999.

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20

Good psychiatric practice: CPD. London: Royal Colege of Psychiatrists, 2001.

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21

O'Brien, James Jerome. CPM in construction management. 6th ed. New York: McGraw-Hill, 2005.

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22

Roddy, Edward, and Michael Doherty. Calcium pyrophosphate crystal deposition (CPPD). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0142.

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Calcium pyrophosphate crystal deposition (CPPD) in articular cartilage is a common age-related phenomenon. Recent important advances in our understanding of the pathophysiology of pyrophosphate metabolism include the identification of a mutation within the ANK gene which associates with familial CPPD, and elucidation of the interleukin-1β‎ (IL-1β‎)-dependent mechanisms by which crystals invoke an inflammatory response. Risk factors for CPPD include age, prior joint damage and osteoarthritis, genetic factors, and occasionally metabolic diseases (hyperparathyroidism, haemochromatosis, hypomagnesaemia, and hypophosphatasia). CPPD is commonly asymptomatic or may present as osteoarthritis with CPPD, acute calcium pyrophosphate (CPP) crystal arthritis, or chronic CPP crystal inflammatory arthritis. Although radiographic chondrocalcinosis is often taken to be synonymous with CPPD, other calcium crystals can also have this appearance and definitive diagnosis requires identification of CPP crystals by compensated polarized light microscopy of aspirated synovial fluid. Recently, the ultrasonographic appearances of CPPD have been described. Treatment of CPPD is targeted to the clinical presentation. Acute CPP crystal arthritis is treated by aspiration and injection of glucocorticosteroid, local ice packs, non-steroidal anti-inflammatory drugs (NSAIDS), low-dose colchicine, oral or parenteral glucocorticosteroids, or adrenocorticotrophic hormone (ACTH). Treatment of osteoarthritis with CPPD is very similar to the treatment of osteoarthritis alone. There is no specific therapy for chronic CPP crystal inflammatory arthritis: options include NSAID, low-dose colchicine, low-dose glucocorticosteroid, methotrexate, and hydroxychloroquine. Recommendations for the management of CPPD are derived from a small evidence base and largely based on clinical experience and extrapolation from gout. Further research into diagnosis and management including novel treatment strategies such as IL-1β‎ blockade is much needed.
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23

Abhishek, Abhishek, and Michael Doherty. Epidemiology and risk factors for calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0048.

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Calcium pyrophosphate crystal deposition (CPPD) is rare in younger adults but becomes increasingly common over the age of 55 years, especially at the knee. Ageing and osteoarthritis (OA) are the main attributable risk factors. Hyperparathyroidism, hypomagnesaemia, haemochromatosis, and hypophosphatasia are other less common risk factors. Rare families with familial CPPD have been reported from many different parts of the world, and mainly present as young-onset polyarticular CPPD. Recent studies suggest that CPPD occurs as the result of a generalized constitutional predisposition and may also associate with low cortical bone mineral density. Previous meniscectomy, joint injury, and constitutional knee malalignment are local biomechanical risk factors specifically for knee chondrocalcinosis. Although associated with OA, current evidence suggests that CPPD does not associate with development or progression of OA.
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24

CPD briefing. Windsor: Barbour Index plc, 1998.

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25

Publication, Newbee. CPD Journal. Independently Published, 2020.

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26

UK Chamber of Shipping Staff, Witherby Publishing Group, and ECDIS Ltd Staff. ECDIS CPD. Witherby & Company, 2019.

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27

Cyliax, Ingo. Designing With Fpga/Cpld. Prompt Pubns, 2000.

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28

Abhishek, Abhishek, and Michael Doherty. Clinical features of calcium pyrophosphate crystal deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0050.

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Calcium pyrophosphate deposition (CPPD) occurs in the elderly, and is commonly asymptomatic. However, it can cause acute calcium pyrophosphate (CPP) crystal arthritis, chronic CPP crystal inflammatory arthritis, and is frequently present in joints with osteoarthritis (OA). Acute CPP crystal arthritis presents with rapid onset of acute synovitis, which frequently affects the knees, wrists, shoulders, and elbows. It can mimic sepsis in the elderly, and may require hospital admission. Patients with CPPD plus OA may have more inflammatory signs and symptoms (e.g. joint swelling, stiffness) than those with OA alone. Additionally, patients with CPPD plus OA may also have intermittent attacks of acute CPP crystal arthritis. Some patients with CPPD may have more chronic inflammatory joint involvement and are classified as chronic CPP crystal inflammatory arthritis. This chapter describes the clinical features and differential diagnosis of common clinical manifestations of CPPD and outlines some of its rarer manifestations.
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29

Group, CPM. The CPM Platinum Yearbook, 2006 (CPM Platinum Yearbook). Wiley, 2006.

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30

Abhishek, Abhishek, and Michael Doherty. Investigations of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0051.

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Joint aspiration and microscopic examination of the aspirated synovial fluid remains the gold standard for the diagnosis of calcium pyrophosphate crystal deposition (CPPD). If synovial fluid aspiration is not feasible, plain radiography and/or ultrasound scanning may be used to detect chondrocalcinosis (CC) which predominantly occurs due to calcium pyrophosphate (CPP) crystals, and this can be used as a diagnostic surrogate for CPPD as suggested by the EULAR Task Force. Acute CPP crystal arthritis often associates with a brisk acute phase response (elevated C-reactive protein (CRP) and/or erythrocyte sedimentation rate (ESR), plasma viscosity) and neutrophilia. A mildly raised CRP and/or ESR may be present in chronic CPP crystal inflammatory arthritis. On the contrary, asymptomatic CC, or CPPD with osteoarthritis does not cause raised acute phase reactants. As CPPD most commonly occurs due to increasing age and osteoarthritis, investigations to screen for underlying metabolic abnormalities should be carried out in those with early-onset CPPD (under 55 years), or in those with florid polyarticular CC. As hyperparathyroidism gets more common with ageing its presence should be specifically sought in all age groups. Tests for other predisposing metabolic conditions should only be carried out in the presence of specific clinical features. Genotyping for mutations, especially in the ANKH gene, may be warranted in those with a family history of premature CPPD and no evidence of inherited metabolic predisposition, but such testing is unavailable to most clinicians.
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31

Murdoch, John, Sandi Murdoch, and Sue Rivers. Cpd Legal Update. Estates Gazette Ltd, 1997.

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32

Murdoch, John, Sandi Murdoch, and Christopher Opstad. CPD Legal Update. Estates Gazette Ltd, 1994.

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33

Murdoch, John, Sandi Murdoch, and Christopher Opstad. CPD Legal Update. Estates Gazette Ltd, 1995.

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34

PILBERY. Standby Cpd: Tachyarrhythmias. Class Publishing, 2016.

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35

MCPHERSON. Standby Cpd: Hyponatraemia. Class Publishing, 2017.

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36

PILBERY. Standby Cpd: Hypoglycaemia. Class Publishing, 2011.

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37

PILBERY. Standby Cpd: Tracheostomies. Class Publishing, 2014.

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38

CPM Algebra Mathematics. CPM, 2000.

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39

Group, CPM. The CPM Platinum Group Metals Yearbook 2007 (CPM Platinum Yearbook). Wiley, 2007.

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40

Components. Ieee/Cpmt International Electronics Manufacturing Technology Symposium 1994 (Ieee/Cpmt International Electronics Manufacturing Technology Symposium). Institute of Electrical & Electronics Enginee, 1994.

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41

Ch36330-tbr. Electronics Manufacturing Technology: 1999 IEEE Cpmt. Ieee, 1999.

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42

Digital Logic Simulation and CPLD Programming. Prentice Hall, 2000.

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43

Staff, IEEE. 2021 IEEE CPMT Symposium Japan (ICSJ). IEEE, 2021.

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44

Ellwein, Christian. Programmierbare Logik mit GAL und CPLD. Oldenbourg, 1998.

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45

Suhail, Saad A. CPM/LOB: New methodology to integrate CPM and line of balance. 1995.

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46

PILBERY. Standby Cpd: Status Epilepticus. Class Publishing, 2012.

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47

Publication, Newbee. Cpd Journal: For Dietitians. Independently Published, 2020.

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48

Group, CPM. CPM PGM Yearbook 2021. CPM Group, 2021.

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49

PILBERY. Standby Cpd: Crush Syndrome. Class Publishing, 2012.

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50

Boyd, Brian. Cpd: The Learning Classroom. Hodder Education Group, 2014.

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