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Journal articles on the topic 'Congenital heart disease'

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Published: 4 June 2021
Last updated: 29 July 2024

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1

Martins, Cristiane. "Congenital heart disease." Clinical Cardiology and Cardiovascular Interventions 3, no. 11 (November 20, 2020): 01–02. http://dx.doi.org/10.31579/2641-0419/097.

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2

Taksande, Amar, and Sachin Dhamke. "Critical Congenital Heart Disease in Newborns." Pediatric Education and Research 5, no. 2 (2017): 87–95. http://dx.doi.org/10.21088/per.2321.1644.5217.17.

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3

Petrovna, Askaryans Vera, and Xikmatov Javoxirbek Sherali ogli. "CONGENITAL HEART DEFECTS." Eurasian Journal of Medical and Natural Sciences 03, no. 02 (February 1, 2023): 194–99. http://dx.doi.org/10.37547/ejmns-v03-i02-p1-32.

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Congenital heart defects (TYN), also known as congenital heart anomaly and congenital heart disease, are defects in the structure of the heart or great vessels present at birth. Congenital heart defects are classified as cardiovascular diseases. Signs and symptoms depend on the specific type of defect. Symptoms can be harmless or life-threatening. If present, symptoms may include rapid breathing, bluish skin (cyanosis), low weight, and fatigue. Congenital heart defects do not cause chest pain. Congenital heart defects are often not associated with other diseases. A complication of congenital heart defects is heart failure.
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4

Siu, S. C. "CONGENITAL HEART DISEASE: Heart disease and pregnancy." Heart 85, no. 6 (June 1, 2001): 710–15. http://dx.doi.org/10.1136/heart.85.6.710.

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5

Kelleher, Andrea A. "Adult congenital heart disease (grown-up congenital heart disease)." Continuing Education in Anaesthesia Critical Care & Pain 12, no. 1 (February 2012): 28–32. http://dx.doi.org/10.1093/bjaceaccp/mkr045.

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6

Allan, L. "CONGENITAL HEART DISEASE: Antenatal diagnosis of heart disease." Heart 83, no. 3 (March 1, 2000): 367. http://dx.doi.org/10.1136/heart.83.3.367.

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7

Rathod, Bipin. "Anthropometric Profiles of Children with Congenital Heart Disease." Pediatric Education and Research 5, no. 1 (2017): 23–27. http://dx.doi.org/10.21088/per.2321.1644.5117.5.

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8

K., Thaslima, and Sunil Mhaske. "Neurodevelopmental Status of Children with Congenital Heart Disease." Indian Journal of Trauma and Emergency Pediatrics 8, no. 2 (2016): 99–102. http://dx.doi.org/10.21088/ijtep.2348.9987.8216.10.

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9

Qureshi, Shakeel A., and Lee Benson. "Congenital heart disease." Future Cardiology 8, no. 2 (March 2012): 143–47. http://dx.doi.org/10.2217/fca.12.22.

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10

Puri, Kriti, Hugh D. Allen, and Athar M. Qureshi. "Congenital Heart Disease." Pediatrics in Review 38, no. 10 (October 2017): 471–86. http://dx.doi.org/10.1542/pir.2017-0032.

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11

MANZOOR, IRAM, SEEMA DAUD, and NOREEN RAHAT HASHMI. "CONGENITAL HEART DISEASE." Professional Medical Journal 17, no. 01 (March 10, 2010): 128–34. http://dx.doi.org/10.29309/tpmj/2010.17.01.2093.

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Objective: To assess the parental knowledge and myths related to etiology of congenital heart disease. Design: A crosssectional descriptive study. Place and Duration of Study: Out patient department, indoor and echocardiography rooms of Children Hospitaland Institute of Child Health (CH & ICH) Lahore, in two weeks of October, 2007. Patients and Methods: Convenient sampling technique wasused to recruit 299 children between 1 month and 14 years of age, suffering from congenital heart disease. Cases of rheumatic fever andassociated congestive cardiac failure were excluded from the study group. A structured questionnaire was used to interview parents of the studygroup. Descriptive statistics was determined in terms of percentages. Results: Out of total 299 patients included in this study, 132 were in theage group of 1 -5 years (44%), with the mean age 1.86 years ± 0.886 SD. 200 were males (67 %) and 99 were females (33%). The respondentsincluded 71% mothers and 27% fathers. Ventricular septal defect (VSD) was the most common disease encountered in 128 children (43%).Regarding the causes of congenital cardiac defects, parents blamed cousin marriage (41%), inheritance (26%), increased maternal age(16%), maternal smoking(16%) and paternal smoking (39%). Other responses included evil spirits, moon and sun eclipses and bad wishes/evileye. 57% of the respondents said that there is no relationship between gender and congenital cardiac malformation and 85% respondentsbelieved that there is no relation with the food taken in pregnancy and subsequent cardiac defects. High Salt intake was considered as anassociated factor by 47 (15%) of the respondents. Naming the cause of cardiac malformations, 41% of the parents said drugs taken inpregnancy, 13% of the respondents blamed alcohol, cocaine and opium intake. Among parents, 73% were aware of the fact that maternal healthis important for disease causation in neonates but could not name any particular disease. Conclusion: The parents of those children, who havecongenital malformation, have different myths and fallacies associated with disease causation. Adequate health education strategies shouldbe adopted towards mass dissemination of information in the community about this important issue.
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12

Scott, Michael, and Ashley E. Neal. "Congenital Heart Disease." Primary Care: Clinics in Office Practice 48, no. 3 (September 2021): 351–66. http://dx.doi.org/10.1016/j.pop.2021.04.005.

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13

Marian, Ali J. "Congenital Heart Disease." Circulation Research 120, no. 6 (March 17, 2017): 895–97. http://dx.doi.org/10.1161/circresaha.117.310830.

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14

HUHTA, JAMES C. "Congenital Heart Disease." Echocardiography 8, no. 4 (July 1991): 439–40. http://dx.doi.org/10.1111/j.1540-8175.1991.tb01005.x.

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15

Ramin, Susan M., Mark C. Maberry, and Larry C. Gilstrap. "Congenital Heart Disease." Clinical Obstetrics and Gynecology 32, no. 1 (March 1989): 41–47. http://dx.doi.org/10.1097/00003081-198903000-00007.

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16

Baker, Cindy. "Congenital Heart Disease." Pediatric Physical Therapy 4, no. 2 (1992): 100. http://dx.doi.org/10.1097/00001577-199200420-00019.

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17

van Dorp, Francoise, and Chantal Simon. "Congenital Heart Disease." InnovAiT: Education and inspiration for general practice 1, no. 1 (January 2008): 34–40. http://dx.doi.org/10.1093/innovait/inm006.

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18

Delphin, E. "Congenital heart disease." Current Opinion in Anaesthesiology 3, no. 1 (February 1990): 14–16. http://dx.doi.org/10.1097/00001503-199002000-00005.

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19

Bruneau, Benoit G., and Deepak Srivastava. "Congenital Heart Disease." Circulation Research 114, no. 4 (February 14, 2014): 598–99. http://dx.doi.org/10.1161/circresaha.113.303060.

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20

Link, Kerry M., Stephen P. Loehr, Eric M. Martin, and Nadja M. Lesko. "Congenital heart disease." Coronary Artery Disease 4, no. 4 (April 1993): 340–44. http://dx.doi.org/10.1097/00019501-199304000-00005.

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21

SOMERVILLE, J. "Congenital heart disease." European Heart Journal 18, no. 3 (March 1, 1997): 533–34. http://dx.doi.org/10.1093/oxfordjournals.eurheartj.a015284.

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22

Ramamoorthy, Chandra. "Congenital heart disease." Pediatric Anesthesia 21, no. 5 (April 12, 2011): 471–72. http://dx.doi.org/10.1111/j.1460-9592.2011.03592.x.

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23

Treasure, Tom. "Congenital heart disease." BMJ 328, no. 7440 (March 11, 2004): 594–95. http://dx.doi.org/10.1136/bmj.328.7440.594.

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24

Burch, Michael. "Congenital heart disease." Medicine 34, no. 7 (July 2006): 274–81. http://dx.doi.org/10.1383/medc.2006.34.7.274.

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25

Burch, Michael. "Congenital Heart Disease." Medicine 30, no. 6 (June 2002): 158–65. http://dx.doi.org/10.1383/medc.30.6.158.28201.

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26

GRAHAM, THOMAS P., J. TIMOTHY BRICKER, FREDERICK W. JAMES, and WILLIAM B. STRONG. "Congenital heart disease." Medicine & Science in Sports & Exercise 26, Supplement (October 1994): S254. http://dx.doi.org/10.1249/00005768-199410001-00006.

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27

Hopkins, Maeve K., Lorraine Dugoff, and Jeffrey A. Kuller. "Congenital Heart Disease." Obstetrical & Gynecological Survey 74, no. 8 (August 2019): 497–503. http://dx.doi.org/10.1097/ogx.0000000000000702.

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28

Watt, Richard H. "Congenital Heart Disease." Lippincott's Case Management 9, no. 4 (2004): 205–8. http://dx.doi.org/10.1097/00129234-200407000-00009.

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29

Burch, Michael. "Congenital heart disease." Medicine 38, no. 10 (October 2010): 561–68. http://dx.doi.org/10.1016/j.mpmed.2010.07.001.

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30

ALDERMAN, LAUREN M. "Congenital heart disease." Nursing 30, no. 5 (May 2000): 41–47. http://dx.doi.org/10.1097/00152193-200030050-00028.

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31

Wilkinson, James L. "Congenital heart disease." International Journal of Cardiology 13, no. 1 (October 1986): 97. http://dx.doi.org/10.1016/0167-5273(86)90090-2.

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32

Dolbec, Katherine, and Nathan W. Mick. "Congenital Heart Disease." Emergency Medicine Clinics of North America 29, no. 4 (November 2011): 811–27. http://dx.doi.org/10.1016/j.emc.2011.08.005.

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33

Kasinskas, Kaitlyn, and Dawn Colomb-Lippa. "Congenital heart disease." Journal of the American Academy of Physician Assistants 27, no. 8 (August 2014): 32–34. http://dx.doi.org/10.1097/01.jaa.0000451416.29847.8c.

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34

Woodard, Pamela K., and Jie Zheng. "Congenital Heart Disease." Current Protocols in Magnetic Resonance Imaging 00, no. 1 (March 2001): A10.1.1—A10.1.19. http://dx.doi.org/10.1002/0471142719.mia1001s00.

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35

Alrasyid, Harun, Suhadi Suhadi, Sutaryo Sutaryo, Rosyid Ridho, A. Samik Wahab, and Ismangoen Ismangoen. "Congenital Heart Disease." Paediatrica Indonesiana 20, no. 7-8 (December 16, 2021): 145–50. http://dx.doi.org/10.14238/pi20.7-8.1980.145-50.

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This paper reports 54 cases of congenital heart disease which were diagnosed by simple methods, without cathetherisation and Angiocardiography. These consisted of : 1). 37% Ventricular septal defect. 2). 24% Atrial Septal defect, second type. 3). 11,11% Patent Ductus Arteriosus Botalli. 4). 9% Tetralogy of Fallot. 5). 7,5% Stenosis pulmonalis. 6). 3,7% Atrial septal defect primary type. 7). 1,85% Aorta stenosis. 8). 2% Others. Early signs and symptoms were: frequent cough, growth retardation, dyspnoe d'effort, Electrocardiographic and Rontgenologic abnormalities. With simple examinations we can almost accurately diagnose congenital heart disease. We suggest to build up more cardiac centres to overcome congenital heart disease problems.
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36

Podzolkov, V. "Congenital heart disease." Patologiya krovoobrashcheniya i kardiokhirurgiya 21, no. 3S (September 22, 2017): 26–27. http://dx.doi.org/10.21688/1681-3472-2017-3s-26-27.

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An iterview with Vladimir P. Podzolkov, Dr. Sci. (Medicine), Prof., Academician, Russian Academy of Sciences, Head of Pediatric Congenital Heart Disease Department, A.N. Bakoulev Scientific Center for Cardiovascular Surgery, Moscow
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37

Shah, Prashant, Kunjang Sherpa, Naveen Kumar Pandey, Bhawani Manandhar, and Sahadeb Prasad Dhungana. "Spectrum of Congenital Heart Diseases in Eastern Nepal: A tertiary care hospital experience." Journal of College of Medical Sciences-Nepal 12, no. 4 (January 19, 2017): 137–42. http://dx.doi.org/10.3126/jcmsn.v12i4.15593.

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Background & Objectives: Congenital heart diseases are neglected especially in world’s poorest nations and appear to be ignored and unexplored dimension of health. The exact prevalence and spectrum of congenital heart diseases in Nepal is largely unknown. The aim of this study was to describe the local experience on the magnitude and the pattern of congenital heart disease in order to increase the awareness of the public and health policy makers on its burden in Nepal.Materials & Methods: This is an observational hospital based study carried out in a tertiary care hospital in Eastern Nepal. The duration of this study was from April 2015 to July 2016. The echocardiography reports of all patients clinically suspected of having congenital heart disease were retrieved, and their diagnostic details were extracted. Only patients of day one of life to 14 years of age were included. Congenital heart diseases like bicuspid aortic valve, mitral valve prolapse and various inherited cardiomyopathies were excluded.Results: A total of 330 echocardiograms were performed for clinically suspected congenital heart disease. The mean age of study population was 22.31±34.08 months with male to female ratio of 1.2:1. 23% of clinically suspected congenital heart disease cases turned out to have normal echocardiography. Acyanotic congenital heart disease was most common (81.5%) followed by cyanotic congenital heart disease (14.2%) and obstructive congenital heart disease (4.3%). Atrial septal defect was found to be the most common form of acyanotic congenital heart disease (52%) which was followed by ventricular septal defect (28.8%) and patent ductus arteriosus (14.8%). Tetralogy of Fallot and double outlet right ventricle were the most common form of cyanotic CHD representing 44.4% of all cyanotic patients. Pulmonary stenosis was the most common obstructive congenital heart disease observed in this study population (63.6%). Rarer entities, like d-transposition of great arteries, congenitally corrected transposition of great arteries, various types of total anomalous pulmonary venous drainage, double inlet left ventricle, interrupted aortic arch, Shone complex, etc. were also observed, however represented only the minority of the study population.Conclusion: The spectrum of congenital heart disease seen in this study very likely and only represents the tip of the iceberg. Public awareness programmes and training of health care personnel needs to be emphasized in order to facilitate its early diagnosis and improve its outcome.
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38

Crossland, David S., Susan P. Jackson, Rosalind Lyall, John Burn, and John J. O'Sullivan. "Employment and advice regarding careers for adults with congenital heart disease." Cardiology in the Young 15, no. 4 (July 13, 2005): 391–95. http://dx.doi.org/10.1017/s104795110500082x.

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Aims: To compare the rates of employment, and advice offered concerning careers, in adults with congenital heart disease and controls. To assess the impact of the severity of the congenital cardiac malformation on the chances for employment. Methods: We solicited responses from 299 adults with congenitally malformed hearts, asking them to answer questions from a questionnaire posed at interview by a trained nurse. The adults were asked to give an identical questionnaire to a friend to act as a control. We received responses from 177 of the controls. Results: The responses showed that 51 of 156 (33 per cent) adults with congenital heart disease were unemployed, and 37 of 151 (25 per cent) had been unemployed for more than a year. This is significantly more than 25 of 156 (16 per cent) matched controls unemployed, and 5 of 151 (3 per cent) controls unemployed for more than a year. Almost one-fifth of the adults with congenital heart disease (19 per cent) had received advice regarding their career which they found helpful, which is significantly fewer than the 31 per cent of controls. More (42 per cent) had been given advice against certain occupations than controls (11 per cent). Receiving career advice was significantly associated with employment in the population with congenitally malformed hearts, with almost three-quarters (73 per cent) of those given advice being employed compared to 46 per cent of those not given advice. This pattern was not seen in controls. The severity of the congenital cardiac malformation did not significantly affect the rates of unemployment. Conclusions: Whatever the severity of their disease, adults with congenitally malformed hearts are more likely to be unemployed than matched controls. They are less likely to receive useful advice regarding potential careers, and find the advice given less helpful, than controls, although receiving suitable advice is associated with being employed in the population with congenital cardiac disease.
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39

O'Brien, Kevin M. "Congenital syndromes with congenital heart disease." Seminars in Roentgenology 20, no. 2 (April 1985): 104–5. http://dx.doi.org/10.1016/0037-198x(85)90059-8.

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40

BAKER, E. "Radionuclide investigation of congenital heart disease." Heart 84, no. 5 (November 1, 2000): 467–68. http://dx.doi.org/10.1136/heart.84.5.467.

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41

Jarvelin, M. R. "CONGENITAL HEART DISEASE: Fetal and infant markers of adult heart diseases." Heart 84, no. 2 (August 1, 2000): 219–26. http://dx.doi.org/10.1136/heart.84.2.219.

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42

Häcker, Anna-Luisa, Barbara Reiner, Renate Oberhoffer, Alfred Hager, Peter Ewert, and Jan Müller. "Increased arterial stiffness in children with congenital heart disease." European Journal of Preventive Cardiology 25, no. 1 (October 23, 2017): 103–9. http://dx.doi.org/10.1177/2047487317737174.

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Objective Central systolic blood pressure (SBP) is a measure of arterial stiffness and strongly associated with atherosclerosis and end-organ damage. It is a stronger predictor of cardiovascular events and all-cause mortality than peripheral SBP. In particular, for children with congenital heart disease, a higher central SBP might impose a greater threat of cardiac damage. The aim of the study was to analyse and compare central SBP in children with congenital heart disease and in healthy counterparts. Patients and methods Central SBP was measured using an oscillometric method in 417 children (38.9% girls, 13.0 ± 3.2 years) with various congenital heart diseases between July 2014 and February 2017. The test results were compared with a recent healthy reference cohort of 1466 children (49.5% girls, 12.9 ± 2.5 years). Results After correction for several covariates in a general linear model, central SBP of children with congenital heart disease was significantly increased (congenital heart disease: 102.1 ± 10.2 vs. healthy reference cohort: 100.4 ± 8.6, p < .001). The analysis of congenital heart disease subgroups revealed higher central SBP in children with left heart obstructions (mean difference: 3.6 mmHg, p < .001), transpositions of the great arteries after arterial switch (mean difference: 2.2 mmHg, p = .017) and univentricular hearts after total cavopulmonary connection (mean difference: 2.1 mmHg, p = .015) compared with the reference. Conclusion Children with congenital heart disease have significantly higher central SBP compared with healthy peers, predisposing them to premature heart failure. Screening and long-term observations of central SBP in children with congenital heart disease seems warranted in order to evaluate the need for treatment.
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43

Brida, Margarita, and Michael A. Gatzoulis. "Adult Congenital Heart Disease." JACC: Case Reports 3, no. 2 (February 2021): 353–55. http://dx.doi.org/10.1016/j.jaccas.2021.01.001.

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44

NISHIBE, Shinichi. "Adult Congenital Heart Disease." JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA 37, no. 1 (2017): 41–48. http://dx.doi.org/10.2199/jjsca.37.41.

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45

TOMIMATSU, Hirofumi. "Adult congenital heart disease." Choonpa Igaku 42, no. 4 (2015): 445–56. http://dx.doi.org/10.3179/jjmu.jjmu.r.51.

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46

Weale, Jonathan, and Andrea A. Kelleher. "Adult congenital heart disease." Anaesthesia & Intensive Care Medicine 22, no. 5 (May 2021): 290–96. http://dx.doi.org/10.1016/j.mpaic.2021.03.003.

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47

Sparacino, Patricia S. A. "ADULT CONGENITAL HEART DISEASE." Nursing Clinics of North America 29, no. 2 (June 1994): 213–19. http://dx.doi.org/10.1016/s0029-6465(22)02728-1.

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48

Strozzi, Maja. "Adult Congenital Heart Disease." Cardiologia Croatica 11, no. 1-2 (February 22, 2016): 3–4. http://dx.doi.org/10.15836/ccar2016.3.

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49

Tsuda, Takeshi. "Cyanotic Congenital Heart Disease." Journal of Heart and Cardiology 2, no. 1 (2016): 1–5. http://dx.doi.org/10.15436/2378-6914.16.014.

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50

Scott, Craig H. "Adult Congenital Heart Disease." Annals of Internal Medicine 129, no. 12 (December 15, 1998): 1084. http://dx.doi.org/10.7326/0003-4819-129-12-199812150-00045.

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