Relevant bibliographies by topics / Control of the porcine proliferative enteropathies

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  2. Dissertations / Theses
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Academic literature on the topic 'Control of the porcine proliferative enteropathies'

Author: Grafiati
Published: 4 June 2021
Last updated: 21 February 2023

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Journal articles on the topic "Control of the porcine proliferative enteropathies"

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McOrist, S., N. MacIntyre, C. R. Stokes, and G. H. Lawson. "Immunocytological responses in porcine proliferative enteropathies." Infection and Immunity 60, no. 10 (1992): 4184–91. http://dx.doi.org/10.1128/iai.60.10.4184-4191.1992.

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McOrist, S., R. Boid, G. Lawson, and I. McConnell. "Monoclonal antibodies to intracellular campylobacter-like organisms of the porcine proliferative enteropathies." Veterinary Record 121, no. 18 (October 31, 1987): 421–22. http://dx.doi.org/10.1136/vr.121.18.421.

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Lawson, G., S. McOrist, A. Rowland, E. McCartney, and L. Roberts. "Serological diagnosis of the porcine proliferative enteropathies: implications for aetiology and epidemiology." Veterinary Record 122, no. 23 (June 4, 1988): 554–57. http://dx.doi.org/10.1136/vr.122.23.554.

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McOrist, Steven, Gordon H. K. Lawson, Douglas J. Roy, and Richard Boid. "DNA analysis of intracellular campylobacter-like organisms associated with the porcine proliferative enteropathies: novel organism proposed." FEMS Microbiology Letters 69, no. 3 (June 1990): 189–93. http://dx.doi.org/10.1111/j.1574-6968.1990.tb04227.x.

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McOrist, S., R. Boid, and G. H. Lawson. "Antigenic analysis of Campylobacter species and an intracellular Campylobacter-like organism associated with porcine proliferative enteropathies." Infection and Immunity 57, no. 3 (1989): 957–62. http://dx.doi.org/10.1128/iai.57.3.957-962.1989.

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McOrist, S., M. F. H. Shearn, and J. Morgan. "Control of porcine proliferative enteropathy by oral administration of chlortetracycline." Veterinary Record 144, no. 2 (January 9, 1999): 48–49. http://dx.doi.org/10.1136/vr.144.2.48.

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Guedes, R. M. C., S. A. Franca, G. S. Machado, M. A. Blumer, and E. C. da Costa Cruz. "Use of tylvalosin-medicated feed to control porcine proliferative enteropathy." Veterinary Record 165, no. 12 (September 19, 2009): 342–45. http://dx.doi.org/10.1136/vr.165.12.342.

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Lee, Sang Won, Tae Jong Kim, Seung Yong Park, Chang Sun Song, Hyung Kwan Chang, Jae Kil Yeh, Hye In Park, and Joong Bok Lee. "Prevalence of porcine proliferative enteropathy and its control with tylosin in Korea." Journal of Veterinary Science 2, no. 3 (2001): 209. http://dx.doi.org/10.4142/jvs.2001.2.3.209.

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9

Holyoake, PK, A. Collins, M. Donahoo, R. Lising, and D. Emery. "Identifying obstacles to reducing the use of antibiotics to control porcine proliferative enteropathy." Australian Veterinary Journal 87, no. 1-2 (January 2009): 33–34. http://dx.doi.org/10.1111/j.1751-0813.2008.00372.x.

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10

Otoni, Luisa V. A., Michelle P. Gabardo, Núbia R. Macêdo, Mariane M. Wagatsuma, Marina M. Pereira, and Roberto Maurício C. Guedes. "Tylosin injectable for the treatment of porcine proliferative enteropathy in experimentally inoculated pigs." Pesquisa Veterinária Brasileira 39, no. 3 (March 2019): 168–74. http://dx.doi.org/10.1590/1678-5150-pvb-6066.

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ABSTRACT: Porcine proliferative enteropathy (PPE) is one of the most common enteric diseases in growing and finishing pigs. PPE is characterized by reduced growth performance, accompanied or not by diarrhea. PPE is highly prevalent in several countries of the Americas, Europe and Asia, causing high economic losses in swine herds. The most common form of PPE control in pigs is antibiotic therapy. The objective of this study was to evaluate a new product based on tylosin injectable (Eurofarma Laboratórios S.A.) to control PPE in experimentally inoculated animals. Sixty 5-week-old pigs with mean weight of 9.5kg were divided into two experimental groups of 30 animals: medication and control. All pigs were challenged with Lawsonia intracellularis, the etiologic agent of PPE, on day zero. Fecal score, body condition score, and behavior were daily evaluated. Pigs were weighted on days -2, 13 and 21 of the experiment. Pigs in the Medication Group received tylosin injectable 13 days after inoculation, in three doses with a 12-hour interval between them. Pigs in the Control Group received injectable saline solution following the same protocol. In the Control Group, 23pigs presented with diarrhea before day 13. After day 13, the number of diarrheic animals in this group was reduced to 17. In the Medication Group, 26 pigs presented with diarrhea in the initial period, and in the period after medication, only 11 animals had diarrhea. The score of gross intestinal PPE lesions in the Medication Group was lower than that in the Control Group (p=0.031). The Medication Group also showed lower score for Lawsonia intracellularis antigen-labeling by immunohistochemistry compared with that of the Control Group (p=0.032), showing lower level of infection. These results demonstrate that tylosin injectable (Eurofarma Laboratórios S.A.), administrated in three doses (1mL/20kg) every 12 hours, was effective for the control of PPE in experimentally inoculated pigs.
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Dissertations / Theses on the topic "Control of the porcine proliferative enteropathies"

1

Collins, Alison Marie. "A Study of the Lawsonia intracellularis-induced porcine proliferative enteropathies." University of Sydney. Veterinary Science, 2001. http://hdl.handle.net/2123/835.

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The porcine proliferative enteropathies (PPE) are a group of diseases ranging from intestinal adenomatosis (PIA), a chronic condition causing reduced growth rates in post weaning pigs, to the often fatal proliferative haemorrhagic enteropathy (PHE), resulting in intestinal haemorrhage. PHE predominantly occurs in older and heavier pigs than the chronic disease PIA. This thesis examined whether the age when susceptible pigs are infected affects the clinical response to L.intracellularis infection. The characteristic pathologic lesion of PPE is the abnormal proliferation of crypt epithelial cells in the ileum and colon. Closely associated with this proliferation is the presence of an obligately intracellular bacterium, Lawsonia intracellularis. Characterisation of L.intracellularis was performed in in-vitro co-cultures of L.intracellularis extracted from PHE-affected mucosa. The efficacy of antimicrobials to inhibit the growth of L.intracellularis in-vitro was evaluated and compared with isolates cultured in the United Kingdom. The results were analysed with respect to medication strategies currently used to control PPE in piggeries. PPE occurs in virtually all piggery management systems, including newly developed systems that are aimed at improving the herd health, such as segregated early weaning and multiple site production. PPE is currently controlled in Australia with the routine addition of antimicrobials in pig feed, in particular olaquindox. Recommendations to reduce the use of feed-based antibiotics in Australia require the development of alternate strategies to control diseases such as PPE. Sequential outbreaks of PHE reported in minimal disease herds suggested that pigs could develop immunity to disease. An experimental model of L.intracellularis infection was developed in this thesis to demonstrate that immunity to re-infection with L.intracellularis could be developed. Infection was monitored by detection of faecal shedding of L.intracellularis and serum IgG antibodies against L.intracellularis. Two in-feed antimicrobial strategies were analysed in this thesis for their ability to induce the development of immunity to L.intracellularis, while avoiding clinical signs of disease. The first strategy evaluated the use of low levels of in-feed antimicrobials to allow subclinical infection and the development of immunity. The second strategy evaluated the use of high levels of in-feed antimicrobials to terminate infection two weeks after exposure to L.intracellularis. Gaining a greater understanding of how L.intracellularis infection is spread both within and between piggeries will enable the development of management strategies to control the spread of infection. This thesis examined the possibility that other species in contact with pigs and piggeries such as rats, mice and birds may transmit infection to pigs. The transmission of infection between pigs via the faecal/oral route was also examined, as was the survival and infectivity of L.intracellularis over time. Ultimately this thesis aimed to understand the pattern of L.intracellularis infection and the survival and transmission of L.intracellularis in order to develop effective control measures for PPE, especially in minimal disease herds.
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2

Collins, Alison Marie. "A Study of the Lawsonia intracellularis-induced porcine proliferative enteropathies." Thesis, The University of Sydney, 2000. http://hdl.handle.net/2123/835.

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The porcine proliferative enteropathies (PPE) are a group of diseases ranging from intestinal adenomatosis (PIA), a chronic condition causing reduced growth rates in post weaning pigs, to the often fatal proliferative haemorrhagic enteropathy (PHE), resulting in intestinal haemorrhage. PHE predominantly occurs in older and heavier pigs than the chronic disease PIA. This thesis examined whether the age when susceptible pigs are infected affects the clinical response to L.intracellularis infection. The characteristic pathologic lesion of PPE is the abnormal proliferation of crypt epithelial cells in the ileum and colon. Closely associated with this proliferation is the presence of an obligately intracellular bacterium, Lawsonia intracellularis. Characterisation of L.intracellularis was performed in in-vitro co-cultures of L.intracellularis extracted from PHE-affected mucosa. The efficacy of antimicrobials to inhibit the growth of L.intracellularis in-vitro was evaluated and compared with isolates cultured in the United Kingdom. The results were analysed with respect to medication strategies currently used to control PPE in piggeries. PPE occurs in virtually all piggery management systems, including newly developed systems that are aimed at improving the herd health, such as segregated early weaning and multiple site production. PPE is currently controlled in Australia with the routine addition of antimicrobials in pig feed, in particular olaquindox. Recommendations to reduce the use of feed-based antibiotics in Australia require the development of alternate strategies to control diseases such as PPE. Sequential outbreaks of PHE reported in minimal disease herds suggested that pigs could develop immunity to disease. An experimental model of L.intracellularis infection was developed in this thesis to demonstrate that immunity to re-infection with L.intracellularis could be developed. Infection was monitored by detection of faecal shedding of L.intracellularis and serum IgG antibodies against L.intracellularis. Two in-feed antimicrobial strategies were analysed in this thesis for their ability to induce the development of immunity to L.intracellularis, while avoiding clinical signs of disease. The first strategy evaluated the use of low levels of in-feed antimicrobials to allow subclinical infection and the development of immunity. The second strategy evaluated the use of high levels of in-feed antimicrobials to terminate infection two weeks after exposure to L.intracellularis. Gaining a greater understanding of how L.intracellularis infection is spread both within and between piggeries will enable the development of management strategies to control the spread of infection. This thesis examined the possibility that other species in contact with pigs and piggeries such as rats, mice and birds may transmit infection to pigs. The transmission of infection between pigs via the faecal/oral route was also examined, as was the survival and infectivity of L.intracellularis over time. Ultimately this thesis aimed to understand the pattern of L.intracellularis infection and the survival and transmission of L.intracellularis in order to develop effective control measures for PPE, especially in minimal disease herds.
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3

Collins, Alison Marie. "A study of the Lawsonia intracellularis-induced porcine proliferative enteropathies." Connect to full text, 2000. http://hdl.handle.net/2123/835.

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Thesis (Ph. D.)--University of Sydney, 2001.
Includes tables. Title from title screen (viewed Apr. 22, 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Dept. of Veterinary Clinical Sciences, Faculty of Veterinary Science. Degree awarded 2001; thesis submitted 2000. Includes bibliography. Also available in print form.
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4

Okereke, Rowland Eugene. "Infection of cell cultures with Campylobacter derived from the porcine proliferative enteropathies." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/30605.

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Books on the topic "Control of the porcine proliferative enteropathies"

1

Ontario. Ministry of Agriculture and Food. Intestinal Adenomatosis Complex (Porcine Proliferative Enteropathies). S.l: s.n, 1987.

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Conference papers on the topic "Control of the porcine proliferative enteropathies"

1

BOUTHERIN-FALSON, O., and N. BLAES. "MODULATION of PROSTACYCLIN PRODUCTION BY HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS WITH ECGF/HEPARIN MEDIUM : ROLE OF CELLULAR DENSITY AT CONFLUENCE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643376.

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Prostacyclin (PGI2) is a major product of arachidonic acid metabolism in vascular endothelial cells. In addition to the role of exogenous agents, its production could be modulated by culture conditions : proliferative state, medium renewal, subcultivation... The use of endothelial cell growth factor (ECGF) associated with heparin has been shown to improve human endothelial cell proliferation. Here we report that human umbilical vein endothelial cells (HUVEC) grown in that medium produce less prostacyclin than without growth factor.HUVEC were cultured in RPMI-199 1:1 + 20% fetal calf serum, added or not with ECGF (Bovine hypothalamus extract BTI Cambridge, 24 ug/ml) and heparin (from porcine intestinal mucosa, Signa, 90 ug/ml). After 4 days in culture, medium was removed and replaced by Tyrode Hepes buffer and basal production was measured after 20 min. Cells were then submitted to 5 min thrombin to assess PGI2 production in stimulated conditions. PGI2 production was estimated by specific radioimmunoassay for 6 keto PGFjalpha. For each point, cell number in the culture was counted after Trypsin EDTA treatment. In the present study, cells grown in ECGF-heparin medium produce lower amount of PGI2, compared to heparin or control medium. This result was observed in both basal and stimulated conditions. For each medium (ECGF-heparin, heparin, control), correlations between PGI2 production per cell and log cell density were shown to be significantly negative.These observations suggest that ECGF effect on PGI2 production could be a consequence of its growth factor activity, notably by the fact that it leads to an endothelial monolayer made of more numerous cells. Since it is now suggested by a number of clinical observations that PGI2 is rather produced in pathological conditions, culture models showing a weak production of PGI2 appear in that connection doser to the physiological conditions.
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