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Books on the topic 'Corticosterone'

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Published: 4 June 2021
Last updated: 1 June 2024

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1

Lanctot, Richard Benjamin. Are corticosterone levels a good indicator of food availability and reproductive performance in a kittiwake colony? Anchorage, Alaska: EVOS Trustee Council, 2003.

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2

Shum, Kar Man Kathy. Effects of antecedent hypoglycemia, antecedent hyperinsulinemia, and antecedent corticosterone on subsequent counterregulation in normal rats. Ottawa: National Library of Canada, 2000.

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3

Harrison, Amanda J. The effects of increased corticosterone levels and auditory stress on food consumption in female wistar rats. Sudbury, Ont: Laurentian University, 2006.

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4

N, Lin Andrew, and Paget Stephen A, eds. Principles of corticosteroid therapy. London: Arnold, 2002.

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5

de, Kloet E. R., Azmitia Efrain C, Landfield Philip W, and New York Academy of Sciences., eds. Brain corticosteroid receptors: Studies on the mechanism, function, and neurotoxicity of corticosteroid action. New York, N.Y: New York Academy of Sciences, 1994.

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6

Parker, James N., and Philip M. Parker. The official patient's sourcebook on antenatal corticosteroid therapy. Edited by Icon Group International Inc and NetLibrary Inc. San Diego, Calif: Icon Health Publications, 2002.

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7

Griffiths, Mark Raymond. Phencyclidine-and corticosteroid-induced apoptosis of striatal neurons. Birmingham: University of Birmingham, 1999.

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8

Enno, Christophers, ed. Topical corticosteroid therapy: A novel approach to safer drugs. New York: Raven Press, 1988.

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9

Verfasser, Kley Hans Kuno, ed. Cortisontherapie: Corticoide in Klinik und Praxis. 9th ed. Stuttgart: Thieme, 1992.

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10

Näsström, Karin. Dentin formation after corticosteroid treatment: A clinical study and an experimental study on rats. Malmö, Sweden: Department of Oral Radiology, Faculty of Odontology, Lund University, 1996.

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11

Whorwood, Christopher B. The pivotal role of 11[beta]-Hydroxysteroid dehydrogenase in the modulation of corticosteroid hormone action. Birmingham: University of Birmingham, 1995.

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12

Ghiculete, Daniela. Initial evaluation of a novel method to assess corticosteroid responsiveness in chronic obstructive pulmonary disease (COPD). Ottawa: National Library of Canada, 2002.

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13

Nyberg, Lena. Glucocorticoid binding proteins: Endocrinological and biochemical studies on the corticosteroid binding globulin and the glucocorticoid receptor. Uppsala: Sveriges Lantbruksuniversitet, 1988.

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14

P, Schleimer Robert, ed. Inhaled steroids in asthma: Optimizing effects in the airways. New York: Marcel Dekker, 2002.

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15

Simmons, Jonathan. Corticosterone: Roles, Research and Insights. Nova Science Publishers, Incorporated, 2017.

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16

Ryan, Jeanne P. The effects of the interaction between corticosterone suppression and peripheral catecholamines on the behavior of brain injured rats. 1985.

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17

Coty, Kevin D. Mechanism of L1210 tumor resistance of protein-malnourished mice: Roles of corticosterone and growth factors in vitro. 1996.

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18

Chiavarini, Katherine E. Rapid effects of corticosterone on stress-related behaviors in an amphibian. 1997.

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19

Newman, Charlotte. The effect of corticosterone and stress on plasma and brain amino acid concentration. 1986.

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20

Youngberg, Jill Annette Meyer. The effect of 3,4,5,3',4',5'- hexachlorobiphenyl on plasma corticosterone and prolactin concentration in the mouse. 1991.

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21

Youngberg, Jill Annette Meyer. The effect of 3,4,5,3',4',5'- hexachlorobiphenyl on plasma corticosterone and prolactin concentration in the mouse. 1991.

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22

Effect of chronic ethanol consumption and moderate intensity endurance training on murine plasma corticosterone concentration. 1992.

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23

Effect of chronic ethanol consumption and moderate intensity endurance training on murine plasma corticosterone concentration. 1992.

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24

Sipp, Tina Leigh. Effect of chronic ethanol consumption and moderate intensity endurance training on murine plasma corticosterone concentration. 1992.

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25

Goldey, Ellen S. Indirect exposure to trichloroethylene during development alters learning and circulating corticosterone levels in male rats. 1986.

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26

Steel, Vivian L. Effects of maternal exposure to trichloroethylene on locomotor activity and circulating corticosterone levels in rat pups. 1985.

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27

Schlabach, Gretchen A. The effects of training and detraining on corticosterone rhythms and dietary fat selection in the Osborne-Mendel rat. 1992.

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28

The effects of training and detraining on corticosterone rhythms and dietary fat selection in the Osborne-Mendel rat. 1991.

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29

Rees, Stephanie Lauranne. Effects of corticosterone on maternal behaviour: Roles of parity, gonadal hormones, and early experience : y Stephanie Lauranne Rees. 2006.

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30

The relation of corticosterone and circadian rhythms in the ages syrian hamster: Their effect on the acquisition of hippocampal-based learning and memory. Ottawa: National Library of Canada, 2000.

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31

Corticosteroid-Induced Osteoporosis. Taylor & Francis Group, 1999.

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32

Lin, A. N., and Stephen A. Paget. Principles of Corticosteroid Therapy. Hodder Education Group, 2000.

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33

Primer on Corticosteroid-Induced Osteoporosis. Lippincott Williams & Wilkins, 2000.

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34

Culinary Dictionary for Corticosteroid Therapy: Food Encyclopedia. Independently Published, 2020.

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35

Publications, ICON Health. The Official Patient's Sourcebook on Antenatal Corticosteroid Therapy. Icon Health Publications, 2002.

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36

(Editor), Efrain C. Azmitia, E. R. De Kloet (Editor), New York Academy of Sciences (Corporate Author), and Philip W. Landfield (Editor), eds. Brain Corticosteroid Receptors: Studies on the Mechanism, Function, and Neurotoxicity of Corticosteroid Action (Annals of the New York Academy of). New York Academy of Sciences, 1995.

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37

Kloet, Ronald De. Brain Corticosteroid Receptors: Studies on the Mechanism, Function, and Neurotoxicity of Corticosteroid Action (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1994.

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38

Enno, Christophers. Topical Corticosteroid Therapy: A Novel Approach to Safer Drugs. Raven Pr, 1988.

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39

Foster, Helen, and Paul A. Brogan, eds. Specialized therapeutic approaches. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199592630.003.0008.

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Corticosteroid intra-articular injections 390Biologic therapies for paediatric rheumatological diseases 393Approvals for use of biologic therapies 399Medicines for children and paediatric rheumatology 401Haematopoietic stem cell transplantation 405(See also BSPAR guidelines for treatments used in paediatric rheumatology, p 415)Intra-articular corticosteroid injections are increasingly used as they allow rapid symptom control and allow time for other therapies, such as methotrexate (MTX) to have their effect. They often remove the need for the use of oral or IV corticosteroids and avoidance of side effects. There is no limit to the number of joints that can be injected at any one time or the total dose of corticosteroid....
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40

Shanahan, Jeanette. FLU0CINONIDE a Strong Contemporary Corticosteroid Capable of Curing Inflammatory Skin Illnesses. Independently Published, 2019.

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41

Bergnaum, Garret. FLUOCINOLONE ACETONIDE: Topical Corticosteroid for the Handling of Inflammatory Skin Conditions. Independently Published, 2019.

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42

Smith, Jay, and Jacob Sellon. Elbow Injections: Ultrasound. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199908004.003.0044.

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This chapter discusses sonographically guided elbow, tendon, and joint procedures used in the management of patients presenting with tendon and joint disorders of the elbow. Two sonographically guided corticosteroid injection techniques have been used for common extensor tendinosis, a superficial technique and a deep technique. Sonographically guided percutaneous longitudinal tenotomy has also been described for chronic flexor-pronator tendinosis. Distal biceps tendinopathy is a broad term that includes both inflammatory (tendinitis) and chronic degenerative (tendinosis) conditions. As the proximal portion of the distal biceps tendon can be easily palpated anteriorly, an anterior injection approach will typically provide uncomplicated access to the tendon and/or adjacent bursal fluid. Common elbow conditions are amenable to intra-articular elbow aspiration and injections (typically with corticosteroid).
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43

Bright, Doctor Amos. Dermovate: An Extra-Powerful Topical Corticosteroid Cream for Treating Inflammations and Any Other Skin Conditions. Independently Published, 2019.

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44

Hosu, Liana G., and Lori A. Aronson. Hypopituitarism. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0048.

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Hypopituitarism refers to decreased secretion of pituitary hormones, which can result from diseases of the pituitary gland or hypothalamus. Surgery in the presence of untreated panhypopituitarism is rare. It is important to recognize the presence of hypopituitarism and most importantly adrenal insufficiency, a condition that can result in mortality from adrenal crisis without stress-dose corticosteroid treatment.
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45

Wilson, John W., and Lynn L. Estes. Select Opportunistic Infections in Adult Patients With HIV. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199797783.003.0143.

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•Risk factors: Ubiquitous organism; CD4 count <200/mcL; chronic corticosteroid or other immunosuppressive drug therapy•Clinical disease• Exertional dyspnea, fever, nonproductive cough, and chest discomfort that gets worse over days to weeks• Hypoxemia; chest radiographs vary (most commonly show diffuse bilateral, symmetrical interstitial infiltrate but may be relatively normal early in course and can have atypical presentation)...
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46

Klocko, Rosanna. CLθBETASθl PRθPIONATE: An Exceptionally Powerful Topical Corticosteroid Treatment for Inflammatory Skin Conditions. Independently Published, 2019.

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47

Miller, Aaron E., and Teresa M. DeAngelis. Hashimoto’s Encephalopathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0018.

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Hashimoto’s encephalopathy (HE) is a rare but potentially corticosteroid responsive disorder, which should be considered in cases of encephalopathy of unclear etiology. Hashimoto’s thyroiditis usually does not accompany HE and thyroid function is generally clinically normal. In this chapter, we review the differential diagnostic suspicions for a case of encephalopathy of unclear etiology and respective aspects that raise concern for HE. In addition, we discuss the utility of diagnostic testing and options for therapeutic management.
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48

Abhishek, Abhishek, and Michael Doherty. Treatment of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0052.

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The treatment of calcium pyrophosphate crystal deposition (CPPD) is mainly symptomatic. Acute calcium pyrophosphate (CPP) crystal synovitis should be treated with rest, local application of ice packs, joint aspiration, and/or intra-articular corticosteroid injection (once joint sepsis has been excluded). Oral colchicine or prednisolone may be used if joint aspiration and/or injection are not feasible. Anti-inflammatory agents (with proton pump inhibitors) may be used but in general these should be avoided as most patients with acute CPP crystal arthritis are elderly, and at a high risk of gastrointestinal and renal complication of non-steroidal anti-inflammatory drug (NSAIDs). Principles of management of CPPD with osteoarthritis (OA) are identical to those for isolated OA. However, patients may have more inflammatory signs and symptoms and periodic joint aspiration and corticosteroid injection may be required more often than in isolated OA. Oral NSAIDs (with gastro-protection), colchicine, low-dose corticosteroids, hydroxychloroquine, and radiosynovectomy have been suggested as options for the treatment of chronic CPP crystal arthritis. There is growing interest in use of anti-interleukin-1 agents for acute or chronic CPP crystal arthritis but the efficacy of these agents has not been formally studied, and their use should be considered on an individual basis.
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49

Keh, Didier. Steroids in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0054.

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The benefit of prolonged application of moderate-dose corticosteroids in systemic inflammatory diseases remains controversial. In critical illness, the endogenous cortisol effect may become insufficient due to adrenal dysfunction and corticosteroid resistance to counterbalance an exaggerated and protracted inflammatory response, which has been termed ‘critical illness-related corticosteroid insufficiency’ (CIRCI). There is evidence that moderate-dose hydrocortisone (200–300 mg/day) significantly fastens shock reversal in patients with septic shock, but may improve survival probably only in patients with high risk of death. Thus, therapy should be considered only in refractory shock with poor response to fluid administration and vasopressor therapy. The indication should be based on clinical judgement and not on cortisol measurement. The application prolonged of moderate-dose methylprednisolone (1 mg/kg/day) was found to be most effective in early acute respiratory distress syndrome, and associated with improved lung function, reduction of mechanical ventilation, and faster discharge from the ICU, but a survival benefit was found only in pooled data, including cohort studies. A continuous infusion and weaning of corticosteroids may be preferable to bolus applications and abrupt withdrawal to avoid side effects such as rebound of inflammation and shock, glucose variability, or respiratory failure. There is currently no evidence that prolonged application of moderate-dose corticosteroids increase the risk of secondary infections or muscle weakness, but infection surveillance should be implemented and combination with muscle relaxants be avoided.
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50

Rajakariar, Ravindra, and Muhammad M. Yaqoob. The patient with sarcoidosis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0156.

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Renal involvement in sarcoidosis is common and often under-recognized. The most frequent manifestation is acute kidney injury secondary to hypercalcaemia and granulomatous tubulointerstitial nephritis. The latter can lead to both acute kidney injury and to slowly progressive chronic renal impairment with concomitant chronic damage seen on histology. This chapter describes the types of renal disease that may occur in sarcoidosis and the pathogenesis, clinical presentation, diagnosis, and treatment of the patient with sarcoidosis. Corticosteroid therapy is the cornerstone of therapy. In patients with granulomatous tubulointerstitial nephritis, the authors recommend long-term, low-dose maintenance steroids.
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